GENU VALGUM AND GENU VARUM
Angular Knee Deformities | Physiological vs Pathological | Blount Disease | Guided Growth
DEFORMITY TYPES
Critical Must-Knows
- Physiological evolution: Birth varus (10-15°) → neutral (18-24mo) → peak valgus (3-4yrs, 10-15°) → adult alignment (7yrs, 5-7° valgus)
- Red flags for pathology: Asymmetry, rapid progression, extreme angle, short stature, onset after walking established
- Blount disease: Tibia vara from disrupted medial proximal tibial physis. Infantile (1-3yrs) vs adolescent types
- Rickets: Systemic cause of angular deformity - nutritional or renal. Bilateral, symmetric, physeal widening
- Guided growth: Hemiepiphysiodesis with 8-plates to correct angular deformity in skeletally immature patients
Examiner's Pearls
- "Measure intercondylar distance (varus) or intermalleolar distance (valgus) clinically
- "Mechanical axis deviation on standing long leg X-ray is the gold standard
- "Drennan metaphyseal-diaphyseal angle greater than 16° suggests Blount disease
- "Always check calcium, phosphate, vitamin D, ALP in bilateral deformity
Clinical Imaging
Imaging Gallery


Critical Angular Deformity Exam Points
Physiological Pattern
Normal evolution: Babies born with 10-15° varus (bowlegs). Straightens by 18-24 months. Peaks at valgus 10-15° (knock-knees) by age 3-4. Gradually corrects to adult alignment (5-7° valgus) by age 7. Symmetric, follows expected pattern.
Pathological Red Flags
Suspect pathology if: Asymmetric deformity, rapid progression beyond expected pattern, extreme angles (varus greater than 15° after age 2, valgus greater than 15° after age 5), short stature, lateral thrust in gait, persisting/worsening after expected correction age.
Blount Disease
Tibia vara from medial proximal tibial physis disorder. Infantile (1-3yrs, often bilateral) vs Adolescent (greater than 10yrs, unilateral, obese). Drennan MDA greater than 16° diagnostic. Infantile may respond to bracing; adolescent requires surgery.
Guided Growth
Hemiepiphysiodesis with tension-band plating (8-plate). Applied to convex side of physis to retard growth and allow correction. Requires at least 2 years growth remaining. Correct approximately 1°/month. Remove hardware after correction achieved.
Quick Decision Guide - Genu Varum
| Feature | Physiological Varus | Blount Disease | Rickets |
|---|---|---|---|
| Age pattern | Less than 2 yrs, resolving | Infantile: 1-3yrs, progressive | Any age, often bilateral |
| Symmetry | Symmetric | Often bilateral but may be asymmetric | Symmetric |
| X-ray findings | Normal physes | Medial tibial beaking, MDA greater than 16° | Physeal widening, cupping, fraying |
| Other features | None | Lateral thrust, internal tibial torsion | Short stature, rachitic rosary, bowing |
| Treatment | Observation | Brace (early) or surgery | Vitamin D/phosphate, guided growth |
ALIGNALIGN - Approach to Angular Deformity
Memory Hook:ALIGN the approach - check age, look for thrust, investigate appropriately, and consider growth.
BLOUNTBLOUNT - Blount Disease Features
Memory Hook:BLOUNT affects the proximal tibia physis, causing Beaking on X-ray and Lateral thrust.
RICKETSRICKETS - Features of Rickets
Memory Hook:RICKETS is systemic - look for rosary, cupping, short stature, and treat the metabolic cause.
Overview and Epidemiology
Angular knee deformities are common presenting concerns in pediatric orthopaedics. Understanding the normal physiological development is essential to differentiate benign patterns from pathological conditions requiring intervention.
Definitions:
- Genu varum: "Bowlegs" - tibiofemoral angle in varus, knees apart when ankles together
- Genu valgum: "Knock-knees" - tibiofemoral angle in valgus, ankles apart when knees together
Physiological Evolution
Birth to adult pattern: Newborns have 10-15° varus (intrauterine positioning). This straightens by 18-24 months to neutral. Maximum valgus (10-15°) occurs at age 3-4 years. Gradual correction to adult alignment (5-7° valgus) by age 6-7 years.
