Re-routing the tibialis anterior to correct dynamic forefoot supination — SPLATT for relapsed clubfoot, cerebral palsy and stroke · advanced
- SPLATT (split anterior tibial tendon transfer) is the standard procedure for dynamic forefoot supination in a relapsed idiopathic clubfoot after Ponseti correction and for flexible supination in cerebral palsy. It re-routes the lateral half (or the whole) of the tibialis anterior tendon to the lateral cuneiform or cuboid, converting a deforming supination force into a balanced dorsiflexion-eversion force. The foot MUST be supple and passively correctable — fixed bony deformity requires an osteotomy, not a tendon transfer.
- The prerequisite for any tendon transfer about the foot is a plantigrade, passively correctable foot. Fixed hindfoot varus, a stiff cavus, or a fixed equinus contracture must be addressed first — usually with a concurrent tendo-Achilles lengthening (TAL), a calcaneal osteotomy, or both. The transfer is tensioned with the foot held plantigrade in neutral dorsiflexion and about 5 degrees of eversion: too much tension produces over-correction (valgus), too little fails to correct the supination.
- Split versus whole: SPLATT preserves the medial half of the TA insertion, maintaining active dorsiflexion and inversion through the retained slip — preferred in patients who walk and need balanced ankle motion. The whole transfer gives a maximal eversion moment at the cost of the retained dorsiflexion-inversion function, and is reserved for severe or recurrent deformity or for non-ambulatory patients.
- The deep peroneal nerve and dorsalis pedis artery run between EHL and the TA tendon at the ankle — identify and protect them, and keep the anterior incision medial to the TA. The transferred tendon must pass SUPERFICIAL to the extensor retinaculum in the subcutaneous plane; a passage deep to the retinaculum tethers the excursion and the transfer functions poorly.
- Always assess the whole limb, not just the foot. The Coleman block test separates forefoot-driven from hindfoot-driven varus; the Silfverskiöld test unmasks a coexisting equinus that needs a concurrent TAL; and in cerebral palsy a hip adduction or hamstring contracture alters gait and often demands combined procedures (TAL, hamstring lengthening, rectus transfer).
When & Why
The indication. A dynamic forefoot supination deformity — the foot rolls into supination during the swing phase of gait but is plantigrade and passively correctable at rest — that has failed non-operative management (an AFO or brace adjustment, and botulinum toxin to the tibialis anterior). Three populations dominate: - Relapsed idiopathic clubfoot after Ponseti correction — dynamic supination develops in roughly 10 to 30 percent of treated feet; SPLATT is indicated when repeat casting and bracing fail. Most authors operate at age 3 to 8 years, before adaptive bony change sets in.
- Cerebral palsy (hemiplegia or diplegia, GMFCS levels I to III) — a dynamic equinovarus or supination pattern from swing-phase overactivity of the TA, often with a stance-phase equinus from the gastrocnemius-soleus.
- Stroke-related equinovarus — an acquired upper-motor-neuron spastic pattern in an adult; the equinus component is usually dominant, so an isolated TA transfer without TAL is rarely sufficient. Relative indications include mild-to-moderate dynamic supination in an ambulatory CP patient with a Silfverskiöld-negative ankle, a whole transfer for severe recurrence after failed SPLATT, dynamic supination in myelomeningocoele at a lower lumbar level with a supple foot, and a combined procedure with a calcaneal osteotomy once the fixed component of a rigid cavovarus foot has been corrected. Contraindications. Absolute: any fixed bony deformity — a rigid hindfoot varus, fixed cavus, or fixed equinus that does not correct passively (address it with a Dwyer calcaneal osteotomy, lateral column lengthening, or first metatarsal dorsiflexion osteotomy first); active infection in the field; and non-functioning peroneals (MRC grade less than 3) without a plan for augmentation (the transfer produces a stiff foot with no active eversion). Relative: severe spasticity uncontrolled by botulinum toxin or baclofen; a non-ambulatory patient without a functional goal the transfer can address (an AFO is simpler); age under 2 years (small tendon, small lateral cuneiform ossific nucleus, physis at risk); and previous anterior-compartment surgery with scarred planes. The one decision — split or whole. Every technique fixes the tendon into a bony tunnel at the lateral cuneiform or cuboid; the only real choice is whether the medial half of the TA is preserved:
