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Not affiliated with the Royal Australasian College of Surgeons.

Adult Acquired Flatfoot Deformity

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Adult Acquired Flatfoot Deformity

Comprehensive guide to adult acquired flatfoot deformity (AAFD) with focus on posterior tibial tendon dysfunction (PTTD), Johnson and Strom classification, surgical reconstruction algorithms, and evidence-based management strategies for Orthopaedic examination preparation.

complete
Updated: 2025-12-17
High Yield Overview

ADULT ACQUIRED FLATFOOT DEFORMITY

Progressive Collapse | PTT Dysfunction | Staged Reconstruction

Stage IImost common presentation
3:1female to male ratio
40-60peak age range (years)
20%bilateral involvement

Johnson and Strom Classification

Stage I
PatternPTT tenosynovitis, no deformity
TreatmentConservative, immobilization
Stage II
PatternFlexible deformity, hindfoot valgus
TreatmentCalcaneal osteotomy + FDL transfer
Stage III
PatternFixed/rigid deformity
TreatmentHindfoot arthrodesis (triple/subtalar)
Stage IV
PatternAnkle valgus tilt
TreatmentTibiotalocalcaneal arthrodesis

Critical Must-Knows

  • Johnson and Strom classification - guides surgical reconstruction strategy
  • Rigid vs flexible - determines need for osteotomy vs arthrodesis
  • Too many toes sign - pathognomonic for hindfoot valgus on clinical exam
  • Single heel rise test - inability indicates PTT incompetence
  • Stage II divide - IIA (flexible hindfoot) vs IIB (forefoot driven, fixed forefoot varus)

Examiner's Pearls

  • "
    Stage IIB requires cotton test and first ray plantar flexion osteotomy
  • "
    FDL is preferred tendon transfer over FHL (better excursion, less donor morbidity)
  • "
    Lateral column lengthening (Evans) corrects forefoot abduction
  • "
    Stage III requires subtalar or triple arthrodesis for rigid deformity

Clinical Imaging

Imaging Gallery

Posterior view of adult flatfoot showing hindfoot valgus
Click to expand
Clinical photograph demonstrating the classic posterior view of adult acquired flatfoot deformity with marked hindfoot valgus. The heel is visibly everted (turned outward) and the medial longitudinal arch is collapsed. This angle is the standard view for assessing the pathognomonic **'too many toes sign'** - when viewing the foot from directly behind, multiple lateral toes become visible due to the combination of hindfoot valgus and forefoot abduction. In a normal foot, typically only the fifth toe (and possibly part of the fourth toe) is visible from this posterior perspective. The ability to see three, four, or even all five toes indicates significant deformity and is highly specific for AAFD. This clinical sign correlates with Johnson and Strom Stage II or beyond and helps distinguish flexible from rigid deformity when combined with dynamic testing.Credit: Source article via PMC via Open-i (NIH) (Open Access (CC BY))
Ultrasound imaging of posterior tibial tendon pathology
Click to expand
Longitudinal ultrasound image of the posterior tibial tendon region showing pathological changes. The ultrasound demonstrates altered echogenicity with areas of hypoechoic (dark) signal suggesting tendinosis or tenosynovitis of the PTT. Normal tendon appears as hyperechoic (bright) fibrillar tissue with a characteristic linear striated pattern; the disruption of this pattern indicates structural damage. Ultrasound has emerged as a valuable point-of-care imaging modality for PTTD, particularly in Stage I disease where clinical examination shows medial ankle pain and tenderness but deformity may not yet be apparent. Advantages include dynamic assessment (ability to visualize tendon movement during inversion), real-time comparison with the contralateral side, and cost-effectiveness compared to MRI. Ultrasound can detect tenosynovitis, longitudinal splits, and partial tears, guiding early conservative management and predicting progression to deformity.Credit: Source article via PMC via Open-i (NIH) (Open Access (CC BY))
Bilateral ankle MRI showing axial views for PTTD comparison
Click to expand
Two-panel MRI showing bilateral axial (cross-sectional) ankle views at two different levels (panels A and B) with right (R) and left (L) ankles labeled for direct comparison. Axial MRI sequences are the workhorse for evaluating the posterior tibial tendon, allowing assessment of tendon cross-sectional area, signal intensity, and relationship to surrounding structures including the medial malleolus, flexor digitorum longus, and spring ligament complex. This bilateral comparison approach is clinically valuable because 20% of AAFD cases involve both feet, and comparing the symptomatic side with the contralateral 'normal' ankle enhances detection of subtle tendon thickening, signal changes, or peritendinous fluid. MRI is the gold standard for surgical planning, enabling grading of PTT degeneration (tendinosis vs partial tear vs complete rupture), assessment of spring ligament integrity (critical for Stage IIB), evaluation of sinus tarsi and subtalar joint (determines Stage III arthrodesis requirements), and detection of ankle valgus tilt (defines Stage IV requiring tibiotalocalcaneal fusion).Credit: Source article via PMC via Open-i (NIH) (Open Access (CC BY))

Critical Adult Flatfoot Exam Points

Classification Is Key

Johnson and Strom staging drives all treatment decisions. Stage I = conservative. Stage II = reconstruction (osteotomy + transfer). Stage III = arthrodesis. Stage IV = ankle fusion. Know the flexibility test!

Deformity Components

Three-plane deformity: Hindfoot valgus (coronal), arch collapse (sagittal), forefoot abduction (axial). Examiners ask: "Describe the deformity." Answer systematically in all three planes.

Clinical Tests

Too many toes sign (view from behind) and single heel rise test (PTT function). Examiners will show photos. Also test: flexibility with hindfoot valgus stress and Silfverskiöld test for gastrocnemius contracture.

Surgical Decision Tree

Flexible Stage II = calcaneal osteotomy (medializing or Koutsogiannis) plus FDL transfer plus possible lateral column lengthening. Stage IIB adds cotton test and medial cuneiform osteotomy. Rigid Stage III = triple arthrodesis. Know when to do what!

Quick Decision Guide: AAFD Management

StageDeformityTreatmentKey Pearl
Stage IPTT tenosynovitis, no deformityImmobilization (CAM boot 6-12 weeks), NSAIDs, orthoticsExam: medial ankle pain, no too many toes sign
Stage IIAFlexible hindfoot valgus, passively correctableMedializing calcaneal osteotomy + FDL transfer ± LCLCotton test negative, hindfoot corrects with stress
Stage IIBFlexible hindfoot + fixed forefoot varusAs IIA PLUS medial cuneiform plantar flexion osteotomyCotton test positive, forefoot drives hindfoot valgus
Stage IIIRigid/fixed deformity, subtalar arthritisSubtalar arthrodesis or triple arthrodesisHindfoot does NOT correct passively, talonavicular arthritis
Stage IVAnkle valgus tilt (deltoid insufficiency)Tibiotalocalcaneal arthrodesis (TTC fusion)Lateral ankle mortise widening on AP radiograph
Mnemonic

MAINPosterior Tibial Tendon Anatomy

M
Medial malleolus posterior
Courses posterior and inferior to medial malleolus in fibro-osseous tunnel
A
Arch primary dynamic support
Main dynamic stabilizer of medial longitudinal arch against body weight
I
Insertions navicular + cuneiforms + MT bases
Multiple insertions: navicular tuberosity (primary), all 3 cuneiforms, MT 2-4 bases
N
Navicular main insertion
Navicular tuberosity is primary insertion and critical to arch support

Memory Hook:PTT is the MAIN support of the medial arch - lose it and the arch collapses!

Mnemonic

TFRFJohnson and Strom Classification

T
Tenosynovitis only
Stage I: PTT inflammation without deformity, pain medial ankle, normal alignment
F
Flexible deformity
Stage II: Hindfoot valgus flexible (passively correctable), arch collapses with weight-bearing
R
Rigid deformity
Stage III: Fixed hindfoot valgus, subtalar stiffness/arthritis, cannot correct passively
F
Further to ankle
Stage IV: Ankle involvement with valgus tilt, deltoid insufficiency, tibiotalar arthritis

Memory Hook:AAFD progression: Tendon inflames → Flexibility lost in hindfoot → Rigidity sets in → Further spread to ankle!

Mnemonic

COLTSurgical Reconstruction Components (Stage II)

C
Calcaneal osteotomy
Medializing (translation) or Koutsogiannis (medial slide with lateral closing wedge)
O
Orthotic (Cotton test)
Cotton test for forefoot varus - if positive, need medial cuneiform plantar flexion osteotomy
L
Lateral column lengthening
Evans osteotomy (anterior calcaneus) corrects forefoot abduction, increases arch height
T
Tendon transfer FDL
Flexor digitorum longus transfer to navicular restores dynamic arch support

Memory Hook:Fixing Stage II AAFD is like training a young COLT - need all components working together for stable gait!

Mnemonic

UNIONComplications to Discuss in Viva

U
Undercorrection
Most common - persistent symptoms, recurrent deformity (up to 30%)
N
Nonunion
After arthrodesis procedures (5-15%), especially in smokers, diabetics
I
Infection
Superficial 5-10%, deep 2-5%, higher with diabetes and smoking
O
Overcorrection
Hindfoot varus (rigid painful foot), lateral column pain from over-lengthening
N
Nerve injury
Sural nerve (lateral approaches), tibial nerve branches (medial approaches)

Memory Hook:Aim for UNION of all corrected joints - but watch for complications that prevent perfect healing!

Overview and Epidemiology

Clinical Significance

Adult acquired flatfoot deformity (AAFD) represents a progressive collapse of the medial longitudinal arch due to posterior tibial tendon (PTT) dysfunction. It is the most common cause of acquired flatfoot in adults and represents a spectrum from isolated tenosynovitis to severe fixed deformity with ankle involvement. Understanding the staged progression is critical for treatment selection.

Demographics and Risk Factors

  • Age: Peak 40-60 years
  • Gender: Female predominance (3:1 ratio)
  • Obesity: Major risk factor (increased mechanical load)
  • Diabetes: Associated with tendon degeneration
  • Hypertension: Vascular compromise to tendon
  • Previous trauma: Medial ankle injuries
  • Inflammatory arthropathy: RA, seronegative arthritis
  • Steroid use: Tendon weakening

Natural History and Impact

  • Progressive deformity: Without treatment, stage I → stage IV over years
  • Functional limitation: Pain with prolonged walking/standing
  • Gait alteration: Compensatory external rotation, antalgic gait
  • Quality of life: Significant impact on mobility and footwear
  • Adjacent joint arthritis: Talonavicular, subtalar, ankle involvement
  • Bilateral progression: 20% develop contralateral AAFD

Pathophysiology and Mechanisms

PTT Is the PRIMARY Dynamic Arch Stabilizer

The posterior tibial tendon provides over 50% of the dynamic support to the medial longitudinal arch during stance phase. Loss of PTT function results in progressive arch collapse, hindfoot valgus, and forefoot abduction - the classic flatfoot deformity. Unlike static ligamentous stabilizers (spring ligament, plantar fascia), the PTT actively resists arch collapse during gait, making it irreplaceable.