Epidemiology:
- Physiological angular deformity is extremely common and usually normal
- Infantile Blount disease: more common in African descent, early walkers, obese
- Rickets: nutritional form rare in developed countries; renal osteodystrophy in chronic kidney disease
Pathophysiology and Mechanisms
Lower Limb Mechanical Axis
Limb Alignment Parameters
| Parameter | Definition | Normal Value |
|---|---|---|
| Mechanical axis | Center of femoral head to center of ankle | Passes through or just medial to knee center |
| Anatomical axis | Femoral and tibial diaphyseal axes | 5-7° valgus tibiofemoral angle |
| MPTA | Medial proximal tibial angle | 85-90° |
| LDFA | Lateral distal femoral angle | 85-90° |
Physeal Growth and Correction
Growth Plate Principles
- Proximal tibia: 55% of tibial growth
- Distal femur: 70% of femoral growth, 37% of leg length
- Asymmetric loading affects physeal growth
- Hueter-Volkmann law: compression retards, tension stimulates growth
Guided Growth Mechanics
- Hemiepiphysiodesis: Retard growth on one side
- 8-plate creates tension band effect
- Screw/staple methods also used
- Correct approximately 1°/month at distal femur
- Need at least 2 years growth remaining
Contribution of Each Physis
Distal femur contributes more to angular correction than proximal tibia due to faster growth rate. Consider site of maximum deformity (CORA - center of rotation of angulation) when planning surgery. Overcorrection is a risk if hardware not removed promptly.
Classification Systems
Etiology-Based Classification
| Category | Examples | Management Approach |
|---|---|---|
| Physiological | Normal developmental pattern | Observation, reassurance |
| Developmental (Blount) | Infantile tibia vara, adolescent tibia vara | Bracing (infantile), surgery |
| Metabolic | Rickets (nutritional, X-linked hypophosphatemic, renal) | Treat metabolic cause, then guided growth |
| Skeletal dysplasia | Achondroplasia, multiple epiphyseal dysplasia | Manage deformity, often complex |
| Post-traumatic | Physeal injury, malunion | Depends on growth remaining |
| Infection | Post-septic physeal damage | May need surgical correction |
Identifying the cause is essential for appropriate management.
Clinical Assessment
Systematic Examination
- Age of onset: When parents first noticed deformity
- Progression: Improving, stable, worsening
- Walking age: Early walkers at risk for Blount
- Family history: Skeletal dysplasia, rickets
- Diet/sun exposure: Vitamin D intake, risk for nutritional rickets
- Medical history: Renal disease, GI malabsorption
- Lateral thrust: Knee appears to "pop out" laterally in stance
- This is pathological and suggests significant varus
- In-toeing or out-toeing: May coexist with angular deformity
- Assess overall gait pattern and foot progression angle
- Intercondylar distance (ICD): Measure with ankles together (varus)
- Intermalleolar distance (IMD): Measure with knees together (valgus)
- Symmetry: Compare both sides
- Rotational profile: May coexist tibial internal torsion
- Height: Short stature suggests skeletal dysplasia or rickets
- Wrist exam: Physeal widening in rickets
- Rachitic rosary: Costochondral swelling
- Dysmorphic features: Skeletal dysplasia
Lateral Thrust = Pathological
A lateral thrust during gait is NEVER physiological. It indicates significant varus with medial collateral laxity or tibial vara. This finding warrants investigation and likely treatment.
Investigations
When to Image
X-ray if: Asymmetric deformity, extreme angles, not following physiological pattern, lateral thrust, short stature, or clinical concern. Request standing AP long leg X-ray for accurate mechanical axis assessment.
