Post-traumatic vs Inflammatory | Conservative First | TEA for End-Stage | Ulnar Nerve
- RA is most common inflammatory cause - bilateral involvement, systemic disease
- Post-traumatic follows fractures, dislocations, or chronic instability
- Conservative management first - NSAIDs, injections, activity modification
- TEA indicated when conservative fails, age over 60, low demand
- Ulnar nerve commonly affected - cubital tunnel syndrome in 30-50%
- “RA elbow arthritis: bilateral, systemic, medical management first
- “Post-traumatic: history of fracture/dislocation, may have instability
- “TEA best outcomes in RA (90% 10-year survival) vs trauma (75%)
- “Ulnar nerve must be assessed and protected in all elbow procedures

Rheumatoid arthritis is the most common inflammatory cause, typically bilateral with systemic features. Post-traumatic follows fractures, dislocations, or chronic instability. Always assess for instability and ulnar nerve involvement.
Medical management is first-line for RA (DMARDs, biologics). Activity modification and injections for post-traumatic. Surgery reserved for failed conservative management in appropriate patients.
Total elbow arthroplasty indicated when conservative fails, age over 60, low demand. RA patients have best outcomes (90% 10-year survival). Trauma patients have higher failure rates (75% 10-year survival).
Ulnar nerve commonly affected in elbow arthritis (30-50% cubital tunnel syndrome). Must assess preoperatively and protect during surgery. Nerve complications occur in 10-15% of TEA procedures.
- Etiology
- Inflammatory (RA)
- Treatment
- TEA both sides (staged)
- Key Consideration
- Best outcomes - 90% 10-year survival
- Etiology
- Post-traumatic OA
- Treatment
- Conservative, debridement if needed
- Key Consideration
- TEA too young - wait until over 60 if possible
- Etiology
- Acute fracture
- Treatment
- Primary TEA
- Key Consideration
- 75% 10-year survival, faster rehab than ORIF
- Etiology
- Post-traumatic OA
- Treatment
- Avoid TEA, consider interposition or fusion
- Key Consideration
- TEA contraindicated - high failure risk
RAPIDElbow Arthritis Causes
Hook:RAPID progression of elbow arthritis - RA and post-traumatic are most common.
FRAILTEA Indications
Hook:FRAIL patients are ideal for TEA - elderly, low demand, inflammatory causes.
Overview and Epidemiology
Elbow arthritis encompasses inflammatory and degenerative conditions affecting the elbow joint, leading to pain, stiffness, and functional limitation. Unlike hip and knee arthritis where primary osteoarthritis predominates, elbow arthritis is most commonly post-traumatic or inflammatory (rheumatoid arthritis).
- Prevalence: Less than 1% of population (much less common than hip/knee)
- Primary OA: Rare, usually in manual laborers with repetitive stress
- RA: Most common inflammatory cause, typically bilateral
- Post-traumatic: Common after fractures, dislocations, or chronic instability
- Age: Peak 50-70 years
- Gender: RA more common in females, post-traumatic equal distribution
Elbow arthritis significantly impacts activities of daily living including eating, personal hygiene, and work. Unlike lower extremity arthritis, elbow involvement affects both dominant and non-dominant arms, with bilateral RA causing severe functional impairment.
Elbow arthritis differs from hip/knee arthritis: primary OA is rare (usually post-traumatic or inflammatory), the joint tolerates less cartilage loss before symptoms, and treatment options are more limited. TEA is less common than hip/knee replacement due to higher complication rates and stricter patient selection criteria.
Anatomy and Pathophysiology
- Ulnohumeral joint: Primary hinge (flexion-extension 0-150°)
- Radiocapitellar joint: Valgus stability, load transmission
- Proximal radioulnar joint: Forearm rotation (pronation-supination)
- Medial collateral ligament (MCL): Primary valgus stabilizer
- Lateral collateral ligament (LCL): Primary varus stabilizer
- Capsule: Anterior and posterior capsular constraints
- Muscles: Dynamic stabilizers (biceps, triceps, brachialis)
- Synovial inflammation → cartilage destruction
- Pannus formation erodes articular surfaces
- Ligamentous laxity → instability
- Bilateral involvement common
- Cartilage damage from initial injury
- Malalignment → abnormal load distribution
- Instability → recurrent trauma
- Osteochondral defects → progressive degeneration
- Rare, usually manual laborers
- Repetitive stress → cartilage wear
- Loose bodies common
- Usually unilateral
Classification Systems
Classification by Cause
Type I: Inflammatory
- Rheumatoid arthritis (most common)
- Systemic lupus erythematosus
- Psoriatic arthritis
- Juvenile idiopathic arthritis
- Features: Bilateral, systemic, synovial inflammation
Type II: Post-traumatic
- After fractures (distal humerus, radial head, olecranon)
- After dislocations (simple or complex)
- Chronic instability
- Features: Unilateral, history of trauma, may have malalignment
Type III: Primary Degenerative
- Primary osteoarthritis (rare)
- Hemophilic arthropathy
- Neuropathic (Charcot)
- Features: Usually unilateral, specific risk factors
Understanding etiology guides treatment approach and predicts outcomes.
