The trap: Placing the finger in a single straight splint that blocks BOTH the PIP and DIP joints — this prevents the lateral bands from being drawn dorsally during DIP flexion and fails to correct the deformity.
The fix: Use a PIP extension splint (Stack, Goldfinger, or custom thermoplastic) that holds the PIP in full extension but leaves the DIP free to flex and extend. The patient must actively flex the DIP against resistance (or passively stretch it) to pull the lateral bands dorsally over the PIP joint. The DIP is the engine of reduction in closed treatment.
PIP Fixed Contracture — Do Not Operate
The pathology: If the PIP joint cannot be passively extended to 0 degrees, the volar plate and accessory collateral ligaments have contracted. Any active extensor reconstruction will fail because the central slip cannot generate enough force to overcome the fixed flexion.
The fix: Serial extension casting or dynamic PIP extension splinting until passive extension is at least 0 degrees. This typically takes 4-8 weeks. Only then is the patient a candidate for central-slip reconstruction. Operating through a fixed contracture produces a stiff, flexed finger.
Swan-Neck Conversion — Overcorrection
Mechanism: Over-tensioning the central slip reconstruction or over-dorsalising the lateral bands (particularly in a Matev-type transfer) can convert the deformity from boutonnière to swan-neck (PIP hyperextension, DIP flexion).
Prevention: Tension the repair in about 20 degrees of PIP flexion, not in full extension. The lateral bands should lie dorsal to the PIP axis but not so far dorsally that they become extensors. Check full passive PIP flexion before closing to ensure the repair does not tether flexion.
Traumatic vs Rheumatoid — Different Treatments
Traumatic pattern: A discrete central-slip rupture (avulsion from dorsal base of middle phalanx, laceration, or closed rupture). Repairable directly in most acute cases. Surgery aims to restore anatomy.
Rheumatoid pattern: Synovitis causes gradual attenuation of the central slip and dorsal capsule without a clean rupture. The lateral bands are displaced volarly by synovial hypertrophy. Direct repair of the attenuated tissue is futile. Treatment is synovectomy plus lateral-band relocation (Matev-type). Managing the synovitis (DMARDs, synovectomy) takes priority.
DIP Hyperextension — Don't Ignore It
Why it matters: The DIP hyperextension component of the boutonnière is cosmetically and functionally disabling — the fingertip cannot flex to grasp small objects. Ignoring it in the reconstruction plan leaves the patient with an incomplete correction.
Options: (1) If the DIP hyperextension is passively correctable and mild (less than 20 degrees), it often resolves spontaneously once the PIP is extended and the lateral bands relocate dorsally. (2) For fixed or severe hyperextension, add a Fowler terminal tenotomy (divide the terminal tendon just distal to the DIP joint). (3) For ORL tightness causing the DIP extension, consider Littler ORL reconstruction.
Hand Therapy Is the Surgery — Rehab Defines Outcome
The principle: In boutonnière reconstruction, the surgical repair is only as good as the post-operative rehabilitation. A technically perfect central-slip repair that is immobilised incorrectly or too briefly will stretch out and recur.
The programme: 6 weeks of continuous PIP extension splinting (removed only for supervised active DIP exercises), followed by 4-6 weeks of night splinting and progressive active PIP flexion. The patient must understand this commitment before surgery. Loss to therapy follow-up is a relative contraindication to complex reconstruction.
