Safe interval between tibialis anterior and EHL β protects the deep peroneal nerve and anterior tibial artery; the workhorse exposure for arthrodesis, total ankle arthroplasty and plafond fixation
- The interval is between tibialis anterior (medial) and extensor hallucis longus (lateral). Both are supplied by the deep peroneal nerve, so this is NOT a true internervous plane.
- It is safe because the deep peroneal nerve and anterior tibial artery travel together 1 to 2 cm lateral to EHL at the ankle β dissection stays medial to the bundle and the EHL is retracted medially WITH the nerve.
- The superficial peroneal nerve branches emerge 10 to 12 cm proximal to the lateral malleolus, are highly variable, and cross the field subcutaneously β they are the structure most often injured (3 to 5 percent).
- The extensor retinaculum must be divided for joint access and REPAIRED at closure (0 Vicryl horizontal mattress) to prevent tendon bowstringing.
- Main uses are ankle arthrodesis, total ankle arthroplasty, and ORIF of pilon and talar fractures; it extends proximally to the tibial shaft and distally to the talar neck.
When & Why
What it exposes. The anterior approach gives direct, extensile access to the tibiotalar joint β the anterior tibial plafond and talar dome β through the interval between the tibialis anterior and extensor hallucis longus (EHL) tendons. It exploits the superficial nature of the anterior ankle and the predictable course of the neurovascular bundle, making it the gold-standard exposure for ankle arthrodesis, total ankle arthroplasty (TAR) and ORIF of pilon and talar fractures. Advantages - Direct visualisation of the anterior plafond and talar dome.
- Extensile β proximally to the tibial shaft, distally to the talar neck.
- Relatively safe when the neurovascular bundle is identified systematically.
- Minimal muscle dissection β a true interval between tendons, no muscle divided.
- Good implant access for arthrodesis hardware and arthroplasty components. Disadvantages - No access to the posterior ankle β a separate posterior approach is needed for posterior pathology.
- Superficial peroneal nerve at risk β its variable course makes injury possible.
- Thin anterior soft-tissue envelope β wound-healing concerns, especially after trauma.
- Limited lateral access β cannot extend to the lateral malleolus through the same incision.
- Typical indications
- End-stage post-traumatic or inflammatory arthritis, failed TAR, deformity, talar AVN
- What the anterior approach provides
- Joint exposure for cartilage removal and fixation
- Typical indications
- Older, lower-demand patients; neutral or correctable alignment; good bone stock
- What the anterior approach provides
- Strict midline exposure for jigs and component placement
- Typical indications
- Tibial plafond fractures needing anatomic articular reduction
- What the anterior approach provides
- Direct joint surface visualisation and anterior plating
- Typical indications
- Talar neck (Hawkins IIβIV) and talar body fractures
- What the anterior approach provides
- Access to the talar neck; medial malleolar osteotomy may be added
- Typical indications
- OCD of the anterior talar dome (greater than 1.5 cm, uncontained), anterior impingement osteophytes, septic washout, synovectomy, loose-body or hardware removal
- What the anterior approach provides
- Focused anterior arthrotomy
Contraindications - Absolute β active infection overlying the surgical site (unless the procedure is debridement for that infection); severe peripheral vascular disease with compromised healing (ankle-brachial index less than 0.5).
- Relative β poor soft-tissue envelope (previous burns, infection, radiation); morbid obesity (BMI greater than 40); severe osteoporosis; active Charcot arthropathy; unrealistic patient expectations. Position & landmarks. Supine on a radiolucent table, with a small bump under the ipsilateral hip to internally rotate the leg and bring the anterior ankle parallel to the floor, and slight knee flexion (10β15 degrees) to relax the gastrocnemius. The foot hangs free off the end of the table or rests on a sterile bolster so it can be manipulated and imaged. Palpate and mark the tibialis anterior tendon (most medial, prominent with dorsiflexion-inversion), the EHL tendon (central, palpated by extending the hallux), the EDL tendons (lateral), the dorsalis pedis pulse between EHL and EDL, and the malleoli β the ankle joint line runs level with the malleolar tips. Tourniquet. Optional. Many experienced surgeons operate without a tourniquet to assess tissue perfusion throughout and avoid ischaemia-reperfusion; if used, a high-thigh cuff at 250β300 mmHg (about 100 mmHg above systolic), deflated before closure, aiming for less than 120 minutes. Anaesthesia & adjuncts. Spinal/epidural or general anaesthesia; tranexamic acid 1 g IV at induction reduces blood loss by 30β50 percent; a popliteal block gives 12β24 hours of analgesia (but blocks motor function, so early nerve assessment is delayed). Skin prep with chlorhexidine 2% in alcohol circumferentially from toes to below knee; image intensifier positioned for AP and lateral views before draping. Global epidemiology. Symptomatic ankle arthritis is most often post-traumatic (about 70 to 80 percent), in contrast to the hip and knee where primary osteoarthritis predominates β so patients presenting for arthrodesis or TAR are typically younger. Ankle arthrodesis is classically a younger, higher-demand, frequently post-traumatic population; TAR volumes are rising worldwide as implants mature and is offered to lower-demand patients with neutral or correctable alignment. Ankle fractures show a bimodal distribution β younger males (high-energy sport and road trauma) and older women (low-energy fragility injuries).
