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Back to Operative Surgery
Spine

Anterior Cervical Discectomy and Fusion (ACDF) - Single Level

Surgical technique guide for Anterior Cervical Discectomy and Fusion (ACDF) - Single Level - FRCS exam preparation

Core Procedure
intermediate
By OrthoVellum Medical Education Team

Reviewed by OrthoVellum Editorial Team

Orthopaedic clinicians and medical editors • Published by OrthoVellum Medical Education Team

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High Yield Overview

ANTERIOR CERVICAL DISCECTOMY AND FUSION (ACDF) - SINGLE LEVEL

Smith-Robinson approach | Left-sided transverse incision | Avascular plane medial to carotid sheath | Discectomy with decompression | Cage + anterior plate fixation

SpineSubspecialty
12Key Steps
8Danger Zones
60-90minDuration

Critical Must-Knows

  • Surface landmarks: C2-C3 mandible angle, C3-C4 hyoid, C4-C5 thyroid cartilage, C6 cricoid
  • Smith-Robinson interval: medial to carotid sheath (avascular plane between SCM and strap muscles)
  • Left-sided approach preferred: left RLN has predictable course under aortic arch vs variable right RLN
  • Complete discectomy to PLL, then remove PLL to visualize dura - no epidural space anteriorly in cervical spine
  • Monitor for post-op airway compromise from retropharyngeal hematoma - catastrophic if missed

Examiner's Pearls

  • "
    Level localization MANDATORY before incision: wrong-level surgery is a never-event - use fluoroscopy with spinal needle in disc
  • "
    RLN injury rate 2-11%: most are temporary neuropraxia (resolve 6-12 weeks), permanent 0.5-1%
  • "
    Dysphagia most common complication: 50% at 1 week, 20% at 1 month, 5% persistent at 1 year
  • "
    Plate adds fusion stability: reduces non-union from 10% to 5% for single level
  • "
    Adjacent segment disease: 2-3% per year radiographic, symptomatic 1%/year - controversial whether fusion causes this

Critical Danger Structures

Danger 1

Recurrent laryngeal nerve - LEFT: predictable course, loops under aortic arch, ascends in tracheoesophageal groove. RIGHT: loops under subclavian, more variable, may be lateral. Injury causes hoarseness.

Danger 2

Superior laryngeal nerve (external branch) - Runs with superior thyroid artery at C3-C4 level. Injury causes voice fatigue, pitch problems (cricothyroid muscle).

Danger 3

Sympathetic chain - On anterolateral vertebral bodies, under lateral longus colli. Injury causes Horner syndrome (miosis, ptosis, anhidrosis).

Danger 4

Carotid sheath (carotid artery, IJV, vagus nerve) - Lateral to operative field. Vagus gives off RLN. Retract gently laterally.

Danger 5

Vertebral artery - In transverse foramen of C1-C6, 14-18mm from midline. Risk during lateral decompression/foraminotomy.

Danger 6

Esophagus - Posterior to trachea, medial retraction. Perforation rate 0.1-0.5%, potentially fatal. Higher risk with revision, long retraction.

Danger 7

Spinal cord and nerve roots - Dura immediately posterior to PLL (no epidural space anteriorly). Decompression must be controlled.

Danger 8

Thoracic duct - Left-sided, enters jugular-subclavian junction. Risk at C6-C7/T1 on left. Injury causes chyle leak.

Primary Indications:

  • Cervical radiculopathy from disc herniation with dermatomal symptoms correlating to imaging
  • Cervical radiculopathy from osteophyte/foraminal stenosis causing nerve root compression
  • Cervical myelopathy with cord compression (single level - ACDF preferred over posterior)
  • Failed 6-12 weeks conservative management for radiculopathy

Relative Indications:

  • Axial neck pain from degenerative disc disease (less predictable outcomes)
  • Soft disc herniation with motor deficit (relative urgent indication)
  • Cervical instability requiring fusion (trauma, tumor, infection - single level)

ACDF vs Posterior Approach:

  • ACDF preferred for 1-2 levels, anterior pathology (soft disc, osteophyte)
  • Posterior preferred for 3+ levels, posterior pathology, kyphosis correction
  • Combined anterior-posterior for severe instability, multilevel with kyphosis

Expected Outcomes:

  • Radiculopathy: 90-95% good/excellent outcomes
  • Myelopathy: 80% improvement or stabilization (less predictable than radiculopathy)
  • Fusion rate: 95%+ with plate at single level

Absolute Contraindications:

  • Active cervical spine infection (relative - may still need surgery with debridement)
  • Severe medical comorbidities precluding general anesthesia
  • No identifiable structural pathology correlating to symptoms

Relative Contraindications:

  • Severe osteoporosis (poor screw purchase, subsidence risk)
  • Previous anterior cervical surgery (increased RLN injury risk on same side)
  • Previous neck irradiation (wound healing risk, esophageal fragility)
  • Severe kyphotic deformity (posterior approach may be better)
  • Multi-level disease (3+ levels - consider posterior or staged)
  • Diffuse idiopathic skeletal hyperostosis (DISH) - ossification risk

Patient Counselling Points:

  • Dysphagia common initially (50%), usually resolves
  • Hoarseness risk (2-11% temporary, <1% permanent)
  • Adjacent segment disease long-term (2-3%/year)
  • Smoking significantly impairs fusion (counsel cessation)

Essential Imaging:

  • Cervical spine X-rays (AP, lateral, flexion-extension): assess alignment, instability, osteophytes
  • MRI cervical spine: disc herniation, cord signal change, nerve root compression, cord compression
  • CT cervical spine (if needed): bony anatomy, osteophyte configuration, foramen dimensions

Clinical Correlation:

  • Radiculopathy: dermatomal pain/numbness, myotomal weakness, reflex changes
  • Myelopathy: gait disturbance, hand clumsiness, hyperreflexia, positive Hoffmann/Babinski
  • Must correlate imaging level to clinical findings (don't operate on asymptomatic degeneration)

Special Investigations:

  • EMG/nerve conduction studies: if diagnosis unclear, differentiate from peripheral neuropathy
  • CT myelogram: if MRI contraindicated or unclear findings
  • Dynamic X-rays: assess stability if concern for listhesis

Pre-operative Optimization:

  • Smoking cessation (minimum 4-6 weeks pre-op, continue post-op)
  • Optimize diabetes control (HbA1c target <8%)
  • Review anticoagulation (bridge vs hold based on thrombotic risk)
  • Laryngoscopy pre-op if previous anterior neck surgery (assess RLN function)

Positioning:

  • Radiolucent table or Jackson frame
  • Shoulder roll (small - extends neck)
  • Head ring or Mayfield attachment (optional)
  • Shoulder tape for caudal traction (lower levels)

Surgical Instruments:

  • Self-retaining retractor system (Caspar, Cloward, or equivalent)
  • Caspar distraction pins and distractor
  • High-speed burr with various sizes (3-5mm)
  • Kerrison rongeurs (1-3mm)
  • Curettes (various sizes)
  • Pituitary rongeurs
  • Nerve hooks and dissectors

Implants:

  • Interbody cage: PEEK (radiolucent), titanium, or allograft bone
  • Cage sizing trials
  • Anterior cervical plate and screws (variable angle preferred)
  • Bone graft: local autograft, allograft chips, synthetic bone substitute

Imaging:

  • C-arm fluoroscopy (essential for level confirmation)
  • Neuromonitoring: MEPs, SSEPs, EMG (strongly recommended)
  • Consider intraoperative CT if available (complex cases)
Mnemonic

MHTCSurface Landmarks for Level Identification

M
Mandible angle
C2-C3 level
H
Hyoid bone
C3-C4 level
T
Thyroid cartilage
C4-C5 level (thyrohyoid notch)
C
Cricoid cartilage
C6 level (most reliable landmark)
Mnemonic

SCOPESmith-Robinson Approach Key Steps

S
SCM medial border
Identify and incise along medial border of sternocleidomastoid
C
Carotid sheath lateral
Retract carotid sheath (carotid, IJV, vagus) laterally
O
Omohyoid crossing
Divide omohyoid if necessary (can be retracted or divided)
P
Prevertebral fascia
Blunt dissection to shiny prevertebral fascia over vertebral bodies
E
Elevate longus colli
Subperiosteal elevation for retractor placement (protects sympathetic chain)

Surgical Anatomy

Surface Anatomy:

  • C2-C3: angle of mandible
  • C3-C4: hyoid bone
  • C4-C5: thyroid cartilage (thyrohyoid notch)
  • C6: cricoid cartilage (most reliable landmark)
  • C7: carotid tubercle (Chassaignac's tubercle) on transverse process

Smith-Robinson Interval:

  • Avascular plane between SCM/carotid sheath laterally and strap muscles/trachea/esophagus medially
  • Strap muscles: sternohyoid, sternothyroid, omohyoid
  • Omohyoid crosses operative field at C5-C6 level - can retract or divide
  • Deep cervical fascia envelopes all structures

Vertebral Anatomy:

  • Uncovertebral joints (joints of Luschka): lateral to disc, form lateral boundary of foramen
  • Vertebral artery: enters transverse foramen at C6 (sometimes C7), courses through C6-C1
  • Distance from midline to vertebral artery: 14-18mm (be aware during lateral decompression)
  • Anterior longitudinal ligament (ALL): tough, covers anterior vertebral bodies
  • Posterior longitudinal ligament (PLL): thinner, covers posterior disc - remove for decompression

Recurrent Laryngeal Nerve:

  • LEFT RLN: loops under aortic arch, ascends in tracheoesophageal groove (predictable)
  • RIGHT RLN: loops under subclavian artery, more variable course, may be more lateral
  • Most surgeons prefer left-sided approach for more predictable RLN anatomy
  • Bilateral RLN injury: acute airway obstruction requiring tracheostomy

Sympathetic Chain:

  • Runs on anterolateral aspect of vertebral bodies
  • Located under lateral edge of longus colli muscle
  • Elevate longus colli subperiosteally and place retractor blades underneath - protects chain
  • Injury causes Horner syndrome: miosis (small pupil), ptosis, anhidrosis

Positioning and Preparation

Patient Position: Supine with small shoulder roll (extends neck slightly - improves anterior exposure). Head in neutral or slight right rotation if left-sided approach (opens the interval). Arms tucked at sides with padding. All pressure points padded.

Anaesthesia Considerations:

  • General anaesthesia with endotracheal intubation
  • Consider awake fibreoptic intubation if severe myelopathy or instability
  • Neuromonitoring (MEPs, SSEPs) - establish baseline after induction

Fluoroscopy Setup:

  • C-arm positioned for lateral imaging
  • Tape shoulders caudally for lower cervical levels (C6-T1)
  • Confirm target level with spinal needle before incision

Skin Marking:

  • Transverse incision in skin crease
  • Center over target level (use surface landmarks as guide)
  • Typical incision 4-5cm for single level

Operative Technique

Step 1: Positioning and Neuromonitoring Baseline

Position supine with shoulder roll (extends neck for exposure). Head neutral or slight right rotation for left-sided approach. Arms tucked, pressure points padded. Tape shoulders caudally for lower cervical levels (C6-T1). Establish neuromonitoring baseline (MEPs, SSEPs) after induction.

Exam Pearl

Positioning Principles:

  • Extension improves anterior exposure but avoid hyperextension in myelopathy (risk of cord compression)
  • Slight right rotation opens left-sided interval (most surgeons prefer left approach)
  • Shoulder taping essential for C6-C7/T1 visualization on fluoroscopy
  • Document neuromonitoring baseline - any deterioration during surgery requires investigation

Dangers at this step

  • Hyperextension in myelopathy patient causing cord compression/injury
  • Pressure injury to occiput, heels, elbows from prolonged positioning
  • Brachial plexus stretch from arm positioning

Step 2: Level Localization and Incision

Fluoroscopy to identify target disc level using surface landmarks (C4-C5 at thyroid cartilage, C6 at cricoid). Place spinal needle into disc space under fluoroscopy to confirm correct level - MUST obtain lateral image to confirm. Mark transverse skin incision in skin crease at target level. This is the most important step to prevent wrong-level surgery.

Exam Pearl

Level Confirmation Protocol:

  • Surface landmarks as initial guide only (can be unreliable)
  • Spinal needle placed in disc under fluoroscopy
  • MUST obtain LATERAL image to confirm level (AP view insufficient)
  • Wrong-level surgery is a "never event" - document level confirmation
  • Some surgeons obtain another image after self-retaining retractors placed

Dangers at this step

  • Wrong-level surgery - never-event, confirm with fluoroscopy before proceeding
  • Incision too proximal or distal limiting exposure
  • Needle injury to esophagus or other structures if not placed correctly

Step 3: Superficial Dissection to Platysma

Transverse skin incision 4-5cm along skin crease. Divide subcutaneous tissue and expose platysma. Divide platysma transversely (fibers run vertically, transverse division reduces tension on closure). Identify deep cervical fascia beneath platysma.

Exam Pearl

Platysma Division:

  • Platysma fibers run obliquely/vertically
  • Divide transversely to minimize tension on wound closure
  • Meticulous hemostasis in subcutaneous layer (prevent post-op hematoma)
  • Protect marginal mandibular branch of facial nerve (superior to operative field)

Dangers at this step

  • External jugular vein injury (anterior to SCM)
  • Marginal mandibular nerve injury (superior - at level of mandible)
  • Inadequate hemostasis causing post-op hematoma

Step 4: Development of Smith-Robinson Interval

Palpate medial border of SCM. Incise deep cervical fascia along medial border of SCM. Develop avascular plane BETWEEN SCM/carotid sheath laterally (retract laterally) and strap muscles/trachea/esophagus medially (retract medially). This plane is bloodless if developed correctly with blunt finger dissection.

Exam Pearl

Smith-Robinson Interval Pearls:

  • Avascular plane exists if dissection stays in correct tissue plane
  • Finger dissection sweeps carotid sheath laterally, trachea/esophagus medially
  • Omohyoid muscle crosses at C5-C6 level - can retract superiorly/inferiorly or divide
  • Feel carotid pulse laterally to confirm correct interval (should be lateral to retractor)
  • If bleeding encountered, wrong plane - reassess and redirect

Dangers at this step

  • Carotid artery injury if plane too lateral
  • Esophageal injury if plane too medial or aggressive retraction
  • IJV injury - retracted with carotid sheath, can tear with aggressive retraction

Step 5: Exposure of Prevertebral Fascia

Continue blunt dissection medially until prevertebral fascia identified (shiny white layer over vertebral bodies). Palpate midline (feel anterior longitudinal ligament and vertebral bodies). Identify disc space (soft) between hard vertebral bodies. Place self-retaining retractor.