- Split transfer (SPLATT)
- Ambulatory, walking children and adults
- Whole transfer
- Non-ambulatory, severe recurrence, or combined deformity
- Split transfer (SPLATT)
- Medial slip of the TA insertion preserved
- Whole transfer
- Entire insertion detached
- Split transfer (SPLATT)
- Medial slip maintains dorsiflexion with some inversion
- Whole transfer
- No residual inversion moment from the TA
- Split transfer (SPLATT)
- Moderate — the lateral half of the tendon
- Whole transfer
- Maximum — the whole tendon excursion
- Split transfer (SPLATT)
- Low — less than 5 percent
- Whole transfer
- Higher — 10 to 15 percent valgus reported
- Split transfer (SPLATT)
- 3 to 8 percent
- Whole transfer
- 8 to 15 percent
- Split transfer (SPLATT)
- Bony tunnel in the lateral cuneiform, pull-out button or interference screw
- Whole transfer
- Same, but a larger 5 to 6 mm tunnel for the whole tendon
Consent specifically for nerve injury (deep and superficial peroneal nerves, less than 2 percent), wound problems (5 to 8 percent), over-correction into valgus (5 to 10 percent, higher in whole transfer), under-correction (3 to 8 percent), growth-plate injury in children (less than 1 percent with fluoroscopy), and the planned period of cast immobilisation and non-weight-bearing. Setup. Supine with a sandbag under the ipsilateral hip to bring the foot to neutral or slight internal rotation (the foot tends to externally rotate in supine); the foot projects beyond the table end so the assistant can hold the ankle. A thigh tourniquet for cases over 60 minutes, or a calf tourniquet at 300 mmHg for shorter ones; exsanguinate with an Esmarch before inflation. General anaesthesia, with a popliteal sciatic block for analgesia (discuss with the anaesthetist — motor blockade of the peroneals can delay the early assessment of active eversion). A single dose of a first-generation cephalosporin at induction for clean bony surgery with hardware.
The Operation
The goal is to harvest the lateral half (or whole) of the tibialis anterior tendon, re-route it subcutaneously across the dorsum of the foot superficial to the extensor retinaculum, and fix it under tension into a bony tunnel in the lateral cuneiform so it acts as a dorsiflexor-evertor. The exposure — an anterior incision kept medial to the TA with the deep peroneal nerve and dorsalis pedis artery identified and protected between EHL and TA — is the heart of the operation.

Operative sequence
- Supine, sandbag under the ipsilateral hip, foot beyond the table end; thigh or calf tourniquet; exsanguinate and inflate.
- Palpate the TA tendon along the dorsomedial foot and trace it to its insertion on the medial cuneiform and the base of the first metatarsal; mark the anterior harvest incision just medial to the tendon, and the dorsal/lateral incision over the lateral cuneiform.
- A 3 to 4 cm longitudinal incision over the TA tendon from just proximal to the medial cuneiform to the naviculocuneiform joint, placed just MEDIAL to the tendon — well away from the deep peroneal nerve and dorsalis pedis artery, which lie laterally between EHL and the TA.
- Incise skin and subcutaneous tissue down to the paratenon (a thin, glistening, vascularised layer — preserve it, it supplies blood and gliding); define the full width of the TA tendon and trace it to its insertion.
- BEFORE any tendon division, identify the deep peroneal nerve and dorsalis pedis artery. Palpate the dorsalis pedis pulse between EHL (medial) and EDL (lateral) at the ankle, then gently retract EHL to expose the bundle lying between EHL and the TA, just deep to the extensor retinaculum.
- Pass a blunt right-angle forceps from medial to lateral UNDER the TA tendon to define the deep plane. If the pulse is not felt, use a Doppler or momentarily deflate the tourniquet to confirm the vessel is intact before proceeding.
- Insert a blunt right-angle forceps between the medial and lateral fibre bundles at the musculotendinous junction (about 5 to 6 cm proximal to the ankle) and spread gently to create the plane; carry the split distally for 4 to 5 cm along the natural fibre cleavage, keeping within the tendon substance.
