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Trigger Finger (Stenosing Tenosynovitis)

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Trigger Finger (Stenosing Tenosynovitis)

Comprehensive guide to trigger finger including diagnosis, Green classification, injection technique, and surgical release.

complete
Updated: 2026-01-02
High Yield Overview

TRIGGER FINGER

A1 Pulley | Stenosing Tenosynovitis | Injection | Release

A1Pulley affected
RingMost common digit
70%Injection success
95%Surgical success

Green Classification

Grade 1
PatternPain and history of catching, no demonstrable catching
TreatmentConservative
Grade 2
PatternDemonstrable catching, able to actively extend
TreatmentInjection
Grade 3
PatternLocking, requires passive extension to unlock
TreatmentInjection or surgery
Grade 4
PatternFixed locked position, unable to passively extend
TreatmentSurgery

Critical Must-Knows

  • A1 Pulley: Location of stenosis at MCP level. Release A1, NOT A2.
  • Ring and Middle Fingers: Most commonly affected digits.
  • Steroid Injection: 70% success rate. Lower in diabetics (50%).
  • A2 Pulley: CRITICAL - never release. Causes bowstringing if released.
  • Diabetics: 10% prevalence, multiple digits, lower injection success.

Examiner's Pearls

  • "
    Palpable nodule at A1 (MCP level)
  • "
    Worse in morning (tissue edema)
  • "
    Diabetics: 10% prevalence, multiple fingers
  • "
    Preserve A2 = fundamental principle

Clinical Imaging

Imaging Gallery

Peritendinous injections for release of trigger finger: (A) Trigger finger is caused by a tenosynovitis of the digital flexor tendons and presents with snapping fingers. Peritendinous steroid injectio
Click to expand
Peritendinous injections for release of trigger finger: (A) Trigger finger is caused by a tenosynovitis of the digital flexor tendons and presents witCredit: Daftary AR et al. via Indian J Radiol Imaging via Open-i (NIH) (Open Access (CC BY))
A–B. (A) Initial photograph after actively flex fingers shows some degree of flexion at MP joint. (B) Initial photograph after actively extend fingers shows extension of MP joint, but there is no rang
Click to expand
A–B. (A) Initial photograph after actively flex fingers shows some degree of flexion at MP joint. (B) Initial photograph after actively extend fingersCredit: Lee YK et al. via Medicine (Baltimore) via Open-i (NIH) (Open Access (CC BY))
A–B. Preoperative AP (A) and oblique (B) radiographs demonstrate a Salter–Harris type II fracture of the small finger proximal phalanx base and volar angulation with callus formation.
Click to expand
A–B. Preoperative AP (A) and oblique (B) radiographs demonstrate a Salter–Harris type II fracture of the small finger proximal phalanx base and volar Credit: Lee YK et al. via Medicine (Baltimore) via Open-i (NIH) (Open Access (CC BY))
Clinical photograph showing bilateral trigger fingers with locked flexion
Click to expand
Clinical presentation of multiple trigger fingers: (A) Patient unable to fully grip - fingers lock in flexed position during forceful grasp. (B) Attempted extension reveals multiple digits (thumbs, middle, ring fingers) locked in flexion bilaterally. This case demonstrates severe (Grade 3-4) trigger finger requiring surgical A1 pulley release.Credit: Lee YK et al. - J. Korean Med. Sci. (CC BY 4.0)

Critical Trigger Finger Exam Points

A1 Pulley Location

Stenosis occurs at A1 pulley (MCP level). Nodule in flexor tendon catches at pulley during motion. Release A1, never A2.

Preserve A2 Pulley

A2 is critical for tendon function. Releasing A2 causes bowstringing and loss of grip. A2 and A4 are the critical pulleys.

Diabetic Trigger Finger

10% prevalence in diabetics. Multiple digits common. Injection success only 50%. May need surgery first-line.

Injection Technique

Inject into tendon SHEATH, not tendon. At A1 pulley level. 70% success. May repeat once. Avoid tendon injection.

