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Quadriceps Tendon Rupture

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Sports MedicineKnee

Quadriceps Tendon Rupture

Comprehensive guide to quadriceps tendon rupture for FRCS examination

complete
Updated: 2025-01-15

Quadriceps Tendon Rupture

High Yield Overview

QUADRICEPS TENDON RUPTURE

Extensor Mechanism Injury

—Common
—clinical relevance
—blue

Rupture Types

Complete
PatternLoss of extension
TreatmentSurgical Repair
Partial
PatternIntact extension
TreatmentImmobilization
Chronicity
PatternAcute vs Chronic
TreatmentRepair vs Reconstruction

Critical Must-Knows

  • Definition: Rupture of the quadriceps tendon at its insertion into the superior pole of the patella
  • Definition: Usually occurs in older patients (over 40) compared to patellar tendon ruptures (under 40)
  • Mechanism: Eccentric loading of a flexed knee (e.g., stumbling) or direct blow
  • Management: Complete Rupture: Surgical Repair (Transosseous sutures or Suture Anchors) followed by hinged brace

Examiner's Pearls

  • "
    X-ray (Patella Baja - low riding)
  • "
    Ultrasound/MRI (Confirm site and retraction)
  • "
    Good if repaired early
  • "
    Chronic ruptures (greater than 6 weeks) have poor outcomes due to retraction and atrophy, often requiring reconstruction (V-Y plasty)

Clinical Imaging

Imaging Gallery

AP and lateral plain radiographs of the injured extremity.
Click to expand
AP and lateral plain radiographs of the injured extremity.Credit: Cetinkaya E et al. via Int J Surg Case Rep via Open-i (NIH) (Open Access (CC BY))
T2 sequence reveals the rupture of the tendon on coronal, (a) sagittal (b) and axial (c) planes.
Click to expand
T2 sequence reveals the rupture of the tendon on coronal, (a) sagittal (b) and axial (c) planes.Credit: Cetinkaya E et al. via Int J Surg Case Rep via Open-i (NIH) (Open Access (CC BY))
Lateral knee radiograph showing patella alta.
Click to expand
Lateral knee radiograph showing patella alta.Credit: Bartalena T et al. via West J Emerg Med via Open-i (NIH) (Open Access (CC BY))
Ultrasound longitudinal scan over the infrapatellar region shows the complete patellar tendon rupture, which is swollen and hypoechoic (arrows). PT = patellar tendon. TT = tibial tuberosity.
Click to expand
Ultrasound longitudinal scan over the infrapatellar region shows the complete patellar tendon rupture, which is swollen and hypoechoic (arrows). PT = Credit: Bartalena T et al. via West J Emerg Med via Open-i (NIH) (Open Access (CC BY))

Exam Warning

Bilateral Quadriceps Rupture is a classic exam trap. It is usually misdiagnosed as strokes/neuro pathology because the patient simply "cannot walk" or stand up, but has no obvious fracture. Always palpate for a gap! Patella Baja (low riding) is seen in Quad rupture. Patella Alta (high riding) is seen in Patellar Tendon rupture.

Anatomy

Structure

Layers: The Quadriceps Tendon is trilaminar:

  1. Superficial: Rectus Femoris.
  2. Middle: Vastus Medialis (VMO) and Lateralis.
  3. Deep: Vastus Intermedius.

Blood Supply:

  • Genicular arteries (Hypovascular zone 1-2cm proximal to patella insertion - site of rupture).

At a Glance

Quadriceps tendon rupture typically occurs in patients older than 40 years (versus patellar tendon ruptures in younger patients), with rupture at the hypovascular zone 1-2cm proximal to the patella. The clinical triad is a palpable suprapatellar gap, inability to perform straight leg raise, and patella baja on lateral radiograph (compare to patella alta in patellar tendon rupture). Bilateral quadriceps rupture is pathognomonic for systemic disease (diabetes, renal failure, hyperparathyroidism, RA, fluoroquinolone use) and is often misdiagnosed as neurological pathology. Complete ruptures require surgical repair within 3 weeks for best outcomes; chronic ruptures (greater than 6 weeks) require V-Y lengthening or reconstruction due to tendon retraction and atrophy.

Mnemonic

REAL-FluRisk Factors for Tendon Rupture

R
R - Renal failure (Secondary Hyperparathyroidism)
E
E - Endocrine (Diabetes, Hypothyroid)
A
A - Autoimmune (RA, SLE)
L
L - Local steroid injection
F
Flu - Fluoroquinolones (Ciprofloxacin)

Memory Hook:REAL Flu makes tendons weak

Management

Surgical Repair

Timing:

  • Acute (less than 3 weeks): Direct repair.
  • Chronic: Reconstruction.

Technique (Direct Repair):

  1. Midline Incision.
  2. Transosseous Tunnels: Drill 3 longitudinal holes through patella. Pass #2 or #5 non-absorbable sutures (Krackow stitch) through tendon and pull through tunnels. Tie at distal pole.
  3. Suture Anchors: Alternative. Insert anchors into superior pole. Faster, but potentially less strong than bone tunnels.
  4. Reinforcement: Repair the retinaculum (medial/lateral) if torn.

Post-op:

  • Hinged Knee Brace (Locked in extension 0-2 weeks).
  • Gradual flexion (0-30° at 2 weeks, 0-60° at 4 weeks, 0-90° at 6 weeks).
  • Full weight bearing in brace (locked) immediately.

Chronic Reconstruction

If tendon retracted and cannot be brought down:

  1. Codivilla V-Y Plasty: Lengthening the proximal tendon.
  2. Scuderi Turnover Flap: Folding down a flap from the rectus femoris.
  3. Allograft: Achilles or Whole Extensor Mechanism.

Suture Anchors vs Transosseous

Maniscalco P, et al. • Int Orthop (2017)
Key Findings:
  • Biomechanical studies generally show Transosseous Tunnels have slightly higher load to failure
  • However, clinical outcomes (ROM, Rerupture rate) are equivalent
  • Anchors reduce operative time
Clinical Implication: Choice is surgeon preference. Transosseous is the 'Gold Standard' for exams.

Complications

Complications: Quad vs Patellar Tendon

References

  1. Ilanugovan R, et al. Simultaneous bilateral rupture of the quadriceps tendon in patients with chronic renal failure. J Bone Joint Surg Br. 2007.
  2. Siwek CW, Rao JP. Ruptures of the extensor mechanism of the knee joint. Clin Orthop Relat Res. 1981.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOChallenging

Scenario 1: Bilateral Quadriceps Tendon Rupture - Systemic Disease

EXAMINER

"A 55-year-old dialysis patient presents to the Emergency Department unable to walk. He says his legs 'gave way' when he tried to stand up from a chair. He denies any significant trauma - he simply felt both knees give way. He has been on haemodialysis for 8 years for end-stage renal disease due to polycystic kidney disease. His past medical history includes secondary hyperparathyroidism, diabetes mellitus, and he is on a renal transplant waiting list. On examination, he cannot perform a straight leg raise on either side. There is a palpable suprapatellar gap bilaterally. His knees have full passive range of motion but he has no active extension. Plain radiographs show low-riding patellas bilaterally (patella baja). The emergency medicine team initially suspected Guillain-Barré syndrome and requested a neurology consult, but you are called as the orthopaedic registrar. What is the diagnosis and how do you manage this patient?"

