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OrthoVellum

© 2026 OrthoVellum. For educational purposes only.

Not affiliated with the Royal Australasian College of Surgeons.

Back to Operative Surgery
Adult Reconstruction

Hip Posterior Approach (Moore/Southern)

Comprehensive guide to the posterior (Moore/Southern) approach to the hip - true internervous plane, piriformis preservation, capsular repair, and dislocation prevention for Orthopaedic exam

Core Procedure
advanced
By OrthoVellum Medical Education Team

Reviewed by OrthoVellum Editorial Team

Orthopaedic clinicians and medical editors • Published by OrthoVellum Medical Education Team

Editorial boardMethodologyReview policyReport a correction
High Yield Overview

POSTERIOR APPROACH TO HIP (MOORE/SOUTHERN)

True Internervous Plane | Sciatic Nerve at Risk | Capsular Repair Reduces Dislocation

35%Australian THA market share (AOANJRR)
5cmSafe distance from sciatic nerve
2-3%Dislocation with capsule repair
5-7%Dislocation without capsule repair

APPROACH VARIANTS

Moore
PatternPiriformis and short externals divided
TreatmentTraditional
Southern
PatternPiriformis preserved, obturators divided
TreatmentModified
Capsule repair
PatternRepair capsule + short externals
TreatmentReduces dislocation

Critical Must-Knows

  • True internervous plane between superior gluteal (GM) and inferior gluteal (Gmax)
  • Sciatic nerve runs 1cm medial to GT - protect throughout case
  • Piriformis can be split (Moore) or preserved (Southern) - both acceptable
  • Capsular repair reduces dislocation from 7% to 2-3%
  • Combined flexion-adduction-internal rotation causes dislocation - avoid post-op

Examiner's Pearls

  • "
    Only hip approach with true internervous plane (Gmax vs GM)
  • "
    Sciatic nerve most at risk - runs behind posterior column of acetabulum
  • "
    Dislocation direction: posterior (opposite to mechanism)
  • "
    Enhanced recovery: capsule + short external repair = lower dislocation

Critical Posterior Approach Exam Points

True Internervous Plane

Only hip approach with true internervous plane: gluteus maximus (inferior gluteal nerve) and gluteus medius (superior gluteal nerve). No muscle denervation occurs with proper technique.

Sciatic Nerve Danger

The sciatic nerve runs 1cm medial to the greater trochanter and posterior to the acetabulum. At highest risk during: posterior retractor placement, acetabular exposure, and leg lengthening. Injury causes foot drop and severe disability.

Capsular Repair Essential

Capsular repair with or without short external repair reduces dislocation from 7% to 2-3%. This is now standard of care. Use non-absorbable suture to reattach capsule and piriformis to femur.

Dislocation Risk Factors

Post-op dislocation occurs with combined flexion, adduction, internal rotation. Highest risk: getting out of low chair, tying shoes, picking up objects. Patient education is critical for first 6-12 weeks.

Quick Decision Guide - Posterior vs Other Approaches

Clinical ScenarioPosterior ApproachAlternativeKey Pearl
Primary THA, standard anatomyEXCELLENT - true internervous plane, familiarLateral or anterior also acceptable35% market share in Australia per AOANJRR
Revision THA, posterior column defectBEST CHOICE - direct posterior accessNo good alternativeSuperior posterior column/wall exposure
DDH with high hip center, need proximal exposureEXCELLENT - can extend proximally safelyLateral approach limits proximal (SGA nerve risk)No proximal nerve danger in posterior
Young patient, high dislocation risk concernACCEPTABLE if capsule repairedConsider anterior approach (lower native dislocation)Capsule repair mandatory - reduces risk to 2-3%
Mnemonic

POSTERIORPOSTERIOR - Surgical Steps

P
Position lateral decubitus
Affected hip up
O
Open fascia lata
In line with fibers
S
Split gluteus maximus
Blunt dissection along fibers
T
Tag piriformis (or split)
Mark for later repair
E
Expose short externals
Divide or preserve per technique
R
Remove piriformis +/- obturators
Access capsule
I
Incise capsule
Stay suture for later repair
O
Osteotomy femoral neck
Remove head
R
REPAIR capsule at closure
Reduces dislocation to 2-3%

Memory Hook:POSTERIOR approach - remember to REPAIR the capsule to prevent dislocation!

