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Mallet Finger

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Mallet Finger

Comprehensive guide to the diagnosis and management of Mallet Finger (Tendinous and Bony), including splinting protocols and surgical indications.

complete
Updated: 2025-12-20
High Yield Overview

MALLET FINGER

Zone I Extensor Tendon Injury

6-8wSplinting Time
30%Articular Surface
DIPJExtension Lag
SwanNeck Deformity

Doyle Classification

Type I
PatternClosed tendinous injury (Classic).
Treatment
Type II
PatternOpen injury (Laceration).
Treatment
Type III
PatternOpen with skin/tendon loss.
Treatment
Type IV
PatternMallet Fracture (A: Peds, B: Adult).
Treatment

Critical Must-Knows

  • Conservative management (Splinting) is effective for MOST cases, including Bony Mallet.
  • Surgery is indicated for DIPJ subluxation (volar) or large fragments (greater than 30-50%).
  • Splinting must be CONTINUOUS. If the finger flexes once, the clock resets.
  • Swan Neck Deformity develops due to proximal retraction of the central slip (Fowler's mechanism).
  • Ishiguro Extension Block Pinning is the standard minimal invasive technique for bony mallet.

Examiner's Pearls

  • "
    A lag of less than 10 degrees is functionally acceptable.
  • "
    Dorsal skin necrosis is the most common complication of splinting (check the fit!).
  • "
    Antibiotics are required for open injuries (Type II/III).

Clinical Imaging

Imaging Gallery

Main theories explaining the mechanism of injuryIn all analyses, an axial force is applied to the tip of a straight digit (black arrows), followed by extreme passive distal interphalangeal joint (DIPJ
Click to expand
Main theories explaining the mechanism of injuryIn all analyses, an axial force is applied to the tip of a straight digit (black arrows), followed by Credit: Salazar Botero S et al. via Arch Plast Surg via Open-i (NIH) (Open Access (CC BY))
Size and displacement calculation on lateral viewRatio of the fractured articular surface over the total articular surface of the distal phalanx base: T=B/A+B=size of fragment in %. The ratio of the g
Click to expand
Size and displacement calculation on lateral viewRatio of the fractured articular surface over the total articular surface of the distal phalanx base:Credit: Salazar Botero S et al. via Arch Plast Surg via Open-i (NIH) (Open Access (CC BY))
Modified Tubiana classificationType I, subcutaneous tendon rupture; type II, bony avulsion at the base of the distal phalanx; type III, fracture >1/3 of the articular surface with volar subluxation
Click to expand
Modified Tubiana classificationType I, subcutaneous tendon rupture; type II, bony avulsion at the base of the distal phalanx; type III, fracture >1Credit: Salazar Botero S et al. via Arch Plast Surg via Open-i (NIH) (Open Access (CC BY))
Post-operative antero-posterior and lateral radiographs showing in-situ extension-block and trans-articular K-wires. DIP joint is in slight extension. Extension-block K-wire is radial-ward and trans-a
Click to expand
Post-operative antero-posterior and lateral radiographs showing in-situ extension-block and trans-articular K-wires. DIP joint is in slight extension.Credit: Agarwal S et al. via J Orthop Case Rep via Open-i (NIH) (Open Access (CC BY))

The Splinting Pitfall

The Problem

Skin Necrosis The dorsal skin over the DIPJ is extremely thin. Tight splints (especially dorsal splints or taped Stacks) can cause pressure necrosis. This turns a closed simple injury into a complex soft tissue defect.

The Solution

Hygiene & Fit Instruct the patient to clean the finger daily while supported in extension. Check the splint for blanching points. Use a slightly looser splint with perforation for aeration.

ConditionPathologyTreatmentPrognosis
Tendinous MalletTendon RuptureSplint 6-8wGood
Bony MalletAvulsion #Splint 6w (Bony union fast)Good (Remodels)
Subluxed BonyVolar SubluxationSurgery (Ishiguro)Variable (OA risk)
Chronic MalletScarred/LongRe-splint or TenotomyFair
Mnemonic

LOSSSurgical Indications

L
Large
Fragment greater than 30-50% articular surface.
O
Open
Open injury requiring washout.
S
Subluxation
Volar subluxation of the distal phalanx.
S
Salter-Harris
Seymour Fracture in children (nail bed incarceration).