Clinical Assessment
- Pain: Location, character, timing (activity-related vs rest)
- Stiffness: Loss of flexion-extension, pronation-supination
- Instability: Feeling of giving way, recurrent dislocations
- Trauma history: Previous fractures, dislocations
- Systemic symptoms: RA features (morning stiffness, bilateral, systemic)
Physical Examination
- Swelling, deformity, scars
- Muscle atrophy (especially triceps)
- Carrying angle (cubitus valgus/varus)
- Joint line tenderness
- Ulnar nerve (cubital tunnel)
- Loose bodies
- Synovial thickening (RA)
- Flexion-extension: Normal 0-150°, functional arc 30-130°
- Pronation-supination: Normal 80° each direction
- Stiffness pattern: Capsular vs mechanical block
- Valgus stress: MCL integrity
- Varus stress: LCL integrity
- Tinel's sign: Ulnar nerve at cubital tunnel
- Instability: Apprehension, pivot shift
- Ulnar nerve: Sensation (ulnar 1.5 digits), motor (interossei, FDP)
- Median nerve: Sensation, motor (thenar muscles)
- Radial nerve: Sensation, motor (wrist/finger extension)
CUBITALUlnar Nerve Assessment
Hook:CUBITAL tunnel syndrome is common - assess and protect the ulnar nerve.
Investigations
- AP and lateral views: Joint space narrowing, osteophytes, loose bodies
- Stress views: Instability assessment
- Comparison views: Contralateral elbow for reference
- 3D reconstruction: Bone stock assessment, version analysis
- Loose body detection: More sensitive than X-ray
- Preoperative planning: For TEA or debridement
- Cartilage assessment: Early changes, osteochondral defects
- Synovial evaluation: RA pannus, synovitis
- Ligament integrity: MCL, LCL assessment
- Ulnar nerve: Cubital tunnel evaluation
- RA workup: RF, anti-CCP, ESR, CRP
- Infection markers: If concern for septic arthritis
- Bone health: If considering TEA
- EMG/NCS: Ulnar nerve function if symptomatic
- Brachial plexus: If neurological symptoms
Differential Diagnosis
- Key Distinguishing Features
- Bilateral, symmetrical, synovitis, morning stiffness, systemic features
- Confirmatory Workup
- RF, anti-CCP, ESR/CRP; periarticular erosions on X-ray
- Key Distinguishing Features
- Prior fracture/dislocation, often unilateral, may have malalignment or instability
- Confirmatory Workup
- History; X-ray asymmetric joint space loss, deformity, loose bodies
- Key Distinguishing Features
- Manual worker/athlete, dominant arm, locking from osteophytes, terminal-arc pain
- Confirmatory Workup
- X-ray: olecranon/coronoid osteophytes, loose bodies, preserved mid-arc space
- Key Distinguishing Features
- Acute hot swollen joint, fever, severe pain on micro-movement
- Confirmatory Workup
- Aspiration (WCC, Gram stain, culture, crystals), CRP — urgent
- Key Distinguishing Features
- Acute self-limiting attacks, tophi, chondrocalcinosis
- Confirmatory Workup
- Aspirate for crystals under polarised light; serum urate
- Key Distinguishing Features
- Ulnar paraesthesia/weakness without true joint destruction
- Confirmatory Workup
- Tinel/elbow flexion test; nerve conduction studies
Primary Osteoarthritis of the Elbow — Clinical Syndrome and Joint-Preserving Surgery
Primary (idiopathic) osteoarthritis of the elbow is uncommon — a small minority of all elbow arthritis — but has a distinctive, high-yield clinical signature that separates it from inflammatory and post-traumatic disease. It classically affects the dominant arm of middle-aged men, particularly manual labourers, throwers, and weight-training athletes, reflecting a degenerative response to repetitive loading.