Exam Pearl

Prevertebral Fascia Exposure:

  • Shiny white prevertebral fascia covers longus colli muscles and vertebral bodies
  • Feel for midline - ALL is palpable longitudinal ridge
  • Disc spaces are soft, vertebral bodies are hard
  • Elevate longus colli subperiosteally BEFORE placing retractor blades
  • Retractor blades placed UNDER longus colli to protect sympathetic chain

Dangers at this step

  • Sympathetic chain injury if retractor blades placed on top of longus colli (Horner syndrome)
  • Recurrent laryngeal nerve injury from excessive medial retraction
  • Wrong level if midline not correctly identified

Step 6: Level Confirmation and Retractor Placement

Insert Caspar distraction pins into vertebral bodies above and below target disc. Confirm level AGAIN with lateral fluoroscopy (second confirmation with retractors in place). Place self-retaining retractor blades UNDER elevated longus colli muscles bilaterally. Apply distraction to open disc space.

Exam Pearl

Retractor Placement Critical Points:

  • Elevate longus colli subperiosteally from midline to 15mm laterally
  • Place retractor blades UNDER longus colli (protects sympathetic chain)
  • Release retractors periodically during case (reduces RLN neuropraxia from prolonged retraction)
  • Caspar pins in vertebral bodies (not endplates) for distraction
  • Limit distraction to avoid overdistraction and nerve root injury

Dangers at this step

  • Sympathetic chain injury if retractor blades superficial to longus colli
  • Vertebral body fracture from pin placement in osteoporotic bone
  • RLN injury from prolonged or excessive medial retraction

Step 7: Discectomy

Incise anterior longitudinal ligament (ALL) over disc with scalpel. Remove disc material with pituitary rongeurs and curettes. Use high-speed burr to remove osteophytes from vertebral body margins if present. Proceed posteriorly systematically, removing all disc material to posterior longitudinal ligament (PLL).

Exam Pearl

Discectomy Technique:

  • Complete discectomy to PLL is essential for adequate decompression
  • Use curettes to scrape disc material from endplates
  • Burr osteophytes but preserve endplate integrity (prevent subsidence)
  • Identify posterior annulus and PLL as posterior limit of initial discectomy
  • Maintain awareness of depth - dura is immediately posterior to PLL

Dangers at this step

  • Endplate violation causing subsidence (overly aggressive curetting)
  • Vertebral artery injury if burr strays too lateral (14-18mm from midline)
  • Spinal cord injury if instruments pushed posterior through PLL without control

Step 8: Posterior Decompression (PLL Removal and Foraminotomy)

Remove posterior longitudinal ligament (PLL) to visualize dura - there is NO epidural space anteriorly in the cervical spine. Use Kerrison rongeurs to remove PLL and visualize dural pulsations. Perform bilateral foraminotomy by removing medial uncinate processes with Kerrison rongeurs - ensure nerve roots are decompressed.

Exam Pearl

Decompression Goals:

  • PLL removal required for adequate decompression and to visualize dura
  • No epidural space anteriorly - dura is immediately posterior to PLL
  • Dural pulsations confirm adequate central decompression
  • Foraminotomy: remove medial uncinates to decompress exiting nerve roots
  • Lateral limit of foraminotomy: 14mm from midline (vertebral artery beyond)
  • Confirm nerve root decompression with nerve hook - should be freely mobile

Dangers at this step

  • Dural tear causing CSF leak (rare but requires repair)
  • Spinal cord contusion from instruments or bone fragments
  • Vertebral artery injury during foraminotomy (stay within 14mm of midline)
  • Nerve root injury from Kerrison rongeur during foraminotomy

Step 9: Endplate Preparation

Prepare superior and inferior vertebral endplates by removing cartilage with curettes. Expose bleeding cancellous bone (enhances fusion). Avoid violating endplate integrity (subchondral bone should remain intact - prevents subsidence). Create parallel surfaces for cage seating.

Exam Pearl

Endplate Preparation Principles:

  • Remove cartilaginous endplate to expose bleeding cancellous bone
  • Preserve subchondral bone plate (prevents cage subsidence)
  • Create flat, parallel surfaces for optimal cage contact
  • Bleeding bone essential for fusion (vascular supply for graft incorporation)
  • Balance between adequate preparation (bleeding bone) and not weakening endplate

Dangers at this step

  • Endplate violation causing subsidence (overly aggressive preparation)
  • Inadequate preparation with residual cartilage (impairs fusion)
  • Asymmetric preparation causing cage tilt

Step 10: Cage Insertion

Select appropriate interbody cage (PEEK most common, titanium, or allograft). Size cage with trials - should be slight press-fit, restore disc height but avoid overdistraction. Pack cage with bone graft material (local autograft, allograft, synthetic). Insert cage and position flush with anterior vertebral body margin. Confirm position with fluoroscopy.

