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Quadriceps Tendinitis

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Quadriceps Tendinitis

Comprehensive guide to Quadriceps Tendinopathy - anatomy, clinical assessment, differential diagnosis, and management strategies for suprapatellar pain

complete
Updated: 2024-12-18
High Yield Overview

QUADRICEPS TENDINITIS

Suprapatellar pain | Deep flexion aggravation | Eccentric loading | Older athlete demographic

SupraSuprapatellar pain location
Over 30yOften older than patellar tendinitis
DeepPain in deep flexion (greater than 90°)
CalcificCalcific tendinitis mimic

BLAZINA CLASSIFICATION

Stage 1
PatternPain only after activity
TreatmentActivity mod + rehab
Stage 2
PatternPain during and after, no performance deficit
TreatmentRehab + injections (PRP)
Stage 3
PatternPain during and after, performance disrupted
TreatmentExtended rest to Surgery
Stage 4
PatternComplete tendon rupture
TreatmentSurgical repair

Critical Must-Knows

  • Location: Tenderness at the superior pole of the patella (distal quadriceps insertion).
  • Demographics: Often affects slightly older athletes compared to Jumper's Knee.
  • Biomechanics: Deep flexion places maximum tensile load on the quadriceps tendon.
  • Differential: Must rule out suprapatellar plica and bipartite patella.
  • Treatment: Eccentric strengthening remains the cornerstone.

Examiner's Pearls

  • "
    Tenderness is at the suprapatellar insertion (superior pole).
  • "
    Pain is reproduced by resisted extension from deep flexion.
  • "
    Often associated with a 'gap' palpable in ruptures (sulcus sign).
  • "
    Calcification is more common than in patellar tendon.

Clinical Imaging

Imaging Gallery

MRI right knee. Sagittal T1 image. Demonstrating rupture patellar tendon attachment from inferior pole of patella, high riding patella, chronic tendinosis of patellar tendon and clustering of sheared
Click to expand
MRI right knee. Sagittal T1 image. Demonstrating rupture patellar tendon attachment from inferior pole of patella, high riding patella, chronic tendinCredit: Vun SH et al. via J Surg Case Rep via Open-i (NIH) (Open Access (CC BY))

Critical Exam Points for Quadriceps Tendinopathy

Location is Key

Differentiation from Jumper's Knee is purely anatomical. Quadriceps tendinopathy is at the superior pole. Patellar tendinopathy is at the inferior pole. Do not confuse them.

Rupture Risk

Bilateral quadriceps tendon rupture is a classic association with systemic disease (Renal failure, Diabetes, RA, Hyperparathyroidism) or fluoroquinolone/steroid use. Always ask about medical history.

Deep Flexion

The quadriceps tendon is under maximal tension in deep flexion. Pain is often worse at the bottom of a squat, whereas patellar tendinopathy pain is often worse during the descent/deceleration phase.

Surgical Nuance

Surgical debridement requires splitting the tendon. The quadriceps tendon is multi-layered (trilaminar). Understanding this anatomy is crucial for partial thickness tears.

Quick Decision Guide - Suprapatellar Pain

ConditionLocationAggravating FactorManagement
Quadriceps TendinopathySuperior pole insertionDeep squat / decelerationEccentric rehab
Suprapatellar PlicaMedial suprapatellarPlausible snap/clickInjection / resection
Bipartite PatellaSuperolateral poleDirect trauma / overuseRest / excision
Quadriceps RuptureSuprapatellar gapUnable to extend kneeSurgical repair
Prepatellar BursitisAnterior to patellaKneeling / direct pressureAspiration / protection
Mnemonic

Rupture Risk Factors - REAL-MD

R
Renal failure
Chronic renal failure (dialysis)
E
Endocrine
Diabetes, Hyperparathyroidism
A
Age
Over 40 years old
L
Local injection
Steroid history
M
Medications
Fluoroquinolones, Anabolic steroids
D
Disease
Systemic inflam (RA, SLE, Gout)

