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Total Hip Replacement Dislocation - Comprehensive Assessment and Management

Operative SurgeryArthroplasty
ArthroplastyAdvancedCore Procedure

Total Hip Replacement Dislocation - Comprehensive Assessment and Management

Evidence-based surgical technique for managing THA instability through systematic assessment, component revision, dual mobility, and soft tissue reconstruction

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Peer-reviewed Β· 2026-06-20
High-yield overview

Systematic approach to THA instability: exclude infection, measure component position (the Lewinnek safe zone, read alongside spinopelvic factors), identify direction and risk factors, then correct malposition and consider a dual-mobility construct. First dislocation with well-positioned components β€” non-operative trial (about 60 to 70 percent remain stable). Recurrent (more than two episodes) β€” operative revision.

75 to 90%Posterior dislocations
60 to 70%Stable after one closed reduction
Dual mobilityLeading revision construct
120 minTypical duration
Critical Must-Knows
  • RULE OUT INFECTION before any revision for instability β€” aspiration with cell count and culture is gold standard (cultures held 14 days); infection can present as recurrent dislocation.
  • The Lewinnek safe zone is 40Β±10Β° inclination and 15Β±10Β° anteversion (Lewinnek 1978: 1.5 percent dislocation inside versus 6.1 percent outside). CRITICAL CAVEAT β€” Abdel 2016 showed 58 percent of dislocated THAs were WITHIN this zone, so the safe zone is necessary but not sufficient and instability is multifactorial.
  • Posterior dislocation accounts for 75 to 90 percent (flexion, adduction, internal rotation); anterior 10 to 25 percent (extension, external rotation, adduction).
  • Treatment algorithm: first dislocation with well-positioned components β€” non-operative trial; recurrent instability (more than two episodes) β€” operative revision, with dual mobility a leading construct (revision-series re-dislocation typically under 5 percent).
  • Stability depends on combined anteversion (cup plus stem, target roughly 25 to 50 degrees), offset restoration, soft-tissue integrity and spinopelvic mobility β€” assess and correct all of them, not the cup alone.

When & Why


The clinical problem. Dislocation after total hip arthroplasty is one of the most common complications and a leading indication for revision surgery. Management is dictated by the number of episodes, the direction, the position of the components and the integrity of the soft tissues β€” so the first job is a systematic assessment, not an operation. Step 1 β€” Rule out infection (mandatory). Before any revision for instability, exclude infection. Infection can present as recurrent dislocation (around 30 percent of "instability" cases in some series), and any revision will fail if infection is missed. Workup is ESR and CRP, plus joint aspiration as the gold standard β€” cell count (greater than 3000 cells per microlitre), differential (greater than 80 percent PMNs), Gram stain, and aerobic and anaerobic cultures held for 14 days. Step 2 β€” Image and measure component position. Standard radiographs (AP pelvis, lateral hip, shoot-through lateral femur) assess cup inclination, leg length and offset. A CT pelvis with metal-artifact reduction is the gold standard for component position: cup inclination (normal 35 to 45 degrees, safe zone 30 to 50 degrees), cup anteversion (normal 15 to 25 degrees, safe zone 5 to 25 degrees), stem version (CT femur, normal 10 to 15 degrees), impingement (anterior wall overhang, posterior offset deficiency), and spinopelvic parameters (pelvic tilt, sacral slope, lumbar lordosis). The Lewinnek safe zone β€” and its critical caveat. Lewinnek (1978) defined the safe zone as 40Β±10Β° inclination and 15Β±10Β° anteversion, with 1.5 percent dislocation inside versus 6.1 percent outside (roughly four-fold higher). The critical caveat is Abdel (2016): 58 percent of dislocated THAs had a cup WITHIN this zone. The safe zone is therefore necessary but not sufficient β€” instability is multifactorial, so read cup position together with stem version, combined anteversion, offset and spinopelvic mobility, never in isolation. Step 3 β€” Classify the direction. - Posterior (75 to 90 percent) β€” mechanism flexion + adduction + internal rotation (rising from a low chair, tying shoes, getting into a car). Component factors: cup or stem retroversion or insufficient combined anteversion, inadequate posterior offset, posterior capsular and short-rotator deficiency.

  • Anterior (10 to 25 percent) β€” mechanism extension + external rotation + adduction (reaching back, pivoting, leg crossing). Component factors: excessive cup and/or stem anteversion (excessive combined anteversion), anterior impingement. Step 4 β€” Identify the correctable risk factors. Component malposition (the most common correctable cause); impingement (bony β€” anterior wall overhang, posterior offset deficiency, femoral neck osteophytes; component β€” neck-cup or liner-rim contact; soft-tissue β€” iliopsoas anteriorly, short external rotators posteriorly); soft-tissue deficiency (capsule deficient or absent, short-rotator attenuation, abductor insufficiency with a positive Trendelenburg); and patient factors (Parkinson's disease, dementia, CVA, cognitive or compliance problems, unrestored offset, leg-length inequality). Document the dislocation history: number of episodes (first versus recurrent), timing (early less than 90 days versus late), direction, energy (high versus low), reduction method (closed versus open), and any prior revisions for instability. The combined-anteversion concept. Stability depends on the sum of cup plus stem anteversion, not the cup alone. The commonly cited target is roughly 25 to 50 degrees (Dorr), with Widmer's optimum near 37 degrees. Excessive combined or stem anteversion (stem over 20 degrees) drives ANTERIOR instability; a retroverted stem (under 5 degrees) or low combined anteversion drives POSTERIOR instability. If the combined value is outside the zone, plan to address BOTH the cup and the stem.
20Β°
Stem anteversion
17Β°
Combined
37Β°
Interpretation
In the target zone β€” ideal (Widmer optimum near 37Β°)
30Β°
Stem anteversion
25Β°
Combined
55Β°
Interpretation
High β€” anterior instability and edge-loading risk in extension and external rotation
10Β°
Stem anteversion
5Β°
Combined
15Β°
Interpretation
Low β€” posterior impingement and instability in flexion and internal rotation
Combined anteversion β€” worked examples
Cup anteversionStem anteversionCombinedInterpretation
20Β°17Β°37Β°In the target zone β€” ideal (Widmer optimum near 37Β°)
30Β°25Β°55Β°High β€” anterior instability and edge-loading risk in extension and external rotation
10Β°5Β°15Β°Low β€” posterior impingement and instability in flexion and internal rotation

The decision. - Non-operative β€” first dislocation with well-positioned components and correctable risk factors: closed reduction under sedation, a hip abduction orthosis for 6 to 12 weeks, and hip precautions for a minimum of 12 weeks. Success is roughly 60 to 70 percent after the first episode, falls to around 30 percent after the second, and is less than 20 percent after the third.

