Tibiotalar fusion — open (anterior/transfibular) or arthroscopic | intermediate
- Ideal fusion position is 0° plantarflexion/dorsiflexion (neutral), 5° hindfoot valgus, 5–10° external rotation matching the contralateral limb, and mild posterior translation of the talus under the tibia — malposition in equinus or varus is the leading cause of poor functional outcome.
- Townshend 2013 multicentre comparative case series (30 arthroscopic vs 30 open) showed greater early improvement in the Ankle Osteoarthritis Scale and a shorter hospital stay with arthroscopic arthrodesis, with similar complications, operative time and radiographic alignment.
- Adjacent joint degeneration (subtalar, talonavicular, midfoot) is the primary long-term concern after successful fusion — Coester 2001 (22-year follow-up) showed significantly more severe ipsilateral subtalar, talonavicular, calcaneocuboid and midfoot arthritis than the contralateral side.
- Non-union occurs in approximately 5–10% overall (higher with smoking, prior infection, open injury and talar avascular necrosis) — obtain a CT scan to differentiate fibrous union from true non-union.
When & Why
Indication. End-stage tibiotalar (ankle) osteoarthritis — pain, stiffness and progressive loss of function — that has failed a minimum of 3–6 months of conservative care (NSAIDs, physiotherapy, an intra-articular corticosteroid injection and an ankle-foot orthosis). The arthritis may be primary, post-traumatic (the commonest cause, for example after a pilon or ankle fracture treated by ORIF), rheumatoid, Charcot neuroarthropathy, or the result of a failed total ankle replacement. Assess the whole hindfoot before committing. Confirm the disease is isolated to the tibiotalar joint, because concomitant disease changes the plan: - Examine the subtalar and midfoot joints clinically — symptomatic subtalar arthritis may require a tibiotalocalcaneal (TTC) fusion rather than an isolated ankle fusion.
- Obtain a weight-bearing AP and lateral radiograph and a CT to characterise the deformity, assess bone stock and the subtalar joint, and identify retained metalwork that may need removal.
- Gauge deformity magnitude and talar vascularity — arthroscopic fusion is reliable only when coronal/sagittal deformity is minimal (generally less than 15°) and there is no talar avascular necrosis. Absolute indications - End-stage tibiotalar osteoarthritis (primary, post-traumatic, or rheumatoid) with failed conservative management.
- Tibiotalar arthritis following failed ORIF with joint destruction.
- Charcot neuroarthropathy of the ankle with instability or ulcer risk.
- Ankle instability with severe arthritis, or arthritic change after failed ligamentous reconstruction.
- Failed total ankle replacement with inadequate bone stock for revision TAR. Relative indications - Avascular necrosis of the talus with joint involvement.
- Septic arthritis with residual tibiotalar destruction (staged — eradicate infection, then fuse).
- Ankle tumour resection requiring reconstruction with fusion.
- Severe coronal or sagittal deformity not correctable by realignment osteotomy alone. Contraindications - Active deep infection (relative — a staged approach is possible).
- Severe ipsilateral hindfoot or midfoot arthritis limiting compensatory motion.
- Contralateral lower-limb amputation (fusion prevents compensatory gait adaptation).
- Unrealistic patient expectations regarding functional outcome. The one decision that matters — choose the approach. Every ankle fusion prepares the joint and fixes it in the same four-point position; what differs is the exposure and fixation:
Preferred for end-stage OA without significant deformity (less than 15°) or talar AVN. Two or three percutaneous cannulated screws. Faster union, fewer wound problems, shorter hospital stay (Townshend 2013).
Required for deformity greater than 15°, Charcot, bone loss needing graft, or revision. Allows differential wedge resection; anterior locking plate or transfibular onlay graft.
For combined ankle and subtalar disease, severe Charcot, revision, or elderly osteoporotic bone. Axial compression and rotational stability across both joints.
A motion-preserving total ankle replacement is the alternative for the older, low-demand patient with preserved subtalar motion, near-normal alignment and good bone stock — the fusion-versus-TAR decision is covered under Background & Evidence. Consent specifically for sural or superficial peroneal numbness/dysaesthesia, anterior wound problems (especially in diabetes and Charcot), non-union (about 5–10%, higher if smoking continues), malposition requiring revision, accelerated adjacent-joint arthritis over decades, and CRPS. Setup. Supine with a sandbag under the ipsilateral hip to bring the foot into neutral rotation, a padded thigh tourniquet, and the image intensifier on the contralateral side for unobstructed AP and lateral views. Mark the superficial peroneal nerve (tense it with plantarflexion and inversion), the dorsalis pedis pulse, and both malleoli before inflating the tourniquet.