- Pain at the extremes of motion (terminal extension and end-range flexion) with a comparatively pain-free mid-arc — the reverse of the diffuse rest-and-motion pain of inflammatory disease
- Loss of terminal extension is usually the first and most consistent deficit
- Mechanical symptoms — catching, locking, and grinding from loose bodies and impinging osteophytes
- Ulnar nerve traction or compression symptoms are common, because posteromedial olecranon osteophytes and a contracted posterior capsule crowd the cubital tunnel — assess the nerve in every case and decompress it if flexion is limited (for example, fewer than 100 degrees) or if there are neuritic symptoms
osteophytes at the tips of the olecranon and coronoid and within their fossae, with loose bodies, but with relative preservation of the ulnohumeral joint space. This retained central space is the single most useful feature distinguishing primary OA from rheumatoid and post-traumatic arthritis, both of which show global joint-space loss.
because these patients are often younger and higher-demand than the ideal TEA candidate, treatment is joint-preserving. The Outerbridge–Kashiwagi ulnohumeral arthroplasty ("OK procedure") is the classic open operation: through a posterior approach a fenestration is created through the floor of the olecranon fossa, giving access to remove both posterior (olecranon) and anterior (coronoid) osteophytes and loose bodies through a single window, with capsular release to restore the arc. The same goals — osteophyte excision, loose-body removal, and osteocapsular release — are achievable arthroscopically in experienced hands, with the systematic-review evidence above showing an average arc gain of roughly 23 degrees and a Mayo Elbow Performance Score improvement of about 24 points. Osteophytes and motion loss may recur over time, but joint preservation defers arthroplasty in a population for whom lifelong TEA load restrictions are poorly suited.
Primary elbow OA is a disease of impinging osteophytes with a preserved central joint space — end-range pain, mechanical symptoms, and terminal extension loss in a middle-aged manual worker's dominant arm. If the ulnohumeral space is globally obliterated, reconsider rheumatoid or post-traumatic arthritis. Because posteromedial osteophytes tether the ulnar nerve, ulnar decompression is frequently combined with debridement, especially when preoperative flexion is limited.
Hemophilic Arthropathy of the Elbow
Hemophilic arthropathy is listed repeatedly as a cause of elbow arthritis on this page but deserves its own logic, because both the pathology and the peri-operative rules differ fundamentally from other causes. The elbow is one of the commonest target joints in haemophilia A and B, alongside the knee and ankle. A target joint is conventionally defined as one that sustains at least three spontaneous bleeds within a six-month period.
- Recurrent haemarthrosis deposits iron (haemosiderin) in the synovium, driving a chronic proliferative, hypervascular synovitis
- The inflamed synovium releases enzymes and cytokines that degrade cartilage, while the friable neovascular tissue bleeds again — a self-perpetuating cycle
- Characteristic changes include an enlarged, overgrown radial head, widening of the trochlear notch, radiocapitellar destruction, and (in children) epiphyseal overgrowth from chronic hyperaemia
- The foundation is factor replacement / prophylaxis to abolish recurrent bleeds and protect the target joint; this is haematologist-led
- For persistent bleeding despite prophylaxis, synovectomy reduces bleed frequency: radiosynovectomy (intra-articular radioactive isotope such as yttrium-90, rhenium-186, or phosphorus-32) is minimally invasive and first-line in many centres, with surgical (arthroscopic or open) synovectomy reserved for failure
- In end-stage radiocapitellar disease, radial head excision with synovectomy relieves pain and improves forearm rotation
- Total elbow arthroplasty is reserved for end-stage destruction but carries higher infection and revision risk in these typically younger patients
Any procedure on a haemophilic elbow — even an aspiration — demands haematologist-guided perioperative factor replacement to safe levels and inhibitor screening beforehand; operating on an uncorrected clotting deficiency risks catastrophic bleeding. Remember the target-joint definition (at least three bleeds in six months) and that radiosynovectomy is the minimally invasive way to break the bleed–synovitis cycle before cartilage is lost.
Management Algorithm

The key decision is conservative vs surgical management. Conservative management is first-line for all patients. Surgery (debridement, TEA) is reserved for failed conservative management in appropriate patients (age over 60, low demand for TEA).