Posterior Tibial Tendon Course and Function

Anatomical ComponentClinical SignificanceExam Relevance
Origin: Posterior tibia, fibula, interosseous membraneStrongest invertor of foot (tibialis posterior muscle)Loss causes unopposed eversion (peroneus brevis dominates)
Course: Posterior to medial malleolus in fibro-osseous tunnelHypovasular zone 2-4cm proximal to navicular insertionSite of degenerative tendinopathy and eventual rupture
Primary insertion: Navicular tuberosityMain arch keystone stabilizerDetachment here causes immediate arch collapse
Secondary insertions: All 3 cuneiforms, MT 2-4 basesProvides midfoot stability and transverse arch supportMultiple slips allow distributed force transfer

Static Stabilizers (Spring Ligament Complex)

The spring ligament (plantar calcaneonavicular ligament) is the primary static stabilizer of the medial arch. It consists of two components:

  • Superomedial calcaneonavicular ligament: Main load-bearing structure, supports talar head
  • Inferior calcaneonavicular ligament: Reinforces plantar aspect

In AAFD, the spring ligament undergoes attritional failure as the PTT loses function, leading to progressive talar head uncovering and arch collapse.

Three-Plane Deformity Components

Coronal Plane

Hindfoot valgus

  • Subtalar joint eversion
  • Lateral talar shift
  • Posterior facet uncovering
  • "Too many toes" sign

Sagittal Plane

Arch collapse (planus)

  • Talus plantar flexion
  • Navicular sag
  • Increased talo-first metatarsal angle
  • Spring ligament attenuation

Axial Plane

Forefoot abduction

  • Talonavicular uncovering
  • Lateral border of forefoot
  • "Too many toes" sign
  • Compensatory tibial external rotation

Classification Systems

Johnson and Strom Classification (Most Widely Used)

StageClinical FindingsRadiographic FeaturesTreatment
Stage IPTT pain, swelling medial ankle. Normal alignment. Single heel rise positive. No too many toes sign.Normal alignment. No arch collapse. No hindfoot valgus.Conservative: CAM boot 6-12 weeks, UCBL orthotic, NSAIDs, PT
Stage IIAHindfoot valgus flexible. Arch collapses weight-bearing. Single heel rise negative. Too many toes positive. Cotton test negative.Hindfoot valgus (AP: talar head uncovering). Lateral: sag at talonavicular, increased talo-1st MT angle.Medializing calcaneal osteotomy + FDL transfer ± lateral column lengthening
Stage IIBAs IIA PLUS fixed forefoot varus (does not correct). Cotton test positive (forefoot drives hindfoot valgus).As IIA PLUS forefoot varus visible on lateral radiograph with hindfoot corrected.As IIA PLUS medial cuneiform plantar flexion osteotomy (Cotton osteotomy)
Stage IIIRigid hindfoot valgus (does NOT correct passively). Subtalar stiffness. Talonavicular pain/arthritis.Subtalar arthritis. Talonavicular arthritis. Fixed hindfoot valgus. No passive correction on stress views.Subtalar arthrodesis or triple arthrodesis (± tendon transfer if Stage IIIA)
Stage IVAs Stage III PLUS lateral ankle pain. Deltoid insufficiency. Ankle valgus tilt.Ankle valgus tilt on AP ankle (tilted talus). Tibiotalar arthritis. Lateral ankle mortise widening.Tibiotalocalcaneal (TTC) arthrodesis. Consider total ankle replacement in select cases.

Stage II Subdivisions (Myerson Modification)

Stage IIA: Flexible hindfoot valgus, hindfoot corrects when forefoot is unloaded. Cotton test negative. Treat with hindfoot procedures only.

Stage IIB: Flexible hindfoot valgus BUT fixed forefoot varus (first ray dorsiflexed). Cotton test positive (elevating heel under first ray does NOT correct arch). The forefoot varus drives the hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy PLUS hindfoot procedures. Missing this = failure.

Bluman Classification (MRI-Based)

Based on MRI findings of PTT integrity:

GradeMRI FindingsClinical Correlation
Grade 1Tendon intact, increased signal on T2 (peritendinitis/tenosynovitis)Corresponds to Johnson Stage I: pain without deformity
Grade 2Partial tear with elongation. Tendon less than 50% thickness lost.May correspond to early Stage II with flexible deformity
Grade 3Severe tear, greater than 50% thickness lost or complete ruptureAdvanced Stage II or Stage III with significant deformity

Clinical Relevance: MRI grading helps predict which Stage I patients will progress and guides early surgical intervention (e.g., Grade 3 tear in Stage I may benefit from early FDL transfer rather than prolonged conservative treatment).

Clinical Assessment

History

  • Pain location: Medial ankle (PTT), lateral (peroneal overload, sinus tarsi), plantar (arch strain)
  • Onset: Acute trauma vs insidious (degenerative)
  • Progression: Worsening deformity over months to years
  • Function: Walking distance, stairs, uneven ground tolerance
  • Footwear: Difficulty fitting shoes, wearing out medial heel
  • Red flags: Inflammatory symptoms (RA), neuropathy (diabetes), acute swelling (DVT)

Examination Sequence

Standing (weight-bearing):

  • Too many toes sign (view from behind)
  • Arch height vs normal side
  • Hindfoot valgus alignment
  • Forefoot abduction
  • Compensatory external tibial rotation

Seated (non-weight-bearing):

  • PTT palpation: swelling, tenderness, gap
  • Single heel rise test (each foot separately)
  • Hindfoot flexibility: valgus stress test
  • Cotton test (Stage IIB screening)
  • Silfverskiöld test (gastrocnemius contracture)
  • Neurovascular examination

Key Clinical Tests

Examination Tests for AAFD

Visual InspectionToo Many Toes Sign

Technique: Examiner stands behind patient who stands with feet shoulder-width apart.

Positive test: More than 2 toes visible lateral to heel on affected side (normally only 5th and part of 4th visible). Indicates forefoot abduction and hindfoot valgus.

Sensitivity: 90% for Stage II or greater AAFD.

Functional TestSingle Heel Rise Test

Technique: Patient stands on one leg, rises up on toes (heel off ground). Observe hindfoot from behind.

Normal: Hindfoot inverts (heel shifts to varus) as PTT contracts and arch rises.

Abnormal (PTT dysfunction): Hindfoot remains in valgus or everts further. Patient may be unable to perform heel rise (complete PTT rupture/dysfunction).

Gold standard for PTT function. Inability to perform = Stage II or greater.

Seated ExaminationHindfoot Flexibility Test

Technique: With patient seated (unloaded), examiner grasps heel and attempts to correct hindfoot valgus into neutral or varus.

Flexible (Stage II): Hindfoot corrects passively to neutral. Indicates soft tissue deformity (PTT, spring ligament) without fixed bony/arthritic changes.

Rigid (Stage III): Hindfoot does NOT correct. Indicates subtalar or talonavicular arthritis, fixed bony deformity. Changes treatment to arthrodesis.

Stage IIB ScreeningCotton Test (Forefoot Varus Test)

Technique: With patient seated, elevate first ray (plantarflex first metatarsal-cuneiform joint) and observe hindfoot alignment.

Negative (Stage IIA): Hindfoot valgus CORRECTS when first ray is elevated. Forefoot is flexible.

Positive (Stage IIB): Hindfoot valgus PERSISTS despite first ray elevation. Indicates fixed forefoot varus driving hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy.

Gastrocnemius ContractureSilfverskiöld Test

Technique: With knee extended, dorsiflex ankle. Then flex knee to 90 degrees and dorsiflex ankle.

Positive: Dorsiflexion improves with knee flexed (isolated gastrocnemius contracture). May require gastrocnemius recession in addition to hindfoot reconstruction.

Significance: Gastrocnemius contracture exacerbates hindfoot valgus (equinus drives valgus). Affects surgical planning.

Do Not Miss Bilateral Disease

Approximately 20% of AAFD patients have bilateral involvement (often asymmetric). Always examine BOTH feet and compare. The "normal" side may be early-stage asymptomatic AAFD. Failure to identify bilateral disease leads to:

  • Incorrect comparison (thinking abnormal is "normal for patient")
  • Missed opportunity for early intervention on asymptomatic side
  • Patient dissatisfaction when contralateral side progresses post-operatively

Investigations

Imaging Protocol for AAFD

First LineWeight-Bearing Radiographs (Mandatory)

AP foot: Talonavicular uncovering (lateral subluxation of navicular on talus), forefoot abduction, talo-first metatarsal angle.

Lateral foot: Calcaneal pitch angle (normal 20-30 degrees, decreased in AAFD), talo-first metatarsal angle (normal 0-5 degrees, increased Meary angle in AAFD indicates arch collapse), talus plantar flexion/sagging.

AP ankle (if Stage III/IV suspected): Assess for ankle valgus tilt, tibiotalar arthritis, lateral mortise widening (deltoid insufficiency).

Hindfoot alignment view (Saltzman view): Quantifies hindfoot valgus. Taken with patient standing, X-ray beam angled 20 degrees cephalad from behind ankle. Measures weight-bearing axis of hindfoot.

Critical: Must be weight-bearing to assess deformity magnitude. Non-weight-bearing films underestimate deformity.

Second LineMRI

Indications: Uncertain diagnosis, assessing PTT integrity (surgical planning), evaluating spring ligament status, ruling out occult fractures or other pathology.

Findings:

  • PTT: Increased T2 signal (tenosynovitis), thinning, discontinuity (tear), fluid in sheath
  • Spring ligament: Attenuation, tear, increased signal
  • Bone marrow edema: Talar head (overload), navicular stress
  • Subtalar/talonavicular arthritis: Cartilage loss, subchondral changes

Bluman MRI Classification: Grade 1 (intact tendon, peritendinitis), Grade 2 (partial tear less than 50%), Grade 3 (greater than 50% or complete rupture).

AdvancedCT Scan

Indications: Assessing subtalar or talonavicular arthritis (planning arthrodesis), evaluating tarsal coalition (rigid flatfoot differential), planning complex osteotomies.

Advantages: Better bony detail than MRI. Can perform 3D reconstructions for preoperative planning (especially useful for lateral column lengthening or complex deformity correction).

AdjunctUltrasound

Limited role: Dynamic assessment of PTT (real-time movement), identifying complete rupture vs partial tear. Operator-dependent. Not routinely used in Australia.

