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Extensor Tendon Injuries - Foot and Ankle

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Extensor Tendon Injuries - Foot and Ankle

Comprehensive guide to extensor tendon injuries of the foot - anatomy, mechanism, acute and chronic management, surgical repair techniques, and rehabilitation for orthopaedic exam

complete
Updated: 2025-12-25
High Yield Overview

EXTENSOR TENDON INJURIES - FOOT AND ANKLE

Laceration or Rupture | Zone Classification | Early Repair | Protected Mobilization

8Extensor zones in foot/ankle
TAMost important extensor (dorsiflexion)
3-4wProtected immobilization duration
6-8wReturn to activities timeline

FOUR MAIN EXTENSOR TENDONS

Tibialis Anterior (TA)
PatternStrongest dorsiflexor
TreatmentAlways repair if divided
Extensor Hallucis Longus (EHL)
PatternIP/MTP hallux extension
TreatmentRepair to restore function
Extensor Digitorum Longus (EDL)
PatternLesser toe extension
TreatmentRepair when feasible
Peroneus Tertius
PatternAccessory dorsiflexor/everter
TreatmentRepair not always required

Critical Must-Knows

  • Tibialis anterior is the most important extensor - always repair if injured
  • Zone classification guides repair technique and prognosis (8 zones from muscle belly to toe)
  • Early repair (within 2 weeks) gives best outcomes - delayed repair more difficult
  • Protected mobilization balances tendon healing with preventing adhesions
  • EDL tendons to lesser toes may not require repair if extensor digitorum brevis intact

Examiner's Pearls

  • "
    TA rupture causes foot drop - unopposed plantar flexion during swing phase
  • "
    EHL injury prevents hallux IP extension - important for propulsion
  • "
    Multiple tendon injuries common with lacerations across dorsum of foot
  • "
    Repair strength depends on suture technique - modified Kessler or Krackow preferred

Critical Extensor Tendon Exam Points

Zone System Critical

Eight zones from muscle belly to toe guide management. Zone 1-3 (muscle/MTJ/distal leg) best prognosis. Zone 4-5 (ankle/dorsal foot) moderate adhesion risk. Zone 6-8 (MTP/phalanges) high adhesion risk but limited excursion.

TA Dominance

Tibialis anterior provides 80% of dorsiflexion power. Loss leads to foot drop gait. Always explore and repair if suspected injury. EHL can partially compensate but inadequate alone.

Repair Timing

Primary repair (within 2 weeks) is ideal - direct end-to-end repair possible. Delayed primary (2-6 weeks) may need tendon advancement. Late reconstruction (more than 6 weeks) often requires graft or transfer.

Rehabilitation Balance

Balance protection (prevent rupture) with early motion (prevent adhesions). Controlled dorsiflexion in first 3-4 weeks. Full weight bearing delayed until 6 weeks. Stiffness more common than re-rupture.

Quick Decision Guide - Extensor Tendon Management by Zone

ZoneLocationInjury PatternManagement Priority
1-2Muscle belly and MTJLaceration or strainDebride, approximate muscle. Good healing potential
3Distal legLaceration commonPrimary repair with core and epitenon sutures
4Ankle and superior retinaculumLaceration or closed rupture (TA)Critical zone - repair essential for TA/EHL
5Dorsum of foot to MTPLaceration, often multiple tendonsRepair all major tendons. EDL may not need repair if EDB intact
6-8MTP and digitsMallet toe, lacerationZone 6: repair. Zone 7-8: splinting often sufficient
Mnemonic

TALE - The Four Main Extensors

T
Tibialis Anterior
Strongest - dorsiflexion and inversion
A
(and)
Working together on dorsum
L
Longus - EHL and EDL
Extensor Hallucis Longus + Extensor Digitorum Longus
E
Eversion assist (Peroneus Tertius)
Weakest extensor, also everts

Memory Hook:Tell a TALE of the four extensors - TA is the hero of the story

Mnemonic

EIGHT ZONES - Foot Extensor Zones

1
Muscle belly
Proximal leg - good healing
2
Musculotendinous junction
Mid leg - transition zone
3
Distal leg
Above ankle - tendinous portion
4
Ankle and retinaculum
Critical zone for TA/EHL
5
Dorsum of foot
Multiple tendons - laceration common
6
MTP joint level
Tendon expansion begins
7
Proximal phalanx
Central slip equivalent
8
DIP level
Terminal tendon - mallet toe

