End-bearing amputation at the ankle level preserving the heel pad | advanced
- Prerequisites: intact heel pad skin, patent posterior tibial artery (via the medial calcaneal branch), and TcPO2 greater than 30 mmHg.
- The dissection of the calcaneus off the heel pad must be strictly subperiosteal β straying into the fat pad destroys its septal architecture and blood supply and the pad dies.
- Bony cut: the malleoli and a thin slice of the tibial plafond are resected flush and parallel to the ground in both planes, preserving the maximum metaphyseal flare for end-bearing and suspension.
- The heel pad must be anchored to the distal tibia through anterior drill holes, otherwise it migrates posteriorly and the stump fails.
When & Why
Indication. A Syme amputation is an end-bearing ankle disarticulation used when the forefoot or midfoot is unsalvageable but the heel pad and its blood supply are pristine. The classic situations are: - Unsalvageable forefoot or midfoot infection or gangrene β severe diabetic foot sepsis that cannot be managed with a partial foot amputation.
- Severe distal foot trauma β crush or blast injuries where the forefoot cannot be reconstructed but the heel pad is uninjured.
- Forefoot or midfoot tumours where ankle disarticulation achieves adequate oncological margins.
- Congenital foot deformities (e.g. fibular hemimelia, severe untreated clubfoot) where a Syme provides a superior prosthetic base than the native deformed foot. Prerequisites for success. Before offering a Syme, three boxes must be ticked: - Vascular β a palpable posterior tibial pulse, Doppler biphasic flow, or TcPO2 greater than 30 mmHg. The posterior tibial artery is the sole supply to the heel pad through its medial calcaneal branches.
- Soft tissue β the heel pad skin must be completely intact, with no deep ulceration, necrosis or active infection, and healthy enough to bear full body weight.
- Functional β the patient must have the cognitive and physical capacity to engage with prosthetic rehabilitation. Contraindications. Absolute: absent posterior tibial artery flow (unless reconstructable by bypass), heel pad necrosis or deep ulceration, inadequate tissue oxygenation (TcPO2 less than 20 mmHg universally predicts failure), and ascending gas gangrene (which needs a higher open amputation for life salvage). Relative: severe neuropathy risking unrecognised heel trauma, and a bedbound non-ambulatory patient β a transtibial amputation often heals more reliably and faster in a severe dysvascular bedbound patient, even though a Syme is end-bearing and can assist with pivot transfers. The one decision that matters β one-stage or two-stage. Every Syme begins the same way, with ankle disarticulation. The only choice is whether to do the definitive malleolar resection and pad closure immediately, or stage it:
James Syme's original 1843 operation. Definitive bony resection and soft-tissue closure in a single sitting. Excellent in trauma, congenital cases and controlled dysvascular cases without active infection β but carries an unacceptably high failure rate in severe forefoot sepsis, because the broad cancellous surface of the distal tibia is exposed to contamination.
Built for the diabetic patient with an infected forefoot. Stage one: disarticulate and remove the infected forefoot but leave the malleoli intact, loosely draping the oversized pad over them. Stage two, two to six weeks later once infection has resolved: resect the malleoli and close definitively. Significantly lowers stump infection and revision to transtibial amputation β the standard of care for infected cases.
- Syme Amputation
- End-bearing β can walk short distances without a prosthesis
- Transtibial Amputation
- Not end-bearing β requires a prosthesis for any weight bearing
- Syme Amputation
- Only 10 to 15 percent increased above baseline
- Transtibial Amputation
- 20 to 40 percent increased above baseline
- Syme Amputation
- Custom socket accommodating the bulbous stump
- Transtibial Amputation
- Patellar tendon bearing or total contact socket
- Syme Amputation
- High β technically demanding heel pad dissection
- Transtibial Amputation
- Moderate β standardised technique
- Syme Amputation
- Lower in severe peripheral vascular disease
- Transtibial Amputation
- Higher β more robust collateral blood supply
Consent specifically for the possibility of heel pad necrosis (which can mean revision to a transtibial amputation), heel pad migration, painful neuroma within the socket, wound infection (highest in diabetics with active sepsis), and the lifelong need for a specialised Syme prosthesis. Setup. Supine with a sandbag under the ipsilateral buttock to bring the leg to neutral and the lateral malleolus anteriorly. A thigh tourniquet may be used in trauma or congenital cases but is generally avoided in severe peripheral vascular disease to avoid further ischaemic insult. Prep and drape the entire leg from the knee down so alignment and rotation can be judged accurately.