Exam Pearl

Cage Selection and Insertion:

  • PEEK cages: radiolucent (allows fusion assessment), tantalum markers for position
  • Titanium cages: better osteointegration but artifact on MRI
  • Allograft bone: biological option but resorption risk
  • Height: restore disc height but avoid overdistraction (>2mm increases radiculopathy risk)
  • Cage should sit flush with or 1-2mm posterior to anterior vertebral body edge
  • Lordotic cages can help restore cervical lordosis

Dangers at this step

  • Cage too large causing overdistraction and radiculopathy
  • Cage retropulsion (migration posterior) - confirm position on fluoroscopy
  • Cage subsidence if endplates violated or osteoporotic bone
  • Inadequate graft packing reducing fusion potential

Step 11: Anterior Plate Fixation

Select plate to span from mid-superior vertebral body to mid-inferior body. Plate provides load-sharing and increases fusion rate (reduces non-union from 10% to 5%). Position plate midline on vertebral bodies. Insert screws with 12-15 degrees convergent trajectory. Confirm plate and screw position with fluoroscopy (AP and lateral).

Exam Pearl

Plate Fixation Principles:

  • Plate provides stability during fusion (load-sharing device)
  • Screw trajectory: 12-15 degrees toward midline (convergent) for best bone purchase
  • Avoid lateral cortex breach (vertebral artery, nerve root injury)
  • Variable-angle screws preferred (allows trajectory adjustment)
  • Check screw length - avoid posterior cortex breach
  • Plate reduces non-union rate from ~10% to ~5% for single level

Dangers at this step

  • Screw lateral cortex breach (nerve root/vertebral artery injury)
  • Screw posterior cortex breach (spinal cord injury)
  • Screw into adjacent disc space (affects adjacent segment)
  • Plate too long encroaching on adjacent disc (accelerates adjacent segment disease)

Step 12: Final Checks, Hemostasis, and Closure

Remove distractor pins. Confirm neuromonitoring unchanged from baseline. Fluoroscopy to confirm final cage and plate position. Release retractors and allow soft tissues to return to anatomic position. Meticulous hemostasis with bipolar cautery (careful near RLN). Irrigate thoroughly. Drain placement optional (controversial). Close platysma (3-0 Vicryl), subcutaneous (3-0 Vicryl), skin (subcuticular 4-0 Monocryl).

Exam Pearl

Closure and Post-operative Considerations:

  • Release retractors before closure - prolonged retraction increases dysphagia
  • Check neuromonitoring before closing - investigate any changes
  • Drain controversial: some evidence reduces hematoma, most surgeons don't use routinely
  • Extubate awake in OR to assess airway and voice immediately
  • Monitor for airway compromise post-op (retropharyngeal hematoma)
  • Soft diet initially (dysphagia common first few days)

Dangers at this step

  • Retropharyngeal hematoma causing airway obstruction (catastrophic if missed)
  • Esophageal injury not recognized (delayed presentation with sepsis)
  • RLN injury from cautery or traction not recognized until extubation
  • Inadequate hemostasis causing post-op hematoma and airway compromise