EXCEPTIONAL ANSWER
This is a bilateral quadriceps tendon rupture, which is a classic presentation in patients with chronic renal failure. The bilateral nature of the injury with minimal trauma is pathognomonic for systemic disease - bilateral simultaneous quadriceps rupture almost never occurs in healthy individuals and should immediately alert you to underlying metabolic or systemic pathology. This is a classic exam scenario where the initial misdiagnosis as neurological pathology (Guillain-Barré, stroke, spinal cord pathology) is the trap - the key is recognizing the mechanical nature of the problem (palpable gap, patella baja) rather than neurological. The patient has multiple risk factors for quadriceps tendon rupture: (1) Chronic renal failure - the most important risk factor, causes tendinopathy through multiple mechanisms: secondary hyperparathyroidism (elevated PTH causes bone resorption at tendon insertion and calcium deposition in tendon), accumulation of β2-microglobulin (forms amyloid deposits in tendons), uraemic toxins impair collagen synthesis; (2) Dialysis-related hyperparathyroidism - PTH levels often markedly elevated (typically 500-1000 pg/mL, normal 10-65) causing profound bone-tendon interface weakening; (3) Diabetes - impairs collagen synthesis and tendon healing; (4) Age 55 - quadriceps rupture typically occurs in patients over 40 (unlike patellar tendon rupture which is younger). The diagnosis is clinical: bilateral inability to perform straight leg raise (complete loss of active knee extension), palpable suprapatellar gaps bilaterally, and patella baja on X-rays. The Insall-Salvati ratio would be less than 0.8 (normal 0.8-1.2), indicating low-riding patella from loss of superior restraint. I would confirm with ultrasound (quick bedside test showing tendon discontinuity and gap size) or MRI if needed, though the clinical diagnosis is clear. Management requires both medical and surgical approaches: (1) Medical optimization: Check renal function (creatinine, eGFR), electrolytes (calcium, phosphate - often deranged in dialysis patients), PTH level (likely very elevated), blood glucose control; Involve nephrology team for dialysis coordination around surgery and PTH management (may need parathyroidectomy if severe); Assess fitness for anaesthesia - dialysis patients are high-risk (cardiac disease, fluid overload); Ensure adequate dialysis pre-operatively to optimize metabolic status and reduce surgical risk; (2) Surgical management: Bilateral quadriceps rupture is an absolute indication for surgery - the patient cannot walk or transfer without active knee extension, so non-operative management is not an option; Staged vs simultaneous repair decision: Generally favor simultaneous bilateral repair in one operation - advantages are single anaesthetic exposure (important in high-risk renal patient), single rehabilitation period, patient can mobilize both legs together, psychologically better; Disadvantages of simultaneous are longer operative time (3-4 hours), higher DVT risk (bilateral surgery), more difficult post-operative care (both legs immobilized); Staged repair (1-2 week interval) allows one leg to be used for transfers but requires two operations in a high-risk patient - only consider if patient medically unstable; Surgical technique: Midline incision bilaterally, identify and mobilize torn quadriceps tendon (expect poor quality friable tissue - 'mop-end' appearance, may be retracted 2-3cm), freshen superior pole of patella (decorticate to bleeding bone to improve healing), transosseous tunnels preferred (drill 3 longitudinal tunnels through patella from superior to inferior pole using 2.5-3mm drill), heavy non-absorbable sutures #2 or #5 Ethibond or FiberWire in Krackow locking pattern (4-5cm of tendon purchase bilaterally), pass through patellar tunnels and tie anteriorly over anterior cortex or tie at inferior pole; Suture anchors alternative but transosseous considered gold standard (higher load to failure, though clinical outcomes similar); Critical: Restore patella height - compare intraoperatively to pre-injury Insall-Salvati ratio or contralateral normal (target 0.9-1.1), avoid over-tightening (creates patella infera/baja, increases patellofemoral contact pressure, causes anterior knee pain); Augmentation: In renal patients with poor tissue quality, consider reinforcement - cerclage wire or suture tape from tibial tubercle to proximal quadriceps, or Scuderi turndown flap (turn down strip of rectus femoris to reinforce repair), or synthetic mesh; Repair medial and lateral retinaculum if torn (important for patellar tracking); (3) Post-operative protocol: Hinged knee brace locked in full extension for ambulation 4-6 weeks (longer than standard due to poor tissue quality in renal patient); Immediate full weight-bearing allowed in locked brace (patients can mobilize and transfer); ROM protocol: 0° only for first 2 weeks, then 0-30° weeks 2-4, 0-60° weeks 4-6, 0-90° weeks 6-8, gradual to full by 12 weeks - much slower than normal patients due to poor healing capacity; Quadriceps strengthening: Isometric quad sets immediately, straight leg raises at 4-6 weeks when brace unlocked, progressive resistance once ROM achieved; DVT prophylaxis critical (mechanical and chemical - renal dose anticoagulation); Coordinate with dialysis schedule (dialysis on non-operative days to avoid heparinization affecting wound); (4) Prognosis and counseling: Explain that outcomes in renal patients are worse than healthy population - re-rupture rate 10-15% (vs 5% normal) due to poor tissue quality and impaired healing, stiffness common (30-40% have some residual limitation), extensor lag possible (inability to achieve last 10-20° of extension), infection risk 5-8% in dialysis patients (vs 2-3% normal); Most patients (70-80%) achieve functional ambulation and independence in ADLs, but return to pre-injury level may take 6-12 months; Long-term: Renal transplant would improve tissue quality and reduce future tendon problems, continue monitoring PTH and consider parathyroidectomy if persistently elevated.
KEY POINTS TO SCORE
Bilateral quadriceps tendon rupture pathognomonic for systemic disease - never occurs in healthy individuals: Chronic renal failure is most common cause (secondary hyperparathyroidism, β2-microglobulin accumulation, uraemic toxins impair collagen); PTH elevation causes bone resorption at tendon-bone interface and calcium deposition in tendon substance; Other systemic causes: Diabetes (impaired collagen synthesis), fluoroquinolones (ciprofloxacin - disrupts collagen crosslinking), chronic steroid use (impairs tendon healing), rheumatoid arthritis, gout, lupus; Bilateral simultaneous rupture with minimal trauma = red flag for metabolic disease; This patient has multiple risk factors: ESRD on dialysis, secondary hyperparathyroidism, diabetes, age over 40
Classic misdiagnosis as neurological pathology - key differences and diagnostic approach: Emergency teams often suspect Guillain-Barré syndrome (ascending paralysis), stroke (bilateral weakness), or spinal cord pathology (cauda equina) when patient 'cannot walk'; Key differentiating features: (1) Mechanical not neurological - palpable gaps (pathognomonic for tendon rupture), patella baja on X-ray, full passive ROM but no active extension; (2) Tendon injury selective for knee extension - patient can flex knee, move ankles/toes, has normal sensation; (3) Guillain-Barré would have ascending paralysis starting feet, areflexia, sensory changes, respiratory involvement; Clinical triad diagnostic: Palpable suprapatellar gap, inability to straight leg raise (SLR), patella baja (Insall-Salvati less than 0.8); Always palpate for gap in patient who 'cannot walk' - crucial exam skill; Imaging: Plain X-rays show patella baja (compare to normal Insall-Salvati 0.8-1.2), ultrasound quick bedside confirmation (tendon discontinuity, gap size), MRI if diagnostic doubt
Staged vs simultaneous bilateral repair - decision-making in high-risk renal patient: Simultaneous (both knees same operation) generally preferred: Single anaesthetic exposure (important - dialysis patients high cardiac risk), single recovery period (less disruption to dialysis schedule), psychological benefit (one ordeal), patient can mobilize both legs together with PT, cost-effective; Disadvantages: Longer operative time (3-4 hours), higher DVT/PE risk (bilateral surgery, renal patients hypercoagulable), post-op care more demanding (both knees immobilized), if complication on one side affects both; Staged repair (1-2 week interval) allows: One leg for transfers and mobility, reassess medical status between operations, less DVT risk per operation; Disadvantages: Two anaesthetic exposures in high-risk patient, prolonged total treatment (two separate recoveries), patient immobile on one leg during interval, psychological impact of two surgeries; Decision factors: Favor simultaneous if medically stable, good cardiac function, young (this patient 55 borderline), motivated patient, good social support; Favor staged if medically unstable (active cardiac issues, severe fluid overload), frail/elderly (over 70), poor social support, patient preference; Most surgeons do simultaneous in renal patients under 65 who are dialysis-stable
Surgical technique in poor tissue quality - modifications for renal patients: Tissue quality in ESRD patients poor - 'mop-end' friable tendon, thin and attenuated, poor healing capacity; Transosseous tunnels preferred over suture anchors: 3 longitudinal tunnels (2.5-3mm drill) superior to inferior through patella, heavy sutures (#2 or #5 Ethibond/FiberWire) in Krackow locking pattern (4-5cm purchase both sides of tear), pass through tunnels and tie anteriorly; Suture anchors acceptable (faster, less bone trauma) but transosseous higher load-to-failure and gold standard for exams; Augmentation MANDATORY in renal patients: Cerclage wire (18-gauge) or suture tape from tibial tubercle through quadriceps tendon proximal to repair - protects repair, shares load, allows earlier safe mobilization; Alternative: Scuderi turndown flap (mobilize strip of rectus femoris, turn distally to reinforce); Synthetic mesh reinforcement if tissue very poor; Restore patella height: Use C-arm intraoperatively to check lateral X-ray, target Insall-Salvati 0.9-1.1, compare to contralateral or pre-injury films if available, avoid overtightening = patella baja = patellofemoral pain; Repair retinaculum: Medial and lateral retinacular tears common, repair with absorbable sutures for tracking
Post-operative rehabilitation and prognosis in dialysis patients - realistic expectations: Slower protocol than healthy patients due to poor healing: Brace locked extension 4-6 weeks (vs 2-4 weeks normal), ROM 0-30° weeks 2-4, 0-60° weeks 4-6, 0-90° weeks 6-8 (vs 0-90° by 6 weeks normal), full ROM by 12 weeks (vs 8 weeks normal), return to baseline function 6-12 months (vs 4-6 months normal); Early mobilization crucial: FWB in locked brace immediately (prevents deconditioning, maintains transfers), quad sets day 1 post-op, SLR at 4-6 weeks when brace unlocked; DVT prophylaxis: Mechanical (TEDs, foot pumps), chemical anticoagulation (renal-dosed LMWH or warfarin), early mobilization; Dialysis coordination: Schedule dialysis non-operative days to avoid heparinization affecting wound healing, ensure adequate dialysis pre-op (reduce uraemic complications), monitor fluid balance (avoid overload but maintain perfusion); Prognosis worse than normal: Re-rupture 10-15% (vs 5% normal) - poor tissue quality, impaired healing, persistent hyperparathyroidism; Extensor lag 20-30% (inability to achieve last 10-20° extension); Stiffness 30-40% (adhesions, poor ROM compliance); Infection 5-8% (vs 2-3%) - immunosuppression, poor wound healing, dialysis access colonization; Despite challenges, 70-80% achieve functional independence (walk, transfers, ADLs) which is successful outcome given alternative is wheelchair-bound; Long-term: Renal transplant improves tissue quality, reduces future tendon problems; Parathyroidectomy if PTH persistently elevated (greater than 800-1000 pg/mL despite medical management)
COMMON TRAPS
✗Misdiagnosing as neurological pathology (Guillain-Barré syndrome, stroke, spinal cord lesion) - bilateral weakness misleads emergency teams; Key is palpating for gaps and recognizing mechanical not neurological problem
✗Missing the second side - focusing on more symptomatic knee and missing bilateral nature; Always examine both knees in extensor mechanism injury
✗Not recognizing that bilateral = systemic disease - isolated traumatic bilateral quad rupture essentially never happens; Must investigate for renal disease, hyperparathyroidism, diabetes, drug history
✗Operating without medical optimization - diving into surgery without checking renal function, PTH, calcium/phosphate, dialysis status, cardiac clearance; High perioperative risk in ESRD patients
✗Using standard rapid rehabilitation protocol - allowing early flexion greater than 30° or rapid progression causes re-rupture in poor quality tendon; Must go slower (4-6 week extension lock, gradual ROM)
✗Not augmenting the repair - transosseous tunnels alone insufficient in poor tissue quality; Cerclage wire or Scuderi flap reinforcement mandatory to prevent re-rupture
✗Over-tightening repair creating patella baja - excessive tension reduces patellar height, causes patellofemoral pain and arthritis; Must restore normal Insall-Salvati ~1.0
✗Overpromising outcomes - telling dialysis patient they'll return to normal or previous activity level; Must counsel about 10-15% re-rupture, possible extensor lag, 6-12 month recovery
LIKELY FOLLOW-UPS
"How does chronic renal failure lead to quadriceps tendon rupture? Explain the pathophysiology of secondary hyperparathyroidism"
"What is the Insall-Salvati ratio and how do you measure it? What are normal values?"
"What is the blood supply to the quadriceps tendon and where is the hypovascular zone?"
"Describe the transosseous tunnel technique for quadriceps tendon repair step-by-step"
"What is the Scuderi turndown flap and when would you use it?"
"What antibiotics are associated with tendon rupture and what is the mechanism?"
"How would your management change if this patient had one complete rupture and one partial rupture?"
"What are the arguments for staged versus simultaneous bilateral repair in this patient?"
VIVA SCENARIOChallenging

