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Back to CIM Cases
OncologyPaediatric Bone Tumours

Langerhans Cell Histiocytosis - Eosinophilic Granuloma

Oncology
Intermediate
6 min
High Yield
LCHeosinophilic granulomaBirbeck granulesCD1aS100vertebra planapunched-out lesionskeletal surveydiabetes insipidus
6:00
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CIM Case: Langerhans Cell Histiocytosis - Eosinophilic Granuloma

Clinical Scenario

Patient: 7-year-old boy Presentation: Localised skull pain and tender swelling over the right parietal region for 3 weeks, worse at night, mild irritability Relevant history: Previously well, no weight loss, no polyuria/polydipsia, normal development, no family history of malignancy Examination findings:

  • 3cm tender, non-mobile swelling over right parietal bone
  • No overlying skin changes
  • No lymphadenopathy
  • No hepatosplenomegaly
  • No skin rash or petechiae
  • No exophthalmos
  • Neurologically intact
  • No other bony tenderness

Investigations Provided

Laboratory Results

TestResultNormal RangeInterpretation
Haemoglobin125 g/L115-145Normal
WCC8.2 x 10⁹/L5-13Normal
Platelets280 x 10⁹/L150-400Normal
ESR25 mm/hr0-10Mildly elevated
CRP8 mg/L<5Mildly elevated
LDH180 U/L120-300Normal
ALP220 U/L150-400Normal (child)
Albumin42 g/L35-50Normal
LFTsNormal-No liver involvement
Urine osmolality650 mOsm/kg500-800Normal (no DI)

Imaging

Image 1: Skull X-ray (AP and Lateral)

Radiological features:

  • Well-defined, punched-out lytic lesion in right parietal bone
  • Size approximately 3 x 2.5 cm
  • Geographic bone destruction
  • No sclerotic rim
  • "Bevelled edge" appearance (outer table more affected than inner)
  • No periosteal reaction
  • No soft tissue calcification
  • No other skull lesions visible

Image 2: CT Head with Bone Windows

CT findings:

  • Sharply marginated lytic lesion
  • Destruction of both inner and outer tables
  • Bevelled margins confirming asymmetric involvement
  • Soft tissue component extending into subgaleal space
  • No intracranial extension
  • No dural involvement

Image 3: Skeletal Survey

Findings:

  • No other bony lesions identified
  • Normal spine (no vertebra plana)
  • Normal pelvis and long bones
  • Unifocal disease confirmed

Questions & Model Answers

Q1

What is the likely diagnosis and describe the spectrum of this disease?

Q2

What investigations would you perform to stage this patient?

Q3

What histological findings would confirm the diagnosis?

Q4

How would you manage this patient with unifocal skull LCH?

Q5

What if this patient had vertebral involvement with vertebra plana?

Q6

What features would indicate multisystem disease and how would management change?


Key Teaching Points

Pattern Recognition

This pattern suggests Eosinophilic Granuloma (Unifocal LCH):

  • Child aged 5-15 years
  • Punched-out lytic lesion without sclerotic margin
  • Bevelled edge on skull (pathognomonic)
  • No periosteal reaction
  • Single lesion on skeletal survey
  • No systemic symptoms

Distinguish from Other Lesions:

FeatureLCHOsteomyelitisEwing Sarcoma
Age5-15 yearsAny10-20 years
MarginPunched-outIll-definedPermeative
Periosteal reactionMinimalAggressiveOnion-skin
Soft tissue massSmallAbscessLarge
Systemic symptomsRareFever, malaiseB symptoms

Critical Management Points

  1. Skeletal survey is ESSENTIAL - bone scan misses 30% of lesions
  2. Screen for diabetes insipidus - urine osmolality in all patients
  3. Unifocal lesions often self-resolve - especially after biopsy
  4. Intralesional steroids are first-line - for persistent lesions
  5. Vertebra plana reconstitutes - avoid surgery when possible
  6. CD1a + S100 positive - confirms diagnosis

Common Examiner Follow-ups

Q: "Why does bone scan miss LCH lesions?"

LCH lesions are often "cold" on bone scan because:

  • Rapid bone destruction outpaces osteoblastic response
  • Bone scan relies on osteoblastic activity for uptake
  • Skull lesions particularly prone to false negatives
  • Up to 30% of lesions missed on bone scan

Therefore, skeletal survey (plain X-rays) is the preferred staging modality.


Q: "What is the role of chemotherapy in LCH?"

IndicationAgents
Not indicatedUnifocal bone lesion
IndicatedMultifocal bone disease
IndicatedAny multisystem involvement
First-lineVinblastine + prednisolone x 12 months
SalvageCladribine, cytarabine

Q: "What are the late effects of LCH?"

Late EffectIncidenceManagement
Diabetes insipidus15-25%DDAVP lifelong
Growth hormone deficiency10%GH replacement
Orthopaedic sequelae20%Usually mild
Hearing loss10-15%If temporal bone involved
Neurodegenerative disease1-4%Cerebellar, rare

Q: "Can LCH recur?"

Disease TypeRecurrence RateNotes
Unifocal10-15%Usually another bone lesion
Multifocal30-40%May need prolonged therapy
Multisystem50-60%May need salvage chemotherapy

Recurrence is most common within 2 years of diagnosis. Follow-up should continue for at least 5 years.


Related Topics

  • Bone Tumours in Children
  • Paediatric Spine Tumours
  • Vertebra Plana Differential
  • Skeletal Survey Indications
  • Ewing Sarcoma
  • Osteomyelitis
Quick Stats
Category
Oncology
DifficultyIntermediate
Time Allowed6 min
Reading Time36 min
Investigation Types
bloodsimaginghistology
Exam Tips

Read the clinical scenario carefully

Structure your answers systematically

Consider differential diagnoses

Justify your investigation choices

Think about management priorities