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Lumbar Disc Herniation

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Lumbar Disc Herniation

Comprehensive guide to lumbar disc herniation - epidemiology, disc anatomy, herniation types, radiculopathy, conservative vs surgical management, microdiscectomy technique, and recurrence prevention

complete
Updated: 2024-12-17
High Yield Overview

LUMBAR DISC HERNIATION

Radicular Pain | Straight Leg Raise | Microdiscectomy for Failed Conservative Care

5-10%Adults experience sciatica in lifetime
90%Resolve with conservative care at 6 weeks
L4-L5/L5-S1Most common levels (95%)
5-10%Recurrence rate after microdiscectomy

HERNIATION TYPES (NASS NOMENCLATURE)

Protrusion
PatternBase wider than dome, contained
TreatmentConservative
Extrusion
PatternBase narrower than dome, may migrate
TreatmentConsider surgery if symptomatic
Sequestration
PatternFragment separated from disc
TreatmentOften surgical

Critical Must-Knows

  • Radicular pain (leg worse than back) is hallmark - back pain alone is NOT disc herniation
  • Straight leg raise (SLR) positive if reproduces radicular pain below knee at less than 60 degrees
  • MRI confirms clinical diagnosis - never operate on imaging alone without matching symptoms
  • Conservative management first - 90% improve by 6 weeks without surgery
  • Surgery indicated for: cauda equina, progressive motor deficit, or failed 6 weeks conservative care
  • Microdiscectomy gold standard - remove herniated fragment, preserve disc space, early mobilization

Examiner's Pearls

  • "
    SLR positive at less than 60 degrees with radicular pain = disc herniation until proven otherwise
  • "
    Crossed SLR (raising opposite leg reproduces ipsilateral leg pain) is highly specific
  • "
    L5-S1 disc herniation affects S1 nerve root (ankle jerk, plantar flexion, lateral foot numbness)
  • "
    Cauda equina syndrome = surgical emergency - saddle anesthesia, bowel/bladder dysfunction, bilateral symptoms
  • "
    SPORT trial: surgery provides faster relief but similar 4-year outcomes to conservative care

Clinical Imaging

Imaging Gallery

A 26-year-old female presented with chronic low back pain and left sciatica. Her ability to walk was significantly decreased. Physical examination revealed positive straight leg rising and weak big to
Click to expand
A 26-year-old female presented with chronic low back pain and left sciatica. Her ability to walk was significantly decreased. Physical examination revCredit: Open-i / NIH via Open-i (NIH) (Open Access (CC BY))
(A, B) Axial magnetic resonance imaging scan showing L5-S1 disc herniation in a patient and L5 sciatica.
Click to expand
(A, B) Axial magnetic resonance imaging scan showing L5-S1 disc herniation in a patient and L5 sciatica.Credit: Mostofi K et al. via Asian Spine J via Open-i (NIH) (Open Access (CC BY))
Axial magnetic resonance imaging scan showing L4-5 disc herniation in a patient with S1 sciatica.
Click to expand
Axial magnetic resonance imaging scan showing L4-5 disc herniation in a patient with S1 sciatica.Credit: Mostofi K et al. via Asian Spine J via Open-i (NIH) (Open Access (CC BY))
On the right side herniated disc (black arrow) and contralateral hypertrophic ligamentum flavum (white arrow) are shown on the T1-weighted axial MRI image
Click to expand
On the right side herniated disc (black arrow) and contralateral hypertrophic ligamentum flavum (white arrow) are shown on the T1-weighted axial MRI iCredit: Karabekir HS et al. via Arch Med Sci via Open-i (NIH) (Open Access (CC BY))
Sagittal T2-weighted MRI showing severe L4-L5 disc herniation
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Lumbar disc herniation: Sagittal T2-weighted MRI demonstrating a severe L4-L5 disc herniation with posterior displacement of disc material into the spinal canal. Note the loss of disc height and compression of neural elements.Credit: Jay Moore - Wikimedia Commons (CC0 Public Domain)

Critical Lumbar Disc Herniation Exam Points

Radicular Pain Dominates

Leg pain worse than back pain is the key feature. Pure back pain without radiculopathy is NOT a disc herniation presentation. Sciatica follows dermatomal distribution below the knee.

Clinical-Radiological Correlation

MRI confirms clinical diagnosis. Never operate based on imaging alone. Up to 30% of asymptomatic adults have disc bulges on MRI. Match imaging level to clinical examination findings.

Conservative Care First

90% improve with conservative management within 6 weeks. Surgery is for failed conservative care, progressive motor deficit, or cauda equina syndrome. Natural history favors non-operative treatment.

Cauda Equina is an Emergency

Saddle anesthesia, bowel/bladder dysfunction, bilateral leg symptoms = cauda equina syndrome. Requires urgent MRI and emergency decompression within 24-48 hours to prevent permanent dysfunction.

Quick Decision Guide - Management Algorithm

PresentationClinical FeaturesManagementKey Pearl
Acute radiculopathy, no red flagsLeg pain > back pain, positive SLR, intact motor/reflexesConservative: NSAIDs, physio, nerve root injection90% resolve by 6 weeks - reassure patient
Persistent radiculopathy 6 weeksFailed conservative care, impacting functionMRI, consider microdiscectomySurgery for quality of life, not cure
Progressive motor deficitWorsening foot drop, EHL weakness grade 3 or lessUrgent MRI, early surgery (within 2 weeks)Footdrop may not fully recover if delayed
Cauda equina syndromeSaddle anesthesia, bowel/bladder dysfunction, bilateralEmergency MRI, immediate decompressionOperate within 24-48h to prevent permanent deficit
Mnemonic

DISCDISC - Herniation Characteristics

D
Dermatomal distribution
Radicular pain follows nerve root pattern
I
Imaging confirmation
MRI shows herniation matching clinical level
S
Straight leg raise positive
Reproduces leg pain at less than 60 degrees
C
Conservative care first
90% improve without surgery by 6 weeks

Memory Hook:DISC reminds you that herniation causes radicular pain confirmed by imaging and SLR

Mnemonic

SCIATICASCIATICA - Red Flags for Surgery

S
Saddle anesthesia
Cauda equina syndrome - emergency
C
Cauda equina symptoms
Bowel/bladder dysfunction
I
Intolerable pain
Failed 6 weeks conservative care
A
Acute motor deficit
Progressive foot drop, EHL grade 3 or less
T
Timing critical
CES needs surgery within 24-48 hours
I
Imaging matches symptoms
MRI level correlates with clinical findings
C
Conservative failed
6 weeks physio, NSAIDs, injections tried
A
Affecting function
Unable to work, significant disability

Memory Hook:SCIATICA reminds you when surgery is indicated for disc herniation

Mnemonic

ROOTSROOTS - Nerve Root Examination

R
Reflex testing
Knee jerk (L4), ankle jerk (S1)
O
Observation for atrophy
Calf wasting in chronic S1 radiculopathy
O
Opposition strength
EHL (L5), plantar flexion (S1)
T
Touch sensation
Dermatomal patterns - dorsum foot (L5), lateral foot (S1)
S
Straight leg raise
SLR positive if less than 60 degrees reproduces leg pain

Memory Hook:Test all ROOTS components to identify which nerve root is compressed

Mnemonic

MICROMICRO - Microdiscectomy Technique

M
Midline incision
2-3cm centered over interspace
I
Interlaminar window
Remove ligamentum flavum, expose disc
C
Careful nerve retraction
Protect nerve root and dura during fragment removal
R
Remove fragment only
Do not excise entire disc - preserve disc space
O
Out same day/next day
Early mobilization, discharge within 24 hours

Memory Hook:MICRO technique is minimally invasive with focus on fragment removal

Overview and Epidemiology

Lumbar disc herniation is the most common cause of sciatica (radicular leg pain). The intervertebral disc herniates posterolaterally, compressing the exiting nerve root in the lateral recess or foramen.

