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Plantar Fasciitis Management

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Plantar Fasciitis Management

Comprehensive guide to plantar fasciitis - anatomy, pathophysiology, diagnosis, conservative management, injection therapies, surgical techniques, and evidence-based treatment for orthopaedic exam

complete
Updated: 2024-12-25
High Yield Overview

PLANTAR FASCIITIS - PLANTAR HEEL PAIN SYNDROME

Degenerative Process | Conservative First | 90% Resolve at 1 Year | Surgery Only After 6-12 Months

10%Lifetime incidence in population
90%Resolve with conservative care
6-12moConservative trial before surgery
4mmNormal fascia thickness on USS

TREATMENT STAGES

Stage 1: Acute (0-6 weeks)
PatternRest, ice, NSAIDs, stretching
TreatmentActivity modification, orthoses
Stage 2: Subacute (6 weeks-3 months)
PatternPT, night splints, ESWT
TreatmentIntensive conservative therapy
Stage 3: Chronic (3-12 months)
PatternInjections (steroid, PRP)
TreatmentConsider interventional therapies
Stage 4: Refractory (over 12 months)
PatternFailed all conservative
TreatmentSurgical release (open vs endoscopic)

Critical Must-Knows

  • Plantar fasciitis is a degenerative process, not inflammatory - prefer term plantar fasciosis or plantar heel pain
  • 90% resolve with conservative management within 12 months - surgery is rarely needed
  • Risk factors: obesity, pes planus, limited ankle dorsiflexion, prolonged standing
  • Diagnosis is clinical - tenderness at medial calcaneal tubercle, pain with first steps
  • Conservative trial 6-12 months mandatory before considering surgery
  • Steroid injection provides short-term relief but risk of fascia rupture - use sparingly
  • Surgical release: partial (medial 50%) vs complete - never release more than 50% to avoid lateral column overload

Examiner's Pearls

  • "
    Plantar fascia is windlass mechanism - passive toe extension tightens fascia and raises arch
  • "
    Heel spur present in 50% but is incidental finding, not cause of pain
  • "
    Fascia rupture causes sudden relief of pain but lateral column pain and arch collapse
  • "
    Nerve entrapment (Baxter nerve) is important differential - burning pain, night pain

Critical Plantar Fasciitis Exam Points

Degenerative Not Inflammatory

Histology shows myxoid degeneration and collagen necrosis, not inflammation. Use term plantar fasciosis or plantar heel pain. This explains why NSAIDs and steroid injections have limited long-term benefit.

Conservative Management First

90% resolve within 12 months with conservative care. Mandatory trial of 6-12 months before surgery. Include stretching (gastrocnemius and plantar fascia), orthoses, activity modification, and PT.

Partial Release Only

Never release more than 50% of the plantar fascia width. Complete release causes lateral column overload, arch collapse, and worse outcomes. Release medial half only from medial calcaneal tubercle.

Differential Diagnosis

Baxter nerve entrapment mimics plantar fasciitis - first branch of lateral plantar nerve compressed between abductor hallucis and quadratus plantae. Look for burning pain, night pain, and positive Tinel sign.

At a Glance

Plantar fasciitis is a degenerative process (not inflammatory—prefer "plantar fasciosis") causing plantar heel pain at the medial calcaneal tubercle, classically worst with first steps after rest. Histology shows myxoid degeneration and collagen necrosis, explaining limited long-term benefit from anti-inflammatory treatments. 90% resolve with conservative management within 12 months—stretching (gastrocnemius and plantar fascia), orthoses, and activity modification are first-line. A 6-12 month conservative trial is mandatory before surgery. Surgical release should preserve the lateral 50% of fascia (release medial half only) to prevent lateral column overload and arch collapse. Baxter nerve entrapment is an important differential with burning pain and night symptoms.

Mnemonic

PLANTAR - Risk Factors

P
Prolonged standing/walking
Occupational risk - teachers, nurses, factory workers
L
Limited ankle dorsiflexion
Tight gastrocnemius increases plantar fascia strain
A
Age 40-60 years
Peak incidence in middle age
N
No arch support (pes planus)
Flat feet or high arches both increase risk
T
Training errors in runners
Sudden increase in distance or intensity
A
Adiposity (obesity)
BMI over 30 is major risk factor
R
Reduced first step pain
Classic symptom - worst pain with first steps after rest

Memory Hook:PLANTAR reminds you of all key risk factors and the classic first-step pain pattern

Mnemonic

STRETCH - Conservative Management Algorithm

S
Stretching exercises
Gastrocnemius, soleus, plantar fascia - 3 times daily
T
Time (6-12 months trial)
Mandatory conservative period before surgery
R
Rest and activity modification
Avoid barefoot walking, prolonged standing
E
ESWT (Extracorporeal shockwave)
2000-4000 shocks at 0.1-0.2 mJ/mm² if refractory
T
Taping and night splints
Maintain dorsiflexion overnight, low-dye taping
C
Corticosteroid injection (cautious)
Limited use - risk of rupture, fat pad atrophy
H
Heel cups and orthoses
Cushioned heel cups, custom orthoses for arch support

Memory Hook:STRETCH emphasizes the conservative approach and the importance of stretching exercises

Mnemonic

RELEASE - Surgical Considerations

R
Refractory to all conservative
Failed 6-12 months of comprehensive treatment
E
Endoscopic vs open approach
Endoscopic has faster recovery but learning curve
L
Lateral 50% must be preserved
Release medial half only to prevent lateral overload
E
Examine for nerve entrapment
Release Baxter nerve if entrapped (inferior calcaneal nerve)
A
Avoid complete release
Complete release causes arch collapse, lateral pain
S
Spur removal not necessary
Heel spur is incidental - focus on fascia release
E
Early mobilization postop
Weight-bearing as tolerated, return to sport 3-6 months

Memory Hook:RELEASE guides surgical decision-making and emphasizes partial release only

Mnemonic

4-4-50 Rule - Diagnostic Criteria

4
4mm fascia thickness
Normal plantar fascia under 4mm on ultrasound
4
4cm from insertion
Tenderness typically within 4cm of medial calcaneal tubercle
50
50% have heel spur
Heel spur is incidental finding in 50%, not cause of pain

Memory Hook:4-4-50 helps remember key diagnostic measurements and the incidental nature of heel spurs

Overview and Epidemiology

Plantar fasciitis (also termed plantar fasciosis or plantar heel pain syndrome) is the most common cause of inferior heel pain in adults, accounting for approximately 1 million patient visits annually in the United States. Despite the suffix "itis" suggesting inflammation, histological studies demonstrate a degenerative process characterized by myxoid degeneration, collagen necrosis, and angiofibroblastic hyperplasia rather than acute inflammation.

Epidemiology:

  • Incidence: Affects approximately 10% of the population at some point in their lifetime
  • Peak age: 40-60 years, though can occur at any age
  • Gender: Slight female predominance (2:1 ratio)
  • Bilateral: Occurs in 30% of cases
  • Athletes: Higher incidence in runners (up to 22% prevalence) and dancers

Terminology Matters

The term plantar fasciitis is entrenched in clinical practice, but plantar fasciosis or plantar heel pain more accurately reflects the pathology. In exams, acknowledge that histology shows degeneration, not inflammation, which explains the limited benefit of anti-inflammatory treatments.

Natural history:

  • Self-limiting condition: 80-90% resolve within 12 months with conservative treatment
  • Spontaneous resolution: Even without treatment, many cases improve over 1-2 years
  • Recurrence: Approximately 25% experience recurrent symptoms
  • Chronic cases: 10% develop chronic, refractory symptoms requiring advanced interventions

The excellent prognosis with conservative care is the foundation of treatment algorithms, with surgery reserved only for the small minority who fail comprehensive non-operative management.

Pathophysiology

Plantar Fascia Anatomy

Structure and function:

  • Origin: Medial calcaneal tubercle on the medial process of the calcaneal tuberosity
  • Insertion: Divides into five bands inserting onto the proximal phalanges and flexor tendon sheaths
  • Composition: Dense fibrous connective tissue arranged in three bands (medial, central, lateral)
  • Central band: Thickest and strongest, most commonly affected in plantar fasciitis
  • Thickness: Normal fascia is 2-4mm thick; in plantar fasciitis often exceeds 4mm
  • Blood supply: Relatively avascular, which contributes to slow healing

Biomechanical function:

  • Windlass mechanism: Passive toe extension during push-off tightens the plantar fascia and raises the medial longitudinal arch
  • Arch support: Provides static support to the longitudinal arch of the foot
  • Shock absorption: Absorbs impact forces during heel strike (up to 2-3 times body weight)
  • Spring ligament: Works in concert with spring ligament and posterior tibial tendon for arch stability

Windlass Mechanism Clinical Test

The windlass test reproduces pain by passively dorsiflexing the hallux, which tightens the plantar fascia and stresses the inflamed origin. This test has good sensitivity (32-100%) but variable specificity, and helps confirm the diagnosis clinically.

Pathophysiology

Degenerative process (not inflammatory):

Histological studies of chronic plantar fasciitis demonstrate:

  • Myxoid degeneration: Disorganization of collagen fibers
  • Collagen necrosis: Breakdown of normal fascial architecture
  • Angiofibroblastic hyperplasia: Proliferation of fibroblasts and small blood vessels
  • Absence of inflammatory cells: Minimal or no lymphocytes, macrophages, or neutrophils

This pathology is similar to other tendinopathies (Achilles tendinosis, lateral epicondylosis) and explains why anti-inflammatory treatments have limited long-term efficacy.