Mnemonic
B.O.U.T.O.N.N.I.E.R.EBOUTONNIERE — Systematic Assessment and Treatment
Open Zone III extensor tendon injury with loss of active PIP extension — primary repair within 24 hours
Bony avulsion of central slip with greater than 2 mm fragment displacement and articular incongruity — ORIF or reattachment
Failed non-operative management — persistent or recurrent deformity after 6-8 weeks of compliant full-time PIP extension splinting
Chronic boutonnière (greater than 12 weeks) with supple PIP (passive extension to 0 degrees) and functional disability
Relative Indications
Patient dissatisfaction with functional or cosmetic appearance after closed treatment
Inflammatory arthropathy (rheumatoid, psoriatic) with persistent deformity despite optimal medical management
Recurrent deformity after previous surgical reconstruction
Concomitant nerve or vascular injury requiring exploration
Contraindications
Absolute:
Fixed PIP flexion contracture (passive extension cannot reach 0 degrees) — must be corrected first
Active infection, poorly controlled wound
Non-compliant patient or inability to participate in structured hand therapy (relative absolute)
Relative:
Mild deformity (less than 30 degrees PIP flexion) with minimal functional limitation — try closed treatment
Elderly or low-demand patient with comfortable, functional range — may choose observation
Severe rheumatoid synovitis not medically optimised — treat the synovitis first
Evidence for Non-Operative Treatment
PIP Extension Splinting for Acute Closed Injuries
Full-time PIP extension splinting for 6 consecutive weeks, with the DIP left completely free, is the standard of care for acute closed central-slip injuries
The splint is removed only for wound care and supervised active DIP flexion exercises
The mechanism of action is mechanical: DIP flexion pulls the lateral bands dorsally across the PIP joint, stretching the ORL and allowing the extensor mechanism to heal with the central slip in correct anatomical position
Reported success rates in compliant patients: 80-90% excellent/good outcomes for acute (less than 2 week) closed injuries treated with full-time splinting (Evans, Green)
Key Evidence (Closed Treatment)
Evans (2006) — detailed the "splint the PIP, mobilise the DIP" protocol; approximately 90% good results in acute closed boutonnière when compliance is assured
Coons and Green (2001) — retrospective series confirming that early full-time PIP extension splinting with DIP exercises is the most reliable closed treatment; outcomes deteriorate if initiation of splinting is delayed beyond 2 weeks
Evidence for Surgery
Timing and Case Selection
For established chronic boutonnière (greater than 12 weeks) with failed closed treatment, multiple surgical options exist but the evidence base is largely Level IV (case series) with no high-quality comparative trials
The key principle consistent across all series: the outcome of reconstruction is determined more by patient selection and post-operative rehabilitation than by the specific surgical technique chosen
A supple PIP with passive extension to 0 degrees is the single most important predictor of a successful outcome
Reconstructive Options — Advantages and Limitations
Key Evidence
Evidence
Extensor tendon: anatomy, injury, and reconstruction
Level V
Rockwell WB, Butler PN, Byrne BA
Clinical implication: One of the most frequently cited references for the non-operative management of acute closed boutonnière deformity; reinforced the critical role of the DIP being free during splinting.
Results of the Matev operation for correction of Boutonnière deformity
Level IV
Gama C
Clinical implication: The Matev lateral-band transfer remains a workhorse procedure for chronic boutonnière when the central slip is irreparable and the PIP is supple.
Correction of the severe nonrheumatoid chronic boutonnière deformity with a modified Matev procedure
Level IV
Terrill RQ, Groves RJ
Clinical implication: A refined version of the classic Matev technique for severe nonrheumatoid boutonnière that can guide contemporary reconstruction planning.
Anatomic repair of the central slip with anchor suture augmentation for treatment of established boutonnière deformity
Level IV
Lee JK, Lee S, Kim M, Jo S
Clinical implication: Suture anchor fixation provides a biomechanically stronger and technically simpler alternative to transosseous tunnels for central-slip reattachment.
Clinical implication: A useful modern reference that integrates both deformities into a single decision framework, reinforcing the importance of aetiology-specific treatment.
The Extensor Mechanism at the PIP Joint (Zone III)
Central Slip — Anatomy and Function
The central slip is the continuation of the common extensor tendon (EDC in fingers, EIP/index, EDM/small) that inserts on the dorsal base of the middle phalanx
It is the primary active extensor of the PIP joint — it provides roughly 80% of PIP extension power
The central slip is a flat, broad tendon approximately 4-6 mm wide at the PIP level, receiving contributions from the interosseous and lumbrical muscles via the lateral bands
The dorsal capsule of the PIP joint lies immediately deep to the central slip — injury to the central slip frequently involves the dorsal capsule, and the two structures should be repaired together
Lateral Bands — Normal and Pathological Position
Normal anatomy:
The two lateral bands (radial and ulnar) run dorsal to the PIP axis of rotation in the uninjured digit