The Operation
The goal is to expose the tibiotalar joint, remove all residual cartilage to bleeding cancellous bone on both surfaces, correct any deformity by differential bone resection, fix the joint in the four-point position, and graft any defects. The exposure — arthroscopic portals or the open anterior/transfibular approach — is laid out as the first operative steps below.



Operative sequence
- Supine with a sandbag under the ipsilateral hip to neutralise rotation; padded thigh tourniquet; image intensifier on the contralateral side for unobstructed AP and lateral views.
- Before inflation, mark the superficial peroneal nerve (tense it with plantarflexion and inversion), the dorsalis pedis pulse, and the medial and lateral malleoli.
- Inflate the tourniquet to 100 mmHg above systolic; deflate before closure to secure haemostasis, especially before applying an anterior plate.
- Anteromedial portal: just medial to the tibialis anterior tendon, about 1 cm above the joint line. Use an 18-gauge needle first to confirm intra-articular position, skin incision only, blunt dissection to the capsule, then introduce a 4.0 mm 30° arthroscope.
- Anterolateral portal: lateral to the EHL tendon at the same joint-line level, created under transillumination — this is the working portal for the shaver and burr. Dorsiflex the foot to open the anterior compartment.
- Posterolateral accessory portal: 1 cm above the fibula tip, just posterior to it — avoid the sural nerve posteriorly; used for posterior gutter débridement.
- Safety: the superficial peroneal nerve's dorsal branch crosses the anterolateral portal — plantarflex and invert the foot to reveal it before marking.
- Longitudinal incision 10–12 cm over the anterior ankle, lateral to the tibialis anterior tendon, deepened through the deep fascia in the EHL–TA interval.
- Identify and protect the dorsalis pedis artery and the deep peroneal nerve (lying immediately lateral to EHL) — mobilise and retract them medially with EHL before any bone work.
- Divide the ankle capsule transversely, elevate the anterior periosteum, and clear the anterior gutter fat pad to expose both joint surfaces.
- Transfibular option: osteotomise the distal fibula 5–7 cm above the joint line, reflect it distally on its soft-tissue attachments for extensile access to a large deformity, and reuse it as onlay bone graft.
- Systematic progression with a 4.0 mm full-radius shaver then a 4.0 mm abrader burr: anterior gutter and anterior osteophytes, central tibial plafond (medial-to-lateral sweeps), medial and lateral gutters, posterior tibial surface (needs the posterolateral portal or curved instruments), then the full talar dome.
- Débride every surface to bleeding cancellous bone — complete removal of all residual cartilage is mandatory for union.
- Pearl: drill multiple 5 mm-deep holes through the subchondral plate with a 2.0 mm drill or K-wire (the fish-scale or Russian technique) to multiply the bleeding surface area.
- For the open technique, resect the plafond and talar dome with a sagittal saw (flat cuts) or cup-and-cone reamers to preserve length and maximise contact; use a femoral distractor or large laminar spreader to reach the posterior tibial margin, which cannot be débrided without distraction.
- Correct coronal and sagittal deformity by differential bone resection before fixation — wedge resections of the tibial plafond or talar dome correct varus/valgus and equinus.
- Aim for the final four-point position (Step 6); retain as much bone length as possible, because over-resection shortens the limb and increases strain on adjacent joints.
- Sagittal plane — 0° (neutral dorsiflexion): aligns joint reaction force through the tibia. Even 5° of equinus shifts loading to the forefoot, increases knee flexion demand and reads as a functional leg-length shortening.
- Coronal plane — 5° hindfoot valgus: preserves the subtalar axis of compensation. Varus is poorly tolerated, causing lateral-border overload, peroneal tendon stress and sural nerve symptoms.
- Axial rotation — 5–10° external rotation matching the contralateral limb (compare the second-toe axis).
- Sagittal translation — mild posterior shift of the talus under the tibia: shortens the anterior lever arm and reduces midfoot stress in terminal stance.
- Confirm on AP and lateral fluoroscopy, then hold the position with two 2.0 mm percutaneous K-wires across the joint before definitive fixation.