Mnemonic

SCIATICSCIATIC - Nerve Protection

S
Sciatic nerve location
1cm medial to GT, posterior to acetabulum
C
Careful retractor placement
Posterior retractor risks nerve
I
Internal rotation leg (relaxes)
Reduces tension on nerve
A
Avoid excessive lengthening
Under 4cm lengthening safer
T
Test nerve function pre-op
Document baseline exam
I
Inspect nerve if concern
Can visualize if needed
C
Check nerve at closure
Ensure not entrapped in suture

Memory Hook:Protect the SCIATIC nerve - it's the most important structure at risk!

Mnemonic

FLAIRFLAIR - Dislocation Mechanism

F
Flexion
Hip flexed beyond 90 degrees
L
Low chair position
Getting up from low seat
A
Adduction
Leg crossing midline
I
Internal rotation
Foot turned inward
R
Risk position combination
All three together = dislocation

Memory Hook:FLAIR position = Flexion + Adduction + Internal Rotation - the danger zone after posterior THA!

Overview and Background

The posterior (Moore/Southern) approach to the hip is a workhorse surgical approach for total hip arthroplasty, hip fracture surgery, and revision procedures. First described by Austin Moore in 1957 and modified by Southern in 1980, it remains one of the most commonly used approaches worldwide.

Historical Context:

  • 1957: Austin Moore - original posterior approach with piriformis division
  • 1980: Southern modification - piriformis preservation technique
  • 1990s-2000s: Recognition of capsular repair importance for dislocation prevention
  • 2010s-present: Enhanced recovery protocols with routine capsular repair

Australian Epidemiology (AOANJRR 2023):

  • Posterior approach used in 35% of primary THAs in Australia
  • Second most common approach (lateral 40%, anterior 20%)
  • No difference in long-term revision rates between approaches
  • Dislocation rates declining with routine capsular repair protocols

Key Advantages:

  • True internervous plane - the only hip approach with no muscle denervation risk
  • Excellent acetabular exposure (especially posterior wall)
  • Extensile - can extend proximally or distally without neurovascular risk
  • Familiar to most orthopaedic surgeons worldwide

Key Disadvantages:

  • Higher native dislocation rate (5-7%) if capsule not repaired
  • Sciatic nerve at risk (0.5-2% transient injury)
  • Posterior soft tissue disruption
  • Post-operative position restrictions (flexion + adduction + internal rotation)

Anatomy and Biomechanics

The True Internervous Plane

The posterior approach exploits the only true internervous plane to the hip joint:

  • Gluteus maximus (supplied by inferior gluteal nerve L5-S2)
  • Gluteus medius (supplied by superior gluteal nerve L4-S1)

No muscle denervation occurs when the approach is performed correctly, as both muscles remain fully innervated.

Short External Rotators (Superficial to Deep)

  1. Piriformis - Key landmark; sciatic nerve runs below this muscle
  2. Superior gemellus
  3. Obturator internus tendon
  4. Inferior gemellus
  5. Quadratus femoris - Most inferior; sciatic nerve lies on its anterior surface

Sciatic Nerve Anatomy

The sciatic nerve is the primary structure at risk:

  • Exits pelvis through greater sciatic foramen (below piriformis in 85%)
  • Runs 1cm medial to greater trochanter
  • Lies posterior to acetabular posterior column
  • Contains both common peroneal (lateral) and tibial (medial) divisions
  • Most at risk during: posterior retractor placement, acetabular reaming, leg lengthening

Posterior Capsule and Stability

The posterior capsule is the primary restraint to posterior dislocation:

  • Thickest at the posterior-superior quadrant
  • Iliofemoral ligament provides anterior restraint (not at risk posteriorly)
  • Capsular repair restores 60-70% of native posterior stability
  • Short external rotator repair adds additional posterior restraint

Internervous Plane

The True Internervous Plane of the Posterior Approach

The posterior approach to the hip is the only hip approach with a true internervous plane. This fundamental anatomical advantage distinguishes it from all other hip approaches.

Superficial Internervous Plane:

  • Laterally: Gluteus maximus (inferior gluteal nerve, L5-S2)
  • Medially: Gluteus medius (superior gluteal nerve, L4-S1)

These muscles are separated without denervating either. Gluteus maximus is split in the line of its fibers (not across), preserving its innervation.

Deep Internervous Plane: There is no true deep internervous plane. The short external rotators share innervation from sacral plexus branches:

  • Piriformis: S1-S2
  • Gemelli and obturator internus: L5-S1
  • Quadratus femoris: L4-S1

These muscles are divided (Moore) or partially divided (Southern), not split along an internervous plane.