Complications

Major Complications - Recognition, Prevention, and Management

ComplicationRecognitionPreventionManagement
Dysphagia (50% early, 5% persistent)Difficulty swallowing, choking, aspiration, odynophagiaMinimize retractor time, release retractors periodically, careful medial retraction, avoid excessive cage heightUsually self-limiting (6-12 weeks), speech pathology assessment, soft diet, severe cases may need gastrostomy
Recurrent laryngeal nerve injury (2-11%)Hoarseness immediately post-op, weak cough, voice changes, aspirationLeft-sided approach (predictable RLN course), minimize retraction force, release retractors periodically, careful cauteryMost temporary (neuropraxia) - recover 6-12 weeks; permanent &lt;1% - ENT referral, voice therapy, medialization procedures if persistent
Retropharyngeal hematoma (0.5-1%)Progressive dyspnea, stridor, neck swelling, anxiety - can progress rapidly to complete obstructionMeticulous hemostasis, consider drain (controversial), monitor closely 24 hours post-opEMERGENCY - call for help, prepare for intubation/cricothyroidotomy, return to OR for evacuation, may need bedside evacuation if impending arrest
Esophageal perforation (0.1-0.5%)May be immediate (recognized intraoperatively) or delayed (fever, neck swelling, mediastinitis)Careful medial retraction, avoid sharp instruments near esophagus, recognize anatomy, gentle tissue handlingIf recognized early: primary repair, NPO, antibiotics. Delayed: often requires drainage, antibiotics, may need flap coverage, high mortality if missed
Vertebral artery injury (0.3-0.5%)Massive arterial bleeding during foraminotomy or lateral decompressionStay within 14mm of midline during foraminotomy, know anomalous artery patterns, careful burringDirect pressure, pack with hemostatic agent, may need interventional radiology for embolization, accept some stenosis rather than repair
Spinal cord injury (&lt;0.5%)Immediate motor/sensory loss, worsened myelopathy post-operativelyControlled decompression, neuromonitoring, avoid instruments posterior to PLL, careful techniqueIf intraoperative neuromonitoring change: stop, check position, consider steroids. Post-op: MRI urgently, may need revision if compression
Dural tear/CSF leak (0.5-1%)Clear fluid in wound, headache with upright position, meningitis signs if infectedCareful PLL removal, recognize dura (blue-grey, pulsating), controlled techniquePrimary repair if possible (patch with muscle/fat, fibrin glue), watertight closure, may need lumbar drain, bedrest if persistent
Horner syndrome (0.1-1%)Ptosis, miosis, anhidrosis on operative side immediately post-opElevate longus colli subperiosteally, place retractor blades UNDER longus colli, avoid lateral dissectionUsually temporary (resolves weeks-months), rarely permanent, patient education and reassurance
Cage subsidence/failure (5-10%)Loss of disc height on follow-up X-rays, recurrent symptoms, kyphosisPreserve endplate integrity, appropriate cage sizing, plate fixation, smoking cessationIf asymptomatic: observe with imaging. If symptomatic or kyphotic: revision surgery with larger cage or corpectomy
Adjacent segment disease (2-3%/year)New radiculopathy or myelopathy at level above or below fusion, years after index surgeryControversial if preventable - may use disc arthroplasty in selected patients, maintain lordosis, avoid long fusionsConservative management first, if fails: extend fusion or decompression, consider arthroplasty at new level

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

EXAMINER

"You are performing a single-level ACDF at C5-C6 on a 45-year-old with radiculopathy. During closure, the anaesthetist mentions the patient is 'a bit stridorous' after extubation. What is your immediate management?"

EXCEPTIONAL ANSWER
This is a potentially life-threatening emergency that requires immediate attention. Stridor post-ACDF suggests airway compromise, most likely from retropharyngeal hematoma, which can progress rapidly to complete airway obstruction. My immediate management would be: First, I would call for senior help and notify the anaesthetic team that this may be an airway emergency. I would ask for an emergency airway trolley and ensure difficult airway equipment is available. I would assess the patient's airway by examining the neck for swelling or bruising, checking voice quality, and assessing respiratory distress. If the patient is maintaining their airway but stridorous, I would keep them calm, upright if possible, and apply supplemental oxygen. If the stridor is progressive or the patient is deteriorating, I would prepare for emergent intubation. However, intubation may be extremely difficult due to hematoma distorting the airway. I would have ENT surgery on standby and prepare for emergency cricothyroidotomy or tracheostomy if intubation fails. In parallel, I would prepare to return to the operating theatre immediately for surgical evacuation. If the patient is deteriorating rapidly and cannot be intubated, I would perform bedside wound evacuation - opening the skin and platysma to release the hematoma and decompress the airway - this can be life-saving even if not in the OR. Once the airway is secured, I would return to theatre for formal wound exploration, evacuation of hematoma, identification and control of the bleeding source, thorough irrigation, and consideration of drain placement before re-closure. The key teaching point is that retropharyngeal hematoma post-ACDF is a surgical emergency. Do not delay intervention hoping it will improve - these can progress from stridor to complete obstruction within minutes. Have a low threshold for re-exploration.
KEY POINTS TO SCORE
Stridor post-ACDF suggests retropharyngeal hematoma - potentially life-threatening emergency
Call for help immediately - senior surgeon, anaesthetics, ENT if available
Prepare for difficult airway - hematoma distorts normal anatomy
If deteriorating rapidly, consider bedside wound evacuation to decompress airway
Return to OR for formal exploration, evacuation, and bleeding source identification
COMMON TRAPS
✗Waiting and observing in a deteriorating patient (can progress to complete obstruction quickly)
✗Assuming intubation will be straightforward (hematoma makes airway very difficult)
✗Not having a backup plan if intubation fails (cricothyroidotomy/tracheostomy)
✗Forgetting that bedside evacuation can be life-saving in impending arrest
LIKELY FOLLOW-UPS
"If you are called at 2am post-operatively and the nurse says the patient has developed progressive stridor, what instructions would you give over the phone while coming in?"
VIVA SCENARIOStandard

EXAMINER

"A patient wakes up from ACDF with a hoarse voice. How do you assess and counsel this patient?"