Memory Hook:Surgery prevents LOSS of the joint.

Mnemonic

COATDoyle Classification

C
Closed
Type I (Tendinous).
O
Open
Type II (Laceration).
A
Abrasion/Avulsion
Type III (Skin loss).
T
Transosseous
Type IV (Fracture).

Memory Hook:Wear a COAT for the cold finger.

Mnemonic

VOLTSwan Neck Component

V
Volar Plate
Laxity at the PIPJ.
O
Overpull
Extensor overpull (central slip).
L
Lateral Bands
Dorsal subluxation of lateral bands.
T
Terminal Tendon
Rupture (Mallet) initiates the imbalance.

Memory Hook:It takes VOLTage to snap into Swan Neck.

Overview

Definition

Mallet Finger is the disruption of the terminal extensor tendon at its insertion into the base of the distal phalanx (Zone I). It results in the inability to actively extend the DIPJ (extensor lag), while passive extension remains intact.

It is commonly caused by a "jamming" injury (e.g., basketball hitting the tip of the extended finger). The force force-flexes the DIPJ against extensor resistance, snapping the tendon or avulsing bone.

Pathophysiology and Mechanisms

Mechanism of mallet finger injury
Click to expand
Mechanism of injury: an axial force applied to the tip of a straight digit causes extreme passive DIPJ flexion, rupturing the terminal extensor tendon or avulsing its bony insertion.Credit: Salazar Botero S et al. via Arch Plast Surg (CC BY)

Extensor Apparatus

  • Terminal Tendon: Formed by the confluence of the two lateral bands. Inserts into the dorsal lip of the distal phalanx.
  • Central Slip: Inserts into the middle phalanx.
  • Triangular Ligament: Prevents lateral bands from subluxing volarly.
  • Oblique Retinacular Ligament (ORL): Links PIPJ extension to DIPJ extension.

The terminal tendon is wide and wafer thin (less than 1mm).

Swan Neck Mechanism

  • Loss of distal anchor (Mallet) causes the extensor force to retract proximally.
  • This increases tension on the Central Slip (hyperextending the PIPJ).
  • If the Volar Plate at the PIPJ is lax, the PIPJ hyperextends.
  • The Lateral Bands subluxate dorsally, locking the PIPJ in extension.
  • Result: PIPJ Hyperextension + DIPJ Flexion.

This must be treated by correcting the DIPJ lag.

Blood Supply

  • The dorsal skin of the DIPJ is supplied by tiny vascular branches.
  • It is very susceptible to pressure necrosis from splints.
  • Germinal Matrix: Lies immediately deep to the extensor tendon insertion. Nails are often damaged in Type IV injuries.

Care must be taken during K-wire insertion to avoid the germinal matrix.

Classification Systems

Doyle Classification

  • Type I: Closed tendinous injury. (Most common).
  • Type II: Open injury (Laceration).
  • Type III: Open injury with loss of skin/tendon substance.
  • Type IV: Mallet Fracture.
    • IVa: Pediatric (Salter-Harris Physeal injury).
    • IVb: Adult, less than 30% articular surface.
    • IVc: Adult, greater than 30% articular surface / Subluxed.

Type IVc is the most debated category regarding surgical management.

Wehbe & Schneider

  • Based on size of fragment and degree of subluxation.
  • Rarely used clinically; the decision is binary (Stable vs Unstable).

Subluxation is the key determinant of instability.

History

History Factors

  • Mechanism: Jamming vs Laceration.
  • Timing: Acute (less than 4 weeks) vs Chronic (greater than 4 weeks).
  • Occupation: Surgeon/Musician (needs perfect glide) vs Labourer (needs stable union).

Late presentation (greater than 4 weeks) can still be treated with splinting.

Examination

Physical Exam

  • Look: Drooping DIPJ. Erythema/Swelling (acute).
  • Move:
    • Active: Loss of active extension (Lag).
    • Passive: Full passive extension (rules out locked joint/fracture block).
  • Swan Neck: Check for PIPJ hyperextension.
  • Open: Check for laceration (don't miss a Type II).