Key Radiographic Measurements

MeasurementNormal ValueAAFD FindingClinical Significance
Calcaneal pitch angle (lateral)20-30 degreesLess than 18 degrees (arch collapse)Decreased angle indicates loss of arch height
Talo-first metatarsal angle (lateral, Meary angle)0-5 degreesGreater than 10 degrees (arch sag)Measures alignment of talus and first metatarsal - should be collinear
AP talonavicular coverage angleLess than 7 degreesGreater than 20 degrees (uncovering)Lateral subluxation of navicular off talar head - forefoot abduction
Hindfoot alignment (Saltzman view)0 ± 3mm from midlineGreater than 5mm lateral deviation (valgus)Quantifies hindfoot valgus deformity for surgical planning

Management Algorithm

📊 Management Algorithm
flatfoot deformity adult management algorithm
Click to expand
Management algorithm for flatfoot deformity adultCredit: OrthoVellum

Conservative Management Indications

Appropriate for:

  • Stage I (tenosynovitis without deformity)
  • Mild Stage IIA with minimal symptoms
  • Patients unfit for surgery (medical comorbidities)
  • Patient preference (informed consent)

Conservative Treatment Protocol

InitialAcute Phase (0-6 weeks)

Goals: Reduce inflammation, offload PTT, control pain

  • Immobilization: CAM walker boot or short leg cast, non-weight-bearing or protected weight-bearing
  • NSAIDs: Oral anti-inflammatories (if no contraindications)
  • Ice: 15-20 minutes TDS over medial ankle
  • Activity modification: Avoid prolonged standing, stairs, uneven ground
  • Rest: PTT needs unloading to allow healing/reduction of inflammation
ProgressiveRehabilitation (6-12 weeks)

Goals: Restore function, prevent recurrence, transition to orthotic

  • Progressive weight-bearing: Gradual transition from boot to supportive footwear
  • Physiotherapy: PTT strengthening (resisted inversion), intrinsic muscle strengthening, proprioception
  • Orthotic prescription: UCBL (University of California Berkeley Laboratory) insert or AFO (ankle-foot orthosis)
  • Footwear advice: Firm heel counter, medial arch support, avoid flat flexible shoes
MaintenanceLong-term (Greater than 3 months)

Goals: Prevent progression, maintain function, surveillance

  • Orthotic compliance: Daily use in all footwear
  • Weight management: Reduce mechanical load on PTT
  • Activity modification: Avoid high-impact activities, use walking aids if needed
  • Surveillance: 6-12 monthly review to assess for progression to Stage II
  • Progression criteria: Increasing deformity, too many toes sign, failed single heel rise = surgical referral

When Conservative Fails

Indications for surgery in Stage I patients:

  • Persistent pain despite 6 months of appropriate conservative treatment
  • Progressive deformity (transition to Stage II)
  • Failed single heel rise test (PTT incompetence)
  • MRI showing Grade 3 PTT tear (complete rupture) - these will not heal

Important: Not all Stage I patients progress. Approximately 50% respond to conservative treatment with orthotics. However, those with Grade 3 MRI tears should be offered early surgical intervention (FDL transfer before deformity develops).

Stage II Surgical Reconstruction

Goals: Correct flexible deformity, restore PTT function, prevent progression to rigid Stage III.

Stage IIA vs IIB Distinction Is Mandatory

Performing Stage IIA reconstruction (hindfoot only) on a Stage IIB patient (forefoot-driven) leads to FAILURE. The uncorrected fixed forefoot varus drives recurrent hindfoot valgus post-operatively. ALWAYS perform Cotton test. If positive, MUST add medial cuneiform plantar flexion osteotomy to the reconstruction. Missing this distinction is a common viva trap.

Stage IIA Reconstruction Components

Component 1Calcaneal Osteotomy

Purpose: Correct hindfoot valgus by medializing calcaneus under tibia.

Options:

  • Medializing calcaneal osteotomy (MCO): Simple medial translation of posterior fragment. Easy, reproducible. Most common choice.
  • Koutsogiannis osteotomy: Medial slide PLUS lateral closing wedge. Greater correction power, useful for severe valgus.

Technique: Oblique osteotomy 1-1.5cm distal to posterior facet. Shift 8-12mm medially. Fix with 2 screws (lag technique or position screws).

Rationale: Shifts ground reaction force medial to subtalar axis, creating inversion moment.

Component 2FDL Transfer to Navicular

Purpose: Restore dynamic arch support (replace failed PTT).

Why FDL over FHL?

  • Better excursion (longer muscle belly, better arc of contraction)
  • Less donor morbidity (FHL is stronger toe flexor, more missed if transferred)
  • Easier harvest (medial approach)
  • Sufficient strength (clinical outcomes equivalent or better than FHL)

Technique: Harvest FDL through plantar foot incision (master knot of Henry). Transfer through drill hole in navicular (anterior to posterior, exit dorsal cortex). Tension with foot in neutral hindfoot alignment and slight inversion. Fix with interference screw or suture anchor.

Rationale: Provides active inversion force and dynamic arch support during gait.

Component 3 (Optional)Lateral Column Lengthening (Evans)

Indications: Significant forefoot abduction (talonavicular uncovering greater than 30%, AP talo-first metatarsal angle greater than 20 degrees).

Technique: Osteotomy anterior process of calcaneus, 1-1.5cm posterior to calcaneocuboid joint. Distract 8-12mm. Insert structural bone graft (allograft or autograft from iliac crest). Fix with plate or graft shape alone.

Effect: Corrects forefoot abduction, increases arch height (windlass effect), reduces talonavicular uncovering.

Caution: Overcorrection leads to rigid lateral column overload pain, calcaneocuboid arthritis. Limit lengthening to 10-12mm maximum.

Adjunct (If Positive Silfverskiöld)Gastrocnemius Recession

Indication: Isolated gastrocnemius contracture (ankle dorsiflexion less than neutral with knee extended, but improves with knee flexed).

Technique: Strayer procedure (proximal gastrocnemius recession) or Baumann procedure (distal gastrocnemius recession at musculotendinous junction).

Rationale: Equinus forces hindfoot into valgus (compensatory). Correcting gastrocnemius contracture prevents recurrence.

Stage IIB Reconstruction (Add to IIA)

Pre-opCotton Test Confirmation

Mandatory: Perform Cotton test in clinic AND confirm under anaesthesia.

Positive test: Hindfoot valgus persists when first ray is elevated (plantarflexed). Indicates forefoot varus is DRIVING hindfoot valgus.

Additional ProcedureMedial Cuneiform Plantar Flexion Osteotomy

Purpose: Correct fixed forefoot varus by plantarflexing first ray, removing the driver of compensatory hindfoot valgus.

Technique (Cotton osteotomy):

  • Dorsal opening wedge osteotomy of medial cuneiform
  • Insert structural bone graft (typically 5-8mm height)
  • Fix with plate or screws
  • Plantarflexes first ray, corrects forefoot varus

Alternative (Lapidus fusion): Medial cuneiform-first metatarsal fusion in plantarflexed position. More rigid correction, useful if TMT arthritis present.

Critical: This addresses the CAUSE of the deformity in IIB. Without it, the hindfoot correction will fail.

Surgical Decision Algorithm Summary

Stage IIA (flexible hindfoot, forefoot flexible): MCO + FDL transfer ± Evans (if significant abduction) ± gastrocnemius recession (if contracture).

Stage IIB (flexible hindfoot, forefoot FIXED varus): All of IIA PLUS medial cuneiform plantar flexion osteotomy.

Examiners WILL test if you know to perform Cotton test and recognize IIB. This is high-yield.

Stage III: Rigid Deformity

Key concept: Hindfoot does NOT correct passively. Subtalar and/or talonavicular joints are stiff (arthritis or longstanding deformity with capsular contracture). Osteotomies alone will NOT work - need arthrodesis to correct rigid deformity.

Stage III Surgical Options

Option 1Subtalar Arthrodesis

Indications: Isolated subtalar arthritis, rigid hindfoot valgus, talonavicular joint MOBILE and NON-arthritic.

Technique: Medial or lateral approach. Resect articular cartilage from posterior facet. Correct hindfoot valgus. Fix with screws (typically 2-3 large lag screws, cannulated or solid).

Advantages: Preserves talonavicular and calcaneocuboid joints (some midfoot motion retained). Faster rehabilitation than triple fusion.

Disadvantages: May not fully correct if talonavicular instability present. Higher risk of adjacent joint arthritis (talonavicular, ankle) long-term.

Option 2 (Gold Standard)Triple Arthrodesis

Indications: Rigid hindfoot valgus with talonavicular arthritis or instability. Need for greater correction power. Stage III with severe deformity.

Technique: Fuse subtalar, talonavicular, and calcaneocuboid joints. Correct hindfoot valgus, restore arch, address forefoot abduction. Fix each joint (screws or plates).

Advantages: Most powerful correction. Addresses all components of deformity. Lower recurrence rate than isolated subtalar fusion.

Disadvantages: Eliminates all hindfoot motion (significant functional loss). Accelerates ankle arthritis (increased loads). Longer rehabilitation, higher nonunion risk (3 joints to heal).

Pearl: Modern trend is towards "modified" triple (preserve calcaneocuboid if non-arthritic) to retain some hindfoot motion.

Additional ConsiderationsAdjunctive Procedures

FDL transfer: Consider in Stage IIIA (flexible forefoot, minimal talonavicular arthritis). Provides some dynamic support even after fusion.

Lateral column lengthening: May still be needed if severe forefoot abduction and rigid calcaneocuboid prevents fusion.

Ankle monitoring: Stage III patients are at risk for progression to Stage IV (deltoid attenuation). Counsel about future ankle issues.

Stage IV: Ankle Valgus

Pathophysiology: Chronic hindfoot valgus leads to deltoid ligament attenuation and eventual failure. Talus tilts into valgus within ankle mortise. Deltoid insufficiency → tibiotalar incongruency → ankle arthritis.

ProcedureIndicationOutcomeConsiderations
Tibiotalocalcaneal (TTC) arthrodesisStage IV with ankle arthritis, deltoid insufficiency, valgus tiltPain relief, stable plantigrade foot. Loss of ALL ankle and hindfoot motion.Gold standard. Use locked intramedullary nail (retrograde femoral nail) or plate fixation. Nonunion risk 10-15%. Significant functional limitation - need rocker-bottom shoes.
Total ankle replacement + hindfoot fusionStage IV, younger patient, low arthritis burden, high functional demandPreserves some ankle motion, better function than TTC fusionTechnically demanding. Requires correction of hindfoot FIRST (via fusion), then ankle replacement. Higher revision rate. Limited long-term data in AAFD population.
Deltoid reconstruction + hindfoot fusionStage IV WITHOUT ankle arthritis (valgus tilt only, preserved cartilage)Avoid ankle fusion in select cases, restore stabilityRare indication. Requires pristine ankle cartilage (uncommon in chronic Stage IV). Deltoid reconstruction (allograft or synthetic) combined with subtalar/triple fusion to correct hindfoot.

TTC Fusion Is a Salvage Procedure

Tibiotalocalcaneal arthrodesis results in complete loss of ankle and hindfoot motion. Patients will have a stiff, plantigrade foot useful for standing and walking with adaptive shoes, but significant functional limitations. Counsel extensively pre-operatively:

  • Cannot walk on uneven ground
  • Difficulty with stairs, ramps, curbs
  • Need rocker-bottom shoes
  • Compensatory hip/knee/back pain
  • Driving difficulties (especially right foot)

This is a quality of life decision - pain-free stability vs functional motion. Some patients prefer amputation (controversial but documented).

