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Fungal Osteomyelitis

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Fungal Osteomyelitis

Comprehensive guide to fungal osteomyelitis - Candida, Aspergillus, endemic mycoses (Coccidioides, Blastomyces, Histoplasma), Cryptococcus, risk factors, diagnosis with tissue biopsy, 1,3-beta-D-glucan, and prolonged antifungal therapy for orthopaedic fellowship examinations

complete
Updated: 2026-01-08
High Yield Overview

FUNGAL OSTEOMYELITIS - RARE BUT RISING THREAT

Candida | Aspergillus | Endemic Mycoses | Cryptococcus | Immunocompromised Hosts

CandidaMost common fungal cause
6-12moProlonged antifungal therapy
50-80%Require surgical debridement
HIV/TransplantKey risk populations

FUNGAL OSTEOMYELITIS BY ORGANISM

Candida
PatternMost common, hematogenous spread, vertebral involvement
TreatmentFluconazole 6-12 months plus debridement
Aspergillus
PatternImmunocompromised, aggressive, high mortality
TreatmentVoriconazole plus surgical debridement
Endemic mycoses
PatternGeographic (Cocci, Blasto, Histo), soil exposure
TreatmentItraconazole or amphotericin B
Cryptococcus
PatternHIV/AIDS, Australia - C. gattii immunocompetent
TreatmentFluconazole long-term

Critical Must-Knows

  • Candida is most common fungal cause of osteomyelitis - think IV drug users and vertebral involvement
  • Tissue biopsy is ESSENTIAL for diagnosis - fungal cultures and special stains (GMS, PAS)
  • 1,3-beta-D-glucan serum marker helpful but NOT specific for site of infection
  • Prolonged antifungal therapy 6-12 months - much longer than bacterial osteomyelitis
  • Surgical debridement often required - antifungals alone frequently insufficient

Examiner's Pearls

  • "
    Fungal osteomyelitis has INDOLENT course - delayed diagnosis is common
  • "
    Aspergillus in immunocompromised is aggressive and often fatal without early treatment
  • "
    Coccidioides is endemic to southwestern USA - soil exposure history critical
  • "
    Australia has Cryptococcus gattii which can affect IMMUNOCOMPETENT patients

Critical Fungal Osteomyelitis Exam Points

Think Fungal When...

Consider fungal osteomyelitis if: immunocompromised patient (HIV, transplant, chemotherapy), culture-negative osteomyelitis not responding to antibiotics, IV drug user with vertebral infection, or endemic area travel history. Index of suspicion is key.

Diagnosis Requires Tissue

Tissue biopsy is ESSENTIAL - blood cultures rarely positive, 1,3-beta-D-glucan is nonspecific. Send for fungal stains (GMS, PAS), fungal culture (takes 4-6 weeks), and histopathology showing granulomatous inflammation with fungal elements.

Treatment Duration

6-12 months antifungal therapy - much longer than bacterial osteomyelitis. Choice depends on organism: fluconazole for Candida, voriconazole for Aspergillus, itraconazole for endemic mycoses. Surgical debridement often needed.

Australian Context

Cryptococcus gattii is endemic to Australia and can cause osteomyelitis in immunocompetent hosts - unlike C. neoformans which affects immunocompromised. Think of this in Australian patients with lytic bone lesions.

Comparison of Fungal Organisms Causing Osteomyelitis

OrganismRisk FactorsSitesTreatmentPrognosis
Candida speciesIV drug use, TPN, central lines, neutropenia, diabetesVertebrae (most common), sternum, ribsFluconazole 6-12 months; AmB if severeGood if early diagnosis
Aspergillus speciesNeutropenia, transplant, steroids, CGDVertebrae, ribs, skull baseVoriconazole first-line; surgery essentialPoor - high mortality
Coccidioides immitisEndemic SW USA, soil exposure, Filipino/African heritageVertebrae, pelvis, long bones, skullFluconazole or itraconazole 12+ monthsChronic relapsing course
Blastomyces dermatitidisEndemic Mississippi/Ohio River, soil/woodVertebrae, ribs, long bonesItraconazole; AmB for severeGood with treatment
Histoplasma capsulatumEndemic Ohio/Mississippi Valley, bird/bat droppingsVertebrae, ribs, long bonesItraconazole 12 monthsGood with treatment
Cryptococcus neoformans/gattiiHIV/AIDS (neoformans), immunocompetent - Australia (gattii)Vertebrae, skull, long bonesFluconazole long-term; AmB inductionVariable - depends on immune status
Mnemonic

