Trauma

Volar and Dorsal Forearm Fasciotomy

Comprehensive surgical technique guide for emergency forearm fasciotomy - releasing all three compartments to prevent Volkmann's contracture

Core Procedure
intermediate
By OrthoVellum Medical Education Team

Reviewed by OrthoVellum Editorial Team

Editorial maintenance, source checking, and correction workflow • Published by OrthoVellum Medical Education Team

High Yield Overview

VOLAR AND DORSAL FOREARM FASCIOTOMY

Emergency three-compartment release - volar and dorsal approaches | intermediate

Critical Danger Structures - 6 Key Zones

Median Nerve

Location: Between FDS and FDP in volar compartment, approximately 2cm radial to midline incision. Most sensitive nerve to ischemia.

Protection: Identify early when releasing volar superficial fascia. Trace distally between FDS tendons. Protect during deep compartment release by gentle retraction.

Ulnar Nerve & Artery

Location: Ulnar border between FCU and FDP. Enters Guyon's canal at wrist between pisiform and hook of hamate. Ulnar artery runs with nerve.

Protection: Volar incision along ulnar border identifies nerve superficially. Oblique wrist incision crosses lateral to Guyon's canal avoiding direct nerve pressure.

Radial Nerve (Superficial Branch)

Location: Deep between brachioradialis and ECRL/ECRB proximally. Becomes subcutaneous 8-10cm proximal to radial styloid, emerging between brachioradialis and ECRL tendons.

Protection: Identify when releasing mobile wad from dorsal approach. Trace carefully in distal third where it becomes superficial. Gentle retraction only.

Posterior Interosseous Nerve

Location: Motor branch of radial nerve. Deep to supinator proximally. Runs between superficial and deep extensor layers in extensor compartment.

Protection: Typically safe during standard dorsal fasciotomy as it runs deep. Risk increases if extensive proximal dissection or supinator release required.

Radial Artery

Location: Radial border of forearm between brachioradialis and FCR proximally. Becomes superficial at wrist 5cm proximal to radial styloid, between FCR and brachioradialis tendons.

Protection: ULNAR volar approach avoids radial artery completely. If radial approach used (not recommended), radial artery lies in direct path - must identify and protect.

Anterior Interosseous Nerve

Location: Pure motor branch of median nerve arising 5cm distal to medial epicondyle. Runs on interosseous membrane with anterior interosseous artery in deep volar compartment.

Protection: Identified during deep volar compartment release. Innervates FPL, radial FDP, pronator quadratus. Injury causes inability to make 'OK' sign (AIN syndrome).

Mnemonic

V.S.D.MCOMPARTMENTS - Three Essential Spaces

Memory Hook:Exam favorite: 'How many compartments in the forearm?' Answer: THREE main (volar superficial, volar deep, dorsal), with mobile wad often considered a FOURTH separate compartment. All four must be assessed and released if tense.

Mnemonic

4 CsFOUR Cs - Muscle Viability Assessment

Memory Hook:Exam favorite: 'How do you assess muscle viability during fasciotomy?' Answer: Four Cs - Color, Contractility, Consistency, Capacity to bleed. Debride clearly necrotic muscle to prevent rhabdomyolysis and infection. Document findings carefully.

Forearm Compartment Syndrome - Recognition and Decision-Making

High-Risk Injuries

Pediatric:

  • Supracondylar humerus fractures (most common cause in children - 10-20% risk)
  • Both-bone forearm fractures with significant displacement
  • Floating elbow (ipsilateral humerus and forearm fractures)

Adult:

  • Both-bone forearm fractures (radius and ulna)
  • Isolated radius or ulna fractures with severe soft tissue injury
  • Crush injuries (machinery, motor vehicle collision)
  • Prolonged limb ischemia (brachial artery injury with delayed repair)
  • Ischemia-reperfusion injury after revascularization
  • Burns (circumferential eschar)
  • Tight casts or dressings
  • Snake bites (rare but recognized)

Clinical Diagnosis - CRITICAL

Exam Pearl

EXAM KEY: Forearm compartment syndrome is a CLINICAL DIAGNOSIS. Do NOT wait for compartment pressure measurements - they delay treatment and increase morbidity. Classic teaching: 'Pain out of proportion to injury' but this is subjective. More reliable: Pain on PASSIVE FINGER EXTENSION (stretches volar flexor compartment) - earliest and most specific sign.