Natural history:

  • Spontaneous resolution is the norm - herniated disc fragments resorb over time
  • 90% of patients improve with conservative care within 6 weeks
  • Surgical intervention is reserved for specific indications, NOT a primary treatment
  • Large sequestered fragments paradoxically have better prognosis (more inflammatory resorption)

Why Disc Herniation Resolves

Disc fragments undergo inflammatory resorption. Extruded and sequestered fragments (exposed to epidural space) have more contact with blood supply and macrophages, leading to faster resorption than contained protrusions. This explains why large herniations may resolve faster than small ones.

Epidemiology:

  • Peak incidence: 30-50 years (disc still has nucleus pulposus hydration)
  • Male to female: 2:1 ratio
  • Risk factors: Heavy lifting, vibration exposure, smoking, obesity, genetics
  • Level distribution: L4-L5 (45%), L5-S1 (50%), L3-L4 (5%), higher levels rare

Pathophysiology and Mechanisms

Anatomical illustration of intervertebral disc structure showing nucleus pulposus, annulus fibrosus, and vertebral endplates
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Intervertebral disc anatomy: (A) Cross-sectional view showing the central nucleus pulposus, surrounding annulus fibrosus, and vertebral endplates. The disc measures approximately 4 cm wide and 7-10 mm thick. (B) Detailed structure of the annulus fibrosus with its 15-25 lamellae comprised of collagen fiber bundles oriented at alternating angles to resist rotational forces.Credit: Doblado LR et al. Materials (Basel) 2019 - CC-BY 4.0 (PMC6356370)

Intervertebral disc structure:

Nucleus Pulposus

  • Composition: 80% water, proteoglycans (aggrecan), type II collagen
  • Function: Absorbs compressive loads, distributes forces
  • Age changes: Dehydration, loss of proteoglycans, decreased height

Annulus Fibrosus

  • Composition: Type I collagen, organized lamellae, 15-20 layers
  • Function: Contains nucleus, resists tensile forces
  • Weak zone: Posterolateral annulus (thinnest, lacks support from ALL)

Why posterolateral herniation?

  • Anterior longitudinal ligament (ALL): Strong, thick, prevents anterior herniation
  • Posterior longitudinal ligament (PLL): Weak centrally, strong midline, narrow laterally
  • Posterolateral annulus: Thinnest region, no PLL reinforcement
  • Result: 95% of herniations occur posterolaterally into lateral recess

Nerve Root Numbering

Lumbar nerve roots exit BELOW their corresponding pedicle. L4-L5 disc herniation compresses the L5 nerve root (traversing root in lateral recess), NOT L4. The L4 root has already exited above. Far lateral herniations compress the exiting root (L4 at L4-L5 level).

Nerve root anatomy:

  • Traversing root: Crosses disc space in lateral recess (e.g., L5 root at L4-L5 level)
  • Exiting root: Exits at same level foramen (e.g., L4 root at L4-L5 level, far lateral)
  • Dural sac: Contains cauda equina, ends at S1-S2 level
  • Conus medullaris: Spinal cord ends at L1-L2 level

Disc herniation zones:

  1. Central: Midline, may compress multiple roots or cauda equina
  2. Paracentral: Most common, compresses traversing root
  3. Foraminal: Within foramen, compresses exiting root
  4. Far lateral (extraforaminal): Lateral to foramen, compresses exiting root

Classification Systems

Diagram showing the four stages of disc herniation: degeneration, prolapse, extrusion, and sequestration
Click to expand
Disc herniation classification: Progressive stages from (1) Degeneration - initial disc weakening with annular fissures, (2) Prolapse/Protrusion - nucleus bulges but remains contained by outer annulus, (3) Extrusion - nucleus breaches the outer annulus fibrosus while maintaining continuity, (4) Sequestration - disc fragment separates completely and may migrate within the spinal canal.Credit: Servier Medical Art - Wikimedia Commons (CC-BY-SA 3.0)

North American Spine Society (NASS) Classification

TypeDefinitionPrognosisTreatment
NormalNo disc extension beyond vertebral bodyNot pathologicalNone
BulgeCircumferential extension, not focalUsually asymptomaticConservative
ProtrusionFocal herniation, base wider than dome, contained by annulusMay resolve spontaneouslyConservative first
ExtrusionFocal herniation, base narrower than dome, through annulusMay migrate, variable resolutionConsider surgery if symptomatic
SequestrationFragment separated from parent disc, free in canalGood prognosis for resorptionOften resolves, surgery if symptomatic

Sequestered Fragments

Sequestered fragments have BETTER prognosis than contained herniations. They have more contact with vascular tissue and macrophages in the epidural space, leading to faster inflammatory resorption. Large sequestrations often resolve without surgery.

Classification by Location (Axial Plane)

ZoneDescriptionRoot AffectedApproach
CentralMidline canalMultiple or cauda equinaLaminectomy if massive
ParacentralLateral recessTraversing root (e.g., L5 at L4-L5)Standard microdiscectomy
ForaminalWithin foramenExiting root (e.g., L4 at L4-L5)Foraminotomy
Far LateralLateral to foramenExiting rootFar lateral approach, may need facetectomy
MRI of L4-L5 paracentral disc herniation
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Three-panel MRI demonstrating classic L4-L5 paracentral disc herniation. Panels A-B: Sagittal T2-weighted images showing posterior disc protrusion narrowing the spinal canal (note decreased CSF signal). Panel C: Axial T2-weighted image at L4-L5 showing left-sided paracentral disc herniation compressing the thecal sac and left L5 traversing nerve root in the lateral recess. This is the most common herniation pattern requiring surgical intervention.Credit: Tarukado K et al., Asian Spine J - CC BY 4.0
MRI of foraminal disc herniation at L5-S1
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Two-panel MRI demonstrating foraminal disc herniation at L5-S1. Panel (a): Axial T2-weighted image showing disc material occupying the left neural foramen (foraminal herniation) - note the loss of normal bright fat signal surrounding the exiting L5 nerve root. Panel (b): Sagittal T2-weighted image demonstrating L5-S1 foraminal stenosis from extruded disc. Foraminal herniations compress the exiting root (L5 at L5-S1) rather than the traversing root, requiring a different surgical approach (foraminotomy).Credit: Open-i (NIH) - CC BY 4.0

Classification by Migration (Sagittal Plane)

Extruded and sequestered fragments may migrate:

DirectionDescriptionClinical Significance
No migrationFragment at disc space levelStandard approach
Cranial migrationFragment migrates upwardHidden behind superior lamina
Caudal migrationFragment migrates downward (most common)Hidden behind inferior lamina
Extreme migrationFragment far from disc spaceMay require extended exposure

Surgical implication: Migrated fragments may require more extensive decompression or approach modification.