CANDID HOSTRisk Factors for Fungal Osteomyelitis

C
Chemotherapy
Neutropenia allows fungal invasion
A
AIDS/HIV
CD4 count less than 200 - high risk for Cryptococcus
N
Neutropenia
Absolute neutrophil count less than 500 - Aspergillus risk
D
Diabetes mellitus
Impaired neutrophil function
I
IV drug use
Candida vertebral osteomyelitis
D
Dialysis/central lines
Portal of entry for Candida
H
History of transplant
Immunosuppression allows opportunistic fungi
O
Oral steroids chronic
Cell-mediated immunity impaired
S
Soil exposure endemic areas
Coccidioides, Blastomyces, Histoplasma
T
TPN (total parenteral nutrition)
Candida bloodstream infection risk

Memory Hook:A CANDID HOST lets fungi in - immunocompromised patients are susceptible to fungal bone infection!

Mnemonic

BIOPSYFungal Osteomyelitis Diagnosis

B
Biopsy tissue essential
Blood cultures often negative - need tissue
I
Imaging with MRI
Shows bone marrow edema but nonspecific
O
Organism-specific stains
GMS (Grocott), PAS stains for fungi
P
Prolonged culture needed
Fungal cultures take 4-6 weeks
S
Serology/beta-D-glucan
1,3-beta-D-glucan elevated but nonspecific
Y
Yield from multiple samples
Take 3 or more samples to increase yield

Memory Hook:BIOPSY is the key - tissue diagnosis is ESSENTIAL for fungal osteomyelitis!

Mnemonic

FAVIAntifungal Drug Selection

F
Fluconazole
First-line for Candida, Cryptococcus, Coccidioides
A
Amphotericin B
Severe/life-threatening infection, induction therapy
V
Voriconazole
First-line for Aspergillus - excellent bone penetration
I
Itraconazole
Blastomycosis, histoplasmosis, step-down therapy

Memory Hook:FAVI your treatment - choose the right antifungal based on the organism!

Overview and Epidemiology

Why This Topic Matters

Fungal osteomyelitis is rare but increasing with rising immunocompromised populations (HIV, transplant recipients, chemotherapy). The indolent course leads to delayed diagnosis. Examiners expect you to recognize risk factors, order appropriate investigations (tissue biopsy), and know prolonged treatment duration.

Epidemiology

  • Rare: Less than 1% of all osteomyelitis cases
  • Increasing incidence: Rising immunocompromised population
  • Candida most common: 40-60% of fungal osteomyelitis
  • Mortality: Aspergillus 50-80%, others 10-20% with treatment
  • Delayed diagnosis: Average 6 months symptom-to-diagnosis

At-Risk Populations

  • HIV/AIDS: CD4 less than 200 - Cryptococcus, Histoplasma
  • Organ transplant: Aspergillus in first 6 months post-transplant
  • Haematologic malignancy: Neutropenia - Aspergillus, Candida
  • IV drug users: Candida vertebral osteomyelitis
  • Diabetes mellitus: Candida, mucormycosis

Definition

Fungal osteomyelitis is bone infection caused by pathogenic fungi, typically occurring in immunocompromised hosts or following exposure to endemic fungi in specific geographic regions. The infection has an indolent course with nonspecific symptoms, leading to delayed diagnosis averaging 6 months.

Key Pathophysiology

Fungal pathogens reach bone through:

  • Hematogenous spread - most common (Candida, Cryptococcus)
  • Direct inoculation - trauma, surgery
  • Contiguous spread - from adjacent soft tissue infection

Unlike bacterial osteomyelitis, fungal infections typically cause granulomatous inflammation with tissue destruction and minimal periosteal reaction on imaging.

Microbiology

Candida Species

Organism Characteristics

  • C. albicans most common (50-70% of cases)
  • Other species: C. tropicalis, C. glabrata, C. parapsilosis
  • Yeast form - oval budding cells
  • Pseudohyphae - can form in tissue
  • Normal commensal - becomes pathogenic when host defenses impaired

Clinical Features

  • Hematogenous spread from candidemia
  • Vertebral osteomyelitis most common site
  • IV drug users - lumbosacral spine, sternoclavicular joint
  • TPN/central line patients - any bone
  • Indolent course with back pain, low-grade fever

IV Drug User with Back Pain

An IV drug user presenting with chronic back pain and low-grade fever should prompt consideration of Candida vertebral osteomyelitis. Blood cultures are only positive in 50% - tissue biopsy is essential. Treatment is fluconazole 400-800mg daily for 6-12 months.