Mechanical overload theory:

The most accepted theory involves repetitive microtrauma to the plantar fascia origin:

  • Repetitive stress: Excessive or repetitive loading exceeds the fascia's capacity for repair
  • Microtears: Develop at the fascial origin from the medial calcaneal tubercle
  • Failed healing response: Instead of inflammation and repair, degeneration occurs
  • Cycle continues: Ongoing stress prevents healing, perpetuating the degenerative process

Contributory factors:

  • Tight gastrocnemius: Limited ankle dorsiflexion increases strain on plantar fascia by up to 15%
  • Obesity: Increased BMI directly increases load on the fascia
  • Abnormal foot mechanics: Both pes planus (overpronation) and pes cavus (poor shock absorption) increase stress
  • Occupational standing: Prolonged standing or walking on hard surfaces
  • Training errors: Sudden increase in activity in runners or athletes

Heel spur - association vs causation:

  • Prevalence: Calcaneal spurs present in 50% of patients with plantar fasciitis
  • Asymptomatic spurs: Also present in 15-25% of asymptomatic individuals
  • Formation: Develops from traction at the origin of flexor digitorum brevis, not the plantar fascia
  • Clinical significance: Spur is an incidental finding, not the cause of pain
  • Surgical implication: Spur excision is unnecessary during plantar fascia release

Risk Factors and Prevention

Risk Factors - Modifiable vs Non-Modifiable

CategoryModifiableNon-Modifiable
BiomechanicalTight gastrocnemius, poor footwear, training errors, abnormal gaitPes planus, pes cavus, leg length discrepancy, age 40-60
Body habitusObesity (BMI over 30), weight managementFemale gender, genetic predisposition to flat feet
Activity-relatedOccupational standing, running volume, surface hardnessNone
SystemicDiabetes control, inflammatory arthritis managementSeronegative spondyloarthropathy, diabetes mellitus

Key modifiable risk factors:

  • Obesity: Strongest modifiable risk factor - BMI over 30 increases risk 5-fold
  • Tight gastrocnemius-soleus complex: Limited ankle dorsiflexion (under 10 degrees) increases plantar fascia strain
  • Inappropriate footwear: Worn-out shoes, lack of arch support, high heels, flip-flops
  • Training errors: Sudden increase in running distance or intensity (over 10% per week increase)
  • Occupational: Prolonged standing or walking on hard surfaces (over 8 hours daily)
  • Poor biomechanics: Overpronation, supination, or other gait abnormalities

Prevention strategies:

  • Weight management: Maintain healthy BMI
  • Stretching program: Daily gastrocnemius and plantar fascia stretching
  • Appropriate footwear: Well-cushioned shoes with arch support, replace running shoes every 400-500 miles
  • Activity progression: Gradual increase in activity level (10% rule)
  • Cross-training: Vary exercise to reduce repetitive stress
  • Early intervention: Address tight calf muscles and biomechanical issues promptly

Classification

Classification by Severity

GradeDescriptionSymptomsImpact on Activity
MildEarly/acuteMorning pain resolving within 10-15 minutesMinimal activity modification
ModerateSubacutePersistent pain affecting daily activitiesModified exercise program
SevereChronicConstant pain with rest painUnable to perform usual activities
RefractoryTreatment-resistantFailed 6+ months conservative careConsider procedural intervention

Clinical Classification Subtypes

By Duration:

  • Acute: Less than 6 weeks duration
  • Subacute: 6 weeks to 6 months
  • Chronic: Greater than 6 months

By Etiology:

  • Mechanical/overuse: Most common - repetitive microtrauma
  • Degenerative (fasciopathy): Chronic changes without inflammation
  • Inflammatory: Associated with seronegative arthropathy (HLA-B27)
  • Neurogenic: Baxter's nerve entrapment component

By Response to Treatment:

  • Responsive: Resolves with first-line conservative care
  • Refractory: Requires second-line interventions
  • Recalcitrant: Failed all conservative measures

Clinical Presentation and Diagnosis

History

Classic presentation:

  • First-step pain: Severe pain with first steps in the morning or after prolonged sitting (95% of cases)
  • Pain location: Inferior heel at the medial calcaneal tubercle
  • Gradual onset: Usually develops over weeks to months, not acute
  • Pain pattern: Improves with initial activity as fascia "warms up," then worsens with prolonged activity
  • Impact of rest: Pain decreases with rest, recurs with weight-bearing
  • Night pain: Typically absent - if present, consider other diagnoses

Red flags requiring further investigation:

  • Severe night pain (possible infection, tumor, inflammatory arthritis)
  • Fever, systemic symptoms (septic arthritis, osteomyelitis)
  • History of trauma (calcaneal fracture)
  • Progressive neurological symptoms (tarsal tunnel, nerve compression)
  • Lack of response to conservative treatment (reconsider diagnosis)

Aggravating factors:

  • Walking barefoot on hard surfaces
  • Climbing stairs
  • Prolonged standing or walking
  • Running or jumping activities
  • First steps after rest

Relieving factors:

  • Rest
  • Sitting with feet elevated
  • Supportive footwear
  • Activity modification

Physical Examination

Inspection:

  • Gait: May demonstrate antalgic gait with shortened stance phase on affected side
  • Foot alignment: Assess for pes planus, pes cavus, or neutral arch
  • Muscle atrophy: Chronic cases may show calf atrophy
  • Swelling: Typically absent - if present, consider alternative diagnosis

Palpation (key finding):

  • Point tenderness: Exquisite tenderness at medial calcaneal tubercle (95% sensitive)
  • Location: Typically 4cm or less from the insertion point
  • Diffuse vs focal: Plantar fasciitis causes focal tenderness; diffuse pain suggests alternative diagnosis
  • Lateral tenderness: Absence of lateral heel tenderness helps distinguish from other conditions

Range of motion:

  • Ankle dorsiflexion: Assess with knee extended (gastrocnemius) and flexed (soleus)
  • Limited dorsiflexion: Less than 10 degrees with knee extended is significant risk factor
  • Hallux dorsiflexion: Normal but may reproduce pain (windlass test)

Special tests:

TestTechniquePositive FindingClinical Significance
Windlass testPassively dorsiflex halluxReproduction of heel painConfirms plantar fascia as pain source
Tinel signTap over tarsal tunnel or Baxter nerveRadiating burning painSuggests nerve entrapment
Dorsiflexion-eversion testDorsiflex ankle and evert footPain medial heelSuggests posterior tibial tendon pathology
Palpation of Baxter nerveDeep palpation between abductor hallucis and quadratus plantaeBurning pain, radiationBaxter nerve entrapment

Windlass Test Technique

With the patient standing or sitting, passively dorsiflex the hallux (great toe) while palpating the plantar fascia. This tightens the fascia through the windlass mechanism. Pain at the medial calcaneal origin is a positive test. Sensitivity varies (32-100%) but specificity is good when combined with history.

Differential Diagnosis

Important alternative diagnoses:

  1. Baxter nerve entrapment (first branch of lateral plantar nerve):

    • Burning pain, often with radiation to medial heel
    • Night pain more common than plantar fasciitis
    • Positive Tinel sign between abductor hallucis and quadratus plantae
    • May coexist with plantar fasciitis
  2. Tarsal tunnel syndrome:

    • Pain and paresthesias in plantar foot
    • Positive Tinel sign posterior to medial malleolus
    • Night pain and numbness common
  3. Calcaneal stress fracture:

    • History of increased activity or trauma
    • Pain with medial-lateral calcaneal squeeze test
    • Tenderness over calcaneal body, not just plantar aspect
  4. Fat pad atrophy or contusion:

    • Diffuse heel pain, not focal at medial tubercle
    • History of repetitive impact or prior steroid injections
    • Pain worse with direct pressure to heel pad
  5. Inflammatory arthritis (seronegative spondyloarthropathy):

    • Bilateral symptoms common
    • Morning stiffness lasting over 30 minutes
    • Associated symptoms (back pain, uveitis, psoriasis)
    • Positive HLA-B27 in many cases
  6. Calcaneal apophysitis (Sever disease) in children:

    • Age 8-15 years
    • Tenderness at calcaneal insertion of Achilles tendon
    • Pain with squeeze test of calcaneus
  7. Tumor or infection (rare):

    • Night pain, systemic symptoms
    • Progressive pain unrelieved by rest
    • Requires imaging to rule out

Investigations and Imaging

Diagnosis is Clinical

Plantar fasciitis is primarily a clinical diagnosis based on history and physical examination. Imaging is reserved for atypical presentations, failed conservative treatment, or when alternative diagnoses are suspected.