- Under direct vision, divide the LATERAL half at its insertion with a number 15 blade, leaving a 1 to 2 mm stump; tag it with a 2-0 stay suture. Leave the MEDIAL half intact at its insertion — it continues to provide active dorsiflexion and inversion.
- For a WHOLE transfer, detach the entire tendon from its insertion; no medial slip is retained, and a larger 5 to 6 mm tunnel is used later.
- Make a second 2 to 3 cm longitudinal incision over the lateral cuneiform (about 1 cm distal to the ankle joint, midway between the bases of the second and fourth metatarsals); the cuboid is an alternative, more lateral site.
- Pass a curved tendon passer or long clamp from the lateral incision, SUBCUTANEOUSLY across the dorsum, to emerge from the anterior incision. The tunnel MUST lie superficial to the extensor retinaculum. Grasp the stay suture and draw the lateral half through from medial to lateral.
- Palpate the dorsum — the transferred tendon should be palpable just under the skin, not fixed to or deep to the retinaculum. If it feels tethered, re-pass it superficial.
- Confirm the lateral cuneiform visually or with fluoroscopy; in children use the image to locate the physis (it lies proximally in the lateral cuneiform).
- Drill a single-pass tunnel (4.5 to 5.0 mm in adults; 3.2 to 4.0 mm in children aged 3 to 8) from dorsolateral to plantaromedial, in the line of pull of the transferred tendon (about 30 to 45 degrees from vertical, angled medially and plantarly). In the growing child, direct the tunnel into the metaphyseal (distal) portion under fluoroscopy and confirm the drill stays within bone on AP and lateral.
- Whip-stitch the tendon end with a 2-0 or 0 braided polyester on a straight needle; pass the suture through the tunnel dorsal to plantar and draw the tendon in until it is snug.
- Pull-out button (classic): the suture exits the plantar skin and is tied over a felt-padded button, guaranteeing compression of the tendon within the tunnel. Interference screw (alternative): a 5 to 6 mm absorbable biotendosis screw secures the tendon — no plantar button, no external suture, but more demanding and not re-tensionable. Suture anchor (young children): avoids a transosseous tunnel across the physis but has lower pull-out strength.
- TENSION is the critical step: hold the foot in NEUTRAL dorsiflexion and about 5 DEGREES of eversion before tying. Over-tension gives a valgus foot; under-tension fails to correct the supination.
- Close the anterior and lateral incisions in layers (3-0 absorbable subcutaneous, 4-0 subcuticular skin). The pull-out suture exits the plantar skin through a separate stab, tied over felt between button and skin.
- Apply a well-padded below-knee cast with the foot plantigrade (neutral dorsiflexion, ankle at 90 degrees to the tibia). If a TAL was performed concurrently, the cast position is dictated by the TAL protocol — initial equinus, gradually brought to neutral.
Concurrent procedures are the rule rather than the exception and are performed BEFORE the TA transfer so the plantigrade position the transfer is tensioned against is defined: - Tendo-Achilles lengthening (TAL) — the commonest combined procedure, performed in an estimated 50 to 70 percent of transfers, for a Silfverskiöld-positive equinus. Either a percutaneous Hoke triple hemisection or an open sagittal Z-lengthening; performed first, with the foot then cast in 15 to 20 degrees of equinus for 3 to 4 weeks before gradual dorsiflexion to neutral.
- Posterior tibial tendon (PTT) lengthening — intramuscular coronal-split lengthening (the Barnes and Herring technique) when there is a mixed swing-phase TA supination AND a stance-phase PTT-driven varus.
- Calcaneal osteotomy — a lateral closing-wedge (Dwyer) or sliding osteotomy for the rigid cavovarus foot; performed and fixed first, then the transfer tensioned against the corrected hindfoot.
The deep peroneal nerve and dorsalis pedis artery run between EHL and the TA tendon at the ankle, just deep to the extensor retinaculum. Keep the anterior incision medial to the TA, identify the bundle between EHL and the TA before any instrumentation, and pass the tendon tunnel SUPERFICIAL to the retinaculum — a deep passer can stretch, contuse or divide the nerve and artery. An intra-operative injury is managed with primary epineural repair (8-0 nylon); a recognised post-operative motor loss is explored within 3 months.
