Skip to main content
OrthoVellum
Knowledge Hub

Study

  • Topics
  • MCQs
  • ISAWE
  • Operative Surgery
  • Flashcards

Company

  • About Us
  • Editorial Policy
  • Contact
  • FAQ
  • Blog

Legal

  • Terms of Service
  • Privacy Policy
  • Cookie Policy
  • Medical Disclaimer
  • Copyright & DMCA
  • Refund Policy

Support

  • Help Center
  • Accessibility
  • Report an Issue
OrthoVellum

© 2026 OrthoVellum. For educational purposes only.

Not affiliated with the Royal Australasian College of Surgeons.

Cam-Type Femoroacetabular Impingement

Back to Topics
Contents
0%

Cam-Type Femoroacetabular Impingement

Comprehensive guide to cam-type FAI diagnosis and management for FRCS exam preparation

complete
Updated: 2025-12-25
High Yield Overview

CAM-TYPE FAI

Aspherical Femoral Head | Males | Anterosuperior

CamFemoral-side pathology
AlphaAngle greater than 55 degrees
MalesPredominantly affected
ASAnterosuperior labrum/cartilage

FAI Types

Cam
PatternAspherical head
TreatmentFemoral bump/pistol grip
Pincer
PatternOvercoverage
TreatmentAcetabular-side
Combined
PatternBoth elements
TreatmentMost common (86%)

Critical Must-Knows

  • Aspherical femoral head with loss of head-neck offset
  • Alpha angle greater than 55° on lateral view is diagnostic
  • Young athletic males predominantly affected
  • Anterosuperior labral and cartilage damage from shear mechanism
  • Hip arthroscopy or surgical dislocation for treatment

Examiner's Pearls

  • "
    Pistol grip deformity on AP pelvis
  • "
    FADIR test positive (flexion, adduction, internal rotation)
  • "
    Outside-in cartilage damage (acetabular side first)
  • "
    Associated with early osteoarthritis

Clinical Imaging

Imaging Gallery

A cam-type impingement lesion (black arrow) and adjacent labrum (white arrow) of the right hip viewed from the peripheral compartment during hip arthroscopy.Figure 1A. Femoral head-neck junction. Figu
Click to expand
A cam-type impingement lesion (black arrow) and adjacent labrum (white arrow) of the right hip viewed from the peripheral compartment during hip arthrCredit: Jayasekera N et al. via PLoS ONE via Open-i (NIH) (Open Access (CC BY))
Radiographs of the right hip demonstrating a case of mixed Cam- and Pincer-type femoroacetabular impingement in a 17-year-old hockey goaltender. (A) The orange arrow points to the Cam bump in the AP r
Click to expand
Radiographs of the right hip demonstrating a case of mixed Cam- and Pincer-type femoroacetabular impingement in a 17-year-old hockey goaltender. (A) TCredit: Open-i / NIH via Open-i (NIH) (Open Access (CC BY))
Anteroposterior radiographs illustrate the case of an 85-year-old female patient who had secondary OA caused by Cam type FAI of left hip. (A) Preoperative radiograph shows typical pistol grip deformit
Click to expand
Anteroposterior radiographs illustrate the case of an 85-year-old female patient who had secondary OA caused by Cam type FAI of left hip. (A) PreoperaCredit: Open-i / NIH via Open-i (NIH) (Open Access (CC BY))
Preoperative computed tomography (CT) 3-dimensional reconstruction in a 24-year-old male professional rugby player (patient 4 in Table 1): (A) anteroposterior (AP) view and (B) lateral view of the rig
Click to expand
Preoperative computed tomography (CT) 3-dimensional reconstruction in a 24-year-old male professional rugby player (patient 4 in Table 1): (A) anteropCredit: Open-i / NIH via Open-i (NIH) (Open Access (CC BY))

Critical Cam FAI Exam Points

Pathoanatomy

Aspherical femoral head creates bump at head-neck junction. Lost offset causes impingement on flexion + internal rotation. Anterosuperior location most common.

Alpha Angle

Measured on lateral view (Dunn, cross-table, MRI). Angle between femoral neck axis and point where head becomes aspherical. Greater than 55° = cam morphology.

Cartilage Damage

Outside-in mechanism: Cam bump shears acetabular cartilage from labrum, causing delamination. Differs from pincer (labral crushing, central cartilage damage).