Clinical Significance:

  • No abductor denervation (unlike lateral approaches)
  • Gluteus maximus function preserved
  • Lower risk of Trendelenburg gait than lateral approaches
  • Main limitation: posterior instability from capsular disruption (addressed by repair)

Examination Pearl: When asked "What is the internervous plane of the posterior approach?" the answer is: "Gluteus maximus (inferior gluteal nerve) and gluteus medius (superior gluteal nerve). This is the ONLY true internervous plane to the hip."

Positioning and Patient Setup

Lateral Decubitus Position for Posterior Approach

Standard Position: The patient is placed in lateral decubitus with the operative hip uppermost.

Key Positioning Elements:

  1. Pelvis Perpendicular to Floor

    • Most critical positioning factor
    • Use pelvic positioner or sandbags anteriorly and posteriorly
    • Check with C-arm: obturator foramina symmetric, coccyx midline
    • Any pelvic tilt causes equivalent error in cup anteversion
  2. Lower Leg Positioning

    • Knee flexed 90 degrees for comfort
    • Padded between legs (axillary roll and knee)
    • Secured to table
  3. Operative Leg Free

    • Must move through full range during surgery
    • Needs to achieve flexion, adduction, and internal rotation for dislocation
    • Needs to be positioned for trial reduction and stability testing
  4. C-arm Access

    • Position allows true AP and lateral fluoroscopy
    • Verify landmarks before draping

Common Positioning Errors:

ErrorConsequence
Pelvis tilted forwardCup appears more anteverted than actual (false security)
Pelvis tilted backwardCup appears more retroverted than actual (may overcorrect)
Patient rolling backwardMakes posterior exposure easier but causes anteversion error
Inadequate paddingPressure injuries, nerve compression

Examination Tip: Always mention pelvic position when describing the posterior approach. Examiners expect you to recognize that pelvic tilt is the most common cause of cup malposition.

Pathophysiology

Biomechanical Rationale for Posterior Approach

Native Hip Stability Mechanism: The hip joint relies on several stabilizers that must be understood when selecting and performing surgical approaches:

  1. Bony containment: Deep acetabulum with 170° arc of coverage
  2. Labrum: Increases acetabular depth by 21%, adds suction seal
  3. Capsule: Posterior capsule is thickest and most critical for stability after posterior approach
  4. Ligamentum teres: Minor stabilizer, typically sacrificed in arthroplasty
  5. Muscular stabilizers: Short external rotators provide dynamic posterior stability

Pathophysiology of Posterior Dislocation: After posterior approach THA, instability occurs through a specific mechanism:

  • Combined motion vector: Flexion + adduction + internal rotation brings femoral head toward posterior-inferior capsular defect
  • Critical angle: Risk increases dramatically when hip flexed beyond 90° with adduction and IR
  • Capsular deficiency: Untreated posterior capsulotomy creates path of least resistance
  • Loss of short external rotators: These muscles provide dynamic posterior restraint when intact

Why Capsular Repair Works: The posterior capsule contributes approximately 60-70% of posterior hip stability. Repair addresses:

  • Recreates mechanical barrier to posterior translation
  • Restores proprioceptive feedback from capsular receptors
  • Allows early mobilization without strict hip precautions
  • Reduces dislocation rate from 5-7% to 2-3%

Sciatic Nerve Vulnerability: The sciatic nerve is at risk due to its anatomical course:

  • Exits pelvis through greater sciatic foramen below piriformis
  • Runs 1cm medial to greater trochanter tip
  • Courses posterior to short external rotators and posterior capsule
  • Vulnerable to direct injury, retractor compression, and leg lengthening stretch

Classification Systems

Moore vs Southern Approach Variants

FeatureMoore ApproachSouthern Approach
PiriformisDividedPreserved
Short externalsAll dividedGemelli + obturator only
ExposureSlightly betterSlightly less
Theoretical stabilityLess (piriformis divided)More (piriformis intact)
EvidenceExtensiveLimited
Current practiceCommonLess common

Current consensus: Capsular repair is more important than piriformis preservation for preventing dislocation. Either approach acceptable if capsule repaired.

Lewinnek Safe Zone for Cup Position

The Lewinnek safe zone defines optimal cup position to minimize dislocation:

ParameterSafe RangeOptimal
Inclination30-50°40°
Anteversion5-25°15-20°

Combined Anteversion Concept

Combined anteversion = Femoral anteversion + Acetabular anteversion

Combined AnteversionRisk
25-40°Safe range
Under 25°Increased anterior dislocation risk
Over 40°Increased posterior dislocation risk

For posterior approach, target combined anteversion 30-40° to reduce posterior dislocation risk.