Investigation Modalities
| Investigation | When Used | What to Look For |
|---|---|---|
| Standing long leg X-ray | Angular deformity requiring assessment | Mechanical axis deviation, site of deformity |
| AP/Lateral knee X-rays | Initial assessment, Blount diagnosis | Physeal changes, metaphyseal beaking, MDA |
| Wrist X-ray | Suspected rickets | Physeal widening, cupping, fraying |
| Bloods: Ca, PO4, Vit D, ALP | Bilateral deformity, suspected metabolic cause | Low Ca/PO4/Vit D, high ALP in rickets |
| MRI | Physeal bar suspected, Blount assessment | Physeal status, bar extent |
Key Radiographic Measurements:
- Mechanical axis deviation (MAD): Distance from mechanical axis to knee center
- Metaphyseal-diaphyseal angle (MDA/Drennan): Greater than 16° suggests Blount disease
- Tibial metaphyseal angle: Assesses tibia vara severity
Management Algorithm
Conservative Management
Physiological Deformity:
- Most common presentation
- Follows expected pattern for age
- No lateral thrust, symmetric
Management:
- Reassurance and education
- Clinical follow-up every 6-12 months
- Serial clinical photos to document
- X-rays rarely needed if following typical pattern
- Expect correction by age 6-7 for valgus, earlier for varus
No bracing or orthotics required for true physiological deformity. They do not hasten correction.
Surgical Technique
Tension-Band Plating (8-Plate)
Standard guided growth technique for angular deformity.
Surgical Steps
- Standing long leg X-ray to assess mechanical axis
- Identify site(s) of deformity (CORA)
- Decide distal femur vs proximal tibia vs both
- Supine on radiolucent table
- Affected side accessible
- Image intensifier available
- Small (2-3cm) incision over physis
- On convex side of deformity
- For valgus: lateral distal femur
- For varus: medial proximal tibia
- Identify physis with fluoroscopy
- Extraperiosteal placement of 8-plate
- One screw in epiphysis, one in metaphysis
- Screws parallel, with plate on tension side
- Confirm position with imaging
- Close in layers
- No immobilization required
Technical Points:
- Screws must be parallel for tension-band effect
- Plate should sit flush on bone
- Do not violate physis with plate, only screws cross it
Post-operative: Weight-bear as tolerated; follow-up X-rays every 3 months.
Complications
Complications of Treatment
| Complication | Incidence | Prevention/Management |
|---|---|---|
| Overcorrection | Common if hardware not removed | Regular monitoring, prompt removal |
| Undercorrection | Variable | Ensure adequate growth remaining |
| Recurrence | Blount: significant if growth remaining | Address physeal bar, consider osteotomy |
| Hardware prominence/irritation | Common (8-plate) | May need removal after correction |
| Physeal arrest (iatrogenic) | Rare with 8-plate | Proper technique, avoid physeal damage |
| Compartment syndrome (osteotomy) | Rare but serious | Prophylactic fasciotomy for large corrections |
Overcorrection Risk
Overcorrection is common if 8-plates are not removed promptly. Follow patients closely (every 3 months) and remove hardware as soon as correction is achieved. Correcting past neutral is easy but creates opposite deformity.
Postoperative Care and Rehabilitation
Post-Guided Growth Protocol
- Wound care
- Weight-bearing as tolerated immediately
- No immobilization needed
- Discharge day 0-1
- Normal activities resume quickly
- First follow-up X-ray at 6-8 weeks
- Assess early correction
- Clinical and radiographic assessment
- Measure mechanical axis on long leg films
- Continue until correction achieved
- Once alignment normalized or slightly overcorrected
- Remove 8-plate as day surgery
- May rebound slightly after removal
- Post-removal X-ray at 3-6 months
Post-Osteotomy Rehabilitation:
- Protected weight-bearing for 6-8 weeks
- Knee ROM exercises
- Progress to full weight-bearing and activity
Outcomes
Physiological Deformity:
- Nearly 100% spontaneous resolution
- No intervention needed in vast majority
Guided Growth:
- High success rate for appropriate candidates
- Predictable correction if adequate growth remaining
- Low morbidity, reversible if overcorrects
Blount Disease:
- Infantile: better prognosis if treated early
- Adolescent: higher recurrence, often needs osteotomy
- Long-term: risk of OA if residual malalignment
Evidence Base
8-Plate Guided Growth
- Tension-band plating effective for angular deformity
- Predictable correction rate approximately 1° per month
- Low complication rate
- Requires adequate growth remaining
Bracing for Infantile Blount Disease
- Bracing effective in Langenskiöld I-II stages
- Higher success in children less than 3 years
- Full-time wear required
- Later stages usually require surgery
Adolescent Blount Disease Outcomes
- Adolescent Blount has higher recurrence than infantile
- Often requires osteotomy rather than guided growth
- Obesity is major risk factor
- Long-term risk of osteoarthritis
Physiological Genu Valgum Natural History
- Documented physiological evolution of tibiofemoral angle
- Peak valgus at 3-4 years (10-15°)
- Adult alignment by age 7
- Basis for current understanding of normal development
Rickets-Related Deformity
- Rickets causes angular deformity through physeal widening
- Both varus and valgus patterns can occur
- Treat metabolic cause first
- Guided growth after metabolic control achieved
Exam Viva Scenarios
Practice these scenarios to excel in your viva examination
"A mother brings her 18-month-old child concerned about 'bowlegs'. The child started walking at 11 months and the mother feels the legs are bowed."