Treatment

Femoral osteochondroplasty: Resect cam bump to restore sphericity. Arthroscopic (most common) or open surgical dislocation. Labral repair/debridement as needed.

At a Glance

Cam-type femoroacetabular impingement (FAI) results from an aspherical femoral head with loss of head-neck offset, creating a bump that impinges on the acetabular rim during hip flexion and internal rotation. It predominantly affects young athletic males and causes anterosuperior labral and cartilage damage through an outside-in shear mechanism (acetabular cartilage delamination from the labrum). Diagnosis requires an alpha angle greater than 55° on lateral radiographs or MRI (Dunn view). The "pistol grip" deformity is visible on AP pelvis, and clinical examination reveals a positive FADIR test. Treatment is femoral osteochondroplasty (arthroscopic or open surgical dislocation) to restore head sphericity, with concurrent labral repair. Combined cam-pincer morphology is actually most common (86% of FAI cases).

Mnemonic

CAM-PINCam vs Pincer

C
Cam = Femoral
Aspherical head
A
Alpha angle elevated
Greater than 55 degrees
M
Males predominant
Young athletes
P
Pincer = Acetabular
Overcoverage
I
In females more common
Middle-aged women
N
Normal femoral head
Problem is socket

Memory Hook:CAM is Males/Femoral, PIN is Females/Acetabular!

Mnemonic

55-CAMAlpha Angle

55
Threshold 55 degrees
Greater than 55 is abnormal
C
Cross-table lateral
Or Dunn view
A
Axis of neck to
Point of asphericity
M
MRI can also measure
Radial sequences best

Memory Hook:55 degrees is the threshold for CAM morphology!

Mnemonic

SHEARCartilage Damage Pattern

S
Shear forces
Cam bump shears cartilage
H
Head-neck junction
Location of cam lesion
E
Edge of labrum
Cartilage stripped from labrum
A
Acetabular cartilage
Outside-in damage pattern
R
Rim damage
Anterosuperior quadrant

Memory Hook:SHEAR describes cam damage: cartilage sheared from labrum outside-in!

Overview and Epidemiology

Femoroacetabular impingement (FAI) is a recognized cause of hip pain and early osteoarthritis, particularly in young adults. Cam-type FAI is the most common form, characterized by an aspherical femoral head.

Epidemiology:

  • Prevalence: Cam morphology present in 10-15% of asymptomatic population
  • Demographics: Young athletic males predominantly affected
  • Age: Typically presents 20-40 years of age
  • Sports: High risk in soccer, hockey, martial arts, rowing
  • Association: Strong link to early-onset hip osteoarthritis

Pathophysiology and Pathoanatomy

Cam-type femoroacetabular impingement (FAI) occurs when an aspherical femoral head impinges on the acetabulum during flexion and internal rotation. The "cam" refers to the bump that acts like a cam in an engine, causing abnormal contact.

Pathoanatomy

The cam lesion is an abnormal bony prominence at the anterosuperior femoral head-neck junction, causing loss of normal head-neck offset. This may result from abnormal epiphyseal development, particularly in young athletes during growth.

Mechanism of Damage

During hip flexion and internal rotation, the cam bump is driven into the anterosuperior acetabulum. This causes shear forces that strip the acetabular cartilage from the labrum ("outside-in" damage). The labrum is also crushed. This differs from pincer mechanism.

Clinical Presentation

History

Young athletic males typically present with groin pain. Pain is activity-related, particularly with sports requiring hip flexion (soccer, hockey, martial arts). May describe mechanical symptoms such as clicking or catching. Prolonged sitting may aggravate symptoms.

Examination

FADIR Test (Anterior Impingement Test): Flexion, Adduction, Internal Rotation reproduces groin pain. This is the most sensitive test for FAI.

FABER Test: Flexion, Abduction, External Rotation assesses SI joint but also stresses the hip.

Range of Motion: Internal rotation in flexion is typically reduced and painful.

Gait: Usually normal unless advanced damage.