Quick Decision Guide

GradePresentationTreatmentKey Action
Grade 1Pain only, no catchingConservative, splintTrial of splint and activity modification
Grade 2Catching, active extension OKInjection first70% success with steroid injection
Grade 3Locking, passive unlock neededInjection or surgeryConsider surgery if injection fails
Grade 4Fixed lockedSurgeryA1 pulley release indicated
Mnemonic

A1-A2-A3-A4-A5Annular Pulley System

A1
MP level
Release for trigger finger
A2
Proximal phalanx
CRITICAL - do NOT release
A3
PIP level
Minor pulley
A4
Middle phalanx
CRITICAL - do NOT release
A5
DIP level
Minor pulley

Memory Hook:A2 and A4 are the CRITICAL pulleys - never release them! A1, A3, A5 are minor and can be released safely.

Mnemonic

DRAGTrigger Finger Risk Factors

D
Diabetes
10% prevalence, multiple digits, lower injection success
R
Rheumatoid arthritis
Inflammatory tenosynovitis
A
Age (50-60 years)
Peak incidence
G
Gripping repetitively
Occupational exposure

Memory Hook:DRAG = Diabetes, RA, Age 50-60, Gripping - know the risk factors for trigger finger!

Mnemonic

SHEATHInjection Technique

S
Steroid
Triamcinolone or methylprednisolone
H
Horizontal at MCP
Inject at MCP crease
E
Enter sheath
Into sheath, NOT tendon
A
Aspirate first
Ensure not in tendon
T
Test resistance
Low resistance = in sheath
H
Half ml steroid
0.5ml steroid + 0.5ml LA

Memory Hook:SHEATH = where you inject! Into the tendon sheath, not the tendon itself.

Overview and Epidemiology

Why Trigger Finger Matters

Trigger finger is extremely common in the exam. Key points: A1 pulley location, preserve A2, injection 70% success, diabetics have higher prevalence and lower injection response.

Trigger Finger (stenosing tenosynovitis) is catching or locking of a digit due to stenosis at the A1 pulley.

Demographics

  • Prevalence: 2-3% general population
  • Gender: Female greater than male (6:1)
  • Age: Peak 50-60 years
  • Digits: Ring greater than middle greater than thumb greater than index greater than small

Middle-aged women are the classic demographic.

Risk Factors

  • Diabetes: 10% prevalence (vs 2% general)
  • Rheumatoid arthritis: Inflammatory tenosynovitis
  • Gout: Crystal-induced
  • Repetitive gripping: Occupational
  • Carpal tunnel: Associated conditions

Screen diabetics for trigger finger.

Pathophysiology and Mechanisms

Pulley Anatomy

There are 5 annular pulleys (A1-A5) and 3 cruciate pulleys (C1-C3). A2 and A4 are CRITICAL for tendon function - never release them. A1, A3, A5 are minor and can be safely released.

Flexor Tendon Sheath Anatomy:

  • A1 Pulley: At MCP joint level - where trigger finger occurs
  • A2 Pulley: Over proximal phalanx - CRITICAL, do not release
  • A3 Pulley: At PIP joint level - minor
  • A4 Pulley: Over middle phalanx - CRITICAL, do not release
  • A5 Pulley: At DIP joint level - minor

Pathophysiology:

  1. Repetitive friction causes tendon sheath thickening at A1
  2. Nodule forms in flexor tendon (FDS or FDP)
  3. Nodule catches at narrowed A1 pulley
  4. Results in catching, triggering, or locking

Classification Systems

Green Severity Grading

GradeDescriptionClinical FeaturesTreatment
Grade 1Pain, palpable noduleHistory of catching, no demonstrable catchingSplinting, activity modification
Grade 2CatchingDemonstrable catching, can actively extendSteroid injection (70% success)
Grade 3LockingRequires passive extension to unlockInjection or surgery
Grade 4Fixed lockedUnable to passively extend the digitSurgical release indicated

Higher grades progressively more likely to need surgery.