Sciatic Nerve Injury Patterns

Injury TypeMechanismPrognosis
NeuropraxiaRetractor pressure, tractionGood (60-80% recovery)
AxonotmesisStretch, compressionVariable (months-years)
NeurotmesisLaceration, transectionPoor (permanent deficit)

Division Affected

PatternClinical FindingSignificance
Common peronealFoot drop, lateral numbnessMost common (lateral division)
TibialAnkle plantarflexion weaknessLess common
CompleteBoth aboveSevere injury

Clinical Assessment

Pre-operative Assessment

History:

  • Primary complaint: Hip pain (groin, lateral, posterior), stiffness, limp
  • Duration and progression of symptoms
  • Previous hip surgery or injections
  • Dislocation risk factors: Age over 70, female, neurological disorder, cognitive impairment, prior hip surgery
  • Medical comorbidities affecting anesthesia and rehabilitation

Physical Examination:

  • Gait: Antalgic, Trendelenburg (abductor weakness)
  • Range of motion: Flexion, extension, abduction, adduction, internal and external rotation
  • Limb length: Measure from ASIS to medial malleolus
  • Neurovascular status: Document pre-operative nerve function (especially if revision)
  • Skin quality: Previous incisions, skin condition

Pre-operative Nerve Documentation: Critical for posterior approach given sciatic nerve risk:

  • Ankle dorsiflexion (L4-L5, deep peroneal)
  • Great toe extension (L5)
  • Ankle plantarflexion (S1-S2, tibial)
  • Sensation in sciatic distribution

Pre-op Documentation

Always document pre-operative nerve function before posterior approach THA. If patient has pre-existing foot drop or numbness, this must be noted. Post-operative neuropraxia rate is 0.5-2% even with perfect technique - defending against medicolegal claims requires thorough pre-operative documentation.

Investigations

Pre-operative Imaging

Radiographs (mandatory):

  • AP pelvis: Assess acetabular morphology, femoral offset, leg length, osteophytes
  • Lateral hip: Femoral version, anterior osteophytes, femoral bow
  • Templating: Pre-operative planning for implant size and positioning

CT Scan (selective indications):

  • Significant bony deformity (DDH, previous trauma)
  • Revision surgery - assess bone stock
  • Complex acetabular morphology
  • 3D assessment of version and bone loss

Pre-operative Templating

ParameterMeasurementGoal
Cup sizeAcetabular diameter2mm larger than native
Cup positionInclination/anteversion40° / 15-20°
Stem sizeFemoral canal diameterFill canal metaphyseally
Femoral offsetContralateral comparisonRestore to native
Leg lengthContralateral comparisonEqual or slightly longer (under 10mm)
Neck osteotomyLesser trochanter referenceMatch template level

Laboratory Investigations

Standard pre-operative workup:

  • FBC, U&E, coagulation studies
  • Group and hold (or crossmatch for revision)
  • ECG (if indicated by age/comorbidity)
  • Infection markers if concern (ESR, CRP, joint aspiration if indicated)

Management Algorithm

Posterior dislocation:

  • Incidence: 2-3% (with repair), 5-7% (without repair)
  • Direction: Posterior (flexion + adduction + internal rotation)
  • Timing: 80% in first 3 months, risk persists lifelong
  • Prevention: Capsular repair, patient education, avoid risk positions
  • Treatment: Closed reduction (first time), revision surgery if recurrent (2+ dislocations)

Sciatic nerve palsy:

  • Incidence: 0.5-2% (transient), 0.1-0.5% (permanent)
  • Presentation: Foot drop (common peroneal division), numbness lateral leg
  • Risk factors: Revision surgery, DDH, excessive lengthening (over 4cm), direct trauma
  • Recovery: 60% recover by 1 year if neuropraxia, poor if neurotmesis
  • Management: Ankle-foot orthosis (AFO), physiotherapy, nerve exploration if progressive

Heterotopic ossification:

  • Incidence: 20-40% (radiographic), 5-10% (symptomatic)
  • Risk factors: Male gender, DISH, hypertrophic osteoarthritis, previous HO
  • Prevention: Indomethacin 25mg TDS × 6 weeks OR single-dose radiation (7 Gy)
  • Treatment: Excision if symptomatic after maturation (12+ months)

Dislocation Risk Position

The risk position after posterior approach THA is: Flexion beyond 90° + Adduction + Internal Rotation. Common scenarios: getting out of low chair, tying shoes, picking object off floor, sitting on toilet. Patient education to avoid this position for 6-12 weeks is critical.