Normal Plantar Fascia Anatomy

Normal plantar fascia appearance on multimodal imaging
Click to expand
Five-panel multimodal imaging showing normal plantar fascia anatomy for comparison. (a) Schematic diagram illustrating the normal thin plantar fascia originating from the calcaneus. (b) Lateral X-ray with arrows indicating the normal thin plantar fascia. (c) Ultrasound demonstrating normal fibrillar echogenic plantar fascia (typically less than 4mm thick). (d-e) MRI T1 and T2-weighted sequences showing normal thin, low-signal plantar fascia. Understanding normal anatomy is essential for recognizing pathological thickening and signal changes in plantar fasciitis.Credit: Open-i (NIH) - CC BY 4.0

Plain Radiographs

Indications:

  • Atypical presentation
  • History of trauma
  • Failed conservative treatment over 3 months
  • Need to rule out fracture, tumor, or other bony pathology

Findings:

  • Heel spur: Present in 50% of plantar fasciitis cases but also in 15-25% of asymptomatic individuals
  • Incidental finding: Spur location is at flexor digitorum brevis origin, not plantar fascia
  • Not diagnostic: Presence or absence does not confirm or exclude diagnosis
  • Other findings: May reveal stress fracture, tumor, or arthritic changes

X-ray technique:

  • Lateral view: Best view for assessing heel spur and soft tissue thickness
  • Weight-bearing: Preferred for assessing foot alignment and arch height
  • Bilateral comparison: May be helpful in assessing arch differences

Ultrasound

Advantages:

  • Non-invasive, no radiation
  • Dynamic assessment: Can assess fascia with foot in different positions
  • Cost-effective: Less expensive than MRI
  • Readily available: Can be performed in clinic
  • Guided injections: Allows real-time visualization for injection therapy

Diagnostic findings:

  • Fascial thickening: Normal fascia less than 4mm; plantar fasciitis typically over 4mm (often 5-7mm)
  • Hypoechoic appearance: Loss of normal fibrillar pattern, indicating degeneration
  • Perifascial edema: Fluid around the fascia
  • Power Doppler: Increased vascularity suggests active process (though less common given degenerative nature)

Ultrasound Thickness Criteria

Plantar fascia thickness over 4mm on ultrasound has sensitivity of 80-95% and specificity of 80-85% for plantar fasciitis. Measurement should be taken at the fascial origin from the medial calcaneal tubercle in sagittal plane.

Differential findings on ultrasound:

  • Fascia rupture: Discontinuity of fascia, often with hematoma
  • Heel pad atrophy: Thinning of the normal 2cm heel fat pad
  • Nerve compression: Hypoechoic mass or swelling along nerve course
  • Plantar fibroma: Hypoechoic nodule within the plantar fascia (more distal)

Magnetic Resonance Imaging (MRI)

Indications:

  • Atypical presentation with concern for alternative diagnosis
  • Failed conservative treatment over 6 months
  • Preoperative planning in rare surgical candidates
  • Suspected stress fracture, tumor, or infection
  • Nerve entrapment evaluation

Findings in plantar fasciitis:

  • Fascial thickening: Greater than 4mm, often 5-8mm
  • Increased T2 signal: Indicates edema and degeneration within the fascia
  • Perifascial edema: High signal around fascia on fluid-sensitive sequences
  • Calcaneal bone marrow edema: Seen in severe cases at the fascial attachment site
  • Fascial discontinuity: Indicates partial or complete rupture (complication)

Differential diagnoses on MRI:

  • Baxter nerve entrapment: Edema or mass between abductor hallucis and quadratus plantae
  • Tarsal tunnel syndrome: Nerve enlargement or mass in tarsal tunnel
  • Calcaneal stress fracture: Bone marrow edema in calcaneal body
  • Tumor: Mass lesion with characteristic signal patterns
  • Infection: Bone marrow edema with cortical destruction and soft tissue involvement

Multimodal Imaging Comparison

Multimodal imaging of plantar fasciitis - X-ray, ultrasound, and MRI comparison
Click to expand
Four-panel multimodal imaging comparison in plantar fasciitis. (a) Lateral X-ray with arrow indicating the plantar fascia insertion at the medial calcaneal tubercle. (b) Ultrasound showing thickened hypoechoic plantar fascia measuring 6.5mm (normal less than 4mm), with loss of normal fibrillar echotexture. (c) Sagittal T1-weighted MRI showing thickened plantar fascia with intermediate signal. (d) Sagittal STIR MRI demonstrating high signal within and around the fascia indicating active inflammation and degeneration. This multimodal approach is valuable when clinical diagnosis is uncertain.Credit: Open-i (NIH) - CC BY 4.0
Classic plantar fasciitis imaging findings
Click to expand
Three-panel imaging demonstrating classic plantar fasciitis findings. (a) Lateral X-ray showing thickened plantar fascia at the calcaneal insertion (yellow arrows), insertional calcification (white arrowhead), and plantar heel spur. Note: heel spurs are an incidental finding present in 50% of cases but also in 15-25% of asymptomatic individuals. (b) Sagittal T1 MRI showing thickened plantar fascia with intermediate signal intensity. (c) Sagittal STIR MRI demonstrating bright signal within the fascia and at the calcaneal enthesis, indicating active inflammation and bone marrow edema.Credit: Open-i (NIH) - CC BY 4.0

Laboratory Tests

Generally not required for typical plantar fasciitis.

Consider when:

  • Bilateral symptoms with systemic features (inflammatory arthropathy)
  • Concern for seronegative spondyloarthropathy
  • Atypical presentation

Potential tests:

  • HLA-B27: If suspecting ankylosing spondylitis or reactive arthritis
  • Inflammatory markers: ESR, CRP if concerned about inflammatory or infectious process
  • Rheumatoid factor, anti-CCP: If rheumatoid arthritis suspected
  • Uric acid: If gout is in differential (though rarely causes isolated heel pain)

Management

📊 Management Algorithm
plantar fasciitis management management algorithm
Click to expand
Management algorithm for plantar fasciitis managementCredit: OrthoVellum

Treatment Philosophy

90% of patients improve with conservative management within 12 months. The goal is to address biomechanical factors, reduce load on the fascia, and allow the degenerative process to resolve. A structured, stepwise approach is recommended with patience and realistic expectations.

Initial treatment:

  • Activity modification (reduce standing, running)
  • Ice massage (15 minutes, 3-4 times daily)
  • NSAIDs (2-week course for pain relief, not long-term)
  • Stretching exercises (calf and plantar fascia)
  • Supportive footwear with cushioned heels
  • Avoid barefoot walking

Expected outcome: 20% significant improvement by 6 weeks.

Escalate conservative treatment:

  • Formal physical therapy referral
  • Night splints (maintain dorsiflexion overnight)
  • Custom orthoses or prefabricated arch supports
  • Low-dye taping technique
  • Continue stretching program (3 times daily)
  • Consider extracorporeal shockwave therapy (ESWT)

Expected outcome: Additional 30-40% improvement by 3 months (total 60% improved).

Advanced conservative therapies:

  • ESWT if not already tried (2000-4000 shocks)
  • Consider corticosteroid injection (use sparingly)
  • Platelet-rich plasma (PRP) injection (emerging evidence)
  • Intensive PT with iontophoresis
  • Assess footwear and biomechanics
  • Weight loss if BMI over 30

Expected outcome: Additional 20-25% improvement by 6 months (total 80-85% improved).

Final conservative measures:

  • Repeat injection therapy if single prior injection
  • Trial of different orthoses or footwear
  • Consider walking boot for 4-6 weeks (rarely needed)
  • Rule out alternative diagnoses with imaging
  • Reassess compliance with stretching and PT

Expected outcome: Additional 5-10% improvement by 12 months (total 90% improved).

Criteria for surgery:

  • Failed ALL conservative measures above
  • Symptoms significantly impair quality of life
  • Minimum 6-12 months of treatment
  • Imaging confirms no alternative diagnosis
  • Patient motivated and understands risks

Surgical success: 70-90% improvement with plantar fascia release.

Specific Conservative Interventions

1. Stretching Exercises (First-Line, Essential)

Gastrocnemius-soleus stretching:

  • Technique: Stand facing wall, affected leg behind, knee straight, lean forward until stretch felt in calf
  • Duration: Hold 30 seconds, repeat 3 times
  • Frequency: 3 times daily (morning, midday, evening)
  • Soleus variation: Repeat with knee bent to isolate soleus
  • Evidence: High-quality evidence supporting stretching as cornerstone of treatment

Plantar fascia-specific stretching:

  • Technique: Seated, cross affected foot over opposite knee, pull toes back toward shin while massaging arch
  • Duration: Hold 10 seconds, repeat 10 times
  • Frequency: Before first steps in morning and before standing after prolonged sitting
  • Evidence: Specific fascia stretching may be superior to calf stretching alone

Stretching Protocol Evidence

A landmark RCT by DiGiovanni et al. demonstrated that plantar fascia-specific stretching was superior to standard Achilles stretching for pain relief and functional improvement at 8 weeks. This has become the foundation of conservative treatment protocols.

2. Orthoses and Footwear Modifications

Heel cups and cushions:

  • Silicon heel cups: Reduce impact forces on heel
  • Gel inserts: Provide cushioning
  • Taping: Low-dye taping supports arch and reduces fascia strain

Prefabricated orthoses:

  • Over-the-counter arch supports: Readily available, inexpensive
  • Cushioned insoles: Reduce heel impact
  • Evidence: Moderate evidence for short-term benefit (3-6 months)

Custom orthoses:

  • Semi-rigid orthoses: Provide arch support and control pronation
  • Indications: Significant pes planus or cavus, failed prefabricated orthoses
  • Cost: More expensive but may be worth trial in refractory cases
  • Evidence: Limited evidence that custom is superior to prefabricated

Footwear recommendations:

  • Avoid: High heels, flip-flops, worn-out shoes, flat unsupportive shoes
  • Prefer: Well-cushioned athletic shoes, shoes with arch support and heel counter
  • Running shoes: Replace every 400-500 miles

3. Night Splints

Mechanism: Maintain ankle in neutral or slight dorsiflexion overnight, preventing fascia from contracting and reducing "first-step" pain in morning.

Types:

  • Posterior leaf spring: L-shaped splint maintaining dorsiflexion
  • Sock-type splints: More comfortable, lower profile
  • Adjustable: Allow gradual increase in dorsiflexion stretch

Protocol:

  • Wear: Nightly for 1-3 months
  • Compliance: Key challenge - 50% discontinue due to discomfort
  • Evidence: Moderate evidence for effectiveness when tolerated

4. Extracorporeal Shockwave Therapy (ESWT)

Mechanism: High-energy acoustic waves induce microtrauma, stimulating neovascularization and healing response in degenerative tissue.

