Progressive Collapse | PTT Dysfunction | Staged Reconstruction
- Johnson and Strom classification - guides surgical reconstruction strategy
- Rigid vs flexible - determines need for osteotomy vs arthrodesis
- Too many toes sign - pathognomonic for hindfoot valgus on clinical exam
- Single heel rise test - inability indicates PTT incompetence
- Stage II divide - IIA (flexible hindfoot) vs IIB (forefoot driven, fixed forefoot varus)
- “Stage IIB requires cotton test and first ray plantar flexion osteotomy
- “FDL is preferred tendon transfer over FHL (better excursion, less donor morbidity)
- “Lateral column lengthening (Evans) corrects forefoot abduction
- “Stage III requires subtalar or triple arthrodesis for rigid deformity
Johnson and Strom staging drives all treatment decisions. Stage I = conservative. Stage II = reconstruction (osteotomy + transfer). Stage III = arthrodesis. Stage IV = ankle fusion. Know the flexibility test!
Three-plane deformity: Hindfoot valgus (coronal), arch collapse (sagittal), forefoot abduction (axial). Examiners ask: "Describe the deformity." Answer systematically in all three planes.
Too many toes sign (view from behind) and single heel rise test (PTT function). Examiners will show photos. Also test: flexibility with hindfoot valgus stress and Silfverskiöld test for gastrocnemius contracture.
Flexible Stage II = calcaneal osteotomy (medializing or Koutsogiannis) plus FDL transfer plus possible lateral column lengthening. Stage IIB adds cotton test and medial cuneiform osteotomy. Rigid Stage III = triple arthrodesis. Know when to do what!
- Deformity
- PTT tenosynovitis, no deformity
- Treatment
- Immobilization (CAM boot 6-12 weeks), NSAIDs, orthotics
- Key Pearl
- Exam: medial ankle pain, no too many toes sign
- Deformity
- Flexible hindfoot valgus, passively correctable
- Treatment
- Medializing calcaneal osteotomy + FDL transfer ± LCL
- Key Pearl
- Cotton test negative, hindfoot corrects with stress
- Deformity
- Flexible hindfoot + fixed forefoot varus
- Treatment
- As IIA PLUS medial cuneiform plantar flexion osteotomy
- Key Pearl
- Cotton test positive, forefoot drives hindfoot valgus
- Deformity
- Rigid/fixed deformity, subtalar arthritis
- Treatment
- Subtalar arthrodesis or triple arthrodesis
- Key Pearl
- Hindfoot does NOT correct passively, talonavicular arthritis
- Deformity
- Ankle valgus tilt (deltoid insufficiency)
- Treatment
- Tibiotalocalcaneal arthrodesis (TTC fusion)
- Key Pearl
- Lateral ankle mortise widening on AP radiograph
MAINPosterior Tibial Tendon Anatomy
Hook:PTT is the MAIN support of the medial arch - lose it and the arch collapses!
Overview and Epidemiology
Adult acquired flatfoot deformity (AAFD) represents a progressive collapse of the medial longitudinal arch due to posterior tibial tendon (PTT) dysfunction. It is the most common cause of acquired flatfoot in adults and represents a spectrum from isolated tenosynovitis to severe fixed deformity with ankle involvement. Understanding the staged progression is critical for treatment selection.
- Age: Peak 40-60 years
- Gender: Female predominance (3:1 ratio)
- Obesity: Major risk factor (increased mechanical load)
- Diabetes: Associated with tendon degeneration
- Hypertension: Vascular compromise to tendon
- Previous trauma: Medial ankle injuries
- Inflammatory arthropathy: RA, seronegative arthritis
- Steroid use: Tendon weakening
- Progressive deformity: Without treatment, stage I → stage IV over years
- Functional limitation: Pain with prolonged walking/standing
- Gait alteration: Compensatory external rotation, antalgic gait
- Quality of life: Significant impact on mobility and footwear
- Adjacent joint arthritis: Talonavicular, subtalar, ankle involvement
- Bilateral progression: 20% develop contralateral AAFD
Pathophysiology and Mechanisms
The posterior tibial tendon provides over 50% of the dynamic support to the medial longitudinal arch during stance phase. Loss of PTT function results in progressive arch collapse, hindfoot valgus, and forefoot abduction - the classic flatfoot deformity. Unlike static ligamentous stabilizers (spring ligament, plantar fascia), the PTT actively resists arch collapse during gait, making it irreplaceable.
Posterior Tibial Tendon Course and Function
- Clinical Significance
- Strongest invertor of foot (tibialis posterior muscle)
- Exam Relevance
- Loss causes unopposed eversion (peroneus brevis dominates)
- Clinical Significance
- Hypovasular zone 2-4cm proximal to navicular insertion
- Exam Relevance
- Site of degenerative tendinopathy and eventual rupture
- Clinical Significance
- Main arch keystone stabilizer
- Exam Relevance
- Detachment here causes immediate arch collapse
- Clinical Significance
- Provides midfoot stability and transverse arch support
- Exam Relevance
- Multiple slips allow distributed force transfer
Static Stabilizers (Spring Ligament Complex)
The spring ligament (plantar calcaneonavicular ligament) is the primary static stabilizer of the medial arch. It consists of two components:
- Superomedial calcaneonavicular ligament: Main load-bearing structure, supports talar head
- Inferior calcaneonavicular ligament: Reinforces plantar aspect
In AAFD, the spring ligament undergoes attritional failure as the PTT loses function, leading to progressive talar head uncovering and arch collapse.
Three-Plane Deformity Components
Hindfoot valgus
- Subtalar joint eversion
- Lateral talar shift
- Posterior facet uncovering
- "Too many toes" sign
Arch collapse (planus)
- Talus plantar flexion
- Navicular sag
- Increased talo-first metatarsal angle
- Spring ligament attenuation
Forefoot abduction
- Talonavicular uncovering
- Lateral border of forefoot
- "Too many toes" sign
- Compensatory tibial external rotation
The Windlass Mechanism and the Arch-Supporting Partnership
The plantar fascia is named above as a static arch stabiliser, and the "windlass effect" is invoked when explaining how lateral column lengthening raises the arch. The mechanism is worth defining, because it is fundamental to how the medial arch is maintained - and how its loss contributes to collapse.