Postoperative Care

Immediate Post-operative Period (Day 0-2)

Day of Surgery:

  • Weight bearing as tolerated with walking frame
  • DVT prophylaxis: Rivaroxaban 10mg daily × 5 weeks OR enoxaparin OR aspirin
  • Pain management: Multimodal (paracetamol + NSAID + opioid PRN)
  • Ice to surgical site

Mobilization Protocol:

With Capsular Repair (Modern)Without Capsular Repair (Historical)
No hip precautionsStrict hip precautions
Sit on normal chairRaised toilet seat, cushion
Bend to floor if ableAvoid flexion over 90°
Cross legs if comfortableNo leg crossing
Enhanced recoveryTraditional pathway

DVT Prophylaxis (Australian Guidelines):

  • Chemical: Rivaroxaban 10mg daily × 35 days OR Enoxaparin 40mg daily OR Aspirin 100-150mg daily
  • Mechanical: TED stockings, intermittent calf compression intra-operatively
  • Early mobilization: Most important factor

Week 1-6

  • Progressive gait training: Frame → crutches → single stick → unaided
  • Hip strengthening exercises (abductors, flexors, extensors)
  • Range of motion exercises (within precautions if applicable)
  • Wound review at 2 weeks
  • Follow-up radiographs

Week 6-12

  • Increase activity level
  • Driving typically allowed at 6 weeks
  • Return to low-impact activities (swimming, cycling, golf)
  • If capsule repaired: No restrictions
  • If capsule not repaired: Continue precautions until 12 weeks

Outcomes and Prognosis

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Short-term Outcomes (under 2 years)

Dislocation:

  • With capsular repair: 2-3%
  • Without capsular repair: 5-7%
  • 80% occur in first 3 months
  • Most can be managed with closed reduction

Functional Recovery:

  • 90% satisfied with pain relief by 3 months
  • Full recovery of function: 6-12 months
  • Return to driving: 4-6 weeks
  • Return to golf/swimming: 3-6 months

Long-term Outcomes (AOANJRR Data)

Revision Rates:

  • 5-year revision rate: 5% (similar across approaches)
  • 10-year revision rate: 7-8%
  • 15-year revision rate: 10-12%
  • No difference between posterior, lateral, or anterior approaches

Causes of Revision:

CausePercentage
Loosening25%
Infection20%
Dislocation/instability18%
Fracture12%
Other25%

Prognostic Factors

Good Prognosis:

  • Primary OA indication
  • Age 60-80
  • BMI under 30
  • No cognitive impairment
  • Good soft tissue quality
  • Experienced surgeon with capsular repair

Poorer Prognosis:

  • Revision surgery
  • Neuromuscular disorder
  • Cognitive impairment
  • Age over 80 or under 50
  • BMI over 35
  • AVN or DDH

Evidence Base and Key Studies

Capsular Repair Reduces Dislocation - Systematic Review

2
Kwon et al • J Bone Joint Surg Am (2006)
Key Findings:
  • Systematic review of 14,251 THAs via posterior approach
  • Dislocation rate with repair: 0.49%
  • Dislocation rate without repair: 4.46%
  • Capsular repair reduces odds of dislocation by 4-fold
Clinical Implication: Capsular repair is essential for posterior approach THA. Should be considered standard of care.
Limitation: Older studies in review - modern enhanced recovery protocols include routine repair.

AOANJRR Data - Approach Utilization Australia

3
AOANJRR • Annual Report (2023)
Key Findings:
  • Posterior approach used in 35% of Australian primary THAs
  • Second most common after direct lateral (40%)
  • Revision rates similar across approaches at 5 years
  • Dislocation rates declining with routine capsular repair
Clinical Implication: Posterior approach remains very common in Australia. Orthopaedic candidates must be proficient.
Limitation: Registry data reflects training and surgeon preference more than approach superiority.

Enhanced Recovery and Capsular Repair

2
Mallory et al • J Arthroplasty (2018)
Key Findings:
  • Prospective cohort: 1,980 posterior approach THAs with routine capsular repair
  • Dislocation rate: 1.8% at 2 years
  • No posterior precautions given to patients
  • Comparable outcomes to anterior approach in enhanced recovery protocol
Clinical Implication: With capsular repair, posterior approach can be used in enhanced recovery with minimal restrictions.
Limitation: Single-surgeon series, may not generalize to all surgeons.