Scenario 2: Acute Complete Quadriceps Tendon Rupture - Surgical Technique

EXAMINER

"A 52-year-old type 2 diabetic man presents to your clinic 5 days after injuring his knee. He was walking down stairs when he missed a step and his right knee 'gave out'. He fell to the ground and has been unable to fully extend his knee or perform a straight leg raise since. He went to his GP initially who gave him crutches and told him he had a 'knee sprain', but when symptoms persisted he self-referred to your clinic. His past medical history includes well-controlled type 2 diabetes (HbA1c 7.2%, on metformin), hypertension, and hyperlipidaemia. He is otherwise fit and active, working as a carpenter. On examination, there is a palpable suprapatellar gap on the right knee. He has full passive range of motion (0-135°) but cannot perform an active straight leg raise - when you ask him to extend against gravity, the knee extends to about 30° short of full extension with visible quadriceps effort but no further. When you support the leg, he can hold it extended passively but it drops immediately when you release. The left knee is normal with full active extension. Plain radiographs show patella baja on the right (Insall-Salvati ratio 0.7) compared to normal on the left (1.0). There is no fracture. You arrange an urgent MRI which confirms complete rupture of the quadriceps tendon 1.5cm proximal to the superior pole of the patella, with 2cm of retraction. The tear appears to involve all three layers (rectus femoris, vasti, intermedius). How do you counsel him and what is your surgical plan?"

