Adult Reconstruction

Total Hip Replacement Dislocation - Comprehensive Assessment and Management

Evidence-based surgical technique for managing THA instability through systematic assessment, component revision, dual mobility, and soft tissue reconstruction - FRACS exam preparation

Core Procedure
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By OrthoVellum Medical Education Team

Reviewed by OrthoVellum Editorial Team

Editorial maintenance, source checking, and correction workflow • Published by OrthoVellum Medical Education Team

High Yield Overview

TOTAL HIP REPLACEMENT DISLOCATION - COMPREHENSIVE ASSESSMENT AND MANAGEMENT

Systematic approach to THA instability: assess infection, measure component position (Lewinnek safe zone), identify direction/risk factors, correct malposition with dual mobility (gold standard). First dislocation with good components → non-operative (60-70% success). Recurrent (>2) → operative revision. | advanced

Critical Danger Structures - 5 Key Neurovascular Zones

Danger Zone 1: Sciatic Nerve

Location: 15-30mm posterior to hip joint (closer in revision due to scarring)

Injury Pattern: Foot drop (tibial division) + foot eversion weakness (peroneal division) + sensory loss posterior calf/foot

Protection: Identify early with vessel loop, keep hip flexed >45° to relax nerve, avoid excessive traction/lengthening >4cm, protect during posterior capsule work

Danger Zone 2: Femoral Neurovascular Bundle

Location: 30-50mm medial to anterior hip joint, beneath iliopsoas

Injury Pattern: Quadriceps paralysis, absent knee extension, anterior thigh numbness, catastrophic vascular injury if femoral artery damaged

Protection: Stay on bone with anterior retractors (Hohmann), avoid medial cement extrusion, fluoroscopy for anterior screw placement

Danger Zone 3: Superior Gluteal Neurovascular Bundle

Location: 30-50mm proximal to greater trochanter, exits sciatic notch above piriformis

Injury Pattern: Gluteus medius/minimus paralysis (Trendelenburg gait), superior buttock numbness

Protection: Avoid proximal dissection beyond GT tip, stay in safe zone (anterior-inferior quadrant), protect during GT osteotomy

Danger Zone 4: Lateral Femoral Cutaneous Nerve

Location: Variable 20-50mm from anterior incisions (ASIS to anterolateral thigh)

Injury Pattern: Meralgia paresthetica (lateral thigh burning/numbness)

Protection: Identify and protect when visible during anterior approach, gentle retraction if encountered

Danger Zone 5: Obturator Neurovascular Bundle

Location: 20-30mm medial to acetabulum, through obturator foramen

Injury Pattern: Adductor weakness, medial thigh numbness, vascular injury rare

Protection: Avoid medial acetabular screws below equator, use posterior-superior quadrant for fixation

Mnemonic

DISLOCATEDISLOCATE - Systematic Assessment for THA Instability

Mnemonic

DUAL MOBILITYDUAL MOBILITY - Gold Standard Advantages for THA Instability

Comprehensive Evaluation Protocol

Infection Workup (MANDATORY - Do NOT Skip)

Laboratory Tests:

  • ESR and CRP (elevated suggests infection)
  • Joint aspiration GOLD STANDARD: Cell count (>3000 cells/µL), differential (>80% PMNs), aerobic/anaerobic cultures (hold 14 days), Gram stain

Rule: Any revision for instability requires infection exclusion - infection can present as recurrent dislocation (30% of 'instability' cases in some series)

Imaging Protocol

Standard Radiographs:

  • AP pelvis (assess cup inclination, leg length, offset)
  • Lateral hip (assess cup anteversion on cross-table lateral)
  • Shoot-through lateral femur (assess stem version)

CT Pelvis with Metal Artifact Reduction (GOLD STANDARD for Component Position):

  • Measure cup inclination (radiographic plane): Normal 35-45°, safe zone 30-50°
  • Measure cup anteversion (radiographic plane): Normal 15-25°, safe zone 5-25°
  • Lewinnek safe zone: 40±10° inclination, 15±10° anteversion
  • Outside safe zone: 4x higher dislocation risk (9% vs 2%)
  • Assess impingement: Anterior wall overhang, posterior offset, neck-cup contact
  • Spinopelvic parameters: Pelvic tilt, sacral slope, lumbar lordosis (especially if spinal fusion or kyphosis)

Femoral Version Assessment:

  • CT femur (measure stem anteversion): Normal 10-15°
  • Combined anteversion: Cup + stem should total 25-40° (Ranawat triangle)
  • Excessive stem anteversion (>20°) → posterior instability
  • Excessive stem retroversion (<5°) → anterior instability

Dislocation Direction Classification

Posterior Dislocation (75-90%):