Five Ps (late signs - do not wait for all):

  1. Pain - out of proportion to injury, progressive
  2. Pain on passive stretch - MOST RELIABLE early sign
  3. Pressure - tense, swollen forearm compartments on palpation
  4. Paresthesias - median nerve most sensitive (thumb-index numbness, thenar weakness)
  5. Pulselessness - LATE sign (compartment syndrome can occur with intact pulses)
  6. Pallor - LATE sign
  7. Paralysis - LATE sign (indicates severe ischemia)

Key Clinical Findings:

  • Pain on passive finger extension (stretches volar compartment) - EARLIEST sign
  • Pain on passive finger flexion (stretches dorsal compartment)
  • Tense, swollen forearm (compartments firm on palpation)
  • Progressive pain despite adequate analgesia
  • Median nerve symptoms (most sensitive) - thenar weakness, thumb-index numbness
  • Ulnar nerve symptoms - intrinsic weakness, ulnar-sided numbness
  • Radial nerve symptoms - wrist drop, thumb extension weakness

Associated Carpal Tunnel Syndrome

Critical Association

Carpal tunnel syndrome occurs in 20-30% of acute forearm compartment syndrome cases. Mechanism: Combined effect of forearm swelling transmitted distally plus local carpal tunnel swelling. EXAM TIP: If median nerve symptoms present (especially if confined to median distribution at hand), assess for Tinel's sign at wrist. Release carpal tunnel FIRST before forearm fasciotomy to prevent ongoing median nerve compression.

Timing - Critical Factor

  • Less than 6 hours: Excellent prognosis with early fasciotomy
  • 6-12 hours: Good prognosis if immediate fasciotomy
  • 12-24 hours: Permanent damage likely, Volkmann's contracture risk
  • More than 24 hours: Established Volkmann's contracture, fasciotomy may not help

EXAM ANSWER: "Forearm compartment syndrome is a CLINICAL DIAGNOSIS - I do not wait for pressure measurements. Classic presentation: pain out of proportion, tense forearm, pain on passive finger extension (stretches volar compartment - earliest sign). High-risk injuries include supracondylar fractures in children and both-bone forearm fractures. I assess for concurrent carpal tunnel syndrome (20-30% of cases) - if present, I release carpal tunnel first. Timing critical - fasciotomy within 6 hours gives excellent prognosis, delay beyond 12 hours risks permanent Volkmann's contracture."

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

EXAMINER

"A 7-year-old child 6 hours after closed reduction and pinning of a supracondylar humerus fracture has increasing forearm pain and pain on passive finger extension. The hand is pink with palpable radial pulse. What is your diagnosis and immediate management?"

EXCEPTIONAL ANSWER
This is ACUTE FOREARM COMPARTMENT SYNDROME until proven otherwise - the most common serious complication of supracondylar fractures in children (10-20% incidence). Pink hand with intact pulses does NOT exclude compartment syndrome. IMMEDIATE MANAGEMENT - STEP BY STEP: 1. **Remove all constricting dressings** - remove splint, cast, any circumferential bandages immediately. This alone may relieve pressure. 2. **Reposition arm** - extend elbow to neutral (flexion increases volar compartment pressure), forearm supination (relaxes flexor compartment). 3. **Clinical Assessment** - examine for key features: - Pain on passive finger extension (stretches volar flexor compartment) - PRESENT in this case - diagnostic - Tense forearm compartments on palpation - Median nerve function: thenar strength (APB), thumb-index sensation - Ulnar and radial nerve function 4. **Decision** - this child has CLINICAL compartment syndrome (pain on passive extension is diagnostic). Do NOT wait for compartment pressure measurements - they delay treatment. 5. **Consent and prepare for URGENT fasciotomy**: - Explain to parents: compartment syndrome = high pressure in muscle compartments causing muscle and nerve death - Needs emergency surgery to release pressure - Without surgery: permanent damage (Volkmann's contracture - claw hand deformity) - Risks of surgery: nerve injury, need for skin graft, scarring, infection 6. **Operative plan**: - Volar (ulnar approach) and dorsal fasciotomies - Release all THREE compartments (volar superficial, volar deep, dorsal + mobile wad) - Assess for concurrent carpal tunnel syndrome (20-30% incidence) - Leave wounds open - return to OR 48-72 hours for reassessment and delayed closure/STSG CRITICAL POINTS: - Clinical diagnosis - do not delay for pressure measurements - Pain on passive finger extension = volar compartment syndrome - Pink hand with pulses does NOT exclude compartment syndrome - Supracondylar fractures = highest risk injury in children - Delay beyond 6-12 hours = Volkmann's contracture risk
VIVA SCENARIOStandard

EXAMINER

"Describe Volkmann's ischemic contracture - what is the pathophysiology, clinical classification, and management options?"