- Standard two-screw construct: a medial malleolar screw (6.5 or 7.3 mm cannulated partially threaded) from the medial face of the medial malleolus directed laterally and slightly anteriorly into the talar body, and a lateral screw from the fibula or anterolateral distal tibia directed medially into the talar body or neck. The threads must fully cross the joint line and purchase the talus; keep the trajectory clear of the subtalar joint.
- Optional third anterior screw: from the anterior distal tibia directed posteriorly across the joint into the talus — adds rotational control, most useful in revision or high-demand patients.
- Anterior blade/locking plate: applied to the anterior tibia after retracting the neurovascular bundle — multiplanar stability, preferred for Charcot arthropathy or bone loss.
- Retrograde tibiotalocalcaneal nail: for combined ankle and subtalar fusion, severe Charcot, revision, or elderly osteoporotic bone.
- Pack autograft (iliac crest or local cancellous bone from reamed cavities) into any gaps; consider demineralised bone matrix or synthetic graft to augment.
- Bone graft is mandatory in revision cases and in talar avascular necrosis.
- Copious irrigation; close the deep fascial layer without tension, then subcutaneous tissue and skin over a suction drain (removed at 24–48 hours).
- In high-risk wounds (diabetic, Charcot, anterior approach) consider leaving the deep fascia open or performing delayed primary closure at 48–72 hours.
- Final fluoroscopy: AP and lateral views to confirm screw position, compression and correct fusion alignment.
The sural nerve runs posterior to the lateral malleolus with the short saphenous vein; the superficial peroneal nerve pierces the deep fascia 10–12 cm above the malleolus and crosses the anterolateral portal; the dorsalis pedis artery and deep peroneal nerve lie between EHL and EDL. Mark the superficial peroneal nerve pre-operatively, identify and retract the dorsalis pedis and deep peroneal nerve medially in the anterior approach, and confirm the four-point fusion position on AP and lateral fluoroscopy before driving the definitive screws — equinus or varus left at this moment is the commonest cause of a poor outcome.
Dorsiflex the foot to open the anterior compartment for arthroscopic access, and use a femoral distractor or large laminar spreader for the open technique. The posterior tibial margin cannot be débrided without adequate distraction, and cartilage left posteriorly is a classic cause of non-union.
Reserve arthroscopic fusion for deformity less than 15° and no talar avascular necrosis. Above that, or in Charcot, revision, or bone loss, an open (anterior or transfibular) approach is needed to correct alignment and graft defects — Zvijac's single non-union had extensive talar avascular necrosis.
Aftercare & Complications
Rehabilitation | Phase | Timing | Immobilisation & weight-bearing | Milestones | |-------|--------|--------------------------------|------------| | 1 | 0–2 weeks | Below-knee backslab, strict non-weight-bearing, elevation above heart level for 48–72 h | Wound check at 48–72 h; drain out at 24–48 h | | 2 | 2–6 weeks | Circumferential below-knee cast; remain non-weight-bearing (Charcot with a nail: protected weight-bearing in a total contact cast from week 2) | Suture removal at 2 weeks | | 3 | 6–12 weeks | CAM boot with progressive weight-bearing if radiographs show bridging; full weight-bearing in the boot by 8–10 weeks | Boot-to-shoe transition at 10–12 weeks; CT at 12 weeks if union uncertain | | 4 | 3–6 months | Supportive footwear with a custom orthosis for the first 6 months | Driving at about 12 weeks; sedentary work 6–8 weeks; light manual 12–16 weeks; heavy manual about 6 months; low-impact sport 4–6 months | Because the ankle is fused, rehabilitation focuses on hip and knee strengthening, balance and proprioception (eyes-closed single-leg stance) and gait retraining, not ankle range of motion. A popliteal nerve block gives 12–24 hours of effective analgesia and reduces early opioid requirements. Union assessment. Declare union when at least three cortices are bridged on plain radiograph, the fusion site is pain-free on stress, and the patient is progressing to full weight-bearing. CT at 12 weeks is the gold standard when the radiograph is equivocal — plain films significantly underestimate non-union. Follow up at 2 weeks (wound), 6 weeks (radiograph and weight-bearing decision), 12 weeks (CT if union uncertain, boot-to-shoe), 6 months (final union, orthosis review) and 12 months, then annually to screen adjacent joints. Review urgently for increasing pain and swelling beyond the expected course, wound breakdown or discharge, worsening neurology, or hardware failure on radiograph. Complications