  • Mechanism: Flexion + adduction + internal rotation
  • Common scenarios: Getting up from low chair, tying shoes, getting into car
  • Component factors: Cup retroversion, excessive stem anteversion, posterior wall deficiency

Anterior Dislocation (10-25%):

  • Mechanism: Extension + external rotation + adduction
  • Common scenarios: Reaching back, pivoting on affected leg, leg crossing
  • Component factors: Excessive cup anteversion, stem retroversion, anterior impingement

Risk Factor Identification

Component Malposition (MOST COMMON CORRECTABLE):

  • Cup outside safe zone (inclination or anteversion)
  • Stem malposition (excessive anteversion/retroversion)
  • Combined anteversion outside 25-40°

Impingement:

  • Bony: Anterior acetabular wall overhang, posterior offset deficiency, femoral neck osteophytes
  • Component: Neck-cup contact, liner rim contact, modular neck-acetabular bone
  • Soft tissue: Iliopsoas over acetabular component (anterior), short external rotators (posterior)

Soft Tissue Deficiency:

  • Capsule deficient or absent (PRIMARY PATHOLOGY in many recurrent dislocations)
  • Short external rotator attenuation (posterior stability)
  • Abductor insufficiency (Trendelenburg positive)

Patient Factors:

  • Neuromuscular: Parkinson's disease (40-50% failure despite optimal surgery), dementia, CVA
  • Compliance: Cognitive impairment, psychiatric disorders, substance abuse
  • Anatomy: Femoral offset not restored, leg length inequality

Dislocation History Documentation

Critical Data:

  • Number of dislocations (first vs recurrent)
  • Time since surgery (early <90 days vs late)
  • Direction (posterior vs anterior)
  • Mechanism (high vs low energy)
  • Reduction method (closed vs open)
  • Previous revisions for instability

Treatment Algorithm Decision Tree

NON-OPERATIVE (First dislocation + well-positioned components + correctable risk factors):

  • Closed reduction under sedation
  • Hip abduction orthosis 6-12 weeks
  • Hip precautions 12 weeks minimum
  • Success rate: 60-70% after first, 30% after second, less than 20% after third

OPERATIVE (Recurrent >2 episodes OR first with malposition/uncorrectable factors):

  • Component revision if malposition
  • Dual mobility liner (gold standard, 85-95% success)
  • Soft tissue reconstruction if deficient
  • GT advancement if abductor insufficiency

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

EXAMINER

"A 68-year-old woman presents with her third posterior dislocation 18 months after primary posterior approach THR. What is your systematic approach to assessment and management?"

EXCEPTIONAL ANSWER
SYSTEMATIC ASSESSMENT: 1) RULE OUT INFECTION first (mandatory before any instability revision) - joint aspiration with cell count (>3000 cells/µL, >80% PMNs suggests infection), differential, cultures held 14 days, ESR/CRP. 2) CT PELVIS to measure component position - Lewinnek safe zone is 40±10° inclination, 15±10° anteversion. Outside safe zone has 4x dislocation risk. 3) Assess DISLOCATION DIRECTION - posterior 75-90% (flexion/adduction/IR mechanism). 4) Identify CORRECTABLE FACTORS: Component malposition (most common), impingement (bony/component), offset restoration, abductor function. 5) TREATMENT: Recurrent >2 episodes = operative indication. If malposition → revise component to safe zone + dual mobility liner. If well-positioned → isolated liner exchange to dual mobility. If abductor insufficiency → GT advancement. DUAL MOBILITY is gold standard with 85-95% success vs 75-85% constrained liner. POST-OP: Hip abduction brace 6-12 weeks, precautions 12 weeks minimum, compliance critical.
VIVA SCENARIOStandard

EXAMINER

"Explain dual mobility - how does it work, what are the advantages, and how does it compare to constrained liner?"

EXCEPTIONAL ANSWER
DUAL MOBILITY CONSTRUCT: Two articulations - 1) Large METAL outer bearing (52-60mm) articulates with acetabular cup. 2) Small POLYETHYLENE inner bearing (28-32mm) articulates with femoral head. MECHANISM: Outer bearing provides most motion initially (large bearing delays impingement). If extreme motion, inner bearing also moves (second articulation prevents dislocation). ADVANTAGES: 1) Delays impingement - large outer bearing (52-60mm) vs standard liner (32-40mm) means more ROM before neck impingement. 2) Reduces dislocation 50-80% vs standard. 3) Preserves head-to-neck ratio with small inner head. 4) Lower dissociation 1-2% vs constrained 3-5%. 5) Less wear - metal outer bearing vs polyethylene constraint in constrained liner. 6) Less torque to fixation vs constrained locking mechanism. COMPARISON TO CONSTRAINED: Dual mobility superior success 85-95% vs constrained 75-85%. Constrained uses locking ring to capture head - dissociation risk higher (3-5%), accelerated wear from polyethylene constraint, higher loosening from torque. I use dual mobility FIRST-LINE for instability revision, constrained only if dual mobility unavailable. EVIDENCE: Berry 2012 (0% re-dislocation in 140 instability revisions), AOANJRR (50% reduction in revision rate).
VIVA SCENARIOStandard

EXAMINER

"The cup position on CT shows inclination 42° and anteversion 18° (within safe zone), but the patient has had 3 posterior dislocations. Intraoperatively the hip is stable with trials. What else should you assess and what is your management?"