Always document the rotational alignment of the finger.

Investigations

Radiology

  • Views: PA and True Lateral.
  • Findings:
    • Avulsion: Triangular fragment from dorsal base of P3.
    • Subluxation: Volar subluxation of P3 relative to P2 head. (Surgical Indication).
    • Apposition: Gap between fragment and bone.

A "True Lateral" is essential to assess joint congruency.

Mallet finger radiograph and splint
Click to expand
(a) Lateral radiograph of a mallet fracture (b) Dorsal splint used for mallet finger. Notice slight extension pre-bent into splint to assist in reduction of the avulsed fragment.Credit: Oetgen ME et al. via Curr Rev Musculoskelet Med (CC BY)
Mallet finger fragment size calculation
Click to expand
Size and displacement calculation on lateral view: ratio of fractured articular surface over total articular surface (T=B/A+B) determines fragment size percentage - critical for surgical decision-making.Credit: Salazar Botero S et al. via Arch Plast Surg (CC BY)

Management Algorithm

📊 Management Algorithm
mallet finger management algorithm
Click to expand
Management algorithm for mallet fingerCredit: OrthoVellum

Conservative (Splinting)

  • Indication: Type I, II, III, IVb (Stable).
  • Protocol:
    • Initial: 6-8 weeks CONTINUOUS extension. (Reset clock if flexed).
    • Weaning: 2-4 weeks night splinting + sports/heavy use.
    • Type: Stack Splint (off the shelf) or Custom Thermoplastic.
  • Outcome: 80-90% success. 5-10 degree lag is common but functional.

Patient education is the single most important factor in success.

Surgical Management

  • Indication: Volar Subluxation, Large Fragment (greater than 30-50%), Open Injury, Failed Splinting (intolerant).
  • Techniques:
    • Extension Block Pinning (Ishiguro): K-wire into P2 head blocks the fragment. P3 is extended to reduce it.
    • Open Reduction (ORIF): Screw/Hook plate. (High complication rate - comminution).
    • Tenodermodesis: For chronic tendon mallet (excise scar + plicate skin/tendon).

Surgical risks often outweigh the benefits for small fragments.

Surgical Considerations

Ishiguro Extension Block Pinning

  • Concept: Uses the P2 head as a backstop.
  • Step 1: Flex DIPJ. Insert 1.0mm K-wire into distal P2 head, aimed dorsally.
  • Step 2: Extend DIPJ. The fragment hits the wire and is reduced onto the P3 base.
  • Step 3: Insert a second longitudinal K-wire across DIPJ to lock flexion.
  • Pros: Percutaneous. No dissection.
  • Cons: Pin track infection. Wire migration.

The blocking wire must be placed dorsal to the fracture fragment.

Extension block pinning post-operative radiographs
Click to expand
Post-operative AP and lateral radiographs showing in-situ extension-block and trans-articular K-wires. DIPJ is in slight extension with the extension-block K-wire positioned radial-ward.Credit: Agarwal S et al. via J Orthop Case Rep (CC BY)

Tenodermodesis (Brooks-Graner)

  • Indication: Chronic tendinous mallet.
  • Technique: Excise elliptical wedge of skin and tendon/scar from dorsal DIPJ.
  • Repair: Close skin and tendon as one layer (Dermodesis).
  • Splint: K-wire fixation for 6 weeks.

This salvage procedure sacrifices some DIPJ flexion for extension.

Complications

  • Skin Necrosis: From tight splint. Most common.
  • Nail Deformity: Ridging from germinal matrix injury (by splint or fracture).
  • Recurrence: If splint removed too early.
  • Stiffness: Loss of flexion. Often worse than the original 10 degree lag.
  • Septic Arthritis: From pin track infection.
  • Functional Deficit: Most patients adapt well to a minor lag, but cannot adapt to a stiff, painful joint.
  • Hypersensitivity: Common at the fingertip. Desensitization therapy is needed.