Make the incision just medial to the TA tendon, not directly over it — this keeps you away from the neurovascular bundle. Identify the full width of the TA first, pass a blunt right-angle forceps from medial to lateral under the tendon to define the deep plane, and always palpate the dorsalis pedis pulse; if it is not felt, use a Doppler or deflate the tourniquet momentarily to confirm the vessel is intact before proceeding.
A tendon passed deep to the extensor retinaculum runs against the retinaculum, loses excursion, and works poorly — the transfer is stiff and ineffectual. The entire re-route from the anterior incision to the dorsolateral foot should be subcutaneous. If the tendon feels tethered or deep, re-pass it superficial to the retinaculum.
Fix the tendon with the foot in neutral dorsiflexion and no more than 5 degrees of eversion. Over-tension (the foot held in excessive eversion when the button is tied) produces a valgus over-correction — the opposite deformity; under-tension (a loose tendon at the tunnel entrance) fails to correct the supination and fills with scar. The tendon must be pulled through until it is snug within the cancellous bone.
Confirm the lateral cuneiform under fluoroscopy and keep the drill parallel to and away from the proximal physis, directing the tunnel into the metaphyseal (distal) portion of the bone. Confirm the drill path is within the bone on both AP and lateral — the lateral cuneiform is narrow mediolaterally, and an entry point placed too far lateral falls off the bone. In the very young, a metaphyseal suture anchor avoids a transphyseal tunnel altogether.
Aftercare & Complications
Rehabilitation | Phase | Timing | Immobilisation | Activity | |-------|--------|----------------|----------| | 1 | 0–4 weeks | Below-knee cast, non-weight-bearing (plantigrade; equinus if concurrent TAL) | Toe-touch weight-bearing with crutches only | | 2 | 4–6 weeks | Cast change — remove the plantar button and pull-out suture; re-cast plantigrade | Weight-bearing as tolerated in the cast | | 3 | 6–8 weeks | Walking boot or solid AFO | Active dorsiflexion and eversion; gentle passive stretching of any residual equinus | | 4 | 8–12 weeks | Stiff-soled shoe or UCBL insert for residual mild deformity | Physiotherapy — gait training, proprioception, strengthening of dorsiflexors and evertors | | 5 | Beyond 12 weeks | Normal footwear | Sport at 4 to 6 months; annual review in children until skeletal maturity | If a TAL was performed, its protocol dictates the first 3 to 4 weeks: the foot is cast in 15 to 20 degrees of equinus to protect the lengthened tendon, then progressively dorsiflexed with serial cast changes to neutral. The TA transfer benefits from early plantigrade positioning but must not compromise the TAL repair. Complications
- Incidence
- 5–10 percent (whole higher than split)
- Recognition
- Foot sits pronated/everted at rest; weight-bearing on the medial border; active dorsiflexion produces ankle valgus
- Prevention & management
- Tension in neutral dorsiflexion and under 5 degrees of eversion; prefer SPLATT; assess peroneals. If under 10 degrees and asymptomatic, observe; otherwise shoe modification, AFO, or revision transfer
- Incidence
- 3–8 percent
- Recognition
- Persistent forefoot supination on active dorsiflexion; swing-phase supination on gait examination
- Prevention & management
- Snug tendon in the tunnel; correct the equinus (TAL); avoid a retinacular tether. Gait analysis; repeat transfer if early and supple; calcaneal osteotomy if late and fixed
- Incidence
- less than 2 percent
- Recognition
- Sensory loss in the first web space; weak dorsiflexion and toe extension; loss of anterior-compartment bulk
- Prevention & management
- Incision medial to the TA; identify the nerve between EHL and the TA; blunt tunnelling. Intra-operative primary neurorrhaphy; post-operative exploration within 3 months; EMG at 3 months if no recovery
- Incidence
- 1–3 percent (lateral incision)
- Recognition
- Numbness over the dorsum (except the first web space); painful neuroma at the lateral scar
- Prevention & management
- Careful lateral skin incision; gentle subcutaneous retraction. Neuroma excision and burial in muscle if pain persists beyond 6 months
- Incidence
- less than 1 percent with fluoroscopy
- Recognition