  • Operative β€” recurrent instability (more than two episodes), or a first dislocation with component malposition or uncorrectable risk factors: correct the cause and add a stability-enhancing construct. The construct choice (in order of preference).
Dual mobility (preferred)

A large metal outer bearing (52 to 60 mm) plus a small polyethylene inner bearing (28 to 32 mm) delays impingement and lengthens jump distance; revision-series re-dislocation is typically under 5 percent, with lower dissociation than a constrained liner.

Component revision (if malpositioned)

Revise an out-of-zone cup (target 40Β° inclination, 15 to 20Β° anteversion) and/or an out-of-version stem to neutral (10 to 15Β° anteversion), restore offset, then add dual mobility. Corrects the underlying cause.

Constrained liner (salvage)

A locking ring captures the head; higher dissociation (3 to 5 percent), accelerated wear and loosening torque. Reserve for the irreparable-abductor or unreconstructable-soft-tissue hip where dual mobility will not control instability.

If the components are well-positioned, an isolated liner exchange to dual mobility is the least invasive option (it preserves well-fixed components). Add capsular reconstruction (Achilles allograft) for a deficient capsule and greater trochanteric advancement for abductor insufficiency. Consent specifically for re-dislocation, infection (2 to 5 percent in revision), sciatic nerve palsy (3 to 7 percent in revision versus 0.5 to 2 percent in primary), intraoperative fracture, leg-length or offset change, and the possibility of salvage (Girdlestone, arthrodesis) if multiple revisions fail. Setup. Lateral decubitus for a posterior approach (around 90 percent of primary THRs), supine if the primary was anterior; re-use the previous surgical approach to minimise further soft-tissue damage; fluoroscopy is essential throughout.

The Operation


The goal is to expose the hip through the previous approach, identify and protect the sciatic nerve early, define the cause of instability (component position, impingement, soft-tissue deficiency), correct any malposition, and rebuild a stable construct β€” most often with a dual-mobility bearing, reinforced by soft-tissue repair or reconstruction. The exposure is laid out in full as the opening steps, because in the scarred, unstable hip the exposure and nerve protection are the whole game.

AP pelvic radiograph of a dislocated hip replacement
AP pelvic radiograph showing a dislocated total hip replacement, the prosthetic head lying outside the acetabular cup.Credit: OrthoVellum surgical illustration

Operative sequence

Step 1Position, approach & setup
  • Lateral decubitus for the posterior approach (around 90 percent of primary THRs); supine if the primary was done anteriorly.
  • Re-use the previous surgical approach to minimise further soft-tissue damage; the posterior approach is most common and is extended for revision.
  • Fluoroscopy is essential for intra-operative component position assessment.
  • Excise the old scar, use the previous incision, and extend proximally and distally as needed β€” revision demands a longer exposure.
Step 2Exposure β€” posterior approach (the key step)
  • Incise the fascia lata over the greater trochanter and split gluteus maximus in line with its fibres, protecting the superior gluteal nerve proximally.
  • IDENTIFY THE SCIATIC NERVE EARLY and protect it with a vessel loop β€” it lies closer in revision because of scarring, and the palsy rate is 3 to 7 percent in revision versus 0.5 to 2 percent in primary surgery.
  • Tag the short external rotators if any are present (they are often attenuated or absent in the unstable hip).
  • Perform an extensive capsulectomy β€” the capsule is usually deficient and is in fact the primary pathology in many recurrent dislocations.
  • Keep the hip flexed more than 45 degrees during deep work to relax the sciatic nerve.
Step 3Dislocate the hip
  • Posterior dislocation: flex 90 degrees, adduct, internally rotate.
  • The hip may be scarred in position and require extensive soft-tissue release.
Step 4Intra-operative assessment (CRITICAL)
  • Component position: fluoroscopy to measure cup inclination and anteversion; direct visualisation to assess stem version; calculate combined anteversion.
  • Impingement testing: reproduce the dislocation mechanism (flexion, adduction, internal rotation for posterior), identify neck-cup or liner-rim contact, and check throughout the range of motion.
  • Soft-tissue quality: capsule (intact versus deficient versus absent), short external rotators (present versus attenuated), abductors (strong versus weak β€” Trendelenburg).
  • Range to dislocation: test stability in all positions and identify the safe versus unsafe arcs.
Step 5Component removal (only if indicated)
  • Acetabular cup: remove the modular liner first; a loose cup levers out with curved osteotomes; a well-fixed cup needs circumferential osteotomes kept on metal to preserve bone (sometimes an Explant system); remove all cement if cemented. Preserve bone stock β€” it is critical for revision.
  • Femoral stem: remove the head and liner first; a loose stem extracts with gentle impaction and extraction devices; a well-fixed stem requires an extended trochanteric osteotomy (ETO) β€” osteotomise the anterior third of the femur for 10 to 12 cm with the greater trochanter left attached to vastus lateralis, repaired with cerclage wires after stem insertion. DO NOT FORCE the stem β€” it will fracture the femur.
Step 6Acetabular and/or femoral revision (if malpositioned or loose)
  • Acetabulum: ream to bleeding bone, size for press-fit (1 to 2 mm under-ream), position to the safe zone (40Β° inclination, 15 to 20Β° anteversion), confirm with fluoroscopy, place multiple screws in the posterior-superior quadrant (avoiding anterior and medial neurovascular structures), use porous-metal augments for bone loss to achieve roughly 50 percent host-bone contact, and insert a dual-mobility liner.
  • Femur: ream and broach the canal; choose a modular stem if metaphyseal bone is adequate (adjusts offset and version independently) or an extensively coated diaphyseal-fit stem if there is bone loss; position in neutral version (10 to 15Β° anteversion); restore offset by comparison with the contralateral side; repair an ETO with 2 to 3 cerclage wires before the final head; use a large head (36 to 40 mm) or a dual-mobility construct.
Step 7Isolated liner exchange (if components are well-positioned)
  • Dislocate the hip and remove the head; remove the modular liner (locking tabs, specialised tools); clean the cup taper and inspect for wear.
  • Insert a dual-mobility liner (large metal outer bearing locks into the cup, small polyethylene inner bearing for the head). Alternatives are a constrained liner (locking ring) or a lipped (elevated-rim) liner.
  • Upsize the head (32 to 36 mm, or 36 to 40 mm) only if polyethylene thickness remains greater than 6 mm.
Step 8Soft-tissue reconstruction
  • Capsule: if adequate, primary repair to the femoral neck or greater trochanter with heavy non-absorbable sutures (number 2 or number 5 Ethibond). If inadequate, an Achilles tendon allograft reconstruction β€” anchored to the acetabular rim through drill holes and to the femoral neck or GT with transosseous sutures β€” recreates anterior and posterior restraints.
  • Short external rotators: repair piriformis, the gemelli, obturators and quadratus femoris to the GT or posterior capsule (transosseous sutures if bone quality allows); these are often attenuated, so do the best repair possible.
  • Greater trochanteric advancement (if abductor insufficiency): osteotomise the GT with the attached gluteus medius and minimis, advance it 1 to 2 cm distally to increase the abductor moment arm and tension, fix with screws and washers or cables, and protect with touch-down weight-bearing for 12 weeks until union.
Step 9Trial reduction and stability testing (CRITICAL)
  • Posterior stability (most important): 90Β° flexion + adduction + internal rotation with a posterior force β€” must be stable with a firm endpoint.
  • Anterior stability: extension + external rotation + adduction β€” must remain reduced.
  • Mid-range: 45Β° flexion, neutral rotation, abduction and adduction.
  • Extremes: full flexion, extension, abduction, adduction β€” identify impingement.
  • Shuck test: quantify translation (should be minimal, less than 5 mm).
  • RULE: do not accept instability on the table β€” it will fail post-operatively. If unstable, increase head size, convert to dual mobility or a constrained liner, adjust component position, or increase soft-tissue tension.
Step 10Closure
  • Layered closure: capsule and short external rotators to the femoral neck or GT (heavy non-absorbable sutures); confirm stable GT fixation if osteotomised; close gluteus maximus fascia securely, then deep fascia, subcutaneous layer and skin (monofilament or staples).
  • Place an abduction pillow between the legs; consider a drain for a large dead space (removed at 24 to 48 hours).
Sciatic nerve β€” the critical safety step