Rehabilitation

Week 0-6
  • Splint: Strict extension (0 degrees). Maintain 24/7 contact with dorsal DIPJ.
  • Hygiene: Clean finger while supported in extension - use support finger or splint.
  • PIPJ: Active PIPJ ROM (flexion/extension) - prevent stiffness.
  • Monitoring: Check splint fit weekly. Look for blanching, pressure marks.
  • Education: Explain that ONE flex episode resets the clock.
Week 6-8
  • Assess: Measure active extension. If lag less than 10 degrees, proceed.
  • Active ROM: Start gentle active flexion (20-30 degrees initially).
  • Splint: Night splinting continues. Wear for heavy tasks, sports.
  • Progression: Increase flexion by 10 degrees per week.
Week 8-12
  • Full ROM: Expect full DIPJ flexion by 10-12 weeks.
  • Strengthening: Putty exercises, grip strengthening.
  • Return to Sport: Usually 10-12 weeks for non-contact, 12-16 for contact.
Week 12+
  • Full Activity: Unrestricted by 12-16 weeks.
  • Lag Management: Slight recurrence of lag (5-10 degrees) is common and acceptable.
  • Re-splint Threshold: If lag exceeds 15 degrees, restart 2-week splint protocol.
  • Long-term: Scar massage, desensitization if hypersenitive.

Key Rehabilitation Principles:

  • Continuous splinting is essential - even brief flexion can undo weeks of healing
  • Patient education is the single most important factor in outcome
  • Monitor skin - dorsal skin necrosis is avoidable with proper splint fit
  • PIPJ exercises prevent stiffness and Swan Neck progression

Prognosis

Outcomes by Treatment Type

Conservative Management (Splinting):

  • Success Rate: 80-90% achieve good/excellent results
  • Residual Lag: Average 5-10 degrees (functionally acceptable)
  • Patient Satisfaction: High, especially with compliant patients
  • Return to Work: 85% return to pre-injury occupation

Surgical Management:

  • Success Rate: 70-80% (higher complication rate offsets benefits)
  • Complications: Nail deformity (15%), infection (5-10%), stiffness (20%)
  • Indication: Reserved for subluxed joints or failed splinting

Factors Affecting Outcome:

  • Compliance: Single most important variable
  • Delay to Treatment: Late presentation still responds to splinting
  • Age: Younger patients heal faster, elderly have thinner skin (necrosis risk)
  • Comorbidities: Diabetes, smoking impair tendon healing

Compliance is the single most important predictor of outcome.

Prognostic Factors

FactorGood PrognosisPoor Prognosis
TypeType I (Tendinous)Type III (Skin loss)
Fragment SizeSmall (under 30%)Large (over 50%) + Subluxed
ComplianceExcellentPoor (repeated flexion)
DelayAcute (under 4 weeks)Chronic (over 12 weeks)
JointCongruentSubluxed (volar)

Key Prognostic Principles:

  • Bony mallet heals better than tendinous (bone-to-bone vs tendon-to-bone)
  • Persistent X-ray gap does NOT correlate with function - treat the patient, not the X-ray
  • Swan Neck corrects when mallet is treated early

Remember: Treat the patient, not the X-ray.

Long-Term Outcomes

5-Year Follow-Up Data:

  • Function: 90% full function (grip, pinch, dexterity)
  • Pain: Rare - if present, suspect OA or Swan Neck
  • Cosmesis: Minor droop (5-10 degrees) is common and accepted
  • Re-injury: Low risk once healed; may need protective splint for contact sports

Return to Activity:

  • Desk Work: 1-2 weeks (with splint)
  • Manual Work: 8-12 weeks
  • Non-Contact Sport: 10-12 weeks
  • Contact Sport: 12-16 weeks (protective splinting recommended initially)

When to Refer Back:

  • Progressive lag despite compliance
  • Swan Neck development
  • Persistent pain at 8-12 weeks
  • Significant functional limitation

Most patients return to full function with minimal residual lag.

Evidence Base

Surgery vs Splinting for Bony Mallet

1
King et al. • J Hand Surg Br (2001)
Key Findings:
  • Systematic review of Bony Mallet management
  • No difference in outcome between Splinting and Surgery even for large fragments
  • Surgery had significantly higher complication rate (infection, nail deformity)
  • Splinting is the Gold Standard unless subluxed
Clinical Implication: Don't operate on bony mallet unless the joint is falling off.