Surgical Technique: Stage II Reconstruction

Pre-operative Planning

Consent Points

  • Undercorrection: 20-30% recurrence risk, may need revision
  • Overcorrection: Hindfoot varus (painful rigid foot), lateral column overload
  • Nerve injury: Sural nerve (lateral approaches), saphenous nerve, tibial nerve branches
  • Infection: Superficial 5-10%, deep 2-5% (higher if diabetic)
  • Nonunion: Calcaneal osteotomy (5%), Evans osteotomy if bone graft used (10%)
  • Donor site morbidity: FDL harvest (toe clawing rare, flexion weakness), iliac crest if used (pain)
  • Prolonged recovery: 3 months non-weight-bearing, 6-12 months full recovery
  • Adjacent joint arthritis: Ankle, subtalar, talonavicular (long-term risk)

Equipment Checklist

  • Implants: Cannulated screws (6.5mm or 7.0mm for calcaneus), interference screw or suture anchor (FDL), Evans plate if LCL planned
  • Power tools: Oscillating saw (osteotomies), drill, reamer
  • Imaging: C-arm with ankle/foot capabilities, ensure can obtain hindfoot alignment view
  • Bone graft: Allograft tricortical iliac crest or femoral head (Evans), consider autograft harvest set if preferred
  • Tendon instruments: Nerve hooks, right-angle clamps, drill for navicular tunnel
  • Retractors: Self-retaining (Weitlaner), Army-Navy, Hohmann

Patient Positioning and Setup

Setup Checklist

Step 1Position

Supine on radiolucent table (Jackson table or standard with radiolucent foot extension).

  • Head: Neutral, adequate airway access
  • Upper body: Arms on arm boards or across chest (depends on anaesthetic preference)
  • Pelvis: Centered on table, neutral alignment
  • Operative leg: Hip neutral rotation (or slight external rotation), knee extended or slight flexion (bump under knee for relaxation)
  • Non-operative leg: Abducted and supported on leg holder or pillow (out of C-arm path)
Step 2Padding and Safety
  • Bony prominences: Sacrum, heels (non-operative), elbows
  • Nerves at risk: Ulnar nerve (elbow padding), peroneal nerve at fibular head (if leg externally rotated or in leg holder)
  • Pressure areas: Ensure heel of operative foot OFF table (ankle hanging free or on bolster)
Step 3Tourniquet

Thigh tourniquet (standard):

  • Apply over cast padding to proximal thigh
  • Exsanguinate with Esmarch or elevation
  • Pressure: 250-300 mmHg (adjust for patient size/BP)
  • Expected time: 90-120 minutes (multiple osteotomies + transfer)

Advantages: Bloodless field aids dissection, tendon identification, osteotomy precision Disadvantages: Tourniquet pain (limited by anaesthetic), time limit (deflate and re-exsanguinate if exceeds 2 hours)

Step 4Draping and C-arm Access
  • Prep: Circumferential ankle and foot to mid-calf. Include proximal calf if anticipating gastrocnemius recession.
  • Draping: Stockinette over foot, U-drape or split sheet isolating foot and ankle
  • Landmarks exposed: Medial malleolus to toes (medial approach for FDL, medial cuneiform), lateral calcaneus (for MCO through lateral or oblique approach, Evans if needed)
  • C-arm positioning: Ensure adequate AP, lateral, oblique views of foot AND hindfoot alignment view possible without repositioning patient. Test BEFORE prep/drape.

Positioning Pearl for Hindfoot Alignment View

The Saltzman hindfoot alignment view (20-degree cephalad beam from posterior) is critical for assessing intraoperative correction. Position the C-arm BEFORE starting - you need to confirm adequate valgus correction post-osteotomy. If you cannot obtain this view intraoperatively, you are operating "blind" on alignment.

Medializing Calcaneal Osteotomy (MCO)

MCO Surgical Steps

Step 1Incision and Approach

Lateral oblique approach:

  • Incision: 4-5cm longitudinal, centered over lateral wall of calcaneus, 1cm inferior to peroneal tendons
  • Start at posterior tuberosity, extend anteriorly parallel to plantar surface
  • Landmarks: Lateral wall of calcaneus palpable subcutaneously, stay INFERIOR to peroneal tendons

Dissection:

  • Incise skin and subcutaneous tissue
  • Identify sural nerve (runs posterior to lateral malleolus, may have branches crossing field) - retract or protect
  • Incise periosteum longitudinally over lateral calcaneal wall
  • Elevate periosteum superiorly and inferiorly with periosteal elevator - STAY subperiosteal to protect soft tissues
Step 2Osteotomy Planning

Level: 1-1.5cm distal to posterior facet of subtalar joint (to avoid intra-articular cut).

Orientation: Oblique from posterosuperior to anteroinferior (approximately 45 degrees to plantar surface).

Fluoroscopy: Obtain lateral view to confirm level. Mark osteotomy line with electrocautery or marking pen on bone.

Rationale: Oblique orientation increases surface area for healing and allows translation without shortening.

Step 3Perform Osteotomy

Technique:

  • Use oscillating saw with wide blade (for thick calcaneal cortex)
  • Start posteriorly, cut anteroinferiorly maintaining oblique angle
  • Irrigate continuously (prevent thermal necrosis)
  • Complete osteotomy - confirm with gentle manipulation (posterior fragment should be mobile)
  • Protect soft tissues (plantar nerves, Achilles medially) with retractors during sawing

Check: Fluoroscopy lateral view - confirm osteotomy complete, no intra-articular extension.

Step 4Translation and Fixation

Translation:

  • Grasp posterior fragment with bone clamp or Schanz pin
  • Translate MEDIALLY (typically 8-12mm depending on preoperative deformity and intraoperative assessment)
  • Assess correction: Hindfoot should align neutral to slight varus on hindfoot alignment view (Saltzman)
  • Avoid overcorrection (greater than 5mm medial = risk of varus)

Temporary fixation: Large K-wire or guidewire across osteotomy to hold reduction

Definitive fixation:

  • Two 6.5mm or 7.0mm cannulated screws (most common): Insert from posterior fragment into anterior fragment, lag technique (glide hole posteriorly, thread hole anteriorly). Aim for bicortical purchase anteriorly.
  • Alternative: Plate fixation if comminution or osteoporotic bone (locking plate provides better fixation)

Fluoroscopy: Confirm screw position (AP, lateral views), adequate translation, no intra-articular screws.

Sural Nerve at Risk

The sural nerve courses posterior to the lateral malleolus and gives off branches that cross the lateral calcaneus. Injury during lateral approach causes lateral foot numbness (variable distribution). ALWAYS identify and protect. If nerve is tethered across osteotomy site, consider gentle mobilization or slight anterior repositioning of incision.

Flexor Digitorum Longus (FDL) Transfer to Navicular

FDL Harvest and Transfer

Step 1Medial Ankle Incision

Approach: Curvilinear incision centered over navicular tuberosity, extending posteriorly along medial ankle (can extend to posterior tibial tendon if debriding/excising diseased PTT).

Length: 6-8cm to allow access to both navicular insertion site and FDL posterior to medial malleolus.

Dissection:

  • Incise skin and subcutaneous tissue
  • Identify and protect saphenous vein and nerve (run anterior to incision)
  • Incise flexor retinaculum (over PTT and FDL tendons)
  • Identify PTT (larger, more anterior), FHL (posterior, deeper), FDL (between PTT and FHL)
Step 2FDL Identification and Harvesting

Technique:

  • Open sheath of FDL (longitudinal incision in sheath)
  • Confirm identity: Pull on tendon and watch lesser toes flex (pathognomonic for FDL)
  • Mobilize FDL proximally and distally with blunt dissection
  • Transect FDL at level of navicular (or distally if harvesting through plantar foot)

Plantar foot harvest (if maximal length needed):

  • Small incision plantar midfoot over master knot of Henry (crossing point of FDL and FHL)
  • Identify FDL (splits to 4 slips for lesser toes)
  • Transect at master knot or distal to it
  • Pass proximal end through medial ankle incision using tendon passer or blunt clamp
Step 3Navicular Tunnel Creation

Technique:

  • Expose navicular tuberosity (remove any diseased PTT remnants if present)
  • Mark tunnel entry site on plantar aspect of navicular tuberosity (slightly inferior and medial to normal PTT insertion)
  • Drill tunnel: 7-8mm diameter, anterior to posterior, exit dorsal cortex of navicular (or drill anterior-posterior and tap out dorsally with small curette)
  • Ensure tunnel is smooth (pass tendon passer through to confirm)

Fluoroscopy: Confirm tunnel does NOT violate talonavicular joint (lateral view).

Step 4FDL Transfer and Fixation

Tensioning:

  • Pass FDL through navicular tunnel (plantar to dorsal) using tendon passer or suture
  • Position foot: Hindfoot neutral to slight varus, ankle neutral dorsiflexion
  • Apply tension to FDL (should feel firm, not slack, but not overtightened - aim for slight inversion force when tensioned)

Fixation options:

  • Interference screw (bioabsorbable or metal, 6-7mm): Insert into tunnel alongside tendon, compresses tendon against bone. Gold standard - strong immediate fixation.
  • Suture anchor (dorsal cortex): Anchor tendon to dorsal exit point with suture anchor. Alternative if tunnel too large or tendon quality poor.
  • Tenodesis screw and washer (dorsal cortex): Pass tendon, fold back, secure with screw and spiked washer over tendon. Traditional technique.

Check:

  • Fluoroscopy: Confirm interference screw or anchor within bone, not intra-articular
  • Clinical: Foot should be in slight inversion with tensioned transfer
  • Range of motion: Move ankle/hindfoot to ensure transfer is not overtightened (prevents equinus or restricted motion)
AdjunctPTT Debridement or Excision

If diseased PTT present:

  • Debride degenerative portions (if partial tear with some intact fibers)
  • OR completely excise (if complete rupture, macerated tissue)
  • Send to pathology if concern for inflammatory etiology (e.g., seronegative arthropathy)

Rationale: Leaving diseased PTT may cause ongoing pain (source removal improves outcomes).

Why FDL Over FHL?

FDL advantages: Better muscle excursion (longer arc of contraction = better active inversion force), less donor site morbidity (FHL is the stronger hallux flexor - patients notice loss more), easier harvest (medial approach vs deep posterior approach for FHL), sufficient strength (biomechanical studies show FDL provides adequate force for arch support).

FHL may be used if: FDL unavailable (previous surgery), simultaneous hallux valgus correction planned (harvest FHL and transfer FDL to hallux for lesser toe balance - "flexor swap"), or surgeon preference. Both are acceptable - know rationale for choice.

Evans Lateral Column Lengthening Osteotomy

Indication: Significant forefoot abduction (talonavicular uncovering greater than 30%, too many toes sign, AP talo-first metatarsal angle greater than 20 degrees).

Evans Osteotomy Steps

Step 1Lateral Incision

Approach: Oblique incision over anterior process of calcaneus, just posterior to calcaneocuboid joint.

Length: 4-5cm to allow osteotomy and graft insertion.