The Operation
The goal is to disarticulate at the ankle, deliver the calcaneus out of the heel pad by strictly subperiosteal dissection (protecting the pad's architecture and the posterior tibial artery), resect the malleoli flush and parallel to the ground, and anchor the pad squarely beneath the distal tibia so it becomes a durable end-bearing surface. The exposure β incision, anterior dissection and ankle disarticulation β is laid out in the first steps below; the calcaneal dissection is the technical heart of the operation.

Operative sequence β classical one-stage Syme
- The heel pad is specialised weight-bearing tissue: closed fat compartments separated by thick, vertical fibrous septa that anchor skin to calcaneus. Dissection must stay on bone β cut the septa and the fat lobules necrose.
- Its lifeline is the medial calcaneal artery, a branch of the posterior tibial artery, which enters the pad posteromedially. Lateral calcaneal branches from the peroneal artery give only a small secondary contribution. Loss of the anterior tibial artery does NOT compromise the pad.
- Five sensory nerves cross the ankle and must be managed to prevent neuroma: the tibial, superficial peroneal, deep peroneal, sural and saphenous nerves.
- Two continuous limbs meet at the malleoli, creating a U-shaped plantar flap and a slightly curved anterior flap.
- Anterior limb: straight across the front of the ankle from the tip of the lateral malleolus to a point 1.5 cm inferior to the tip of the medial malleolus. The medial start is deliberately lower to protect the posterior tibial neurovascular bundle.
- Plantar limb: a vertical cut straight down to bone across the sole, passing directly under the heel. It must be a perfectly straight vertical line β too anterior and the pad is too long and redundant, too posterior and it is too short to cover the tibia.
- Deepen the anterior incision straight down to bone and the anterior ankle capsule.
- Identify and ligate the anterior tibial artery and vein.
- Identify the superficial peroneal, deep peroneal and saphenous nerves, apply gentle traction, transect them cleanly and let them retract proximally.
- Divide the extensor tendons (tibialis anterior, extensor hallucis longus, extensor digitorum longus) under distal traction so they retract out of the field.
- Incise the anterior joint capsule to enter the ankle joint and plantarflex the foot firmly to open the anterior space.
- Divide the medial (deltoid) and lateral collateral ligaments from inside the joint, cutting close against the talus to protect surrounding soft tissues.
- The talus is now free to subluxate anteriorly, exposing the posterior capsule, which is then incised.
- Place a bone hook in the dome of the talus and pull the foot strongly anteriorly and inferiorly, placing the posterior soft tissues under tension.
- With a heavy knife or sharp periosteal elevator, dissect the soft tissues off the superior, posterior and inferior aspects of the calcaneus. The dissection must be strictly subperiosteal β hug the bone tightly, working superior to inferior. Straying into the fat pad destroys the septal architecture and blood supply and the pad necroses.
- Take extreme care on the posteromedial corner: the posterior tibial artery and its medial calcaneal branches lie immediately adjacent. Stay firmly on bone.
- Divide the Achilles tendon close to its insertion on the calcaneal tuberosity, taking care not to buttonhole the very thin posterior skin over the insertion.
- Continue the subperiosteal dissection inferiorly until the entire calcaneus and foot are delivered out of the heel pad.
- Clear soft tissues 1 to 2 cm proximally from the articular margin of the distal tibia and fibula, using retractors to protect the flaps.
- With a broad oscillating saw, remove the medial and lateral malleoli and a thin (3 to 5 mm) slice of the distal tibial articular surface.
- The cut must be perfectly parallel to the ground in both coronal and sagittal planes β check alignment with the knee fully extended. A non-parallel cut causes shear forces and pressure sores in the socket.
- Preserve the broad metaphyseal flare β do not cut too proximally or the end-bearing surface and suspension are lost.
- Smooth all sharp bony edges with a rasp, especially the anterior tibial cortex.
- Identify the tibial nerve (posteromedial, with the posterior tibial artery) and the sural nerve (posterolateral, behind the lateral malleolus) in the posterior flap.
- The tibial nerve is often thick and may need ligation for bleeding from its vasa nervorum.
- Transect the nerves high under tension so they retract well proximal to the weight-bearing surface to prevent terminal neuromas.
- To stop the pad migrating posteriorly or medially on weight-bearing, anchor it firmly to bone.
- Drill several small holes (usually 3 to 5) in the anterior and anterolateral cortex of the distal tibia and fibula, about 1 cm proximal to the cut edge.