Memory Hook:REAL-MDs check for systemic causes

Mnemonic

Quadriceps Anatomy - RF-VL-VM-VI

RF
Rectus Femoris
Superficial layer
VL/VM
Vastus Lat/Med
Middle layer
VI
Vastus Intermedius
Deep layer

Memory Hook:Three layers of the tendon

Mnemonic

Rehab Progression - IEM

I
Isometric
Pain relief
E
Eccentric
Tendon remodeling
M
Movement
Functional energy storage

Memory Hook:I Eat Meat (muscle/tendon fuel)

Overview and Epidemiology

Quadriceps tendinopathy is a degenerative condition affecting the suprapatellar insertion of the quadriceps tendon. While less common than patellar tendinitis (Jumper's Knee), it is a significant cause of anterior knee pain, particularly in older athletes and weightlifters.

Epidemiology:

  • Prevalence: Lower than patellar tendinitis (approx 1/10th frequency in some series).
  • Age: Typically older cohort (greater than 35-40 years) compared to patellar tendinitis (under 30 years).
  • Sports: Volleyball, basketball, weightlifting (deep squats).
  • Gender: Male > Female.

The Age Distinction

The "Jumper's Knee" spectrum moves proximal with age. Young/Adolescents = Tibial Tubercle (Osgood-Schlatter). Young Adults = Patellar Tendon (Inf pole). Older Adults = Quadriceps Tendon (Sup pole).

Pathophysiology and Mechanisms

Anatomy:

  • Insertion: Base of patella (superior pole).
  • Layers (Trilaminar):
    1. Superficial: Rectus femoris
    2. Middle: Vastus lateralis and Vastus medialis (conjoined)
    3. Deep: Vastus intermedius
  • Hypovascular Zone: 1-2cm proximal to the superior pole of the patella is the "watershed" area, most prone to rupture and degeneration.

Pathophysiology:

  • Identical to patellar tendinopathy: Angiofibroblastic tendinosis.
  • Microtearing formed by repetitive eccentric loading outpacing repair.
  • Calcification: More common in quadriceps tendon than patellar tendon (Calcific tendinitis).
  • Bone Spur: Traction enthesophyte at superior pole is common (tooth sign).

Classification Systems

Blazina Classification (Modified)

Originally described for Jumper's knee, applied to Quads tendon.

StageSymptomsFunctionTreatment
Phase 1Pain only after activityNo functional impairmentIce, NSAIDs, eccentric rehab
Phase 2Pain during and after activityCan still compete/performActivity mod + intense rehab
Phase 3Pain during and afterUnable to compete at levelProlonged rest, consider surgery
Phase 4Complete tendon ruptureLoss of extensionSurgical repair

Clinical Utility

Blazina aids in decision making. Stage 1/2 is rehab. Stage 3 warrants imaging and discussion of intervention.

Simple, clinically relevant.

MRI Grading of Tendinosis

  • Grade 1: Signal intensity change (T1/T2 increased) only. No dimension change.
  • Grade 2: Signal change + Tendon thickening/fusiform swelling.
  • Grade 3: Partial thickness tearing (fluid signal in tendon).
  • Grade 4: Full thickness rupture.

Differentiation between Grade 2 (tendinosis) and Grade 3 (partial tear) is important for rehab aggression.

VISA-P Score Note

While designed for patellar tendinopathy, the questions (squatting, lunging, sitting) are highly relevant for quadriceps tendinopathy and it is often used as a surrogate outcome measure in studies, though technically validated for the patellar tendon.

  • Interpretation: Less than 80 suggests significant functional deficit.

The VISA-P remains the standard outcome measure for clinical tracking.

Clinical Presentation and Assessment

History:

  • Suprapatellar pain.
  • Pain with deep knee flexion (e.g., squatting past 90 degrees).
  • "Start-up pain" (stiffness after sitting).
  • History of jumping or heavy lifting.