- The windlass mechanism: the plantar aponeurosis runs from the calcaneal tuberosity forwards to the plantar plates and proximal phalanges, passing beneath the metatarsal heads. During terminal stance / push-off, dorsiflexion of the metatarsophalangeal joints (especially the hallux) winds the aponeurosis around the metatarsal heads like a cable around a drum (the "windlass"), shortening the calcaneus-to-forefoot distance. This raises and rigidifies the medial longitudinal arch, converting a flexible foot into a stable lever for push-off.
- The arch-supporting partnership: arch height depends on three contributors acting together - the dynamic stabiliser (posterior tibial tendon), the static ligamentous stabilisers (spring ligament, plantar aponeurosis and plantar ligaments), and the bony architecture. As the PTT fails and the spring ligament attenuates, load is transferred onto the plantar fascia; an intact windlass partly compensates early, but with progressive collapse and forefoot abduction the windlass becomes mechanically inefficient (the lever arm and hallux purchase are lost) and the arch can no longer be re-tensioned at push-off.
- Surgical relevance: realigning the medial column and forefoot (for example by lateral column lengthening, Cotton osteotomy or medial column fusion) re-establishes an effective windlass, which is part of why these procedures raise the arch and improve push-off - not simply by changing static alignment.
The windlass mechanism describes how hallux / MTP dorsiflexion at push-off winds the plantar aponeurosis around the metatarsal heads, shortening it, raising the medial arch and locking the midfoot into a rigid lever. The arch is supported by a partnership of the dynamic PTT, the static spring ligament and plantar aponeurosis, and the bony architecture - flatfoot is the failure of this partnership, and a foot that has lost an efficient windlass cannot re-tension its arch for push-off.
Classification Systems
Johnson and Strom Classification (Most Widely Used)
- Clinical Findings
- PTT pain, swelling medial ankle. Normal alignment. Single heel rise positive. No too many toes sign.
- Radiographic Features
- Normal alignment. No arch collapse. No hindfoot valgus.
- Treatment
- Conservative: CAM boot 6-12 weeks, UCBL orthotic, NSAIDs, PT
- Clinical Findings
- Hindfoot valgus flexible. Arch collapses weight-bearing. Single heel rise negative. Too many toes positive. Cotton test negative.
- Radiographic Features
- Hindfoot valgus (AP: talar head uncovering). Lateral: sag at talonavicular, increased talo-1st MT angle.
- Treatment
- Medializing calcaneal osteotomy + FDL transfer ± lateral column lengthening
- Clinical Findings
- As IIA PLUS fixed forefoot varus (does not correct). Cotton test positive (forefoot drives hindfoot valgus).
- Radiographic Features
- As IIA PLUS forefoot varus visible on lateral radiograph with hindfoot corrected.
- Treatment
- As IIA PLUS medial cuneiform plantar flexion osteotomy (Cotton osteotomy)
- Clinical Findings
- Rigid hindfoot valgus (does NOT correct passively). Subtalar stiffness. Talonavicular pain/arthritis.
- Radiographic Features
- Subtalar arthritis. Talonavicular arthritis. Fixed hindfoot valgus. No passive correction on stress views.
- Treatment
- Subtalar arthrodesis or triple arthrodesis (± tendon transfer if Stage IIIA)
- Clinical Findings
- As Stage III PLUS lateral ankle pain. Deltoid insufficiency. Ankle valgus tilt.
- Radiographic Features
- Ankle valgus tilt on AP ankle (tilted talus). Tibiotalar arthritis. Lateral ankle mortise widening.
- Treatment
- Tibiotalocalcaneal (TTC) arthrodesis. Consider total ankle replacement in select cases.
Stage IIA: Flexible hindfoot valgus, hindfoot corrects when forefoot is unloaded. Cotton test negative. Treat with hindfoot procedures only.
Stage IIB: Flexible hindfoot valgus BUT fixed forefoot varus (first ray dorsiflexed). Cotton test positive (elevating heel under first ray does NOT correct arch). The forefoot varus drives the hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy PLUS hindfoot procedures. Missing this = failure.
TFRFJohnson and Strom Classification
Hook:AAFD progression: Tendon inflames → Flexibility lost in hindfoot → Rigidity sets in → Further spread to ankle!
Clinical Assessment
- Pain location: Medial ankle (PTT), lateral (peroneal overload, sinus tarsi), plantar (arch strain)
- Onset: Acute trauma vs insidious (degenerative)
- Progression: Worsening deformity over months to years
- Function: Walking distance, stairs, uneven ground tolerance
- Footwear: Difficulty fitting shoes, wearing out medial heel
- Red flags: Inflammatory symptoms (RA), neuropathy (diabetes), acute swelling (DVT)
Standing (weight-bearing):
- Too many toes sign (view from behind)
- Arch height vs normal side
- Hindfoot valgus alignment
- Forefoot abduction
- Compensatory external tibial rotation
Seated (non-weight-bearing):
- PTT palpation: swelling, tenderness, gap
- Single heel rise test (each foot separately)
- Hindfoot flexibility: valgus stress test
- Cotton test (Stage IIB screening)
- Silfverskiöld test (gastrocnemius contracture)
- Neurovascular examination
Key Clinical Tests
Examination Tests for AAFD
Technique: Examiner stands behind patient who stands with feet shoulder-width apart.
Positive test: More than 2 toes visible lateral to heel on affected side (normally only 5th and part of 4th visible). Indicates forefoot abduction and hindfoot valgus.
Sensitivity: 90% for Stage II or greater AAFD.
Technique: Patient stands on one leg, rises up on toes (heel off ground). Observe hindfoot from behind.
Normal: Hindfoot inverts (heel shifts to varus) as PTT contracts and arch rises.
Abnormal (PTT dysfunction): Hindfoot remains in valgus or everts further. Patient may be unable to perform heel rise (complete PTT rupture/dysfunction).
Gold standard for PTT function. Inability to perform = Stage II or greater.
Technique: With patient seated (unloaded), examiner grasps heel and attempts to correct hindfoot valgus into neutral or varus.
Flexible (Stage II): Hindfoot corrects passively to neutral. Indicates soft tissue deformity (PTT, spring ligament) without fixed bony/arthritic changes.
Rigid (Stage III): Hindfoot does NOT correct. Indicates subtalar or talonavicular arthritis, fixed bony deformity. Changes treatment to arthrodesis.
Technique: With patient seated, elevate first ray (plantarflex first metatarsal-cuneiform joint) and observe hindfoot alignment.
Negative (Stage IIA): Hindfoot valgus CORRECTS when first ray is elevated. Forefoot is flexible.