Clinical Presentation and Assessment

Cardinal features of lumbar disc herniation:

History

  • Chief complaint: Leg pain worse than back pain (radiculopathy)
  • Distribution: Dermatomal pattern below knee
  • Character: Sharp, shooting, electric, burning
  • Aggravating factors: Sitting, bending forward, Valsalva (cough, sneeze)
  • Relieving factors: Standing, lying, walking
  • Red flags: Saddle anesthesia, bowel/bladder changes, bilateral symptoms, progressive weakness

Examination

  • Gait: Antalgic, footdrop gait if L5 weakness
  • Inspection: Scoliosis (sciatic scoliosis - away from side of pain)
  • Range of motion: Decreased lumbar flexion
  • Straight leg raise: Positive if less than 60 degrees reproduces radicular pain
  • Neurological exam: Motor, sensory, reflexes by dermatome/myotome

Straight leg raise (SLR) test:

  • Technique: Patient supine, examiner lifts extended leg
  • Positive test: Reproduces radicular leg pain (NOT back pain) at less than 60 degrees elevation
  • Sensitivity: 90% for disc herniation
  • Specificity: 25% (many false positives)
  • Crossed SLR: Raising opposite leg reproduces ipsilateral leg pain - highly specific (95%) for disc herniation

SLR Interpretation

SLR is positive only if it reproduces LEG PAIN below the knee, not back pain or hamstring tightness. Pain at greater than 60 degrees is often hamstring or sacroiliac joint pain. Crossed SLR (raising contralateral leg causes ipsilateral sciatica) is highly specific for disc herniation.

MRI comparison showing mild disc bulge versus full lumbar disc herniation with spinal stenosis
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Lumbar disc herniation on MRI: Comparison of (A) mild disc bulge without severe neural impingement, and (B) full lumbar disc herniation with substantial spinal stenosis and nerve root compression. The severe herniation demonstrates the posterolateral location typical of most disc herniations, with significant narrowing of the spinal canal.Credit: Doblado LR et al. Materials (Basel) 2019 - CC-BY 4.0 (PMC6356370)

Neurological examination by level:

Lumbar Nerve Root Examination

LevelMotorSensationReflexSLR
L4 (L3-L4 disc)Quadriceps, knee extensionMedial leg, medial ankleKnee jerk (patellar)Femoral stretch test
L5 (L4-L5 disc)EHL, ankle dorsiflexion, foot eversionDorsum of foot, first web spaceNone (or medial hamstring)Positive SLR
S1 (L5-S1 disc)Ankle plantar flexion, toe plantar flexionLateral foot, sole of footAnkle jerk (Achilles)Positive SLR

Red flag symptoms requiring urgent assessment:

Cauda Equina Syndrome

Cauda equina syndrome is a surgical emergency:

  • Saddle anesthesia (perianal and perineal numbness)
  • Bowel dysfunction (incontinence or retention)
  • Bladder dysfunction (urinary retention, overflow incontinence, loss of sensation)
  • Bilateral leg symptoms (weakness or numbness)
  • Sexual dysfunction (erectile dysfunction, loss of sensation)

Management: Urgent MRI, emergency decompression within 24-48 hours. Delayed surgery results in permanent bowel/bladder dysfunction.

MRI of large central disc herniation causing cauda equina compression
Click to expand
Two-panel MRI demonstrating a large central disc herniation at L5-S1 causing significant cauda equina compression. Left: Sagittal T2-weighted image showing multi-level degenerative changes with a large posterior disc protrusion at L5-S1. Right: Axial T2-weighted image at L5-S1 showing a massive central disc herniation severely compressing the thecal sac and cauda equina (note almost complete obliteration of CSF signal). This imaging pattern correlates with cauda equina syndrome requiring emergency surgical decompression.Credit: Akca N et al., J Craniovertebr Junction Spine - CC BY 4.0

Investigations

Imaging Protocol

Acute (0-6 weeks)Clinical Diagnosis

No imaging needed initially for typical radiculopathy without red flags. Natural history favors spontaneous resolution. Treat conservatively and observe.

If Red FlagsUrgent MRI

Immediate MRI for:

  • Cauda equina syndrome
  • Progressive motor deficit
  • Bilateral symptoms
  • Severe or rapidly worsening symptoms
After 6 WeeksMRI for Persistent Symptoms

MRI lumbar spine if:

  • Symptoms persist despite 6 weeks conservative care
  • Considering surgical intervention
  • Confirm level and type of herniation
AlternativeCT Myelography

CT myelogram if MRI contraindicated (pacemaker, claustrophobia). Shows nerve root compression and disc herniation. Less detail than MRI for soft tissues.

MRI findings:

T1-Weighted Images

  • Normal disc: Intermediate signal
  • Degenerated disc: Decreased signal
  • Herniation: Intermediate signal, displaces thecal sac or nerve root
  • Use: Anatomy, foraminal stenosis, bone marrow changes

T2-Weighted Images

  • Normal disc: Bright signal (high water content)
  • Degenerated disc: Dark signal (loss of water)
  • Herniation: Intermediate signal, may enhance with contrast
  • Use: Disc hydration, nerve root compression, CSF bright

MRI and Clinical Correlation

Up to 30% of asymptomatic adults have disc bulges or herniations on MRI. NEVER operate based on imaging alone. The herniation level MUST correlate with clinical examination findings (dermatomal pain, motor weakness, reflex changes). Mismatch between imaging and exam suggests alternate diagnosis.

When to consider EMG/NCS:

  • Atypical presentations: Symptoms not matching expected dermatomal pattern
  • Peripheral neuropathy suspected: Diabetes, alcoholism
  • Timing neurological deficit: Distinguish acute (2-3 weeks for denervation changes) vs chronic
  • Medicolegal: Document baseline function before surgery

Management Algorithm

📊 Management Algorithm
Lumbar Disc Herniation Management Algorithm
Click to expand
Management algorithm for lumbar disc herniation. Conservative care (6 weeks), then MRI and Specialist Referral if failed or red flags (Cauda Equina or Motor Deficit).Credit: OrthoVellum

First-line treatment for 6 weeks (unless red flags)

Goal: Symptom relief, facilitate natural history of disc resorption

Conservative Protocol

Acute PhaseWeek 0-2
  • Activity modification: Avoid prolonged sitting, heavy lifting, bending
  • Analgesia: NSAIDs (if no contraindications), paracetamol, neuropathic agents (gabapentin, pregabalin)
  • Short rest: 1-2 days if severe, then encourage mobilization
  • Education: Reassure natural resolution, avoid bed rest
Subacute PhaseWeek 2-6
  • Physiotherapy: Core strengthening, posture education, nerve glides
  • Activity progression: Gradual return to activities as tolerated
  • Nerve root injection: Consider if symptoms persist, diagnostic and therapeutic
  • Review progress: If improving, continue conservative care
Persistent PhaseWeek 6+
  • Reassess: MRI if not already done
  • Multidisciplinary input: Pain clinic, psychology, functional restoration
  • Consider surgery: If symptoms persist and impacting function/quality of life

Natural History Wins

90% of disc herniations improve with conservative care by 6 weeks. The SPORT trial showed that while surgery provides faster pain relief, outcomes at 4 years are similar between surgical and conservative groups. Surgery is for quality of life and faster return to function, NOT a cure.