The Exposure
Work down through the layers directly over EHL, protecting the superficial peroneal nerve at the skin, opening and tagging the extensor retinaculum, then identifying the neurovascular bundle lateral to EHL and retracting it medially together with EHL to expose the capsule.

- Position
- Most medial, large tendon in its own tunnel
- What you do with it
- Retract medially; muscular belly proximally
- Position
- Central, passing to the hallux
- What you do with it
- The index tendon β retract medially WITH the neurovascular bundle
- Position
- 1 to 2 cm lateral to EHL at the ankle
- What you do with it
- Loop together; retract medially with EHL (the critical protective move)
- Position
- Lateral, multiple slips to toes 2β5
- What you do with it
- Retract laterally
Exposure sequence
- A 10 to 12 cm longitudinal incision centered between the tibialis anterior and EDL tendons, directly over or just lateral to the EHL tendon β beginning 8 to 10 cm proximal to the joint line and curving onto the talar neck 3 to 4 cm distal.
- Hold the scalpel perpendicular to the skin and make a single pass through skin and subcutaneous tissue to deep fascia; avoid beveling, which undermines the skin edges.
- Look for the superficial peroneal nerve branches (white glistening cords, usually lateral and highly variable); mobilise each gently and protect it with a vessel loop.
- Identify the extensor retinaculum β the white glistening fascial band crossing the anterior ankle (superior band 2β3 cm wide, 2β4 cm proximal to the joint; inferior band Y-shaped with a lateral stem on the calcaneus).
- Incise both the superior and inferior retinaculum longitudinally in line with the skin to mobilise the tendons.
- Tag each edge with a 0 Vicryl suture (long tails, clamped) so it can be retrieved and repaired at closure.
- Tibialis anterior (medial) β the largest, most medial tendon, in its own tunnel.
- Extensor hallucis longus (central) β thinner, passing to the hallux; the incision lies over it and it is the landmark for the neurovascular bundle (which lies lateral to it).
- Extensor digitorum longus (lateral) β multiple thin slips to toes 2β5.
- Palpate the dorsalis pedis pulse between EHL and EDL (absent in 8 to 12 percent of normal people β an anatomic variant).
- Dissect along the lateral border of EHL: the deep peroneal nerve lies 1 to 2 cm lateral to EHL, travelling with the anterior tibial artery and its two venae comitantes.
- Place a vessel loop around nerve and artery together; handle gently β excessive manipulation causes neuropraxia.
- Retract the tibialis anterior medially, the EHL together WITH the neurovascular bundle medially (the critical protective move), and the EDL laterally.
- This exposes the anterior ankle joint capsule.
- Use gentle tension only β never seat a self-retaining retractor directly on the bundle, and release retraction periodically.
- Make a longitudinal capsulotomy over the joint, extending proximally onto the anterior distal tibia and distally onto the talar neck; tag the edges with stay sutures.
- Plantarflex the ankle to deliver the talar dome into view and expose the anterior plafond.
- The anterior tibiotalar joint is now fully exposed for cartilage removal (arthrodesis), bone cuts (arthroplasty), fracture reduction and fixation, or OCD and osteophyte management.
Both tibialis anterior and EHL are deep-peroneal-nerve muscles, so the safety of this approach does not come from an internervous plane. It comes from the consistent course of the deep peroneal nerve and anterior tibial artery 1 to 2 cm lateral to EHL: identify the bundle early, keep dissection medial to it, and retract the EHL and bundle together medially at every step.
Retract the EHL tendon and the neurovascular bundle together medially as a single unit. Because the deep peroneal nerve and anterior tibial artery travel with EHL, moving them together keeps the bundle out of the working field and away from retractors, saw cuts and bone work throughout the case.