First-Line Treatment
- DMARDs (methotrexate, sulfasalazine)
- Biologics (anti-TNF, anti-IL6)
- Corticosteroids (oral or intra-articular)
- Continue until pain uncontrolled or function severely limited
- Activity modification
- Physiotherapy (maintain ROM, strengthen)
- Bracing (elbow brace for support)
- Weight management
- Corticosteroid injection (temporary relief, 3-6 months)
- Hyaluronic acid (limited evidence)
- Maximum 2-3 lifetime injections
- RA: 60-70% achieve adequate control with medical management
- Post-traumatic: 40-50% improve with conservative measures
- Primary OA: 50-60% respond to conservative treatment
Conservative management should be exhausted before considering surgery.
Surgical Technique
Arthroscopic Elbow Debridement
- Loose bodies
- Osteophytes causing impingement
- Early arthritis with mechanical symptoms
- Capsular contracture
- Standard arthroscopic portals (anteromedial, anterolateral, posterolateral)
- Remove loose bodies
- Debride osteophytes (coronoid, olecranon)
- Capsular release if stiff
- Radial head excision if arthritic
- 60-70% improvement
- Temporary benefit (2-5 years)
- May delay need for TEA
Arthroscopic debridement provides temporary relief and may delay definitive surgery.
Complications
- Incidence
- 10-15% (TEA)
- Risk Factors
- Cubital tunnel disease, previous surgery, transposition
- Management
- Observation if neuropraxia, exploration if transection, may need transposition
- Incidence
- 15% at 10 years
- Risk Factors
- High demand, trauma indication, young age
- Management
- Revision TEA with longer stems, bone graft
- Incidence
- 5-10%
- Risk Factors
- Triceps-reflecting approach, inadequate repair
- Management
- Extension lag, may need revision repair or tendon transfer
- Incidence
- 2-3%
- Risk Factors
- Previous surgery, RA, immunosuppression
- Management
- Debridement, antibiotics, may need explant
- Incidence
- 2-4%
- Risk Factors
- Unlinked designs, ligament deficiency
- Management
- Revision to linked design or ligament reconstruction
- Incidence
- 5-10%
- Risk Factors
- Trauma history, extensive dissection
- Management
- Prophylaxis with indomethacin, may need excision
Ulnar nerve complications occur in 10-15% of TEA procedures. The nerve must be identified and protected at the start of surgery. Transposition is not always required and may increase complications. If symptomatic preoperatively, transposition should be considered. Postoperative neuropraxia usually resolves but may be permanent.
Postoperative Care and Rehabilitation
Total Elbow Arthroplasty Rehabilitation
- Splint at 90° flexion
- Elevation to reduce swelling
- Pain control
- Ulnar nerve monitoring
- Remove splint at 2 weeks
- Gentle passive motion (avoid forced extension)
- Active-assisted motion
- No active extension until 6 weeks (protect triceps)
- Active extension strengthening
- Progressive ROM exercises
- Light activities (under 2 kg)
- Return to ADLs
- Lifelong weight restrictions (under 5 kg single, under 2 kg repetitive)
- Avoid impact activities
- Regular follow-up for loosening
Proper rehabilitation optimizes outcomes and prevents complications.
Outcomes and Prognosis
- RA: 60-70% achieve adequate control with medical management
- Post-traumatic: 40-50% improve with conservative measures
- Duration: Variable, may delay surgery for years
- Improvement: 60-70% of patients
- Duration: 2-5 years of benefit
- May delay: Need for TEA
- RA patients: 90% 10-year survival, 90% pain relief, 100° functional arc
- Trauma patients: 75% 10-year survival, 85% pain relief
- Satisfaction: 85-90% satisfied at 10 years
- Functional: 30-130° flexion arc for ADLs
- Aseptic loosening: Most common long-term failure (15% at 10 years)
- Revision: 15% revision rate at 10 years
- Weight restrictions: Lifelong (under 5 kg single, under 2 kg repetitive)
Good outcomes are associated with: RA etiology (better than trauma), age over 60, low demand, compliant with restrictions, and optimal surgical technique. Poor outcomes are associated with: young age, high demand, trauma indication, non-compliance, and complications.