Protocol:

  • Energy level: 0.1-0.2 mJ/mm² (low to medium energy)
  • Shocks per session: 2000-4000 shocks
  • Sessions: Typically 3 sessions, 1-2 weeks apart
  • Anesthesia: Usually performed without anesthesia (mild discomfort)

Evidence:

  • Meta-analyses: Show moderate benefit over placebo for pain reduction
  • Success rate: 60-80% improvement in refractory cases
  • Timing: Best used after 3-6 months of failed conservative care
  • Cochrane review: Moderate evidence for effectiveness

Contraindications:

  • Infection at treatment site
  • Malignancy
  • Pregnancy
  • Coagulopathy or anticoagulation
  • Open growth plates in children

Level I
📚 Cochrane Review: Shockwave therapy for plantar heel pain
(2015)
Key Findings:
  • ESWT showed statistically significant improvement in pain reduction compared to placebo
  • Moderate quality evidence supports use in chronic plantar fasciitis after conservative measures fail
  • Most effective in patients with symptoms over 6 months who have failed other conservative measures

5. Activity Modification

Reduce aggravating activities:

  • Limit standing: Take regular sitting breaks if occupation requires prolonged standing
  • Modify exercise: Replace running with swimming, cycling, or elliptical
  • Avoid barefoot walking: Always wear supportive footwear, even at home
  • Weight management: Target BMI under 30 if overweight

Gradual return to activity:

  • 10% rule: Increase activity by no more than 10% per week
  • Cross-training: Vary activities to reduce repetitive stress
  • Appropriate surfaces: Avoid hard surfaces; prefer grass or synthetic tracks

This completes the conservative management overview.

Corticosteroid Injection

Indications:

  • Moderate to severe pain limiting function
  • Failed initial conservative measures (stretching, orthoses, PT)
  • Typically reserved for 3+ months of symptoms
  • Use sparingly due to risks

Technique:

  • Approach: Medial approach to avoid puncturing heel pad
  • Needle: 22-25 gauge, 1.5 inch needle
  • Medication: 1 mL of corticosteroid (triamcinolone 40mg or methylprednisolone 40mg) mixed with 1-2 mL local anesthetic (lidocaine 1%)
  • Landmark: Palpate medial calcaneal tubercle; insert needle just plantar to tubercle, directing laterally
  • Ultrasound guidance: Preferred for accuracy and to avoid heel pad injection

Post-injection protocol:

  • Rest: Avoid prolonged standing or high-impact activities for 48-72 hours
  • Ice: Apply ice for 15 minutes several times daily for 2-3 days
  • Activity: Gradual return to normal activity over 1 week
  • Repeat: No more than 2-3 injections total; repeat only after 6 weeks minimum

Risks of corticosteroid injection:

  • Plantar fascia rupture (5-10% risk): Sudden relief of pain followed by acute arch collapse, lateral column pain, and worse function. Risk increases with multiple injections.
  • Heel pad fat atrophy: Permanent thinning of protective heel fat pad if injection placed into fat pad rather than fascia.
  • Skin depigmentation: Especially in darker-skinned individuals.
  • Infection: Rare with sterile technique.
  • Hyperglycemia: Transient elevation in diabetic patients.

Evidence:

  • Short-term benefit: Good evidence for pain relief at 1 month
  • Long-term outcomes: No benefit over placebo at 6-12 months
  • Complications: Higher rupture rate compared to conservative treatment alone
  • Conclusion: Use for short-term symptom relief only; not a definitive treatment

Level I
📚 Systematic Review: Corticosteroid injection for plantar heel pain
(2012)
Key Findings:
  • Corticosteroid injections provide short-term pain relief at 1 month
  • No benefit over placebo at 3-6 months follow-up
  • Risk of plantar fascia rupture increases with repeated injections
  • Recommend maximum of 2 injections total

Platelet-Rich Plasma (PRP) Injection

Rationale: Autologous platelets contain growth factors (PDGF, VEGF, TGF-beta) that may stimulate healing in degenerative tissue. Emerging therapy with increasing evidence.

Technique:

  • Preparation: Draw 20-60 mL patient blood, centrifuge to concentrate platelets (3-5x baseline)
  • Injection volume: 2-3 mL of PRP
  • Approach: Medial approach under ultrasound guidance
  • Target: Inject into degenerative areas of fascia (hypoechoic regions on ultrasound)
  • Peppering technique: Multiple small injections throughout affected area

Post-injection protocol:

  • Initial rest: 48 hours minimal weight-bearing
  • Activity restriction: Avoid high-impact activities for 2 weeks
  • Stretching: Continue stretching program
  • Repeat injections: Some protocols use 2-3 injections, 2-4 weeks apart

Evidence:

  • Emerging positive data: Several RCTs show superiority over corticosteroid at 3-6 months
  • Comparison to steroid: PRP may have slower onset but better long-term outcomes
  • Safety: Lower risk of rupture compared to corticosteroid
  • Limitations: Not standardized (varying platelet concentrations, preparation methods)
  • Cost: More expensive than steroid; often not covered by insurance

Level II
📚 RCT: PRP versus corticosteroid for chronic plantar fasciitis
(2014)
Key Findings:
  • PRP injection showed inferior pain relief at 1 month compared to corticosteroid
  • PRP demonstrated superior outcomes at 3 and 6 months follow-up
  • No fascia ruptures in PRP group versus 3 ruptures in steroid group
  • PRP may have slower onset but better long-term safety profile

Contraindications:

  • Active infection
  • Malignancy
  • Platelet disorders or anticoagulation (relative)
  • Immunosuppression

Other Injection Therapies (Limited Evidence)

Botulinum toxin:

  • Mechanism: Paralyzes gastrocnemius-soleus, reducing tension on fascia
  • Evidence: Very limited; small case series show benefit but no high-quality RCTs
  • Not standard practice

Prolotherapy (dextrose injection):

  • Mechanism: Hyperosmolar dextrose induces inflammatory response
  • Evidence: Limited; small studies suggest possible benefit
  • Not widely adopted

This completes the injection therapies section.

Surgical Management

Indications for Surgery

Strict criteria must be met before considering surgical intervention:

Essential requirements:

  • Failed comprehensive conservative treatment for minimum 6-12 months
  • All non-operative measures attempted: stretching, PT, orthoses, night splints, ESWT, and at least one injection
  • Imaging confirmation: No alternative diagnosis (MRI if needed)
  • Significant functional impairment: Pain limiting activities of daily living
  • Patient motivated and compliant: Understanding of postoperative rehabilitation
  • Realistic expectations: Surgery is not guaranteed success

Relative contraindications:

  • Active infection
  • Peripheral neuropathy (diabetic neuropathy increases risk of complications)
  • Venous insufficiency or PAD (poor healing)
  • Inflammatory arthropathy (may have systemic component)
  • Worker's compensation or litigation (poorer outcomes)
  • Non-compliance with prior treatment

Only 10% of plantar fasciitis patients are surgical candidates after appropriate conservative management. Surgery should be considered a last resort for chronic, refractory cases only.

Surgical Options

Open Plantar Fascia Release

Indications:

  • Failed conservative treatment over 6-12 months
  • Surgeon preference or lack of endoscopic equipment
  • Need for nerve decompression (Baxter nerve)
  • Concern for atypical anatomy or pathology

Surgical Technique:

Patient positioning:

  • Supine with bump under ipsilateral hip
  • Tourniquet on thigh
  • Foot and ankle prepped and draped

Incision:

  • Medial longitudinal incision: 3-4 cm incision centered over medial calcaneal tubercle
  • Start just distal to tubercle, extend distally along medial arch
  • Avoid plantar surface to prevent painful scar

Approach and dissection:

  • Incise skin and subcutaneous tissue
  • Identify abductor hallucis muscle along medial border
  • Retract abductor hallucis plantarly to expose plantar fascia origin
  • Identify thick, glistening white fascia inserting onto medial tubercle

Plantar fascia release:

  • Partial release only: Release medial 50% of fascia width
  • Transect fascia sharply 1-2 cm from calcaneal origin
  • Preserve lateral 50%: Essential to prevent lateral column overload and arch collapse
  • Palpate release with finger to confirm adequate release but lateral half intact

Baxter nerve decompression (if indicated):

  • Identify first branch of lateral plantar nerve (Baxter nerve) deep between abductor hallucis and quadratus plantae
  • Release fascia compressing nerve if entrapment suspected
  • Decompress nerve distally to medial calcaneal tuberosity

Heel spur management:

  • Do NOT routinely excise - spur is incidental finding
  • If large and prominent, may remove with rongeur
  • Not necessary for symptom relief

Closure:

  • Irrigate wound
  • Close subcutaneous layer with absorbable suture
  • Skin closure with non-absorbable suture or staples
  • Apply soft dressing and posterior splint in neutral

Postoperative protocol:

  • Weight-bearing: Non-weight-bearing or toe-touch for 2 weeks, then progressive weight-bearing in boot
  • Immobilization: Posterior splint or boot for 2-4 weeks
  • Range of motion: Gentle ankle ROM exercises starting at 2 weeks
  • Physical therapy: Formal PT at 4-6 weeks for strengthening
  • Return to sport: 3-6 months depending on activity level

This completes the open release technique description.