Memory Hook:EIGHT zones progress from proximal to distal - remember ankle (4) and MTP (6) are critical transitions

Mnemonic

REPAIR - Primary Repair Principles

R
Recognize early
Within 2 weeks ideal for primary repair
E
Explore completely
Identify all injured structures
P
Preserve blood supply
Gentle tissue handling, minimal stripping
A
Adequate suture technique
Core suture (Krackow/Kessler) plus epitenon
I
Immobilize appropriately
Ankle in neutral or slight dorsiflexion
R
Rehabilitate progressively
Protected motion at 3-4 weeks

Memory Hook:REPAIR guides you from recognition through rehabilitation

Mnemonic

DROP FOOT - TA Rupture Clinical Features

D
Dorsiflexion weak or absent
Cannot lift foot against gravity
R
Running difficult
Toe catches during swing phase
O
Obvious gap palpable
Anterior ankle contour loss
P
Plantarflexed position at rest
Gravity foot drop in sitting
F
Footslap on heel strike
Uncontrolled plantarflexion
O
Overpowered by plantar flexors
Unopposed gastrocsoleus
O
Obvious during single limb stance
Cannot stand on heel
T
Testing shows grade 0-1 power
MRC grading

Memory Hook:DROP FOOT describes the clinical picture when TA is ruptured

Overview and Epidemiology

Extensor tendon injuries of the foot and ankle encompass a spectrum from acute lacerations to chronic degenerative ruptures. Unlike flexor tendons, extensor tendons are more superficial and vulnerable to laceration but have better healing potential due to abundant vascular supply and looser paratenon.

Mechanisms of injury:

  • Laceration - most common, often from glass, machinery, or sharp objects across dorsum of foot
  • Closed rupture - tibialis anterior most susceptible, usually at musculotendinous junction or insertion
  • Avulsion - terminal extensor avulsion causes mallet toe
  • Attrition rupture - chronic inflammatory arthropathy or repetitive trauma

Epidemiology:

  • Lacerations most common in young active males (20-40 years)
  • Closed TA rupture typically older patients (40-60 years) with degenerative changes
  • Multiple tendon injuries common with extensive lacerations
  • EHL injuries often associated with TA injuries due to anatomical proximity

Why Extensors Different from Flexors?

Better healing potential than flexor tendons because: (1) More robust vascular supply from paratenon, (2) Less critical gliding requirements, (3) Lower tensile loads. However, still require formal repair for optimal function, especially tibialis anterior.

Clinical significance by tendon:

Tibialis Anterior:

  • Most powerful dorsiflexor (80% of dorsiflexion strength)
  • Critical for toe clearance in swing phase
  • Loss causes foot drop and altered gait
  • Always requires repair

Extensor Hallucis Longus:

  • Extends hallux IP and MTP joints
  • Important for terminal stance and push-off
  • Loss impairs propulsion and causes clawing
  • Repair strongly recommended

Extensor Digitorum Longus:

  • Extends lesser toes at MTP and IP joints
  • Extensor digitorum brevis provides backup extension at MTP
  • Repair when feasible but functional loss less severe

Peroneus Tertius:

  • Accessory dorsiflexor and everter
  • Least important of the four
  • Repair not always necessary

Anatomy and Biomechanics

Gray's anatomy illustration showing the anterior compartment muscles of the leg including tibialis anterior, extensor hallucis longus, and extensor digitorum longus
Click to expand
Anterior compartment muscles of the leg (Gray's Anatomy). The tibialis anterior (most medial) provides primary dorsiflexion. The extensor digitorum longus lies laterally, with extensor hallucis longus emerging between them distally.Credit: Gray's Anatomy. Public Domain

The extensor compartment of the leg contains four muscles and tendons:

1. Tibialis Anterior (TA)

  • Origin: Lateral tibial condyle, proximal lateral tibia, interosseous membrane
  • Insertion: Medial cuneiform and base of first metatarsal
  • Function: Dorsiflexion (primary), inversion (secondary)
  • Nerve: Deep peroneal nerve (L4, L5)
  • Unique features: Largest cross-sectional area, most medial tendon at ankle