- Incidence
- 5–10% overall, up to 40% in Charcot, smoking, talar AVN or revision; CT differentiates fibrous from true non-union
- Prevention
- Débride both surfaces to bleeding cancellous bone; correct position; graft gaps over 5 mm; rigid fixation; mandate smoking cessation
- Management
- Pain-free fibrous union: observe. True non-union: revise — refresh surfaces, iliac crest autograft, anterior plate or retrograde nail; optimise diabetes and enforce smoking cessation
- Incidence
- 5–8% clinically significant; under 5° often tolerated, over 10° impairs function
- Prevention
- Confirm the four-point position on AP and lateral fluoroscopy before final screws; compare rotation to the other side; watch for equinus creep on screw insertion
- Management
- Functional equinus under 10°: heel raise and shoe modification. Symptomatic: corrective osteotomy through the fusion mass with wedge resection and re-fixation
- Incidence
- Wound complications 5–15% (higher with anterior approach, diabetes, Charcot); deep infection 2–4%
- Prevention
- Full-thickness flaps; tension-free closure; deflate tourniquet to check perfusion; delayed primary closure at 48–72 h in high-risk; cefazolin prophylaxis
- Management
- Superficial: wound care, antibiotics, VAC. Deep with stable hardware: debride, washout, IV antibiotics, retain until union then remove. Chronic with failed union: two-stage exchange
- Incidence
- Superficial peroneal nerve 3–8% (arthroscopic); sural nerve 2–5%; vascular injury rare but devastating
- Prevention
- Mark the superficial peroneal nerve pre-operatively (plantarflexion-inversion test); identify and retract the dorsalis pedis and deep peroneal nerve medially; avoid portal leverage; plan screw trajectories
- Management
- Neuropraxia: observe. Painful dysaesthesia: desensitisation, gabapentin. Neuroma: excision and proximal burial at 12 months. Dorsalis pedis injury: vascular surgery, possible bypass or flap
- Incidence
- Radiographic progression near-universal at long-term follow-up; a subset becomes symptomatic
- Prevention
- Optimise fusion position (avoid equinus and varus); preserve subtalar motion during débridement; use arthroscopic technique where possible
- Management
- Activity modification, orthosis, injection. Severe subtalar: subtalar fusion (becomes tibiotalocalcaneal). Widespread: triple arthrodesis or extended TTC fusion
- Incidence
- 2–5%; increased by prolonged immobilisation, long tourniquet time and pre-existing neuropathy
- Prevention
- Minimise tourniquet time; early mobilisation within protocol; multimodal analgesia; popliteal block; vitamin C 500 mg daily for 50 days
- Management
- Early diagnosis (Budapest criteria); multidisciplinary care — physiotherapy, sympathetic nerve blocks, gabapentin or amitriptyline, pain psychology; spinal cord stimulation if refractory
Viva & Exam Focus
FVERTFVERT — the ideal fusion position
SCANSCAN — non-union risk factors
Runs posterior to the lateral malleolus with the short saphenous vein, about 1 cm posterior to the fibula. At risk during the lateral approach, posterolateral portal and fibula osteotomy. Identify it in the posterolateral quarter before incising and retract it with the vein. Injury causes painful dysaesthesia over the lateral foot and fifth toe.
Pierces the deep fascia 10–12 cm proximal to the lateral malleolus tip; its dorsal branch runs anterior to the fibula toward the fourth toe and crosses the anterolateral portal. Plantarflex and invert the foot to make it taut and visible, then mark and avoid it. Injury causes dysaesthesia over the dorsum of the foot.
The artery lies between EHL and EDL with the deep peroneal nerve immediately lateral to it. At risk during the anterior approach, anterior plating and aggressive débridement. Identify and retract the bundle medially with EHL; apply any anterior plate lateral to it. Injury causes foot ischaemia or dorsal sensory loss.
The commonest cause of poor functional outcome and revision. Even 5° of equinus equals roughly a 1.5 cm functional leg-length discrepancy and abnormal knee loading; varus overloads the lateral border and stresses the peroneals. Confirm the four-point position on AP and lateral fluoroscopy before definitive fixation, using the contralateral limb as the rotation reference.