When surgery is indicated:

IndicationUrgencyRationale
Cauda equina syndromeEmergency (within 24-48h)Prevent permanent bowel/bladder dysfunction
Progressive motor deficitUrgent (within 2 weeks)Prevent permanent weakness, footdrop may not recover
Failed conservative care (6 weeks)ElectiveQuality of life, faster return to function
Intolerable painElectiveSeverely impacting function despite maximal medical management

Contraindications to surgery:

  • Predominant back pain: Not a disc herniation indication
  • Non-concordant imaging: MRI does not match clinical level
  • Psychosocial issues: Secondary gain, litigation, poor coping
  • Medical comorbidities: ASA 4 or above, prohibitive surgical risk

Surgery is NOT for Back Pain

Microdiscectomy treats radicular leg pain, NOT back pain. Patients with predominant axial back pain are poor surgical candidates. If back pain is the main complaint, consider alternate diagnoses (facet arthropathy, sacroiliac joint, discogenic pain).

Nerve root injection (transforaminal epidural steroid injection):

  • Indication: Persistent radiculopathy, diagnostic uncertainty
  • Technique: Fluoroscopy-guided, contrast confirmation, corticosteroid + local anesthetic
  • Success rate: 50-70% pain relief, often temporary
  • Role: Bridge to spontaneous resolution, avoid surgery in some cases

Other interventions:

  • Epidural steroid injection (interlaminar): Less targeted than transforaminal
  • Caudal epidural: Useful for S1 radiculopathy
  • Discography: Controversial, not routinely used for disc herniation

These interventional options can provide temporary relief and may help avoid surgery in selected patients.

Surgical Technique - Microdiscectomy

Pre-operative Checklist

Consent Points

  • Recurrence: 5-10% risk of recurrent herniation
  • Dural tear: 1-2% risk, usually repaired primarily
  • Nerve root injury: Less than 1%, may cause new deficit
  • Infection: 1-2% superficial, less than 1% deep (discitis)
  • No guarantee: Pain may persist, surgery does not prevent future disc disease
  • Cauda equina: Rare postoperative complication from hematoma

Equipment Checklist

  • Microscope or loupes: Magnification for nerve root visualization
  • Fluoroscopy: Confirm correct level intraoperatively
  • Retractors: Self-retaining (e.g., Taylor, Caspar) for exposure
  • Curets and pituitary rongeurs: Fragment removal
  • Bipolar cautery: Hemostasis around nerve root
  • Nerve root retractor: Protect nerve during fragment retrieval

Level localization:

  • L4-L5: Most common level - identify iliac crest (L4-L5 level)
  • L5-S1: Lumbosacral junction - palpate for sacral prominence
  • Fluoroscopy mandatory: Confirm level before incision (wrong level surgery is never event)

Accurate level confirmation prevents wrong-site surgery, which is a never event.

Patient Positioning

Setup Checklist

Step 1Position

Prone position on Wilson frame or kneeling frame.

  • Hips flexed: Opens interlaminar space
  • Abdomen free: Minimizes epidural venous pressure
  • Pressure points padded: Eyes, chest, knees, toes
Step 2Fluoroscopy
  • AP view: Confirm spinous processes midline
  • Lateral view: Identify disc space and level
  • Mark skin: Indicate incision site over interspace
Step 3Draping
  • Prepare widely: Allow for extension if needed
  • Fluoroscopy draping: Access for intraoperative imaging

Surgical Approach: Posterior Midline/Paramedian

Incision: 2-3cm midline or paramedian incision centered over interspace

Step-by-Step Approach

Step 1Skin and Fascia
  • Midline skin incision
  • Incise fascia just off midline (ipsilateral to herniation)
  • Minimizes contralateral muscle dissection
Step 2Muscle Dissection
  • Subperiosteal dissection along lamina
  • Preserve midline interspinous ligament if possible
  • Retract paraspinal muscles laterally with self-retaining retractor
Step 3Bony Landmarks
  • Identify: Inferior edge of superior lamina, superior edge of inferior lamina
  • Interlaminar window: Space between laminae
  • Confirm level: Fluoroscopy if any doubt

Microdiscectomy Decompression Steps

Decompression Technique

Step 1Ligamentum Flavum Removal
  • Identify yellow ligament spanning interlaminar window
  • Incise with scalpel or Kerrison rongeur
  • Remove carefully - epidural fat/nerve beneath
  • Expose: Dura and nerve root now visible
Step 2Nerve Root Identification
  • Locate traversing nerve root (e.g., L5 root at L4-L5 level)
  • Assess degree of compression - root may be displaced or tense
  • Protect with cottonoid patty or nerve root retractor
Step 3Annulotomy
  • Retract nerve root gently medially with blunt retractor
  • Identify herniated disc fragment (blue-white, tense)
  • Incise annulus with scalpel (cruciate or H-incision)
  • Deliver extruded fragment with pituitary rongeur
Step 4Disc Space Exploration
  • Remove loose fragments from disc space
  • Do NOT perform aggressive discectomy - removes only loose material
  • Preserve disc height - do not remove entire nucleus
  • Inspect for free fragments in epidural space
Step 5Nerve Root Decompression Confirmation
  • Assess mobility: Nerve root should move freely
  • Check axilla: Ensure no residual fragment compressing root
  • Hemostasis: Bipolar cautery for epidural bleeding

Nerve Root Protection

Never pull on the nerve root. Use gentle retraction with blunt instruments. Excessive traction can cause permanent neurological injury. If the nerve root does not mobilize easily, look for residual disc fragments or far lateral compression.

Closure

Closure Steps

Step 1Hemostasis
  • Ensure dry field: Bipolar cautery for bleeding
  • Valsalva maneuver: Check for dural tear (CSF leak)
  • Dural repair: If tear identified, repair with 5-0 or 6-0 suture
Step 2Fascial Closure
  • Layers: Fascia closed with absorbable suture (e.g., Vicryl 0 or 1)
  • Water-tight: Prevents CSF leak if occult dural tear
Step 3Skin
  • Subcuticular closure: Absorbable suture (Monocryl 3-0 or 4-0)
  • Dressing: Simple dressing, no drain needed in most cases

Drain decision:

  • Usually NOT needed: Routine microdiscectomy does not require drain
  • Consider drain: If significant epidural bleeding, large dead space, revision surgery
  • Removal: Within 24 hours to minimize infection risk

Most microdiscectomies do not require postoperative drainage.