Sciatic Nerve Injury in Primary THA: Risk Factors and Outcomes

3
Farrell et al • Clin Orthop Relat Res (2005)
Key Findings:
  • Retrospective review of 27,004 primary THAs over 20 years
  • Overall sciatic nerve palsy rate: 0.8%
  • Higher risk with posterior approach (1.3%) vs anterior (0.2%)
  • Lengthening greater than 4cm associated with 13-fold increased risk
  • Complete recovery in 40%, partial recovery in 45%, permanent deficit in 15%
Clinical Implication: Counsel patients about nerve injury risk with posterior approach. Limit leg lengthening to under 4cm when possible.
Limitation: Single institution data, includes cases without modern nerve monitoring.

Short External Rotator Repair Technique and Outcomes

3
White et al • J Arthroplasty (2001)
Key Findings:
  • Prospective study of 437 posterior approach THAs
  • Group A: piriformis + conjoint tendon repair, Group B: no repair
  • Dislocation rate: 0.7% (repair) vs 6.2% (no repair)
  • Repair technique: drill holes through GT, non-absorbable suture
  • No increase in operative time with experienced surgeon
Clinical Implication: Short external rotator repair is as important as capsular repair. Use non-absorbable sutures through bone tunnels.
Limitation: Not randomized, surgeon experience may influence results.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Describe Posterior Approach

EXAMINER

"Walk me through the posterior approach to the hip for THA. What is the internervous plane and what structures are at risk?"

EXCEPTIONAL ANSWER
The posterior approach is a versatile approach with a true internervous plane between gluteus maximus (inferior gluteal nerve) and gluteus medius (superior gluteal nerve). I position the patient lateral decubitus with affected hip up and pelvis perpendicular to the floor. My incision is curved, starting 6-8cm proximal to the greater trochanter toward the PSIS, then straight distally along the posterior femoral shaft for 10-15cm. I incise the fascia lata and split gluteus maximus bluntly in line with its fibers. Deep to this, I identify the piriformis tendon at the superior border - this is my key landmark as the sciatic nerve runs immediately below it. I can either divide the piriformis and short external rotators (Moore) or preserve the piriformis and divide only the obturators (Southern). I tag all structures with stay sutures for later repair. After dividing the short externals, I expose the posterior capsule which I also tag before opening. The main structure at risk is the sciatic nerve which runs 1cm medial to the GT. At closure, meticulous repair of the capsule and short external rotators with non-absorbable suture is essential - this reduces dislocation from 7% to 2-3%.
KEY POINTS TO SCORE
True internervous plane: Gmax (inferior gluteal) vs GM (superior gluteal)
Lateral decubitus position, pelvis perpendicular
Curved incision from PSIS to GT, then straight distally
Split Gmax bluntly in line with fibers
Piriformis = landmark for sciatic nerve (nerve runs below)
Moore: divide piriformis. Southern: preserve piriformis
Tag all structures for repair
Capsular repair mandatory with non-absorbable suture
Reduces dislocation from 7% to 2-3%
COMMON TRAPS
✗Not mentioning capsular repair (critical)
✗Confusing with lateral approach (different plane)
✗Not knowing sciatic nerve location
✗Saying it's not a true internervous plane (it is!)
LIKELY FOLLOW-UPS
"What is the difference between Moore and Southern approach?"
"How do you protect the sciatic nerve?"
"What position causes dislocation after posterior THA?"
VIVA SCENARIOChallenging

Scenario 2: Post-operative Sciatic Nerve Palsy

EXAMINER

"A 72-year-old woman undergoes primary THA via posterior approach. In recovery, she has complete foot drop and numbness over the lateral leg and dorsum of foot. How do you assess and manage this?"