EXCEPTIONAL ANSWER
Hoarseness immediately post-ACDF suggests recurrent laryngeal nerve dysfunction. This is the most common nerve injury following ACDF, occurring in 2-11% of cases. My assessment and counselling would be systematic. First, I would document the voice change, including severity and whether they can phonate at all. I would ask about cough strength (weak cough suggests aspiration risk) and any swallowing difficulties. I would examine for stridor or respiratory distress, which would suggest more severe bilateral injury or airway compromise. Importantly, the vast majority of RLN injuries post-ACDF are temporary neuropraxia from retraction rather than transection. Approximately 90-95% recover fully within 6-12 weeks as the nerve recovers from the traction insult. For counselling, I would explain that temporary voice changes are common after this surgery, occurring in up to 1 in 10 patients. I would reassure them that this is usually due to nerve stretching from retractors rather than permanent damage, and that recovery is expected over the coming weeks to months. I would advise them to avoid straining their voice and that we will monitor their recovery. I would arrange ENT referral for laryngoscopy to document vocal cord function - this serves as a baseline and determines if the cord is paretic versus paralyzed. If there is concern about aspiration (weak cough, choking with liquids), I would request a speech pathology swallowing assessment. I would schedule follow-up at 6 weeks and 3 months to document recovery. If hoarseness persists beyond 6 months with confirmed vocal cord paralysis on laryngoscopy, I would refer to ENT for consideration of medialization procedures (injection laryngoplasty or thyroplasty). I would also document this complication thoroughly in the medical record, including the discussion with the patient, and notify my consultant.
KEY POINTS TO SCORE
RLN injury occurs in 2-11% of ACDF cases, vast majority (90-95%) temporary neuropraxia
Document severity, assess cough strength, rule out bilateral injury/airway compromise
Arrange ENT referral for laryngoscopy to document vocal cord function as baseline
Reassure patient that recovery expected over 6-12 weeks for most cases
If persistent beyond 6 months with confirmed paralysis, ENT may offer medialization procedures
COMMON TRAPS
✗Dismissing the symptom without proper documentation and follow-up
✗Not assessing for aspiration risk (weak cough, choking)
✗Forgetting to document and communicate with the patient about this complication
✗Not arranging appropriate specialist follow-up (ENT, speech pathology)
LIKELY FOLLOW-UPS
"If laryngoscopy shows left vocal cord paralysis and the voice hasn't recovered at 6 months, what surgical options might ENT offer?"
VIVA SCENARIOStandard

EXAMINER

"Why do many surgeons prefer a left-sided approach for ACDF, and in what situations would you consider a right-sided approach?"

EXCEPTIONAL ANSWER
The preference for a left-sided approach to ACDF relates primarily to the anatomy of the recurrent laryngeal nerve, though other factors also influence the decision. The left recurrent laryngeal nerve has a more predictable course. It loops under the aortic arch in the chest and then ascends in the tracheoesophageal groove to reach the larynx. This consistent path means the nerve is reliably located in the groove between the trachea and esophagus throughout the cervical approach. The right recurrent laryngeal nerve has a more variable course. It loops under the subclavian artery, which is higher in the neck than the aortic arch, resulting in a shorter recurrent course. More importantly, the right RLN may take a more lateral path before entering the tracheoesophageal groove, potentially placing it more at risk during the lateral retraction required for exposure. Some studies have suggested a higher rate of RLN injury with right-sided approaches, though this remains controversial, and other studies show no significant difference. I would consider a right-sided approach in the following situations: First, if the patient has had previous left-sided anterior cervical surgery, I would use the right side to avoid scar tissue and the potentially already-injured left RLN. A second injury to the same nerve could convert hoarseness to complete paralysis, and bilateral RLN injury causes acute airway obstruction requiring tracheostomy. Second, if pre-operative laryngoscopy shows pre-existing left vocal cord paralysis from any cause, I would approach from the right to protect the functioning right cord. Third, if the pathology is predominantly right-sided (right-sided disc herniation or right foraminal stenosis), some surgeons prefer approaching from the side of the pathology for better visualization of the nerve root. Fourth, for surgeons who are experienced and comfortable with right-sided approaches, there may be no meaningful difference in outcomes. Regardless of approach side, the key protective measures remain the same: careful retraction, periodic release of retractors, and meticulous surgical technique.
KEY POINTS TO SCORE
Left RLN has predictable course: loops under aortic arch, ascends in tracheoesophageal groove
Right RLN more variable: loops under subclavian (higher), may take more lateral path
Left side preferred by many surgeons for this anatomic predictability
Right side indicated if: previous left surgery, pre-existing left cord paralysis, or right-sided pathology
Bilateral RLN injury catastrophic (airway obstruction) - avoid same side if previous injury
COMMON TRAPS
✗Stating definitively that right side has higher injury rate (evidence is mixed)
✗Not knowing when to use the right side (previous surgery, pre-existing paralysis)
✗Forgetting that bilateral injury causes airway obstruction requiring tracheostomy
✗Not mentioning that technique and retraction are more important than side
LIKELY FOLLOW-UPS
"If a patient had previous right-sided anterior cervical surgery and now needs ACDF at a different level, what special precautions would you take?"