EXCEPTIONAL ANSWER
This is an acute complete quadriceps tendon rupture presenting at 5 days post-injury, which is within the ideal window for primary surgical repair. The delayed presentation due to initial misdiagnosis as a 'knee sprain' is unfortunately common - quadriceps ruptures are frequently missed in primary care because the knee may still have some passive extension and the diagnosis requires specific examination for the suprapatellar gap and active extension loss. I would counsel him that this is a complete rupture requiring surgical repair - non-operative management is not an option for complete ruptures as he will have permanent inability to extend the knee, difficulty with stairs and rising from chairs, and eventual quadriceps atrophy. The good news is that presentation at 5 days is ideal timing - acute repairs (less than 2-3 weeks) have excellent outcomes with primary repair, whereas chronic ruptures (greater than 6 weeks) develop tendon retraction, scarring, and quadriceps contracture requiring complex reconstruction with worse outcomes. His risk factors include age 52 (quadriceps ruptures typically occur over 40, unlike patellar tendon ruptures in younger patients) and diabetes (impairs collagen synthesis and tendon healing, increases rupture risk 2-3x). Fortunately, he has no other major systemic disease (unilateral rupture in otherwise healthy diabetic is typical, unlike bilateral which would suggest renal disease or severe metabolic disorder), and his diabetes is well-controlled (HbA1c 7.2% is acceptable, ideally less than 7.5% for elective surgery). Surgical timing: I would aim to operate within the next 3-7 days (total 10-14 days post-injury) which is the sweet spot - tissues are still mobile (less scarring than chronic), inflammation has settled (lower infection risk than first 48 hours), and tendon quality allows primary repair without reconstruction. Operating earlier than 3-5 days risks bleeding and higher infection, operating later than 3 weeks risks chronic changes. My surgical plan: (1) Pre-operative optimization: Ensure diabetes well-controlled (check HbA1c if not recent, optimize glucose less than 10 mmol/L), no active infection (check for skin lesions, wound on leg - diabetics prone to ulcers), medical clearance for GA (BP control, cardiac assessment); (2) Surgical technique: Position supine, tourniquet (inflate after exsanguination but only if diabetic vascular disease not severe - check pedal pulses), midline longitudinal incision centered on superior pole of patella (8-12cm), extend through prepatellar fascia and identify torn quadriceps tendon ends, mobilize torn tendon proximally (may need to release adhesions from vastus lateralis/medialis off femoral shaft laterally to gain length - can mobilize 2-3cm without weakening muscle), freshen tendon edges (debride to healthy viable tissue, should see good bleeding and solid tendon not friable) and superior pole of patella (decorticate cortex with rongeur to bleeding cancellous bone - improves healing interface); Transosseous tunnel repair: Drill 3 longitudinal tunnels through patella (I typically use 2.5mm drill bit, create tunnels from superior pole to inferior pole exiting anteriorly near midpoint of patella), use heavy non-absorbable braided sutures (#2 Ethibond or FiberWire - need strong suture for load), place sutures in Krackow locking pattern in tendon (enter tendon about 4cm proximal to tear, create locking stitches across width of tendon, 4-6 passes per side gives strong purchase), pass sutures through patellar tunnels from superior to inferior, bring out anteriorly, tension appropriately (restore patella height to normal Insall-Salvati ~1.0, check with C-arm lateral X-ray intraoperatively comparing to contralateral), tie sutures anteriorly over anterior patella cortex or bring through to inferior pole and tie there (some surgeons prefer inferior pole tie to avoid prominent anterior knot); Suture anchor alternative: Place 2-3 suture anchors in superior pole of patella (I would use 5.0-5.5mm anchors, Ethibond or high-strength suture), attach sutures to tendon in Krackow pattern as above, tension and tie; Evidence shows transosseous and anchors have equivalent clinical outcomes (Maniscalco 2017), transosseous slightly higher load-to-failure biomechanically but anchors faster (saves 10-15 minutes); For exams, state transosseous is 'gold standard' but both acceptable; Augmentation: Consider cerclage wire or suture tape augmentation in diabetic patient to protect repair - pass 18-gauge wire or FiberTape from tibial tubercle (drill tunnel) through quadriceps tendon 3-4cm proximal to repair, tension with knee in full extension, acts as 'safety belt' sharing load and protecting repair during early flexion; Not mandatory in acute repair of healthy tissue but prudent in diabetic (impaired healing); Repair retinaculum: The medial and lateral retinacular expansions of the quadriceps are often torn when the central tendon ruptures - repair these with absorbable sutures (#1 Vicryl) to restore tracking and reinforce the construct; Closure: Repair prepatellar fascia, subcutaneous, skin; (3) Post-operative protocol: Hinged knee brace locked in full extension for ambulation 2-4 weeks (some surgeons use 0-6 weeks but 2-4 is standard if secure repair with augmentation), immediate full weight-bearing allowed in locked brace (early mobilization prevents stiffness, muscle atrophy, DVT), ROM progression: 0° only first 2 weeks (brace locked), then 0-30° weeks 2-4, 0-60° weeks 4-6, 0-90° weeks 6-8, unrestricted by 8-10 weeks; Physiotherapy: Quadriceps isometric sets (quad sets) starting day 1 post-op (in extension, no risk to repair), straight leg raises once brace unlocked at 2-4 weeks (active knee extension in brace), progressive resistance strengthening once good quad control (8-12 weeks), closed-chain exercises (leg press, squats) at 12 weeks, return to work as carpenter 3-4 months (may need modified duties avoiding deep squatting initially), return to unrestricted activity 6 months; DVT prophylaxis (mechanical + chemical for 2 weeks minimum, diabetics higher risk); Monitor wound healing closely (diabetics prone to wound complications); (4) Expected outcomes: With acute repair, excellent outcomes expected - 80-90% return to pre-injury function, 5-8% re-rupture risk, 10-15% residual extensor lag (inability to achieve last 5-10° extension), 20-30% develop some anterior knee pain (from hardware, patellofemoral changes, or overtight repair), nearly all patients (greater than 95%) can return to work as carpenter within 4-6 months. His diabetes increases infection risk (5% vs 2-3% normal) and may impair healing slightly, but with good glucose control prognosis remains very good.
KEY POINTS TO SCORE
Acute complete quadriceps tendon rupture - clinical diagnosis and imaging confirmation: Classic clinical triad diagnostic: (1) Palpable suprapatellar gap (1-2cm above patella, may be obscured by swelling but careful palpation reveals), (2) Loss of active knee extension (cannot perform straight leg raise, knee sags when try to hold extended against gravity), (3) Patella baja on X-ray (Insall-Salvati ratio less than 0.8, compare to contralateral normal 0.8-1.2); Patient may retain some active extension if partial tear or intact retinaculum compensating (this patient extends to 30° off full suggesting severe but retinaculum partially intact); Full passive ROM preserved (differentiates from arthrofibrosis, locked knee) but no active extension; X-ray: Lateral view shows low-riding patella (patella baja), measure Insall-Salvati (patellar tendon length divided by patellar height, normal 0.8-1.2, less than 0.8 = baja); Compare to contralateral (this patient 0.7 vs 1.0 confirms baja); MRI gold standard for confirmation: Shows complete tear of all three layers (rectus femoris superficial, vasti middle, intermedius deep), quantifies retraction (2cm in this case = moderate, can mobilize for primary repair), assesses tendon quality (look for degenerative changes, thinning, areas of high T2 signal indicating pre-existing tendinopathy); MRI not always necessary if clinical diagnosis clear, but useful for surgical planning and documentation
Timing of surgical repair - acute vs subacute vs chronic management: Acute (less than 2-3 weeks): Primary repair with transosseous tunnels or suture anchors, excellent outcomes (80-90% return to function), tendon mobile and good quality, ideal window 5-14 days post-injury (inflammation settled but before significant scarring); Subacute (3-6 weeks): Primary repair still possible but more difficult (tendon retracts 3-5cm, early scarring, quadriceps begins to shorten), may need mobilization techniques (release vastus lateralis/medialis from femoral shaft, flexing knee to 90-110° during surgery to relax muscle), augmentation more important (cerclage wire or Scuderi flap); Chronic (greater than 6 weeks): Direct repair often impossible (tendon retracted 5-8cm+, dense scar, quadriceps contracture, muscle fatty infiltration/atrophy), requires reconstruction (Codivilla V-Y lengthening, Scuderi turndown, allograft), outcomes worse (60-70% good results vs 80-90% acute, permanent extensor lag common 10-30°); This patient at 5 days is ideal timing - acute repair with excellent prognosis; Key teaching: 'The clock is ticking' - delays worsen outcomes, must identify and refer urgently (less than 2 weeks)
Transosseous tunnel vs suture anchor repair - technical comparison and exam answer: Transosseous tunnel technique (gold standard for exams): Drill 3 longitudinal tunnels (2.5-3mm) from superior to inferior pole of patella (exit anteriorly or at inferior pole), heavy non-absorbable braided sutures #2 or #5 Ethibond or FiberWire, Krackow locking stitch in tendon (4-6 passes, 4cm proximal purchase), pass through tunnels and tie anteriorly over bone bridge or at inferior pole; Biomechanically highest load-to-failure (400-600N), distributes force across three bone bridges, no implant-related complications; Disadvantages: Requires multiple drill holes in patella (theoretically weakens but not clinically significant), technically more time-consuming (15-20 minutes vs 10 minutes anchors), suture knots may be prominent anteriorly (palpable, rarely symptomatic); Suture anchor technique: Insert 2-3 suture anchors (5.0-5.5mm) in superior pole of patella, attach sutures to tendon in Krackow pattern, tension and tie; Faster (saves 10-15 minutes), less bone trauma, lower profile (no anterior knots); Biomechanically adequate (300-500N load-to-failure), clinical outcomes equivalent to transosseous (Maniscalco 2017 - no difference in re-rupture, ROM, or function); Disadvantages: Implant cost (£300-500 vs £20 for sutures), theoretical anchor pullout risk (especially osteoporotic bone), may complicate future surgery if anchor migrates; Exam answer: 'Both techniques acceptable, transosseous considered gold standard with slightly higher biomechanical strength, but clinical outcomes equivalent; I would use transosseous for robust fixation in this case'
Augmentation techniques - when to reinforce repair to prevent failure: Cerclage wire or suture tape augmentation: Pass 18-gauge stainless steel wire or synthetic suture tape (FiberTape, Internal Brace) from tibial tubercle (drill 3-4mm tunnel) through quadriceps tendon 3-4cm proximal to repair, tension with knee in full extension, acts as load-sharing 'seatbelt' protecting primary repair; Indications: Poor tissue quality (diabetic, elderly, degenerative tendon), delayed presentation (subacute 3-6 weeks), revision repair after failure, high-demand patient returning to heavy labor; Evidence: Reduces re-rupture rate from 8-10% to 3-5%, allows earlier safe mobilization, removes as outpatient at 6-12 months if symptomatic (palpable, may irritate with kneeling); Scuderi turndown flap: Turn down a proximally based flap of rectus femoris tendon (typically 2cm wide, 8-10cm long), fold distally over repair, suture to patella and tendon; Provides biologic reinforcement, more robust than wire but technically complex, permanent (cannot remove); Indications: Chronic tears requiring lengthening, poor tissue quality, revision; Synthetic mesh: Rarely used, risk of infection, rejection; This patient (diabetic, acute repair with 2cm retraction): I would use cerclage wire augmentation given diabetes (impaired healing) and need to return to heavy carpentry work - provides safety margin during rehabilitation
Post-operative rehabilitation and return to work - balancing protection with mobilization: Early phase (0-2 weeks): Brace locked in full extension for all ambulation, immediate FWB allowed in locked brace (studies show no increased re-rupture with early WB), quadriceps isometric sets (quad sets) in extension safe (no flexion forces on repair), ice and elevation for swelling; Flexion phase (2-8 weeks): Gradual ROM progression - unlock brace to 30° weeks 2-4 (low tension on repair in early flexion), progress to 60° weeks 4-6 (moderate tension), 90° weeks 6-8 (high tension at end range so delay), unrestricted 8-10 weeks; Straight leg raises once brace unlocked (2-4 weeks) with brace on initially, then without; Rationale for slow flexion: Quadriceps tendon under maximal tension in deep flexion (90-120°), too early flexion risks gapping repair or stretching sutures causing elongation and extensor lag; Strengthening phase (8-16 weeks): Progressive resistance exercises (ankle weights, leg press) starting 8-10 weeks once ROM adequate, closed-chain exercises (squats, leg press) safer than open-chain (knee extension machine) for patellofemoral joint, functional training (stairs, hills, balance) 10-12 weeks; Return to work: Light desk work 2-4 weeks, modified carpentry 3-4 months (avoid deep squatting, kneeling, heavy lifting initially), full unrestricted work 4-6 months depending on demands; Return to sport: Recreational sports (cycling, swimming) 3-4 months, impact sports (running, basketball) 6 months minimum, require full ROM, quad strength 85-90% contralateral, no extensor lag, surgeon clearance; Stiffness prevention: Early ROM within protocol, avoid prolonged immobilization (greater than 6 weeks increases adhesions), aggressive PT if flexion contracture develops (less than -5° at 8 weeks needs intensive stretching)
COMMON TRAPS
✗Misdiagnosing as 'knee sprain' and missing the rupture - failure to palpate for suprapatellar gap; Always examine for gap in any patient with knee 'giving way' or loss of extension
✗Confusing with patellar tendon rupture - both have loss of extension but opposite X-ray findings (patella baja in quad rupture vs patella alta in patellar tendon); Remember 'quad = baja, patellar = alta'
✗Operating too early (less than 3-5 days) when tissues inflamed and bleeding risk high, or too late (greater than 3 weeks) risking chronic changes
✗Poor surgical technique - inadequate tendon purchase with sutures (need 4cm Krackow), insufficient bone preparation (must decorticate patella), not restoring patella height (over-tightening creates baja)
✗Not augmenting repair in diabetic patient - diabetes impairs healing, should use cerclage wire or suture tape to protect repair
✗Aggressive early rehabilitation - allowing flexion greater than 30° before 4 weeks risks repair failure; Must follow gradual protocol despite patient pressure to progress faster
✗Not recognizing partial vs complete tear - if patient has ANY active extension (even 10-20° short of full), consider partial tear which may be managed non-operatively with brace; Complete = absolutely no active extension
✗Operating with poorly controlled diabetes (HbA1c greater than 8-9%, glucose greater than 12 mmol/L) - high infection risk (10-15%); Must optimize glucose first
LIKELY FOLLOW-UPS
"How would your management change if this was a partial tear with 50% tendon continuity on MRI?"
"What is the Krackow locking stitch and why is it used for tendon repairs?"
"How do you measure the Insall-Salvati ratio on lateral X-ray? What are the normal values?"
"Describe step-by-step how you would perform the transosseous tunnel repair"
"What would you do if intraoperatively you found you could not bring the tendon down to the patella even with mobilization?"
"Why does diabetes increase the risk of tendon rupture and impair healing?"
"What are the biomechanical differences in outcomes between transosseous tunnels and suture anchors?"
"If this patient presented at 8 weeks instead of 5 days, how would your surgical approach change?"
VIVA SCENARIOCritical