Guidelines, Registries & Global Practice
Global epidemiology:
- Symptomatic elbow arthritis affects under 1% of the population — far rarer than hip or knee disease
- Inflammatory (RA) and post-traumatic causes predominate worldwide; primary OA accounts for only a minority and is overrepresented in male manual workers and athletes
- TEA is a comparatively low-volume procedure globally (a few cases per surgeon per year), concentrated in specialist upper-limb units
- Falling RA disease burden in high-income settings (driven by early DMARD/biologic therapy) has shifted the dominant TEA indication toward acute distal humeral fracture and post-traumatic arthritis
- Focus
- Medical RA control
- Key Position
- Treat-to-target with MTX plus short-course steroids, escalate to biologic/JAK; optimise before surgical referral
- Focus
- Surgical decision-making
- Key Position
- TEA reserved for low-demand patients with end-stage disease; emphasise lifelong load restriction and infection vigilance
- Focus
- Service & referral
- Key Position
- Elbow arthroplasty concentrated in specialist units; primary TEA endorsed for unreconstructible distal humeral fractures in the elderly
- Focus
- Trauma & fracture
- Key Position
- Attempt stable fixation in younger patients; TEA where comminution precludes fixation in low-demand elderly
- Norwegian Arthroplasty Register and the Australian (AOANJRR) and New Zealand joint registries track elbow implants alongside hip/knee
- Registry data consistently show higher revision rates than hip/knee arthroplasty, with aseptic loosening and deep infection the leading reasons
- RA and inflammatory indications historically show better implant survival than post-traumatic OA in pooled registry series
- Low annual volumes limit statistical power, so single-centre cohorts (Mayo, BESS units) remain important evidence sources
- High-resource settings: early biologic RA control, ready access to TEA, fluoroscopy and arthroscopic debridement
- Limited-resource settings: later presentation with advanced destruction; interposition arthroplasty, debridement, and arthrodesis retain a larger role where implants, revision capacity, and infection management are constrained
- Hemophilic arthropathy is more prominent where factor replacement is limited
- Lifelong load restrictions after TEA may be impractical for manual labourers, shifting selection toward joint-preserving options
Controversies and Areas of Uncertainty
Linked (semiconstrained) designs tolerate ligament deficiency and bone loss but transfer load to the cement-bone interface, risking loosening. Unlinked designs preserve bone but demand competent soft tissues and risk instability. No high-level trial establishes superiority; choice is patient- and surgeon-specific.
Routine anterior transposition versus simple in situ release (or leaving the nerve undisturbed) remains debated. Evidence favours selective transposition for stiff elbows or preoperative symptoms rather than transposing every case.
No durable solution exists for end-stage arthritis in active patients under 60. Options — debridement, interposition arthroplasty, arthrodesis, or accepting TEA with strict restrictions — all carry significant trade-offs and limited evidence.
Triceps-reflecting, triceps-splitting, and triceps-sparing (paratricipital) approaches each have advocates. Triceps insufficiency remains a recognised complication and the optimal exposure to minimise it is unsettled.
MCQ Practice Points
Q: What is the most common cause of inflammatory elbow arthritis? A: Rheumatoid arthritis. Unlike hip and knee where primary OA predominates, elbow arthritis is most commonly inflammatory (RA) or post-traumatic. Primary OA of the elbow is rare.
Q: What is the first-line treatment for rheumatoid elbow arthritis? A: Medical management with DMARDs and biologics. Surgery (TEA) is reserved for failed medical management in appropriate patients (age over 60, low demand).
Q: What is the 10-year survival rate of total elbow arthroplasty in rheumatoid arthritis patients? A: 90% 10-year survival in RA patients. This is better than trauma patients (75% 10-year survival). RA is the primary indication with best outcomes.
Q: What is the most common complication after total elbow arthroplasty? A: Ulnar nerve complications occur in 10-15% of procedures. Most are neuropraxias that recover, but some may be permanent. The nerve must be identified and protected at the start of surgery.
Q: What are the weight restrictions after total elbow arthroplasty? A: Lifelong restrictions: under 5 kg for single lift, under 2 kg for repetitive activities. These restrictions are essential to prevent aseptic loosening, which is the most common long-term failure (15% at 10 years).
Exam Viva Scenarios
Practise clinical reasoning and management decisions out loud
“A 65-year-old patient presents with bilateral elbow pain and stiffness. Examination shows synovial thickening, limited ROM, and ulnar nerve symptoms. How do you differentiate between rheumatoid arthritis and post-traumatic arthritis, and what is your management approach?”
“A 72-year-old patient with rheumatoid arthritis presents with severe elbow pain and stiffness despite optimal medical management. Walk me through your decision-making for total elbow arthroplasty, including patient selection, surgical approach, and key technical points.”