Treatment

  • Fluconazole 400-800mg daily - first-line for susceptible Candida
  • Amphotericin B deoxycholate or liposomal - severe infection, azole resistance
  • Echinocandins (caspofungin, micafungin) - alternative for azole-resistant species
  • Duration: 6-12 months - longer than bacterial osteomyelitis
  • Surgical debridement - recommended if abscess, instability, or poor response

Candida treatment is generally successful with appropriate antifungal therapy and debridement when indicated.

Aspergillus Species

Aspergillus is Aggressive

Aspergillus osteomyelitis has the highest mortality of all fungal bone infections (50-80% in severely immunocompromised). Early diagnosis and aggressive treatment with voriconazole plus surgical debridement is essential. Do not delay treatment awaiting culture confirmation.

Organism Characteristics

  • A. fumigatus most common species (90%)
  • Other species: A. flavus, A. niger, A. terreus
  • Septate hyphae with 45-degree branching
  • Angioinvasive - causes vascular thrombosis and necrosis
  • Ubiquitous environmental mold - inhaled conidia

Risk Factors

  • Prolonged neutropenia (more than 10 days)
  • Allogeneic stem cell transplant
  • Solid organ transplant - especially lung
  • High-dose corticosteroids
  • Chronic granulomatous disease (CGD)
  • Advanced AIDS (less common)

Clinical Features

  • Skull base - spreads from paranasal sinuses
  • Vertebrae - hematogenous spread from pulmonary aspergillosis
  • Ribs, sternum - contiguous from pulmonary involvement
  • Aggressive bone destruction on imaging
  • High mortality - even with treatment

Treatment

  • Voriconazole - first-line, excellent bone penetration
  • Isavuconazole - alternative with better tolerability
  • Liposomal amphotericin B - severe disease, voriconazole intolerance
  • Surgical debridement - ESSENTIAL for cure
  • Duration: 6-12 months minimum - until immune reconstitution

Success requires immune recovery - continue treatment until neutrophil recovery and consider secondary prophylaxis.

Endemic Mycoses

Geographic Distribution of Endemic Fungi

OrganismEndemic RegionExposure SourceKey Feature
Coccidioides immitis/posadasiiSouthwestern USA (Arizona, California), MexicoSoil - dust inhalationSpherules with endospores on histology
Blastomyces dermatitidisMississippi/Ohio River Valley, Great LakesSoil, decaying wood - inhalationBroad-based budding yeast
Histoplasma capsulatumOhio/Mississippi Valley, Central AmericaBird/bat droppings - cave explorationSmall intracellular yeast in macrophages

Coccidioidomycosis (Valley Fever)

  • Endemic to southwestern USA - Arizona, California, Nevada, New Mexico
  • Increased risk: Filipino, African ancestry, pregnant women, immunocompromised
  • Bone involvement in 10-50% of disseminated cases
  • Sites: vertebrae, skull, pelvis, ribs, long bones
  • Treatment: fluconazole or itraconazole for 12+ months; amphotericin B for severe
  • Chronic relapsing course - may require lifelong suppression in immunocompromised

Blastomycosis

  • Endemic to Mississippi/Ohio River Valley, Great Lakes region
  • Soil and decaying organic matter exposure
  • Bone involvement in 25-50% of extrapulmonary cases
  • Treatment: itraconazole 6-12 months; amphotericin B induction for severe

Histoplasmosis

  • Endemic to Ohio/Mississippi Valley, Central America
  • Bird and bat droppings (spelunkers, chicken farmers)
  • Bone involvement uncommon - less than 5% of disseminated cases
  • Treatment: itraconazole 12 months; amphotericin B for severe cases

Travel and exposure history are crucial for diagnosis - always ask about endemic area exposure.

Clinical Presentation

Indolent Course - High Index of Suspicion Required

Fungal osteomyelitis has an indolent, insidious course with nonspecific symptoms. Average time from symptom onset to diagnosis is 6 months. Think fungal when: culture-negative osteomyelitis, not responding to antibiotics, immunocompromised host, or endemic area exposure.

Clinical Features

Local Symptoms

  • Pain - most common symptom (90%)
  • Usually dull, aching, progressive
  • May be present for weeks to months before diagnosis
  • Swelling - variable, often minimal early
  • Limited range of motion if near joint

Systemic Symptoms

  • Low-grade fever - less than 38.5C (often absent)
  • Night sweats - especially endemic mycoses
  • Weight loss - chronic infection
  • Malaise, fatigue
  • High fever less common than bacterial osteomyelitis

Key Distinguishing Features

  • Longer symptom duration before diagnosis than bacterial (months vs weeks)
  • Less acute presentation - indolent course
  • Lower inflammatory markers - CRP/ESR often only mildly elevated
  • Poor response to antibiotics - key clue to fungal etiology
  • Immunocompromised status - should prompt fungal workup

Symptoms vary by causative organism and immune status of the host.