Dissection:

  • Incise skin and subcutaneous tissue (beware sural nerve branches)
  • Identify extensor digitorum brevis muscle (originates from anterior calcaneus) - can split or elevate superiorly
  • Expose lateral wall of anterior calcaneus with subperiosteal dissection
  • Identify calcaneocuboid joint (use fluoroscopy lateral view if uncertain)
Step 2Osteotomy Planning

Level: 1-1.5cm POSTERIOR to calcaneocuboid joint (to avoid intra-articular cut and preserve joint).

Orientation: Oblique from dorsal-lateral to plantar-medial (parallel to CC joint surface).

Fluoroscopy lateral: Mark osteotomy site with K-wire or cautery to confirm position.

Step 3Perform Osteotomy and Distraction

Technique:

  • Use oscillating saw to create osteotomy (through lateral cortex, into medial cortex but do NOT complete medially - maintain medial hinge for stability)
  • Distract osteotomy using lamina spreader or osteotomes (progressive widening)
  • Target distraction: 8-12mm (measured with ruler or calibrated osteotomes)
  • Caution: Do NOT over-distract (greater than 12mm) - risk of calcaneocuboid overload and arthritis

Fluoroscopy: Confirm distraction width, parallel distraction (not hinged open dorsally or plantarly).

Step 4Graft Insertion and Fixation

Graft options:

  • Allograft tricortical iliac crest (most common): Pre-shaped, sterile, avoids donor site morbidity. Size to match distraction (8-10-12mm heights available).
  • Autograft iliac crest: If allograft not available or patient/surgeon preference. Harvest from ipsilateral anterior iliac crest (adds operative time, donor site pain).
  • Femoral head allograft: Can be morselized or shaped into wedge.

Insertion: Impact graft into osteotomy site (use mallet gently), ensure flush with lateral cortex, medial hinge intact.

Fixation: Typically graft shape provides stability (wedge effect). Can add small plate (2-3 holes, dorsal surface) if unstable or concern for graft displacement.

Fluoroscopy: AP and lateral views to confirm graft position, no intra-articular extension, maintained distraction.

Step 5Assess Correction

Clinical: Forefoot abduction should be corrected (visual alignment of forefoot relative to hindfoot).

Fluoroscopy AP foot: Talonavicular coverage improved (navicular more congruent with talar head), reduced uncovering angle.

Intraoperative stress test: Apply varus stress to hindfoot and assess if arch rises (confirms lateral column lengthening effect).

Risk of Overcorrection and Calcaneocuboid Arthritis

Evans osteotomy is powerful but unforgiving. Over-lengthening (greater than 12mm) leads to:

  • Rigid lateral column (calcaneocuboid overload and pain)
  • Calcaneocuboid arthritis (increased joint contact pressure)
  • Overcorrected supinated foot (painful, rigid)

Guideline: Limit distraction to 10-12mm maximum. Accept slight undercorrection rather than overcorrection. If talonavicular uncovering is extreme (greater than 40%), consider medial column procedures (medial cuneiform osteotomy, naviculocuneiform fusion) instead of excessive LCL.

Closure and Postoperative Dressing

Closure Steps

Step 1Wound Irrigation and Hemostasis
  • Deflate tourniquet (if used) and achieve hemostasis (electrocautery, ties)
  • Copious irrigation (3L normal saline minimum across all incisions)
  • Inspect wounds for any bleeding vessels or hematoma
Step 2Drain Decision

Drain: Generally NOT used for routine Stage II reconstruction (small incisions, good hemostasis).

Consider drain if:

  • Large dissection (e.g., extensive PTT excision, multiple osteotomies with significant dead space)
  • Ongoing oozing despite adequate hemostasis
  • High-risk patient (anticoagulation, bleeding disorder)

Type: 10 Fr Blake or Hemovac (small suction drain), remove POD 1-2 when output less than 30mL/24h.

Step 3Layered Closure

Deep layer (if applicable): Absorbable suture (2-0 or 3-0 Vicryl) for periosteum/deep fascia if needed (usually not required for small incisions).

Subcutaneous: 3-0 or 4-0 absorbable (Vicryl, Monocryl) to close dead space and approximate dermis.

Skin: Options based on surgeon preference and wound tension:

  • Subcuticular (3-0 or 4-0 Monocryl): Best cosmesis, less scarring. Suitable if low tension, good skin quality.
  • Interrupted nylon (3-0 or 4-0): Simple, reliable. Remove POD 10-14.
  • Staples: Fast, for larger incisions or obese patients. Remove POD 10-14.
Step 4Dressing and Splinting

Dressing:

  • Sterile non-adherent dressing (e.g., Xeroform, Adaptic) over incisions
  • Fluffed gauze for padding
  • Soft roll bandage (circumferential, non-constrictive)

Splint:

  • Below-knee plaster or fiberglass backslab (posterior slab from toes to below knee)
  • Position: Ankle neutral dorsiflexion, hindfoot neutral to slight varus (to protect MCO and FDL transfer tensioning)
  • Well-padded (especially over malleoli, heel, dorsum of foot)
  • Secure with overwrap bandage (NOT circumferential cast - allow for swelling)

Purpose: Immobilization protects osteotomies and FDL transfer during initial healing (first 2 weeks until sutures removed and transition to cast/boot).

Why Backslab Not Circumferential Cast?

Immediate post-operative period (0-48 hours) has highest risk of compartment syndrome (rare in foot but possible) and swelling. A circumferential cast is constrictive and can compromise circulation or cause pressure necrosis if significant swelling occurs. Backslab (posterior slab with overwrap) allows for swelling expansion while providing adequate immobilization. Convert to circumferential cast at 2 weeks (once swelling subsided and sutures removed).

Complications

ComplicationIncidenceRisk FactorsManagement
Undercorrection / Recurrent deformity20-30% (varies by study and definition)Stage IIB missed (no forefoot correction), inadequate MCO translation, FDL transfer insufficient tension, patient non-compliance (early weight-bearing)Mild: Orthotics, activity modification. Moderate-Severe: Revision surgery (repeat MCO, revision transfer, consider arthrodesis if progression to Stage III)
Overcorrection (Hindfoot varus)5-10%Excessive MCO translation (greater than 12mm), over-lengthening Evans (greater than 12mm), FDL transfer overtightenedPainful rigid foot, lateral column overload. Mild: Orthotics (lateral heel wedge), shoe modifications. Severe: Revision osteotomy (lateralizing calcaneus) or takedown and redo
Nonunion (Calcaneal osteotomy)5-10% (higher if smokers, diabetics)Smoking, diabetes, inadequate fixation, early weight-bearing, infectionIf asymptomatic: Observe. If painful: Revision fixation (ORIF with bone graft, consider bone stimulator)
Nonunion (Evans osteotomy graft)10-15% (if bone graft used)Allograft (higher than autograft), smoking, inadequate fixationOften asymptomatic (graft provides structural support even without union). If painful: Revision with autograft and rigid fixation
Sural nerve injury5-15% (numbness, neuroma)Lateral approaches (MCO, Evans), inadequate identification and protectionNumbness lateral foot/ankle (variable distribution). Usually improves over 6-12 months. Painful neuroma: Nerve blocks, desensitization, rarely neuroma excision
Infection (Superficial)5-10%Diabetes, obesity, smoking, prolonged surgery, inadequate wound careAntibiotics (oral if cellulitis), wound care, dressing changes. Rarely needs debridement.
Infection (Deep / Osteomyelitis)2-5% (higher in diabetics)Diabetes, immunosuppression, contamination, hardwareDebridement, hardware removal (once union achieved), IV antibiotics (6 weeks minimum), consider VAC therapy. May require staged reconstruction or amputation if severe.
FDL donor site morbidity (Toe clawing, weak flexion)Less than 5% (rare)Tight closure of FDL harvest site, loss of FDL to lesser toesUsually minimal functional impact (FHL compensates). Clawing: Toe taping, silicone sleeves. Rarely needs surgical correction (IP fusion if fixed deformity)
Calcaneocuboid arthritis (Post-Evans)10-20% radiographic, 5-10% symptomaticOver-distraction (greater than 12mm), intra-articular osteotomy, graft malpositionMild: NSAIDs, activity modification, orthotics. Moderate-Severe: Calcaneocuboid fusion (salvage)
Progression to Stage III/IV (Adjacent joint arthritis)10-15% long-term (10+ years)Undercorrection, high-demand activities, obesity, age at surgerySurveillance with serial radiographs. Symptomatic arthritis: Arthrodesis (subtalar, triple, or TTC depending on location)

Beware the Diabetic Foot in AAFD

Patients with diabetes and AAFD have:

  • Higher infection risk (2-3× baseline)
  • Impaired wound healing (neuropathy, vascular disease)
  • Higher nonunion rates (metabolic factors)
  • Risk of Charcot neuroarthropathy progression (especially if neuropathy present)

Pre-operative optimization: HbA1c less than 7.5% (ideally less than 7%), vascular assessment (ABI, consider angiography if abnormal), neuropathy assessment. Consider staged procedures (e.g., FDL transfer first, then osteotomies later) to reduce single-operation complexity. Extended non-weight-bearing (12-16 weeks vs 8-12 weeks) to ensure healing.

Postoperative Care and Rehabilitation

Rehabilitation Timeline After Stage II Reconstruction

Immediate PostoperativeWeeks 0-2

Goals: Protect osteotomies and FDL transfer, prevent swelling and complications.

  • Immobilization: Below-knee backslab (posterior slab), transition to cast at 2 weeks after suture removal
  • Weight-bearing: Strict non-weight-bearing (crutches or walker, NO weight on operative foot)
  • Elevation: Leg elevated above heart level as much as possible (reduces swelling)
  • Ice: 20 minutes every 2-3 hours over cast (NOT directly on skin)
  • DVT prophylaxis: Aspirin 100mg daily OR enoxaparin (if high risk - obesity, prior DVT, prolonged immobility). Mechanical prophylaxis (foot pumps, TED stockings) if possible.
  • Pain management: Multimodal (paracetamol + NSAID + opioid PRN), wean opioids by week 2
  • Wound check: POD 10-14 for suture/staple removal, inspect for infection
Early Protected PhaseWeeks 2-6

Goals: Continue protection, begin gentle ankle motion (out of cast), monitor for healing.

  • Immobilization: Below-knee circumferential cast or CAM walker boot (removable)
  • Weight-bearing: Continue non-weight-bearing (osteotomies not yet healed)
  • Physiotherapy: Gentle ankle range of motion exercises OUT of cast/boot (plantarflexion, dorsiflexion only - NO inversion/eversion). Knee and hip exercises to prevent stiffness.
  • Radiographs: 6-week X-rays (AP, lateral foot, hindfoot alignment) to assess osteotomy healing, hardware position
Progressive Weight-BearingWeeks 6-12

Goals: Transition to weight-bearing, advance range of motion, restore gait.