Endoscopic Plantar Fascia Release (EPR)

Advantages over open:

  • Smaller incision (5mm portal)
  • Less soft tissue dissection
  • Faster recovery and return to activity
  • Reduced postoperative pain
  • Lower infection risk

Disadvantages:

  • Learning curve: Technically demanding
  • Limited visualization: Cannot address Baxter nerve
  • Equipment required: Specialized endoscopic instruments
  • Risk of incomplete release or injury to adjacent structures

Surgical Technique:

Patient positioning:

  • Supine with thigh tourniquet
  • Bump under ipsilateral hip for neutral foot position

Portal placement:

  • Medial portal: 1 cm incision at medial border of heel, just plantar to medial malleolus
  • Lateral portal: 1 cm incision at lateral border of heel, mirror image of medial portal
  • Portals should be at level of plantar fascia origin

Endoscopic dissection:

  • Insert obturator through medial portal to create space
  • Insert endoscope (4mm, 30-degree) through medial portal
  • Visualize plantar fascia under direct vision
  • Insert knife or scissors through lateral portal

Fascia release:

  • Under direct visualization, release medial 50% of plantar fascia
  • Transect fascia from medial to lateral, stopping at midpoint
  • Do not release entire width - risk of lateral column overload
  • Confirm release by palpating fascia externally and visualizing endoscopically

Closure:

  • Remove instruments
  • Close portals with single suture each
  • Apply soft dressing

Postoperative protocol:

  • Weight-bearing: Weight-bearing as tolerated in surgical shoe or boot
  • Earlier mobilization than open: Many protocols allow immediate protected weight-bearing
  • Return to activity: 6-12 weeks depending on activity level
  • Physical therapy: Start at 2-4 weeks

This completes the endoscopic release technique description.

Gastrocnemius Recession (Strayer Procedure)

Rationale: Isolated gastrocnemius contracture increases plantar fascia strain. Releasing tight gastrocnemius may reduce fascia tension without directly cutting fascia.

Indications:

  • Plantar fasciitis with isolated gastrocnemius contracture (less than 10 degrees dorsiflexion with knee extended, normal with knee flexed)
  • Patient preference for procedure that does not violate plantar fascia
  • Failed conservative treatment

Technique (Strayer procedure):

  • Incision: Medial longitudinal incision at mid-calf
  • Dissection: Identify gastrocnemius aponeurosis (distinct from soleus)
  • Release: Transect gastrocnemius aponeurosis transversely, preserving underlying soleus
  • Result: Lengthens gastrocnemius, allows increased dorsiflexion

Evidence:

  • Limited high-quality data: Small case series suggest benefit
  • Theoretical advantage: Preserves plantar fascia integrity
  • Not standard first-line surgical option for plantar fasciitis

This completes the gastrocnemius recession section.

Surgical Outcomes

Success rates:

  • Overall: 70-90% of patients report good to excellent outcomes after plantar fascia release
  • Pain relief: 80-85% achieve significant pain reduction
  • Return to activity: Most return to normal activities by 3-6 months
  • Endoscopic vs open: Similar outcomes in experienced hands; endoscopic may have faster return

Factors predicting success:

  • Appropriate patient selection (failed all conservative measures)
  • Correct diagnosis (no alternative pathology)
  • Partial (not complete) fascia release
  • Compliance with postoperative rehabilitation
  • Non-worker's compensation cases

Factors predicting failure:

  • Incomplete conservative trial
  • Worker's compensation or litigation
  • Secondary gain issues
  • Peripheral neuropathy or systemic disease
  • Complete fascia release (causes new problems)

Level III
📚 Systematic Review: Surgical treatment of chronic plantar fasciitis
(2018)
Key Findings:
  • Plantar fascia release results in good to excellent outcomes in 70-90% of carefully selected patients
  • Required failure of comprehensive conservative treatment for 6-12 months minimum
  • Partial release (medial 50% only) is essential to avoid lateral column overload
  • Both open and endoscopic techniques show similar outcomes

Complications

Conservative Treatment Complications

Steroid injection complications:

  • Plantar fascia rupture (5-10%): Sudden relief of pain, followed by arch collapse, lateral column pain, and worse function
  • Fat pad atrophy: Permanent thinning of heel fat pad if injection misplaced
  • Skin depigmentation: More common in darker-skinned patients
  • Infection: Rare with sterile technique
  • Hyperglycemia: Transient in diabetic patients

ESWT complications:

  • Pain during treatment: Usually mild and tolerable
  • Bruising: Temporary, resolves in 1-2 weeks
  • Rare: Nerve injury, skin burns (very rare)

Night splint complications:

  • Poor compliance: 50% discontinue due to discomfort
  • Skin irritation: Friction at points of contact
  • Calf cramping: From prolonged dorsiflexion stretch

Surgical Complications

Intraoperative:

  • Nerve injury: Baxter nerve (lateral plantar nerve) injury causing lateral heel numbness or burning pain
  • Incomplete release: Under-release fails to relieve symptoms
  • Excessive release: Over-release (more than 50%) causes lateral column overload
  • Vascular injury: Rare; medial plantar artery at risk

Early postoperative (less than 6 weeks):

  • Wound infection: 1-2% incidence
  • Hematoma: Usually resolves spontaneously
  • Wound dehiscence: More common in diabetics, smokers, or with excessive early activity
  • Deep vein thrombosis: Rare; consider prophylaxis in high-risk patients

Late postoperative (over 6 weeks):

  • Lateral column pain and overload (10-15%): From excessive release (more than 50%), causes lateral foot pain and stress fractures
  • Arch collapse: Loss of medial longitudinal arch from complete release
  • Continued heel pain (10-30%): Incomplete release, scar tissue, or incorrect diagnosis
  • Calcaneal stress fracture: Rare; from altered biomechanics post-release
  • Tarsal tunnel syndrome: From scarring around posterior tibial nerve
  • Neuromas: Entrapment of small sensory nerves in scar tissue
  • Painful scar: Especially if incision placed on plantar surface

Lateral column overload syndrome is the most serious complication of plantar fascia release. It results from releasing more than 50% of the fascia width, causing loss of arch support, lateral foot pain, cuboid stress fractures, and peroneal tendinitis. This complication can be more disabling than the original plantar fasciitis. Prevention is essential - never release more than 50% of fascia.

Plantar Fascia Rupture (Non-Surgical)

Causes:

  • Corticosteroid injection (most common)
  • Acute trauma in chronically degenerated fascia
  • Spontaneous in severe plantar fasciitis

Presentation:

  • Sudden "pop" or tearing sensation in arch
  • Immediate relief of heel pain (diagnostic clue)
  • Acute arch collapse: Foot flattens
  • Bruising and swelling: Develops over 24-48 hours along medial arch
  • New lateral column pain: From altered biomechanics

Diagnosis:

  • Clinical: History of pop, sudden relief of plantar heel pain, new arch collapse
  • Ultrasound: Discontinuity of fascia, hematoma
  • MRI: Fascial disruption with surrounding edema
Plantar fascia rupture on ultrasound and MRI
Click to expand
Two-panel imaging demonstrating plantar fascia rupture, a complication of chronic plantar fasciitis or corticosteroid injection. (a) Ultrasound showing thickened hypoechoic plantar fascia with an arrow indicating the site of discontinuity/tear within the degenerative fascia. (b) Sagittal MRI confirming complete rupture with fascial discontinuity and surrounding edema. Rupture typically presents with sudden relief of heel pain followed by arch collapse and new lateral column symptoms.Credit: Open-i (NIH) - CC BY 4.0

Treatment:

  • Conservative: Most ruptures are treated non-operatively
  • Boot immobilization: 4-6 weeks in walking boot
  • Arch support: Custom orthoses to support collapsed arch
  • Activity modification: Avoid high-impact activities for 3-6 months
  • Physical therapy: Strengthen intrinsic foot muscles, peroneal tendons
  • Surgical repair: Rarely needed; consider if complete rupture with severe symptoms

Prognosis:

  • Variable outcomes: Some patients do well, others develop chronic lateral column pain
  • Arch may not fully recover
  • Return to sport often delayed 6-12 months

Postoperative Rehabilitation

Immediate Postoperative Period (0-2 Weeks)

Open release protocol:

  • Weight-bearing: Non-weight-bearing or toe-touch weight-bearing in boot or splint
  • Immobilization: Posterior splint or walking boot
  • Elevation: Keep foot elevated above heart level to reduce swelling
  • Ice: Apply ice 15-20 minutes every 2-3 hours
  • Wound care: Keep dressing clean and dry until suture removal at 10-14 days
  • Pain management: Oral analgesics as needed

Endoscopic release protocol:

  • Weight-bearing: Protected weight-bearing as tolerated in surgical shoe or boot
  • Earlier mobilization: Small incisions allow faster recovery
  • Suture removal: 7-10 days

Rehabilitation Phase (2-6 Weeks)

Progressive weight-bearing:

  • Week 2-4: Gradual increase to full weight-bearing in boot
  • Week 4-6: Transition from boot to supportive athletic shoe

Range of motion:

  • Gentle ankle ROM: Begin at 2 weeks (dorsiflexion, plantarflexion, circumduction)
  • Avoid forced dorsiflexion: To protect healing fascia

Physical therapy:

  • Start formal PT at 2-4 weeks
  • Goals: Restore ROM, begin gentle strengthening, normalize gait

Strengthening Phase (6-12 Weeks)

Progressive strengthening:

  • Intrinsic foot muscles: Towel curls, marble pickups
  • Calf strengthening: Heel raises (bilateral progressing to unilateral)
  • Peroneal strengthening: Resistance band eversion exercises
  • Balance training: Single-leg stance, wobble board

Return to activities:

  • Low-impact activities: Walking, swimming, cycling at 6-8 weeks
  • Gradual progression: 10% increase in activity per week

Return to Sport Phase (3-6 Months)

Sport-specific training:

  • Running: Begin interval training at 3 months if pain-free with walking
  • Jumping and cutting: Delay until 4-6 months
  • Gradual return: Use 10% rule for increasing activity