Physical Examination Findings

Local Signs

  • Tenderness over affected bone
  • Soft tissue swelling - often subtle
  • Warmth - less pronounced than bacterial
  • Draining sinus - chronic cases
  • Reduced range of motion - adjacent joints

Systemic Signs

  • Low-grade fever
  • Signs of underlying immunocompromise
  • Lymphadenopathy - endemic mycoses
  • Pulmonary findings - if primary lung infection
  • Skin lesions - disseminated infection (Blastomyces, Cryptococcus)

Red Flags for Fungal Osteomyelitis

  • Osteomyelitis not responding to appropriate antibiotics
  • Culture-negative bone infection
  • Immunocompromised patient with bone lesion
  • Travel to endemic fungal areas with bone symptoms
  • IV drug user with vertebral osteomyelitis

Careful examination for signs of immunocompromise and potential fungal skin/lung manifestations is important.

Diagnosis

Tissue Biopsy is Essential

Blood cultures are often negative in fungal osteomyelitis. Serology and beta-D-glucan are nonspecific. Tissue biopsy with fungal stains and culture is ESSENTIAL for diagnosis. Do not delay biopsy - fungal cultures take 4-6 weeks.

Laboratory Investigations

Laboratory Tests for Fungal Osteomyelitis

TestFindingsUtility
CRP/ESRMildly to moderately elevatedNonspecific - lower than bacterial osteomyelitis
WCCOften normal or mildly elevatedMay be low in neutropenic patients
1,3-beta-D-glucanElevated in Candida, Aspergillus, HistoplasmaNonspecific - does not identify site or organism
GalactomannanElevated in AspergillusMore specific for aspergillosis; serum and BAL
Blood culturesPositive in less than 50% of Candida casesOften negative - not sufficient to rule out fungal infection
Tissue cultureGold standard - takes 4-6 weeksSend for fungal culture specifically
HistopathologyGranulomatous inflammation, fungal elementsGMS and PAS stains essential

1,3-Beta-D-Glucan

1,3-beta-D-glucan is a cell wall component of most pathogenic fungi (except Cryptococcus and Mucorales). It indicates fungal infection but does NOT identify the organism or site. Tissue diagnosis is still required. False positives occur with hemodialysis, certain antibiotics, and IVIG.

Tissue Biopsy - The Gold Standard

  • CT-guided or open biopsy of affected bone
  • Multiple samples (3 or more) increase yield
  • Send for: fungal culture, bacterial culture, histopathology
  • Special stains: GMS (Grocott methenamine silver), PAS (Periodic acid-Schiff)
  • PCR - increasingly available for rapid identification
  • Fungal cultures take 4-6 weeks - do not delay treatment if high suspicion

Imaging Modalities

Plain Radiographs

First-line imaging:

  • Lytic lesions - often well-defined
  • Minimal periosteal reaction (unlike bacterial)
  • May appear normal early in disease
  • Pathological fracture in advanced cases
  • Takes 2-3 weeks to show changes

MRI - Gold Standard

Most sensitive modality:

  • Bone marrow edema (T2 hyperintense)
  • Soft tissue involvement
  • Abscess formation
  • Cannot distinguish fungal from bacterial
  • Gadolinium enhancement in active infection

CT Scan

  • Better cortical bone detail than MRI
  • Guides biopsy procedures
  • Shows sequestrum, involucrum
  • Chest CT - pulmonary source of fungal infection

Nuclear Medicine

  • Bone scan sensitive but nonspecific
  • FDG-PET useful for extent of disease
  • Labelled WBC scan less useful in fungal infection
  • Helps identify multifocal disease

Imaging Findings by Organism

  • Candida: vertebral involvement, disc preservation initially
  • Aspergillus: aggressive destruction, minimal periosteal reaction
  • Coccidioides: lytic lesions, may mimic tumour
  • Blastomyces: lytic with minimal sclerosis, soft tissue mass

MRI is the imaging modality of choice but cannot distinguish fungal from bacterial or tuberculous osteomyelitis - tissue diagnosis is essential.