Splint Type Comparison

2
O'Brien et al. • Hand (2007)
Key Findings:
  • Comparison of Stack vs Dorsal vs Custom splints
  • No significant difference in extensor lag correction
  • Dorsal splints had higher rate of skin complications
  • Patient preference (comfort) dictates choice
Clinical Implication: The best splint is the one the patient wears.

Ishiguro Technique

4
Ishiguro et al. • J Hand Surg Br (1997)
Key Findings:
  • Description of extension block pinning
  • Excellent reduction of fracture fragment
  • Good functional outcomes
  • Avoids open dissection of the delicate germinal matrix area
Clinical Implication: If you must operate, pin it percutaneously.

Seymour Fractures

4
Seymour N. • JBJS Br (1966)
Key Findings:
  • Pediatric mallet equivalent
  • Open Salter-Harris physeal fracture with nail bed incarceration
  • High risk of osteomyelitis if missed and not washed out
  • Requires open reduction and nail bed repair
Clinical Implication: A kid with a mallet/nail bleeding needs antibiotics and washout.

Antibiotics for Open Mallet

5
Human Bites Evidence (Extrapolated) • Various (2010)
Key Findings:
  • Open mallet injuries often involve crushing
  • High risk of infection in Type II/III
  • Prophylactic antibiotics recommended for open fractures
Clinical Implication: Cover open mallets with antibiotics.

Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: The Chronic Mallet

EXAMINER

"A 30-year-old presents 4 months after a mallet injury. He ignored it. Now has a 40 degree lag and finds it catchy. Examination shows a Swan Neck deformity. Management?"

EXCEPTIONAL ANSWER
This is a Chronic Mallet with Swan Neck. The first line is STILL SPLINTING. I would try serial casting or splinting for 8 weeks. It often tightens the tendon effectively. If that fails, surgery is an option. Tenodermodesis is suitable. If the Swan Neck is passive correctable, treating the mallet usually corrects the PIPJ. If PIPJ is stiff, it needs addressing vertically.
KEY POINTS TO SCORE
Splinting works for chronic cases too
Swan Neck mechanism correction
Tenodermodesis
COMMON TRAPS
✗Jumping straight to surgery
✗Ignoring the Swan Neck
LIKELY FOLLOW-UPS
"Does splinting work at 4 months?"
"Yes, scar remodeling can occur for months. It is low risk and worth a try."
VIVA SCENARIOStandard

Scenario 2: The Bony Mallet

EXAMINER

"X-ray shows a mallet fracture with a fragment involving 40% of the articular surface. The joint is congruent (no subluxation). What do you do?"

EXCEPTIONAL ANSWER
Despite the large fragment (40%), the joint is congruent. Evidence (King et al) shows that splinting yields identical results to surgery with fewer complications. The bone remodels and the lag resolves. I would treat strictly in a splint for 6 weeks. I would obtain weekly X-rays for the first 2 weeks to ensure no subluxation develops.
KEY POINTS TO SCORE
Congruency is more important than fragment size
Remodeling potential
High complication rate of surgery
COMMON TRAPS
✗Operating just because fragment is greater than 30%
✗Using '1/3 rule' dogmatically
LIKELY FOLLOW-UPS
"When would you operate?"
"If the P3 was subluxed volarly (joint incongruent)."
VIVA SCENARIOStandard

Scenario 3: The Seymour Fracture

EXAMINER

"A 7-year-old crushes his finger. The nail plate is avulsed proximally (lying on top of the fold). The tip is flexed. X-ray shows a physeal widening."

EXCEPTIONAL ANSWER
This is a Seymour Fracture (Salter-Harris I/II of distal phalanx). It is effectively an OPEN fracture because the physis often communicates with the nail bed laceration. This is an emergency (urgency). It needs Washout, Reduction (removing the interposed nail bed/plate), Repair of the nail bed, and antibiotics. Splinting alone will lead to osteomyelitis and growth arrest.
KEY POINTS TO SCORE
Seymour fracture definition
Open fracture path (Nail bed)
Risk of Osteomyelitis
Need for Washout
COMMON TRAPS
✗Treating as a simple mallet
✗Missing the open nature
LIKELY FOLLOW-UPS
"What organism are we worried about?"
"Staph aureus and oral flora (if bitten), but also soil organisms."
VIVA SCENARIOStandard

Scenario 4: The Skin Complication

EXAMINER

"A 65-year-old diabetic presents 3 weeks into splint treatment for a mallet finger. She has a 1cm area of full thickness skin necrosis over the dorsal DIPJ. The underlying tendon is exposed. What is your management?"