Scenario 3: Chronic Neglected Quadriceps Tendon Rupture - Reconstructive Challenge

EXAMINER

"You are seeing a 60-year-old man in your reconstructive clinic who was referred from a rural area. He sustained a quadriceps tendon rupture 10 weeks ago when he fell on icy steps. He was seen at a local emergency department where X-rays were performed and showed no fracture, and he was told he had a 'severe knee contusion' and given crutches and analgesia. He was unable to walk properly or extend his knee, but because he lives alone on a farm and has limited mobility, he did not seek further medical attention. Over the past 10 weeks, he has been largely immobile, using crutches and a wheelchair, and has developed significant quadriceps atrophy. His daughter recently visited and insisted he see a specialist, which led to this referral. On examination, you note severe quadriceps wasting with the thigh circumference 8cm less than the contralateral side. There is a palpable suprapatellar depression. He cannot perform any active knee extension - when you ask him to try, you see quadriceps muscle contraction proximally but no movement at the knee. His passive range of motion is limited to 30-100° due to a fixed flexion contracture that has developed. The patella is palpable very high in the suprapatellar region. Plain radiographs show severe patella baja with Insall-Salvati ratio of 0.5 (compared to 1.1 on the contralateral side). MRI shows complete quadriceps tendon rupture with 6cm of retraction and scarring. The tendon stump is retracted and adherent to the distal femur. There is significant quadriceps muscle atrophy with fatty infiltration and contracture. The superior pole of the patella shows bone resorption. He is desperate to regain some function as he cannot manage his farm independently. How do you counsel him and what are the surgical options?"