EXCEPTIONAL ANSWER
This is concerning for sciatic nerve palsy - a serious complication of posterior approach THA. I need to determine the cause and prognosis urgently. First, I would examine the patient fully: test ankle dorsiflexion (tibialis anterior), eversion (peroneus longus), and sensation in common peroneal and tibial distributions. I would check post-operative X-rays immediately for: (1) implant malposition (cup retroversion causing posterior impingement), (2) excessive leg lengthening (over 4cm increases nerve stretch), (3) cement or hardware in sciatic notch, (4) fracture or hematoma. If imaging shows a correctable cause like malposition or cement in notch, I would discuss urgent return to theatre for revision. If imaging is normal, the mechanism is likely traction injury during surgery. I would document the deficit carefully and start conservative management: ankle-foot orthosis (AFO) for foot drop, physiotherapy, reassure patient that 60% recover within 12 months. I would arrange nerve conduction studies and EMG at 3-4 weeks to differentiate neuropraxia (good prognosis) from axonotmesis or neurotmesis (poor prognosis). If deficit is progressive or there is concern for hematoma, I would get urgent MRI and consider surgical exploration. I would have an honest discussion with the patient about recovery timeline and likelihood - complete recovery uncertain, some permanent deficit possible.
KEY POINTS TO SCORE
Urgent assessment: full neurological exam, post-op X-rays
Check for correctable causes: malposition, lengthening, cement, fracture, hematoma
If correctable cause: urgent revision surgery
If traction injury (no correctable cause): conservative management
AFO for foot drop, physiotherapy
EMG/NCS at 3-4 weeks to assess severity
60% recover if neuropraxia, poor prognosis if neurotmesis
If progressive or hematoma suspected: MRI and exploration
Honest discussion about recovery prognosis
COMMON TRAPS
✗Assuming it will resolve without investigation
✗Not checking X-rays for correctable causes
✗Promising full recovery (60% recover, not 100%)
✗Not providing AFO for function
LIKELY FOLLOW-UPS
"What would you see on EMG at 3 weeks if this were neuropraxia vs axonotmesis?"
"If you found a large hematoma compressing the nerve, what would you do?"
"How much leg lengthening is safe to avoid nerve injury?"
VIVA SCENARIOCritical

Scenario 3: Recurrent Posterior Dislocation

EXAMINER

"A 65-year-old man has had three posterior dislocations in 6 months following primary THA via posterior approach. Each has been closed reduced successfully. How do you manage this?"

EXCEPTIONAL ANSWER
Recurrent dislocation (3 or more episodes) after posterior approach THA is a failure of the initial surgery and requires systematic assessment and likely revision surgery. My assessment includes: (1) detailed history - what position causes dislocation, any trauma, compliance with precautions, (2) examination - range of motion, Trendelenburg sign (abductor function), leg length, (3) imaging - AP pelvis and lateral hip X-rays to assess component position, CT if needed for 3D assessment. The key radiographic assessment is: cup anteversion (target 15-20 degrees), cup inclination (target 40 degrees), combined anteversion (femoral anteversion + acetabular anteversion should equal 25-40 degrees), femoral offset restoration, and any impingement. Common causes of recurrent posterior dislocation are: (1) cup malposition (excessive retroversion or high inclination), (2) femoral malposition (excessive anteversion or short neck), (3) insufficient offset, (4) abductor dysfunction, (5) component impingement, or (6) inadequate soft tissue repair. My management approach: non-operative management has failed with 3 dislocations, so revision surgery is indicated. Options include: (1) revision of malpositioned components to optimal position, (2) use of larger femoral head (36mm or 40mm) for improved stability, (3) dual mobility articulation if bone stock allows, (4) constrained liner as last resort. I would also consider trochanteric advancement if abductors are deficient. Post-operatively, I would use hip abduction brace for 6 weeks and intensive physiotherapy.
KEY POINTS TO SCORE
Recurrent dislocation = surgical problem, needs revision
Systematic assessment: history, exam, imaging (X-ray, CT if needed)
Assess cup position: anteversion (15-20°), inclination (40°)
Assess combined anteversion: femoral + acetabular = 25-40°
Assess offset, leg length, impingement
Common causes: cup retroversion, high inclination, insufficient offset, abductor dysfunction
Revision options: reposition components, larger head (36-40mm), dual mobility, constrained liner (last resort)
Consider trochanteric advancement if abductors deficient
Post-op: abduction brace, intensive physio
COMMON TRAPS
✗Trying more conservative management after 3 dislocations
✗Not getting CT to assess 3D component position
✗Using constrained liner as first option (should be last resort)
✗Not considering dual mobility (excellent option for recurrent instability)
LIKELY FOLLOW-UPS
"What is the target range for combined anteversion?"
"What are the downsides of a constrained liner?"
"How does dual mobility work and what are its advantages?"

MCQ Practice Points

Internervous Plane Question

Q: What is the internervous plane for the posterior approach to the hip? A: Between gluteus maximus (inferior gluteal nerve) and gluteus medius (superior gluteal nerve). This is the only hip approach with a true internervous plane.

Sciatic Nerve Location Question

Q: Where does the sciatic nerve run in relation to the greater trochanter? A: Approximately 1cm medial to the greater trochanter and posterior to the acetabulum. It exits the pelvis below the piriformis muscle (85% of patients).