Identify the sciatic nerve early and protect it with a vessel loop before any deep revision work. It lies 15 to 30 mm posterior to the hip joint and is closer in the scarred revision hip, where the palsy rate is 3 to 7 percent (versus 0.5 to 2 percent in primary surgery). Keep the hip flexed more than 45 degrees to relax the nerve, avoid excessive traction or lengthening more than 4 cm, and protect it during posterior capsular work. If a palsy is recognised intra-operatively: remove all traction, flex the hip, and assess perfusion; a laceration needs immediate microsurgical repair with a neurosurgery consult. Most palsies are neuropraxias β€” fit an AFO for foot drop, baseline the EMG at 3 weeks and repeat at 6 to 12 weeks; roughly 60 to 70 percent recover partially, 20 to 30 percent completely.

Dual mobility vs constrained liner β€” the exam answer

For instability revision, favour a dual-mobility construct (85 to 95 percent success, 1 to 2 percent dissociation) over a constrained liner (75 to 85 percent success, 3 to 5 percent dissociation, accelerated wear and loosening torque). Reserve the constrained liner for the salvage hip with irreparable abductors or unreconstructable soft tissue. The dual-mobility mechanism is NOT simply a jumbo head β€” it is two articulations (a large metal outer bearing with the cup, plus a small polyethylene inner bearing with the head) that delay impingement and lengthen jump distance.

Do not revise well-positioned components 'because the hip dislocates'

Recall Abdel 2016: most dislocated THAs sit within the Lewinnek zone, so instability is multifactorial. If components are well-positioned and stable on trial, the answer is an isolated liner exchange to dual mobility plus soft-tissue reconstruction β€” not revision of stable components, which only burns bone stock and adds risk.

Aftercare & Complications


Post-operative protocol | Phase | Timing | Brace / weight-bearing | Activity | |-------|--------|------------------------|----------| | 1 | 0 to 6 weeks | Hip abduction orthosis day and night; WBAT (touch-down weight-bearing 12 weeks if GT advancement) | Strict hip precautions | | 2 | 6 to 12 weeks | Orthosis night-time only; advance weight-bearing as able | Wean brace, advance activity | | 3 | 3 months and beyond | None | Precautions lifted if stable; graded return to function | Hip precautions apply for a minimum of 12 weeks (consider lifelong if high risk). Posterior approach: no flexion more than 90Β°, no adduction past midline, no internal rotation. Anterior approach: no extension, no external rotation, no adduction. DVT prophylaxis is extended 4 to 6 weeks in the high-risk patient (LMWH or DOAC per protocol). Follow-up schedule | Interval | Review | |----------|--------| | 2 weeks | Wound check | | 6 weeks | Radiographs, advance activity | | 3 months | Radiographs, wean brace | | 6 months | Radiographs and function | | 1 year | Radiographs | | Annually for life | Radiographs and function (Oxford Hip Score, HOOS) | Surveillance watches for late dislocation, accelerated wear (dual-mobility metal-on-poly, constrained poly), component loosening (especially the torque on a constrained liner), and neuromuscular progression. Outcomes. Non-operative management of a first dislocation keeps roughly 60 to 70 percent stable, but success falls steeply with each further episode (weak evidence base). Dual mobility delivers low re-dislocation in revision series β€” Philippot 2009 (3.7 percent overall, 0 percent in the recurrent-instability subgroup, 7-year cup survival 96.1 percent) and Guyen 2009 (54 unstable hips restored to stability) β€” and registries report lower revision-for-dislocation than single bearings. The constrained liner carries a recognised catastrophic mode (locking-ring or liner dissociation). Worse prognosis attends neuromuscular or cognitive disorders (a relative risk only β€” Meek 2006 found no increased dislocation with Parkinson's or stroke in their cohort), severe abductor deficiency, multiple prior revisions, and a stiff or fused spine.