Each lateral band receives: fibres from the interosseous muscle (on its respective side), the lumbrical (radial side only, in the index to little fingers), and contributions from the extensor tendon
Distal to the PIP, the lateral bands converge to form the terminal tendon, which inserts on the dorsal base of the distal phalanx
Pathological migration in boutonnière deformity:
When the central slip is disrupted, the lateral bands lose their dorsal restraint and migrate volarly — they slide past the PIP axis of rotation
Once volar to the PIP axis, the lateral bands become flexors of the PIP joint — each contraction of the intrinsic muscles (which are meant to extend the PIP via the lateral bands) now increases the PIP flexion deformity
The volar migration also tethers the oblique retinacular ligament, which contributes to DIP hyperextension
The ORL is a slender, oblique ligament that runs from the volar aspect of the flexor tendon sheath at the proximal phalanx level to the dorsal aspect of the lateral bands or terminal tendon distally
In the uninjured finger, the ORL acts as a passive coupling between PIP and DIP motion: when the PIP extends, the ORL slackens — when the PIP flexes, the ORL tightens and helps flex the DIP
In boutonnière deformity, the ORL becomes pathologically tight because the lateral bands are volarly displaced — this pulls the terminal tendon into extension and contributes to the DIP hyperextension
DIP hyperextension in boutonnière is therefore a passive mechanical phenomenon (from ORL tightness) rather than an active one
The Transverse Retinacular Ligament (TRL)
Also known as the Cleland's ligament in its oblique part, the TRL runs transversely from the lateral band to the flexor tendon sheath at the PIP level
It is one of the structures that normally holds the lateral bands in a dorsal position
Disruption or attenuation of the TRL is thought to contribute to volar lateral-band migration in boutonnière deformity
Some surgical techniques incorporate TRL reefing to help restore lateral-band position
Zones of the Extensor Mechanism
The Verdan classification divides the extensor tendon into 8 zones (odd = joint, even = bone):
Zone I
Location
DIP joint
Structures at Risk
Terminal tendon
Relevance to Boutonnière
Fowler tenotomy site; terminal tendon rupture in chronic cases
Zone II
Location
Middle phalanx
Structures at Risk
Lateral band confluence
Relevance to Boutonnière
Lateral band dissection during Matev transfer
Zone III
Location
PIP joint
Structures at Risk
Central slip insertion
Relevance to Boutonnière
PRIMARY injury site for boutonnière deformity
Zone IV
Location
Proximal phalanx
Structures at Risk
Extensor hood, lateral bands
Relevance to Boutonnière
Lateral band mobilisation during reconstruction
Zone
Location
Structures at Risk
Relevance to Boutonnière
Zone I
DIP joint
Terminal tendon
Fowler tenotomy site; terminal tendon rupture in chronic cases
Zone II
Middle phalanx
Lateral band confluence
Lateral band dissection during Matev transfer
Zone III
PIP joint
Central slip insertion
PRIMARY injury site for boutonnière deformity
Zone IV
Proximal phalanx
Extensor hood, lateral bands
Lateral band mobilisation during reconstruction
Vascular Anatomy and Wound Healing
The dorsal skin over the PIP joint has a segmental blood supply from the dorsal digital branches of the radial and ulnar digital arteries
The central slip itself is relatively hypovascular — this is why it has limited healing potential and requires prolonged protection after repair
The lateral bands are better vascularised (intrinsic muscle contributions) — this makes them a good choice for local transfer (Matev) in chronic cases, as they bring their own blood supply into the reconstruction
Structures at Risk During Surgical Exposure
Dorsal digital nerves — small-calibre sensory branches of the radial nerve (index, middle, ring radial) and ulnar nerve (ring ulnar, small) that run in the dorsal subcutaneous tissue over the PIP — they can be divided during skin incision if the incision is placed too laterally or dorsally
The extensor tendon itself (the repaired/reconstructed structure) — the most common "injured" structure is the repair itself, by overly aggressive post-operative mobilisation
The articular surface of the PIP joint — the joint is entered during dorsal capsulectomy or bony preparation for central-slip reattachment; inadvertent chondral injury must be avoided
The opposite-side lateral band — during Matev transfer, the intact lateral band must be protected from iatrogenic injury, as both bands are needed for balanced PIP function
Positioning and Preparation
Patient position: Supine, arm abducted on a hand table. A well-padded tourniquet is applied to the upper arm (or forearm for better comfort: 200 mmHg). Exsanguinate with an Esmarch bandage.
Anaesthesia: Regional anaesthesia (supraclavicular or infraclavicular brachial plexus block) with sedation — this provides excellent muscle relaxation and a bloodless field while allowing the patient to tolerate tourniquet times of 60-90 minutes. WALANT is not ideal for complex reconstructions because the patient may move during delicate steps, but can be used in selected patients.
Consent: Specifically counsel regarding risk of recurrence (approximately 15% at 5 years), residual stiffness (flexion loss of 10-20 degrees common), swan-neck conversion (overcorrection), DIP extension lag (if Fowler tenotomy added), infection (less than 1%), and need for prolonged therapy compliance.