- Progressive lateral-column shortening or angular deformity on follow-up radiographs
- Prevention & management
- Fluoroscopy to locate the physis; metaphyseal (distal) tunnel; suture anchor to avoid a transphyseal tunnel. Monitor; guided growth or corrective osteotomy if deformity develops
- Incidence
- 5–8 percent (lateral incision and plantar button site)
- Recognition
- Wound breakdown; exposed tendon or bone; pressure necrosis at the button
- Prevention & management
- Gentle skin handling; well-padded cast; felt-pad the button; avoid an incision over a bony prominence. Dressings and antibiotics for superficial loss; debridement and NPWT or flap for full-thickness; remove the button early if necrosis
- Incidence
- 1–3 percent
- Recognition
- Loss of correction; palpable gap at the lateral cuneiform; cannot actively dorsiflex and evert
- Prevention & management
- Adequate tunnel (at least 4.5 mm); non-absorbable whip-stitch; plantigrade tension; 6 weeks in a cast. Acute rupture (under 4 weeks) — re-explore and re-fix; chronic failure — revision with an interference screw or suture anchor at a different site
- Incidence
- 1–2 percent
- Recognition
- Disproportionate pain, allodynia, swelling, temperature and colour change in the foot
- Prevention & management
- Minimise trauma; aggressive analgesia; early motion within the cast. Pain-team referral, sympathetic blockade, gabapentinoids; vitamin C 500 mg daily for 50 days to reduce incidence
Viva & Exam Focus
TATTTATT — the key principles of the transfer
SPLATTSPLATT — patient selection and prerequisites
Critical danger structures and exam traps
Location: between EHL and the TA at the ankle, deep to the extensor retinaculum. Risk: stretched, contused or divided during the anterior approach and the subcutaneous tunnelling. Protection: identify the bundle between EHL and the TA before instrumenting; the tunnel must pass SUPERFICIAL to the retinaculum; palpate the pedicle before passing the tendon passer.
Location: the ossification centres of the lateral cuneiform and cuboid are present in the skeletally immature foot, with the physis proximal in the lateral cuneiform. Risk: an eccentrically drilled tunnel near the physis causes growth arrest or progressive deformity. Protection: use intra-operative fluoroscopy; drill the metaphyseal (distal) portion of the lateral cuneiform, or use a metaphyseal suture anchor instead of a transphyseal tunnel.
Location: the superior and inferior extensor retinacula overlie the anterior-compartment tendons. Risk: a tendon passed deep to them tethers — the transfer works against the retinaculum rather than with the excursion of the muscle, giving a stiff, ineffectual result. Protection: pass the transferred tendon superficial to the retinaculum in the subcutaneous plane throughout the re-route.
Mechanism: the transferred TA — especially a whole transfer, or one tensioned in excessive eversion — pulls the foot into valgus, the opposite deformity. Recognition: the foot sits pronated/everted at rest; active dorsiflexion produces valgus; the patient walks on the medial border. Prevention: tension with no more than 5 degrees of eversion; prefer SPLATT in ambulatory patients; weak peroneals raise the risk even with a split transfer.
Why concurrent TAL is critical: dynamic supination frequently coexists with equinus. The TA is recruited as a secondary dorsiflexor; once transferred, its equinus contribution is removed and a fixed equinus is unmasked. The trap: an isolated TA transfer without examining for a Silfverskiöld-positive equinus. If the ankle cannot dorsiflex to neutral with the knee extended, a TAL (or a gastrocnemius recession if the contracture is gastrocnemius-only) must be added to reach a plantigrade foot.
Pathophysiology: dynamic supination is a relative imbalance between the TA and the peroneals. Weakness of peroneus brevis or longus — from nerve injury, myelomeningocoele, or CP — unmasks the TA's supination moment. The trap: a patient with an apparently pure dynamic supination may have underlying peroneal weakness; an anatomically perfect SPLATT then produces a stiff foot with no balanced active eversion. Prevention: manually test the peroneals (foot eversion against resistance). If peroneal strength is less than MRC grade 3, consider augmentation or a posterior tibial tendon transfer.
