InfectionSpinal Infection

Spinal Tuberculosis (Pott's Disease)

Infection
Advanced
6 min
High Yield
tuberculosispott diseasespinal infectionkyphosisgibbus deformityRIPE therapy
6:00
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CIM Case: Spinal Tuberculosis (Pott's Disease)

Clinical Scenario

Patient: 45-year-old man from India Presentation: 4 months of progressive back pain, weight loss (8kg), and night sweats Relevant history: Immigrated to Australia 2 years ago. No known TB contacts or previous treatment. Non-smoker, no HIV risk factors disclosed. Examination findings:

  • Tender thoracolumbar junction with localised kyphosis at T12-L1
  • No gibbus deformity palpable
  • Full power in lower limbs (MRC 5/5)
  • Sensation intact, reflexes normal, plantar responses flexor
  • No bladder or bowel symptoms

Investigations Provided

Laboratory Results

TestResultNormal RangeInterpretation
Hb108 g/L130-170 g/L↓ Anaemia of chronic disease
WCC8.2 ×10⁹/L4-11 ×10⁹/LNormal
Platelets385 ×10⁹/L150-400 ×10⁹/LNormal
CRP28 mg/L<5 mg/L↑ Mildly elevated
ESR65 mm/hr<20 mm/hr↑↑ Elevated
Albumin28 g/L35-50 g/L↓ Hypoalbuminaemia
QuantiFERON GoldPositiveNegativeIndicates TB exposure
HIVNegativeNegativeImportant to exclude

Imaging

Image 1: Lateral Radiograph of Thoracolumbar Spine

Radiological features:

  • Anterior vertebral body destruction at T12 and L1
  • Reduced disc height at T12-L1 (late finding)
  • Localised kyphotic angulation at T12-L1
  • Bilateral paravertebral soft tissue shadow (psoas shadow widening)
  • No obvious calcification

Image 2: Chest X-ray PA

Findings:

  • No active pulmonary TB changes visible
  • No upper lobe cavitation or consolidation
  • Normal cardiac silhouette

Image 3: MRI Thoracolumbar Spine (T1, T2/STIR, Post-contrast)

MRI findings:

  • Destruction of T12 and L1 vertebral bodies with low T1, high STIR signal
  • Relative preservation of T12-L1 disc in early images (classic for TB)
  • Subligamentous spread along anterior longitudinal ligament extending to T11 and L2
  • Large bilateral psoas abscesses (5cm right, 4cm left)
  • Epidural soft tissue extending into spinal canal causing mild cord compression
  • No myelomalacia signal within cord
  • Kyphotic deformity measuring 25°

Questions & Model Answers

Q

Describe the investigation findings and differential diagnosis

Q

How would you confirm the diagnosis?

Q

What are the indications for surgery in spinal TB?

Q

The biopsy confirms TB with ZN-positive AFB. What is your antibiotic regimen?

Q

At 3 months, the patient develops new bilateral lower limb weakness. What is your management?

Q

What is the prognosis and follow-up for spinal TB?


Key Teaching Points

Pattern Recognition

This pattern suggests Spinal Tuberculosis:

  • Patient from TB-endemic area with constitutional symptoms
  • Insidious onset back pain with weight loss and night sweats
  • Thoracolumbar junction involvement (most common site)
  • Deceptively normal inflammatory markers (WCC often normal)
  • MRI: Subligamentous spread with relative disc preservation

Distinguish from Pyogenic Spondylodiscitis:

FeatureTBPyogenic
OnsetInsidious (months)Acute (days-weeks)
WCC/CRPOften normal/mildly elevatedUsually markedly elevated
Disc involvementPreserved earlyDestroyed early
Spread patternSubligamentous (under ALL)Direct disc destruction
AbscessCommon (psoas)Less common
CalcificationMay be presentRare

Distinguish from Spinal Metastasis:

  • Metastasis: Usually posterior elements involved, no disc changes
  • TB: Anterior vertebral body, subligamentous spread, disc relatively preserved
  • Both can cause weight loss - look for primary tumour

Critical Management Points

  1. Tissue diagnosis before treatment - never treat empirically
  2. Medical treatment is curative - surgery for complications only
  3. RIPE regimen for 12-18 months - shorter courses have high failure rates
  4. Monitor for drug toxicity - especially hepatotoxicity
  5. Compliance is crucial - consider DOT if adherence concerns

Common Examiner Follow-ups

Q: "What is the most common site for spinal TB?"

The thoracolumbar junction (T12-L1) is most commonly affected, followed by lower thoracic and upper lumbar spine. Cervical involvement is less common but carries higher risk of neurological complications.


Q: "What is a 'cold abscess' and why is it called that?"

A cold abscess is a collection of caseous material and pus that forms without the classical inflammatory signs of warmth and erythema. It occurs because TB produces a granulomatous rather than acute inflammatory response. Can track along fascial planes (e.g., psoas abscess presenting in groin).


Q: "What is Frankel grading and how would you use it?"

Frankel grading assesses spinal cord function:

  • A: Complete motor and sensory loss
  • B: Incomplete - sensation present, no motor function
  • C: Motor function present but not useful (power <3)
  • D: Motor function useful (power ≥3)
  • E: Normal motor and sensory function

Used for documenting baseline deficit and monitoring improvement.


Q: "What are the risk factors for kyphosis progression?"

  • Age <10 years (greatest risk)
  • Loss of >2 vertebral bodies
  • Thoracic spine involvement
  • Pan-vertebral disease
  • Initial kyphosis >30°
  • Posterior element involvement

Children with >2 vertebral bodies destroyed may need prophylactic fusion to prevent severe kyphosis.


  • Pyogenic Spondylodiscitis
  • Spinal Cord Compression
  • Psoas Abscess
  • Kyphosis Correction Surgery
  • TB in Orthopaedics