EXCEPTIONAL ANSWER
This is a significant complication of splinting - full thickness skin necrosis with exposed tendon. Priorities: 1) Stop the current splint - it has failed. 2) Wound care - dressing to protect tendon. 3) May need plastic surgery input for coverage (full thickness graft or local flap if tendon desiccated). 4) Once wound healed, reassess if mallet needs further treatment. Often the scar tissue provides enough tension to reduce the lag. If surgical coverage was needed, K-wire fixation may be better than splint for future immobilization.
KEY POINTS TO SCORE
Immediate splint removal
Wound assessment and soft tissue coverage
Exposed tendon needs moist dressing (prevent desiccation)
Plastic surgery input for complex wounds
COMMON TRAPS
✗Continuing splinting despite wound
✗Delayed referral for coverage
✗Ignoring diabetes as risk factor
LIKELY FOLLOW-UPS
"What would you have done differently?"
"Weekly reviews to check skin, patient education about blanching, looser splint in elderly/diabetic."

MCQ Practice Points

Anatomy

Q: What structure balances the lateral bands and prevents volar subluxation? A: The Triangular Ligament.

Pathology

Q: What is the primary cause of Swan Neck deformity in chronic mallet finger? A: Proximal retraction of the extensor apparatus causing increased tension on the central slip.

Management

Q: What is the most common complication of surgical treatment for mallet finger? A: Nail deformity (ridging) and infection.

Management

Q: A patient wearing a stack splint complains of dorsal pain and white skin. What is the action? A: Adjust/Replace splint immediately (Risk of necrosis).

Surgical Threshold

Q: What percentage of articular surface involvement indicates surgery for bony mallet? A: Greater than 30-50% WITH volar subluxation. Size alone is NOT an indication - congruency is key.

Splint Duration

Q: How long should a mallet finger be splinted? A: 6-8 weeks continuous extension, then 2-4 weeks weaning. Clock resets if finger flexes.

Australian Context

Mallet finger is one of the most common hand injuries presenting to Australian emergency departments, comprising 1-2% of all upper limb injuries. Peak incidence occurs in young males (sports-related) and middle-aged manual workers. Common causative sports include basketball, volleyball, cricket (fielding), and AFL (hand-ball injuries). Seasonal variation shows higher incidence in summer months due to increased outdoor activities.

In the Australian healthcare system, most mallet injuries are managed conservatively by Hand Therapists in public hospital outpatient clinics. Plastic and Hand surgeons in the private sector typically manage surgical cases. Remote and regional areas effectively utilize telehealth supervision for splinting protocols, which has been shown to be safe and effective.

For open mallet injuries (Type II/III) or Seymour fractures, the eTG recommends first-generation cephalosporin prophylaxis (Cephalexin 500mg QID). Seymour fractures require additional anti-staphylococcal coverage (Flucloxacillin) given the high risk of osteomyelitis. WorkCover claims are common in manual workers, and thorough documentation of mechanism, X-ray findings, splint type, and patient education is essential for medicolegal purposes.

High-Yield Exam Summary

Classification

  • •Type I: Tendon (Splint)
  • •Type II: Laceration (Wash/Repair)
  • •Type III: Coverage loss (Flap)
  • •Type IV: Bony (Splint unless subluxed)

Management

  • •Splint: 6-8 weeks straight
  • •Surgery: If Subluxed or greater than 50%
  • •Tech: Ishiguro Pinning
  • •Chronic: Tenodermodesis

Complications

  • •Skin Necrosis (Splint)
  • •Nail Ridge (Surgery)
  • •Stiffness
  • •Recurrence
Quick Stats
Reading Time63 min
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