Diagnosis

AP X-ray of the right hip demonstrating cam-type femoroacetabular impingement with pistol grip deformity
Click to expand
AP X-ray of the right hip showing cam-type femoroacetabular impingement. The classic 'pistol grip' deformity is visible - note the loss of normal concavity at the femoral head-neck junction, creating an aspherical femoral head with a bump at the anterosuperior head-neck junction. This morphology causes impingement on the acetabular rim during hip flexion and internal rotation. On lateral views (Dunn, cross-table), the alpha angle can be measured - greater than 55 degrees confirms cam morphology. The mechanism involves this bony prominence being driven into the anterosuperior acetabulum during hip flexion, causing shear forces that strip acetabular cartilage from the labrum (outside-in damage pattern).Credit: Ruiz Santiago et al., Radiology Research and Practice 2016 - CC BY 4.0

AP Pelvis: May show "pistol grip" deformity (loss of concavity at head-neck junction). Assess for coxa profunda or other acetabular abnormalities suggesting combined morphology.

Lateral View (Dunn, Cross-Table, Frog Lateral): Essential for measuring alpha angle. Shows anterosuperior bump.

Alpha Angle: Angle between the femoral neck axis and a line from the center of the femoral head to the point where the head becomes aspherical. Greater than 55° indicates cam morphology.

MRI/MRA: Shows labral and cartilage damage. Radial sequences best for assessing cam lesion circumferentially. Arthrogram improves sensitivity for labral tears.

Alpha Angle Classification:

  • Normal: Less than 50°
  • Borderline: 50-55°
  • Cam morphology: Greater than 55°

Combined FAI: Most common presentation (approximately 86%). Both cam and pincer elements present.

Severity: Based on cartilage and labral damage on MRI/arthroscopy.

Management

📊 Management Algorithm
Management algorithm for Cam Fai
Click to expand
Management algorithm for Cam FaiCredit: OrthoVellum

Activity Modification: Avoid provocative activities (deep hip flexion, impact sports).

Physiotherapy: Core strengthening, hip stability, range of motion exercises. Address any muscular imbalances.

NSAIDs/Analgesia: Symptomatic relief.

Injection: Intra-articular local anesthetic/steroid can confirm hip as pain source and provide temporary relief.

Conservative treatment may provide symptom relief but does not address underlying morphology or prevent progression of cartilage damage.

Indications: Symptomatic cam FAI with confirmed morphology and labral/cartilage damage. Failed conservative treatment. Realistic expectations.

Femoral Osteochondroplasty: Resection of cam lesion to restore spherical head-neck junction.

Approaches:

  • Hip Arthroscopy: Most common. Peripheral compartment accessed, cam resected with burr.
  • Surgical Dislocation: Open approach with trochanteric osteotomy. Protected blood supply. Allows direct visualization.

Adjunct Procedures: Labral repair (preferred over debridement), microfracture for cartilage defects.

Outcomes: Good outcomes in patients with preserved joint space and repairable labrum. Poor prognostic factors: advanced cartilage loss, older age, high BMI.

Evidence Base

Conceptual
📚 Ganz et al
Key Findings:
  • Defined FAI as cause of hip OA
  • Described cam and pincer mechanisms
  • Foundation for modern understanding
  • Led to surgical treatment development
Clinical Implication: Landmark paper establishing FAI concept.
Source: Clin Orthop 2003

I (RCT)
📚 UK FASHIoN Trial
Key Findings:
  • Hip arthroscopy vs conservative for FAI
  • Arthroscopy superior at 12 months
  • Improved hip function scores
  • 35% of conservative group crossed to surgery
Clinical Implication: Supports hip arthroscopy for FAI in appropriate patients.
Source: BMJ 2018

II
📚 Agricola et al
Key Findings:
  • Cam morphology predicts hip OA development
  • Higher alpha angles = higher OA risk
  • Longitudinal cohort data
  • Supports early intervention concept
Clinical Implication: Cam FAI is a risk factor for osteoarthritis.
Source: Osteoarthritis Cartilage 2013

I (RCT)
📚 FIRST Trial (Griffin et al)
Key Findings:
  • Arthroscopy vs physiotherapy-led care for FAI
  • 12-month primary endpoint showed no significant difference
  • Both groups improved from baseline
  • High crossover rate in physiotherapy group (24%)
Clinical Implication: Emphasizes importance of patient selection and shared decision-making in FAI management.
Source: Lancet 2018

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Young Male with Groin Pain

EXAMINER

"A 25-year-old male soccer player has right groin pain worse with kicking. FADIR test is positive. How do you assess and manage him?"