Positive (Stage IIB): Hindfoot valgus PERSISTS despite first ray elevation. Indicates fixed forefoot varus driving hindfoot valgus (compensatory). MUST address forefoot with medial cuneiform plantar flexion osteotomy.
Technique: With knee extended, dorsiflex ankle. Then flex knee to 90 degrees and dorsiflex ankle.
Positive: Dorsiflexion improves with knee flexed (isolated gastrocnemius contracture). May require gastrocnemius recession in addition to hindfoot reconstruction.
Significance: Gastrocnemius contracture exacerbates hindfoot valgus (equinus drives valgus). Affects surgical planning.
Approximately 20% of AAFD patients have bilateral involvement (often asymmetric). Always examine BOTH feet and compare. The "normal" side may be early-stage asymptomatic AAFD. Failure to identify bilateral disease leads to:
- Incorrect comparison (thinking abnormal is "normal for patient")
- Missed opportunity for early intervention on asymptomatic side
- Patient dissatisfaction when contralateral side progresses post-operatively
Differential Diagnosis of the Adult Flatfoot
- Distinguishing features
- Acquired, usually unilateral and progressive; flexible early then rigid; positive too-many-toes and failed single heel rise
- Key test / investigation
- Weight-bearing radiographs + MRI (PTT and spring ligament); flexibility on examination
- Distinguishing features
- Younger or rigid flatfoot, painful peroneal spasm, restricted subtalar motion from the outset
- Key test / investigation
- CT (bony bar) or MRI (fibrous/cartilaginous coalition); often bilateral
- Distinguishing features
- Neuropathy (diabetes), warm swollen foot, rocker-bottom collapse, often painless out of proportion to deformity
- Key test / investigation
- Loss of protective sensation; radiographs/MRI of midfoot; check inflammatory and metabolic status
- Distinguishing features
- Polyarticular symptoms, morning stiffness, synovitis, often bilateral and symmetrical
- Key test / investigation
- Inflammatory markers, autoantibodies (RF, anti-CCP), erosive changes on radiographs
- Distinguishing features
- History of injury, deformity localised to midfoot, tarsometatarsal malalignment
- Key test / investigation
- Weight-bearing radiographs (Lisfranc alignment), CT for articular detail
- Distinguishing features
- Lifelong, flexible, painless, bilateral and symmetrical; normal single heel rise
- Key test / investigation
- Clinical - normal PTT function and no progression; no imaging needed if asymptomatic
- Distinguishing features
- Talar head sag with relatively preserved PTT strength; isolated medial sag
- Key test / investigation
- MRI shows spring ligament tear; PTT may be intact - changes reconstruction emphasis

Investigations
Imaging Protocol for AAFD
AP foot: Talonavicular uncovering (lateral subluxation of navicular on talus), forefoot abduction, talo-first metatarsal angle.
Lateral foot: Calcaneal pitch angle (normal 20-30 degrees, decreased in AAFD), talo-first metatarsal angle (normal 0-5 degrees, increased Meary angle in AAFD indicates arch collapse), talus plantar flexion/sagging.
AP ankle (if Stage III/IV suspected): Assess for ankle valgus tilt, tibiotalar arthritis, lateral mortise widening (deltoid insufficiency).
Hindfoot alignment view (Saltzman view): Quantifies hindfoot valgus. Taken with patient standing, X-ray beam angled 20 degrees cephalad from behind ankle. Measures weight-bearing axis of hindfoot.
Critical: Must be weight-bearing to assess deformity magnitude. Non-weight-bearing films underestimate deformity.
Indications: Uncertain diagnosis, assessing PTT integrity (surgical planning), evaluating spring ligament status, ruling out occult fractures or other pathology.
Findings:
- PTT: Increased T2 signal (tenosynovitis), thinning, discontinuity (tear), fluid in sheath
- Spring ligament: Attenuation, tear, increased signal
- Bone marrow edema: Talar head (overload), navicular stress
- Subtalar/talonavicular arthritis: Cartilage loss, subchondral changes
Bluman MRI Classification: Grade 1 (intact tendon, peritendinitis), Grade 2 (partial tear less than 50%), Grade 3 (greater than 50% or complete rupture).
Indications: Assessing subtalar or talonavicular arthritis (planning arthrodesis), evaluating tarsal coalition (rigid flatfoot differential), planning complex osteotomies.
Advantages: Better bony detail than MRI. Can perform 3D reconstructions for preoperative planning (especially useful for lateral column lengthening or complex deformity correction).
Limited role: Dynamic assessment of PTT (real-time movement), identifying complete rupture vs partial tear. Operator-dependent and not routinely used for staging in most centres, though availability and reliance vary by region and local expertise.
Key Radiographic Measurements
- Normal Value
- 20-30 degrees
- AAFD Finding
- Less than 18 degrees (arch collapse)
- Clinical Significance
- Decreased angle indicates loss of arch height
- Normal Value
- 0-5 degrees
- AAFD Finding
- Greater than 10 degrees (arch sag)
- Clinical Significance
- Measures alignment of talus and first metatarsal - should be collinear
- Normal Value
- Less than 7 degrees
- AAFD Finding
- Greater than 20 degrees (uncovering)
- Clinical Significance
- Lateral subluxation of navicular off talar head - forefoot abduction
- Normal Value
- 0 ± 3mm from midline
- AAFD Finding
- Greater than 5mm lateral deviation (valgus)
- Clinical Significance
- Quantifies hindfoot valgus deformity for surgical planning


Management Algorithm

Conservative Management Indications
Appropriate for:
- Stage I (tenosynovitis without deformity)
- Mild Stage IIA with minimal symptoms
- Patients unfit for surgery (medical comorbidities)
- Patient preference (informed consent)
Conservative Treatment Protocol
Goals: Reduce inflammation, offload PTT, control pain
- Immobilization: CAM walker boot or short leg cast, non-weight-bearing or protected weight-bearing
- NSAIDs: Oral anti-inflammatories (if no contraindications)
- Ice: 15-20 minutes TDS over medial ankle
- Activity modification: Avoid prolonged standing, stairs, uneven ground
- Rest: PTT needs unloading to allow healing/reduction of inflammation
Goals: Restore function, prevent recurrence, transition to orthotic
- Progressive weight-bearing: Gradual transition from boot to supportive footwear
- Physiotherapy: PTT strengthening (resisted inversion), intrinsic muscle strengthening, proprioception
- Orthotic prescription: UCBL (University of California Berkeley Laboratory) insert or AFO (ankle-foot orthosis)
- Footwear advice: Firm heel counter, medial arch support, avoid flat flexible shoes
Goals: Prevent progression, maintain function, surveillance
- Orthotic compliance: Daily use in all footwear
- Weight management: Reduce mechanical load on PTT
- Activity modification: Avoid high-impact activities, use walking aids if needed
- Surveillance: 6-12 monthly review to assess for progression to Stage II
- Progression criteria: Increasing deformity, too many toes sign, failed single heel rise = surgical referral
Indications for surgery in Stage I patients:
- Persistent pain despite 6 months of appropriate conservative treatment
- Progressive deformity (transition to Stage II)
- Failed single heel rise test (PTT incompetence)
- MRI showing Grade 3 PTT tear (complete rupture) - these will not heal
Important: Not all Stage I patients progress. Approximately 50% respond to conservative treatment with orthotics. However, those with Grade 3 MRI tears should be offered early surgical intervention (FDL transfer before deformity develops).