Surgical Pearls and Pitfalls

Do's (Pearls)

  • Confirm level with fluoroscopy before incision
  • Gentle nerve retraction - never pull forcefully
  • Remove fragment only - do not perform complete discectomy
  • Check for migrated fragments - cranial and caudal exploration
  • Test nerve mobility before closure
  • Valsalva to check for dural tear before closing

Don'ts (Pitfalls)

  • Wrong level surgery - always confirm with imaging
  • Excessive nerve retraction - can cause permanent injury
  • Aggressive discectomy - accelerates adjacent segment disease
  • Missing far lateral fragment - causes persistent symptoms
  • Incomplete decompression - axilla is a common blind spot
  • Unrecognized dural tear - may cause CSF leak, meningitis

Avoiding Recurrence

Balance fragment removal with disc preservation. Aggressive discectomy (removing all nucleus pulposus) does NOT reduce recurrence rate and may accelerate disc degeneration. Remove loose fragments only, preserve disc height and annulus integrity.

Complications

Microdiscectomy Complications

ComplicationIncidencePrevention/Management
Recurrent herniation5-10%Preserve disc height, patient education (avoid heavy lifting), smoking cessation
Dural tear1-5%Primary repair if identified, bed rest if unrecognized, reoperation if CSF leak persists
Nerve root injuryLess than 1%Gentle retraction, avoid excessive traction, microsurgical technique
Infection (superficial)1-2%Perioperative antibiotics, sterile technique, early debridement if deep
Discitis0.5-1%Prolonged antibiotics (6-12 weeks), rest, rarely requires debridement
Wrong level surgeryLess than 1%Fluoroscopy confirmation, preoperative imaging review
Epidural hematomaLess than 1%Meticulous hemostasis, early recognition (cauda equina symptoms), emergency evacuation
Persistent pain10-20%Realistic expectations, not all pain resolves, consider other pain sources

Recurrent disc herniation:

  • Definition: Recurrence of radiculopathy after initial improvement
  • Timing: Can occur months to years after surgery
  • Same level: Most common (80%)
  • Risk factors: Smoking, obesity, heavy lifting, young age, large annular defect
  • Management: Conservative first (as per primary herniation), revision discectomy if failed conservative care

Postoperative Cauda Equina

Epidural hematoma can cause acute postoperative cauda equina syndrome. Suspect if patient develops:

  • New onset bowel/bladder dysfunction
  • Bilateral leg weakness
  • Saddle anesthesia

Requires immediate MRI and emergency reoperation for hematoma evacuation. Time-sensitive - operate within hours.

Dural tear management:

  • Intraoperative recognition: Repair primarily with 5-0 or 6-0 suture, water-tight fascial closure
  • Small tear: May seal with fascial closure and bed rest
  • Postoperative CSF leak: Persistent headache, wound drainage - reoperation for repair if conservative measures fail
  • Consequences: Meningitis risk, CSF fistula, pseudomeningocele

Postoperative Care and Rehabilitation

Postoperative Protocol

ImmediateDay 0-1
  • Mobilization: Get out of bed same day, walk within hours
  • Analgesia: Simple analgesia (paracetamol, NSAIDs), minimize opioids
  • Wound care: Keep dressing clean and dry
  • Discharge: Usually next day (day case in some centers)
EarlyWeek 1-2
  • Activity: Light activities, short walks, avoid prolonged sitting
  • Driving: After 2 weeks (when safe to perform emergency stop)
  • Work: Desk work after 2 weeks, modify heavy work
  • No restrictions: On bending, twisting, lifting light objects
ProgressiveWeek 2-6
  • Physiotherapy: Core strengthening, posture education
  • Activity progression: Gradual return to normal activities
  • Avoid: Heavy lifting (greater than 10kg), repetitive bending
  • Return to work: Most patients by 6 weeks
RecoveryWeek 6-12
  • Full activities: Most activities unrestricted by 3 months
  • Sports: Contact sports after 3 months
  • Lifting: Gradual progression, proper technique education
OngoingLong-term
  • Lifestyle modification: Smoking cessation, weight loss, core strengthening
  • Recurrence prevention: Avoid prolonged sitting, heavy lifting
  • Follow-up: Discharge at 6 weeks if no concerns

Key rehabilitation principles:

  • Early mobilization: Reduces stiffness, improves outcomes
  • No prolonged bed rest: Bed rest delays recovery
  • Core strengthening: Protects spine from recurrence
  • Patient education: Proper lifting technique, ergonomics, weight management

Postoperative Expectations

Leg pain improves immediately, back pain may persist. Microdiscectomy decompresses the nerve root (treats radiculopathy), but does not address disc degeneration or back pain. Set realistic expectations: surgery is for leg pain relief, NOT back pain cure.

Outcomes and Prognosis

Outcomes with microdiscectomy:

Outcome MeasureResult
Leg pain relief85-90% good to excellent at 1 year
Back painVariable, often persists (not primary indication)
Return to work80-95% by 3 months (depends on occupation)
Patient satisfaction80-90% satisfied at 1 year
Recurrence5-10% within 2 years

Predictors of good outcome:

  • Leg pain dominant: Radiculopathy worse than back pain
  • Positive SLR: Clear nerve root tension sign
  • MRI-clinical correlation: Imaging matches examination
  • Acute symptoms: Less than 6 months duration
  • Large sequestration: Paradoxically better outcomes (fragment removable)
  • First episode: Not revision surgery

Predictors of poor outcome:

  • Predominant back pain: Not a microdiscectomy indication
  • Chronic symptoms: Greater than 12 months duration
  • Psychosocial factors: Secondary gain, litigation, depression
  • Multiple level disease: Adjacent segment pathology
  • Smoking: Impairs healing, increases recurrence

SPORT Trial Key Findings

The SPORT trial (Spine Patient Outcomes Research Trial) showed:

  • Surgery provides faster pain relief than conservative care
  • At 4 years, outcomes are similar between surgical and conservative groups
  • Crossover was common: Many conservative patients eventually had surgery
  • Conclusion: Surgery is for quality of life and faster recovery, NOT superior long-term outcomes

Surgery accelerates what would happen naturally in most cases.

Evidence Base and Key Trials

SPORT Trial (Spine Patient Outcomes Research Trial)

1
Weinstein JN et al • JAMA (2006)
Key Findings:
  • Randomized trial comparing surgery (microdiscectomy) vs conservative care for lumbar disc herniation
  • Surgery group had greater improvement in pain and function at 3 months
  • By 4 years, outcomes were similar between groups (high crossover rate)
  • 50% of conservative group eventually had surgery
  • Surgery provides faster relief but not superior long-term outcomes
Clinical Implication: Surgery is indicated for faster pain relief and return to function. Long-term outcomes similar to conservative care due to natural history of disc resorption.
Limitation: High crossover rate (50% of conservative patients had surgery) makes true comparison difficult. Reflects real-world practice.

Cochrane Review: Surgery for Lumbar Disc Prolapse

1
Jacobs WC et al • Cochrane Database Syst Rev (2011)
Key Findings:
  • Meta-analysis of surgical vs conservative treatment for sciatica due to lumbar disc herniation
  • Surgery provides faster pain relief than conservative care
  • Differences diminish over time (1-2 years)
  • No difference in long-term outcomes (greater than 2 years)
  • Surgery appropriate for persistent disabling symptoms
Clinical Implication: Surgery accelerates recovery but does not change natural history. Indicated for quality of life when conservative measures fail.
Limitation: Heterogeneity in studies, different surgical techniques, variable conservative care protocols.