EXCEPTIONAL ANSWER
This presentation in a young male athlete with groin pain and positive FADIR suggests femoroacetabular impingement, likely cam-type given his demographics. My history would confirm pain location, aggravating activities (hip flexion, internal rotation), duration, and mechanical symptoms. On examination, the positive FADIR (flexion, adduction, internal rotation) suggests anterior impingement. I would assess range of motion, particularly internal rotation in flexion which is typically reduced in cam FAI. I would obtain AP pelvis and lateral hip radiographs. On AP I would look for pistol grip deformity. On lateral (Dunn or cross-table), I would measure the alpha angle - greater than 55 degrees confirms cam morphology. I would also assess for pincer elements as most FAI is combined type. MRI with or without arthrogram would show labral and cartilage damage. Cam FAI causes outside-in cartilage damage due to the aspherical head shearing acetabular cartilage from the labrum. For management, I would initially trial conservative treatment with activity modification and physiotherapy. However, if symptoms persist and imaging confirms correctable morphology with repairable damage, surgical treatment is indicated. Hip arthroscopy with femoral osteochondroplasty (resection of cam bump) and labral repair would address the pathology. Evidence from the UK FASHIoN trial supports arthroscopy over conservative management.
KEY POINTS TO SCORE
FADIR positive suggests FAI
Alpha angle greater than 55 degrees = cam
Outside-in cartilage damage pattern
Arthroscopic osteochondroplasty is treatment
COMMON TRAPS
✗Not measuring alpha angle
✗Not knowing difference between cam and pincer
✗Missing combined pathology
LIKELY FOLLOW-UPS
"What is the mechanism of cartilage damage in cam FAI?"
"What are poor prognostic factors for surgery?"
VIVA SCENARIOChallenging

Scenario 2: Bilateral Cam Morphology with Unilateral Symptoms - Diagnostic Dilemma

EXAMINER

"You are seeing a 26-year-old professional footballer in your sports clinic with 9 months of right groin pain. The pain is worse with running, cutting, and deep squatting. He has failed 4 months of physiotherapy and activity modification. His plain X-rays show bilateral cam morphology with alpha angles of 62° on the right and 58° on the left. He has no symptoms in his left hip whatsoever. An MRI arthrogram of his right hip shows an anterosuperior labral tear and Outerbridge grade II chondral changes to the acetabulum. He is asking about hip arthroscopy for his right hip, but is also concerned because he has 'the same bone problem' in his left hip and wants to know if he should have both hips operated on at the same time to prevent future problems. How do you counsel this patient about surgical management?"