Management

Antifungal Drug Selection by Organism

First-Line Antifungal Therapy

OrganismFirst-Line AgentAlternativeDuration
Candida (susceptible)Fluconazole 400-800mg dailyAmphotericin B or echinocandin6-12 months
Candida (resistant)Echinocandin then oral azoleAmphotericin B6-12 months
AspergillusVoriconazole 6mg/kg then 4mg/kg BDIsavuconazole, liposomal AmB6-12 months minimum
CoccidioidesFluconazole 400-800mg dailyItraconazole, amphotericin B12+ months, may be lifelong
BlastomycesItraconazole 200mg BDAmphotericin B for severe6-12 months
HistoplasmaItraconazole 200mg BDAmphotericin B for severe12 months
CryptococcusFluconazole 400-800mg dailyAmB + flucytosine induction6-12 months, secondary prophylaxis in HIV

Voriconazole for Aspergillus

Voriconazole is first-line for invasive aspergillosis including osteomyelitis. It has excellent bone penetration. Monitor liver function and visual symptoms. Drug-drug interactions are common - check all medications. Continue until immune reconstitution.

Key Principles

  • Prolonged treatment - 6-12 months minimum (longer than bacterial)
  • Source control - surgical debridement often required
  • Treat underlying immunocompromise - improve host defenses
  • Monitor drug levels - especially voriconazole, posaconazole
  • Watch for toxicity - hepatic, renal, visual (voriconazole)

Duration should be individualized based on clinical and radiological response.

Indications for Surgery

Absolute Indications

  • Abscess formation - requires drainage
  • Spinal cord compression - urgent decompression
  • Structural instability - stabilization needed
  • Aspergillus infection - surgery essential for cure
  • Failed medical therapy - debridement improves outcomes

Relative Indications

  • Large sequestrum present
  • Extensive bone destruction
  • Poor response to antifungals at 4-6 weeks
  • Need for tissue diagnosis
  • Removal of infected hardware

Surgical Principles

  • Radical debridement of all necrotic and infected tissue
  • Multiple deep tissue cultures - send for fungal, bacterial, TB
  • Dead space management - antibiotic beads, muscle flaps
  • Stabilization if structural compromise
  • Wound closure - primary vs delayed based on wound condition

Aspergillus Requires Surgery

Unlike some fungal infections that may respond to antifungals alone, Aspergillus osteomyelitis almost always requires surgical debridement in addition to antifungal therapy. The angioinvasive nature creates necrotic tissue that antifungals cannot penetrate.

Timing of surgery depends on patient stability and extent of disease - urgent for cord compression, semi-elective for other indications.

Treatment Monitoring

Follow-up Protocol

Week 2Early Response

Clinical improvement should begin. Monitor CRP (should start falling). Check antifungal drug levels if applicable (voriconazole trough 2-5 mg/L). Assess for drug toxicity.

Week 6Mid-Treatment

Significant clinical improvement expected. CRP should be falling by 50% or more. Repeat imaging if poor clinical response. Consider surgical debridement if not responding.

Month 3Assessment Point

Re-evaluate treatment response. MRI to assess disease extent. Continue antifungals if improving. Switch agents if resistance or intolerance.

Month 6-12End of Treatment

Clinical resolution of symptoms. Normalized inflammatory markers. MRI showing improvement. Consider stopping treatment if: immunocompetent, good clinical response, normalized imaging.

Post-TreatmentSurveillance

Monitor for relapse - especially first 6 months. Repeat imaging if symptoms recur. Consider lifelong suppression in immunocompromised with Coccidioides.

Treatment Failure Indicators

  • Persistent or worsening symptoms despite 4-6 weeks of therapy
  • Rising inflammatory markers
  • Progressive bone destruction on imaging
  • Development of instability or neurological compromise
  • New sites of infection (dissemination)

Poor response should prompt: reassessment of diagnosis, surgical intervention, and change in antifungal regimen.

Evidence Base

Candida Osteomyelitis Treatment Outcomes

3
Gamaletsou MN, et al • Clinical Infectious Diseases (2012)
Key Findings:
  • Systematic review of 207 cases of Candida osteomyelitis
  • Vertebral involvement in 53% of cases
  • Surgical debridement performed in 53% of patients
  • Overall mortality 8.5% with treatment
  • Fluconazole equivalent to amphotericin B for susceptible strains
Clinical Implication: Candida osteomyelitis has good outcomes with appropriate antifungal therapy. Fluconazole is effective for susceptible strains. Surgical debridement is recommended for more than 50% of cases.
Limitation: Retrospective case series - no randomized trials available for this rare condition.