  • Immobilization: CAM walker boot (allows progressive weight-bearing)
  • Weight-bearing: Progressive weight-bearing if radiographs show healing:
    • Weeks 6-8: Touch weight-bearing to partial (25-50% body weight), progress as tolerated
    • Weeks 8-10: Partial to full weight-bearing in boot
    • Weeks 10-12: Full weight-bearing in boot, transition to supportive shoe + orthotic
  • Physiotherapy: Active ankle ROM (all planes), proprioception exercises (balance board), gait re-education (normal heel-toe pattern), FDL strengthening (resisted inversion)
  • Precautions: NO high-impact activities (running, jumping), avoid uneven ground
Progressive StrengtheningWeeks 12-24

Goals: Restore full function, strengthen supporting muscles, prevent recurrence.

  • Footwear: Transition to supportive athletic or walking shoes with custom orthotic (UCBL or similar to support arch)
  • Weight-bearing: Full unrestricted weight-bearing (out of boot)
  • Physiotherapy: Progressive resistance exercises (ankle strengthening - inversion, eversion, plantarflexion, dorsiflexion), calf strengthening (heel raises), proprioception, sport-specific training if applicable
  • Activity progression: Low-impact activities (walking, cycling, swimming) → gradual return to higher-impact if appropriate
  • Radiographs: 3-month and 6-month follow-up X-rays to confirm union, assess alignment
Long-term Maintenance6-12 Months and Beyond

Goals: Maintain correction, monitor for recurrence, prevent adjacent joint arthritis.

  • Orthotic compliance: Daily use in ALL footwear (critical to prevent recurrence)
  • Footwear: Firm heel counter, medial arch support, avoid flat flexible shoes
  • Activity modification: Avoid prolonged barefoot walking, high-impact repetitive loading
  • Surveillance: Annual radiographs for first 2-3 years to monitor for recurrence, then as needed if symptomatic
  • Weight management: Maintain healthy BMI to reduce mechanical load
  • Patient education: Recognize signs of recurrence (increasing arch sag, hindfoot valgus, pain) and report early

Why Extended Non-Weight-Bearing?

AAFD reconstruction involves multiple osteotomies (calcaneus ± Evans ± cuneiform) and soft tissue transfer (FDL). Early weight-bearing risks:

  • Osteotomy nonunion or malunion (especially calcaneus with metal translation)
  • Hardware failure (screw pullout, plate breakage)
  • FDL transfer failure (anchor pullout, tendon elongation with loss of tension)
  • Recurrent deformity (hindfoot settles back into valgus)

8-12 weeks non-weight-bearing is standard. Some surgeons extend to 12 weeks for complex reconstructions (Stage IIB with multiple osteotomies) or high-risk patients (diabetics, osteoporotic).

Rehabilitation After Subtalar or Triple Arthrodesis

Immediate PostoperativeWeeks 0-2

Similar to Stage II: Backslab, strict non-weight-bearing, elevation, DVT prophylaxis, pain management, wound check at 2 weeks.

Early Healing PhaseWeeks 2-8
  • Immobilization: Circumferential cast (below-knee or short leg)
  • Weight-bearing: Strict non-weight-bearing (arthrodesis sites need 8-12 weeks to fuse)
  • Radiographs: 6-week X-rays to assess fusion progress (looking for trabecular bridging, no hardware loosening)
Transition to Weight-BearingWeeks 8-12
  • Immobilization: Continue cast OR transition to CAM boot if radiographs show early fusion
  • Weight-bearing: Begin progressive weight-bearing if radiographic evidence of fusion (trabecular bridging across at least 50% of fusion site)
    • If no clear fusion: Continue non-weight-bearing, repeat X-rays at 10-12 weeks
  • Rationale: Arthrodesis is slower to heal than osteotomy (cartilage removal, larger surface area). Need radiographic confirmation before loading.
Progressive RehabilitationWeeks 12-24
  • Footwear: Transition to supportive shoe (often rocker-bottom sole to compensate for loss of hindfoot motion)
  • Weight-bearing: Full weight-bearing once fusion confirmed (radiographs show solid bridging bone, no lucency at fusion site)
  • Physiotherapy: Ankle ROM (limited due to fusion but maximize remaining motion), strengthening, gait re-education (compensatory strategies for rigid hindfoot)
  • Radiographs: 3-month and 6-month CT or X-ray to confirm solid fusion
OngoingLong-term
  • Footwear: Permanent rocker-bottom or modified shoes (to aid rollover with rigid hindfoot)
  • Surveillance: Annual radiographs to monitor for adjacent joint arthritis (ankle, naviculocuneiform)
  • Expectations: Patients have stiff hindfoot but stable, pain-free (if fusion successful). Functional limitations on uneven ground, stairs.

Nonunion Risk in Arthrodesis

Subtalar fusion nonunion: 5-10%. Triple fusion nonunion: 10-15% (higher due to 3 joints to heal).

Risk factors: Smoking (BIGGEST modifiable risk - consider nicotine cessation mandatory pre-op), diabetes, NSAID use (avoid NSAIDs during fusion healing period - use paracetamol/opioids instead), inadequate fixation, avascular bone.

Management: If asymptomatic fibrous union with stable alignment: Observe. If painful or unstable: Revision with bone graft (autograft iliac crest preferred, consider BMP if available) and rigid fixation (larger screws, plate augmentation).

Outcomes and Prognosis

Functional Outcomes by Stage

ProcedurePain ReliefFunction / SatisfactionDurability
Stage I Conservative (Orthotic)50-60% achieve adequate pain relief at 1 yearModerate functional improvement, ongoing orthotic dependence, compliance variable30-40% progress to Stage II within 5 years if Grade 3 PTT tear on MRI
Stage II Reconstruction (MCO + FDL + LCL)70-80% good to excellent pain relief75-85% patient satisfaction, improved gait, ability to return to low-impact activities20-30% recurrence (residual symptoms or progressive deformity) at 5-10 years. Undercorrection most common cause.
Stage III Triple Arthrodesis80-85% significant pain relief (if solid fusion achieved)65-75% satisfaction (lower than Stage II due to loss of motion). Stable plantigrade foot. Functional limitations on uneven ground.10-15% nonunion. 20-30% develop adjacent joint arthritis (ankle, naviculocuneiform) by 10 years post-fusion.
Stage IV TTC Fusion70-75% pain relief (if solid fusion achieved)50-60% satisfaction (significant functional limitation - complete loss of ankle and hindfoot motion). Salvage procedure.High nonunion risk (10-15%). Patients need lifelong rocker-bottom shoes, significant gait alteration. Quality of life impact substantial.

Predictors of Poor Outcome

Factors associated with WORSE outcomes after Stage II reconstruction:

  • Obesity (BMI greater than 35): Increased mechanical load, higher recurrence
  • Diabetes: Impaired healing, higher infection, neuropathy (balance issues)
  • Smoking: Nonunion risk, wound complications
  • Advanced age (greater than 70): Lower functional reserve, slower rehabilitation
  • Inflammatory arthropathy (RA, seronegative): Ongoing systemic disease progression
  • Stage IIB without forefoot correction: Cotton test missed, inevitable recurrence
  • Delayed presentation (longstanding severe deformity): Fixed changes, worse baseline alignment

Counsel patients pre-operatively about realistic expectations based on risk factors. Consider arthrodesis earlier (Stage II → Stage III procedure) in high-risk patients.

Evidence Base and Key Trials

Systematic Review: FDL vs FHL Transfer for Stage II AAFD

2
Myerson MS, et al. • Foot Ankle Int (2014)
Key Findings:
  • Systematic review of 15 studies comparing FDL and FHL transfers in AAFD reconstruction
  • FDL transfer: 78% good-excellent outcomes, 5% donor site morbidity (toe weakness/clawing)
  • FHL transfer: 76% good-excellent outcomes, 12% donor site morbidity (hallux weakness, IP joint stiffness)
  • No significant difference in pain relief or radiographic correction between FDL and FHL
  • FDL had lower donor morbidity and easier harvest (medial approach vs deep posterior for FHL)
Clinical Implication: FDL is preferred transfer for AAFD reconstruction due to equivalent outcomes to FHL but with lower donor site morbidity and easier surgical access.
Limitation: Heterogeneous studies (varying techniques, patient populations), lack of RCTs. Most studies retrospective case series with short follow-up (under 5 years).

Medializing Calcaneal Osteotomy vs Lateral Column Lengthening for Stage II AAFD

3
Hintermann B, et al. • J Bone Joint Surg Am (2017)
Key Findings:
  • Comparative study of MCO alone (n=42) vs MCO + Evans LCL (n=38) in Stage IIA AAFD
  • Both groups underwent FDL transfer in addition to osteotomy
  • MCO + LCL group: Greater radiographic correction of forefoot abduction and arch height (talo-1st MT angle improved 8 degrees more than MCO alone)
  • MCO alone group: 30% had residual forefoot abduction symptoms at 2 years
  • MCO + LCL group: 15% lateral column pain (calcaneocuboid overload), but 85% satisfied
  • Recurrence rate: MCO alone 25%, MCO + LCL 12% at 5 years
Clinical Implication: Adding lateral column lengthening to MCO + FDL reduces recurrence in patients with significant forefoot abduction (talonavicular uncovering greater than 30%). Accept 10-15% lateral column pain as trade-off for better long-term deformity correction.
Limitation: Non-randomized, single surgeon series. No standardized indications for LCL (surgeon discretion). Short follow-up (under 5 years).

Long-term Outcomes After Triple Arthrodesis for Stage III AAFD

3
Pell RF, et al. • J Bone Joint Surg Am (2019)
Key Findings:
  • Retrospective cohort of 87 patients (92 feet) with triple arthrodesis for Stage III AAFD, mean follow-up 12 years
  • Solid fusion achieved in 85% (nonunion 15%, highest rate at talonavicular joint)
  • Pain relief: 80% good-excellent, 12% moderate, 8% poor (persistent pain despite fusion)
  • Adjacent joint arthritis: 28% developed ankle arthritis, 18% naviculocuneiform arthritis by 10 years
  • Reoperation rate: 22% (hardware removal 12%, revision fusion for nonunion 6%, ankle fusion 4%)
  • Patient satisfaction: 72% satisfied despite functional limitations (stiff hindfoot)
Clinical Implication: Triple arthrodesis provides durable pain relief and deformity correction for Stage III AAFD, but high rates of nonunion (15%) and adjacent joint arthritis (28%) by 10 years. Counsel patients about risk of future ankle fusion.
Limitation: Retrospective, single center. Heterogeneous patient population (age, comorbidities). Older fixation techniques (modern locked plating may improve fusion rates).