EXCEPTIONAL ANSWER
This patient presents with **symptomatic cam FAI on the right** and **asymptomatic cam morphology on the left** - a common diagnostic dilemma that tests understanding of the difference between **morphology** (structural abnormality) and **disease** (symptomatic impingement). The key principle is that **cam morphology is common in the asymptomatic population (10-15% prevalence)** and the presence of morphology alone is **NOT an indication for surgery**. I would counsel as follows: **Right Hip - Symptomatic FAI Management:** The right hip has **clear indications for arthroscopic treatment**: (1) Persistent symptoms greater than 6 months despite conservative management, (2) Positive FADIR test, (3) Alpha angle greater than 55° (62°), (4) Labral tear confirmed on MRI arthrogram, (5) Outerbridge grade I-II cartilage (favorable prognostic factor). Before proceeding, I would perform a **diagnostic intra-articular injection** (10ml 0.5% bupivacaine ± 40mg triamcinolone) - this is **critical** because: (1) It confirms the hip joint is the pain source (greater than 80% temporary relief expected), (2) Sets realistic expectations for surgical outcomes, (3) Rules out extra-articular pathology (iliopsoas tendinopathy, athletic pubalgia). **Hip Arthroscopy Technique:** Central and peripheral compartment evaluation, femoral osteochondroplasty to reduce alpha angle to less than 50°, labral repair preferred over debridement (better long-term outcomes), capsular closure. **Evidence base:** UK FASHIoN trial (BMJ 2018, Level I RCT) showed arthroscopy superior to conservative management at 12 months for symptomatic FAI with labral tears. FIRST trial (Lancet 2018, Level I RCT) showed no significant difference but had 24% crossover to surgery, suggesting patient selection critical. **Left Hip - Asymptomatic Morphology Management:** The left hip should be **observed only** - prophylactic surgery is **not indicated**. Counseling points: (1) Cam morphology prevalence 10-15% in asymptomatic population, particularly high in athletes (up to 30% in certain sports), (2) **Morphology does not equal disease** - many people with cam morphology never develop symptoms, (3) No evidence that prophylactic surgery prevents future FAI or OA, (4) Surgery carries risks (1-2% complications including iatrogenic cartilage damage, infection, nerve injury), (5) **Observation protocol**: Annual clinical review, educate about FAI symptoms (groin pain, FADIR positive, reduced internal rotation), advise to return if symptoms develop. **Prognostic Factors for Right Hip Surgery:** **Good prognostic factors** (this patient has most): Age less than 40 (26 years), Tonnis grade 0-1 (preserved joint space), Outerbridge I-II cartilage, labral tear pattern suitable for repair, realistic expectations. **Poor prognostic factors to assess**: BMI greater than 30, workers' compensation, psychiatric comorbidities. Expected outcomes: 75-85% good-excellent results at 5 years, 10-15% conversion to THA at 10 years if advanced cartilage damage present. **Return to Sport:** For professional footballer: Graded rehabilitation protocol 4-6 months, expect 80-90% return to pre-injury sport level at 12 months (studies in elite athletes), maintain hip ROM and core strength lifelong.
KEY POINTS TO SCORE
Cam morphology common in asymptomatic population (10-15%, up to 30% in athletes): Morphology alone NOT indication for surgery, many people with cam morphology never develop symptoms, prophylactic surgery not indicated
Symptomatic vs asymptomatic FAI - key distinction: Symptomatic FAI requires (1) symptoms greater than 6 months, (2) positive clinical tests (FADIR), (3) morphology on imaging (alpha greater than 55°), (4) labral/chondral pathology on MRI; Asymptomatic morphology requires observation only
Diagnostic intra-articular injection critical before surgery: Confirms hip joint as pain source (greater than 80% temporary relief), sets realistic surgical expectations, rules out extra-articular causes (iliopsoas, athletic pubalgia, lumbar referred pain)
MRI arthrogram assesses surgical suitability: Labral tear pattern (anterosuperior typical for cam), Outerbridge grading (I-II favorable, III-IV poor prognosis), Tonnis grade (0-1 required, ≥2 contraindication to arthroscopy)
Evidence-based surgical decision: UK FASHIoN trial (BMJ 2018) - arthroscopy superior to conservative at 12 months; FIRST trial (Lancet 2018) - no difference but 24% crossover; Good prognostic factors: age less than 40, Tonnis 0-1, Outerbridge I-II, realistic expectations
COMMON TRAPS
✗Operating on asymptomatic morphology 'prophylactically' - no evidence base, exposes patient to unnecessary surgical risk, violates principle of primum non nocere
✗Not performing diagnostic injection before surgery - injection confirms hip as pain source and sets expectations (if injection gives less than 50% relief, question diagnosis)
✗Ignoring cartilage grading on MRI - Outerbridge III-IV poor prognostic factor for arthroscopy, may require counseling about risk of progression to THA
✗Not knowing evidence base (UK FASHIoN vs FIRST trials) - examiners expect awareness of Level I evidence for common sports procedures
✗Promising elite athlete 100% return to sport - realistic counseling: 80-90% return to pre-injury level at 12 months, some require sport modification
LIKELY FOLLOW-UPS
"What is the prevalence of cam morphology in asymptomatic athletes?"
"What Outerbridge grade on MRI would make you reconsider arthroscopy?"
"What is the difference in findings between the UK FASHIoN and FIRST trials?"
VIVA SCENARIOCritical

Scenario 3: Failed Cam Osteochondroplasty - Progression to Early Osteoarthritis

EXAMINER

"You are seeing a 28-year-old man in your clinic who underwent right hip arthroscopy with cam osteochondroplasty and labral repair 18 months ago. He initially improved for 6 months post-operatively but has had progressive worsening of groin pain over the past year. He now has constant pain with activities of daily living, difficulty with stairs, and uses a cane for walking. On examination, he has a positive FADIR test, marked restriction of internal rotation (5° vs 30° on the left), and an antalgic gait. His pre-operative X-rays (which you obtain from the referring surgeon) show cam morphology with an alpha angle of 68° but also Tonnis grade 1 osteoarthritis. His new X-rays show Tonnis grade 2 osteoarthritis with joint space narrowing to 2mm (previously 3mm), subchondral sclerosis, and early cyst formation. MRI shows complete loss of acetabular cartilage (Outerbridge grade IV) in the anterosuperior quadrant. The patient is devastated that the surgery 'failed' and is now reading online about revision hip arthroscopy. What is your assessment and management plan?"