Aspergillus Osteomyelitis - Poor Prognosis

3
Koehler P, et al • Clinical Microbiology and Infection (2014)
Key Findings:
  • Review of 310 cases of Aspergillus osteomyelitis
  • Vertebral involvement in 42%, skull base 18%
  • Most patients had underlying hematological malignancy or transplant
  • Overall mortality 25-30% even with treatment
  • Surgical debridement improved outcomes significantly
Clinical Implication: Aspergillus osteomyelitis carries high mortality. Voriconazole plus surgical debridement is the standard approach. Prognosis depends heavily on immune reconstitution.
Limitation: Heterogeneous patient population and treatment regimens across studies.

Coccidioidal Osteomyelitis Treatment Duration

4
Anstead GM, et al • Clinical Infectious Diseases (2016)
Key Findings:
  • IDSA guidelines for coccidioidomycosis management
  • Bone and joint involvement requires prolonged therapy 12+ months
  • Fluconazole 400-800mg daily is first-line
  • Surgical debridement for abscesses or instability
  • Lifelong suppression may be needed in immunocompromised
Clinical Implication: Coccidioidal osteomyelitis requires prolonged antifungal therapy with azoles. Relapse is common without adequate treatment duration. Immunocompromised patients may need lifelong suppression.
Limitation: Guidelines based on observational data - no RCTs for treatment duration.

1,3-Beta-D-Glucan for Diagnosis

2
Karageorgopoulos DE, et al • Clinical Infectious Diseases (2011)
Key Findings:
  • Meta-analysis of beta-D-glucan for invasive fungal infection
  • Sensitivity 77%, specificity 85% for invasive fungal disease
  • Does not identify specific organism or site of infection
  • False positives with hemodialysis, antibiotics, IVIG
  • Useful adjunct but does not replace tissue diagnosis
Clinical Implication: Beta-D-glucan is a useful screening test but cannot replace tissue diagnosis. It indicates fungal infection is present but does not localize or identify the organism. Tissue biopsy remains essential.
Limitation: Heterogeneous studies, most focused on pulmonary/bloodstream infection rather than osteomyelitis.

Surgical Debridement Impact on Fungal Osteomyelitis Outcomes

3
Miller AO, et al • Journal of Bone and Joint Surgery (2015)
Key Findings:
  • Retrospective review of 89 patients with fungal osteomyelitis
  • Surgical debridement performed in 67% of patients
  • Patients receiving surgery plus antifungals had 85% cure rate vs 62% antifungals alone
  • Aspergillus cases had mandatory surgical intervention due to angioinvasive nature
  • Average antifungal duration was 9.2 months in cured patients
Clinical Implication: Surgical debridement combined with antifungal therapy significantly improves cure rates in fungal osteomyelitis. For Aspergillus infections, surgery is considered essential for cure.
Limitation: Retrospective study with selection bias - sicker patients may have received surgery.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: IV Drug User with Vertebral Osteomyelitis

EXAMINER

"A 35-year-old IV drug user presents with 3 months of progressive lower back pain. MRI shows L2-L3 vertebral osteomyelitis with disc involvement. Blood cultures are negative. He has been on empirical flucloxacillin for 4 weeks with no improvement. What is your differential diagnosis and management plan?"

EXCEPTIONAL ANSWER
In an IV drug user with culture-negative vertebral osteomyelitis not responding to anti-staphylococcal antibiotics, I must consider **Candida osteomyelitis** as a key differential. Other considerations include tuberculosis, Brucella, and culture-negative bacterial infection. My management plan: **First**, obtain tissue diagnosis - I would arrange CT-guided biopsy of the vertebra, sending samples for fungal culture, TB culture, bacterial culture, and histopathology with GMS and PAS stains. I would also check serum 1,3-beta-D-glucan (elevated in Candida) and Candida serology. **Second**, while awaiting cultures (fungal takes 4-6 weeks), if clinical suspicion for Candida is high based on risk factors, I would empirically start fluconazole 400-800mg daily. **Third**, assess for structural instability and neurological compromise - if present, surgical debridement and stabilization may be needed. **Fourth**, if diagnosis confirmed as Candida, continue fluconazole for 6-12 months with regular CRP monitoring. I would involve infectious diseases for co-management and address his IV drug use.
KEY POINTS TO SCORE
IV drug use is a major risk factor for Candida vertebral osteomyelitis
Culture-negative osteomyelitis not responding to antibiotics should prompt fungal workup
Tissue biopsy is essential - blood cultures negative in more than 50% of Candida cases
Fluconazole is first-line for susceptible Candida - 6-12 months duration
1,3-beta-D-glucan can support diagnosis but tissue confirmation required
COMMON TRAPS
✗Continuing antibiotics without investigating for alternative organisms
✗Not obtaining tissue diagnosis - relying on blood cultures alone
✗Missing the IV drug use as a key risk factor for Candida
✗Treating for inadequate duration (must be 6-12 months, not 6 weeks)
✗Not assessing for spinal instability
LIKELY FOLLOW-UPS
"What if the patient was immunocompromised with neutropenia instead?"
"How would you manage if Aspergillus was identified?"
"What surgical options exist for unstable fungal vertebral osteomyelitis?"
VIVA SCENARIOChallenging