Approach
Dorsal Approach to the PIP Joint
The standard approach is a dorsal curvilinear or lazy-S incision centred over the PIP joint:
A 3-4 cm dorsal longitudinal incision is made centred over the PIP joint, gently curving to avoid crossing the joint crease at 90 degrees (which can cause hypertrophic scarring)
Alternatively, a midaxial approach (midline of the finger on the ulnar or radial border) can be used — this gives access to the dorsal structures through a laterally based flap and avoids the dorsal scar
Elevate skin flaps carefully in the subcutaneous plane, protecting the dorsal sensory nerves
Surface landmark: The PIP joint line is identified by flexing and extending the finger — the centre of rotation lies at the level of the PIP flexion crease, midpoint between the proximal and middle phalanges.
Deep Dissection
Identify the extensor mechanism — in established boutonnière, the central slip is attenuated or discontinuous, and the lateral bands are visible in their volarly displaced position
The attenuated central slip may appear as a thin, scarred sheet rather than a discrete tendon
Enter the PIP joint through the dorsal capsule (which is often disrupted alongside the central slip) to assess the articular surfaces — if cartilage loss is severe (greater than 50%), consider salvage options (PIP arthrodesis or arthroplasty) rather than reconstruction
Identify the lateral bands and mobilise them gently from their volar position back to the dorsal aspect of the PIP joint — use a fine elevator to separate them from the underlying capsule and collateral ligaments
Prerequisites Satisfied — Intraoperative Check
Before proceeding to any specific reconstruction, confirm:
The PIP joint can be passively extended to 0 degrees — if not, perform a dorsal capsulectomy and release of the accessory collateral ligaments
The articular surface is in satisfactory condition (less than 50% cartilage loss)
The lateral bands are of adequate quality for transfer if a Matev procedure is planned
At least one lateral band can be mobilised without disrupting the contralateral band
Central-Slip Repair (Acute or Repairable Chronic)
Indication
The central slip is identifiable and can be advanced to its insertion without excessive tension (gap less than 5 mm with the PIP in 20 degrees of flexion).
Technique
Prepare the insertion site: Expose the dorsal base of the middle phalanx. If a bony fragment is present, mobilise it with care. If the tendon end is soft tissue only, freshen the footprint to bleeding bone with a small curette or rongeur
Suture repair: Use a 3-0 braided polyester (Ethibond) or 4-0 braided polyester suture in a modified Kessler or Bunnell configuration, depending on tendon integrity:
Good-quality tendon: Modified Kessler with epitendinous repair
Poor-quality/attenuated tendon: Multiple figure-of-eight sutures to spread the tension across a wider area; consider augmentation with a lateral-band slip
Fixation to bone:
Bony avulsion fragment larger than 2 mm: reduce and fix with a 1.3 mm or 1.5 mm mini-screw or K-wire
Soft-tissue reattachment: Drill two 1.1 mm transosseous tunnels from the dorsal footprint to the volar cortex of the middle phalanx base; pass the suture ends through the tunnels and tension over a button on the volar aspect (tension the repair in 20 degrees of PIP flexion)
Suture anchor alternative: A 1.3 mm or 1.8 mm suture anchor loaded with 3-0 braided polyester can be placed in the dorsal base of the middle phalanx as an alternative to transosseous tunnels — this is simpler but may not achieve equivalent pull-out strength in early active motion protocols
Tensioning
Tie the repair with the PIP joint in 20-30 degrees of flexion — do NOT tension in full extension, as this will produce a swan-neck deformity
The repair should be snug but not tight — the PIP should still passively flex to at least 70 degrees
The lateral bands should sit dorsal to the PIP axis of rotation — if they remain volar, they must be relocated
Clinical Pearl
Technical Tip: 'I repair the central slip with 3-0 Ethibond in a modified Kessler pattern and pass the suture ends through two transosseous tunnels in the dorsal base of the middle phalanx. I tension the suture over a volar button with the PIP in 20 degrees of flexion. Before tying the knot, I check that the lateral bands lie dorsally and that the DIP can passively flex fully — if the DIP is stiff, the repair is too tight and I need to re-tension it in slightly more flexion.'
Lateral-Band Transfer (Matev Reconstruction)
Indication
Chronic boutonnière with attenuated central slip that cannot be primarily repaired (gap greater than 5 mm or poor tissue quality). A supple PIP with passive extension to 0 degrees remains an absolute prerequisite.