Surgical Technique: Stage II Reconstruction
Pre-operative Planning
- Undercorrection: 20-30% recurrence risk, may need revision
- Overcorrection: Hindfoot varus (painful rigid foot), lateral column overload
- Nerve injury: Sural nerve (lateral approaches), saphenous nerve, tibial nerve branches
- Infection: Superficial 5-10%, deep 2-5% (higher if diabetic)
- Nonunion: Calcaneal osteotomy (5%), Evans osteotomy if bone graft used (10%)
- Donor site morbidity: FDL harvest (toe clawing rare, flexion weakness), iliac crest if used (pain)
- Prolonged recovery: 3 months non-weight-bearing, 6-12 months full recovery
- Adjacent joint arthritis: Ankle, subtalar, talonavicular (long-term risk)
- Implants: Cannulated screws (6.5mm or 7.0mm for calcaneus), interference screw or suture anchor (FDL), Evans plate if LCL planned
- Power tools: Oscillating saw (osteotomies), drill, reamer
- Imaging: C-arm with ankle/foot capabilities, ensure can obtain hindfoot alignment view
- Bone graft: Allograft tricortical iliac crest or femoral head (Evans), consider autograft harvest set if preferred
- Tendon instruments: Nerve hooks, right-angle clamps, drill for navicular tunnel
- Retractors: Self-retaining (Weitlaner), Army-Navy, Hohmann
Patient Positioning and Setup
Setup Checklist
Supine on radiolucent table (Jackson table or standard with radiolucent foot extension).
- Head: Neutral, adequate airway access
- Upper body: Arms on arm boards or across chest (depends on anaesthetic preference)
- Pelvis: Centered on table, neutral alignment
- Operative leg: Hip neutral rotation (or slight external rotation), knee extended or slight flexion (bump under knee for relaxation)
- Non-operative leg: Abducted and supported on leg holder or pillow (out of C-arm path)
- Bony prominences: Sacrum, heels (non-operative), elbows
- Nerves at risk: Ulnar nerve (elbow padding), peroneal nerve at fibular head (if leg externally rotated or in leg holder)
- Pressure areas: Ensure heel of operative foot OFF table (ankle hanging free or on bolster)
Thigh tourniquet (standard):
- Apply over cast padding to proximal thigh
- Exsanguinate with Esmarch or elevation
- Pressure: 250-300 mmHg (adjust for patient size/BP)
- Expected time: 90-120 minutes (multiple osteotomies + transfer)
Advantages: Bloodless field aids dissection, tendon identification, osteotomy precision Disadvantages: Tourniquet pain (limited by anaesthetic), time limit (deflate and re-exsanguinate if exceeds 2 hours)
- Prep: Circumferential ankle and foot to mid-calf. Include proximal calf if anticipating gastrocnemius recession.
- Draping: Stockinette over foot, U-drape or split sheet isolating foot and ankle
- Landmarks exposed: Medial malleolus to toes (medial approach for FDL, medial cuneiform), lateral calcaneus (for MCO through lateral or oblique approach, Evans if needed)
- C-arm positioning: Ensure adequate AP, lateral, oblique views of foot AND hindfoot alignment view possible without repositioning patient. Test BEFORE prep/drape.
The Saltzman hindfoot alignment view (20-degree cephalad beam from posterior) is critical for assessing intraoperative correction. Position the C-arm BEFORE starting - you need to confirm adequate valgus correction post-osteotomy. If you cannot obtain this view intraoperatively, you are operating "blind" on alignment.
COLTSurgical Reconstruction Components (Stage II)
Hook:Fixing Stage II AAFD is like training a young COLT - need all components working together for stable gait!
Complications
- Incidence
- 20-30% (varies by study and definition)
- Risk Factors
- Stage IIB missed (no forefoot correction), inadequate MCO translation, FDL transfer insufficient tension, patient non-compliance (early weight-bearing)
- Management
- Mild: Orthotics, activity modification. Moderate-Severe: Revision surgery (repeat MCO, revision transfer, consider arthrodesis if progression to Stage III)
- Incidence
- 5-10%
- Risk Factors
- Excessive MCO translation (greater than 12mm), over-lengthening Evans (greater than 12mm), FDL transfer overtightened
- Management
- Painful rigid foot, lateral column overload. Mild: Orthotics (lateral heel wedge), shoe modifications. Severe: Revision osteotomy (lateralizing calcaneus) or takedown and redo
- Incidence
- 5-10% (higher if smokers, diabetics)
- Risk Factors
- Smoking, diabetes, inadequate fixation, early weight-bearing, infection
- Management
- If asymptomatic: Observe. If painful: Revision fixation (ORIF with bone graft, consider bone stimulator)
- Incidence
- 10-15% (if bone graft used)
- Risk Factors
- Allograft (higher than autograft), smoking, inadequate fixation
- Management
- Often asymptomatic (graft provides structural support even without union). If painful: Revision with autograft and rigid fixation
- Incidence
- 5-15% (numbness, neuroma)
- Risk Factors
- Lateral approaches (MCO, Evans), inadequate identification and protection
- Management
- Numbness lateral foot/ankle (variable distribution). Usually improves over 6-12 months. Painful neuroma: Nerve blocks, desensitization, rarely neuroma excision
- Incidence
- 5-10%
- Risk Factors
- Diabetes, obesity, smoking, prolonged surgery, inadequate wound care
- Management
- Antibiotics (oral if cellulitis), wound care, dressing changes. Rarely needs debridement.