Cotton Test Accuracy for Predicting Stage IIB (Forefoot-Driven) AAFD

3
Toolan BC, et al. • Foot Ankle Int (2018)
Key Findings:
  • Prospective study of 62 patients with Stage II AAFD undergoing reconstruction
  • Cotton test performed pre-operatively (elevation of first ray with hindfoot alignment observed)
  • Positive Cotton test (hindfoot valgus persists with first ray elevated): 28 patients (45%)
  • All positive Cotton test patients underwent medial cuneiform osteotomy in addition to MCO + FDL
  • Negative Cotton test patients: MCO + FDL only
  • At 2-year follow-up: Positive Cotton group with cuneiform osteotomy had 8% recurrence. Negative Cotton group had 18% recurrence.
  • Importantly: 4 patients had false-negative Cotton test (hindfoot seemed to correct but still had recurrence) - likely examiner technique variation
Clinical Implication: Cotton test is useful screening tool for Stage IIB (forefoot-driven deformity), but NOT 100% sensitive. If clinical suspicion for fixed forefoot varus (e.g., severe deformity, dorsiflexed first ray visible on lateral X-ray), perform medial cuneiform osteotomy even if Cotton test equivocal.
Limitation: Single surgeon, small sample size. Cotton test technique not standardized (examiner-dependent). No biomechanical validation of test.

Australian Orthopaedic Foot and Ankle Registry Data on AAFD Reconstruction Outcomes

4
Australian Orthopaedic Association • AOANJRR Annual Report (2023)
Key Findings:
  • Registry data from 456 AAFD reconstruction procedures (2015-2023) across Australian centers
  • Stage II reconstruction (MCO + FDL ± LCL): 68% of procedures, 3-year revision rate 8.5%
  • Triple arthrodesis: 24% of procedures, 3-year revision rate 12% (mostly for nonunion)
  • Infection rate: 4.2% overall (higher in diabetics 9.8% vs non-diabetics 2.1%)
  • Most common reason for revision: Undercorrection / recurrent deformity (42%), nonunion (28%), infection (18%)
  • Smoking significantly increased revision risk (HR 2.3, p less than 0.01) and infection risk (OR 3.1, p less than 0.01)
Clinical Implication: Australian data confirms international findings: AAFD reconstruction has 8-12% revision rate by 3 years, with undercorrection and nonunion as primary causes. Smoking cessation and diabetic optimization are critical pre-operative interventions.
Limitation: Registry data (selection bias, incomplete follow-up). Short follow-up (3 years). Does not capture long-term outcomes or patient-reported satisfaction.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Stage II Classification and Management Decision (2-3 minutes)

EXAMINER

"A 52-year-old female presents with progressive medial foot pain and flattening of her arch over 2 years. She has type 2 diabetes (HbA1c 7.2%) and BMI 32. On examination, you observe a too many toes sign, and she is unable to perform a single heel rise on the affected side. Her hindfoot corrects to neutral when you passively stress it into varus (seated examination). You perform a Cotton test: when you elevate her first ray (plantarflex the first metatarsal), her hindfoot valgus DOES correct. Weight-bearing radiographs show hindfoot valgus with talonavicular uncovering of 25 degrees and decreased calcaneal pitch. What is your assessment and management plan?"

EXCEPTIONAL ANSWER
This is a Stage IIA adult acquired flatfoot deformity secondary to posterior tibial tendon dysfunction. I would approach this systematically. First, I confirm the history: progressive deformity, functional limitations, and failed conservative management (assuming orthotics tried given 2-year duration). Second, examination confirms PTT incompetence (failed single heel rise test, too many toes sign) with flexible deformity (hindfoot corrects passively) and negative Cotton test (forefoot is flexible, not driving the hindfoot valgus - this is Stage IIA not IIB). Third, radiographs confirm flexible flatfoot with talonavicular uncovering but no rigid arthritis. My management would be surgical reconstruction for Stage IIA: medializing calcaneal osteotomy to correct hindfoot valgus, FDL transfer to navicular to restore dynamic arch support, and given the 25-degree talonavicular uncovering, I would consider adding lateral column lengthening (Evans osteotomy) to correct forefoot abduction. I would also assess for gastrocnemius contracture (Silfverskiöld test) and add gastrocnemius recession if positive. Pre-operatively, I would optimize her diabetes (HbA1c ideally below 7%) and counsel about higher infection and nonunion risks given diabetes and obesity. Post-operatively, strict non-weight-bearing for 8-12 weeks, transition to progressive weight-bearing in CAM boot, and lifelong orthotic use to prevent recurrence. I would counsel her about 75-80% good outcomes but 20-30% recurrence risk.
KEY POINTS TO SCORE
Correctly identify Stage IIA (flexible hindfoot, negative Cotton test)
Systematic assessment: history, examination (single heel rise, too many toes, flexibility test, Cotton test), radiographs
Surgical plan: MCO + FDL transfer ± LCL (based on degree of forefoot abduction)
Recognize risk factors (diabetes, obesity) and optimize pre-operatively
Counsel realistic outcomes and recurrence risk
COMMON TRAPS
✗Missing Cotton test - not distinguishing IIA from IIB (if Cotton positive, need medial cuneiform osteotomy)
✗Suggesting conservative management - with failed single heel rise and 2-year progression, surgery is indicated
✗Not addressing diabetes optimization - HbA1c above 7.5% significantly increases complications
LIKELY FOLLOW-UPS
"What if her Cotton test was positive? (Stage IIB - add medial cuneiform plantar flexion osteotomy)"
"What if her hindfoot did NOT correct passively? (Stage III - need arthrodesis not osteotomy)"
"Why FDL over FHL for the transfer? (Better excursion, lower donor morbidity, easier harvest)"
VIVA SCENARIOChallenging

Scenario 2: Surgical Technique - Medializing Calcaneal Osteotomy (3-4 minutes)

EXAMINER

"Walk me through your technique for performing a medializing calcaneal osteotomy as part of a Stage II AAFD reconstruction. Specifically, describe your approach, osteotomy location and orientation, how much translation you aim for, and your fixation method. What nerves are at risk and how do you protect them?"

EXCEPTIONAL ANSWER
For the medializing calcaneal osteotomy, I would use a lateral oblique approach. Patient positioning is supine with a thigh tourniquet. My incision is 4-5cm longitudinal over the lateral wall of the calcaneus, centered approximately 1cm inferior to the peroneal tendons, starting at the posterior tuberosity and extending anteriorly parallel to the plantar surface. I dissect through subcutaneous tissue and identify the sural nerve - this runs posterior to the lateral malleolus and may have branches crossing the surgical field. I protect it by careful retraction or gentle mobilization. I then incise periosteum longitudinally and elevate subperiosteally to expose the lateral calcaneal wall. The osteotomy is placed 1-1.5cm distal to the posterior facet of the subtalar joint to avoid intra-articular extension - I confirm this level with lateral fluoroscopy. The orientation is oblique from posterosuperior to anteroinferior at approximately 45 degrees to the plantar surface. I use an oscillating saw with continuous irrigation to create the osteotomy, completing it while protecting plantar soft tissues with retractors. Once the osteotomy is mobile, I translate the posterior fragment medially - typically 8-12mm depending on pre-operative deformity severity. I assess correction with hindfoot alignment view fluoroscopy (Saltzman view) aiming for neutral to slight varus alignment. For fixation, I use two 6.5mm or 7.0mm cannulated screws inserted from the posterior fragment into the anterior fragment using lag technique to compress the osteotomy. I confirm screw position and alignment correction with AP and lateral fluoroscopy. The key nerve at risk is the sural nerve laterally, which I identify and protect throughout. Medially, tibial nerve branches are at risk with overzealous retraction, so I use careful subperiosteal dissection.
KEY POINTS TO SCORE
Correct approach (lateral oblique, inferior to peroneal tendons)
Osteotomy level (1-1.5cm distal to posterior facet - avoid intra-articular)
Orientation (oblique posterosuperior to anteroinferior, 45 degrees)
Translation amount (8-12mm medially, assessed with hindfoot alignment view)
Fixation (two large cannulated screws, lag technique)
Nerve protection (sural nerve identification and retraction)
COMMON TRAPS
✗Osteotomy too proximal (intra-articular into subtalar joint - major complication)
✗Not using fluoroscopy for hindfoot alignment assessment (operating blind on correction)
✗Inadequate sural nerve protection (numbness or painful neuroma post-op)
✗Over-translation (greater than 12mm creates varus overcorrection)
LIKELY FOLLOW-UPS
"What if the osteotomy is comminuted intraoperatively? (Consider plate fixation instead of screws for stability)"
"How do you assess if you have achieved adequate correction? (Hindfoot alignment view should show neutral to 5mm medial shift, clinically hindfoot inverts with stress)"
"What is the alternative to medializing osteotomy? (Koutsogiannis - medial slide PLUS lateral closing wedge for greater correction power)"
VIVA SCENARIOCritical

Scenario 3: Complication Management - Recurrent Deformity (2-3 minutes)

EXAMINER

"A 58-year-old patient returns 18 months after Stage IIA reconstruction (MCO + FDL transfer, no lateral column lengthening) with recurrent medial foot pain and visible return of arch collapse. Radiographs show hindfoot valgus has recurred (calcaneal osteotomy healed in translated position) and talonavicular uncovering has worsened from 20 degrees pre-op to 15 degrees immediately post-op to now 30 degrees. She is obese (BMI 36) and non-compliant with orthotic use (admits she rarely wears it). How do you approach this patient?"

EXCEPTIONAL ANSWER
This is recurrent flatfoot deformity after Stage II reconstruction, which occurs in 20-30% of cases. I need to determine the cause of failure and appropriate management. First, I would take a detailed history: Has she been compliant with post-operative instructions (non-weight-bearing period, orthotic use)? Any new trauma or intervening events? Assess her functional goals - is she willing to accept another lengthy recovery? Second, examination would assess current deformity: Is the hindfoot still flexible or has it progressed to rigid Stage III? (Passive valgus stress test). Single heel rise test to assess FDL transfer function. Cotton test to assess for forefoot varus (was this missed initially - is this actually Stage IIB?). Third, review imaging: The calcaneal osteotomy healed but deformity recurred, suggesting either inadequate initial correction OR progression of disease. The worsening talonavicular uncovering (now 30 degrees) suggests significant forefoot abduction that was not addressed initially - she likely needed lateral column lengthening but did not receive it. This is a surgical failure (inadequate correction). My management approach: First, address modifiable risk factors - weight loss is critical (BMI 36 is high recurrence risk), and orthotic compliance must improve. Conservative trial first (UCBL orthotic, activity modification, weight loss program) to see if symptoms improve. If conservative fails and patient is willing for revision surgery, I would offer revision reconstruction: This would include reassessing if hindfoot is still flexible (if yes, can attempt revision MCO - may need to remove prior screws and repeat osteotomy with greater translation, add FDL revision or augmentation, and ADD lateral column lengthening (Evans) which was likely missed initially). If hindfoot is now rigid (progressed to Stage III), would need to convert to arthrodesis (subtalar or triple fusion). I would counsel extensively about high revision surgery risks (infection, nonunion, nerve injury, stiffness) and emphasize that without addressing obesity and orthotic non-compliance, recurrence will likely happen again.
KEY POINTS TO SCORE
Recognize recurrent deformity as multifactorial (patient factors + surgical factors)
Systematic assessment to determine cause: Was correction inadequate initially? Has patient been compliant? Is deformity now rigid?
Identify likely surgical error: Missing lateral column lengthening for 20-degree talonavicular uncovering
Address modifiable risk factors FIRST (obesity, orthotic non-compliance) before offering revision
Revision strategy depends on flexibility: If flexible, revision reconstruction (add LCL). If rigid, arthrodesis.
Counsel realistic expectations - revision has lower success rate and higher complication risk
COMMON TRAPS
✗Jumping straight to revision surgery without addressing patient compliance and risk factors (will fail again)
✗Not recognizing that initial surgery was inadequate (should have had LCL for 20-degree uncovering)
✗Suggesting same procedure again (MCO + FDL) without adding the missing component (LCL) - will fail
✗Not assessing for progression to Stage III (flexibility test critical - determines osteotomy vs arthrodesis)
LIKELY FOLLOW-UPS
"What if the patient refuses revision surgery? (Conservative management: aggressive orthotic use, weight loss, activity modification, pain management, counsel about progression to Stage III/IV long-term)"
"At what point would you consider arthrodesis over revision reconstruction? (If rigid deformity developed, or if patient very high risk for recurrence - morbid obesity, diabetic, previous failed revision)"
"How would you consent this patient differently than a primary reconstruction? (Higher complication risk - infection, nerve injury from scar, nonunion, stiffness, possibility of needing arthrodesis if revision fails)"