Principle
One lateral band is transected proximally and transferred to the dorsal base of the middle phalanx to reconstruct the central-slip insertion. The transferred lateral band then acts as an active PIP extensor while the remaining lateral band continues to provide DIP extension.
Technique (Matev)
Identify the lateral bands — in chronic cases they are volarly displaced and may be adherent. Mobilise them with a fine elevator, preserving the transverse retinacular ligaments where possible
Select the donor band: On the side of the scar (previous injury) — use the more accessible lateral band. If neither side is scarred, choose the ulnar lateral band (more robust in most digits)
Transect the lateral band at the level of the proximal phalanx midshaft (Zone IV) — a full-thickness cut. The proximal end will be transferred
Route the distal stump: The distal stump of the transected lateral band is left attached to the terminal tendon — it continues to provide DIP extension through the intact contralateral lateral band
Transfer the proximal end: Pass the proximal cut end of the lateral band through a transosseous tunnel drilled at the dorsal base of the middle phalanx (aim: exit volarly, just proximal to the FDP insertion, to avoid flexor tendon tethering)
Fixation: Tension the transferred lateral band with the PIP in 20 degrees of flexion. Suture it to itself or secure it over a volar button. Alternatively, suture the transferred band end-to-side to the contralateral lateral band after routing it through the bone tunnel
Checkbalance: The PIP should extend to 0 degrees passively and flex to at least 70 degrees. The DIP should still actively extend (via the intact contralateral lateral band and the terminal tendon)
Matev Reconstruction — Critical Checks
Do NOT transfer BOTH lateral bands — the DIP will lose all active extension
Do NOT tension the transfer in full PIP extension — this forces the DIP into hyperextension and creates a swan-neck deformity
If the transferred band is too short to reach the bone tunnel, consider a palmaris longus tendon graft interposed between the lateral band and the bone tunnel
Verify that the transferred lateral band glides freely in its subcutaneous route — if it is caught by scar, it will not function as an active extensor
Terminal-Tendon Tenotomy (Fowler)
Indication
Persistent DIP hyperextension (greater than 20 degrees) after the PIP has been corrected to neutral. The tenotomy is performed as a separate or adjunctive procedure — it can be done through the same dorsal exposure or as a percutaneous step.
Technique
Extend the PIP to neutral (or keep the PIP splinted in extension) to tension the terminal tendon
Identify the terminal tendon over the middle phalanx, approximately 3-5 mm distal to the DIP joint (NOT over the joint itself — tenotomising over the joint violates the dorsal capsule)
Make a transverse division of the terminal tendon through a small stab incision (percutaneous) or through the open dorsal wound
Confirm correction: the DIP should now have a 10-20 degree passive extension lag. The active DIP flexion should be preserved (the FDP provides DIP flexion independently)
Do NOT repair the tenotomy — the separation is intentional to release the ORL tether
Clinical Pearl
Technical Tip: 'For Fowler tenotomy, I identify the terminal tendon distal to the DIP joint by flexing and extending the DIP — the terminal tendon moves visibly. I divide it transversely with a number 15 blade through a 3 mm longitudinal skin incision. The DIP should immediately drop into 10-15 degrees of flexion. I do not repair or splint the tenotomy. I counsel the patient pre-operatively that the fingertip will permanently lag in extension by about 10-15 degrees — this is the trade-off for gaining the ability to flex the DIP to grasp small objects.'
Persistent DIP hyperextension in a young, high-demand patient where a Fowler tenotomy (which creates a permanent extension lag) is undesirable. The ORL reconstruction aims to reconstruct the Landsmeer ligament to dynamically link PIP extension with DIP flexion.
Principle
A free tendon graft (palmaris longus) is routed from the base of the proximal phalanx (volar-ulnar/radial) to the dorsal terminal tendon, parallel to the normal ORL. When the PIP extends, the graft tightens and passively flexes the DIP — restoring the natural PIPC-DIP coupling.
Technique (simplified)
Harvest palmaris longus tendon (or plantaris, or extensor indicis proprius if palmaris is absent)
Create a transosseous tunnel at the base of the proximal phalanx from volar-lateral to dorsal
Create a second tunnel through the terminal tendon just proximal to the DIP joint
Weave the graft from the volar tunnel at the proximal phalanx base, along the lateral aspect of the finger, to the terminal tendon
Tension with the PIP in 0 degrees and the DIP in 20 degrees of flexion
Secure the graft to the terminal tendon with 5-0 Prolene
This procedure is rarely performed in contemporary practice — Fowler tenotomy is simpler, more predictable, and selected in the vast majority of cases requiring DIP correction.