Re-dislocation (most common failure)
Recognition
Acute pain, deformity, inability to bear weight; radiographs confirm the head outside the socket. Early less than 90 days is technical; late is patient factors
Prevention
Correct component malposition (Lewinnek safe zone), use dual mobility (reduces dislocation 50 to 80 percent), repair soft tissues, GT advancement if abductor insufficiency, educate the patient on precautions
Management
Closed reduction under sedation if the FIRST episode and components well-positioned. Operative if recurrent (more than 2) β€” systematic reassessment (infection, position, impingement, compliance), conversion to dual mobility, salvage (Girdlestone, arthrodesis) if multiple failures
Infection (2 to 5 percent in revision)
Recognition
Persistent pain, fever, wound drainage, elevated ESR and CRP; aspiration cell count greater than 3000, PMN greater than 80 percent, positive cultures
Prevention
Meticulous sterile technique, weight-adjusted first-generation cephalosporin within 60 minutes of incision, minimise operative time, avoid haematoma, optimise the patient (diabetes, BMI, smoking)
Management
Acute (less than 3 weeks): irrigation and debridement with liner exchange, retain well-fixed components, 6 weeks IV antibiotics. Chronic (more than 3 weeks): two-stage revision (component removal, spacer, 6 to 12 weeks antibiotics, reimplantation). Suppression if non-operative candidate
Sciatic nerve palsy (3 to 7 percent revision versus 0.5 to 2 percent primary)
Recognition
Foot drop (tibial division), foot eversion weakness (peroneal division), sensory loss in the posterior calf and lateral foot. Examine immediately post-op
Prevention
Identify the nerve early with a vessel loop, keep the hip flexed more than 45Β° to relax the nerve, avoid traction or lengthening more than 4 cm, protect during posterior capsule work, gentle tissue handling
Management
Immediate recognition: remove all traction, flex the hip, assess perfusion; intra-operative laceration needs immediate microsurgical repair. Most are neuropraxias: AFO for foot drop, physiotherapy, EMG at 3 weeks and 6 to 12 weeks. 60 to 70 percent recover partially, 20 to 30 percent completely
Intraoperative fracture (femur 2 to 5 percent, acetabulum 1 to 2 percent)
Recognition
Femur: crack or pop during broaching or impaction, sudden subsidence. Acetabulum: crack during reaming or cup impaction, loss of press-fit
Prevention
Femur: gentle technique, sequential broaching, ETO for well-fixed stems (never force), avoid varus positioning. Acetabulum: controlled reaming, assess bone quality, do not over-ream osteoporotic bone
Management
Femur: cerclage wires proximal to the fracture, a long fully-coated stem bypassing the fracture by 2 cortical diameters, touch-down weight-bearing 12 weeks. Acetabulum: stop reaming, screws for stability, cup-cage or reconstruction plate if unstable
Dual-mobility liner dissociation (1 to 2 percent)
Recognition
Acute pain and instability (different from standard dislocation); radiographs show the head separated from the metal outer bearing, or the outer bearing dissociated from the cup
Prevention
Ensure the correct liner taper match (same manufacturer as the cup), confirm full seating (audible and tactile click), intra-operative stability testing, avoid excessive impingement forces
Management
Immediate reoperation (cannot closed-reduce): open reduction, assess and replace a damaged liner, ensure proper taper engagement, test stability. High recurrence risk if components are malpositioned
Constrained liner dissociation (3 to 5 percent)
Recognition
Acute pain and instability; radiographs show the locking ring dissociated from the liner with the head escaped from constraint
Prevention
Prefer dual mobility over constrained, ensure the locking ring is fully engaged, avoid excessive impingement, correct component malposition (constraint fails if impingement persists)
Management
Reoperation: remove the failed constrained liner, convert to dual mobility (preferred) or a new constrained liner with component repositioning if malpositioned. Address impingement and malposition or it will recur
Greater trochanteric non-union or migration (10 to 15 percent after advancement)
Recognition
Persistent lateral hip pain, Trendelenburg gait, palpable GT mobility; radiographs show GT displacement more than 2 cm proximal or non-union at 6 months
Prevention
Rigid fixation (2 to 3 screws and washer, or cables), avoid over-advancement (1 to 2 cm maximum), touch-down weight-bearing 12 weeks, optimise bone health (vitamin D, calcium, bisphosphonates if osteoporotic)
Management
If asymptomatic and less than 1 cm displacement: observe (fibrous union may be adequate). If symptomatic or more than 2 cm migration: revision ORIF with cables or plate and bone graft. Chronic severe abductor deficiency: accept the limitation or a gluteus maximus transfer (less effective)
Accelerated polyethylene wear (5 to 10 percent at 5 years with dual mobility or constrained)
Recognition
Increasing pain, radiographic wear (decreased poly thickness), osteolysis (progressive lucencies or cysts around components)
Prevention
Use highly cross-linked polyethylene, optimise component position to reduce impingement, ensure adequate poly thickness (greater than 6 mm), avoid oversizing the head with thin poly
Management
Surveillance with annual radiographs; liner exchange if progressive wear before osteolysis is severe; component revision if osteolysis with loosening (bone-graft lytic lesions, revise loose components)
Major complications β€” recognition, prevention and management
ComplicationRecognitionPreventionManagement
Re-dislocation (most common failure)Acute pain, deformity, inability to bear weight; radiographs confirm the head outside the socket. Early less than 90 days is technical; late is patient factorsCorrect component malposition (Lewinnek safe zone), use dual mobility (reduces dislocation 50 to 80 percent), repair soft tissues, GT advancement if abductor insufficiency, educate the patient on precautionsClosed reduction under sedation if the FIRST episode and components well-positioned. Operative if recurrent (more than 2) β€” systematic reassessment (infection, position, impingement, compliance), conversion to dual mobility, salvage (Girdlestone, arthrodesis) if multiple failures
Infection (2 to 5 percent in revision)Persistent pain, fever, wound drainage, elevated ESR and CRP; aspiration cell count greater than 3000, PMN greater than 80 percent, positive culturesMeticulous sterile technique, weight-adjusted first-generation cephalosporin within 60 minutes of incision, minimise operative time, avoid haematoma, optimise the patient (diabetes, BMI, smoking)Acute (less than 3 weeks): irrigation and debridement with liner exchange, retain well-fixed components, 6 weeks IV antibiotics. Chronic (more than 3 weeks): two-stage revision (component removal, spacer, 6 to 12 weeks antibiotics, reimplantation). Suppression if non-operative candidate
Sciatic nerve palsy (3 to 7 percent revision versus 0.5 to 2 percent primary)Foot drop (tibial division), foot eversion weakness (peroneal division), sensory loss in the posterior calf and lateral foot. Examine immediately post-opIdentify the nerve early with a vessel loop, keep the hip flexed more than 45Β° to relax the nerve, avoid traction or lengthening more than 4 cm, protect during posterior capsule work, gentle tissue handlingImmediate recognition: remove all traction, flex the hip, assess perfusion; intra-operative laceration needs immediate microsurgical repair. Most are neuropraxias: AFO for foot drop, physiotherapy, EMG at 3 weeks and 6 to 12 weeks. 60 to 70 percent recover partially, 20 to 30 percent completely
Intraoperative fracture (femur 2 to 5 percent, acetabulum 1 to 2 percent)Femur: crack or pop during broaching or impaction, sudden subsidence. Acetabulum: crack during reaming or cup impaction, loss of press-fitFemur: gentle technique, sequential broaching, ETO for well-fixed stems (never force), avoid varus positioning. Acetabulum: controlled reaming, assess bone quality, do not over-ream osteoporotic boneFemur: cerclage wires proximal to the fracture, a long fully-coated stem bypassing the fracture by 2 cortical diameters, touch-down weight-bearing 12 weeks. Acetabulum: stop reaming, screws for stability, cup-cage or reconstruction plate if unstable
Dual-mobility liner dissociation (1 to 2 percent)Acute pain and instability (different from standard dislocation); radiographs show the head separated from the metal outer bearing, or the outer bearing dissociated from the cupEnsure the correct liner taper match (same manufacturer as the cup), confirm full seating (audible and tactile click), intra-operative stability testing, avoid excessive impingement forcesImmediate reoperation (cannot closed-reduce): open reduction, assess and replace a damaged liner, ensure proper taper engagement, test stability. High recurrence risk if components are malpositioned
Constrained liner dissociation (3 to 5 percent)Acute pain and instability; radiographs show the locking ring dissociated from the liner with the head escaped from constraintPrefer dual mobility over constrained, ensure the locking ring is fully engaged, avoid excessive impingement, correct component malposition (constraint fails if impingement persists)Reoperation: remove the failed constrained liner, convert to dual mobility (preferred) or a new constrained liner with component repositioning if malpositioned. Address impingement and malposition or it will recur
Greater trochanteric non-union or migration (10 to 15 percent after advancement)Persistent lateral hip pain, Trendelenburg gait, palpable GT mobility; radiographs show GT displacement more than 2 cm proximal or non-union at 6 monthsRigid fixation (2 to 3 screws and washer, or cables), avoid over-advancement (1 to 2 cm maximum), touch-down weight-bearing 12 weeks, optimise bone health (vitamin D, calcium, bisphosphonates if osteoporotic)If asymptomatic and less than 1 cm displacement: observe (fibrous union may be adequate). If symptomatic or more than 2 cm migration: revision ORIF with cables or plate and bone graft. Chronic severe abductor deficiency: accept the limitation or a gluteus maximus transfer (less effective)
Accelerated polyethylene wear (5 to 10 percent at 5 years with dual mobility or constrained)Increasing pain, radiographic wear (decreased poly thickness), osteolysis (progressive lucencies or cysts around components)Use highly cross-linked polyethylene, optimise component position to reduce impingement, ensure adequate poly thickness (greater than 6 mm), avoid oversizing the head with thin polySurveillance with annual radiographs; liner exchange if progressive wear before osteolysis is severe; component revision if osteolysis with loosening (bone-graft lytic lesions, revise loose components)