Anterior Cervical Discectomy and Fusion (ACDF) - Exam Summary

High-Yield Exam Summary

Key Numbers

  • •Surface landmarks: C2-C3 mandible angle, C3-C4 hyoid, C4-C5 thyroid, C6 cricoid (most reliable)
  • •Vertebral artery: 14-18mm from midline (stay within this during foraminotomy)
  • •RLN injury rate: 2-11% total, 90-95% temporary neuropraxia, &lt;1% permanent
  • •Dysphagia: 50% at 1 week, 20% at 1 month, 5% persistent at 1 year
  • •Fusion rate: 95%+ with plate (reduces non-union from 10% to 5%)

Critical Anatomy

  • •Smith-Robinson interval: avascular plane between SCM/carotid laterally and strap muscles/trachea/esophagus medially
  • •Left RLN: loops under aortic arch, predictable course in tracheoesophageal groove (preferred approach)
  • •Right RLN: loops under subclavian, more variable, may be more lateral
  • •Sympathetic chain: under lateral longus colli - elevate subperiosteally and place retractor blades UNDER
  • •No epidural space anteriorly in cervical spine - dura immediately posterior to PLL

Wrong-Level Prevention

  • •Surface landmarks as INITIAL guide only (can be unreliable)
  • •Spinal needle in disc under fluoroscopy before incision
  • •MUST obtain LATERAL image to confirm level (AP insufficient)
  • •Second confirmation after retractors placed recommended
  • •Wrong-level surgery is a 'never event' - document confirmation

Emergencies

  • •Retropharyngeal hematoma: stridor, dyspnea, neck swelling - EMERGENCY, prepare for difficult airway, may need bedside evacuation
  • •Bilateral RLN injury: complete airway obstruction, requires tracheostomy
  • •Vertebral artery injury: massive bleeding during foraminotomy, pack with hemostatic agent, may need IR embolization
  • •Esophageal perforation: delayed presentation with fever, sepsis - high mortality if missed

Technique Essentials

  • •Left-sided approach preferred (predictable RLN anatomy)
  • •Elevate longus colli subperiosteally, retractor blades UNDER (protects sympathetic chain)
  • •Release retractors periodically (reduces RLN neuropraxia, dysphagia)
  • •Complete discectomy to PLL, then remove PLL to see dura (no epidural space anteriorly)
  • •Foraminotomy: stay within 14mm of midline (vertebral artery beyond)
  • •Screw trajectory: 12-15 degrees convergent (midline) for best bone purchase

References

  1. Smith GW, Robinson RA. The treatment of certain cervical-spine disorders by anterior removal of the intervertebral disc and interbody fusion. J Bone Joint Surg Am. 1958;40-A(3):607-624.

  2. Fountas KN, Kapsalaki EZ, Nikolakakos LG, et al. Anterior cervical discectomy and fusion associated complications. Spine. 2007;32(21):2310-2317.

  3. Bazaz R, Lee MJ, Yoo JU. Incidence of dysphagia after anterior cervical spine surgery: a prospective study. Spine. 2002;27(22):2453-2458.

  4. Netterville JL, Koriwchak MJ, Winkle M, et al. Vocal fold paralysis following the anterior approach to the cervical spine. Ann Otol Rhinol Laryngol. 1996;105(2):85-91.

  5. Hilibrand AS, Robbins M. Adjacent segment degeneration and adjacent segment disease: the consequences of spinal fusion? Spine J. 2004;4(6 Suppl):190S-194S.

  6. Kaiser MG, Haid RW Jr, Subach BR, et al. Anterior cervical plating enhances arthrodesis after discectomy and fusion with cortical allograft. Neurosurgery. 2002;50(2):229-238.

  7. Winslow CP, Winslow TJ, Wax MK. Dysphonia and dysphagia following the anterior approach to the cervical spine. Arch Otolaryngol Head Neck Surg. 2001;127(1):51-55.

  8. Apfelbaum RI, Kriskovich MD, Haller JR. On the incidence, cause, and prevention of recurrent laryngeal nerve palsies during anterior cervical spine surgery. Spine. 2000;25(22):2906-2912.

  9. Fraser JF, Härtl R. Anterior approaches to fusion of the cervical spine: a metaanalysis of fusion rates. J Neurosurg Spine. 2007;6(4):298-303.

  10. Epstein NE. Airway complications of multilevel anterior cervical surgery. J Spinal Disord. 2001;14(2):135-140.

Quick Stats
Complexityintermediate
Reading Time50 min
Updated2025-12-25
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