The anterior ankle has sparse soft-tissue cover and a tenuous blood supply, so dehiscence and deep infection are serious risks, especially in diabetic and Charcot patients. Raise full-thickness flaps, close without tension, deflate the tourniquet to check perfusion, and consider staged closure or a local flap in high-risk patients. Early aggressive management prevents limb-threatening deep infection.
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
“A 48-year-old construction worker presents with 3 years of right ankle pain following a pilon fracture treated with ORIF 5 years ago. He has failed 12 months of NSAIDs, physiotherapy, intra-articular steroid injections, and an ankle-foot orthosis. Weight-bearing AP and lateral radiographs show complete tibiotalar joint space loss with subchondral sclerosis and a 10° varus deformity. He smokes 20 cigarettes per day. What is your assessment and plan?”
“A 58-year-old woman had an ankle arthrodesis 8 months ago. She never became pain-free. CT scan shows no osseous bridging across the tibiotalar joint. She has fibrous union. What are the risk factors for non-union, how do you investigate this patient, and what is your management?”
“A 62-year-old retired teacher has end-stage primary ankle osteoarthritis with mild-to-moderate tibiotalar joint space loss, 5° valgus deformity, preserved subtalar motion, and no prior ankle surgery. She asks you to compare ankle arthrodesis with total ankle replacement and help her decide. What are the key differences and how would you counsel her?”
Indications
- End-stage tibiotalar OA (primary, post-traumatic, RA) after 3–6 months failed conservative care: NSAIDs, physiotherapy, injection, AFO
- Charcot neuroarthropathy with instability or ulcer risk; failed TAR without bone stock for revision
- Avascular necrosis of the talus, sequalae of septic arthritis, tumour reconstruction (relative indications)
Fusion position — FVERT
- Flat: neutral dorsiflexion (0°) — equinus is the cardinal malposition
- Valgus 5° — matches the hindfoot loading axis
- External rotation 5–10° matching the other limb
- Retro-displacement — mild posterior talar shift under the tibia
- Confirm on AP and lateral fluoroscopy before final fixation
Arthroscopic vs open
- Townshend 2013 (30 vs 30): comparable complications and alignment; greater early AOS gain and shorter stay arthroscopically
- Zvijac 2002: 20/21 (95%) united at mean 8.9 weeks; Ogilvie-Harris 1993: 17/19 united
- Arthroscopic for deformity less than 15° and no talar AVN; open for greater than 15°, Charcot, revision, bone loss
- Retrograde TTC nail for combined ankle and subtalar disease, Charcot or osteoporotic bone
Danger structures
- Sural nerve: posterior to lateral malleolus with short saphenous vein — posterolateral portal and lateral approach
- Superficial peroneal nerve: pierces fascia 10–12 cm above the malleolus — mark with the plantarflexion-inversion test
- Dorsalis pedis artery and deep peroneal nerve: EHL–EDL interval — retract medially in the anterior approach
- Fusion malposition: commonest cause of poor outcome — confirm on fluoroscopy
- Anterior wound: sparse coverage — meticulous handling; delayed closure in diabetic/Charcot
Non-union risk — SCAN
- Smoking roughly doubles non-union risk — enforce cessation pre-operatively
- Contact inadequate: incomplete débridement, sclerotic bone — drill to bleeding cancellous bone
- Avascular necrosis of talus: MRI pre-operatively; longer protection, upgraded fixation
- Non-compliance / Charcot biology; CT is mandatory for union assessment — plain films underestimate non-union
Complications
- Non-union 5–10% (up to 40% high-risk); CT workup, revise with iliac crest graft and plate or nail
- Malunion (equinus/varus) 5–8%; confirm position intra-op; corrective osteotomy if symptomatic
- Wound dehiscence 5–15% (anterior approach, diabetic, Charcot); deep infection 2–4%