Dangers & Extensions
Structures at risk, by layer
- Incidence and behaviour
- 3 to 5 percent β the commonest nerve injury; variable course, crosses the field subcutaneously
- Protection
- Perpendicular skin incision; identify and loop every crossing branch; keep the incision more medial if anatomy permits
- Incidence and behaviour
- 1 to 3 percent, mostly neurapraxia recovering in 3 to 6 months; permanent injury under 1 percent
- Protection
- Identify early via the dorsalis pedis pulse; retract medially with EHL; gentle retraction; blade guards for saw work; no cautery within 5 mm
- Incidence and behaviour
- Under 1 percent; isolated injury usually tolerated when posterior tibial and peroneal arteries are intact
- Protection
- Travels with the nerve β retract medially together; never seat a retractor directly on the artery
- Incidence and behaviour
- Tendon bowstringing if unrepaired β loss of mechanical efficiency and poor cosmesis
- Protection
- Tag edges on the way in; repair with 0 Vicryl horizontal mattress at closure; test by dorsiflexing the ankle
If the neurovascular bundle is injured - Deep peroneal nerve β neurapraxia: release tension, observe (most recover in 3β6 months), AFO to prevent equinus, EMG at 6β8 weeks if not recovering. Complete transection: primary epineural repair with 8-0 or 9-0 nylon. A gap: tag the ends and refer for delayed nerve grafting within 6 months.
- Anterior tibial artery β obtain proximal and distal control. Partial laceration: primary repair with 6-0 or 7-0 Prolene. Complete transection: assess foot perfusion (capillary refill, posterior tibial pulse, handheld Doppler); if perfusion is adequate, ligate both ends β most patients tolerate this when the posterior tibial and peroneal arteries are intact. If perfusion is inadequate, this is an emergency: immediate vascular surgery consultation for vein-graft reconstruction. Monitor with hourly neurovascular checks for 24 hours (compartment-syndrome risk).
- Superficial peroneal nerve β recognised transection: primary repair or bury the proximal stump in muscle to prevent neuroma. Delayed painful neuroma: desensitisation, scar massage and gabapentin/pregabalin; if refractory, excision and burial of the proximal stump. Extensile options. Extend proximally along the EHL border to reach the distal tibial shaft and anterior tibia; extend distally onto the talar neck and head. You cannot extend laterally to the fibula through this incision β a separate lateral incision is required. Variants include the anteromedial approach (between TA and EHL) and the anterolateral approach (between EHL and EDL) for specific lateral pathology. Closure (layer by layer). Deflate the tourniquet (if used) and achieve haemostasis with bipolar cautery β avoid cautery near the deep peroneal nerve β then irrigate with 3β6 L of saline. A drain is optional for TAR (reduces haematoma) but avoided in arthrodesis (interferes with fusion biology) and trauma (infection risk). Close in layers: capsule with 0 or 1 Vicryl; extensor retinaculum (critical) with 0 Vicryl horizontal mattress using the tagged edges; subcutaneous with 2-0 or 3-0 Vicryl inverted sutures to eliminate dead space; skin with 3-0 or 4-0 Monocryl subcuticular plus a Dermabond sealant. Apply a non-circumferential posterior splint with the ankle in neutral dorsiflexion and the hindfoot neutral, from toes to below the knee. Early complications (less than 6 weeks). Superficial wound infection 2β5 percent; deep infection 1β2 percent (higher in trauma and the immunocompromised); wound dehiscence 2β4 percent; DVT/PE under 1 percent with prophylaxis. Late complications (greater than 6 weeks). Arthrodesis nonunion 5β15 percent overall and up to 40 percent in smokers β confirm with CT, and revise with bone graft and augmented fixation (blade plate or nail). TAR loosing/subsidence 10β15 percent at 10 years β revision arthroplasty if bone stock is good, salvage arthrodesis if not. Post-traumatic arthritis in 25β40 percent after pilon fractures despite anatomic reduction. Adjacent-joint arthritis (subtalar, talonavicular) in 20β30 percent at 10 years after arthrodesis, from increased stress on neighbouring joints.
Procedures Through This Approach
- Ankle arthrodesis β gold-standard access for end-stage post-traumatic or inflammatory arthritis, failed TAR, deformity correction and talar AVN.
- Pilon fracture ORIF β anatomic articular reduction of the tibial plafond; often combined with a posterolateral approach and staged for soft-tissue management.
- Total ankle arthroplasty β strict midline anterior exposure for instrumentation and component implantation.
- Talar neck and body fractures (Hawkins IIβIV) β primary anterior access; a medial malleolar osteotomy may be added for the body.
- Osteochondral lesions of the anterior talar dome (greater than 1.5 cm, uncontained), anterior ankle impingement osteophytes, septic washout, synovectomy for inflammatory arthropathy, and loose-body or hardware removal.