2. Extensor Hallucis Longus (EHL)

  • Origin: Middle anterior fibula, interosseous membrane
  • Insertion: Base of hallux distal phalanx
  • Function: Hallux IP and MTP extension, weak dorsiflexion
  • Nerve: Deep peroneal nerve (L5, S1)
  • Unique features: Passes between TA and EDL at ankle

3. Extensor Digitorum Longus (EDL)

  • Origin: Lateral tibial condyle, proximal fibula, interosseous membrane
  • Insertion: Middle and distal phalanges of lesser toes (2-5)
  • Function: Toe extension at MTP and IP joints, weak dorsiflexion
  • Nerve: Deep peroneal nerve (L5, S1)
  • Unique features: Divides into four slips on dorsum of foot

4. Peroneus Tertius

  • Origin: Distal anterior fibula (part of EDL complex)
  • Insertion: Base of 5th metatarsal (dorsal aspect)
  • Function: Dorsiflexion, eversion
  • Nerve: Deep peroneal nerve (L5, S1)
  • Unique features: Often absent (10-15% of population)

Anatomical relationships:

At the ankle (Zone 4):

  • Tendons pass deep to superior and inferior extensor retinaculum
  • Order from medial to lateral: TA, EHL, EDL, Peroneus Tertius ("THE DEPT")
  • Deep peroneal nerve and anterior tibial artery pass deep to inferior retinaculum
  • Retinaculum prevents bowstringing and provides mechanical advantage

On dorsum of foot (Zone 5):

  • Tendons diverge as they progress distally
  • Extensor digitorum brevis (EDB) originates from calcaneus, inserts on EDL tendons
  • EDB provides MTP extension even if EDL divided
  • EHL crosses superficial to dorsalis pedis artery
Gray's anatomy illustration of the dorsum of the foot showing extensor tendons and their relationships
Click to expand
Dorsum of the foot (Gray's Anatomy) showing the extensor tendons as they diverge distally. Note the extensor digitorum brevis muscle belly arising from the calcaneus, which provides backup MTP extension if EDL is divided.Credit: Gray's Anatomy. Public Domain
Anatomical photograph showing the tendons of extensor digitorum longus and peroneus tertius on the dorsum of the foot
Click to expand
Cadaveric dissection showing the four slips of extensor digitorum longus (EDL) diverging to the lesser toes, with peroneus tertius inserting on the base of the fifth metatarsal laterally. This Zone 5 region is commonly injured by lacerations across the dorsum.Credit: Wikimedia Commons. CC BY-SA 4.0

Vascular supply:

  • Paratenon provides diffuse blood supply
  • Better vascularity than flexor tendons
  • Anterior tibial artery gives small branches
  • Critical to preserve during repair

Zone 4 Critical Zone

Zone 4 (ankle/retinaculum) is critical because: (1) Tendons confined in tight compartments under retinaculum, (2) TA and EHL most vulnerable here, (3) Adhesions to retinaculum common, (4) Deep peroneal nerve at risk during exploration.

Biomechanics:

Force requirements:

  • Tibialis anterior: 300-500N during normal gait
  • EHL: 50-100N during push-off
  • EDL: 30-50N per toe
  • Peak loads occur during heel strike and toe-off

Tendon excursion:

  • TA: 20-30mm excursion during gait cycle
  • EHL: 15-20mm excursion
  • EDL: 10-15mm excursion
  • Limited excursion compared to hand extensors

Gait mechanics:

  • Swing phase: Extensors contract to lift foot (dorsiflexion) and clear toes
  • Heel strike: Extensors eccentrically control plantarflexion
  • Midstance: Extensors stabilize ankle
  • Terminal stance: EHL maintains hallux extension for push-off

Classification Systems

Zone Classification (adapted from Kleinman and Dustmann)

The foot and ankle extensor tendons are divided into 8 zones from proximal to distal:

Zone 1: Muscle Belly

  • Location: Proximal and mid leg
  • Injury: Usually laceration or direct trauma
  • Characteristics: Excellent healing potential, muscle can reapproximate
  • Treatment: Debridement, loose approximation of muscle and fascia
  • Prognosis: Excellent

Zone 2: Musculotendinous Junction (MTJ)

  • Location: Distal leg
  • Injury: Closed rupture more common (especially TA)
  • Characteristics: Transition zone with variable strength
  • Treatment: Direct repair with nonabsorbable suture
  • Prognosis: Good but risk of re-rupture higher than pure tendinous zones