"A 2.5-year-old child presents with progressive bowlegs. They have been walking since 10 months. On examination, there is a noticeable lateral thrust during gait, and the varus appears worse on the right side."
"A 9-year-old child presents with bilateral knock-knees that have been present for several years. The parents are concerned it is getting worse. The child is otherwise well and of normal height."
MCQ Practice Points
Physiological Evolution
Q: At what age is peak physiological genu valgum expected? A: Age 3-4 years, with approximately 10-15° valgus. This corrects to adult alignment (5-7° valgus) by age 7.
Blount Diagnosis
Q: What Drennan metaphyseal-diaphyseal angle suggests Blount disease? A: Greater than 16°. This measurement helps distinguish Blount disease (tibia vara) from physiological varus.
Guided Growth
Q: Where is an 8-plate placed to correct genu valgum? A: On the medial side of the distal femur (convex side). This retards medial growth and allows valgus to correct.
Correction Rate
Q: What is the expected correction rate with 8-plate at the distal femur? A: Approximately 1° per month. Correction at the proximal tibia is slightly slower.
Medicolegal Considerations
Documentation Points:
- Age, height, and clinical measurements (ICD/IMD)
- Gait assessment including lateral thrust
- Family history and dietary assessment
- Radiographic findings and measurements
Consent for Surgery:
- Need for hardware removal after correction
- Risk of overcorrection or undercorrection
- May need additional surgery (recurrence, other side)
- Osteotomy risks: compartment syndrome, nerve injury
Follow-up Compliance
Missing follow-up after 8-plate insertion can lead to overcorrection. Emphasize the importance of regular monitoring and document the follow-up plan clearly.
Australian Context
Epidemiology:
- Physiological deformity common presenting concern
- Blount disease less common than in some other populations
- Rickets rare but seen in high-risk groups (covered skin, dark complexion, low sun exposure)
Access to Care:
- Pediatric orthopaedic surgery at tertiary children's hospitals
- Long leg X-ray facilities in major centers
- Guided growth well-established technique
Cultural Considerations:
- Parental concern may be driven by cultural norms about leg shape
- Important to reassure about physiological patterns
- Vitamin D supplementation recommendations for at-risk groups
High-Yield Exam Summary
Physiological Pattern
- •Birth: 10-15° varus (bowlegs)
- •18-24 months: Neutral
- •3-4 years: Peak valgus (10-15°)
- •7 years: Adult alignment (5-7° valgus)
Red Flags for Pathology
- •Lateral thrust during gait
- •Asymmetry between sides
- •Extreme angle beyond expected
- •Short stature or systemic features
Blount Disease
- •Tibia vara from medial proximal tibial physis
- •Infantile (1-3yrs) vs Adolescent (greater than 10yrs)
- •MDA greater than 16° diagnostic
- •Langenskiöld staging determines treatment
Guided Growth (8-Plate)
- •On convex side of deformity
- •Valgus: medial distal femur
- •Varus: lateral proximal tibia
- •Correct approximately 1°/month
- •Remove promptly after correction
Key Numbers
- •MDA greater than 16° = Blount disease
- •5-7° = Normal adult tibiofemoral angle
- •1°/month = 8-plate correction rate
- •At least 2 years growth needed for guided growth