Frequency by Digit

DigitFrequencyNotes
RingMost common30% of cases
MiddleSecond25% of cases
ThumbThird20% - different anatomy
IndexFourth15% of cases
SmallLeast10% of cases

Multiple digits involved in 20% (especially diabetics).

Clinical Assessment

History

  • Catching or clicking: During flexion/extension
  • Locking: May need passive unlock
  • Worse in morning: Tissue edema after rest
  • Pain at palm: Over MCP crease
  • Progressive: Gets worse over time

Ask about diabetes and occupation.

Examination

  • Palpable nodule: At A1 pulley (MCP crease)
  • Tenderness: Over A1 pulley
  • Triggering: Demonstrable with flexion/extension
  • Grade: Can actively extend, needs passive, or fixed?
  • Other fingers: Check all digits

Always palpate at the MCP crease, not PIP.

Differential Diagnosis

Consider: Dupuytren's contracture (cord, not nodule at A1), FDS/FDP anomaly (absent FDS in small finger), MCP joint pathology (arthritis, locking from loose body), tenosynovitis (RA, infection).

Investigations

Investigation Protocol

ClinicalPrimary Diagnosis

Clinical diagnosis in most cases. Palpable nodule at A1 with triggering is pathognomonic. No imaging needed routinely.

BloodsScreen for Diabetes

HbA1c or fasting glucose if not known diabetic. Diabetics have 10% trigger finger prevalence and need careful counseling about lower injection success.

ImagingUltrasound (If Needed)

Rarely required. Can show tendon sheath thickening, A1 pulley thickening, tendon nodule. Useful if diagnosis uncertain.

Ultrasound of trigger finger showing A1 pulley thickening
Click to expand
Longitudinal ultrasound of the palm demonstrating trigger finger: (A) Shows thickening of the A1 pulley region with the flexor tendon visible beneath. (B) Dynamic imaging showing the tendon gliding beneath the constricted pulley. Ultrasound can confirm diagnosis in uncertain cases and may be used to guide injection, though trigger finger remains primarily a clinical diagnosis based on palpable nodule and characteristic triggering.Credit: Open-i/PMC - CC BY 4.0

No imaging is required for typical trigger finger with clear clinical findings.

Management Algorithm

📊 Management Algorithm
trigger finger management algorithm
Click to expand
Management algorithm for trigger fingerCredit: OrthoVellum

Non-Operative Management

Conservative Options

First LineActivity Modification

Reduce gripping activities. Especially repetitive gripping. Modify work tasks if occupational.

SplintingExtension Splint

Night splint keeping MCP in extension. Prevents tendon nodule catching overnight. Variable success.

AdjunctsNSAIDs

Short-term anti-inflammatory. May reduce symptoms but won't resolve stenosis.

Conservative treatment has limited long-term success as sole treatment.

Corticosteroid Injection

Technique:

  • Insert needle at MCP crease at 45 degrees
  • Direct into tendon SHEATH (not tendon)
  • Feel needle slide under A1 pulley
  • Inject 0.5ml steroid + 0.5ml lidocaine
  • Should flow easily (no resistance)

Success Rate: 70% cure with 1-2 injections

Diabetics: 50% success (consider surgery first-line)

May repeat once if partial response.

When to Operate

Indications for Surgery:

  • Grade 4 (fixed locked)
  • Failed 2 injections
  • Diabetic with high-grade triggering
  • Patient preference

Not indicated: Mild symptoms, no prior conservative/injection trial.

Surgical Technique

Surgical landmarks for percutaneous A1 pulley release
Click to expand
Surgical planning for trigger finger release: Palmar hand showing surface landmarks for A1 pulley location. Purple markings indicate the proximal interphalangeal (PIP) crease distance and palmar digital crease (PDC) used to predict A1 pulley boundaries. The distal edge of A1 lies approximately 5mm proximal to the PDC. Critical to identify these landmarks to ensure complete A1 release while preserving A2.Credit: Hazani R et al. - Eplasty (CC BY 4.0)

Open A1 Pulley Release

Surgical Steps

1Anesthesia and Positioning

Local anesthesia. Wide-awake local anesthesia no tourniquet (WALANT) preferred. Hand on table.