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
“A 4-year-old boy with a history of Ponseti-corrected idiopathic clubfoot presents with dynamic supination of the forefoot during the swing phase of gait. The foot is plantigrade at rest and can be passively corrected into valgus. His ankle dorsiflexes to neutral with the knee flexed but to 5 degrees of plantarflexion with the knee extended. How do you manage this child?”
“A 12-year-old boy with spastic diplegic cerebral palsy (GMFCS II, independent community ambulator) has a dynamic supination deformity of his right foot. The foot is supple and correctable, with a 10-degree equinus contracture positive on Silfverskiöld testing (gastrocnemius contracture). The peroneals are graded MRC 4. He trips frequently outdoors and has had no previous surgery. Describe your management.”
“You have just performed a SPLATT with a pull-out suture over a plantar button for a 6-year-old girl with relapsed clubfoot. The foot was positioned in neutral dorsiflexion and 5 degrees of eversion when the button was tied. At the first cast change (4 weeks) the foot is in 10 degrees of valgus. The button is intact and the wound is clean. What has gone wrong and how do you manage it?”
Indications
- Dynamic forefoot supination in relapsed idiopathic clubfoot after Ponseti, failing repeat casting and bracing
- Spastic dynamic supination in cerebral palsy (hemiplegia, GMFCS I to III) — a supple, correctable foot
- Stroke-related supination with functional impairment and tripping
- Prerequisite: the foot must be SUPPLE and passively correctable — fixed bony deformity needs an osteotomy first
- Prerequisite: the ankle must dorsiflex to neutral — if not, a concurrent TAL or gastrocnemius recession is mandatory
Pre-operative assessment
- Passive correctability — if the foot cannot be corrected to neutral or valgus, a transfer alone will fail
- Silfverskiöld test — gastrocnemius versus gastrocnemius-soleus equinus; TAL for combined, Strayer for gastrocnemius-only
- Coleman block test — forefoot-driven versus rigid hindfoot varus
- Peroneal strength (MRC grade) — if less than 3, consider a whole transfer or augmentation
- Selective motor control of the TA — the patient must be able to activate it independently in swing for the transfer to work
- Standing foot radiographs — document alignment, exclude tarsal coalition, assess the physis in children
Surgical anatomy
- TA origin: lateral tibial condyle, proximal lateral tibial shaft, interosseous membrane; belly in the anterior compartment
- TA insertion: medial cuneiform and base of the first metatarsal
- Deep peroneal nerve and dorsalis pedis: between EHL (medial) and the TA (lateral) — identify before instrumenting
- Extensor retinaculum: the transferred tendon must pass SUPERFICIAL to it — a deep passage tethers excursion
- Lateral cuneiform: the landing zone — the tunnel is drilled dorsolateral to plantaromedial in the line of pull
- In children the physis is proximal in the lateral cuneiform — drill the metaphyseal (distal) portion under fluoroscopy
Operative technique — key steps
- Position supine with a sandbag under the ipsilateral hip; tourniquet; general anaesthesia
- Anterior incision medial to the TA — identify and protect the deep peroneal nerve and dorsalis pedis
- Split the TA: a right-angle forceps between the fibre bundles; the lateral half is detached at the insertion
- For a whole transfer, detach the entire tendon — no retained medial slip
- Create a subcutaneous tunnel to the dorsolateral foot — MUST be superficial to the extensor retinaculum
- Pass the lateral half through the tunnel, tagged with a 2-0 stay suture
- Drill a 4.0 to 5.0 mm tunnel in the lateral cuneiform — under fluoroscopy in children to avoid the physis
- Fix with a whip-stitched 2-0 braided polyester: pull-out button, or a biotendosis interference screw
- Tension in NEUTRAL DORSIFLEXION and 5 degrees of EVERSION when the button is tied
- Close in layers; below-knee cast — equinus for 3 to 4 weeks if concurrent TAL, then neutral
Technique variants — SPLATT versus whole
- SPLATT (split): medial half preserved — active dorsiflexion and inversion retained; moderate eversion; lower over-correction risk
- Whole: entire TA detached — maximal eversion moment but loss of dorsiflexion-inversion; higher valgus risk (10 to 15 percent)