Salvage options for failed revision (multiple recurrent dislocations despite optimal revision, unreconstructable bone loss, or chronic infection): - Girdlestone resection arthroplasty β€” remove all components and accept a shortened, painful limb. Best for the elderly, low-demand, walker-dependent patient; significant functional limitation but pain relief.

  • Hip arthrodesis β€” for the young (under 40), unilateral, high-demand patient without hip or spine arthritis. Position in 20 to 25Β° flexion, 0Β° adduction or abduction, neutral rotation. Stable and painless but causes back and knee pain; a rare last resort.
  • Bipolar hemiarthroplasty β€” when severe acetabular bone loss precludes a stable cup; accepts a higher dislocation rate but avoids a complex acetabular reconstruction.

Viva & Exam Focus


Mnemonic

DISLOCATEDISLOCATE β€” systematic assessment for THA instability

D
Direction
Posterior 75 to 90 percent (flexion, adduction, IR) versus anterior 10 to 25 percent (extension, ER, adduction)
I
Infection
Rule out with aspiration (cell count greater than 3000, PMN greater than 80 percent), ESR and CRP, cultures held 14 days
S
Safe zones
Lewinnek 40Β±10Β° inclination, 15Β±10Β° anteversion β€” CT to measure, outside the zone is roughly 4-fold risk
L
Levering or impingement
Bony (anterior wall overhang, posterior offset), component (neck-cup), soft-tissue impingement
O
Offset restoration
Compare with the contralateral side β€” femoral offset is critical for abductor tension and stability
C
Combined anteversion
Cup plus stem anteversion in the target range (roughly 25 to 50Β°, Widmer optimum near 37Β°) β€” correct both components, not the cup alone
A
Abductor function
Trendelenburg test β€” if positive, consider GT advancement (1 to 2 cm distal, increases the moment arm)
T
Tissues or capsule
Capsule often deficient or absent β€” repair if present, reconstruct with an Achilles allograft if inadequate
E
Episodes or recurrence
First dislocation 60 to 70 percent success non-operative, drops to 30 percent after the second, less than 20 percent after the third β€” operative if more than 2 episodes
Mnemonic

DUAL MOBILITYDUAL MOBILITY β€” advantages for the unstable THA

D
Delays impingement
Large outer bearing (52 to 60 mm) increases ROM before neck impingement versus a standard liner (32 to 40 mm)
U
Useful low re-dislocation
Revision series report re-dislocation typically under 5 percent (Philippot 2009: 3.7 percent), generally lower than constrained liners
A
Articulations (two)
Large metal outer bearing with the cup plus a small polyethylene inner bearing with the head β€” reduces dislocation 50 to 80 percent
L
Lower dissociation
1 to 2 percent dissociation versus 3 to 5 percent for a constrained liner (locking-mechanism failure)
M
Metal outer bearing
The large metal (not poly) bearing articulates with the cup, preserving polyethylene for the inner bearing
O
Offset preserved
Small inner head (28 to 32 mm) maintains a normal head-to-neck ratio and offset restoration
B
Better than constraint
Less wear (metal-on-poly versus poly constraint), less loosening (lower torque), higher success rate
I
Isolated liner exchange
Can replace a standard liner with dual mobility if components are well-positioned β€” the least invasive option
L
Less torque
Lower torque to fixation versus a constrained liner (no locking mechanism), reducing loosening risk
I
Instability workhorse
Leading construct for revision for instability on the evidence (Guyen 2009, Philippot 2009)
T
Two sizes
Large outer 52 to 60 mm (depends on cup size), small inner 28 to 32 mm (matches the femoral head taper)
Y
Yields stability
Large effective head lengthens jump distance (Berry 2005 head-size data; Philippot 2009 revision series)
Sciatic nerve
Location
15 to 30 mm posterior to the joint (closer in revision)
Injury pattern
Foot drop (tibial) plus foot eversion weakness (peroneal) plus posterior calf and foot sensory loss. Risk 3 to 7 percent revision versus 0.5 to 2 percent primary
Protection
Identify early with a vessel loop, keep the hip flexed more than 45Β°, avoid traction or lengthening more than 4 cm, protect during posterior capsule work
Femoral neurovascular bundle
Location
30 to 50 mm medial to the anterior joint, beneath iliopsoas
Injury pattern
Quadriceps paralysis, absent knee extension, anterior thigh numbness, catastrophic vascular injury if the femoral artery is damaged
Protection
Stay on bone with anterior retractors (Hohmann), avoid medial cement extrusion, use fluoroscopy for anterior screw placement
Superior gluteal neurovascular bundle
Location
30 to 50 mm proximal to the GT, exits the sciatic notch above piriformis
Injury pattern
Gluteus medius and minimis paralysis (Trendelenburg gait), superior buttock numbness
Protection
Avoid proximal dissection beyond the GT tip, stay in the anterior-inferior quadrant, protect during a GT osteotomy
Lateral femoral cutaneous nerve
Location
Variable, 20 to 50 mm from anterior incisions (ASIS to anterolateral thigh)
Injury pattern
Meralgia paraesthetica (lateral thigh burning or numbness)
Protection
Identify and protect when visible during the anterior approach, retract gently if encountered
Obturator neurovascular bundle
Location
20 to 30 mm medial to the acetabulum, through the obturator foramen
Injury pattern
Adductor weakness, medial thigh numbness, vascular injury rare
Protection
Avoid medial acetabular screws below the equator, use the posterior-superior quadrant for fixation
Five neurovascular danger zones of the revision hip
ZoneLocationInjury patternProtection
Sciatic nerve15 to 30 mm posterior to the joint (closer in revision)Foot drop (tibial) plus foot eversion weakness (peroneal) plus posterior calf and foot sensory loss. Risk 3 to 7 percent revision versus 0.5 to 2 percent primaryIdentify early with a vessel loop, keep the hip flexed more than 45Β°, avoid traction or lengthening more than 4 cm, protect during posterior capsule work
Femoral neurovascular bundle30 to 50 mm medial to the anterior joint, beneath iliopsoasQuadriceps paralysis, absent knee extension, anterior thigh numbness, catastrophic vascular injury if the femoral artery is damagedStay on bone with anterior retractors (Hohmann), avoid medial cement extrusion, use fluoroscopy for anterior screw placement
Superior gluteal neurovascular bundle30 to 50 mm proximal to the GT, exits the sciatic notch above piriformisGluteus medius and minimis paralysis (Trendelenburg gait), superior buttock numbnessAvoid proximal dissection beyond the GT tip, stay in the anterior-inferior quadrant, protect during a GT osteotomy
Lateral femoral cutaneous nerveVariable, 20 to 50 mm from anterior incisions (ASIS to anterolateral thigh)Meralgia paraesthetica (lateral thigh burning or numbness)Identify and protect when visible during the anterior approach, retract gently if encountered
Obturator neurovascular bundle20 to 30 mm medial to the acetabulum, through the obturator foramenAdductor weakness, medial thigh numbness, vascular injury rareAvoid medial acetabular screws below the equator, use the posterior-superior quadrant for fixation