- Incidence
- 2-5% (higher in diabetics)
- Risk Factors
- Diabetes, immunosuppression, contamination, hardware
- Management
- Debridement, hardware removal (once union achieved), IV antibiotics (6 weeks minimum), consider VAC therapy. May require staged reconstruction or amputation if severe.
- Incidence
- Less than 5% (rare)
- Risk Factors
- Tight closure of FDL harvest site, loss of FDL to lesser toes
- Management
- Usually minimal functional impact (FHL compensates). Clawing: Toe taping, silicone sleeves. Rarely needs surgical correction (IP fusion if fixed deformity)
- Incidence
- 10-20% radiographic, 5-10% symptomatic
- Risk Factors
- Over-distraction (greater than 12mm), intra-articular osteotomy, graft malposition
- Management
- Mild: NSAIDs, activity modification, orthotics. Moderate-Severe: Calcaneocuboid fusion (salvage)
- Incidence
- 10-15% long-term (10+ years)
- Risk Factors
- Undercorrection, high-demand activities, obesity, age at surgery
- Management
- Surveillance with serial radiographs. Symptomatic arthritis: Arthrodesis (subtalar, triple, or TTC depending on location)
Patients with diabetes and AAFD have:
- Higher infection risk (2-3× baseline)
- Impaired wound healing (neuropathy, vascular disease)
- Higher nonunion rates (metabolic factors)
- Risk of Charcot neuroarthropathy progression (especially if neuropathy present)
Pre-operative optimization: HbA1c less than 7.5% (ideally less than 7%), vascular assessment (ABI, consider angiography if abnormal), neuropathy assessment. Consider staged procedures (e.g., FDL transfer first, then osteotomies later) to reduce single-operation complexity. Extended non-weight-bearing (12-16 weeks vs 8-12 weeks) to ensure healing.
UNIONComplications to Discuss in Viva
Hook:Aim for UNION of all corrected joints - but watch for complications that prevent perfect healing!
Postoperative Care and Rehabilitation
Rehabilitation Timeline After Stage II Reconstruction
Goals: Protect osteotomies and FDL transfer, prevent swelling and complications.
- Immobilization: Below-knee backslab (posterior slab), transition to cast at 2 weeks after suture removal
- Weight-bearing: Strict non-weight-bearing (crutches or walker, NO weight on operative foot)
- Elevation: Leg elevated above heart level as much as possible (reduces swelling)
- Ice: 20 minutes every 2-3 hours over cast (NOT directly on skin)
- DVT prophylaxis: Aspirin 100mg daily OR enoxaparin (if high risk - obesity, prior DVT, prolonged immobility). Mechanical prophylaxis (foot pumps, TED stockings) if possible.
- Pain management: Multimodal (paracetamol + NSAID + opioid PRN), wean opioids by week 2
- Wound check: POD 10-14 for suture/staple removal, inspect for infection
Goals: Continue protection, begin gentle ankle motion (out of cast), monitor for healing.
- Immobilization: Below-knee circumferential cast or CAM walker boot (removable)
- Weight-bearing: Continue non-weight-bearing (osteotomies not yet healed)
- Physiotherapy: Gentle ankle range of motion exercises OUT of cast/boot (plantarflexion, dorsiflexion only - NO inversion/eversion). Knee and hip exercises to prevent stiffness.
- Radiographs: 6-week X-rays (AP, lateral foot, hindfoot alignment) to assess osteotomy healing, hardware position
Goals: Transition to weight-bearing, advance range of motion, restore gait.
- Immobilization: CAM walker boot (allows progressive weight-bearing)
- Weight-bearing: Progressive weight-bearing if radiographs show healing:
- Weeks 6-8: Touch weight-bearing to partial (25-50% body weight), progress as tolerated
- Weeks 8-10: Partial to full weight-bearing in boot
- Weeks 10-12: Full weight-bearing in boot, transition to supportive shoe + orthotic
- Physiotherapy: Active ankle ROM (all planes), proprioception exercises (balance board), gait re-education (normal heel-toe pattern), FDL strengthening (resisted inversion)
- Precautions: NO high-impact activities (running, jumping), avoid uneven ground
Goals: Restore full function, strengthen supporting muscles, prevent recurrence.
- Footwear: Transition to supportive athletic or walking shoes with custom orthotic (UCBL or similar to support arch)
- Weight-bearing: Full unrestricted weight-bearing (out of boot)
- Physiotherapy: Progressive resistance exercises (ankle strengthening - inversion, eversion, plantarflexion, dorsiflexion), calf strengthening (heel raises), proprioception, sport-specific training if applicable
- Activity progression: Low-impact activities (walking, cycling, swimming) → gradual return to higher-impact if appropriate
- Radiographs: 3-month and 6-month follow-up X-rays to confirm union, assess alignment
Goals: Maintain correction, monitor for recurrence, prevent adjacent joint arthritis.
- Orthotic compliance: Daily use in ALL footwear (critical to prevent recurrence)
- Footwear: Firm heel counter, medial arch support, avoid flat flexible shoes
- Activity modification: Avoid prolonged barefoot walking, high-impact repetitive loading
- Surveillance: Annual radiographs for first 2-3 years to monitor for recurrence, then as needed if symptomatic
- Weight management: Maintain healthy BMI to reduce mechanical load
- Patient education: Recognize signs of recurrence (increasing arch sag, hindfoot valgus, pain) and report early
AAFD reconstruction involves multiple osteotomies (calcaneus ± Evans ± cuneiform) and soft tissue transfer (FDL). Early weight-bearing risks:
- Osteotomy nonunion or malunion (especially calcaneus with metal translation)
- Hardware failure (screw pullout, plate breakage)
- FDL transfer failure (anchor pullout, tendon elongation with loss of tension)
- Recurrent deformity (hindfoot settles back into valgus)
8-12 weeks non-weight-bearing is standard. Some surgeons extend to 12 weeks for complex reconstructions (Stage IIB with multiple osteotomies) or high-risk patients (diabetics, osteoporotic).
Outcomes and Prognosis
Functional Outcomes by Stage
- Pain Relief
- 50-60% achieve adequate pain relief at 1 year
- Function / Satisfaction
- Moderate functional improvement, ongoing orthotic dependence, compliance variable
- Durability
- 30-40% progress to Stage II within 5 years if Grade 3 PTT tear on MRI
- Pain Relief
- 70-80% good to excellent pain relief
- Function / Satisfaction
- 75-85% patient satisfaction, improved gait, ability to return to low-impact activities
- Durability
- 20-30% recurrence (residual symptoms or progressive deformity) at 5-10 years. Undercorrection most common cause.