EXCEPTIONAL ANSWER
Volkmann's ischemic contracture is the late consequence of UNTREATED or INADEQUATELY treated forearm compartment syndrome - represents IRREVERSIBLE ischemic injury to muscles and nerves. PATHOPHYSIOLOGY: - Prolonged muscle ischemia (greater than 6-12 hours) causes IRREVERSIBLE muscle necrosis - Necrotic muscle is replaced by fibrous scar tissue which contracts over time - Ischemic nerves develop intraneural fibrosis and permanent damage - Contracture develops over weeks to months as scar tissue matures - Final result: flexion contracture of wrist and fingers, nerve deficits, functional disability CLINICAL CLASSIFICATION - Tsuge (most commonly used): **Mild (most common)**: - Flexion contracture of 2-3 fingers (typically index, middle) - Localized to part of one muscle group (usually FDP) - Minimal sensory loss - Can make functional fist with wrist extension - Treatment: splinting, therapy, +/- flexor slide or lengthening **Moderate**: - All fingers involved in flexion contracture - Multiple muscle groups involved (FDP, FPL, sometimes FDS) - Moderate sensory loss (median/ulnar) - Cannot make functional fist - Treatment: muscle slide procedures, tendon transfers, +/- wrist fusion **Severe (worst prognosis)**: - Severe wrist and finger flexion contracture (claw hand) - All flexor and intrinsic muscles involved - Severe sensory loss, neuropathic pain - Virtually no hand function - Treatment: free functional muscle transfer (gracilis), wrist fusion, digit arthrodesis MANAGEMENT OPTIONS: **Non-operative** (mild cases): - Intensive hand therapy - Dynamic splinting - Serial casting - Tendon lengthening (FDP, FPL) **Operative - Muscle Procedures**: - Flexor origin slide (Page procedure) - releases all flexor origins from medial epicondyle - Muscle debridement - remove fibrotic muscle segments - Limitations: only works if some viable muscle remains **Operative - Tendon Procedures**: - Tendon lengthening (fractional or Z-lengthening) - Tendon transfers for weak muscles - Tenolysis if adhesions present **Operative - Nerve**: - Nerve decompression (late carpal tunnel release) - Neurolysis if intraneural fibrosis - Nerve grafting if complete nerve loss **Operative - Salvage** (severe cases): - Free functional muscle transfer (gracilis to forearm with nerve coaptation) - Wrist fusion for stability - Digit arthrodesis or amputation - Goal: pain relief and basic function, not normal hand PREVENTION: - Early diagnosis of compartment syndrome - Urgent fasciotomy (within 6 hours optimal) - Complete release of all compartments - Never delay for pressure measurements OUTCOME: - Mild: good function with conservative treatment - Moderate: fair function with surgery - Severe: poor function despite extensive reconstruction
VIVA SCENARIOStandard

EXAMINER

"Explain anterior interosseous nerve (AIN) syndrome - anatomy, clinical presentation, relationship to compartment syndrome, and diagnostic test."