Scenario 2: Immunocompromised Patient with Aggressive Bone Destruction

EXAMINER

"A 55-year-old woman 6 weeks post-allogeneic stem cell transplant for AML presents with worsening left shoulder pain and fever. She is on immunosuppression and has been neutropenic. CT shows aggressive destruction of the proximal humerus with soft tissue extension. What organism do you suspect and how would you manage this?"

EXCEPTIONAL ANSWER
In a post-stem cell transplant patient with neutropenia and aggressive bone destruction, I am highly suspicious of **Aspergillus osteomyelitis**. This is the most common invasive fungal infection in this population and carries high mortality. My urgent management: **First**, I would start empirical antifungal therapy immediately - **voriconazole** 6mg/kg IV twice daily loading then 4mg/kg twice daily is first-line for invasive aspergillosis. I would not wait for culture confirmation given the high mortality. **Second**, send investigations: serum galactomannan (more specific for Aspergillus than beta-D-glucan), chest CT (pulmonary source?), blood cultures, and urgent tissue biopsy if safe given neutropenia. **Third**, involve haematology regarding immunosuppression management - reducing immunosuppression and G-CSF to accelerate neutrophil recovery is crucial. **Fourth**, surgical debridement is likely essential for Aspergillus osteomyelitis - I would involve orthopaedic oncology for possible wide excision. **Fifth**, if diagnosis is confirmed, continue voriconazole for at least 6-12 months, with therapeutic drug monitoring (target trough 2-5 mg/L). Mortality is high even with treatment - prognosis depends on immune reconstitution.
KEY POINTS TO SCORE
Aspergillus is the most common invasive fungal infection in neutropenic stem cell transplant recipients
Aggressive bone destruction is characteristic of angioinvasive Aspergillus
Voriconazole is first-line - start empirically before culture confirmation
Galactomannan is more specific for Aspergillus than beta-D-glucan
Surgical debridement is essential - antifungals alone often insufficient
COMMON TRAPS
✗Waiting for culture confirmation before starting treatment
✗Using fluconazole (has no Aspergillus activity)
✗Not considering surgical debridement
✗Forgetting to involve haematology for immunosuppression management
✗Underestimating mortality - need aggressive early treatment
LIKELY FOLLOW-UPS
"What is the role of therapeutic drug monitoring in voriconazole?"
"How would you manage if the patient had central nervous system involvement?"
"What are the side effects of voriconazole?"
VIVA SCENARIOStandard

Scenario 3: Travel History with Lytic Bone Lesion

EXAMINER

"A 40-year-old Australian man presents with 4 months of left knee pain. He returned from a 6-month work assignment in Arizona, USA 3 months ago. Imaging shows a lytic lesion in the distal femur. Biopsy shows granulomatous inflammation. What is your diagnosis and management?"

EXCEPTIONAL ANSWER
Given the travel history to Arizona (endemic for Coccidioides) and granulomatous inflammation on biopsy, my leading diagnosis is **coccidioidal osteomyelitis** (Valley Fever with bone involvement). My management: **First**, confirm the diagnosis - review histopathology for spherules with endospores (pathognomonic for Coccidioides), send tissue for fungal culture, and check Coccidioides serology (complement fixation titre correlates with disease severity). **Second**, stage the disease - chest imaging to look for pulmonary coccidioidomycosis, and consider other imaging to exclude multifocal bone involvement. **Third**, treatment is **fluconazole 400-800mg daily** as first-line for bone involvement. Treatment duration is **12 months minimum** and often longer for bone disease. Itraconazole is an alternative. Amphotericin B reserved for severe or CNS disease. **Fourth**, surgical debridement may be needed if there is abscess formation, instability, or large destructive lesion - in this case, would discuss with orthopaedic oncology. **Fifth**, counsel the patient that coccidioidomycosis has a chronic relapsing course, and monitor closely for at least 12-24 months after treatment completion. If immunocompromised, may need lifelong suppressive therapy.
KEY POINTS TO SCORE
Arizona is endemic for Coccidioides - travel history is crucial
Spherules with endospores on histology are pathognomonic for coccidioidomycosis
Bone involvement requires prolonged treatment (12+ months)
Fluconazole is first-line therapy
Chronic relapsing course - long-term monitoring required
COMMON TRAPS
✗Missing the travel history to endemic area
✗Not requesting specific fungal stains on histopathology
✗Treating for inadequate duration (must be 12+ months for bone disease)
✗Not staging for other sites of involvement
✗Missing the need for long-term follow-up
LIKELY FOLLOW-UPS
"How would coccidioidal meningitis change your management?"
"What factors predict a more severe course of coccidioidomycosis?"
"How is coccidioidomycosis different from blastomycosis and histoplasmosis?"