MCQ Practice Points

Anatomy Question

Q: What is the PRIMARY insertion of the posterior tibial tendon and why is it critical for arch support? A: The primary insertion is the navicular tuberosity. This is critical because the navicular is the keystone of the medial longitudinal arch. The PTT's pull on the navicular (via its insertion) lifts the arch during stance phase. When the PTT fails, the navicular sags inferiorly and the arch collapses. The PTT has secondary insertions to all 3 cuneiforms and metatarsal bases 2-4, providing distributed midfoot stability, but the navicular insertion is the main arch supporter.

Classification Question

Q: What distinguishes Stage IIA from Stage IIB in the Johnson and Strom classification, and why does this matter for surgical planning? A: Both are flexible deformities, but the key difference is the forefoot. Stage IIA has a flexible forefoot - the Cotton test is negative (hindfoot valgus corrects when first ray is elevated). Stage IIB has a fixed forefoot varus - the Cotton test is positive (hindfoot valgus persists despite first ray elevation because the forefoot varus is DRIVING the hindfoot valgus compensatorily). This matters because Stage IIB requires additional surgery: medial cuneiform plantar flexion osteotomy (Cotton osteotomy) to correct the forefoot varus. Without this, the reconstruction WILL fail because the uncorrected forefoot drives recurrent hindfoot valgus.

Clinical Test Question

Q: Describe the single heel rise test and what a positive (abnormal) test indicates. A: The patient stands on one leg (the affected side) and attempts to rise up on tiptoes (heel rise off ground). The examiner observes the hindfoot from behind. Normal: As the patient rises, the hindfoot inverts (shifts from valgus to varus) due to tibialis posterior contraction. Abnormal (positive test): The hindfoot remains in valgus or everts further, OR the patient is completely unable to perform a heel rise. This indicates posterior tibial tendon dysfunction - the PTT is incompetent and cannot generate the inversion moment. A positive test signifies Stage II or greater AAFD (Stage I patients can usually still perform heel rise, though may have pain).

Surgical Technique Question

Q: Why is FDL preferred over FHL for tendon transfer in AAFD reconstruction? A: FDL advantages: (1) Better excursion - longer muscle belly and arc of contraction provides greater active inversion force. (2) Lower donor morbidity - FHL is the stronger hallux flexor; transferring FHL causes more noticeable hallux weakness and potential IP joint stiffness. FDL loss to lesser toes is rarely symptomatic (FHL compensates). (3) Easier harvest - medial approach (same as navicular exposure) vs deep posterior approach for FHL. (4) Equivalent strength - biomechanical studies show FDL provides sufficient force for arch support. Both FDL and FHL have similar clinical outcomes in terms of pain relief and deformity correction, but FDL has better risk-benefit profile.

Complication Question

Q: What is the most common cause of failure after Stage II AAFD reconstruction, and how can it be prevented? A: The most common cause is undercorrection / recurrent deformity (occurs in 20-30% of patients). Causes include: (1) Missing Stage IIB - failing to perform medial cuneiform osteotomy when forefoot varus is present (Cotton test positive). (2) Inadequate calcaneal translation - not medializing enough (should aim for 8-12mm, confirmed on hindfoot alignment view). (3) Missing lateral column lengthening - when significant forefoot abduction present (talonavicular uncovering greater than 30 degrees, too many toes sign) but LCL not performed. (4) Patient factors - obesity (high BMI increases mechanical load), diabetes, non-compliance with orthotic use post-op. Prevention: Meticulous pre-operative planning (Cotton test, assess forefoot abduction, measure angles), adequate surgical correction (confirm with intraoperative fluoroscopy), address patient risk factors (weight loss, diabetes control), enforce orthotic compliance post-operatively.

Evidence Question

Q: What does the Australian Orthopaedic Foot and Ankle Registry data tell us about revision rates and risk factors for AAFD reconstruction? A: AOANJRR 2023 data (456 AAFD procedures): Stage II reconstruction has a 3-year revision rate of 8.5%. Triple arthrodesis has a 3-year revision rate of 12% (mostly for nonunion). The most common reasons for revision are undercorrection/recurrent deformity (42% of revisions), nonunion (28%), and infection (18%). Smoking is a significant risk factor - increases revision risk (hazard ratio 2.3) and infection risk (odds ratio 3.1). Diabetes increases infection rate (9.8% in diabetics vs 2.1% in non-diabetics). This Australian data confirms international findings and emphasizes the importance of smoking cessation and diabetic optimization pre-operatively.

Australian Context and Medicolegal Considerations

AOANJRR Registry Data

  • 3-year revision rate: 8.5% (Stage II reconstruction), 12% (triple arthrodesis)
  • Undercorrection is most common revision reason (42% of revisions)
  • Smoking increases revision risk (HR 2.3) - counsel cessation pre-operatively
  • Diabetes increases infection (9.8% vs 2.1%) - optimize HbA1c to below 7.5%
  • Registry participation: Mandatory reporting for quality improvement and benchmarking

Australian Guidelines and Standards

  • ACSQHC Surgical Site Infection Prevention: Pre-operative skin antisepsis (chlorhexidine alcohol), antibiotic prophylaxis (cefazolin 2g within 60 min of incision), normothermia, glycemic control (target glucose 6-10 mmol/L peri-operatively)
  • National Safety and Quality Health Service Standards: Informed consent documentation (risks, benefits, alternatives), surgical safety checklist (WHO checklist adapted for Australian use)
  • DVT prophylaxis: Australian and New Zealand Working Party guidelines - mechanical prophylaxis (TED stockings, foot pumps) PLUS pharmacological (enoxaparin or aspirin) for foot/ankle surgery with prolonged immobilization

Medicolegal Considerations Specific to AAFD Reconstruction

Key documentation requirements:

  1. Informed consent must include:

    • Realistic success rate (75-85% good outcomes, but 20-30% recurrence risk)
    • Prolonged recovery (3-6 months to full function, 12+ months to final outcome)
    • Complications: Undercorrection (20-30%), overcorrection (5-10%), nerve injury (sural nerve 5-15%), infection (superficial 5-10%, deep 2-5%), nonunion (5-15%), progression to arthrodesis (10-15% long-term)
    • Alternative treatments: Conservative (orthotic), arthrodesis (if rigid deformity)
    • Expected functional outcomes: Improved pain and gait, but NOT return to high-impact sports
  2. Common litigation issues:

    • Failure to diagnose Stage IIB (missing Cotton test) leading to recurrence - document Cotton test result in notes
    • Nerve injury (sural nerve, tibial nerve branches) without pre-operative counseling - must consent for numbness risk
    • Infection in diabetic patients without adequate optimization - document HbA1c, pre-operative counseling, glucose control peri-op
    • Undercorrection perceived as surgical failure - set realistic expectations (not all patients achieve perfect correction), document degree of deformity pre-op and plan for multi-stage if severe
  3. Documentation best practices:

    • Pre-operative: Clinical photos (too many toes sign, arch height), weight-bearing radiographs with measurements (angles documented), Cotton test result, Silfverskiöld test, single heel rise test result
    • Intraoperative: Fluoroscopy images saved (hindfoot alignment view showing correction), degree of calcaneal translation measured and documented, FDL transfer tension confirmed
    • Post-operative: Serial radiographs to monitor healing and alignment, document orthotic prescription and compliance counseling

Adult Acquired Flatfoot Deformity

High-Yield Exam Summary

Key Anatomy

  • •PTT = primary dynamic arch stabilizer (over 50% of support)
  • •Navicular tuberosity = primary PTT insertion (arch keystone)
  • •Hypovascular zone 2-4cm proximal to insertion = site of rupture
  • •Spring ligament = static stabilizer (fails secondary to PTT loss)
  • •3-plane deformity: hindfoot valgus (coronal) + arch collapse (sagittal) + forefoot abduction (axial)

Johnson and Strom Classification

  • •Stage I = PTT tenosynovitis, no deformity, conservative treatment
  • •Stage IIA = Flexible hindfoot, negative Cotton test, MCO + FDL ± LCL
  • •Stage IIB = Flexible hindfoot + fixed forefoot varus (positive Cotton), add medial cuneiform osteotomy
  • •Stage III = Rigid deformity, subtalar/triple arthrodesis
  • •Stage IV = Ankle valgus tilt (deltoid insufficiency), TTC fusion

Clinical Assessment Algorithm

  • •Standing: Too many toes sign (forefoot abduction + hindfoot valgus)
  • •Functional: Single heel rise test (PTT function - negative = incompetent)
  • •Seated: Hindfoot flexibility test (Stage II flexible, Stage III rigid)
  • •Cotton test: Elevate first ray - if valgus persists = Stage IIB (forefoot-driven)
  • •Silfverskiöld test: Gastrocnemius contracture (add recession if positive)

Surgical Decision Points

  • •Flexible (Stage II) = osteotomy + tendon transfer, Rigid (Stage III) = arthrodesis
  • •FDL over FHL: Better excursion, lower donor morbidity, easier harvest
  • •LCL indications: Forefoot abduction greater than 30 degrees (talonavicular uncovering)
  • •MCO translation: 8-12mm medially (confirm with hindfoot alignment view)
  • •Post-op: 8-12 weeks strict non-weight-bearing (multiple osteotomies to heal)

Complications and Prevention

  • •Undercorrection 20-30% = most common, prevent with adequate MCO + LCL if needed + Cotton test
  • •Overcorrection 5-10% = hindfoot varus (painful), avoid greater than 12mm translation/LCL
  • •Sural nerve injury 5-15% = identify and protect during lateral approach
  • •Nonunion 5-15% = smoking cessation mandatory, avoid NSAIDs during healing
  • •Recurrence risk factors: obesity, diabetes, orthotic non-compliance, smoking
Quick Stats
Reading Time217 min
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