Recurrent Disc Herniation Risk Factors

3
Carragee EJ et al • Spine (2006)
Key Findings:
  • Cohort study of risk factors for recurrent herniation after microdiscectomy
  • Recurrence rate: 7% at 2 years, 10% at 5 years
  • Risk factors: Large annular defect (greater than 6mm), younger age, smoking, heavy occupation
  • Aggressive discectomy did NOT reduce recurrence rate
  • Preserving disc height important for adjacent segment health
Clinical Implication: Remove loose fragments only, preserve disc height. Patient education on lifestyle modification (smoking cessation, avoid heavy lifting) may reduce recurrence.
Limitation: Retrospective study, variable surgical techniques, patient selection bias.

Natural History of Lumbar Disc Herniation

3
Saal JA, Saal JS • Spine (1990)
Key Findings:
  • Prospective study of conservative treatment for lumbar disc herniation
  • 90% of patients improved with aggressive conservative care (physio, epidural injections)
  • Only 10% required surgery
  • Larger herniations (sequestrated) had better prognosis than protrusions
  • MRI showed disc resorption in many cases
Clinical Implication: Natural history favors spontaneous resolution. Conservative care is first-line treatment. Large sequestrations paradoxically have better outcomes than contained protrusions.
Limitation: No control group, selection bias (motivated patients), aggressive conservative protocol not universally available.

Early Surgery vs Prolonged Conservative Care

1
Peul WC et al • NEJM (2007)
Key Findings:
  • RCT comparing early surgery (within 2 weeks) vs prolonged conservative care (minimum 6 months) for sciatica
  • Early surgery group had faster pain relief (6-8 weeks earlier)
  • By 1 year, outcomes were similar between groups
  • Quality of life improved faster in surgery group
  • No difference in complication rates
Clinical Implication: Early surgery appropriate for patients with severe symptoms impacting quality of life. Prolonged suffering is unnecessary if conservative care fails early.
Limitation: Dutch healthcare system (different access to care), patient selection, crossover allowed after 6 months.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Classic L5 Radiculopathy

EXAMINER

"A 45-year-old manual laborer presents with 8 weeks of right leg pain radiating to the dorsum of the foot. Pain is worse than back pain, aggravated by sitting and coughing. On examination, he has weak ankle dorsiflexion (EHL grade 3/5), numbness in the first web space, and positive straight leg raise at 40 degrees. MRI shows a right paracentral disc herniation at L4-L5 compressing the L5 nerve root. What is your assessment and management?"

EXCEPTIONAL ANSWER
This gentleman has a **right L5 radiculopathy** secondary to a paracentral L4-L5 disc herniation. The clinical and radiological findings are concordant. **Clinical Assessment:** The key features are: - **Radicular pain worse than back pain** - this is classic for disc herniation - **L5 dermatomal distribution** - dorsum of foot, first web space - **L5 motor weakness** - EHL weakness (grade 3/5), ankle dorsiflexion - **Positive SLR at 40 degrees** - highly suggestive of nerve root compression - **MRI correlation** - L4-L5 disc herniation compressing the L5 traversing root **Management:** He has had 8 weeks of symptoms, which suggests he has **failed initial conservative management**. However, I would first ensure he has had adequate conservative care: - **Medical management**: NSAIDs, neuropathic agents (gabapentin or pregabalin) - **Physiotherapy**: Core strengthening, nerve glides, posture education - **Activity modification**: Avoid heavy lifting, prolonged sitting Given the **progressive motor weakness** (EHL grade 3/5), I would discuss surgical options. Motor deficit is a relative indication for earlier surgery to prevent permanent footdrop. **Surgical Option:** I would offer **microdiscectomy** at the L4-L5 level: - **Approach**: Posterior midline or paramedian incision - **Technique**: Remove herniated fragment, decompress L5 nerve root, preserve disc height - **Goals**: Decompress nerve root, improve leg pain (may not fully restore motor function) - **Outcomes**: 85-90% good results for leg pain, motor recovery variable depending on duration **Consent Discussion:** - **Benefits**: Leg pain relief, prevent further motor deterioration - **Risks**: Recurrence (5-10%), dural tear (1-2%), infection (1-2%), nerve injury (less than 1%) - **Realistic expectations**: Leg pain improves, motor recovery may be incomplete, back pain may persist I would also counsel about **postoperative rehabilitation** (early mobilization, core strengthening) and **lifestyle modification** (smoking cessation, weight management, proper lifting technique) to reduce recurrence risk.
KEY POINTS TO SCORE
L5 radiculopathy from L4-L5 disc herniation (traversing root)
Clinical features: EHL weakness, first web space numbness, positive SLR
MRI confirms diagnosis and matches clinical level
Failed 8 weeks conservative care - surgical option appropriate
Progressive motor deficit is a relative indication for earlier surgery
Microdiscectomy is the gold standard surgical treatment
Remove herniated fragment, decompress nerve root, preserve disc height
Realistic expectations: leg pain relief, motor recovery variable, back pain may persist
Consent includes recurrence risk (5-10%), dural tear (1-2%), infection (1-2%)
Postoperative: early mobilization, core strengthening, lifestyle modification
COMMON TRAPS
✗Operating based on MRI alone without clinical correlation
✗Not assessing adequacy of conservative management
✗Promising full motor recovery (EHL weakness may not fully resolve)
✗Not counseling about recurrence risk and prevention
✗Not addressing back pain expectations (microdiscectomy treats leg pain)
LIKELY FOLLOW-UPS
"How would you perform the microdiscectomy? Walk me through the steps."
"The patient asks about recurrence - what do you tell him?"
"What would you do if you encountered a dural tear intraoperatively?"
VIVA SCENARIOChallenging

Scenario 2: Cauda Equina Syndrome

EXAMINER

"A 55-year-old woman presents to ED with 48 hours of bilateral leg weakness, numbness around her perineum, and urinary retention requiring catheterization. She has a background of chronic low back pain and left leg sciatica for 6 months. On examination, she has bilateral ankle dorsiflexion weakness (grade 3/5), absent ankle jerks, saddle anesthesia, and reduced anal tone. How do you assess and manage this patient?"