Physical Examination:

Physical Exam Findings

ManoeuvreFindingSignificance
PalpationTenderness at superior pole of patellaConfirm anatomic location
Resisted ExtensionPain, especially from deep flexionLoads quad tendon maximally
Gap TestPalpable sulcus suprapatellarSigns of rupture
Active Straight Leg RaiseLag / InabilityComplete rupture mechanism failure
Suprapatellar PlicaMedial snapping / tendernessDifferential diagnosis

Check the Extensor Mechanism:

  • Always confirm the integrity of the mechanism. A "painful" tendon can also be a "partially ruptured" tendon.
  • Lag: Any extensor lag with active straight leg raise is a red flag.

Bilateral Signs

If a patient presents with bilateral symptoms, particularly if acute-on-chronic pain, screen for systemic risk factors (Renal, Diabetes, Gout).

Investigations

1. Radiographs (X-ray):

  • Views: AP, Lateral, Skyline.
  • Findings:
    • Tooth Sign: Traction spur at superior patellar pole (enthesophyte).
    • Calcification: Loose bodies or intratendinous calcification (calcific tendinitis).
    • Patella Alta/Baja: Assessing height.
    • Bipartite Patella: Usually superolateral (saupe type III).

2. Ultrasound (US):

  • Excellent for tendon architecture.
  • Findings: Thickening, hypoechogenicity, loss of fibrillar pattern.
  • Dynamic: Can assess impairment with flexion/extension.
  • Calcification: High sensitivity for calcium deposits.

3. MRI:

  • Gold Standard for quantifying degeneration and ruling out intra-articular pathology.
  • Sequence: T2/STIR hyperintensity in the distal quadriceps tendon.
  • Partial Tears: Fluid signal intensity interrupting fibers.

Imaging Modality Comparison

ModalityProsCons
UltrasoundDynamic, cheap, good for guided injectionOperator dependent
MRIShows internal derangement, partial tears, edemaCost, static
X-rayShows calcification and spursSoft tissue invisible

Management

Cornerstone of Treatment: Management mirrors patellar tendinopathy but focuses on deep flexion control.

Protocol:

  1. Phase 1 (Analgesia): Isometric holds (e.g., wall sit at 60 deg). Ice. NSAIDs.
  2. Phase 2 (Isotonic): Heavy Slow Resistance (HSR). Leg press, squats.
  3. Phase 3 (Eccentric):
    • Note: Standard eccentric decline squats may impinge the superior pole if deep flexion is forced too early.
    • Controlled eccentric lowering (e.g., single leg press lowering).
  4. Phase 4 (Plyometric): Return to jumping.

Deep Flexion Modification

In quadriceps tendinopathy, deep flexion (past 90 deg) compresses the tendon against the femur/trochlea. Early rehab should often limit flexion to 0-60 degrees to avoid compressive load, progressing to deep flexion late.

📊 Management Algorithm
Illustration of quadriceps tendon compression against the trochlea in deep flexion
Click to expand
Deep flexion (greater than 90 degrees) significantly increases the compressive load on the deep surface of the quadriceps tendon against the femoral trochlea, exacerbating insertional tendinopathy.Credit: OrthoVellum

Standard therapy duration: 3-6 months.

Injections:

  1. PRP (Platelet-Rich Plasma): Secondary option for recalcitrant cases. Level 1 evidence is weak/mixed.
  2. Prolotherapy: Hyperosmolar dextrose to stimulate healing.
  3. Dry Needling: Stimulation of healing response.
  4. Polidocanol: Sclerosing of neovessels (if present on Doppler).

NO STEROIDS

Just like the patellar tendon, corticosteroid injection into the quadriceps tendon carries a high risk of rupture and is CONTRAINDICATED.

Calcific Lavage:

  • If calcific tendinitis is present (calcium deposit), US-guided barbotage (lavage) can be curative.

Interventional options should only be considered after adequate load management.

Indications:

  • Failure of prolonged conservative care (greater than 6 months).
  • Partial tear greater than 50% with functional deficit.
  • Calcific tendinitis resistant to lavage.
  • Rupture (acute).