Closure and Dressing
Close the skin with 4-0 or 5-0 nylon interrupted sutures (avoid horizontal mattress sutures that can compromise dorsal skin vascularity)
Apply a non-adherent dressing (Jelonet)
Apply a PIP extension splint as part of the dressing — use a dorsal or volar thermoplastic splint that holds the PIP in full extension but leaves the DIP completely free
No MCP immobilisation is required unless the procedure was combined with an intrinsic release
Clinical Pearl
Technical Tip: 'I incorporate the PIP extension splint into the dressing at the time of surgery. I use a custom thermoplastic volar splint that extends from the proximal phalanx to the middle phalanx, holding the PIP in 0 degrees. I cut the splint short of the DIP — the DIP must be free to flex from day 1 post-operatively. I write the therapy prescription before the patient leaves the operating theatre.'
The rehabilitation protocol is the single most important determinant of outcome in boutonnière reconstruction. The programme below is a generic guideline — specific protocols should be tailored to the intraoperative findings, repair quality, and the hand therapist's preference.
Phase 1: Protection (Weeks 0-6)
Continuous PIP extension splinting — the PIP is held in full extension at all times, day and night
DIP free and actively exercised — the patient performs 10-15 active DIP flexions every waking hour using the opposite hand to block the PIP in the splint while flexing the DIP against resistance
The DIP exercise is the treatment: each DIP flexion cycle pulls the lateral bands dorsally, stretches the ORL, and maintains the correct biomechanical environment for the central-slip reconstruction to heal
Splint removal only for supervised wound care and therapy sessions — never remove the splint for more than 15 minutes at a time
Wound care: Keep the dorsal incision dry; change the dressing every 3-5 days
Suture removal: at 14 days
Phase 2: Transition (Weeks 6-8)
The PIP extension splint is removed for supervised active PIP extension exercises — the patient actively extends the PIP against gravity (no resistance) with the DIP free
Begin active PIP flexion within a comfortable range — typically starting at 30-40 degrees, progressing to 60 degrees by week 8
Night splinting continues for at least 4 more weeks
Never force passive PIP flexion — the reconstruction is weakest at weeks 4-8 and can be stretched out
Phase 3: Strengthening (Weeks 8-12)
Progressive active PIP flexion and extension against gentle resistance (putty, elastic bands)
Scar massage and desensitisation
Grip strengthening begins at week 10
Night splinting continues until week 12
Phase 4: Return to Function (Weeks 12+)
Full active ROM expected by 12-16 weeks
Return to heavy manual work: usually at 12-16 weeks
Final ROM remains relatively stable after 6 months
Patient should expect some permanent flexion loss (10-20 degrees at the PIP) in most cases
Therapy Compliance
The single most common cause of failure in boutonnière reconstruction is loss to therapy or non-compliance with the splinting protocol
Assess the patient's social situation, occupation, and motivation before offering reconstruction
A written therapy contract signed by the patient is reasonable practice for complex reconstructions
Special Case: Traumatic Boutonnière (Acute, Closed)
Clinical Scenario
A patient presents within 2 weeks of a closed PIP injury with an Elson test positive for central-slip disruption. The PIP is supple with no fixed flexion deformity.