2Incision

Transverse incision at MCP crease. 1-1.5cm. Follow skin crease for cosmesis.

3Dissection

Blunt spread to expose A1 pulley. Identify and protect digital nerves (lateral).

4Release

Incise A1 pulley longitudinally. Divide completely. STOP at A2.

5Check Glide

Test tendon excursion. Ask patient to flex/extend. Confirm no triggering.

6Closure

Skin only. Interrupted sutures or steri-strips. Soft dressing.

Simple, quick procedure (less than 10 minutes). LA suitable.

Percutaneous Release

Technique:

  • Needle-based A1 division
  • 18G needle or blade through skin
  • Sweep to divide pulley

Advantages: No incision, office-based

Disadvantages:

  • Blind technique
  • Higher nerve risk (especially thumb)
  • Incomplete release possible

Less commonly performed. Requires experience.

Structures at Risk

StructureLocationInjury Consequence
Digital nerveLateral to flexor sheathNumbness, neuroma
A2 pulleyJust distal to A1Bowstringing, weakness
Flexor tendonDeep to A1Tendon damage, adhesions
Digital arteryWith digital nerveBleeding, ischemia (rare)

Neurovascular bundles are lateral. Stay central.

Complications

Complications of Trigger Finger Treatment

ComplicationIncidencePrevention/Management
Incomplete release5%Ensure complete A1 division, check glide
Digital nerve injuryLess than 1%Direct vision, stay central, protect nerves
BowstringingRareNEVER release A2 - critical pulley
InfectionLess than 1%Sterile technique
Stiffness5%Early motion, hand therapy if needed
RecurrenceLess than 3%Complete release, address underlying disease

A2 pulley injury causing bowstringing is a serious complication. Prevention is key - know anatomy.

Postoperative Care

Postoperative Protocol

Day 0Immediate

Soft dressing. Immediate finger ROM encouraged. No splint needed.

Day 2-7Early Motion

Active and passive ROM. Full motion as tolerated. Keep wound dry.

Week 2Suture Removal

Remove sutures. Most patients fully functional by this point. Scar massage.

Week 4Full Activity

Return to all activities. Including gripping and lifting. Scar tenderness may persist.

Return to work: Light duties immediately. Full duties 2-4 weeks.

Outcomes and Prognosis

Success Rates:

  • Conservative (splint): Variable (30-40%)
  • Injection: 70% (lower in diabetics - 50%)
  • Surgery: 95-100%

Prognostic Factors:

FactorBetter OutcomeWorse Outcome
DurationShort (under 6 months)Long (over 1 year), fixed locked
DiabetesNon-diabeticDiabetic (50% injection success)
GradeLower gradeHigher grade (4 = surgery)
DigitsSingle digitMultiple digits

Most patients do very well with injection or surgery.

Evidence Base

Systematic Review
📚 Peters-Veluthamaningal et al
Key Findings:
  • Steroid injection effective vs placebo
  • 70% success rate with single injection
  • Multiple injections may improve outcomes
  • Limited high-quality evidence for splinting
Clinical Implication: Steroid injection is effective first-line treatment.
Source: Cochrane 2009

RCT
📚 Kazuki et al
Key Findings:
  • Open vs percutaneous release
  • Similar success rates (greater than 95%)
  • Percutaneous higher recurrence in some studies
  • Open provides direct visualization
Clinical Implication: Both techniques are effective; open is safer and more reliable.
Source: J Hand Surg Am 2006

Cohort Study
📚 Ring et al
Key Findings:
  • Diabetics have 10% trigger finger prevalence
  • Multiple digits common in diabetics
  • 50% injection success (vs 70% non-diabetic)
  • May need surgery as first-line
Clinical Implication: Counsel diabetics about lower injection success; consider early surgery.
Source: J Hand Surg Am 2008