Procedures:

  1. Debridement: Excision of angiofibroblastic tissue.
  2. Repair: If significant tendon removed, side-to-side suture or anchor repair.
  3. Osteoplasty: Removal of prominent superior pole spur (if causing impingement).

Post-operative recovery is lengthy (4-6 months).

Surgical Technique

Open Debridement and Repair:

Approach:

  • Midline longitudinal incision extending proximal to patella.
  • Expose paratenon.

Technique:

  1. Inspection: Incise paratenon. Pre-operative MRI guides location (often deep/central).
  2. Tenotomy: Longitudinal split in tendon to access deep layers.
  3. Debridement: Excise grey/friable/mucinoid tissue. "Scrape" the superior pole if cystic change present.
  4. Spur Removal: If a traction spur exists ("tooth"), remove with rongeur/burr to prevent impingement.
  5. Calcium: If calcific deposit, excise white, chalky material.
  6. Closure:
    • Carefully repair the longitudinal split (Vicryl/PDS).
    • If debridement leaves a defect, side-to-side closure.
    • Close paratenon to prevent adhesions.

Post-op:

  • Protected weight bearing in brace (locked extension) for 2 weeks.
  • Gradual ROM increase.
  • Slower than arthroscopic patellar procedures due to muscle violation.

Open debridement provides the most reliable access to the multi-layered pathology.

Arthroscopic Evaluation:

  • Suprapatellar Pouch: View the articular side of the quadriceps tendon.
  • Undersurface Tear: Can identify partial articular-sided tears (PASTA equivalent of knee).
  • Debridement: Shaver debridement of articular surface fraying.
  • Limitation: Cannot access the substance of the tendon or superficial layers easily. Open approach is generally preferred for classic tendinosis.

Arthroscopy is primarily diagnostic or for intra-articular pathology (plica).

Complications

Complications Management

ComplicationRisk FactorsManagement
RuptureSteroid use, aggressive early loadSurgical Repair
Persistant PainIncomplete resection, wrong diagnosisRe-imaging / Revision
StiffnessProlonged immobilizationMUA / Lysis of adhesions
InfectionOpen surgeryAntibiotics +/- Washout

Quadriceps Rupture:

  • The ultimate failure of tendinosis.
  • Requires urgent repair (suture anchors or transosseous tunnels).
  • Delayed repair (greater than 3 weeks) significantly worsens prognosis due to retraction.

Postoperative Care and Rehabilitation

Phase 1 (Week 0-2)
  • Goal: Protect repair, wound healing.
  • Brace in extension.
  • WBAT in extension.
  • Isometric quads.
Phase 2 (Week 2-6)
  • Goal: Regain ROM.
  • Unlock brace 0-30, then 0-60, then 0-90.
  • Active flexion, passive extension.
  • Avoid active extension against resistance.
Phase 3 (Week 6-12)
  • Goal: Strength.
  • Wean brace.
  • Start concentric strengthening.
  • Progress to eccentric loading.
Phase 4 (3-6 Months)
  • Goal: Power and Sport.
  • Plyometrics.
  • Return to sport criteria (limb symmetry index greater than 90%).

Rehab vs Patellar

Post-op rehab for quadriceps tendon debridement is typically slower than patellar tendon debridement because the muscle belly itself is often more affected by the surgical approach and inhibition.

Outcomes and Prognosis

  • Conservative: High success rate (70-90%) with compliance to activity modification and rehab.
  • Surgical: Good results (80-90% satisfaction) in appropriately selected patients.
  • Recurrence: Can occur if biomechanics (training errors, flexibility) are not addressed.
  • Calcific Tendinitis: excellent prognosis with lavage or excision.