Zone 3: Distal Leg (Tendinous)

  • Location: Above ankle, before retinaculum
  • Injury: Laceration, closed rupture of TA
  • Characteristics: Full tendon, good healing
  • Treatment: End-to-end repair with core and epitenon sutures
  • Prognosis: Excellent with appropriate repair

Zone 4: Ankle and Retinaculum

  • Location: Beneath superior and inferior extensor retinaculum
  • Injury: Laceration or closed rupture
  • Characteristics: Critical zone - tendons confined, adhesion risk
  • Treatment: Careful repair, consider retinaculum release if tight
  • Prognosis: Good but adhesions to retinaculum can limit motion

Zone 2 Closed Rupture

Tibialis anterior most commonly ruptures at the MTJ (Zone 2) in middle-aged patients with degenerative changes. Often occurs during eccentric loading (deceleration or downhill walking). Presents with sudden pain and foot drop.

Zone 4 is the critical zone at the ankle.

Zone 5: Dorsum of Foot to MTP Joints

  • Location: From inferior retinaculum to MTP joint
  • Injury: Laceration common (glass, lawn mowers)
  • Characteristics: Multiple tendons often injured, EDB provides backup for EDL
  • Treatment: Repair TA and EHL, consider EDL repair
  • Prognosis: Good for major tendons

Zone 6: MTP Joint Level

  • Location: Over metatarsal heads and MTP joints
  • Injury: Laceration, sagittal band injury
  • Characteristics: Tendon becomes broader, forms expansion
  • Treatment: Repair expansion carefully
  • Prognosis: Good but stiffness possible

Zone 7: Proximal and Middle Phalanx

  • Location: Over proximal phalanx
  • Injury: Laceration or central slip disruption
  • Characteristics: Equivalent to central slip in hand
  • Treatment: Repair and splint in extension
  • Prognosis: Good with immobilization

Zone 8: Distal Phalanx (Terminal Tendon)

  • Location: DIP joint level
  • Injury: Mallet toe - avulsion or laceration
  • Characteristics: Similar to mallet finger
  • Treatment: Splinting in extension for 6 weeks if closed, repair if open
  • Prognosis: Excellent with conservative treatment for closed injuries

Zone 5 Lacerations

Zone 5 lacerations often involve multiple tendons. Essential to identify all injured structures. TA and EHL should always be repaired. EDL to lesser toes may not require repair if EDB is intact (test by passive toe extension).

Zone 8 mallet toe injuries often respond well to splinting alone.

Zone Characteristics and Management

ZoneLocationAdhesion RiskRepair Priority
1-2Muscle/MTJLowModerate
3Distal legLowHigh for TA/EHL
4Ankle/retinaculumHighCritical for all
5Dorsum footModerateHigh for TA/EHL
6-7MTP/phalanxModerateModerate
8DIP levelLowLow - splint often sufficient

The zone system helps predict adhesion risk and guide treatment priorities.

Injury Classification by Mechanism:

Laceration injuries:

  • Clean laceration: Sharp object, minimal contamination - best for primary repair
  • Contaminated laceration: Dirty wound, delayed presentation - washout and delayed repair
  • Extensive laceration: Multiple tendons, nerve/vessel injury - staged reconstruction

Closed ruptures:

  • Acute rupture: Sudden event, distinct timeline - primary repair possible
  • Chronic rupture: Gradual onset or delayed presentation - reconstruction often needed
  • Degenerative rupture: Underlying tendinopathy, older patients - poor tissue quality

Severity classification:

  • Partial laceration (less than 50%): May not require repair if tendon continuity maintained
  • Complete laceration: Always requires repair
  • Complete rupture with retraction: May require tendon advancement or graft

Clinical Assessment

History:

Acute laceration:

  • Mechanism: Sharp object, glass, machinery
  • Timing: Important for deciding primary vs delayed repair
  • Contamination: Assess tetanus status and infection risk
  • Hand dominance and occupation (for functional demands)

Closed rupture:

  • Onset: Sudden pop vs gradual weakness
  • Precipitating activity: Eccentric loading, sports activity
  • Previous symptoms: Antecedent tendinopathy or pain
  • Medical history: Diabetes, inflammatory arthropathy, fluoroquinolone use