- Pain Relief
- 80-85% significant pain relief (if solid fusion achieved)
- Function / Satisfaction
- 65-75% satisfaction (lower than Stage II due to loss of motion). Stable plantigrade foot. Functional limitations on uneven ground.
- Durability
- 10-15% nonunion. 20-30% develop adjacent joint arthritis (ankle, naviculocuneiform) by 10 years post-fusion.
- Pain Relief
- 70-75% pain relief (if solid fusion achieved)
- Function / Satisfaction
- 50-60% satisfaction (significant functional limitation - complete loss of ankle and hindfoot motion). Salvage procedure.
- Durability
- High nonunion risk (10-15%). Patients need lifelong rocker-bottom shoes, significant gait alteration. Quality of life impact substantial.
Factors associated with WORSE outcomes after Stage II reconstruction:
- Obesity (BMI greater than 35): Increased mechanical load, higher recurrence
- Diabetes: Impaired healing, higher infection, neuropathy (balance issues)
- Smoking: Nonunion risk, wound complications
- Advanced age (greater than 70): Lower functional reserve, slower rehabilitation
- Inflammatory arthropathy (RA, seronegative): Ongoing systemic disease progression
- Stage IIB without forefoot correction: Cotton test missed, inevitable recurrence
- Delayed presentation (longstanding severe deformity): Fixed changes, worse baseline alignment
Counsel patients pre-operatively about realistic expectations based on risk factors. Consider arthrodesis earlier (Stage II → Stage III procedure) in high-risk patients.
Guidelines, Registries & Global Practice
Adult flatfoot from posterior tibial tendon failure is a worldwide condition with no dedicated joint registry and no high-level (RCT) guideline: management is driven by expert consensus (the 2020 PCFD nomenclature) and retrospective case series, so the world standard is a deformity-specific reconstruction tailored to flexibility and the planes of collapse.
Global Epidemiology
- Figure
- Most present in the 5th-6th decade
- Source / note
- Reflected across surgical series (e.g. mean age 54 in PCFD reliability cohort, PMID 34852647)
- Figure
- Female predominance (≈ 62% female in PCFD cohort)
- Source / note
- PMID 34852647 (single-centre, surgical population)
- Figure
- Multiplanar collapse is the norm; isolated single-plane deformity is uncommon
- Source / note
- Class A (hindfoot valgus) 89.5%, class C (forefoot varus / medial column instability) 86.2% (PMID 34852647)
- Figure
- Reviews estimate several million affected adults and rising prevalence with ageing and obesity
- Source / note
- Narrative estimate (PMID 39036227); precise population prevalence is not robustly established
Nomenclature and Classification - Side by Side
- Year / status
- 1989, still widely examined
- What it adds
- Original 4-stage tendon-centred staging (I tenosynovitis to IV ankle valgus)
- Evidence level
- Level V (historical, unvalidated)
- Year / status
- 1997
- What it adds
- Splits Stage II into IIA / IIB (forefoot-driven), adds Stage IV subtypes
- Evidence level
- Level V expert opinion
- Year / status
- 2007 (PMID 17561198)
- What it adds
- Subcategories for forefoot supination, abduction, medial column and ankle; MRI grading
- Evidence level
- Level V expert opinion
- Year / status
- 2020 (PMID 32856474) - current international standard
- What it adds
- Renames to Progressive Collapsing Foot Deformity; class + components A-E, flexible/rigid
- Evidence level
- Level V consensus; reliability tested (PMID 34852647)
Guidelines and Peri-operative Standards (Region-Neutral)
- No AAOS, NICE or EFORT disease-specific guideline exists for adult flatfoot reconstruction - this is consensus-driven (PCFD 2020), unlike arthroplasty topics
- Staged escalation is universal: trial non-operative care (orthoses/bracing, activity and weight management) before reconstruction in flexible disease
- Flexible vs rigid divide drives surgery everywhere: osteotomy plus tendon transfer for flexible deformity, arthrodesis for rigid/arthritic deformity
- Surgical safety: WHO Surgical Safety Checklist is the global standard; prophylactic cephalosporin within 60 min of incision and peri-operative normoglycaemia are near-universal SSI-prevention measures
- Imaging: weight-bearing CT is increasingly used in North America and Europe to quantify peritalar subluxation (PMID 33358266) but remains unavailable in many centres, where weight-bearing radiographs ± MRI remain standard
- VTE prophylaxis: foot/ankle surgery with prolonged immobilisation has no global consensus - chemical prophylaxis (LMWH or aspirin) is used more readily in some systems than others; decisions are individualised on risk
- Lateral column lengthening vs medial column procedures for forefoot abduction varies by surgeon and school rather than by guideline
- Resource setting: in limited-resource settings, bracing and arthrodesis predominate over multi-osteotomy reconstruction owing to implant, graft and rehabilitation costs
Unlike hip and knee arthroplasty, adult flatfoot reconstruction is not captured by any national joint registry. The major registries - NJR (England & Wales), AJRR (USA), AOANJRR (Australia), SHAR (Sweden), the Norwegian and New Zealand registries - track arthroplasty implants, not foot/ankle deformity surgery. Any "flatfoot registry revision rate" should be treated with suspicion. Revision and complication figures in this topic come from case series and reviews, which is the correct level of evidence to quote in a viva.
Key documentation requirements:
-
Informed consent must include:
- Realistic success rate (75-85% good outcomes, but 20-30% recurrence risk)
- Prolonged recovery (3-6 months to full function, 12+ months to final outcome)
- Complications: Undercorrection (20-30%), overcorrection (5-10%), nerve injury (sural nerve 5-15%), infection (superficial 5-10%, deep 2-5%), nonunion (5-15%), progression to arthrodesis (10-15% long-term)
- Alternative treatments: Conservative (orthotic), arthrodesis (if rigid deformity)
- Expected functional outcomes: Improved pain and gait, but NOT return to high-impact sports
-
Common litigation issues:
- Failure to diagnose Stage IIB (missing Cotton test) leading to recurrence - document Cotton test result in notes
- Nerve injury (sural nerve, tibial nerve branches) without pre-operative counseling - must consent for numbness risk
- Infection in diabetic patients without adequate optimization - document HbA1c, pre-operative counseling, glucose control peri-op
- Undercorrection perceived as surgical failure - set realistic expectations (not all patients achieve perfect correction), document degree of deformity pre-op and plan for multi-stage if severe
-
Documentation best practices:
- Pre-operative: Clinical photos (too many toes sign, arch height), weight-bearing radiographs with measurements (angles documented), Cotton test result, Silfverskiöld test, single heel rise test result
- Intraoperative: Fluoroscopy images saved (hindfoot alignment view showing correction), degree of calcaneal translation measured and documented, FDL transfer tension confirmed
- Post-operative: Serial radiographs to monitor healing and alignment, document orthotic prescription and compliance counseling
MCQ Practice Points
Q: What is the PRIMARY insertion of the posterior tibial tendon and why is it critical for arch support? A: The primary insertion is the navicular tuberosity. This is critical because the navicular is the keystone of the medial longitudinal arch. The PTT's pull on the navicular (via its insertion) lifts the arch during stance phase. When the PTT fails, the navicular sags inferiorly and the arch collapses. The PTT has secondary insertions to all 3 cuneiforms and metatarsal bases 2-4, providing distributed midfoot stability, but the navicular insertion is the main arch supporter.