EXCEPTIONAL ANSWER
The anterior interosseous nerve (AIN) is a PURE MOTOR branch of the median nerve - critical to understand for forearm compartment syndrome management. ANATOMY: - Branch of median nerve arising approximately 5cm distal to medial epicondyle - Courses distally on the INTEROSSEOUS MEMBRANE in the DEEP VOLAR compartment - Accompanied by anterior interosseous artery - Pure motor - NO sensory component - Terminal branch to pronator quadratus has sensory fibers to wrist joint (but not skin) INNERVATION - Three Muscles: 1. **FPL** (flexor pollicis longus) - thumb IP flexion 2. **FDP to index and middle fingers** (radial FDP) - index/middle DIP flexion 3. **Pronator quadratus** - forearm pronation (PQ is the main pronator in elbow-flexed position) CLINICAL PRESENTATION - AIN Syndrome: - Inability to make 'OK' sign (pathognomonic finding) - Patient asked to make circle with thumb and index finger - Normal: 'O' shape (circle formed) - AIN palsy: 'pinch' shape (thumb IP and index DIP remain extended - cannot flex) - Weak forearm pronation (PQ affected, but PT intact so some pronation remains) - NO SENSORY LOSS (pure motor nerve) - Intact thumb MCP flexion (FPB - thenar), index PIP flexion (FDS - median nerve) RELATIONSHIP TO COMPARTMENT SYNDROME: - AIN runs in DEEP VOLAR compartment - Compressed during forearm compartment syndrome - May be injured during fasciotomy (deep volar compartment release) - AIN syndrome can occur from: 1. Ischemia during compartment syndrome (most common) 2. Iatrogenic injury during fasciotomy 3. Isolated AIN compression (anomalous muscles, fracture, etc.) DIAGNOSTIC TEST - 'OK Sign': - Ask patient to make circle with thumb tip and index finger tip - Normal: Full 'O' shape with thumb IP flexed, index DIP flexed - AIN palsy: Pinch posture with thumb IP extended, index DIP extended - Only thumb MCP and index PIP flex (muscles NOT innervated by AIN) OTHER TESTS: - Thumb IP flexion against resistance (isolates FPL) - Index/middle DIP flexion against resistance (isolates radial FDP) - Hold index PIP extended, ask patient to flex DIP (cannot if AIN palsy) - Pronation strength with elbow flexed 90 degrees (isolates PQ) DIFFERENTIAL DIAGNOSIS: - Median nerve palsy: would have sensory loss, thenar weakness - FPL tendon rupture: would have history of laceration, tendon not palpable - FDP tendon rupture: would have history of closed jersey finger injury MANAGEMENT: - If ischemic (compartment syndrome): usually recovers in 3-6 months - If iatrogenic laceration: immediate microscopic repair with 8-0 or 9-0 nylon - If no recovery by 6 months: consider tendon transfers - FDP index to FDP long (borrow long finger DIP flexion) - Brachioradialis to FPL (restore thumb IP flexion) EXAM KEY POINT: - AIN syndrome = pure motor, inability to make OK sign - Always test after forearm fasciotomy - Differentiate from complete median nerve injury (AIN has no sensory loss, intact thenar)

Volar and Dorsal Forearm Fasciotomy - Exam Quick Reference

High-Yield Exam Summary

References

  1. Shadgan B, Menon M, O'Brien PJ, et al. Diagnostic techniques in acute compartment syndrome of the leg. J Orthop Trauma. 2008;22(8):581-587. PMID: 18758292. Systematic review of diagnostic techniques - emphasizes clinical diagnosis over compartment pressure measurements

  2. Leversedge FJ, Moore TJ, Peterson BC, et al. Compartment syndrome of the upper extremity. J Hand Surg Am. 2011;36(3):544-559. PMID: 21371630. Comprehensive review of upper extremity compartment syndrome including forearm - discusses anatomy, diagnosis, surgical technique

  3. Prasarn ML, Ouellette EA, Livingstone A, et al. Acute pediatric upper extremity compartment syndrome in the absence of fracture. J Pediatr Orthop. 2009;29(3):263-268. PMID: 19305277. Pediatric series highlighting supracondylar fractures as leading cause - discusses diagnosis and outcomes

  4. Kostler W, Strohm PC, Sudkamp NP. Acute compartment syndrome of the limb. Injury. 2005;36(8):992-998. PMID: 16005007. Evidence-based review of compartment syndrome management - discusses timing of fasciotomy and outcomes

  5. Mubarak SJ, Owen CA, Hargens AR, et al. Acute compartment syndromes: diagnosis and treatment with the aid of the wick catheter. J Bone Joint Surg Am. 1978;60(8):1091-1095. PMID: 701337. Classic article describing compartment pressure measurement - but emphasizes clinical diagnosis remains primary

  6. Ouellette EA. Compartment syndromes in obtunded patients. Hand Clin. 1998;14(3):431-450. PMID: 9742423. Discusses upper extremity compartment syndrome diagnosis and management - includes volar and dorsal approach technique

  7. Gelberman RH, Zakaib GS, Mubarak SJ, et al. Decompression of forearm compartment syndromes. Clin Orthop Relat Res. 1978;(134):225-229. PMID: 729244. Original description of forearm fasciotomy technique - volar and dorsal approaches

  8. Finkelstein JA, Hunter GA, Hu RW. Lower limb compartment syndrome: course after delayed fasciotomy. J Trauma. 1996;40(3):342-344. PMID: 8601846. Discusses outcomes based on timing of fasciotomy - emphasizes importance of early intervention to prevent Volkmann's contracture

  9. Tsuge K. Treatment of established Volkmann's contracture. J Bone Joint Surg Am. 1975;57(7):925-929. PMID: 1184640. Classic article describing Tsuge classification of Volkmann's contracture (mild, moderate, severe) and treatment options

  10. Seddon HJ. Volkmann's ischaemia in the lower limb. J Bone Joint Surg Br. 1966;48(4):627-636. PMID: 5953797. Classic description of pathophysiology of ischemic contracture following compartment syndrome - applicable to forearm