Evidence Base

Level I
📚 Filardo et al. - PRP in Tendinopathy
Key Findings:
  • Systematic review of PRP.
  • No clear benefit of PRP over placebo or standard care in chronic tendinopathy.
  • Use remains controversial despite popularity.
Clinical Implication: PRP should not be first-line. Optimizing load management is primary.
Source: Sports Med 2011

Level II
📚 Rudavsky et al. - Tendinopathy Rehab
Key Findings:
  • Comparison of isometric, eccentric, and HSR (heavy slow resistance).
  • All methods effective.
  • Isometrics superior for immediate pain relief.
  • HSR may have better patient compliance than pure eccentrics.
Clinical Implication: Tailor the load type to the patient phase. Isometrics for pain, HSR/Eccentrics for remodeling.
Source: J Orthop Sports Phys Ther 2014

Level III
📚 Bono et al. - Rupture Outcomes
Key Findings:
  • Outcomes of quad tendon rupture repair.
  • Early repair (less than 2 weeks) yields significantly better ROM and functional scores than delayed.
  • Midsubstance tears heal better than avulsions.
Clinical Implication: Diagnosis of rupture must not be delayed. 'Strain' diagnosis in a rupture is a major error.
Source: Am J Sports Med 2007

Level IV
📚 Scuderi - Rupture Classification
Key Findings:
  • Notable for classification of ruptures.
  • Underscored the importance of diagnosing partial vs complete tears.
  • Lag is the critical differentiator.
Clinical Implication: Physical exam (lag test) is the most critical diagnostic tool for rupture.
Source: Clin Orthop 1978

Level III
📚 Ippolito - Histopathology
Key Findings:
  • Classic paper describing angiofibroblastic dysplasia.
  • Confirmed degenerative nature over inflammatory nature.
Clinical Implication: Basis for modern 'tendinosis' vs 'tendinitis' terminology.
Source: J Bone Joint Surg 1986

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Weightlifter with Suprapatellar Pain

EXAMINER

"A 45-year-old weightlifter presents with chronic pain just above the patella. It hurts deep in the squat. X-rays show a small spur at the superior pole. How do you manage him?"

EXCEPTIONAL ANSWER
**Assessment:** - **History:** Confirm pain character (mechanical), duration. Rule out systemic history (age 45 is borderline). - **Exam:** Palpate superior pole. Test extension power. Check for gap (rule out chronic partial tear). - **Imaging:** X-ray confirms spur (enthesophyte). Ultrasound would be my next step (dynamic, check for calcification/tears). **Diagnosis:** Quadriceps Tendinopathy (Insertional). **Management:** 1. **Modification:** Stop deep squats greater than 90 degrees temporarily. Box squats to limit depth. 2. **Rehab:** Isometrics for pain to Heavy Slow Resistance (HSR). 3. **Adjuncts:** Heel lifts (reduce ankle DF demand), shockwave therapy. 4. **Failure:** If 6 months fail + spur is large, consider surgical debridement + spur removal (osteoplasty).
KEY POINTS TO SCORE
Age over 40 raises suspicion for Quads vs Patellar
Deep flexion exacerbates load
Spur indicates chronicity
Rule out rupture
COMMON TRAPS
✗Ignoring the spur (cause of impingement)
✗Diagnosing patellar tendinitis (wrong location)
✗Injecting steroids
LIKELY FOLLOW-UPS
"He feels a 'pop' and can't lift his leg. What happened?"
"Rupture. Needs surgery."
VIVA SCENARIOAdvanced

Diabetic Patient with Bilateral Pain

EXAMINER

"A 55-year-old diabetic male presents with bilateral suprapatellar pain and difficulty climbing stairs. He has a history of renal disease. What is your concern?"

EXCEPTIONAL ANSWER
**Concern:** Impending or chronic bilateral quadriceps tendon rupture/tendinopathy due to systemic disease. **Mechanism:** - Uremia (Renal) and Diabetes affect collagen cross-linking. - High risk of spontaneous rupture. **Workup:** - **Exam:** CRITICAL to test active extension. Is there a lag? Palpable defects? - **Imaging:** Ultrasound/MRI to look for substance quality and partial tearing. - **Medical:** Optimize glucose, renal status. **Management:** - Conservative if intact: Very gentle rehab. - **Warning:** High-risk patient. Low threshold for protected weight bearing. - If ruptured: Surgical repair with possible augmentation (allograft/wire) due to poor tissue quality.
KEY POINTS TO SCORE
Systemic associations (Renal/Diabetes)
Bilateral presentation is a red flag
Poor tissue quality affects repair options
COMMON TRAPS
✗Treating as simple overuse tendinitis
✗Missed bilateral rupture (patient can still walk with stiff knees)
✗Not optimizing medical comorbidities
LIKELY FOLLOW-UPS
"What other systemic conditions are risks?"
"Hyperparathyroidism, RA, SLE, Gout, Fluoroquinolones."
VIVA SCENARIOStandard