Clinical Decision Scenarios

Practise clinical reasoning and management decisions out loud

Viva scenarioStandard
Clinical prompt

β€œA 68-year-old woman presents with her third posterior dislocation 18 months after a primary posterior-approach THR. What is your systematic approach to assessment and management?”

Viva scenarioStandard
Clinical prompt

β€œExplain dual mobility β€” how does it work, what are the advantages, and how does it compare to a constrained liner?”

Viva scenarioStandard
Clinical prompt

β€œThe cup position on CT shows inclination 42Β° and anteversion 18Β° (within the safe zone), but the patient has had 3 posterior dislocations. Intra-operatively the hip is stable with trials. What else should you assess and what is your management?”

Exam day cheat sheet
THA dislocation management β€” rapid exam review

Systematic assessment (DISLOCATE)

  • Direction: posterior 75 to 90 percent (flexion, adduction, IR) versus anterior 10 to 25 percent (extension, ER, adduction)
  • Infection: MANDATORY rule out β€” aspiration (cell count greater than 3000, PMN greater than 80 percent), ESR and CRP, cultures held 14 days
  • Safe zones: Lewinnek 40Β±10Β° inclination, 15Β±10Β° anteversion β€” CT to measure; outside the zone is roughly 4-fold risk
  • Levering or impingement: bony (anterior overhang, posterior offset), component (neck-cup), soft-tissue
  • Offset restoration: compare with the contralateral side β€” critical for abductor tension and stability
  • Combined anteversion: cup plus stem within the target zone (roughly 25 to 50Β°, Widmer optimum near 37Β°) β€” correct BOTH components if outside the range
  • Abductor function: Trendelenburg test β€” if positive, GT advancement (1 to 2 cm distal, increases the moment arm)
  • Tissues or capsule: often deficient or absent β€” repair if present, Achilles allograft reconstruction if inadequate
  • Episodes or recurrence: first 60 to 70 percent success non-operative, after the second drops to 30 percent, after the third less than 20 percent β€” operative if more than 2

Treatment algorithm (evidence-based)

  • FIRST dislocation plus well-positioned components means NON-OPERATIVE: closed reduction, hip abduction orthosis 6 to 12 weeks, precautions 12 weeks. Success 60 to 70 percent
  • RECURRENT more than 2 episodes means an OPERATIVE indication (non-operative success less than 20 percent after the third)
  • Well-positioned, stable, well-fixed components means an ISOLATED LINER EXCHANGE to dual mobility (85 to 95 percent success, least invasive)
  • Malpositioned cup (outside the safe zone) means ACETABULAR REVISION to the safe zone (40Β° inclination, 15 to 20Β° anteversion) plus dual mobility (80 to 90 percent success)
  • Malpositioned stem (excessive anteversion over 20Β° or retroversion under 5Β°) means FEMORAL REVISION to neutral (10 to 15Β° anteversion) plus a large head or dual mobility (80 to 90 percent success)
  • Abductor insufficiency (Trendelenburg) means GT ADVANCEMENT (osteotomise the GT with the abductors, advance 1 to 2 cm distally, screw fixation, touch-down weight-bearing 12 weeks) plus dual mobility
  • Capsule deficient means CAPSULAR RECONSTRUCTION with an Achilles allograft (attach the acetabular rim to the femoral neck, recreate the restraints)
  • DUAL MOBILITY is a leading construct for instability revision (Philippot 2009: 163 revision THAs, 3.7 percent dislocation, 0 percent in the recurrent-instability subgroup; Guyen 2009: 54 unstable hips restored to stability)

Dual mobility advantages (exam favourite)

  • TWO articulations: a large metal outer bearing (52 to 60 mm) with the cup plus a small polyethylene inner bearing (28 to 32 mm) with the head
  • Delays impingement: the large outer bearing increases ROM before neck contact versus a standard liner (32 to 40 mm) β€” the KEY mechanism
  • Reduces dislocation by lengthening jump distance and delaying impingement (large effective head β€” Berry 2005 head-size data; Philippot 2009 revision series)
  • Generally favoured over constrained liners for instability: lower dislocation and it avoids the constrained-liner catastrophic dissociation mode; reserve constraint for the salvage hip
  • Isolated liner exchange option: if components are well-positioned, replace the standard liner with dual mobility β€” preserves bone stock
  • Evidence: Philippot 2009 (163 revision THAs, 3.7 percent dislocation, 0 percent in the recurrent-instability subgroup, 7-year cup survival 96.1 percent), Guyen 2009 (54 unstable hips restored to stability)
  • Trade-off: surveil for polyethylene wear and rare intraprosthetic (inner-bearing) dissociation