Q: What distinguishes Stage IIA from Stage IIB in the Johnson and Strom classification, and why does this matter for surgical planning? A: Both are flexible deformities, but the key difference is the forefoot. Stage IIA has a flexible forefoot - the Cotton test is negative (hindfoot valgus corrects when first ray is elevated). Stage IIB has a fixed forefoot varus - the Cotton test is positive (hindfoot valgus persists despite first ray elevation because the forefoot varus is DRIVING the hindfoot valgus compensatorily). This matters because Stage IIB requires additional surgery: medial cuneiform plantar flexion osteotomy (Cotton osteotomy) to correct the forefoot varus. Without this, the reconstruction WILL fail because the uncorrected forefoot drives recurrent hindfoot valgus.
Q: Describe the single heel rise test and what a positive (abnormal) test indicates. A: The patient stands on one leg (the affected side) and attempts to rise up on tiptoes (heel rise off ground). The examiner observes the hindfoot from behind. Normal: As the patient rises, the hindfoot inverts (shifts from valgus to varus) due to tibialis posterior contraction. Abnormal (positive test): The hindfoot remains in valgus or everts further, OR the patient is completely unable to perform a heel rise. This indicates posterior tibial tendon dysfunction - the PTT is incompetent and cannot generate the inversion moment. A positive test signifies Stage II or greater AAFD (Stage I patients can usually still perform heel rise, though may have pain).
Q: Why is FDL preferred over FHL for tendon transfer in AAFD reconstruction? A: FDL advantages: (1) Better excursion - longer muscle belly and arc of contraction provides greater active inversion force. (2) Lower donor morbidity - FHL is the stronger hallux flexor; transferring FHL causes more noticeable hallux weakness and potential IP joint stiffness. FDL loss to lesser toes is rarely symptomatic (FHL compensates). (3) Easier harvest - medial approach (same as navicular exposure) vs deep posterior approach for FHL. (4) Equivalent strength - biomechanical studies show FDL provides sufficient force for arch support. Both FDL and FHL have similar clinical outcomes in terms of pain relief and deformity correction, but FDL has better risk-benefit profile.
Q: What is the most common cause of failure after Stage II AAFD reconstruction, and how can it be prevented? A: The most common cause is undercorrection / recurrent deformity (occurs in 20-30% of patients). Causes include: (1) Missing Stage IIB - failing to perform medial cuneiform osteotomy when forefoot varus is present (Cotton test positive). (2) Inadequate calcaneal translation - not medializing enough (should aim for 8-12mm, confirmed on hindfoot alignment view). (3) Missing lateral column lengthening - when significant forefoot abduction present (talonavicular uncovering greater than 30 degrees, too many toes sign) but LCL not performed. (4) Patient factors - obesity (high BMI increases mechanical load), diabetes, non-compliance with orthotic use post-op. Prevention: Meticulous pre-operative planning (Cotton test, assess forefoot abduction, measure angles), adequate surgical correction (confirm with intraoperative fluoroscopy), address patient risk factors (weight loss, diabetes control), enforce orthotic compliance post-operatively.
Q: What is the evidence base for the FDL transfer plus medialising calcaneal osteotomy in flexible Stage II disease? A: The most-cited series is Myerson, Badekas and Schon (Foot Ankle Int 2004), who reviewed 129 patients with Stage II posterior tibial tendon deficiency treated with medialising calcaneal osteotomy plus FDL transfer to the navicular at a mean 5.2 years. They reported 97% pain relief, a mean postoperative AOFAS hindfoot score of 79, significant radiographic correction in all parameters, and high satisfaction with few complications. This underpins the modern soft-tissue-plus-osteotomy approach. Limitations to quote: it is a retrospective, single-surgeon series with no control arm, AOFAS is not a validated score, and forefoot-driven (Stage IIB) feet were excluded. Note there is no joint registry capturing flatfoot reconstruction (registries such as AOANJRR, NJR and AJRR track arthroplasty, not foot/ankle deformity surgery), so the evidence base is case series and reviews rather than registry data.
Exam Viva Scenarios
Practise clinical reasoning and management decisions out loud
“A 52-year-old female presents with progressive medial foot pain and flattening of her arch over 2 years. She has type 2 diabetes (HbA1c 7.2%) and BMI 32. On examination, you observe a too many toes sign, and she is unable to perform a single heel rise on the affected side. Her hindfoot corrects to neutral when you passively stress it into varus (seated examination). You perform a Cotton test: when you elevate her first ray (plantarflex the first metatarsal), her hindfoot valgus DOES correct. Weight-bearing radiographs show hindfoot valgus with talonavicular uncovering of 25 degrees and decreased calcaneal pitch. What is your assessment and management plan?”
“Walk me through your technique for performing a medializing calcaneal osteotomy as part of a Stage II AAFD reconstruction. Specifically, describe your approach, osteotomy location and orientation, how much translation you aim for, and your fixation method. What nerves are at risk and how do you protect them?”
“A 58-year-old patient returns 18 months after Stage IIA reconstruction (MCO + FDL transfer, no lateral column lengthening) with recurrent medial foot pain and visible return of arch collapse. Radiographs show hindfoot valgus has recurred (calcaneal osteotomy healed in translated position) and talonavicular uncovering has worsened from 20 degrees pre-op to 15 degrees immediately post-op to now 30 degrees. She is obese (BMI 36) and non-compliant with orthotic use (admits she rarely wears it). How do you approach this patient?”