Calcific Tendinitis

EXAMINER

"Ultrasound reports a 1cm calcific deposit in the quadriceps tendon of a 35-year-old female. She has failed physiotherapy. Options?"

EXCEPTIONAL ANSWER
**Diagnosis:** Calcific Quadriceps Tendinitis. **Natural History:** - Formative phase to Resting phase to Resorptive phase (very painful). **Management Options:** 1. **Barbotage (Lavage):** Ultrasound-guided needling and lavage to wash out the calcium (toothpastelike). High success rate. 2. **Shockwave:** Can break up calcification (painful but effective). 3. **Surgery:** Arthroscopic or Open excision of deposit. **Recommendation:** - US-guided barbotage is less invasive than surgery and often curative. I would refer for this first.
KEY POINTS TO SCORE
Calcific tendinitis differs from degenerative tendinosis
Barbotage is a key treatment modality
Resorptive phase is the most painful
COMMON TRAPS
✗Immediate surgery without trying lavage
✗Ignoring the intense pain of resorptive phase
LIKELY FOLLOW-UPS
"What describes the calcium appearance?"
"Toothpaste-like in resorptive phase, chalky in resting."

MCQ Practice Points

Anatomy

Q: Which layer of the quadriceps tendon is deepest? A: Vastus Intermedius. (Superficial: RF, Middle: VL/VM, Deep: VI).

Risk Factors

Q: Which antibiotic class is associated with tendon rupture? A: Fluoroquinolones (e.g., Ciprofloxacin).

Location

Q: Where is the 'watershed' zone of hypovascularity in the quad tendon? A: 1-2 cm proximal to the superior pole of the patella.

Metabolic Association

Q: A patient on dialysis with knee pain likely has pathology in which tendon? A: Quadriceps tendon (strongly associated with renal failure/secondary hyperparathyroidism).

Imaging

Q: What is the 'Tooth Sign' on lateral knee X-ray? A: A traction enthesophyte (spur) at the superior pole of the patella, indicative of chronic quadriceps tendinopathy.

Differential

Q: Where is a bipartite patella typically located? A: Superolateral pole (Saupe Type III).

Australian Context

  • Epidemiology: Common in Masters athletes (running, tennis) and weightlifters.
  • Referral Pathways: Ultrasound-guided therapies (barbotage) typically performed by Musculoskeletal Radiologists or Sports Physicians.
  • WorkCover: Common claim in laborers/tradesmen involving heavy lifting/squatting.
  • Diabetes: High prevalence of T2DM in Australia increases population risk for rupture complications.

Quadriceps Tendinitis Essentials

High-Yield Exam Summary

Key Facts

  • •Insertion: Superior pole of patella
  • •Age group: Older (over 40) than Jumper's Knee
  • •Deep flexion (greater than 90°) aggravates pain
  • •Systemic link: Renal, Diabetes, Steroids

Imaging

  • •X-ray: Tooth sign (spur) at superior pole
  • •US/MRI: Hypoechoic/High signal in critical zone
  • •Hypovascular zone: 1-2cm proximal to insertion
  • •Always rule out partial tear

Management

  • •Isometrics to HSR to Eccentrics
  • •Limit deep flexion in early phase
  • •Barbotage for calcific deposits
  • •Surgery for failed conservative / rupture

Red Flags

  • •Bilateral symptoms (Systemic cause)
  • •Extensor lag (Rupture)
  • •Inability to SLR (Rupture)
  • •Fluoroquinolone use
Quick Stats
Reading Time61 min
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