Maintenance:

  • Stretching: Continue gastrocnemius and plantar fascia stretching indefinitely
  • Footwear: Supportive shoes with arch support
  • Orthoses: May need continued use of arch supports

Full recovery timeline:

  • Open release: 4-6 months for return to full unrestricted activity
  • Endoscopic release: 3-4 months for return to full activity

Outcomes

Natural History and Conservative Outcomes

  • Self-limiting: 80-90% resolve within 12-18 months with conservative treatment
  • 6-month outcomes: 70% improved with structured conservative protocol
  • 12-month outcomes: 85-90% satisfied with conservative management alone

Treatment-Specific Outcomes

TreatmentSuccess RateTime to EffectDuration of Benefit
Stretching + orthoses70-80%6-8 weeksLong-term if maintained
Night splints60-70%4-8 weeksVariable
Corticosteroid injection70-85%1-2 weeks3-6 months
ESWT60-75%6-12 weeks12+ months
PRP injection70-80%8-12 weeks12+ months
Plantar fasciotomy75-90%6-12 weeksLong-term
Gastrocnemius release80-90%3-6 monthsLong-term

Surgical Outcomes

Open plantar fasciotomy:

  • Success rate: 75-90%
  • Patient satisfaction: 80-85%
  • Complications: 10-15% (nerve injury, lateral column overload)
  • Return to full activity: 3-6 months

Endoscopic plantar fasciotomy:

  • Success rate: 80-90%
  • Faster recovery: 4-8 weeks to regular footwear
  • Lower complication rate: 5-10%

Prognostic Factors

Favorable:

  • Duration less than 6 months
  • BMI less than 30
  • No systemic inflammatory disease
  • Good compliance with stretching

Unfavorable:

  • Bilateral symptoms
  • Workers' compensation claims
  • Obesity (BMI greater than 35)
  • Inflammatory arthropathy
  • Previous failed surgery

Evidence Base and Guidelines

Level I
📚 Cochrane Review: Interventions for plantar heel pain
(2020)
Key Findings:
  • Stretching exercises, particularly plantar fascia-specific stretching, effective for short-term pain relief
  • Moderate evidence supports use of orthoses, night splints, and extracorporeal shockwave therapy
  • Limited long-term benefit from corticosteroid injections beyond 3-6 months
  • Conservative management remains cornerstone of treatment

Level I
📚 RCT: Plantar fascia-specific stretching vs Achilles tendon stretching
(2006)
Key Findings:
  • Plantar fascia-specific stretching superior to standard Achilles stretching for pain relief
  • 82% in fascia stretch group versus 52% in Achilles group reported improvement at 8 weeks
  • Functional improvement significantly better with fascia-specific stretching
  • Established plantar fascia stretching as cornerstone of conservative treatment

Level I
📚 Meta-analysis: Extracorporeal shockwave therapy for plantar fasciitis
(2018)
Key Findings:
  • ESWT significantly reduces pain and improves function compared to placebo
  • Effect size moderate across multiple randomized controlled trials
  • Most effective in patients with symptoms over 6 months who have failed conservative measures
  • Low to medium energy protocols (0.1-0.2 mJ/mm²) preferred

Level II
📚 Prospective cohort: Natural history of plantar fasciitis
(2007)
Key Findings:
  • 80% of patients treated conservatively reported resolution of symptoms by 12 months
  • Only 5% progressed to surgical intervention
  • Most improvement occurred in first 3 months of conservative treatment
  • Supports conservative management as first-line approach

Level III
📚 Systematic review: Plantar fascia release outcomes
(2019)
Key Findings:
  • Surgical release effective in 70-90% of carefully selected patients
  • Required failure of comprehensive conservative treatment for 6-12 months
  • Endoscopic and open techniques show similar outcomes in experienced hands
  • Lateral column overload is main complication from excessive release (over 50%)
  • Partial release (medial 50%) essential to prevent complications

Clinical Practice Guidelines

American Academy of Orthopaedic Surgeons (AAOS) Recommendations:

  • Diagnosis: Clinical diagnosis based on history and examination; imaging reserved for atypical cases
  • Initial treatment: Stretching exercises, orthoses, and activity modification
  • Moderate evidence: Supports night splints, ESWT, and short-term steroid injection
  • Surgery: Only after minimum 6 months of conservative treatment failure

American College of Foot and Ankle Surgeons (ACFAS) Guidelines:

  • Conservative first: 90% resolve with conservative management by 12 months
  • Injection therapy: Limit corticosteroid injections to maximum of 2-3 due to rupture risk
  • Surgical indications: Failed 6-12 months of comprehensive conservative treatment
  • Partial release: Release medial 50% only to prevent lateral column overload

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOModerate

Scenario 1: Classic Presentation and Initial Management

EXAMINER

"A 48-year-old female school teacher presents with 3 months of left heel pain. She describes severe pain with the first steps in the morning that improves after walking for 10 minutes but returns after prolonged standing at work. She is otherwise healthy with BMI of 32. Examination reveals exquisite tenderness at the medial calcaneal tubercle. Ankle dorsiflexion is 5 degrees with knee extended. How would you manage this patient?"

EXCEPTIONAL ANSWER
This is a classic presentation of plantar fasciitis - a degenerative condition of the plantar fascia origin characterized by first-step pain. My approach would be systematic: First, I would confirm the diagnosis clinically. The history of first-step pain worse in the morning, improvement with initial activity, and point tenderness at the medial calcaneal tubercle are pathognomonic. I would perform a windlass test - passive dorsiflexion of the hallux which tightens the fascia and reproduces pain. The limited ankle dorsiflexion (5 degrees) is a significant risk factor as tight gastrocnemius increases plantar fascia strain. I would reassure the patient that 90% of cases resolve with conservative management within 12 months. My initial management would include: 1. **Stretching program**: This is the cornerstone of treatment. I would teach plantar fascia-specific stretching - pulling toes back toward shin while seated, holding 10 seconds, repeating 10 times before first steps and after sitting. Also gastrocnemius stretching against a wall, 30 seconds, 3 times, 3 times daily. Evidence shows fascia-specific stretching is superior to calf stretching alone. 2. **Activity modification**: Avoid barefoot walking, use supportive footwear at all times, take regular sitting breaks at work. Consider a cushioned heel cup or over-the-counter arch support. 3. **Weight reduction**: Target BMI under 30, which would reduce load on the fascia. 4. **Ice massage**: Apply ice for 15 minutes, 3-4 times daily for pain relief. 5. **NSAIDs**: Short 2-week course for symptom relief, acknowledging limited long-term benefit since this is a degenerative not inflammatory process. I would review at 6 weeks and, if inadequate improvement, escalate to physical therapy, night splints, and potentially custom orthoses. If symptoms persist beyond 3 months, I would consider ESWT or a single corticosteroid injection, though I would counsel about rupture risk with steroids. Surgery would only be considered after 6-12 months of failed comprehensive conservative treatment.
KEY POINTS TO SCORE
Emphasize diagnosis is clinical - first-step pain and medial tubercle tenderness are pathognomonic
Reassure 90% resolve with conservative care - set realistic timeline of 6-12 months
Stretching is the cornerstone - both plantar fascia-specific and gastrocnemius stretching
Address modifiable risk factors - tight calf (5 degrees dorsiflexion) and obesity (BMI 32)
Stepwise escalation - simple measures first, injections and ESWT reserved for refractory cases
COMMON TRAPS
✗Ordering imaging immediately - diagnosis is clinical; X-rays only if atypical or failed treatment
✗Early steroid injection - this should be reserved for cases failing initial conservative care due to rupture risk
✗Focusing on heel spur - spurs are incidental findings in 50%, not the cause of pain
✗Promising quick cure - must set realistic expectations of 6-12 month treatment course
LIKELY FOLLOW-UPS
"Q: What if she has already failed 6 months of conservative treatment including PT, orthoses, and stretching? A: I would ensure all conservative measures have been tried including night splints and ESWT. If truly refractory, I would obtain MRI to rule out alternative diagnoses like Baxter nerve entrapment, stress fracture, or tumor. If MRI confirms isolated plantar fasciitis, I would discuss surgical options - partial plantar fascia release of the medial 50% only, with expectation of 70-90% success rate and 3-6 month recovery."
"Q: She asks about steroid injection - what do you tell her? A: I would explain that corticosteroid injection can provide short-term pain relief, often within days to weeks, but does not provide long-term benefit beyond 3-6 months. There is a 5-10% risk of plantar fascia rupture with injection, which can cause sudden arch collapse and lateral column pain that may be worse than the original problem. I would use injection sparingly - maximum 2 injections - and prefer to try other conservative measures first."
"Q: How do you perform the windlass test? A: With the patient standing or sitting, I passively dorsiflex the hallux (great toe) while palpating the plantar fascia along the medial arch. This tightens the fascia through the windlass mechanism. Reproduction of pain at the medial calcaneal origin is a positive test confirming plantar fascia as the pain source."
VIVA SCENARIOStandard

Scenario 2: Failed Conservative Treatment - Surgical Candidate

EXAMINER

"A 52-year-old recreational runner presents with 18 months of right heel pain despite comprehensive conservative treatment including PT, custom orthoses, night splints, two cortisone injections, and ESWT. Pain significantly limits his running and daily activities. MRI shows plantar fascia thickening to 7mm with no other pathology. He is motivated and understands recovery time. Would you offer surgery, and if so, what procedure?"