Danger zones (5 critical neurovascular)

  • Sciatic nerve: 15 to 30 mm posterior (closer in revision); injury causes foot drop plus sensory loss. PROTECT: identify early, vessel loop, hip flexed more than 45Β°, avoid traction or lengthening more than 4 cm. Risk 3 to 7 percent revision versus 0.5 to 2 percent primary
  • Femoral neurovascular: 30 to 50 mm medial to the anterior joint; injury catastrophic. PROTECT: stay on bone with anterior retractors, avoid medial cement, fluoroscopy for anterior screws
  • Superior gluteal neurovascular: 30 to 50 mm proximal to the GT; injury causes abductor weakness. PROTECT: avoid proximal dissection beyond the GT, stay in the anterior-inferior quadrant
  • Lateral femoral cutaneous: variable, 20 to 50 mm from anterior incisions; injury causes meralgia paraesthetica. PROTECT: identify and retract gently
  • Obturator neurovascular: 20 to 30 mm medial to the acetabulum; injury causes adductor weakness. PROTECT: avoid medial screws below the equator, use the posterior-superior quadrant

Intra-operative stability testing (CRITICAL)

  • Posterior stability (MOST IMPORTANT for posterior dislocation): 90Β° flexion plus adduction plus IR with a posterior force β€” must be stable with a firm endpoint
  • Anterior stability: extension plus ER plus adduction β€” must remain reduced
  • Mid-range: 45Β° flexion, neutral rotation, abduction and adduction β€” assess soft-tissue tension
  • Extremes: full flexion, extension, abduction, adduction β€” identify impingement zones
  • Shuck test: quantify translation (should be minimal, less than 5 mm)
  • If UNSTABLE β€” options: increase head size, change to dual mobility, adjust component position, increase soft-tissue tension
  • RULE: DO NOT ACCEPT instability on the table β€” it will fail post-operatively. The hip must be rock-solid throughout ROM before closing

Combined anteversion

  • Principle: cup anteversion plus stem anteversion should fall in a target zone (roughly 25 to 50Β°; Widmer optimum near 37Β°)
  • Example IN-ZONE: cup 20Β° plus stem 17Β° equals 37Β° total
  • Example HIGH (over-anteverted): cup 30Β° plus stem 25Β° equals 55Β° β€” ANTERIOR instability and edge-loading risk (the head levers out in extension and ER)
  • Example LOW (under-anteverted or retroverted): cup 10Β° plus stem 5Β° equals 15Β° β€” POSTERIOR instability and impingement risk in flexion and IR
  • Application: correct BOTH components if combined anteversion is outside the target zone β€” one malpositioned component may not be enough
  • Assessment: CT to measure cup anteversion AND stem version β€” both contribute to overall stability
  • Management: if combined anteversion is wrong, you may need to revise BOTH the cup and the stem even if each component individually appears acceptable

Post-operative protocol (compliance critical)

  • Hip abduction orthosis (brace): 6 to 12 weeks total (day and night 6 weeks, then night-time only 6 weeks)
  • Hip precautions MINIMUM 12 weeks (consider lifelong if high risk): posterior approach β€” no flexion more than 90Β°, no adduction past midline, no IR. Anterior β€” no extension, no ER, no adduction
  • Weight-bearing: WBAT immediately UNLESS GT advancement was performed (then touch-down weight-bearing 12 weeks until union is confirmed)
  • DVT prophylaxis: extended 4 to 6 weeks (high risk) β€” LMWH or DOAC per protocol
  • Follow-up: 2 weeks (wound), 6 weeks (X-ray, advance activity), 3 months (X-ray, wean brace), 6 months, annually for life
  • Patient education: compliance determines success β€” non-compliance is the most common cause of failure after revision

Outcomes and salvage (realistic expectations)

  • Non-operative (first dislocation): roughly 60 to 70 percent remain stable; success falls steeply with each further episode (weak evidence base)
  • Dual mobility: low re-dislocation in revision series (Philippot 2009: 3.7 percent, 0 percent in the recurrent-instability subgroup) β€” the leading construct
  • Constrained liner: a recognised catastrophic failure mode (locking-ring or liner dissociation) β€” reserve for salvage (irreparable abductors or soft tissue)
  • Component revision alone (no large head, dual mobility or constraint): higher re-dislocation β€” correcting position is necessary but often not sufficient
  • WORSE prognosis: neuromuscular or cognitive disorders (Parkinson's, stroke, dementia β€” a relative risk; registry data are mixed, do not quote a fixed failure rate), severe abductor deficiency, multiple prior revisions, a stiff or fused spine
  • SALVAGE options after failed multiple revisions: Girdlestone resection (elderly, low demand, accept a shortened painful limb), hip arthrodesis (young, unilateral, high demand, rare), bipolar hemiarthroplasty (severe bone loss)
  • Counsel realistic expectations: recurrent instability after multiple revisions may need acceptance of limitation or salvage β€” there is no perfect solution

Background & Evidence


Epidemiology. Dislocation is consistently one of the leading indications for revision THA across the major registries (AOANJRR, the National Joint Registry for England, Wales, Northern Ireland and the Isle of Man, the Nordic Arthroplasty Register Association, and AJRR). Posterior dislocation predominates (75 to 90 percent), and the cumulative dislocation risk after a primary THA rises over time. Why the safe zone is necessary but not sufficient. Lewinnek (1978) established the 40Β±10Β° inclination and 15Β±10Β° anteversion target from 300 THAs (9 dislocations, 3 percent), finding 1.5 percent dislocation inside versus 6.1 percent outside. Abdel (2016) re-examined 206 dislocated THAs from 9784 primaries and found 58 percent sat WITHIN the zone β€” instability is multifactorial, and for some patients the ideal cup position lies outside the historical zone. The exam message: quote Lewinnek for the numbers, then Abdel for the nuance. Why dual mobility works β€” the head-size rationale. Berry (2005) showed in 21,047 primary THAs that larger femoral heads reduce dislocation (relative risk 1.7 for 22 mm versus 32 mm heads), with the greatest benefit in the posterolateral approach (10-year dislocation 12.1 percent for 22 mm versus 3.8 percent for 32 mm heads). Dual mobility exploits this by providing a large effective head (the 52 to 60 mm outer bearing) that lengthens jump distance and delays impingement, while the small inner bearing preserves the head-to-neck ratio. Spinopelvic stiffness β€” the 'well-positioned but unstable' hip. Pelvic tilt changes the functional cup orientation between standing and sitting. A stiff or fused spine (a long lumbar fusion, flatback deformity, or loss of lordosis in the elderly) that cannot posteriorly tilt on sitting markedly raises posterior dislocation risk. Assess with lateral standing and sitting radiographs (the pelvic tilt change) and individualise cup anteversion accordingly (more anteversion if anterior tilt, less if posterior tilt). Registries and global practice. The major registries report lower revision-for-dislocation rates with dual mobility versus conventional single bearings, with rising dual-mobility use in high-risk primary and revision-for-instability settings; the trade-off is polyethylene wear and rare intraprosthetic (inner-bearing) dissociation, mandating lifelong radiographic surveillance. For surgical antibiotic prophylaxis the globally concordant principle is a weight-adjusted first-generation cephalosporin (e.g. cefazolin) within 60 minutes of incision (AAOS, NICE and SIGN, WHO surgical site infection guidance), with vancomycin or clindamycin for true beta-lactam allergy and a single pre-incision dose (routine continuation beyond 24 hours is not supported). Name the principle rather than country-specific brand names, codes or billing items.