EXCEPTIONAL ANSWER
This is a case of **rapid progression to osteoarthritis following hip arthroscopy** - likely representing **patient selection error** rather than technical failure. The critical issue is that this patient had **pre-existing Tonnis grade 1 OA** and has now progressed to **Tonnis grade 2 OA** within 18 months, suggesting **advanced chondral damage was present at the time of initial surgery**. Tonnis grade 2 OA is a **contraindication to revision hip arthroscopy**. The patient needs counseling about **conservative management to delay THA** given his young age (28 years), as THA in patients less than 55 has a **15-20% revision rate at 15 years** (AOANJRR data). **Assessment of 'Failed' Arthroscopy:** **Patient selection error:** Pre-operative Tonnis grade 1 OA is a **relative contraindication** to hip arthroscopy - should have prompted detailed counseling about risk of progression. The pre-operative MRI (if performed) likely showed Outerbridge grade III-IV changes that predicted poor outcome. **Poor prognostic factors** in this case: (1) Tonnis grade 1 pre-operatively, (2) Rapid progression to grade 2 (suggests advanced cartilage damage), (3) Young age with early OA (mechanical factors, ? genetics). **Technical factors to assess**: Was adequate femoral osteochondroplasty performed (post-op alpha angle should be less than 50°)? Was labral repair performed vs debridement? Any iatrogenic cartilage damage during surgery? **Current Status - Tonnis Grade 2 OA:** **Tonnis grading system**: Grade 0 (normal), Grade 1 (sclerosis, slight narrowing, marginal osteophytes), Grade 2 (small cysts, moderate narrowing, moderate sclerosis), Grade 3 (large cysts, severe narrowing, deformity). This patient has **Grade 2** with joint space 2mm (normal 4-6mm), subchondral sclerosis, early cysts. **MRI Outerbridge IV** (exposed bone) in anterosuperior acetabulum confirms advanced cartilage loss. **Revision arthroscopy is contraindicated** - Tonnis ≥2 and Outerbridge IV predict failure of further joint preservation surgery. **Management - Conservative to Delay THA:** Given age 28 years, goal is to **delay THA as long as possible** to reduce lifetime revision burden. **Conservative measures**: (1) **Activity modification** - avoid high-impact activities (running, jumping), low-impact exercise encouraged (cycling, swimming, walking), weight management critical, (2) **Physiotherapy** - hip ROM exercises, core strengthening, gait training to reduce antalgic pattern, (3) **Pharmacological** - regular paracetamol, NSAIDs (celecoxib 200mg daily, PPI cover), tramadol for breakthrough pain, (4) **Intra-articular corticosteroid injection** - may provide 3-6 months symptom relief (triamcinolone 40mg or methylprednisolone 40mg), temporary measure, repeat 6-monthly maximum, (5) **Walking aids** - single point stick reduces hip joint load by 20-30%. **Counseling About THA:** **Timing**: THA indicated when conservative measures fail and pain affects quality of life/function. **No absolute age cutoff**, but younger age increases lifetime revision risk. **THA outcomes in young patients**: 90-95% survivorship at 10 years, but **15-20% revision rate at 15 years in patients less than 55** (AOANJRR data). **Revision burden**: If THA performed at age 30, likely requires 2-3 revisions over lifetime (revisions at 50, 65, 80). **Each revision more complex** with bone loss, worse outcomes. **Goal**: Delay THA by even 5-10 years significantly reduces lifetime revision burden. **Patient Communication:** Address devastation about 'failed' surgery with empathy: (1) Explain this likely **patient selection issue** not technical failure - advanced cartilage damage pre-existed surgery, (2) Hip arthroscopy has **limitations** - does not regenerate cartilage, only addresses mechanical impingement, (3) Rapid progression suggests **aggressive disease biology**, (4) At 28 years, delaying THA critical to reduce lifetime surgeries, (5) Many patients manage symptoms conservatively for years before THA needed. Discuss **realistic timeline**: Try conservative measures 12-24 months, if pain disabling despite maximal conservative treatment, consider THA.
KEY POINTS TO SCORE
Patient selection error - operating on Tonnis grade 1 OA: Tonnis ≥1 relative contraindication to hip arthroscopy (high risk progression to THA), should have prompted detailed pre-op counseling about outcomes, pre-op MRI likely showed Outerbridge III-IV predicting poor prognosis
Tonnis grade 2 OA contraindication to revision arthroscopy: Grade 2 = small cysts + moderate joint space narrowing + sclerosis, combined with Outerbridge IV on MRI = advanced irreversible cartilage loss, revision arthroscopy will fail - only definitive treatment is THA
THA in young patients (age 28) has high revision burden: 15-20% revision rate at 15 years in patients less than 55 (AOANJRR data), if THA at age 30 likely requires 2-3 revisions over lifetime (at 50, 65, 80), each revision more complex with bone loss and worse outcomes
Conservative management goal to delay THA: Activity modification (no running, low-impact exercise), physiotherapy (ROM, core strength), NSAIDs with PPI cover, intra-articular corticosteroid (3-6 months relief, repeat 6-monthly max), walking aids reduce load 20-30%, delaying THA by 5-10 years significantly reduces lifetime revision burden
Poor prognostic factors for hip arthroscopy: Tonnis grade ≥1, Outerbridge grade III-IV, age greater than 40, BMI greater than 30, workers' compensation, unrealistic expectations; Good prognostic factors: Tonnis 0, Outerbridge I-II, age less than 40, isolated labral tear
COMMON TRAPS
✗Offering revision hip arthroscopy for Tonnis grade 2 OA - contraindicated, will fail, causes further cartilage damage and patient disappointment
✗Not recognizing pre-operative patient selection error - Tonnis grade 1 with rapid progression to grade 2 suggests advanced chondral damage was present initially
✗Rushing to THA without trial of conservative management - patient only 28 years old, every year delayed reduces lifetime revision burden, try conservative 12-24 months first
✗Not knowing AOANJRR revision data for young THA patients - examiners expect awareness of Australian registry data for arthroplasty topics (15-20% revision at 15 years if less than 55)
✗Not counseling about lifetime revision burden in young THA - if THA at 30, likely 2-3 revisions needed over lifetime, each revision more complex with worse outcomes and bone loss
LIKELY FOLLOW-UPS
"What are the Tonnis grades of hip osteoarthritis?"
"What is the revision rate for THA in patients under 55 at 15 years (AOANJRR)?"
"What are absolute contraindications to hip arthroscopy for FAI?"