Viva & Exam Focus
TENEAnterior ankle structures β medial to lateral
SANDStructures at risk β SAND
The deep peroneal nerve and anterior tibial artery travel together 1 to 2 cm lateral to EHL. Identify the bundle early and retract it medially together with the EHL tendon β the defining protective move of the approach. Injury rate is 1 to 3 percent when it is not protected systematically.
Branches emerge from the lateral compartment about 10 to 12 cm proximal to the lateral malleolus and are highly variable. They cross the field subcutaneously β identify them at the skin incision and protect each with a vessel loop. Injury (3 to 5 percent) causes dorsal foot numbness and a painful neuroma.
The interval is between tibialis anterior and EHL, both supplied by the deep peroneal nerve. It is safe because the nerve travels with EHL laterally, so dissection medial to the nerve avoids it β not because of an internervous plane.
Repair the extensor retinaculum at closure with 0 Vicryl horizontal mattress sutures. Failure causes tendon bowstringing in dorsiflexion, loss of mechanical efficiency and poor cosmesis. Tag the retinacular edges on the way in for easy repair.
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
βDescribe your approach to the anterior ankle. What structures are at risk?β
βA 55-year-old presents with end-stage ankle arthritis. How do you decide between total ankle replacement and ankle arthrodesis?β
βYou are performing an ankle arthrodesis via the anterior approach. During deep dissection you see brisk arterial bleeding. How do you manage this?β
Interval and plane
- Interval between tibialis anterior (medial) and EHL (lateral), both deep peroneal nerve β NOT a true internervous plane
- Safe because the deep peroneal nerve and anterior tibial artery travel lateral to EHL; retract EHL and bundle together medially
Incision
- 10 to 12 cm longitudinal, centered between TA and EDL, directly over or just lateral to EHL
- From 8 to 10 cm proximal to the joint line onto the talar neck 3 to 4 cm distal; scalpel perpendicular to the skin
Structures at risk (SAND)
- Superficial peroneal nerve: emerges 10 to 12 cm proximal to the lateral malleolus, variable, 3 to 5 percent injury
- Anterior tibial artery: with the deep peroneal nerve lateral to EHL, under 1 percent injury; check dorsalis pedis (absent in 8 to 12 percent)
- Deep peroneal nerve: 1 to 2 cm lateral to EHL, 1 to 3 percent injury (mostly neurapraxia); causes foot drop and first-web-space numbness
- Dorsalis pedis: confirm pre- and post-operatively
Exposure sequence
- Skin over EHL; protect the superficial peroneal nerve branches
- Open and tag the extensor retinaculum
- Identify TA (medial), EHL (central), EDL (lateral)
- Find and loop the neurovascular bundle lateral to EHL
- Retract TA medially; EHL plus bundle medially; EDL laterally
- Longitudinal capsulotomy; plantarflex to deliver the talar dome
Closure
- Capsule 0/1 Vicryl; retinaculum 0 Vicryl horizontal mattress (CRITICAL); subcutis 2-0/3-0 Vicryl; skin 3-0/4-0 Monocryl subcuticular plus Dermabond
- Drain optional for TAR, avoided in arthrodesis and trauma; posterior splint in neutral dorsiflexion
Outcomes and complications
- Arthrodesis union 85 to 95 percent; nonunion 5 to 15 percent (up to 40 percent in smokers)
- TAR 10-year survival about 77 to 89 percent; loosening 10 to 15 percent at 10 years
- Post-traumatic arthritis 25 to 40 percent after pilon; adjacent-joint arthritis 20 to 30 percent at 10 years after arthrodesis
- Wound infection 2 to 5 percent superficial, 1 to 2 percent deep
References
Guidelines, registries and global practice National arthroplasty registries provide the most robust real-world implant-survival data and consistently show better survivorship for modern (third-generation) implants, with aseptic loosening and subsidence dominating revision indications across registries. Outcomes also improve with surgeon and centre volume.
- Reported TAR survival
- Around 90 percent at 5 years, declining at 10 years
- Reported TAR survival
- Approximately 80 to 85 percent cumulative survival at 10 years
- Reported TAR survival
- Comparable mid-term survival; revision mainly for loosening
- Reported TAR survival
- Pioneering data; identified high early-generation failure
Named-society guidance (side by side) - Surgeon and centre volume β AOFAS (US), BOFAS (UK) and other national foot-and-ankle societies recommend that TAR be performed by fellowship-trained foot-and-ankle surgeons in higher-volume centres, with registry participation for surveillance.
- Antibiotic prophylaxis (AAOS / NICE / WHO aligned) β first-generation cephalosporin (cefazolin 2 g IV, 3 g if over 120 kg) within 60 minutes of incision; clindamycin or vancomycin for severe beta-lactam allergy or MRSA risk.