- Pass heavy non-absorbable sutures (e.g. number 2 FiberWire or Ethibond) through the deep fascia of the anterior edge of the heel pad and secure them through the drill holes.
- The pad should sit squarely and centrally beneath the cut end of the tibia.
- Insert a deep suction drain to prevent haematoma, which compromises flap viability and fuels infection.
- Trim small dog-ears medially and laterally only if it does not narrow the flap base or threaten its blood supply β often they remodel and are best left alone.
- Close skin and subcutaneous tissue with interrupted monofilament (e.g. 3-0 nylon) to avoid tissue strangulation.
- Apply a rigid or semi-rigid dressing (such as a plaster cast) to hold the pad central and control post-operative oedema.
At the danger zone (Step 5) the operation is won or lost. Three errors each threaten the whole flap: (1) violating the heel pad septa by straying off bone, which causes fat necrosis and loss of the weight-bearing cushion; (2) damaging the posterior tibial artery medially, which causes complete flap necrosis; and (3) buttonholing the thin posterior skin near the Achilles insertion, which compromises the flap and opens a portal for infection. The defence in every case is the same β stay strictly subperiosteal, hug the bone, and keep the soft tissues on tension with a bone hook in the talus.
Do not angle the plantar incision. It must run in a perfectly straight vertical line down to the sole so the heel pad is the correct size. Angled anteriorly, the pad is too long and redundant; angled posteriorly, it is too short to cover the tibia. Mark it before you cut and check it sits directly under the heel.
In the diabetic patient with an actively infected forefoot, do everything in Step 1 to Step 5 but do NOT resect the malleoli or the tibial articular cartilage. Leave the oversized heel pad loosely draped over the intact malleoli, insert a drain and close loosely (or leave partially open if heavily contaminated). Two to six weeks later, once infection has cleared and inflammatory markers have normalised, reopen and complete Step 6 (bony resection), Step 7 (nerves) and Step 8 (pad anchoring) with definitive closure over a drain.
Aftercare & Complications
Rehabilitation | Phase | Timing | Management | Weight-bearing | |-------|--------|------------|----------------| | Early | 0β2 weeks | Rigid or semi-rigid cast applied in theatre; deep drain removed at 48 hours or when output is minimal; limb elevated | Strictly non-weight-bearing | | Intermediate | 2β6 weeks | Cast change at 10 to 14 days for wound check and suture removal (diabetics up to 3 weeks); re-cast snugly to shape the stump and control oedema | Remains non-weight-bearing until 6 weeks | | Late | 6 weeks and beyond | Once healed and the pad is stable (typically 6 to 8 weeks), refer to prosthetics for casting; gait training with a Syme prosthesis | Progression to full prosthetic weight-bearing | Instruct the patient never to rest the stump directly on the bed, which causes posterior pad migration. Prosthetic considerations - The Syme stump is naturally bulbous because the metaphyseal flare is preserved β this gives excellent suspension but needs a specialised socket (a medial window or expandable wall) for donning and doffing.
- A Syme amputation shortens the limb by about 5 to 7 cm (the combined height of the calcaneus and talus). The prosthesis must make up this height, which limits the vertical clearance for complex prosthetic feet β high-energy-return feet can be hard to fit.