MCQ Practice Points

Alpha Angle

Q: What alpha angle indicates cam morphology? A: Greater than 55°. Measured on lateral view. Angle between neck axis and point where head becomes aspherical.

Cartilage Damage

Q: What is the pattern of cartilage damage in cam FAI? A: Outside-in - the cam bump shears acetabular cartilage from the labrum, starting at the labral chondral junction. This differs from pincer (labral crushing, central acetabular damage).

Demographics

Q: Who typically gets cam FAI? A: Young athletic males. Pincer is more common in middle-aged females. Combined morphology is most common overall (86%).

Australian Context

Clinical Practice: FAI is commonly managed in Australia. Hip arthroscopy is widely available at major centers. The Australian Orthopaedic Association supports appropriate patient selection.

CAM-TYPE FAI

High-Yield Exam Summary

Key Facts

  • •Aspherical femoral head (cam bump)
  • •Alpha angle greater than 55 degrees
  • •Young athletic males
  • •Anterosuperior location

Clinical

  • •Groin pain with hip flexion
  • •FADIR test positive
  • •Reduced internal rotation
  • •Sports with deep flexion provocative

Damage Pattern

  • •Outside-in mechanism
  • •Acetabular cartilage delamination
  • •Labral damage
  • •Leads to OA

Treatment

  • •Conservative first
  • •Arthroscopic osteochondroplasty
  • •Labral repair
  • •Good outcomes with preserved joint
Quick Stats
Reading Time68 min
Related Topics

Hip Labral Tears

Pincer-Type Femoroacetabular Impingement

Trunnionosis and Taper Corrosion

Blood Supply of the Hip