- Adjacent joint arthritis: Coester 2001 (22-year) accelerated subtalar/midfoot arthritis — screen annually
- CRPS 2–5%; Budapest criteria; multidisciplinary care; vitamin C 500 mg daily for 50 days
Post-op protocol
- 0–2 weeks: backslab, strict non-weight-bearing, elevation
- 2–6 weeks: cast, non-weight-bearing (Charcot with nail: protected weight-bearing)
- 6–12 weeks: CAM boot, progressive weight-bearing if bridging; CT at 12 weeks if uncertain
- Union: three cortices bridged and pain-free on stress
- Driving about 12 weeks; heavy manual about 6 months
Fusion vs TAR
- Fusion: young high-demand, bone loss, Charcot, severe deformity, infection, failed TAR — about 90% union, durable
- TAR: older low-demand, preserved subtalar motion, near-normal alignment, good bone stock
- TAR survival about 85–90% at 7–10 years (NJR; Clough and Ring 2021)
- Daniels COFAS 2014: comparable PROs but TAR reoperation 17% vs 7%, major complications 19% vs 7%
- TAR conversion to fusion is difficult and inferior to primary fusion
Background & Evidence
Background. End-stage tibiotalar osteoarthritis is most often post-traumatic — after an intra-articular pilon or ankle fracture treated by ORIF — and less often primary, rheumatoid, haemophilic, Charcot, or the result of a failed total ankle replacement. The tibial plafond is slightly wider anteriorly and the talar dome is trapezoidal (wider anteriorly), which is why the dome is unconstrained in plantarflexion and relevant to how the joint surfaces are prepared; the mechanical axis of the tibia lies about 3° of valgus relative to the anatomical axis, informing the desired fusion position. Arthroscopic versus open — the key evidence. Three series establish the arthroscopic technique and its limits. Ogilvie-Harris (1993) reported 19 arthroscopically assisted fusions in ankles with minimal or no deformity fixed with three percutaneous cannulated screws; 17 of 19 united (2 non-unions), with excellent or good results in 16 — establishing the technique for non-deformed arthritic ankles. Zvijac (2002) reported 21 arthroscopically assisted fusions with 20 of 21 (95%) united at a mean of 8.9 weeks, the single failure having extensive talar avascular necrosis (approximately half the talus) — reinforcing AVN and deformity as relative contraindications. Townshend (2013), the comparative benchmark, studied 60 patients (30 arthroscopic, 30 open) over 2 years: both groups improved significantly in the Ankle Osteoarthritis Scale and SF-36 physical component, with significantly greater early improvement and a shorter hospital stay in the arthroscopic group and similar complications, operative time and radiographic alignment. Current recommendation: arthroscopic arthrodesis is preferred for end-stage OA without significant deformity (less than 15°) or talar AVN; open technique is required for major deformity correction, Charcot, bone loss requiring graft, or revision. Long-term outcomes — the price of a successful fusion. Coester (2001) followed 23 patients with isolated post-traumatic ankle arthrodesis a mean of 22 years (the longest follow-up reported) and found ipsilateral subtalar, talonavicular, calcaneocuboid, naviculocuneiform, tarsometatarsal and first MTP arthritis all significantly more severe than the contralateral side (each p less than 0.01), with worse activity limitation, pain and disability — the knee was not significantly affected. Hendrickx (2011) reported 60 patients (66 ankles) fused with a two-incision, three-screw technique: 91% primary union (six required re-arthrodesis), mean AOFAS 67 and 91% satisfaction at a mean 9 years, with significant radiographic progression of arthritis in all adjacent joints. This accelerated adjacent-joint degeneration is the central argument for offering motion-preserving TAR to suitable candidates.