Key Anatomy
- PTT = primary dynamic arch stabilizer (over 50% of support)
- Navicular tuberosity = primary PTT insertion (arch keystone)
- Hypovascular zone 2-4cm proximal to insertion = site of rupture
- Spring ligament = static stabilizer (fails secondary to PTT loss)
- 3-plane deformity: hindfoot valgus (coronal) + arch collapse (sagittal) + forefoot abduction (axial)
Johnson and Strom Classification
- Stage I = PTT tenosynovitis, no deformity, conservative treatment
- Stage IIA = Flexible hindfoot, negative Cotton test, MCO + FDL ± LCL
- Stage IIB = Flexible hindfoot + fixed forefoot varus (positive Cotton), add medial cuneiform osteotomy
- Stage III = Rigid deformity, subtalar/triple arthrodesis
- Stage IV = Ankle valgus tilt (deltoid insufficiency), TTC fusion
Clinical Assessment Algorithm
- Standing: Too many toes sign (forefoot abduction + hindfoot valgus)
- Functional: Single heel rise test (PTT function - negative = incompetent)
- Seated: Hindfoot flexibility test (Stage II flexible, Stage III rigid)
- Cotton test: Elevate first ray - if valgus persists = Stage IIB (forefoot-driven)
- Silfverskiöld test: Gastrocnemius contracture (add recession if positive)
Surgical Decision Points
- Flexible (Stage II) = osteotomy + tendon transfer, Rigid (Stage III) = arthrodesis
- FDL over FHL: Better excursion, lower donor morbidity, easier harvest
- LCL indications: Forefoot abduction greater than 30 degrees (talonavicular uncovering)
- MCO translation: 8-12mm medially (confirm with hindfoot alignment view)
- Post-op: 8-12 weeks strict non-weight-bearing (multiple osteotomies to heal)
Complications and Prevention
- Undercorrection 20-30% = most common, prevent with adequate MCO + LCL if needed + Cotton test
- Overcorrection 5-10% = hindfoot varus (painful), avoid greater than 12mm translation/LCL
- Sural nerve injury 5-15% = identify and protect during lateral approach
- Nonunion 5-15% = smoking cessation mandatory, avoid NSAIDs during healing
- Recurrence risk factors: obesity, diabetes, orthotic non-compliance, smoking
Evidence Base and Key Trials
FDL Transfer with Medialising Calcaneal Osteotomy for Stage II PTT Deficiency
- Retrospective series of 129 patients with Stage II posterior tibial tendon deficiency (flexible flatfoot without fixed forefoot supination), operated 1990-1997, mean follow-up 5.2 years
- Procedure: medial translational calcaneal osteotomy PLUS flexor digitorum longus transfer to the navicular
- Mean postoperative AOFAS hindfoot score 79 (range 54-93); significant radiographic correction in all four parameters measured
- 97% (125/129) experienced pain relief, 94% improved function, 84% wore shoes comfortably without modification or orthotic support
- Patient satisfaction: entirely satisfied 118, partially satisfied 7, dissatisfied 4; seven significant complications in six patients
Outcomes of Reconstruction of the Flexible Adult-Acquired Flatfoot Deformity
- Narrative review synthesising contemporary clinical and radiographic outcomes for the major procedures used in flexible AAFD reconstruction
- Confirms that combined procedures - FDL transfer, medialising calcaneal osteotomy (MCO), gastrocnemius recession, lateral column lengthening (LCL), Cotton osteotomy or first tarsometatarsal fusion, and spring ligament reconstruction - are frequently used together
- Reports consistent significant improvement in patient-reported outcomes after operative treatment of flexible deformity
- Emphasises tailoring the combination of osteotomies and soft-tissue work to the individual deformity rather than a single fixed algorithm
- Reviews the relative correction power and complications of MCO, LCL and Cotton osteotomies
PCFD Consensus: Classification and Nomenclature for Progressive Collapsing Foot Deformity
- Expert consensus group renamed adult acquired flatfoot deformity (AAFD) / posterior tibial tendon dysfunction (PTTD) to Progressive Collapsing Foot Deformity (PCFD)
- Frames the condition as a complex 3-dimensional deformity with varying hindfoot valgus, forefoot abduction and midfoot varus, rather than a single tendon disease
- Replaces the older numeric staging (Johnson and Strom 1989; Myerson 1997) with a class (severity) and flexible/rigid descriptor system reporting deformity components A-E
- Unanimous consensus (9/9) that prior classification systems are incomplete or outdated and none had been validated
- Strong consensus (8/9) to adopt the new system to stage deformity and guide treatment
Reliability of the New PCFD Classification System
- Retrospective study of 92 feet (84 patients) from a prospective registry assessing intra- and interobserver reliability of the PCFD classification
- Class A (hindfoot valgus) was the most frequent component (89.5%), followed by class C (forefoot abduction) in 86.2%
- Most patients had combined rather than isolated deformity; the commonest subclass was 1ABC (25.4%)
- Interobserver reliability was moderate (Fleiss kappa 0.561); intraobserver reliability was very good (Cohen kappa 0.851)
- Confirms that isolated single-component deformity is uncommon - most feet collapse in multiple planes simultaneously
Posterior Tibial Tendon Rupture: A Refined (Bluman-Myerson) Classification System
- Refines the Johnson and Strom 1989 staging to capture the wider spectrum of deformity seen with posterior tibial tendon rupture
- Adds subcategories accounting for ankle and hindfoot valgus, forefoot supination, forefoot abduction and medial column instability
- Provides stage-specific treatment recommendations, including for early disease and for ankle (deltoid) involvement
- Underpins the MRI-based grading of tendon integrity widely quoted in viva preparation
- Bridges the gap between the simple 4-stage system and the later PCFD consensus
Weight-bearing CT and MRI Correlation of Soft-Tissue Failure in Flexible PCFD
- Retrospective comparative study of 54 patients with flexible PCFD using weight-bearing CT (WBCT) markers of peritalar subluxation and MRI markers of soft-tissue failure
- Posterior tibial tendon degeneration on MRI correlated significantly with sinus tarsi impingement (p = 0.04)
- Spring ligament degeneration correlated with subtalar joint subluxation (p = 0.04)
- Talocalcaneal interosseous ligament involvement was the only finding significantly correlated with subfibular impingement (p = 0.02)
- Demonstrates that bony collapse (peritalar subluxation) and soft-tissue (PTT, spring and interosseous ligament) failure progress together