EXCEPTIONAL ANSWER
This is a **cauda equina syndrome** presenting as a surgical emergency. I would take immediate action. **Immediate Assessment:** The red flag features are: - **Saddle anesthesia** - perianal and perineal numbness - **Urinary retention** - requiring catheterization - **Bilateral leg symptoms** - bilateral weakness and sensory changes - **Reduced anal tone** - indicates cauda equina involvement This is **cauda equina syndrome** until proven otherwise, and requires **urgent imaging and emergency decompression**. **Immediate Management (within hours):** **Step 1: Clinical Assessment** - **Detailed neurological exam**: Document motor, sensory, reflexes, rectal exam (tone, perianal sensation) - **Bladder function**: Assess for urinary retention (already catheterized), overflow incontinence - **Bowel function**: Assess for fecal incontinence or constipation - **Timing**: Document when symptoms started (critical for prognosis) **Step 2: Urgent MRI Lumbar Spine** - **Within 1-2 hours** - this is an emergency investigation - **Look for**: Massive central disc herniation, epidural mass, spinal canal stenosis - **Exclude**: Epidural abscess, hematoma, tumor (require different management) **Step 3: Emergency Decompression** If MRI confirms cauda equina compression from disc herniation: - **Timing**: Emergency surgery **within 24-48 hours** (sooner is better) - **Approach**: Bilateral laminectomy at affected level(s) to decompress cauda equina - **Technique**: Wide decompression, remove compressing disc fragments, decompress all affected nerve roots - **Goals**: Prevent permanent bowel/bladder dysfunction, maximize neurological recovery **Surgical Details:** - **Anesthesia**: General anesthesia, patient already catheterized - **Position**: Prone on Wilson frame, abdominal decompression - **Incision**: Midline incision, wider exposure than standard microdiscectomy - **Decompression**: Bilateral laminectomy (not just hemilaminotomy), remove ligamentum flavum, decompress dural sac - **Disc removal**: Remove all compressing disc fragments centrally, may need to explore both sides - **Confirmation**: Dural sac should be lax, pulsatile, nerve roots mobile **Prognosis Discussion:** - **Timing is critical**: Surgery within 48 hours gives best chance of recovery - **Bladder recovery**: 50-70% regain normal function if operated within 48 hours - **Motor recovery**: Often incomplete, depends on severity and duration - **Permanent dysfunction possible**: May have residual bowel/bladder issues - **Realistic expectations**: Even with emergency surgery, full recovery is not guaranteed **Postoperative Management:** - **Catheter**: May need to remain for days to weeks until bladder function recovers - **Bowel regimen**: Manage constipation, educate about bowel care - **Urodynamics**: Follow-up assessment if bladder dysfunction persists - **Physiotherapy**: Intensive rehab for motor recovery - **MDT approach**: Urology, spinal surgeon, physio, OT involvement
KEY POINTS TO SCORE
Cauda equina syndrome is a surgical EMERGENCY
Red flags: saddle anesthesia, urinary retention, bilateral leg symptoms, reduced anal tone
Urgent MRI within 1-2 hours to confirm diagnosis
Emergency decompression within 24-48 hours (sooner is better)
Wide bilateral laminectomy, remove all compressing disc fragments
Prognosis depends on timing - operate as soon as possible
50-70% bladder recovery if surgery within 48 hours
Realistic expectations - full recovery not guaranteed even with emergency surgery
Postoperative: may need prolonged catheterization, bowel regimen, intensive rehab
MDT approach involving urology, spinal surgeon, physiotherapy
COMMON TRAPS
✗Delaying MRI or surgery (every hour counts)
✗Performing standard microdiscectomy instead of wide decompression
✗Not documenting baseline function (medico-legal)
✗Promising full recovery (realistic expectations critical)
✗Not involving MDT (urology, OT, physio) postoperatively
LIKELY FOLLOW-UPS
"What is the prognosis if surgery is delayed beyond 48 hours?"
"How would you counsel the patient preoperatively about outcomes?"
"What would you do differently if MRI showed an epidural abscess instead of disc herniation?"
VIVA SCENARIOCritical

Scenario 3: Recurrent Disc Herniation

EXAMINER

"A 38-year-old man presents with recurrent right L5 radiculopathy 18 months after successful microdiscectomy at L4-L5. He had complete resolution of leg pain after the initial surgery and returned to his job as a builder. The pain recurred 2 weeks ago after lifting a heavy object at work. MRI shows a recurrent right paracentral disc herniation at L4-L5. He asks what went wrong and what can be done. How do you manage this?"

EXCEPTIONAL ANSWER
This is a case of **recurrent lumbar disc herniation** at the same level (L4-L5) 18 months after primary microdiscectomy. I need to address both the patient's concerns about why this happened and the management options. **Discussion of Recurrence:** I would have a transparent conversation with the patient: "I understand your concern. Recurrent disc herniation occurs in **5-10% of patients** after microdiscectomy. This is not a failure of the surgery - your initial surgery was successful (complete pain relief). What has happened is that **another fragment from the same disc has herniated**, likely related to the heavy lifting incident." **Why Recurrence Happens:** - **Disc degeneration is progressive**: The disc that herniated once is weakened and prone to further herniation - **Biomechanical stress**: Your occupation (builder) involves heavy lifting and repetitive loading - **Large annular defect**: The original herniation left a defect in the disc annulus - **Risk factors**: Smoking (if applicable), age (young patients have more nucleus material), heavy occupation "The good news is that recurrent herniations can be treated successfully, though the outcomes are slightly less predictable than primary surgery." **Management Options:** **Option 1: Conservative Management (First-Line)** - **Try conservative care first** - same as primary herniation - **Medical management**: NSAIDs, neuropathic agents - **Physiotherapy**: Core strengthening, activity modification - **Nerve root injection**: Diagnostic and therapeutic - **Duration**: 6 weeks trial **Rationale**: Some recurrent herniations resolve spontaneously (same natural history as primary), and reoperation carries slightly higher risks (scar tissue, dural adhesions). **Option 2: Revision Microdiscectomy** If conservative care fails after 6 weeks: - **Approach**: Same posterior approach, more challenging due to scar tissue - **Technique**: Careful dissection through scar, identify anatomy, remove recurrent fragment - **Considerations**: Higher risk of dural tear (5-10% vs 1-2%), nerve root injury - **Outcomes**: 70-80% good results (slightly lower than primary surgery) **Option 3: Fusion (Not First-Line)** - **Indication**: Only if multiple recurrences, instability, or severe disc degeneration - **Technique**: Remove disc entirely, insert cage, posterior instrumentation - **Considerations**: More invasive, longer recovery, adjacent segment disease risk - **Reserve for**: Failed revision microdiscectomy or documented instability **My Recommendation for This Patient:** I would recommend: 1. **Initial conservative care (6 weeks)** - rule out spontaneous resolution 2. **If failed**: Revision microdiscectomy 3. **Counsel about risks**: Higher dural tear risk, scar tissue, outcomes slightly less predictable 4. **Lifestyle modification**: Discuss occupational change (avoid heavy lifting long-term) 5. **Smoking cessation**: If applicable - impairs healing and increases recurrence **Preventing Further Recurrence:** - **Occupational modification**: Consider changing to lighter duties, avoid repetitive heavy lifting - **Core strengthening**: Physiotherapy to strengthen lumbar spine support - **Smoking cessation**: If applicable - **Weight management**: Reduce biomechanical load - **Proper lifting technique**: Educate about safe manual handling **Realistic Expectations:** - **Success rate**: 70-80% for revision surgery (vs 85-90% for primary) - **Recurrence risk**: Slightly higher after revision (10-15%) - **Alternative**: If multiple recurrences, may need to consider fusion - **Occupation**: May need to consider career change if heavy manual work
KEY POINTS TO SCORE
Recurrent disc herniation occurs in 5-10% after microdiscectomy
Not a surgical failure - disc degeneration is progressive
Risk factors: heavy occupation, smoking, large annular defect, young age
Conservative management first - same natural history as primary herniation
Revision microdiscectomy if failed conservative care (6 weeks)
Revision surgery has higher risks: dural tear (5-10%), scar tissue, nerve injury
Outcomes slightly lower than primary surgery (70-80% vs 85-90%)
Fusion is NOT first-line - reserve for multiple recurrences or instability
Lifestyle modification critical: occupational change, smoking cessation, core strengthening
Realistic expectations about success rate and further recurrence risk
COMMON TRAPS
✗Promising same outcomes as primary surgery (outcomes are slightly worse)
✗Not trying conservative care first (some recurrences resolve spontaneously)
✗Rushing to fusion (not indicated for first recurrence)
✗Not addressing occupational risk factors (heavy manual work)
✗Not counseling about higher surgical risks (dural tear, scar tissue)
LIKELY FOLLOW-UPS
"What are the indications for fusion after recurrent disc herniation?"
"How would you minimize the risk of dural tear during revision surgery?"
"If the patient had three recurrences, what would you recommend?"