Capsular Repair Question

Q: What is the effect of capsular repair on dislocation rate after posterior approach THA? A: Capsular repair reduces dislocation rate from approximately 7% to 2-3%. This is now considered standard of care for posterior approach.

Dislocation Position Question

Q: What combination of hip positions causes posterior dislocation after posterior approach THA? A: Flexion (over 90 degrees) + Adduction + Internal Rotation. Common scenarios: getting out of low chair, tying shoes, picking up objects from floor.

Short External Rotators Question

Q: What is the difference between the Moore and Southern modifications of the posterior approach? A: Moore approach: Divides piriformis and all short external rotators. Southern approach: Preserves piriformis, divides only obturator internus and gemelli. Both require capsular repair - more important than piriformis preservation.

Australian Context and Exam Preparation

AOANJRR Data:

  • Posterior approach: 35% of primary THAs in Australia
  • Second most common approach (lateral is 40%, anterior 20%)
  • Dislocation rates declining with routine capsular repair protocols
  • No difference in long-term revision rates between approaches

Training implications:

  • Essential skill for Orthopaedic exam and practice
  • Most Australian fellowship programs teach posterior as one of two primary approaches
  • Understanding of both posterior and lateral approaches expected

Enhanced Recovery Protocols:

  • Australian units increasingly adopting enhanced recovery (ERAS)
  • Posterior approach with capsular repair compatible with ERAS
  • Reduced or no hip precautions if capsule repaired

Orthopaedic Exam Preparation

For Orthopaedic clinical and viva exam, you must know: (1) True internervous plane (Gmax vs GM), (2) Sciatic nerve location and protection, (3) Moore vs Southern modifications, (4) Mandatory capsular repair to reduce dislocation, (5) Dislocation risk position (flexion + adduction + IR), (6) Management of sciatic nerve palsy and recurrent dislocation.

POSTERIOR APPROACH TO HIP

High-Yield Exam Summary

Key Anatomy

  • •TRUE internervous plane: Gmax (inferior gluteal) vs GM (superior gluteal)
  • •Sciatic nerve: 1cm medial to GT, below piriformis (85%)
  • •Short external rotators (superior to inferior): piriformis, sup gemellus, obturator internus, inf gemellus, quadratus femoris
  • •Posterior capsule: primary restraint to posterior dislocation

Surgical Steps

  • •1. Lateral decubitus, pelvis perpendicular
  • •2. Curved incision: PSIS to GT, then straight distally
  • •3. Split Gmax bluntly in line with fibers
  • •4. Identify piriformis (landmark for sciatic nerve)
  • •5. Tag and divide short externals (Moore) OR preserve piriformis (Southern)
  • •6. Tag capsule, perform capsulotomy
  • •7. Femoral neck osteotomy, proceed with THA
  • •8. CRITICAL: Repair capsule + short externals with non-absorbable suture

Structures at Risk

  • •Sciatic nerve (1cm medial to GT): protect with internal rotation, careful retractors, avoid over-lengthening
  • •Superior gluteal nerve (if extend proximally): limit proximal dissection to under 5cm from GT
  • •Inferior gluteal artery: hemostasis during Gmax split
  • •Medial femoral circumflex artery: at risk near quadratus femoris
  • •First perforating artery: near distal exposure

Advantages vs Disadvantages

  • •PRO: ONLY true internervous plane to hip (Gmax vs GM)
  • •PRO: Extensile proximally for DDH, distally for femoral fracture
  • •PRO: Excellent posterior column/wall exposure for revision and fracture
  • •CON: Higher dislocation if capsule NOT repaired (addressed by modern repair)
  • •CON: Sciatic nerve at risk (0.5-2% palsy rate)
  • •CON: Cannot use with patient supine (needs lateral positioning)

Complications

  • •Dislocation: 2-3% (with repair), 5-7% (without) - position: flex + add + IR
  • •Sciatic palsy: 0.5-2%, 60% recover if neuropraxia
  • •HO: 20-40% radiographic, prevent with indomethacin or radiation
  • •Capsular repair reduces dislocation by 4-fold - standard of care

Key Pearls

  • •CAPSULE REPAIR MANDATORY (non-absorbable suture, 3-4 interrupted)
  • •Risk position: flexion + adduction + internal rotation
  • •Piriformis = landmark (nerve runs below)
  • •AOANJRR: 35% of Australian THAs use posterior approach
  • •Enhanced recovery compatible with capsule repair
Quick Stats
Complexityadvanced
Reading Time25 min
Updated2024-12-24
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