“A patient presents 3 months after total elbow arthroplasty with numbness and weakness in the ulnar distribution. How do you assess and manage this?”
Key Etiology
- RA: most common inflammatory, bilateral, systemic
- Post-traumatic: after fractures/dislocations, unilateral
- Primary OA: rare, manual laborers
- Hemophilic: bleeding arthropathy
Management Algorithm
- Conservative first: medical management (RA), activity modification, injections
- Surgical: debridement (temporary), TEA (definitive)
- TEA indications: failed conservative, age over 60, low demand
- Weight restrictions: under 5 kg single, under 2 kg repetitive (lifelong)
TEA Outcomes
- RA: 90% 10-year survival, 90% pain relief
- Trauma: 75% 10-year survival, 85% pain relief
- Functional arc: 30-130° flexion for ADLs
- Aseptic loosening: 15% at 10 years (most common failure)
Ulnar Nerve
- 30-50% of elbow arthritis patients have cubital tunnel syndrome
- Must assess preoperatively in all cases
- 10-15% complication rate in TEA
- Protect at start of surgery, transposition if symptomatic
Evidence Base and Key Trials
Linked Semiconstrained TEA in RA — 461 Elbows Over Three Decades
- 461 Coonrad-Morrey TEAs in 387 RA patients; median follow-up 10 years
- Survivorship free of revision/removal: 92% at 10 yr, 83% at 15 yr, 68% at 20 yr
- Median Mayo Elbow Performance Score 90 in surviving implants
- Bushing wear seen radiographically in 23%, but revision for isolated wear uncommon (2%)
- Risk factors for revision: male sex, concomitant trauma, PMMA-surfaced ulnar component
EULAR Recommendations for RA Management (2019 Update)
- Start methotrexate plus short-term glucocorticoids as first-line; treat-to-target remission
- Add a bDMARD or JAK inhibitor if poor prognostic factors or csDMARD failure at 3-6 months
- Early, tight disease control limits structural joint destruction including the elbow
- On sustained remission DMARDs may be tapered but not stopped
Arthroscopic Debridement for Primary Elbow OA — Systematic Review
- 9 studies, 213 elbows, mean age 46 yr, mean follow-up 42 months
- Global arc of motion improved by 23 degrees (94.5 to 117.6 degrees)
- Mayo Elbow Performance Score improved by 24 points (61 to 85)
- Low complication rate 2.8% and reoperation rate 4.2%
Open vs Arthroscopic Debridement in Primary Elbow OA — Meta-analysis
- 21 studies, 586 elbows (286 arthroscopic, 300 open)
- Both techniques improved MEPS and range of motion
- Open debridement gave greater flexion gain; arthroscopic improved pain VAS
- Complication rate lower for arthroscopy (6%) than open (12%)
ORIF vs Primary TEA for Distal Humeral Fracture in the Elderly — RCT
- Multicentre RCT, 42 patients over 65 yr with comminuted intra-articular distal humeral fractures
- TEA gave superior Mayo Elbow Performance Scores at 3, 6, 12 and 24 months
- 25% of fractures randomised to ORIF were intraoperatively converted to TEA (unfixable)
- Reoperation rates not significantly different (TEA 12% vs ORIF 27%)
Long-Term Implant Survival of TEA for Fracture (RCT Follow-Up)
- Long-term follow-up of the McKee RCT cohort; mean 12.5 yr for survivors
- Of 25 fracture TEAs, only 1 required revision; 15 died with a well-functioning implant in situ
- No patient required a late revision arthroplasty
- For most elderly fracture patients, a well-performed TEA is the last elbow procedure needed
Ulnar Nerve In Situ Release During TEA
- 83 primary TEAs; routine in situ release, transposition only when nerve tracked abnormally (5%)
- 3% incidence of significant ulnar nerve complications, comparable to systematic reviews
- Preoperative flexion under 100 degrees was associated with postoperative nerve symptoms
- Routine transposition increases nerve handling and operative time without clear benefit
Kudo Type-5 Cementless-Humeral TEA in RA — Minimum 10-Year Follow-Up
- 41 unlinked Kudo type-5 TEAs in RA; mean follow-up 141 months
- Kaplan-Meier survival 87.8% at 5 yr but fell to 70.7% at 10 yr
- Aseptic loosening of the cemented ulnar component was the dominant failure mode
- RA duration under 15 yr and preoperative arc over 85 degrees were revision risk factors