EXCEPTIONAL ANSWER
This patient meets strict criteria for surgical intervention - he has failed comprehensive conservative treatment for 18 months, has confirmed diagnosis on MRI with no alternative pathology, and is motivated with realistic expectations. However, I would still approach this carefully. First, I would review his conservative treatment in detail to ensure it was truly comprehensive. This means: - Daily plantar fascia-specific and gastrocnemius stretching for at least 6 months - Trial of both prefabricated and custom orthoses - Night splints used for at least 8 weeks (compliance is key) - Adequate ESWT protocol (2000-4000 shocks per session, typically 3 sessions) - Formal physical therapy - Activity modification If all measures have been exhausted and symptoms significantly impair his quality of life, I would offer surgery. My preferred procedure would be **partial plantar fascia release** - releasing the medial 50% of the fascia only. **Surgical options discussion:** I would discuss both open and endoscopic approaches: **Open release** - through a 3-4cm medial incision, I can directly visualize and release the medial half of the plantar fascia from the medial calcaneal tubercle. This also allows me to decompress the Baxter nerve if there is any suspicion of nerve entrapment. The advantage is direct visualization and ability to address nerve. Recovery is slightly longer - non-weight-bearing for 2 weeks, then progressive weight-bearing in a boot. Return to running typically 4-6 months. **Endoscopic release** - using medial and lateral 1cm portals, I can release the medial 50% of the fascia with smaller incisions, less soft tissue disruption, and potentially faster recovery. Weight-bearing as tolerated in a boot with return to running at 3-4 months. However, there is a learning curve, and I cannot address Baxter nerve endoscopically. For this patient, given no suggestion of nerve entrapment on history (no burning pain, night pain, or radiation), I would offer endoscopic release as my preference for faster recovery. However, I would be comfortable with either approach. **Critical surgical principle**: I would emphasize that I will release only the **medial 50%** of the fascia. Complete release causes lateral column overload, arch collapse, and can result in worse pain than the original problem. This is the most important technical point. I would counsel realistic expectations: - **Success rate**: 70-90% achieve good to excellent pain relief - **Recovery**: 3-4 months to return to running (endoscopic) or 4-6 months (open) - **Risks**: Infection, nerve injury, continued pain (10-30%), lateral column overload if over-released - **Not guaranteed cure**: 10-30% continue to have some symptoms Finally, I would ensure he understands postoperative rehabilitation is critical - must comply with weight-bearing restrictions, PT, and gradual return to running protocol.
KEY POINTS TO SCORE
Confirm strict surgical criteria met - 12+ months comprehensive conservative treatment failed
Imaging to rule out alternative diagnosis - MRI shows isolated plantar fasciitis
Both open and endoscopic are valid options - discuss pros/cons of each
Critical principle: partial release (medial 50%) only - complete release causes lateral overload
Realistic expectations - 70-90% success, 3-6 month recovery, not guaranteed cure
COMMON TRAPS
✗Offering surgery too early - must document failure of ALL conservative measures for 6-12 months minimum
✗Complete plantar fascia release - this causes lateral column overload and arch collapse
✗Routinely excising heel spur - spur is incidental, does not need to be removed
✗Promising 100% cure - 10-30% continue to have some symptoms even after surgery
✗Ignoring Baxter nerve - if patient has burning pain or radiation, consider nerve entrapment and need for open decompression
LIKELY FOLLOW-UPS
"Q: Describe your open surgical technique. A: I position the patient supine with a thigh tourniquet and bump under the ipsilateral hip. I make a 3-4cm medial longitudinal incision centered over the medial calcaneal tubercle, avoiding the plantar surface. I incise skin and subcutaneous tissue, then identify and retract the abductor hallucis plantarly to expose the plantar fascia. The fascia appears as a thick, glistening white structure. I sharply transect the medial 50% of the fascia 1-2cm from the calcaneal origin, palpating to confirm adequate release while preserving the lateral 50%. If Baxter nerve entrapment is suspected, I decompress the first branch of the lateral plantar nerve between the abductor hallucis and quadratus plantae. I do NOT routinely excise the heel spur. After irrigation, I close in layers and apply a posterior splint in neutral."
"Q: What is the most common complication of plantar fascia release? A: The most serious complication is lateral column overload syndrome from excessive release (more than 50% of fascia width). This causes loss of arch support, lateral foot pain, cuboid stress fractures, and peroneal tendinitis. It can be more disabling than the original plantar fasciitis. Prevention is essential - never release more than 50% of the fascia width. Other complications include nerve injury (Baxter nerve), continued heel pain (10-30%), infection, and calcaneal stress fracture."
"Q: How would your approach differ if MRI showed Baxter nerve entrapment? A: If MRI showed edema or mass effect around the first branch of the lateral plantar nerve (Baxter nerve) between the abductor hallucis and quadratus plantae, I would favor an open approach to allow direct visualization and decompression of the nerve. Endoscopic release does not allow nerve decompression. Baxter nerve entrapment can coexist with or mimic plantar fasciitis - symptoms include burning heel pain often radiating laterally, night pain, and positive Tinel sign. I would release the compressing fascia along the nerve course during open plantar fascia release."
VIVA SCENARIOStandard

Scenario 3: Differential Diagnosis - Baxter Nerve Entrapment

EXAMINER

"A 45-year-old male presents with 6 months of medial heel pain that is worse at night and described as burning. Pain radiates to the lateral heel. He has undergone 3 months of PT and stretching without improvement. Examination reveals tenderness at the medial heel but also a positive Tinel sign deep between the abductor hallucis and quadratus plantae. How does this change your management?"

EXCEPTIONAL ANSWER
This presentation raises concern for **Baxter nerve entrapment** (entrapment of the first branch of the lateral plantar nerve) rather than or in addition to plantar fasciitis. The key distinguishing features are: 1. **Burning quality** of pain rather than sharp pain 2. **Night pain** - atypical for plantar fasciitis 3. **Radiation to lateral heel** - following nerve distribution 4. **Positive Tinel sign** deep between abductor hallucis and quadratus plantae Baxter nerve entrapment can occur in isolation or coexist with plantar fasciitis, as both conditions share risk factors like overpronation and prolonged standing. **Baxter nerve anatomy**: The first branch of the lateral plantar nerve (inferior calcaneal nerve) travels between the deep fascia of abductor hallucis and quadratus plantae, then passes inferior to the medial calcaneal tuberosity to supply abductor digiti minimi. It can be compressed at this site by hypertrophied muscle, fascia, or bone spur. **My management approach would differ**: **Diagnostic confirmation**: - **MRI**: To assess for edema or mass effect around Baxter nerve, thickening of plantar fascia, and rule out other pathology (stress fracture, tumor) - **Nerve conduction studies**: May show denervation of abductor digiti minimi, though limited sensitivity - **Diagnostic injection**: Local anesthetic injection around nerve under ultrasound guidance - temporary relief suggests nerve entrapment **Conservative treatment** (trial first): - **Activity modification**: Reduce aggravating activities - **Orthoses**: Custom orthoses to correct overpronation if present - **Physical therapy**: Nerve gliding exercises, strengthen intrinsic muscles - **NSAIDs**: Limited benefit but trial reasonable - **Corticosteroid injection**: Consider injection around nerve (NOT into nerve) but less effective than for plantar fasciitis **Surgical treatment** (if conservative fails): If conservative treatment fails after 3-6 months and MRI/clinical findings confirm nerve entrapment, I would offer **surgical decompression of Baxter nerve**: - **Approach**: Open medial approach (cannot decompress endoscopically) - **Technique**: Release deep fascia of abductor hallucis and identify Baxter nerve between abductor hallucis and quadratus plantae. Decompress nerve by releasing compressing fascia along its course to the lateral border of the medial calcaneal tuberosity. - **Combined procedure**: If plantar fasciitis also present, combine with partial (medial 50%) plantar fascia release **Prognosis**: - Nerve decompression has good success rates (70-85%) when diagnosis is correct - Recovery slower than plantar fascia release - 4-6 months - Residual numbness along lateral heel may persist **Key point for examiners**: Baxter nerve entrapment is an important differential for heel pain that is often missed. Clues are burning pain, night pain, radiation to lateral heel, and positive Tinel sign deep in the medial heel. MRI can help confirm but diagnosis is largely clinical.
KEY POINTS TO SCORE
Recognize atypical features suggesting nerve entrapment - burning pain, night pain, radiation
Baxter nerve is first branch of lateral plantar nerve compressed between abductor hallucis and quadratus plantae
Can coexist with plantar fasciitis - not mutually exclusive diagnoses
MRI helpful to assess for nerve edema and rule out alternative diagnoses
Surgical decompression requires open approach - cannot be done endoscopically
COMMON TRAPS
✗Missing the diagnosis - assuming all medial heel pain is plantar fasciitis without considering differentials
✗Not asking about night pain or burning quality - key clues to nerve entrapment
✗Attempting endoscopic release - Baxter nerve decompression requires open approach for visualization
✗Injecting INTO the nerve - injection should be around nerve for diagnostic purposes, not into nerve which can cause injury
✗Giving up after conservative treatment fails - nerve decompression can be very successful when diagnosis is correct
LIKELY FOLLOW-UPS
"Q: What other differentials would you consider for heel pain? A: Important differentials include: (1) Tarsal tunnel syndrome - posterior tibial nerve compression posterior to medial malleolus with positive Tinel sign and radiation to plantar foot; (2) Calcaneal stress fracture - pain with medial-lateral squeeze, history of increased activity; (3) Fat pad atrophy - diffuse heel pain, may follow steroid injections; (4) Inflammatory arthritis - bilateral symptoms, morning stiffness over 30 minutes, systemic features; (5) Sever disease in children - calcaneal apophysitis; (6) Tumor or infection - night pain, progressive symptoms, systemic features."
"Q: How do you differentiate Baxter nerve from tarsal tunnel on examination? A: Tarsal tunnel syndrome involves compression of the posterior tibial nerve or its branches posterior to the medial malleolus. Tinel sign is positive POSTERIOR to the medial malleolus, and symptoms include pain and paresthesias radiating to the plantar foot in the distribution of medial and lateral plantar nerves. Baxter nerve entrapment has Tinel sign more distally and plantarly - deep between abductor hallucis and quadratus plantae at the medial heel. Pain is more localized to medial and lateral heel rather than diffuse plantar foot. Tarsal tunnel often has night pain and numbness more prominently than Baxter entrapment."
"Q: Describe the anatomy of Baxter nerve. A: The Baxter nerve is the first branch of the lateral plantar nerve (also called the inferior calcaneal nerve). It arises from the lateral plantar nerve distal to the tarsal tunnel and travels anteriorly between the deep fascia of abductor hallucis (superiorly) and quadratus plantae (inferiorly). It then passes along the medial border of the calcaneus, deep to the medial calcaneal tuberosity, before turning laterally to supply the abductor digiti minimi muscle. The nerve can be compressed at several points: (1) between abductor hallucis and quadratus plantae, (2) at the medial calcaneal tuberosity by bone spurs or hypertrophied fascia, or (3) at the origin of abductor digiti minimi."