Prospective Study
📚 Bain et al
Key Findings:
  • A1 pulley release technique
  • 97% complete resolution
  • Low complication rate
  • Outpatient procedure effective
Clinical Implication: Open A1 release is safe and highly effective.
Source: J Hand Surg Br 1995

Systematic Review
📚 Colbourn et al
Key Findings:
  • Comparison of injection vs surgery
  • Injection 57% long-term success
  • Surgery greater than 95% success
  • Surgery more cost-effective if injection fails
Clinical Implication: Surgery is definitive; injection may avoid surgery in majority.
Source: J Hand Surg Eur 2008

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Classic Trigger Finger

EXAMINER

"A 55-year-old woman has catching in her ring finger for 2 months. She feels a click at the palm and sometimes needs to straighten the finger with her other hand. There is a tender nodule at the MCP crease. What is your management?"

EXCEPTIONAL ANSWER
This is trigger finger (stenosing tenosynovitis) at the A1 pulley of the ring finger - the most commonly affected digit. The catching, need for passive extension, and palpable nodule at MCP level are classic findings. This is Grade 3 (locking requiring passive unlock). My first-line treatment would be corticosteroid injection into the tendon sheath at the A1 pulley level. Technique: I inject using a 25G needle at the MCP crease at 45 degrees, into the tendon sheath (not the tendon itself). I use 0.5ml triamcinolone plus 0.5ml local anesthetic. There should be low resistance if in the sheath. Success rate is approximately 70%. I would review in 4-6 weeks. If symptoms persist, I would offer a second injection or surgical release. At surgery, I would perform open A1 pulley release under WALANT: transverse incision at MCP crease, expose A1 pulley protecting digital nerves laterally, divide A1 longitudinally, check tendon glide, close skin only. Critical: I would preserve A2 pulley which is essential for tendon function.
KEY POINTS TO SCORE
A1 pulley at MCP level
Injection 70% success
Inject into sheath, not tendon
Surgery: release A1, preserve A2
COMMON TRAPS
✗Injecting into the tendon
✗Releasing A2 pulley at surgery
✗Not screening for diabetes
LIKELY FOLLOW-UPS
"Which pulley is critical?"
"What about trigger thumb in children?"
VIVA SCENARIOChallenging

Scenario 2: Diabetic with Multiple Triggers

EXAMINER

"A 60-year-old diabetic man has triggering in his ring, middle, and index fingers. He has had one injection to each finger with only partial improvement. What is your approach?"

EXCEPTIONAL ANSWER
This patient has multiple trigger fingers in the context of diabetes, which has 10% prevalence of trigger finger. Diabetics have lower injection success (50% vs 70%) and commonly have multiple digits involved. Given he has failed one injection to each finger with only partial response, I would counsel him that surgery is likely the best next step. For diabetics, some surgeons offer surgery as first-line given the lower injection response rate. My approach would be open A1 pulley release to all three affected digits. This can be done as a single procedure under WALANT or a wrist block. I would make separate transverse incisions at each MCP crease, release each A1 pulley fully while preserving A2, confirm tendon glide in each digit, and close with skin sutures. I would optimize his diabetes perioperatively, check HbA1c, and warn about higher infection risk. Post-operatively, immediate active motion is encouraged.
KEY POINTS TO SCORE
Diabetics: 10% prevalence, 50% injection success
Multiple digits common in diabetics
Consider surgery first-line in diabetics
Can release multiple digits in one procedure
COMMON TRAPS
✗Repeating injections indefinitely in a diabetic
✗Not checking diabetes control
LIKELY FOLLOW-UPS
"What is the injection success rate in diabetics?"
"What are the surgical risks?"
VIVA SCENARIOStandard

Scenario 3: Fixed Locked Finger

EXAMINER

"A 70-year-old woman presents with her middle finger locked in flexion for 2 weeks. She cannot straighten it actively or passively. Her MCP is tender. What is your management?"