- VTE prophylaxis (AAOS / NICE / ACCP) β mechanical prophylaxis and early mobilisation for all; pharmacological prophylaxis individualised. Foot-and-ankle societies note that routine chemical prophylaxis for isolated below-knee immobilisation is less strongly supported than for hip and knee arthroplasty β guidance genuinely differs and should be risk-stratified.
- Modifiable risk optimisation (universal) β smoking cessation before elective arthrodesis (nonunion rates rise markedly in smokers) and glycaemic optimisation (HbA1c under 7 to 8 percent) before elective surgery. Representative rehabilitation protocols - Arthrodesis β strict non-weight-bearing for 6 to 8 weeks, then protected weight-bearing in a boot, progressing once union is confirmed radiologically (often around 12 weeks).
- Total ankle replacement β implant- and surgeon-dependent, from a short non-weight-bearing period to early weight-bearing as tolerated in a boot, with physiotherapy in the early weeks.
- Fracture ORIF β dictated by fracture configuration and fixation stability; talar and high-energy pilon injuries are kept non-weight-bearing longer (8 to 12 weeks) to mitigate AVN and reduction loss.
Anterior ankle approach β anatomy and safety
Describes the anterior approach through the interval between tibialis anterior and extensor hallucis longus. This is not a true internervous plane (both muscles are supplied by the deep peroneal nerve) but is safe because the neurovascular bundle β the deep peroneal nerve plus the anterior tibial artery β lies just lateral to EHL and is retracted medially together with the EHL tendon. - Interval between tibialis anterior and EHL, both deep peroneal nerve β not a true internervous plane
- Deep peroneal nerve and anterior tibial artery lie lateral to EHL β retract medially with EHL
- Superficial peroneal nerve branches highly variable and most commonly injured (3 to 5 percent)
- Extensor retinaculum must be repaired at closure to prevent tendon bowstringing
Open versus arthroscopic ankle arthrodesis
Systematic review and meta-analysis of 10 cohort studies (507 patients) comparing arthroscopic and open ankle arthrodesis. Arthroscopic fusion achieved a significantly higher fusion rate, with less blood loss and a shorter hospital stay. The open anterior approach remains essential where deformity, bone loss or hardware preclude arthroscopic fusion. - Arthroscopic fusion higher fusion rate than open (odds ratio 0.25, 95% CI 0.11 to 0.57)
- Less estimated blood loss and shorter tourniquet time with arthroscopy
- Shorter length of stay and better 1-year recovery scores
- Open anterior approach still required for deformity, bone loss or hardware
Total ankle replacement survivorship and outcomes
Systematic review and meta-analysis of modern total ankle replacements (58 papers, 7942 implants). TAR reliably improves pain, function and motion for at least a decade, but the supporting evidence is weak and biased, and high-quality randomised trials comparing TAR with fusion are still needed. - Overall survivorship 89% at 10 years; annual failure rate 1.2% (95% CI 0.7 to 1.6)
- Mean AOFAS improved from 40 pre-operatively to 80 at a mean 8.2-year follow-up
- Mean total range of movement improved from 23 to 34 degrees
- Periprosthetic radiolucencies in up to 23% at a mean of 4.4 years
Outcomes after high-energy tibial plafond (pilon) fractures
Retrospective cohort of 80 high-energy pilon fractures evaluated at a mean of 3.2 years. High-energy pilon fractures carry a guarded prognosis β even technically successful fixation may leave persistent pain, stiffness and functional limitation due to the initial cartilage and soft-tissue injury. - SF-36 general health significantly worse than population norms at 3.2 years
- Persistent stiffness in 35%, swelling in 29% and ongoing pain in 33%
- 43% of previously employed patients were not working at follow-up
- External fixation and adverse socioeconomic factors were associated with poorer outcomes
Total ankle arthroplasty versus ankle arthrodesis
Systematic review and meta-analysis pooling 852 total ankle arthroplasty and 1262 ankle arthrodesis patients. Intermediate outcomes of TAR and arthrodesis appear similar; neither is universally superior and the choice should be individualised to age, alignment, bone stock, adjacent-joint arthritis and activity demands through shared decision-making. - Similar mean AOFAS scores (TAR 78.2 versus arthrodesis 75.6)
- TAR 5-year survival 78% and 10-year survival 77%
- Revision 7% after TAR (mainly loosening) versus 9% after arthrodesis; nonunion accounted for 65% of arthrodesis revisions
- Below-knee amputation in 1% after TAR versus 5% after arthrodesis