Management
Full-time PIP extension splinting with DIP free for 6 weeks — the standard of care
Follow the Phase 1 protocol described above
Success rate: 80-90% good/excellent with compliant splinting
If deformity recurs after 6 weeks of compliant splinting, consider reconstruction as for chronic boutonnière
Surgical Indication in the Acute Setting
Open Zone III laceration — primary repair within 24 hours
Failed closed treatment despite compliance (persistent deformity at 8 weeks)
Bony avulsion with greater than 2 mm displacement
Special Case: Rheumatoid Boutonnière
Why It Is Different
The pathology is synovitis-driven attenuation of the central slip and dorsal capsule, not a discrete tendon rupture
The lateral bands are displaced volarly by hypertrophic synovium
Bony erosions and joint destruction may coexist
Direct repair of the attenuated central slip tissue is futile — it will stretch out
Management Algorithm
Optimise medical management of synovitis (DMARDs, biologics) — do not operate on an actively inflamed joint
If synovitis is controlled but deformity persists (supple PIP):
Dorsal synovectomy to decompress the PIP joint
Lateral-band relocation (Matev-type) without attempting direct central-slip repair
Fowler tenotomy if DIP hyperextension persists after PIP correction
If the PIP joint is destroyed (radiographic loss of greater than 50% joint space, erosions):
PIP arthrodesis (in 20-30 degrees of flexion for functional hand position)
PIP arthroplasty (silicone) in selected low-demand patients with good bone stock and intact collateral ligaments
Outcome Expectation
Results are less predictable than traumatic boutonnière — approximately 50-70% satisfactory outcomes
Recurrence risk is higher due to ongoing synovitis
Managing patient expectations is critical
Special Case: Failed Primary Reconstruction
Patterns of Failure
Recurrence within 6 weeks = inadequate splinting or therapy non-compliance — resume full-time PIP extension splinting for another 4-6 weeks
Recurrence at 6-12 weeks = repair stretched out — assess passive correction; if supple, consider re-exploration and revision with augmentation (tendon graft, lateral-band transfer)
Recurrence after 12 weeks = established failure — assess PIP suppleness. Options: (a) revision reconstruction with palmaris longus graft if supple, (b) PIP arthrodesis if stiff or if patient has failed two prior reconstructions
Revision Strategy
If the lateral bands are intact and the PIP is supple, a palmaris longus tendon graft woven through the base of the middle phalanx and the lateral bands can reconstruct the central slip
If the PIP is stiff (less than 30 degrees passive extension), serial casting or dynamic splinting first, then reassess
If all else fails or the joint is arthritic: PIP arthrodesis in 20-30 degrees of flexion — this provides a stable, pain-free finger at the cost of PIP motion
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
Viva scenarioStandard
Clinical prompt
“A 34-year-old electrician presents 4 weeks after a closed jam injury to his right ring finger PIP joint while playing cricket. He has a boutonnière posture of the finger. The PIP can be passively extended to 0 degrees and Elson test is positive. How do you manage him?”
Practical approach
This is an acute (less than 6 weeks) closed central-slip disruption with a fully supple PIP — the ideal candidate for closed non-operative management. I would treat him with full-time PIP extension splinting for 6 continuous weeks with the DIP joint left completely free to flex.\n\n**Splinting protocol**: I would fit a custom thermoplastic PIP extension splint (or a Stack-type splint) that holds the PIP in full extension. The DIP must be free. The patient must flex the DIP actively 10-15 times every waking hour — each DIP flexion pulls the lateral bands dorsally across the PIP joint and stretches the oblique retinacular ligament, which is the mechanism by which the deformity corrects.\n\n**Monitoring**: I would review him at 2 weeks to check skin condition and compliance, then at 6 weeks to remove the splint and assess active PIP extension. At 6 weeks, if he has active PIP extension to within 10 degrees of full extension, I would transition to night splinting for another 4 weeks and begin gentle active PIP flexion. If the deformity persists or recurs after 6 weeks of compliant splinting, I would consider central-slip reconstruction.\n\n**Return to work**: As an electrician, he cannot return to work until the splinting phase is complete (6 weeks) and then gradually over the next 4-6 weeks with light duties. Heavy manual work is typically at 12-16 weeks. I would counsel him on this timeline at the first visit to set realistic occupational expectations.
Viva scenarioAdvanced
Clinical prompt
“A 58-year-old woman with well-controlled seropositive rheumatoid arthritis has a chronic boutonnière deformity of her right long finger. The PIP can be passively extended to 0 degrees. There is 25 degrees of DIP hyperextension that is fixed in this position. Radiographs show preserved PIP joint space. How would you reconstruct this finger?”