EXCEPTIONAL ANSWER
This is Grade 4 (fixed locked) trigger finger - the most severe grade. The finger is locked in flexion and cannot be passively extended, indicating a mature, fixed stenosis at A1 with the tendon nodule trapped proximal to the pulley. For Grade 4, I would recommend surgical release as first-line - injection is unlikely to succeed for a fixed locked digit. I would perform open A1 pulley release: transverse incision at MCP crease, careful dissection protecting digital nerves, complete release of A1 pulley longitudinally, and then gently extending the finger while watching the tendon to ensure it glides freely. I may need to break up adhesions between the nodule and pulley. I would preserve A2. Post-operatively, immediate motion is critical to prevent stiffness, and I would involve hand therapy if there is residual stiffness. I would also check for diabetes and other underlying conditions.
KEY POINTS TO SCORE
Grade 4 = fixed locked = surgery first-line
Injection unlikely to succeed
May need adhesion release
Early motion critical post-op
COMMON TRAPS
✗Trying injection for fixed locked finger
✗Aggressive manipulation without surgery
LIKELY FOLLOW-UPS
"What is the Green classification?"
"What if the finger is still stiff after release?"

MCQ Practice Points

A1 Pulley Location

Q: At what level does trigger finger occur? A: A1 pulley at the MCP joint level. This is where the stenosis and nodule catching occurs.

Critical Pulley

Q: Which pulley must be preserved during trigger finger release? A: A2 pulley. A2 and A4 are critical for tendon function. Releasing A2 causes bowstringing.

Injection Success

Q: What is the success rate of steroid injection for trigger finger? A: 70% in general population. Only 50% in diabetics.

Diabetes Prevalence

Q: What is the prevalence of trigger finger in diabetics? A: 10% (vs 2-3% general population). Multiple digits common. Lower injection success.

Most Common Digit

Q: Which digit is most commonly affected in trigger finger? A: Ring finger (30%), followed by middle (25%), thumb (20%), index (15%), small (10%).

WALANT Technique

Q: What is WALANT and why is it ideal for trigger finger release? A: Wide Awake Local Anesthesia No Tourniquet. Allows patient to actively flex/extend to confirm complete release and resolution of triggering.

Australian Context

Australian Guidelines:

  • RACS supports injection as first-line for Grades 1-3
  • Surgery indicated for Grade 4 or failed injections
  • Medicare rebates available for injection (item 30206) and release (item 30070)

Medicolegal Considerations:

  • Document grade and prior treatment
  • Consent: discuss nerve injury, incomplete release, bowstringing
  • Document diabetes status and counseling about lower success

PBS/Prescribing:

  • Corticosteroid injections are not PBS-restricted
  • Triamcinolone, methylprednisolone commonly used
  • Can be performed in rooms or operating theater

Australian surgeons should document comprehensive consent including A2 preservation.

TRIGGER FINGER

High-Yield Exam Summary

Pathology

  • •Stenosing tenosynovitis at A1 pulley
  • •Ring greater than middle most common
  • •Nodule catches at narrowed pulley
  • •Diabetics: 10% prevalence

Clinical

  • •Catching/locking with flexion
  • •Worse in morning
  • •Palpable nodule at MCP crease
  • •Tender at A1 pulley

Green Classification

  • •1: Pain, no catching
  • •2: Catching, active extension
  • •3: Locking, passive unlock
  • •4: Fixed locked = surgery

Treatment

  • •Injection: 70% success (50% diabetics)
  • •Surgery: 95%+ success
  • •Grade 4 = surgery first-line
  • •Can repeat injection once if partial response

Surgical Principle

  • •Release A1 pulley completely
  • •PRESERVE A2 pulley (critical)
  • •Check tendon glide
  • •Protect digital nerves (lateral)

Key Points

  • •A2 and A4 = critical pulleys
  • •Diabetics: 10% prevalence
  • •Ring finger most common
  • •Multiple digits in diabetics
Quick Stats
Reading Time64 min
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