- SPLATT preferred for: ambulatory patients, mild-to-moderate deformity, first-time surgery
- Whole indicated for: severe recurrence after failed SPLATT, non-ambulatory patients, combined procedures
- Fixation: pull-out button (classic, tension check at cast change), interference screw (no external button, higher cost), suture anchor (young children, avoids a transosseous tunnel)
Complications
- Over-correction valgus (5 to 10 percent): tension in neutral to 5 degrees eversion; prefer SPLATT; treat with neutral cast, AFO, or surgical release
- Under-correction / recurrence (3 to 8 percent): inadequate tension, unrecognised equinus, or retinacular tether; revise, or calcaneal osteotomy if fixed
- Deep peroneal nerve injury (less than 2 percent): identify before instrumenting; keep medial to the TA; explore within 3 months for complete motor loss
- Growth-plate injury (less than 1 percent): fluoroscopy in children; avoid the proximal physis of the lateral cuneiform
- Wound dehiscence (5 to 8 percent): commonest at the lateral incision and button site — gentle handling, well-padded cast, early button removal
- Tendon rupture / pull-out (1 to 3 percent): protect in a cast for 6 weeks; acute rupture under 4 weeks needs re-exploration
- CRPS (1 to 2 percent): early mobilisation, analgesia, consider vitamin C prophylaxis
Post-operative protocol
- 0 to 4 weeks: non-weight-bearing cast (neutral; equinus if concurrent TAL); plantar button intact
- 4 to 6 weeks: cast change — remove button and pull-out suture; weight-bearing as tolerated in a neutral cast
- 6 to 8 weeks: walking boot or AFO; active dorsiflexion-eversion; progress to full weight-bearing
- 8 to 12 weeks: physiotherapy — gait training, proprioception, strengthening of evertors and dorsiflexors
- Beyond 12 weeks: normal footwear; sport at 4 to 6 months; annual review in children until skeletal maturity
- Long term: watch for late recurrence with growth or progressive neurological disease
Clinical contexts
- Relapsed clubfoot after Ponseti: SPLATT at 3 to 8 years for dynamic supination; concurrent TAL in 50 to 70 percent; over 90 percent satisfactory
- Cerebral palsy: SPLATT for ambulatory patients (GMFCS I to III); often with TAL, PTT lengthening, hamstring or rectus work; operate before bony adaptation
- Stroke: acquired spastic equinovarus — SPLATT plus TAL is the rule; pre-operative gait analysis is essential
- Myelomeningocoele: dynamic supination at lower lumbar levels needs careful assessment of peroneal strength first
- Non-ambulatory patient: SPLATT is rarely indicated — AFO management is simpler and avoids surgical morbidity
Background & Evidence
Epidemiology. Dynamic forefoot supination develops in roughly 10 to 30 percent of feet after successful Ponseti correction of an idiopathic clubfoot — the foot is plantigrade at rest but rolls into supination during the swing phase of gait. In cerebral palsy it is the characteristic swing-phase deformity of hemiplegic and diplegic gait, and in adults it follows an upper-motor-neuron stroke as an acquired spastic equinovarus. It must be distinguished from residual or recurrent bony deformity (a stiff, uncorrected clubfoot, or fixed cavovarus), which is not a dynamic problem and is not corrected by a tendon transfer. Pathoanatomy. The tibialis anterior is the primary ankle dorsiflexor but also inverts the foot in dorsiflexion. Dynamic supination is a relative imbalance: overactivity of the TA in swing, often compounded by weakness of the peroneals (peroneus brevis and longus), produces swing-phase supination. Equinus frequently coexists — the TA is recruited as a secondary dorsiflexor, and once it is transferred its equinus contribution is removed, unmasking a fixed equinus contracture. This is why a Silfverskiöld-positive equinus is addressed first. Surgical anatomy of the transfer. The TA originates from the lateral tibial condyle, the proximal half of the lateral tibial shaft, the interosseous membrane and the deep fascia; its tendon becomes tendinous about 5 to 6 cm proximal to the ankle, passes under the extensor retinacula lying medial to EHL, and inserts into the medial cuneiform and the base of the first metatarsal. At the ankle the tendon is 8 to 12 mm wide (enough for a longitudinal split into two 4 to 6 mm slips) with an excursion of 15 to 25 mm. The split follows the natural cleavage between the