MCQ Practice Points

Windlass Mechanism

Q: What is the windlass mechanism and how does it relate to plantar fasciitis?

A: Passive toe dorsiflexion tightens the plantar fascia, raising the medial longitudinal arch and converting the foot to a rigid lever for push-off. This mechanism creates repetitive tension at the calcaneal origin, predisposing to degeneration. Dorsiflexion of the great toe reproduces pain (windlass test).

Conservative Management

Q: What percentage of plantar fasciitis cases resolve with conservative management and what is the recommended duration before considering surgery?

A: 90% resolve with conservative management within 12 months. A minimum of 6-12 months of conservative treatment is mandatory before considering surgical intervention. Conservative measures include stretching, orthoses, night splints, and activity modification.

Heel Spur Significance

Q: What is the clinical significance of a calcaneal heel spur in a patient with plantar heel pain?

A: Heel spurs are an incidental finding present in 50% of asymptomatic individuals. They are NOT the cause of pain and do not correlate with symptom severity. The spur originates from the flexor digitorum brevis, not the plantar fascia. Spur excision is NOT routinely performed with plantar fascia release.

Surgical Release Principles

Q: In plantar fascia release surgery, what percentage of the fascia should be released and why?

A: Release the medial 50% ONLY. Complete release causes lateral column overload with lateral foot pain and arch collapse. The windlass mechanism is eliminated, causing push-off weakness. Partial release preserves some mechanical function while decompressing the medial insertion.

Australian Context

Epidemiology in Australia

  • Prevalence: Affects approximately 10% of the Australian population at some point
  • Primary care presentations: One of the most common musculoskeletal complaints
  • Peak incidence: 40-60 years of age
  • Risk occupations: Prolonged standing (retail, healthcare, hospitality)

Australian Healthcare Access

Primary care:

  • General practitioner first point of contact for most patients
  • Podiatrists widely accessible for orthotic prescription and conservative management
  • Physiotherapy covered under various health insurance plans

Specialist referral:

  • Sports medicine physicians for complex cases
  • Orthopaedic foot and ankle surgeons for refractory cases
  • Average wait time for public orthopaedic review: 6-12 months

RACS/AOA Guidelines Alignment

  • Conservative care first: Minimum 6 months conservative treatment recommended before surgical consideration
  • Multimodal approach: Combination of stretching, orthoses, and activity modification as first-line
  • Evidence-based interventions: PRP and ESWT considered when available for refractory cases

WorkCover Considerations

  • Plantar fasciitis may be compensable if work-related (prolonged standing, hard surfaces)
  • Detailed documentation of occupational factors required
  • Return-to-work programs should include workplace modifications
  • Typical time off work: 1-2 weeks for injection, 6-12 weeks post-surgery

PLANTAR FASCIITIS MANAGEMENT

High-Yield Exam Summary

Key Definitions

  • •**Plantar fasciitis**: Degenerative condition of plantar fascia origin causing inferior heel pain - prefer term plantar fasciosis or plantar heel pain
  • •**Windlass mechanism**: Passive toe extension tightens fascia and raises arch - basis for windlass test
  • •**Baxter nerve**: First branch of lateral plantar nerve between abductor hallucis and quadratus plantae - can mimic plantar fasciitis
  • •**Partial release**: Surgical release of medial 50% of fascia only - complete release causes lateral column overload

Epidemiology Numbers

  • •**10%** lifetime incidence in population
  • •**40-60 years** peak age of onset
  • •**90%** resolve with conservative management within 12 months
  • •**4mm** normal plantar fascia thickness on ultrasound (over 4mm in plantar fasciitis)
  • •**50%** have heel spur but it is incidental finding, not cause of pain
  • •**70-90%** success rate with surgical release in properly selected patients

Risk Factors (PLANTAR)

  • •**P** - Prolonged standing/walking occupations
  • •**L** - Limited ankle dorsiflexion (less than 10 degrees)
  • •**A** - Age 40-60 years
  • •**N** - No arch support (pes planus or cavus)
  • •**T** - Training errors in runners
  • •**A** - Adiposity (BMI over 30)
  • •**R** - Reduced first step pain (classic symptom)

Clinical Diagnosis

  • •**First-step pain**: Severe pain with first steps in morning or after sitting (95% sensitive)
  • •**Point tenderness**: Medial calcaneal tubercle tenderness (most specific finding)
  • •**Windlass test**: Passive hallux dorsiflexion reproduces pain - positive test confirms fascia as source
  • •**Limited dorsiflexion**: Less than 10 degrees with knee extended is risk factor
  • •**Red flags**: Night pain, burning pain, radiation suggest alternative diagnosis (nerve entrapment)

Conservative Treatment Stages

  • •**0-6 weeks (Acute)**: Stretching (fascia-specific and gastrocnemius), ice, NSAIDs, activity modification, supportive footwear
  • •**6 weeks-3 months (Subacute)**: Add PT, night splints, orthoses, consider ESWT
  • •**3-6 months (Chronic)**: ESWT (2000-4000 shocks), consider steroid injection (max 2 due to rupture risk)
  • •**6-12 months (Refractory)**: Repeat injection or trial PRP, rule out alternative diagnoses with MRI
  • •**Over 12 months**: Consider surgery if failed ALL conservative measures

Injection Therapies

  • •**Steroid**: Short-term relief (1 month) but no long-term benefit; 5-10% rupture risk; max 2 injections; use medial approach to avoid fat pad
  • •**PRP**: Slower onset but better long-term outcomes vs steroid at 3-6 months; lower rupture risk; 2-3 mL under ultrasound guidance
  • •**Technique**: Medial approach under ultrasound guidance to visualize fascia and avoid fat pad injection

Surgical Indications (Strict)

  • •**Failed 6-12 months** of comprehensive conservative treatment
  • •**All measures tried**: Stretching, PT, orthoses, night splints, ESWT, at least one injection
  • •**Imaging confirmation**: MRI to rule out alternative diagnosis if needed
  • •**Significant impairment**: Pain limiting ADLs and quality of life
  • •**Motivated patient**: Understands recovery time and realistic expectations

Surgical Technique Principles

  • •**Partial release only**: Release medial 50% of fascia - NEVER complete release
  • •**Complete release complication**: Causes lateral column overload, arch collapse, worse pain than original problem
  • •**Open approach**: 3-4cm medial incision, visualize and release medial half, allows Baxter nerve decompression
  • •**Endoscopic**: Medial and lateral 1cm portals, faster recovery but cannot address nerve
  • •**Heel spur**: Do NOT routinely excise - incidental finding not requiring removal
  • •**Baxter nerve**: Decompress if burning pain, night pain, or positive Tinel sign suggesting entrapment

Surgical Outcomes

  • •**Success**: 70-90% good to excellent outcomes in properly selected patients
  • •**Recovery**: 3-4 months return to running (endoscopic) or 4-6 months (open)
  • •**Continued pain**: 10-30% have residual symptoms despite surgery
  • •**Complications**: Lateral column overload (10-15% if over-released), nerve injury, infection, continued pain

Differential Diagnosis

  • •**Baxter nerve entrapment**: Burning pain, night pain, radiation to lateral heel, Tinel sign deep between abductor hallucis and quadratus plantae
  • •**Tarsal tunnel**: Posterior tibial nerve compression, Tinel posterior to medial malleolus, plantar foot radiation
  • •**Calcaneal stress fracture**: Pain with medial-lateral squeeze, history of increased activity
  • •**Fat pad atrophy**: Diffuse heel pain, history of steroid injections, no focal tenderness
  • •**Inflammatory arthritis**: Bilateral symptoms, morning stiffness over 30 minutes, systemic features

Exam Viva Pearls

  • •**Plantar fasciosis not fasciitis**: Histology shows degeneration (myxoid, collagen necrosis) not inflammation
  • •**Heel spur is incidental**: Present in 50% of plantar fasciitis but also 15-25% of asymptomatic people - not the cause
  • •**Fascia rupture presentation**: Sudden pop, immediate relief of heel pain, arch collapse, bruising, new lateral column pain
  • •**50% rule**: Never release more than 50% of fascia width to prevent lateral column overload
  • •**Baxter nerve anatomy**: First branch of lateral plantar nerve between abductor hallucis and quadratus plantae - important differential
  • •**Conservative is key**: 90% resolve without surgery - set realistic 6-12 month timeline expectations

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