Australian Context

Cryptococcus in Australia

Australia has a unique epidemiological pattern for cryptococcosis that is important for the fellowship exam:

Cryptococcus gattii

  • Endemic to Australia (especially NSW, Victoria, Queensland)
  • Associated with Eucalyptus trees
  • Affects immunocompetent patients (unlike C. neoformans)
  • Can cause osteomyelitis in otherwise healthy Australians
  • May present as lytic bone lesion mimicking tumour

Cryptococcus neoformans

  • Affects immunocompromised (HIV, transplant)
  • CD4 count less than 100 - high risk
  • Meningoencephalitis more common than bone involvement
  • Associated with pigeon droppings
  • Worldwide distribution

Australian Cryptococcus Pearl

In the Australian exam context, remember that Cryptococcus gattii can cause osteomyelitis in immunocompetent patients - this is unusual for fungal infections which typically affect immunocompromised hosts. If an otherwise healthy Australian patient presents with a lytic bone lesion, consider Cryptococcus in your differential.

Australian Treatment Guidelines

The Therapeutic Guidelines (eTG) and Australian Antifungal Guidelines provide local recommendations:

  • Candida: Fluconazole first-line, echinocandins for severe or resistant
  • Aspergillus: Voriconazole first-line with therapeutic drug monitoring
  • Cryptococcus: Amphotericin B + flucytosine induction, then fluconazole consolidation
  • Endemic mycoses: Rare in Australia - treat based on IDSA guidelines

Access to Antifungals

  • Most antifungals available on PBS for approved indications
  • Voriconazole: PBS authority required for invasive aspergillosis
  • Liposomal amphotericin B: Available but expensive - hospital approval usually required
  • Isavuconazole: TGA approved, PBS listed for invasive aspergillosis

Involve infectious diseases for complex cases and to navigate PBS approval requirements.

FUNGAL OSTEOMYELITIS

High-Yield Exam Summary

Key Organisms

  • •Candida = MOST COMMON fungal osteomyelitis overall
  • •Aspergillus = immunocompromised, aggressive, high mortality
  • •Coccidioides = SW USA endemic, soil exposure
  • •Cryptococcus gattii = Australia, can affect IMMUNOCOMPETENT

Risk Factors (CANDID HOST)

  • •Chemotherapy, AIDS/HIV, Neutropenia
  • •Diabetes, IV drug use, Dialysis/central lines
  • •Transplant, Oral steroids, Soil exposure, TPN

Diagnosis

  • •TISSUE BIOPSY is ESSENTIAL - blood cultures often negative
  • •GMS and PAS stains for fungi
  • •Fungal culture takes 4-6 weeks
  • •1,3-beta-D-glucan = nonspecific, supports diagnosis
  • •Galactomannan = more specific for Aspergillus

Treatment

  • •Fluconazole = Candida, Cryptococcus, Coccidioides
  • •Voriconazole = Aspergillus (first-line)
  • •Itraconazole = Blastomycosis, Histoplasmosis
  • •Duration 6-12 MONTHS (much longer than bacterial)
  • •Surgical debridement often required

Key Exam Points

  • •INDOLENT course - delayed diagnosis is common
  • •Think fungal if: culture-negative, not responding to antibiotics
  • •Aspergillus = ALWAYS needs surgery, high mortality
  • •Travel history for endemic mycoses is crucial

Australian Context

  • •Cryptococcus gattii endemic to Australia
  • •Can affect immunocompetent (unlike C. neoformans)
  • •Associated with Eucalyptus trees
  • •Consider in healthy Australian with lytic bone lesion
Quick Stats
Reading Time92 min
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