References


Evidence

Dislocations after total hip-replacement arthroplasties (origin of the Lewinnek safe zone)

Level IV
Lewinnek GE, Lewis JL, Tarr R, Compere CL, Zimmerman JR β€’ J Bone Joint Surg Am (1978)
Key Findings:
  • 300 THAs; 9 dislocations (3 percent)
  • Dislocation rate 1.5 percent when cup anteversion was 15Β±10Β° and inclination 40Β±10Β°, versus 6.1 percent outside this range (roughly 4-fold higher)
  • Anterior dislocation was associated with increased cup anteversion; the greatest overall risk was in the first 30 days and in hips with prior surgery
Clinical implication: Defines the classic acetabular orientation target (40Β±10Β° inclination, 15Β±10Β° anteversion). Useful as a starting reference but, as later work shows, not a guarantee of stability.
Verify on PubMed (PMID 641088)
Evidence

What Safe Zone? The vast majority of dislocated THAs are within the Lewinnek safe zone for acetabular component position

Level III
Abdel MP, von Roth P, Jennings MT, Hanssen AD, Pagnano MW β€’ Clin Orthop Relat Res (2016)
Key Findings:
  • 206 dislocated THAs from a cohort of 9784 primaries
  • 58 percent of dislocated hips had a cup WITHIN the Lewinnek safe zone (84 percent within the inclination target, 69 percent within the anteversion target)
  • Instability is multifactorial; the ideal cup position for some patients lies outside the historical zone
Clinical implication: A cup inside the Lewinnek zone does NOT exclude a positional cause of instability. Interpret cup position together with stem version, combined anteversion, offset and spinopelvic mobility rather than relying on the safe zone alone.
Verify on PubMed (PMID 26150264)
Evidence

Effect of femoral head diameter and operative approach on risk of dislocation after primary total hip arthroplasty

Level IV
Berry DJ, von Knoch M, Schleck CD, Harmsen WS β€’ J Bone Joint Surg Am (2005)
Key Findings:
  • 21,047 primary THAs; cumulative dislocation 2.2 percent at 1 year rising to 6.0 percent at 20 years
  • Larger femoral head reduced dislocation: relative risk 1.7 for 22 mm versus 32 mm heads
  • The head-size benefit was greatest with the posterolateral approach (10-year dislocation 12.1 percent for 22 mm versus 3.8 percent for 32 mm heads)
Clinical implication: Increasing the effective head diameter (large heads or a dual-mobility outer bearing) lengthens jump distance and reduces dislocation β€” the biomechanical rationale underpinning dual mobility in the unstable hip.
Verify on PubMed (PMID 16264121)
Evidence

Use of a dual mobility socket to manage total hip arthroplasty instability

Level IV
Guyen O, Pibarot V, Vaz G, Chevillotte C, BΓ©jui-Hugues J β€’ Clin Orthop Relat Res (2009)
Key Findings:
  • 54 unstable THAs revised to an unconstrained dual mobility (tripolar) cup
  • Stability restored and maintained; one early re-dislocation managed by closed reduction, two intraprosthetic dissociations from technical error
  • Mean follow-up roughly 4 years with satisfactory osseointegration and no reported osteolysis
Clinical implication: Supports dual mobility as an effective option for the unstable THA, while highlighting that intraprosthetic dissociation and technical accuracy matter. The evidence is short-to-mid-term, single-centre.
Verify on PubMed (PMID 18780135)
Evidence

Prevention of dislocation in total hip revision surgery using a dual mobility design

Level IV
Philippot R, Adam P, Reckhaus M, Delangle F, Verdot FX, Curvale G, Farizon F β€’ Orthop Traumatol Surg Res (2009)
Key Findings:
  • 163 revision THAs with a dual mobility cup (110 cementless, 53 cemented in a Kerboull ring)
  • Overall dislocation 3.7 percent at a mean 60 months; 7-year cup survivorship 96.1 percent
  • Dislocation 2.9 percent in aseptic loosening, 9 percent in revision for infection, 0 percent in revision for recurrent instability
Clinical implication: In revision THA, dual mobility achieves dislocation rates comparable to primary single-bearing implants and is especially valuable in the high-instability-risk hip where constrained components were traditionally used.
Verify on PubMed (PMID 19656750)
Evidence

Epidemiology of dislocation after total hip arthroplasty

Level III
Meek RM, Allan DB, McPhillips G, Kerr L, Howie CR β€’ Clin Orthop Relat Res (2006)
Key Findings:
  • Epidemiological study of dislocation after primary THA
  • In their cohort, Parkinson's disease and prior stroke were NOT associated with an increased rate of dislocation
  • Challenges the traditional teaching of a fixed high failure rate in neuromuscular patients
Clinical implication: Neuromuscular conditions are relative, not absolute, risk factors for instability β€” treat them as a reason to favour a stability-enhancing construct rather than a fixed quoted failure rate.
Verify on PubMed (PMID 16672897)
Evidence

The dislocating hip arthroplasty: prevention and treatment

Level V
Patel PD, Potts A, Froimson MI β€’ J Arthroplasty (2007)
Key Findings:
  • Review of the prevention and treatment of the dislocating THA
  • Instability is multifactorial and should be assessed with a systematic algorithm
  • Operative intervention is generally reserved for more than two dislocations and must correct the identified underlying cause
Clinical implication: Provides the simple, widely taught framework for the workup and the threshold for surgery in the unstable THA.
Verify on PubMed (PMID 17570285)
Editorially reviewed β€” transparent references and correction processPublished by OrthoVellum Medical Education TeamEditorial boardMethodologyReview policy
Educational disclosure

Educational content is reviewed for source visibility, editorial coherence, and correction readiness.

No individual clinician credential is claimed unless a named person is shown.

Verify before clinical use; this is not medical advice or a substitute for local guidance.

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