EXCEPTIONAL ANSWER
This is a chronic neglected quadriceps tendon rupture presenting at 10 weeks post-injury, which represents a complex reconstructive challenge with a worse prognosis than acute repair. The delayed presentation due to misdiagnosis as a 'knee contusion' and patient's social isolation has led to the development of chronic changes that make simple primary repair impossible: (1) Tendon retraction (6cm) with dense scar tissue adherent to femur, (2) Quadriceps muscle contracture with fatty infiltration and atrophy (8cm thigh circumference loss), (3) Fixed flexion contracture of the knee (30° - cannot fully extend passively), (4) Severe patella baja (Insall-Salvati 0.5 indicating massive superior migration), (5) Bone resorption at superior pole from chronic traction. These chronic changes mean that simple suture repair is not possible - even if I could mobilize the retracted tendon down to the patella, the tension would be enormous causing either immediate re-rupture or severe patella infera, and the degenerated atrophic muscle may not generate adequate force even if repaired. I need to counsel him with realistic expectations: (1) This is a salvage procedure, not restoration to normal - at 10 weeks with these chronic changes, the best we can hope for is functional improvement allowing assisted ambulation and transfers, not return to normal or heavy farm work; (2) Even with surgery, he will likely have permanent extensor lag (inability to achieve full active extension), residual weakness (atrophied muscle with fatty infiltration does not fully recover), and need for mobility aids long-term; (3) Surgical options are complex reconstructive procedures with significant risks (infection 10-15%, re-rupture 20-30%, stiffness, need for further procedures); (4) Non-operative management (accepting current disability, using wheelchair and mobility aids, strengthening remaining function) is a valid alternative if he is not medically fit or does not accept surgical risks. However, given his age (60, relatively young), desire to return to farm independence, and current complete loss of function, I would offer him surgical reconstruction with appropriate counseling about realistic outcomes. Surgical options for chronic quadriceps rupture: (1) Codivilla V-Y quadriceps lengthening/plasty: The most commonly used technique for chronic ruptures - involves creating a V-shaped incision in the quadriceps muscle-tendon unit proximally (typically starting 8-10cm above the patella), extending distally to the rupture site; The V incision allows mobilization of the distal tendon stump down to the patella (the two arms of the V separate, lengthening the construct); Once the tendon is advanced and repaired to the patella (using transosseous tunnels or suture anchors as in acute repair), the proximal defect where the V was created is closed in a Y configuration (hence V-Y); This effectively lengthens the extensor mechanism by 4-6cm, allowing repair under acceptable tension; Advantages: Uses patient's own tissue, establishes continuity, well-described technique; Disadvantages: Weakens quadriceps (cutting muscle), limited lengthening (maximum 6cm, may be insufficient for this patient with 6cm retraction plus contracture), residual weakness common (30-40% loss of strength); (2) Scuderi quadriceps turndown flap: Turn down a proximally-based flap of the rectus femoris tendon (typically 2cm wide, 10-12cm long) and fold it distally to bridge the gap; The flap is sutured to the retracted tendon stump and to the patella, providing biologic reinforcement and bridging the defect; Often combined with V-Y lengthening for large gaps; Advantages: Provides additional tissue for reinforcement, biologic augmentation, can bridge 6-8cm gaps when combined with V-Y; Disadvantages: Weakens rectus femoris, complex technically, adds operative time; (3) Achilles tendon allograft: Use fresh-frozen Achilles allograft with attached calcaneus bone block (10-11mm diameter Achilles, 2-3cm calcaneus bone block); Fix the calcaneus bone block to the superior pole of patella with large interference screw (7-9mm), weave the Achilles tendon through the quadriceps muscle proximally and suture to muscle with heavy non-absorbable sutures; Augment with cerclage wire or suture tape from tibial tubercle through quadriceps; Advantages: Can span any gap size, strong thick graft, single-stage procedure, does not further weaken patient's quadriceps muscle; Disadvantages: Allograft risks (disease transmission minimal with modern screening but theoretical, incorporation failure 10-15%, cost, availability), more complex surgery, graft may stretch over time; (4) Whole extensor mechanism allograft: Rarely used, involves quadriceps-patella-patellar tendon-tibial tubercle allograft construct replacing entire mechanism; Reserved for massive loss or multiple failed reconstructions; My recommendation for this patient: I would favor Achilles tendon allograft reconstruction given the 6cm gap, severe muscle atrophy, and need for robust construct. The patient's own quadriceps is already severely atrophied and weak (8cm circumference loss, fatty infiltration on MRI), so further weakening it with V-Y or Scuderi flap is not ideal. The allograft provides strong tissue that can span the gap without further compromising his muscle. Surgical plan: (1) Pre-operative: Medical optimization (check comorbidities, anesthetic risk - 60yo farmer likely has cardiac disease, diabetes), physiotherapy to improve passive ROM and reduce flexion contracture (intensive stretching for 2-4 weeks pre-op, aim to get extension to at least 10-15° short of full - currently 30° contracture makes surgery very difficult), plan for prolonged surgery (3-4 hours) and recovery; (2) Surgical technique: Position supine with tourniquet, midline incision (15-20cm) centered on superior pole of patella extending proximally, identify and mobilize retracted quadriceps tendon stump (will be densely adherent to femur with scar - sharp dissection required, may need to release vastus lateralis/medialis from femoral shaft to gain any length), prepare superior pole of patella (debride soft tissue, decorticate to healthy bleeding bone - may have significant bone loss requiring grafting or remodeling), prepare Achilles allograft (thaw per protocol, trim calcaneus bone block to appropriate size for patella, prepare tendon end); Fix calcaneus bone block to superior pole of patella: Create trough in superior pole with rongeur and burr to accept bone block (2-3cm deep), position block and compress, fix with large interference screw (7-9mm bioabsorbable or metal) directed anteroinferiorly - screw should achieve rigid fixation (bone-to-bone healing most reliable part of construct); Weave Achilles tendon through quadriceps muscle: Pass sutures through Achilles in Krackow pattern, create tunnels/windows through quadriceps muscle and tendon stump proximally (about 8-10cm above patella), pull Achilles through and suture to quadriceps with heavy non-absorbable sutures under appropriate tension; Tension: Flex knee to 60-90° (as much as flexion contracture allows), apply moderate tension to restore patella height toward normal (target Insall-Salvati 0.8-0.9, up from 0.5 - full correction may not be possible), use intraoperative C-arm to check position; CRITICAL: Must accept some residual patella baja - trying to fully correct to 1.0-1.1 will create excessive tension and fail; Augmentation: MANDATORY - cerclage wire (18-gauge) or suture tape from tibial tubercle through graft and quadriceps acts as safety belt, shares load, protects graft during healing and early rehabilitation; Repair retinaculum if any salvageable tissue; Closure: May need Z-plasty or V-Y closure of skin if significant shortening makes primary closure tight; (3) Post-operative protocol (very conservative given chronic changes and reconstruction): Brace locked in extension 6-8 weeks minimum (longer than acute repair - graft needs time to incorporate), non-weight-bearing or touch-toe weight-bearing for 6 weeks (protect bone-bone healing of calcaneus block), ROM extremely gradual: 0° only first 4-6 weeks, then 0-30° weeks 6-10, 0-60° weeks 10-14, 0-90° weeks 14-20 (may never achieve beyond 90° given pre-existing contracture and muscle damage), passive ROM first with therapist, active-assisted once muscle reactivates, full active quad strengthening delayed 12-16 weeks (must allow graft incorporation before loading); DVT prophylaxis critical (prolonged immobilization, surgery time, age 60); Serial X-rays to monitor patella position and bone block healing (bone block should heal by 8-12 weeks, graft incorporation takes 6-9 months); Expected timeline: 12-18 months to maximal recovery; (4) Realistic prognosis and counseling: Outcomes for chronic quadriceps reconstruction worse than acute repair: Only 50-60% achieve functional extensor mechanism (defined as ability to perform straight leg raise and ambulate without brace), 40-50% have permanent extensor lag (typically 10-30° - cannot achieve last 10-30° of active extension, compensate by hyperextending hip), quadriceps strength recovery limited (at best 50-70% of normal - muscle has undergone atrophy and fatty infiltration which is irreversible), return to farm work very unlikely (may achieve light duties, but heavy labor, climbing, kneeling not realistic), complications high: Re-rupture 20-30% (vs 5-8% acute), infection 10-15% (long surgery, dead space, allograft), stiffness 40-50% (pre-existing contracture, prolonged immobilization, adhesions), graft failure or stretching 15-20% (persistent lag despite intact graft), need for revision surgery 20-25%, permanent disability 30-40% (persistent severe lag or failure requiring brace or mobility aids); Success definition: Goal is functional independence for basic ADLs (transfers, walking with walker or cane, light household tasks) NOT return to normal or farm work; If he can achieve SLR and walk with assistive device, that is successful outcome given baseline of wheelchair-dependent; Must counsel that surgery is salvage attempt, not cure, and even with surgery he may need long-term mobility aids; Alternative: Non-operative management with bracing (locked knee brace or KAFO), wheelchair, home modifications - may be appropriate if medically unfit or unwilling to accept surgical risks and prolonged recovery. This is a difficult decision requiring extensive counseling and shared decision-making with patient and family.
KEY POINTS TO SCORE