- Ankle arthrodesis
- Young high-demand; bone loss; Charcot; severe deformity; infection history; failed TAR
- Total ankle replacement
- Older low-demand; preserved subtalar motion; near-normal alignment; intact ligaments; good bone stock
- Ankle arthrodesis
- About 90% primary union; permanent pain control, no wear or loosening
- Total ankle replacement
- About 85–90% implant survival at 7–10 years (NJR; Clough and Ring 2021 — 88% Zenith at 7 years)
- Ankle arthrodesis
- None — compensated by subtalar and midfoot joints
- Total ankle replacement
- Preserved plantarflexion–dorsiflexion arc; more natural gait
- Ankle arthrodesis
- Accelerated arthritis long-term (Coester 2001)
- Total ankle replacement
- Theoretically protected by preserved motion
- Ankle arthrodesis
- Lower — about 7%
- Total ankle replacement
- Higher — about 17%; conversion to fusion is difficult and inferior to primary fusion
- Ankle arthrodesis
- Townshend 2013 (arthroscopic vs open); Hendrickx 2011; Patel 2021 (non-union risk)
- Total ankle replacement
- Daniels COFAS 2014; Saltzman STAR 2009; Clough and Ring 2021
References
Arthroscopic versus open ankle arthrodesis: a multicentre comparative case series
- Comparative case series of 60 patients (30 arthroscopic, 30 open) followed for 2 years across two institutions
- Both groups improved significantly in the Ankle Osteoarthritis Scale and SF-36 physical component score at 1 and 2 years
- Arthroscopic group had significantly greater Ankle Osteoarthritis Scale improvement at 1 and 2 years and a shorter hospital stay
- Complications, operative time and radiographic alignment were similar between groups
Analysis of arthroscopically assisted ankle arthrodeses
- Retrospective series of 21 arthroscopically assisted ankle fusions
- 20 of 21 (95%) united at a mean of 8.9 weeks
- The single failure had extensive talar avascular necrosis (approximately half the talus)
- Reinforces talar AVN and major deformity as relative contraindications to the arthroscopic technique
Arthroscopically assisted arthrodesis for osteoarthrotic ankles
- 19 arthroscopically assisted fusions in ankles with minimal or no deformity, fixed with three percutaneous cannulated screws
- 17 of 19 united (2 non-unions), with excellent or good results in 16
- Established the arthroscopic technique as viable for non-deformed arthritic ankles
Long-term results following ankle arthrodesis for post-traumatic arthritis
- 23 patients with isolated, successful post-traumatic ankle fusion followed a mean of 22 years (range 12–44) — the longest follow-up reported
- Ipsilateral subtalar, talonavicular, calcaneocuboid, naviculocuneiform, tarsometatarsal and first MTP arthritis were all significantly more severe than the contralateral side (each p less than 0.01)
- Significantly worse activity limitation, pain and disability on the fused side; the ipsilateral knee was not significantly affected
- Demonstrates accelerated adjacent-joint degeneration distal to the fusion as the price of long-term success
Risk factors for nonunion following ankle arthrodesis: a systematic review and meta-analysis
- Meta-analysis of 13 studies and 987 patients evaluating 37 candidate risk factors for non-union
- Strong evidence: smoking (OR 2.89, 95% CI 1.23–6.76), male sex (OR 1.96) and prior operative-site infection (OR 2.40)
- Moderate evidence: history of open injury (OR 5.95); limited evidence: pre-operative talar avascular necrosis (OR 13.16)
- Confirms smoking roughly doubles non-union risk, supporting mandatory pre-operative cessation
Intermediate-term results of total ankle replacement and ankle arthrodesis: a COFAS multicentre study
- Prospective multicentre cohort of 388 ankles (281 TAR, 107 arthrodesis) reviewed at a mean of 5.5 years
- Ankle Osteoarthritis Scale and SF-36 scores were comparable between TAR and fusion after adjustment for baseline characteristics and surgeon
- Revision was needed in 17% of replacements versus 7% of fusions; major complications 19% versus 7%
- Treatment was tailored to presentation — fusion patients were younger, more often diabetic and more often smokers
Medium- to long-term outcome of ankle arthrodesis
- Single-centre retrospective study of 60 patients (66 ankles) fused with a two-incision, three-screw technique
- Primary union 91% (six required re-arthrodesis to achieve fusion); 91% patient satisfaction
- Mean AOFAS Ankle–Hindfoot 67 and FAAM 69 at a mean follow-up of 9 years; one infection, no other serious adverse events
- Significant radiographic progression of arthritis seen in all contiguous joints
Total ankle replacement and tibiotalocalcaneal references 1. Saltzman CL, Mann RA, Ahrens JE, et al. Prospective controlled trial of STAR total ankle replacement versus ankle fusion: initial results. Foot Ankle Int. 2009;30(7):579–596. PMID 19589303. Non-randomised multicentre controlled (concurrent fusion controls) non-inferiority trial; equivalent pain relief and better function with STAR TAR at 24 months, with more secondary procedures. 2. Clough TM, Ring J. Total ankle arthroplasty. Bone Joint J. 2021;103-B(4):696–703. PMID 33789488. Single-centre series of 118 Zenith TAAs with 88% implant survival at 7 years, comparable to National Joint Registry data. 3. Myerson MS, Alvarez RG, Lam PW. Tibiocalcaneal arthrodesis for the management of severe ankle and hindfoot deformities. Foot Ankle Int. 2000;21(8):643–650. Technique description and outcomes for tibiotalocalcaneal arthrodesis using retrograde intramedullary nailing for complex hindfoot pathology including Charcot arthropathy.