- Because the native heel pad sits directly under the tibia, the stump is end-bearing and can take direct axial load β superior proprioception and the ability to walk short distances barefoot (e.g. to the bathroom at night), a major functional advantage over a transtibial amputation. Complications
- Incidence
- 10 to 15 percent (higher in severe PVD)
- Recognition
- Black, eschar-covered or dusky pad post-op; progressive breakdown; foul odour
- Prevention and management
- Prevention: confirm a patent posterior tibial artery pre-op, dissect the calcaneus strictly subperiosteally, avoid tight closure or casting. Management: minor marginal necrosis demarcates and heals by secondary intention; major full-thickness necrosis needs revision to transtibial amputation
- Incidence
- 5 to 10 percent
- Recognition
- Pad displaced posteriorly or medially, leaving the distal tibia covered only by thin skin; pain on weight-bearing; cannot tolerate the prosthesis
- Prevention and management
- Prevention: anchor the deep fascia to anterior tibial drill holes; use a rigid post-op cast to hold the pad central. Management: revision to mobilise and re-anchor the pad, or revision to transtibial amputation
- Incidence
- 5 to 15 percent
- Recognition
- Sharp, shooting pain on tapping the stump (Tinel sign) or when wearing the prosthesis; pain out of proportion to examination
- Prevention and management
- Prevention: identify all five sensory nerves, apply traction, cut sharply and let them retract well proximal to the stump end. Management: socket modification, ultrasound-guided injection, surgical excision with targeted muscle reinnervation (TMR) or burial in muscle or bone
- Incidence
- 5 to 20 percent (highest in diabetics with active sepsis)
- Recognition
- Erythema, swelling, purulent discharge, increasing pain, fever, systemic sepsis
- Prevention and management
- Prevention: use the two-stage technique for infected diabetic feet; meticulous haemostasis and a drain to prevent haematoma. Management: aggressive antibiotics, early washout and debridement; often needs revision to a higher level if bone is involved
- Incidence
- 5 to 10 percent
- Recognition
- Palpable bony prominence at the distal stump margins; point tenderness; skin irritation in the socket
- Prevention and management
- Prevention: rasp all bone edges smooth at the index operation; ensure the cut is perfectly horizontal. Management: socket relief; surgical excision of the spur if conservative measures fail
Viva & Exam Focus
SYMESYME β key technical principles
PADPAD β heel pad management
Danger structures β what threatens the stump, and the fix
The danger: it gives off the medial calcaneal branches that supply the pad. The fix: a patent posterior tibial artery is an absolute prerequisite β avoid dissection in the posteromedial corner behind the medial malleolus, and if Doppler shows no flow, a Syme is contraindicated.
The danger: damage to the fat lobules or their blood supply destroys the weight-bearing surface. The fix: dissect the calcaneus entirely subperiosteally β hug the bone with a sharp blade or elevator from superior to inferior and never incise into the fat pad.
The danger: without bony attachment the Achilles tendon and weight-bearing pull the pad posteriorly, leaving the tibia covered only by thin skin. The fix: anchor the deep fascia of the pad to drill holes in the anterior distal tibia and fibula with heavy non-absorbable suture.
The danger: the ankle has five major sensory nerves, and unaddressed ones form painful neuromas in the rigid socket. The fix: identify the tibial, sural, saphenous, superficial and deep peroneal nerves, apply gentle distal traction, transect cleanly and let them retract well off the stump end.
The danger: resecting too much tibia converts the procedure into a very short transtibial amputation, losing the metaphyseal flare. The fix: resect only the malleoli and a maximum of 1 cm of distal tibial articular surface, cut parallel to the ground in both planes.
The danger: definitive one-stage closure over severe forefoot sepsis leads to stump infection and failure. The fix: use the two-stage technique β disarticulate and loosely drape the pad over intact malleoli first, then resect and close definitively once infection is cleared.
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
βA 65-year-old diabetic man with a chronic infected midfoot ulcer and toe gangrene is referred for amputation. He was previously mobile and his pulses are absent. Explain your workup to decide whether he is a candidate for a Syme amputation.β
βDuring a classical Syme you are about to resect the calcaneus from the heel pad. What is the most critical technical principle at this stage, and which structure are you trying to protect?β
βSix months after a one-stage Syme for trauma, your patient has severe sharp shooting pain at the anterolateral stump every time they don the prosthesis, radiating proximally and exquisitely tender to light tapping. What is the most likely diagnosis and how would you manage it?β
Indications and prerequisites
- Indications: unsalvageable forefoot gangrene or infection, severe trauma, tumour, congenital deformity
- Prerequisite 1: patent posterior tibial artery (supplies the medial calcaneal branches)
- Prerequisite 2: pristine heel pad skin without ulceration
- Prerequisite 3: TcPO2 greater than 30 mmHg for healing
- Contraindication: ischaemic heel pad or absent posterior tibial flow
Surgical anatomy
- Heel pad: fat lobules in vertical fibrous septa β dissect subperiosteally to preserve them
- Arterial supply: medial calcaneal artery, a branch of the posterior tibial artery