Chronic quadriceps tendon rupture (10 weeks) - pathoanatomic changes preventing primary repair: Tendon retraction and scarring: Torn tendon retracts proximally (typically 5-8cm in chronic cases, 6cm in this patient), becomes densely adherent to distal femur with scar tissue, stump atrophies and degenerates; Quadriceps muscle contracture: Muscle shortens over weeks-months (natural resting length decreases), develops fatty infiltration (MRI shows fat replacing muscle fibers), atrophies significantly (8cm circumference loss indicating 30-40% muscle volume loss); Fixed flexion contracture: Knee cannot be passively fully extended (30° in this patient), develops from prolonged positioning in flexion (sitting, wheelchair), posterior capsule and hamstrings tighten; Severe patella baja: Insall-Salvati 0.5 (normal 0.8-1.2) indicates massive proximal migration due to loss of superior restraint, bone resorption at superior pole from chronic abnormal traction; These changes make simple primary repair impossible - cannot bring tendon down without massive tension, muscle too weak/degenerate to function even if repaired; Chronic defined as greater than 6 weeks (some say 3 weeks) - requires reconstruction not simple repair
Surgical options for chronic reconstruction - comparing techniques and indications: Codivilla V-Y quadriceps lengthening: Create V-shaped incision in quadriceps 8-10cm proximal, extend distally to rupture, mobilize distal limb of V down to patella, repair with transosseous tunnels/anchors, close proximal defect in Y configuration; Lengthens mechanism 4-6cm, uses patient's own tissue; Indications: Moderate retraction (3-5cm), some viable quadriceps muscle remaining, gap less than 6cm; Outcomes: 60-70% functional recovery, 30-40% residual weakness from muscle incision; Scuderi turndown flap: Proximally-based rectus femoris flap (2cm wide, 10-12cm long) turned distally to bridge gap and reinforce; Often combined with V-Y for large gaps; Provides biologic augmentation; Indications: Large gaps (6-8cm) when combined with V-Y, poor tendon quality needing reinforcement; Outcomes: Similar to V-Y, adds complexity but may improve healing; Achilles allograft: Fresh-frozen Achilles with calcaneus bone block, fix bone to superior patella pole (interference screw), weave Achilles through quadriceps proximally; Can span any gap, does not weaken patient's quadriceps, strong graft; Indications: Large retraction (greater than 6cm), severe muscle atrophy (patient's quadriceps already weak - don't weaken further with V-Y), failed previous repair, need for robust construct; Outcomes: 50-60% functional, 15-20% graft failure/stretching, allograft risks; I favor allograft in this case given 6cm gap and severe pre-existing atrophy
Achilles allograft reconstruction technique - step-by-step surgical approach: Pre-operative: Pre-op PT to improve flexion contracture (intensive stretching 2-4 weeks, aim to get extension within 10-15° of full - currently 30° contracture too severe for surgery), plan for 3-4 hour surgery; Graft preparation: Fresh-frozen Achilles allograft thawed per protocol, trim calcaneus bone block to fit patella (typically 2-3cm long, 1.5-2cm wide), prepare Achilles tendon end (Krackow sutures if weaving through muscle); Exposure: Midline incision 15-20cm, mobilize retracted quadriceps stump from femur (sharp dissection, dense scar), release vastus lateralis/medialis from femoral shaft if needed for length, prepare superior pole of patella (debride soft tissue, rongeur cortex to bleeding bone, may need to remodel if bone resorbed); Bone block fixation: Create trough in superior pole with rongeur/burr (2-3cm deep, slightly undersize bone block for compression fit), position calcaneus block and compress into trough, fix with large interference screw 7-9mm (bioabsorbable or metal) directed anteroinferiorly, screw must achieve rigid fixation - this is most critical part (bone-to-bone healing is most reliable); Tendon weaving: Pass Achilles proximally through quadriceps muscle (create tunnels 8-10cm above patella, may weave through or around muscle belly), suture Achilles to quadriceps with heavy non-absorbable (#2 or #5 Ethibond) in multiple locations for broad contact; Tensioning: Flex knee to tolerance (60-90° depending on contracture), restore patella height toward normal (target IS 0.8-0.9, NOT full correction to 1.0+ which creates excessive tension), use C-arm lateral X-ray intraoperatively to assess; Augmentation MANDATORY: Cerclage wire or suture tape from tibial tubercle through graft/quadriceps as safety belt; Closure: May need plastic surgery techniques if skin tight (Z-plasty, V-Y advancement)
Post-operative rehabilitation for chronic reconstruction - prolonged conservative protocol: Much slower than acute repair due to: (1) Graft needs incorporation (6-9 months vs 3-4 months native tendon), (2) Bone block healing critical (6-12 weeks NWB/TTWB), (3) Pre-existing muscle atrophy and contracture limit recovery, (4) Higher failure risk requires caution; Immobilization phase (0-6 weeks): Brace locked extension, NWB or TTWB (protect bone block), 0° ROM only (no flexion to protect graft-bone interface), quad isometrics in extension only, ice/elevation; Early motion phase (6-14 weeks): Unlock brace to 30° weeks 6-10 (once bone block healed on X-ray at 8-10 weeks), progress to 60° weeks 10-14, passive ROM by therapist first, active-assisted once muscle activates; PWB to FWB weeks 8-14; Strengthening phase (14-24 weeks): Progress to 90° flexion weeks 14-20 (may not achieve beyond 90° given pre-existing contracture), active quad strengthening 12-16 weeks (isometric, SLR, resistance), closed-chain exercises 16-20 weeks (leg press, mini squats); Functional phase (24+ weeks): Wean brace 20-24 weeks if adequate quad strength and no lag, gait training with assistive device (walker progressing to cane), ADL training (transfers, stairs with rail); Maximal recovery 12-18 months; Serial X-rays: Monitor patella height (should remain stable, if progressively migrating superior = graft stretching/failure), bone block healing (should see bridging bone 8-12 weeks), hardware position (screw migration = loosening); Complications to monitor: Graft failure (sudden loss of extension, gap reappears, patella migrates superior - 15-20% incidence), infection (10-15% - long surgery, allograft, dead space), stiffness (40-50% - pre-existing contracture, prolonged immobilization, aggressive PT needed)
Realistic outcomes and prognostic counseling - managing expectations in salvage surgery: Success rate chronic reconstruction MUCH worse than acute repair: Functional recovery 50-60% (vs 80-90% acute) - defined as ability to perform SLR and ambulate without brace; Permanent extensor lag 40-50% (vs 10-15% acute) - typically 10-30°, cannot achieve last degrees of extension, compensate with hip hyperextension; Quadriceps strength severely limited 50-70% of normal at best (vs 80-85% acute) - muscle has undergone irreversible atrophy and fatty infiltration visible on MRI, 8cm circumference loss indicates 30-40% muscle volume lost which does NOT fully recover; Complications high: Re-rupture 20-30% (vs 5-8% acute) especially in first 6 months before full graft incorporation, infection 10-15% (vs 2-3% acute) from prolonged surgery and allograft, stiffness 40-50% (pre-existing contracture exacerbated by prolonged immobilization), graft failure/stretching 15-20% (graft elongates over time causing progressive lag), need for revision 20-25% (further reconstruction or conversion to brace); Functional outcomes: Return to farm work (heavy manual labor) extremely unlikely (less than 10-20%), may achieve light ADLs (walking with assistive device, transfers, household tasks) in 60-70%, 30-40% remain significantly disabled requiring long-term brace (KAFO) or wheelchair despite surgery; Success definition must be realistic: Goal is functional independence for BASIC activities (transfers, ambulation with walker/cane, self-care) NOT return to normal function or work; If patient achieves SLR and walks with cane, that is SUCCESSFUL outcome given baseline of wheelchair-dependent; Surgery is salvage attempt not cure - must emphasize this repeatedly; Alternative: Non-operative with bracing and mobility aids valid option if medically unfit or unwilling to accept risks and prolonged recovery; This requires extensive shared decision-making
COMMON TRAPS
✗Attempting simple primary repair in chronic rupture - tendon retracted 6cm, massive tension would cause immediate re-rupture or severe patella infera; Must recognize chronic changes require reconstruction
✗Not counseling realistic expectations - telling patient he will return to farm work or normal function is cruel and sets up disappointment; Must emphasize salvage goal of basic function
✗Operating without pre-operative physiotherapy - attempting reconstruction with 30° fixed flexion contracture makes surgery extremely difficult and risks failure; Must stretch contracture first
✗Over-tensioning graft to fully correct patella baja - trying to restore Insall-Salvati from 0.5 to 1.1 creates excessive tension causing graft failure; Accept residual baja at 0.8-0.9
✗Not augmenting with cerclage wire/suture tape - chronic reconstruction without augmentation has 30-40% failure rate; Augmentation mandatory
✗Aggressive early rehabilitation - allowing flexion or weight-bearing before bone block healed (8 weeks) risks pulling out interference screw and graft failure
✗Using V-Y plasty in severely atrophic muscle - patient's quadriceps already atrophied 30-40% with fatty infiltration; Further weakening with V-Y incision compromises already poor muscle
✗Not obtaining informed consent for poor outcomes - must discuss 20-30% re-rupture, 40-50% permanent lag, 30-40% persistent disability despite surgery; Failure to consent for realistic outcomes is medicolegal risk
LIKELY FOLLOW-UPS
"Describe the Codivilla V-Y plasty technique step-by-step - where do you make the V incision and how do you close in a Y?"
"What is the Scuderi turndown flap and how does it differ from the V-Y lengthening?"
"Why does fatty infiltration of muscle seen on MRI indicate poor prognosis even if you reconstruct the tendon?"
"What is the blood supply to the quadriceps tendon and where is the hypovascular zone of rupture?"
"How would you counsel a patient choosing between attempting reconstruction versus accepting permanent disability with bracing?"
"If the interference screw pulled out at 4 weeks post-op, how would you manage this complication?"
"What are the risks of using allograft tissue and how do you counsel patients about disease transmission?"
"At what point (time post-injury) would you say a quadriceps rupture is too chronic to even attempt reconstruction?"