Practical approach
This is a classic rheumatoid boutonnière pattern — synovitis has attenuated the central slip without a discrete rupture, and the lateral bands are displaced volarly. The preserved joint space on radiographs and controlled synovitis are favourable. The fixed DIP hyperextension (25 degrees) suggests the oblique retinacular ligament is tight.\n\n**Pre-operative**: I would confirm that her rheumatoid disease is medically optimised (rheumatologist review, stable DMARD regimen, no active synovitis in the PIP joint). I would re-examine the PIP to confirm passive extension to 0 degrees — if there is any residual contracture, I would treat it with serial extension casting or dynamic splinting first.\n\n**Operative plan**: Through a dorsal curvilinear approach over the PIP joint:\n1. **Dorsal synovectomy** first — excise the hypertrophic synovium to decompress the joint and reduce the inflammatory load\n2. **Do NOT attempt direct central-slip repair** — in rheumatoid disease the attenuated central-slip tissue does not hold sutures. Instead, I will perform a lateral-band relocation (Matev-type reconstruction)\n3. **Matev lateral-band transfer**: I would identify both lateral bands, select the ulnar band for transfer (more robust), transect it at the proximal phalanx level, and transfer the proximal end to a transosseous tunnel at the dorsal base of the middle phalanx. I would tension the transfer with the PIP in 20 degrees of flexion\n4. **Address the DIP hyperextension**: Because the 25 degrees of DIP hyperextension is fixed (not passively correctable), the DIP will not resolve with PIP correction alone. I would add a **Fowler terminal tenotomy** — divide the terminal tendon 3-5 mm distal to the DIP joint through the same incision or percutaneously. This will release the ORL tether and allow the DIP to flex\n5. **Post-operative**: PIP extension splint with DIP free for 6 weeks; then transition to night splinting and gentle active flexion\n\n**Outcome expectation**: I would counsel her that results in rheumatoid disease are less predictable than traumatic cases — approximately 60-70% chance of satisfactory correction. She should expect some residual PIP stiffness (10-20 degrees flexion loss) and a permanent 10-15 degree DIP extension lag from the Fowler tenotomy. The tenotomy trade-off is acceptable: she gains the ability to flex her fingertip to grasp small objects, which is functionally more important than full DIP extension.
Viva scenarioAdvanced
Clinical prompt
“A 22-year-old university student underwent a Matev lateral-band transfer for a chronic boutonnière deformity of the left index finger 4 months ago. She has been compliant with therapy. The PIP is now 15 degrees into hyperextension (swan-neck posture) with the DIP resting in 20 degrees of flexion. She is distressed about the appearance and has difficulty picking up small objects. How do you manage this complication?”
Practical approach
This is a swan-neck deformity resulting from overcorrection of the boutonnière — the lateral-band transfer was tensioned too tightly or the transferred band has been positioned too far dorsally, converting the PIP from flexion to hyperextension.\n\n**Assessment**: First, I would determine whether the deformity is passively correctable (supple) or fixed. I would examine: (1) passive PIP hyperextension — can it be reduced to neutral? (2) DIP passive flexion — is the terminal tendon tight? (3) active flexion arc of both PIP and DIP — what is the total active motion? (4) stability of the PIP in the lateral plane — are the collateral ligaments intact? (5) X-rays to exclude bony changes, joint destruction, or arthritis from the surgery.\n\n**Initial management (non-operative)**: If the deformity is mild (less than 20 degrees of PIP hyperextension) and the PIP is supple, I would start with a hand-therapy programme: (1) PIP flexion blocking exercises — strengthen the FDS to actively resist hyperextension, (2) DIP flexion stretching to overcome any terminal tendon tightness, (3) a PIP flexion-assist splint or figure-of-eight splint that prevents full PIP extension, (4) buddy-taping to the adjacent index PIP to encourage flexion.\n\n**Surgical options if non-operative management fails**:\n1. **If the deformity is fixed** (less than 30 degrees passive PIP flexion): Check-rein ligament release (volar PIP capsulotomy) plus terminal-tendon tenolysis\n2. **If the deformity is supple** (passive flexion greater than 70 degrees): The least invasive option is a **Fowler tenotomy** of the terminal tendon — this converts the DIP from flexion back to a more extended posture and reduces the hyperextension force on the PIP via the lateral bands. Alternatively, the lateral-band transfer can be taken down and re-tensioned more loosely\n3. **If all else fails or the patient is unwilling to accept additional recovery time**: The easiest salvage is a **PIP arthrodesis** in 20 degrees of flexion — a stable, pain-free finger with excellent function for grasp and pinch\n\n**Management in this patient**: Given she is 4 months post-operative and the deformity is moderate (15 degrees hyperextension, supple), I would begin with 4-6 weeks of hand therapy (FDS strengthening, flexion splinting). If there is no improvement, I would offer a Fowler tenotomy under local anaesthetic as a 15-minute procedure, which would correct the DIP flexion component and indirectly reduce the PIP hyperextension. I would counsel her that tenotomy creates a permanent DIP extension lag of 10-15 degrees (she already has 20 degrees of DIP flexion, so the lag will change from a flexed to a neutral or slightly lagging posture — functionally better).
Exam day cheat sheet
Boutonnière Deformity — Reconstruction: Exam Day Summary
References
Evidence
Surgical treatment of the boutonniere deformity in rheumatoid arthritis
Level IV
Nalebuff EA, Millender LH
Clinical implication: Rheumatoid boutonnière requires a fundamentally different approach to traumatic boutonnière.