- Five sensory nerves: tibial, sural, saphenous, superficial and deep peroneal
- Bone: the distal tibial metaphyseal flare is preserved for end-bearing and suspension
Operative steps (one-stage)
- Incision: lateral malleolus tip, across the ankle, to 1.5 cm below the medial malleolus; straight vertical plantar cut
- Anterior dissection: transect tendons and nerves (cut nerves high under traction)
- Disarticulation: open the capsule, cut the collateral ligaments, subluxate the talus
- Calcaneal dissection: strictly subperiosteal to protect the pad and blood supply
- Bony cut: remove the malleoli flush, parallel to the ground, preserve the flare
- Anchor the pad fascia to anterior tibial drill holes; close over a drain
Two-stage technique (Pinzur)
- Indication: active forefoot infection in diabetics
- Stage 1: disarticulate, remove the infected forefoot, leave the malleoli intact, loose approximation
- Stage 2: two to six weeks later when infection clears β resect the malleoli, anchor the pad, close
- Benefit: markedly reduces stump infection and failure in infected cases
Complications and management
- Heel pad necrosis: poor vascularity or septal damage β often needs revision to transtibial
- Posterior pad migration: failure to anchor β needs revision surgery
- Neuroma: failure to transect and bury nerves β causes prosthetic pain
- Stump infection: high risk in diabetics β mitigate with the two-stage procedure
Prosthetic considerations
- End-bearing: the patient can walk short distances barefoot, unlike a transtibial amputation
- Bulbous stump: excellent suspension but needs a windowed or expandable socket
- Leg length: shortens the limb by 5 to 7 cm, limiting clearance for complex prosthetic feet
Background & Evidence
History. The operation was described by the Scottish surgeon James Syme in 1843 as an end-bearing alternative to a below-knee amputation. Wagner (1977) established the modern principles of patient selection and the critical preoperative vascular assessment, and Pinzur and Smith (1995) developed the two-stage modification specifically for the infected diabetic foot. Biomechanics β why the level matters. Three properties make the Syme level functionally superior to a transtibial amputation when it heals. Energy expenditure: ambulation with a Syme increases energy cost by only 10 to 15 percent above normal gait, compared with 20 to 40 percent for a transtibial amputation β a difference that matters for elderly or dysvascular patients with limited cardiopulmonary reserve. Proprioception: the preserved natural heel pad, with its thick fibrous septa and fat lobules, compresses and distributes load uniformly across the distal tibial metaphysis and gives excellent sensory feedback. Lever arm: the Syme stump is a very long lever arm for the prosthesis, reducing the force the quadriceps and hip extensors must generate to control the limb in stance. Evidence summary. The highest-level summary is the systematic review of Braaksma and colleagues (2018), which confirmed the Syme level's superior energy efficiency over transtibial amputation and reported overall healing rates with predictors of success and failure. In the diabetic, infected subgroup the randomised work of Pinzur, Smith and Osterman (1995) established the two-stage modification as the standard of care by showing significantly lower stump infection and failure rates. Functional and satisfaction data (Morrison 2020; Slater 2024) confirm high satisfaction and end-bearing capacity, and inform modern prosthetic foot selection for the bulbous Syme stump.
References
Syme's amputation; the technical details essential for success
- Detailed the classical surgical technique for Syme amputation
- Emphasised the absolute necessity of subperiosteal dissection of the calcaneus
- Highlighted the importance of anchoring the heel pad to prevent posterior migration
Amputations of the foot and ankle. Current status
- Comprehensive review of amputation levels in the foot and ankle with emphasis on patient selection
- Stressed the importance of the posterior tibial artery for heel pad viability and the critical role of vascular assessment
- Established principles for managing diabetic foot infections requiring amputation at various levels
Syme ankle disarticulation in peripheral vascular disease and diabetic foot infection: the one-stage versus two-stage procedure
- Randomised controlled trial comparing one-stage and two-stage Syme amputations in a diabetic and dysvascular cohort
- Found significantly lower rates of stump infection and failure in the two-stage group
- Established the two-stage technique as the standard of care for infected diabetic forefeet
Syme Amputation: A Systematic Review
- Systematic review of all published evidence on Syme amputation outcomes
- Reported overall healing rates and identified predictors of success and failure
- Confirmed superior energy efficiency of the Syme level compared with transtibial amputation
Syme Amputation: Function, Satisfaction, and Prostheses
- Reported functional outcomes, patient satisfaction, and prosthetic use following Syme amputation
- High satisfaction rates with end-bearing capacity allowing unmounted ambulation
- Described modern prosthetic socket designs accommodating the bulbous distal stump
Effects of high-profile crossover feet on gait biomechanics in 2 individuals with Syme amputation
- Randomised crossover trial comparing prosthetic foot types specifically in Syme amputees
- Demonstrated that high-profile crossover feet improve gait biomechanics and walking speed
- Highlighted the unique prosthetic challenges of the bulbous Syme stump compared with transtibial amputees