MCQ Practice Points

Anatomy Question

Q: An L4-L5 disc herniation typically compresses which nerve root? A: L5 nerve root (the traversing root). Lumbar nerve roots exit BELOW their corresponding pedicle. At L4-L5 level, the L4 root has already exited, and the L5 root crosses the disc space in the lateral recess. Far lateral herniations at L4-L5 level would compress the exiting L4 root.

Clinical Diagnosis Question

Q: What is the most specific clinical sign for lumbar disc herniation? A: Crossed straight leg raise (raising the unaffected leg reproduces pain in the symptomatic leg). This has 95% specificity for disc herniation. Standard SLR has high sensitivity (90%) but low specificity (25%).

Imaging Question

Q: When is MRI indicated for acute sciatica? A: MRI is NOT needed in the first 6 weeks unless red flags are present (cauda equina, progressive motor deficit, bilateral symptoms, severe or rapidly worsening pain). 90% of disc herniations resolve with conservative care, and MRI findings do not change initial management. MRI is indicated if considering surgery after failed conservative care.

Management Question

Q: What is the evidence for surgery vs conservative care for lumbar disc herniation? A: SPORT trial showed that surgery provides faster pain relief, but outcomes at 4 years are similar between surgical and conservative groups due to high crossover rate (50% of conservative patients eventually had surgery). Surgery is indicated for quality of life and faster return to function, NOT superior long-term outcomes. Natural history favors spontaneous resolution.

Surgical Indication Question

Q: What are the absolute indications for surgery in lumbar disc herniation? A: Cauda equina syndrome is the only absolute indication (emergency surgery within 24-48 hours). Progressive motor deficit (e.g., worsening foot drop) is a relative indication for earlier surgery. Failed conservative care (6 weeks) with persistent disabling symptoms is an elective indication. Predominant back pain alone is NOT an indication for microdiscectomy.

Complication Question

Q: What is the most common complication after microdiscectomy? A: Recurrent disc herniation (5-10% within 2 years). Risk factors include large annular defect, smoking, heavy occupation, young age. Aggressive discectomy (removing all nucleus) does NOT reduce recurrence rate. Revision surgery is performed if conservative management fails for recurrence.

Australian Context and Medicolegal Considerations

Epidemiology in Australia:

  • Common presentation: Disc herniation is one of the most frequent reasons for spine referral
  • Occupational: Workers' compensation claims common for work-related lifting injuries
  • Healthcare access: Public hospital waitlists for elective microdiscectomy can be 6-12 months; private faster
  • Return to work: WorkCover/workers' compensation implications for occupational injuries

Guidelines and Standards:

ACSQHC Guidelines

  • Conservative care first: Minimum 6 weeks unless red flags
  • Avoid overimaging: No MRI in first 6 weeks for typical sciatica
  • Opioid stewardship: Minimize opioid use, favor NSAIDs and neuropathic agents
  • Return to work focus: Early mobilization and functional restoration

Medicolegal Considerations:

Documentation is Critical

Key documentation requirements:

  • Pre-operative: Document failed conservative care (6 weeks physio, medications, injections), informed consent including recurrence risk
  • Intra-operative: Level confirmation (fluoroscopy images), nerve root decompression achieved, any complications (dural tear)
  • Post-operative: Immediate motor/sensory exam, discharge planning, follow-up arrangements
  • Cauda equina cases: Time of symptom onset, time of MRI, time to theatre - timing is medicolegally scrutinized

Common litigation issues:

  • Wrong level surgery: Always confirm level with fluoroscopy (never event)
  • Delayed cauda equina surgery: Document time of presentation, MRI, and surgery; delays beyond 48 hours are scrutinized
  • Informed consent: Discuss recurrence risk, persistent pain, nerve injury - document in notes
  • Nerve root injury: Rare but serious; ensure gentle technique, document any intraoperative concerns

Occupational considerations:

  • Return to work planning: Discuss with patient and employer; may need modified duties
  • WorkCover claims: Coordinate with insurer, functional capacity assessment
  • Vocational rehabilitation: Consider referral if unable to return to previous occupation

LUMBAR DISC HERNIATION

High-Yield Exam Summary

Key Anatomy

  • •Nucleus pulposus = 80% water, proteoglycans, type II collagen
  • •Annulus fibrosus = type I collagen, posterolateral region thinnest
  • •PLL weak laterally = posterolateral herniation in 95%
  • •L4-L5 disc compresses L5 root (traversing), L4 root already exited
  • •Cauda equina starts below conus (L1-L2 level), ends at S1-S2

Classification

  • •Protrusion = base wider than dome, contained
  • •Extrusion = base narrower than dome, through annulus
  • •Sequestration = fragment separated, free in canal, best prognosis
  • •Paracentral = most common (70%), compresses traversing root
  • •Far lateral = compresses exiting root, needs different approach

Clinical Diagnosis

  • •Radicular pain > back pain = hallmark feature
  • •SLR positive if leg pain at less than 60 degrees (not back pain)
  • •Crossed SLR = 95% specificity for disc herniation
  • •L5: EHL weakness, first web space numbness, no reflex
  • •S1: Plantar flexion weakness, lateral foot numbness, absent ankle jerk
  • •Cauda equina = saddle anesthesia + bowel/bladder + bilateral symptoms

Management Algorithm

  • •Conservative first (6 weeks) unless red flags - 90% improve
  • •MRI only after 6 weeks or if red flags present
  • •Surgery for: CES (emergency), progressive motor deficit, failed conservative care
  • •Microdiscectomy = remove fragment, preserve disc height, early mobilization
  • •Never operate on imaging alone - must match clinical findings

Surgical Technique

  • •Confirm level with fluoroscopy before incision
  • •Interlaminar window: remove ligamentum flavum, expose nerve root
  • •Gentle nerve retraction - never pull forcefully
  • •Remove loose fragments only - do not excise entire disc
  • •Valsalva before closure to check for dural tear
  • •Early mobilization - discharge same day or next day

Outcomes and Evidence

  • •85-90% good outcomes for leg pain (back pain may persist)
  • •Recurrence rate 5-10% at 2 years
  • •SPORT trial: surgery faster relief, similar 4-year outcomes to conservative care
  • •CES: Operate within 24-48h for best bladder recovery (50-70%)
  • •Revision surgery: 70-80% success (vs 85-90% primary)
  • •Fusion NOT indicated for first recurrence
Quick Stats
Reading Time148 min
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