medially and laterally derived fibre bundles. The deep peroneal nerve (L4, L5, S1) and dorsalis pedis artery run between EHL and the TA, just deep to the retinaculum — the structures at risk throughout the operation; the superficial peroneal nerve emerges distally between peroneus longus and EDL and is at risk at the lateral incision. The lateral cuneiform is the preferred landing zone because it lies in the line of pull of the TA, has adequate bone stock in children over 3 years, and is dorsal to the subtalar joint axis so the transfer restores eversion; the cuboid is more lateral (more eversion moment) but needs a longer tunnel and has thinner stock. Key evidence. Hoffer et al. (1974) described the split transfer in 26 patients with spastic hemiplegia and established the SPLATT as the definitive procedure for dynamic supination in CP. Barnes and Herring (1991) reported 82 percent good-to-excellent results in 28 patients who had SPLATT combined with intramuscular PTT lengthening, with a concurrent TAL in 54 percent — establishing that the equinus must be addressed at the same sitting. For relapsed clubfoot, Mubarak and Van Valin (2009) reported complete correction in 91 percent of 32 feet at a mean 3.5-year follow-up, and Thompson et al. (2009) a 96 percent satisfactory outcome in 23 children at 5 years. Biomechanically, Knutsen et al. (2015) showed the split gives the most balanced correction of forefoot supination while preserving hindfoot motion, whereas the whole transfer over-everts. Mindler et al. (2020) confirmed with instrumented gait analysis that the transfer normalises forefoot supination (12.4 down to 5.2 degrees relative to the tibia), approaching the kinematics of healthy children. Patient selection is the key determinant of success: Limpaphayom et al. (2015) reported an 84 percent success rate in 73 CP feet, with failure predicted by fixed contracture, poor selective motor control and severe spasticity.
References
The split anterior tibial tendon transfer in the treatment of spastic varus hindfoot of childhood
- Original description of the split anterior tibial tendon transfer (SPLATT) — 26 patients with spastic hemiplegia
- The procedure converts the TA from a supinator to a dorsiflexor-evertor by transferring the lateral half to the lateral cuneiform
- Demonstrated durable correction of varus/supination during gait in the ambulatory CP population
Combined split anterior tibial-tendon transfer and intramuscular lengthening of the posterior tibial tendon in spastic cerebral palsy
- 28 patients with CP (spastic hemiplegia or diplegia) underwent SPLATT combined with intramuscular lengthening of the posterior tibial tendon
- 82 percent good-to-excellent results at a mean 4.2-year follow-up; a concurrent TAL was performed in 54 percent of cases
- Demonstrated the importance of addressing the equinus contracture at the same sitting
Normalization of Forefoot Supination After Tibialis Anterior Tendon Transfer for Dynamic Clubfoot Recurrence
- Gait-analysis study of 25 clubfeet (17 children) with dynamic recurrence after Ponseti treatment who underwent a TATT
- Forefoot supination relative to the tibia at initial contact decreased from 12.4 to 5.2 degrees post-operatively (controls 6.0 degrees)
- Demonstrated normalisation of dynamic clubfoot-recurrence kinematics with joint-sparing TATT surgery
How do different anterior tibial tendon transfer techniques influence forefoot and hindfoot motion?
- Biomechanical comparison of whole transfer, split transfer (SPLATT), and medial transfer of the anterior tibial tendon
- SPLATT produced the most balanced correction of forefoot supination while preserving hindfoot motion
- Whole transfer generated excessive eversion moment; medial transfer was insufficient to correct forefoot supination
The split anterior tibialis tendon transfer procedure for spastic equinovarus foot in children with cerebral palsy: results and factors associated with a failed outcome
- Retrospective study of 73 feet in 58 children with CP who underwent a split anterior tibialis tendon transfer for a spastic equinovarus foot
- Overall success rate of 84 percent; failure was associated with fixed contracture, poor selective motor control and severe spasticity
- Demonstrated that patient selection (dynamic versus fixed deformity, GMFCS level) is the key determinant of outcome