MCQ Practice Points

Exam Pearl

Q: What is the classic clinical triad of quadriceps tendon rupture?

A: 1) Palpable suprapatellar gap, 2) Loss of active knee extension (inability to perform straight leg raise), 3) Patella baja on lateral X-ray. The gap is palpable at the superior pole of the patella. Unlike patellar tendon rupture (patella alta), quad rupture shows low-riding patella.

Exam Pearl

Q: What are the major risk factors for quadriceps tendon rupture?

A: Age over 40 (most common extensor mechanism rupture in this group), chronic renal failure (accumulation of β2-microglobulin), diabetes mellitus, hyperparathyroidism, steroid use (local or systemic), fluoroquinolone antibiotics, and gout. Bilateral simultaneous rupture strongly suggests systemic disease.

Exam Pearl

Q: What is the layered anatomy of the quadriceps tendon?

A: The quadriceps tendon has three layers: Superficial - rectus femoris, Middle - vastus lateralis and medialis converging, Deep - vastus intermedius. The rectus femoris continues over the patella as the prepatellar fascia. Most ruptures occur 0-2 cm above the superior pole of the patella in a relatively hypovascular zone.

Exam Pearl

Q: What is the treatment of choice for complete quadriceps tendon rupture?

A: Primary surgical repair through transosseous tunnels in the patella. Technique involves passing heavy non-absorbable sutures (Krackow or Bunnell pattern) through the tendon and through three longitudinal drill holes in the patella. Repair is augmented with medial and lateral retinacular repair. Must restore patella height (compare to contralateral).

Exam Pearl

Q: What is the Codivilla V-Y plasty used for in quadriceps tendon surgery?

A: Used for chronic ruptures with retraction where direct repair is not possible. The technique involves creating a V-shaped incision in the quadriceps muscle proximally, allowing distal mobilization of the tendon. The resulting proximal defect is closed in a Y configuration. Restores tendon length but may result in some extensor weakness.

Australian Context

Australian Epidemiology and Practice

Australian Quadriceps Tendon Rupture Epidemiology:

  • Quadriceps tendon rupture is relatively uncommon compared to other knee injuries
  • Incidence higher in populations with chronic kidney disease - a significant issue given Australia's high prevalence of end-stage renal disease
  • Bilateral quadriceps tendon rupture associated with dialysis patients is encountered at major tertiary centres

RACS Orthopaedic Training Relevance:

  • Quadriceps tendon rupture is a core FRCS Orthopaedic examination topic
  • Examiners commonly test the clinical diagnosis (palpable gap, patella baja, loss of active extension)
  • Key differentiators from patellar tendon rupture (patella alta) must be known
  • Transosseous repair technique is the gold standard answer for examination purposes
  • Candidates must be able to discuss V-Y lengthening for chronic ruptures

Australian Surgical Practice:

  • Major orthopaedic trauma centres manage quadriceps tendon repairs
  • Transosseous tunnels remain the most commonly used fixation technique
  • Suture anchors increasingly used as an alternative with equivalent outcomes
  • Allograft reconstruction available through Australian tissue banks for chronic cases

Australian Rehabilitation Services:

  • Outpatient physiotherapy services coordinate post-operative rehabilitation
  • Hinged knee bracing readily available through orthopaedic suppliers
  • Early mobilisation protocols with protected weight-bearing are standard practice

DVT Prophylaxis (eTG Recommendations):

  • Chemical prophylaxis with enoxaparin recommended for lower limb surgery with reduced mobility
  • Mechanical prophylaxis (graduated compression stockings, intermittent pneumatic compression) used adjunctively
  • Duration typically 10-14 days post-operatively or until adequate mobility achieved

Antibiotic Prophylaxis:

  • Cefazolin 2g IV at induction for tendon repair procedures
  • Single dose prophylaxis is standard for uncomplicated cases

Management Algorithm

📊 Management Algorithm
Management algorithm for Quadriceps Tendon Rupture
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Management algorithm for Quadriceps Tendon RuptureCredit: OrthoVellum

Quad Rupture Quick Reference

High-Yield Exam Summary

Triad

  • •1. Palpable Gap (Supra-patellar)
  • •2. Loss of Active Extension (SLR)
  • •3. Patella Baja (X-ray)

Risk Factors

  • •Age over 40
  • •Renal Failure
  • •Diabetes
  • •Fluoroquinolones

Treatment

  • •Complete: Repair (Transosseous)
  • •Partial: Splint 6w
Quick Stats
Reading Time142 min
Related Topics

Patellar Tendon Rupture

Distal Biceps Rupture

TKA Extensor Mechanism Failure

Extensor Tendon Injuries