EXCEPTIONAL ANSWER
COMPONENTS IN SAFE ZONE BUT RECURRENT INSTABILITY - must assess OTHER factors: 1) COMBINED ANTEVERSION - cup 18° + stem version (measure on CT). Target total 25-40° (Ranawat triangle). If stem excessive anteversion >20° → posterior instability despite cup in safe zone. 2) IMPINGEMENT - test flex/adduct/IR intraoperatively. Identify if femoral neck impinges on cup/acetabular bone (levering out). Assess on CT for anterior wall overhang, posterior offset deficiency. 3) SOFT TISSUE DEFICIENCY - capsule often deficient/absent in recurrent dislocation (PRIMARY PATHOLOGY in many cases). Assess capsule quality - if inadequate, needs reconstruction. 4) ABDUCTOR INSUFFICIENCY - Trendelenburg test preop, assess intraop (pull on gluteus medius). If weak → GT advancement needed. 5) SPINOPELVIC PARAMETERS (advanced) - spinal fusion or kyphosis changes pelvic tilt, alters functional cup position. 6) PATIENT FACTORS - neuromuscular (Parkinson's, dementia), compliance. MANAGEMENT: Well-positioned components + recurrent instability = ISOLATED LINER EXCHANGE to DUAL MOBILITY (85-95% success, least invasive). Add SOFT TISSUE RECONSTRUCTION if capsule deficient (Achilles allograft). Add GT ADVANCEMENT if abductor insufficiency. DO NOT revise well-positioned components - increases bone loss and risk. POST-OP: Brace 6-12 weeks, precautions 12 weeks, education critical.

THA Dislocation Management - Rapid Exam Review

High-Yield Exam Summary

References

  1. Berry DJ, von Knoch M, Schleck CD, Harmsen WS. Effect of femoral head diameter and operative approach on risk of dislocation after primary total hip arthroplasty. J Bone Joint Surg Am. 2005;87(11):2456-2463. doi:10.2106/JBJS.D.02860

  2. Lewinnek GE, Lewis JL, Tarr R, Compere CL, Zimmerman JR. Dislocations after total hip-replacement arthroplasties. J Bone Joint Surg Am. 1978;60(2):217-220. PMID: 641088

  3. Berry DJ, von Knoch M, Schleck CD, Harmsen WS. The cumulative long-term risk of dislocation after primary Charnley total hip arthroplasty. J Bone Joint Surg Am. 2004;86(1):9-14. doi:10.2106/00004623-200401000-00003

  4. Guyen O, Pibarot V, Vaz G, Chevillotte C, Bejui-Hugues J. Use of a dual mobility socket to manage total hip arthroplasty instability. Clin Orthop Relat Res. 2009;467(2):465-472. doi:10.1007/s11999-008-0476-0

  5. Philippot R, Adam P, Reckhaus M, et al. Prevention of dislocation in total hip revision surgery using a dual mobility design. Orthop Traumatol Surg Res. 2009;95(6):407-413. doi:10.1016/j.otsr.2009.04.016

  6. Lombardi AV Jr, Mallory TH, Kraus TJ, Vaughn BK. Preliminary report on the S-ROM constraining acetabular insert: a retrospective clinical experience. Orthopedics. 1991;14(3):297-303. PMID: 2020623

  7. Anderson MJ, Murray WR, Skinner HB. Constrained acetabular components. J Arthroplasty. 1994;9(1):17-23. doi:10.1016/0883-5403(94)90132-5

  8. Patel PD, Potts A, Froimson MI. The dislocating hip arthroplasty: prevention and treatment. J Arthroplasty. 2007;22(4 Suppl 1):86-90. doi:10.1016/j.arth.2007.03.031

  9. Meek RM, Allan DB, McPhillips G, Kerr L, Howie CR. Epidemiology of dislocation after total hip arthroplasty. Clin Orthop Relat Res. 2006;447:9-18. doi:10.1097/01.blo.0000218754.12311.4a

  10. Australian Orthopaedic Association National Joint Replacement Registry (AOANJRR). Hip, Knee & Shoulder Arthroplasty: 2023 Annual Report. Adelaide: AOA; 2023. Available at: https://aoanjrr.sahmri.com/annual-reports-2023