Physical examination:

Inspection:

  • Wound: Location, extent, contamination if acute laceration
  • Resting position: Foot drop (plantarflexed) if TA ruptured
  • Swelling: Hematoma or edema at injury site
  • Gait: Foot drop, slapping gait, toe drag during swing phase
  • Contour: Loss of anterior ankle contour with TA rupture

Palpation:

  • Gap: Palpable defect in tendon continuity
  • Tenderness: Along tendon course
  • Crepitus: Suggests tendinopathy or partial tear

Active movement testing:

Tibialis Anterior:

  • Active dorsiflexion: Ask patient to dorsiflex foot against gravity
  • Grading: 0 = no movement, 1 = flicker, 2 = movement with gravity eliminated, 3 = against gravity, 4 = against resistance, 5 = normal
  • Inversion strength: TA also inverts foot
  • Inability to heel walk: Pathognomonic for TA dysfunction

Extensor Hallucis Longus:

  • Hallux IP extension: Ask patient to extend great toe IP joint against resistance
  • MTP extension: Test separately (EHB can compensate at MTP)
  • Loss indicates EHL injury

Extensor Digitorum Longus:

  • Lesser toe extension: Test each toe individually
  • MTP vs IP extension: EDB extends MTP, so preserved MTP with weak IP suggests EDL injury
  • Intact EDB can mask EDL injury

Special tests:

Thompson test equivalent for TA:

  • With patient prone or sitting, squeeze anterior compartment
  • Normal: Should see dorsiflexion
  • Positive (no dorsiflexion): Suggests TA rupture

Tenodesis effect:

  • Passive plantarflexion should make toes extend (if extensors intact)
  • Passive dorsiflexion should make toes flex
  • Loss of effect suggests complete tendon disruption

Resisted testing:

  • Manual resistance to dorsiflexion and toe extension
  • Grade strength using MRC scale
  • Compare to contralateral side

Neurovascular Assessment Mandatory

Deep peroneal nerve runs with anterior tibial artery beneath inferior extensor retinaculum. Assess: (1) Sensation in first web space, (2) EDB function (extends toes at MTP), (3) Dorsalis pedis pulse. Injuries to nerve/artery require urgent vascular surgery consultation.

Examination findings by tendon:

TendonActive TestLoss of FunctionCompensation
TADorsiflexion, inversionFoot drop, slapping gaitEHL weak dorsiflexion (inadequate)
EHLHallux IP extensionWeak push-off, claw halluxEHB at MTP only
EDLLesser toe extensionWeak toe extensionEDB at MTP preserved
Peroneus TertiusDorsiflexion, eversionMinimal functional lossTA dorsiflexes, peroneals evert

Investigations

Clinical diagnosis is usually sufficient for acute lacerations with obvious tendon injury. Imaging helps in closed ruptures, delayed presentations, and surgical planning.

Radiographs:

Indications:

  • All trauma to exclude fracture
  • Chronic injuries to assess for bony avulsion
  • Mallet toe to identify terminal phalanx avulsion

Views:

  • AP, lateral, and oblique foot
  • AP and lateral ankle if injury near ankle

Findings:

  • Usually normal in tendon injury
  • May show avulsion fragment (insertion injuries)
  • Soft tissue swelling or gas if infected

Ultrasound:

Advantages:

  • Dynamic assessment
  • Can visualize gap and retraction
  • Assess tendon continuity and quality
  • Real-time comparison to contralateral side
  • Inexpensive and readily available

Findings:

  • Complete rupture: Gap with retracted tendon ends
  • Partial tear: Hypoechoic defect, thickening
  • Tendinopathy: Thickened, hypoechoic, loss of fibrillar pattern

Limitations:

  • Operator dependent
  • Limited for deep structures
  • Difficult with extensive swelling

MRI:

Indications:

  • Closed rupture with uncertain diagnosis
  • Chronic injuries for surgical planning
  • Multiple tendon involvement suspected
  • Associated soft tissue or bone injury

Sequences:

  • T1: Anatomical detail
  • T2/STIR: Edema, fluid in tendon sheath
  • Proton density: Tendon morphology

Findings:

  • Acute rupture: High signal, gap, retraction, surrounding edema
  • Chronic rupture: Tendon ends, scar tissue, muscle atrophy
  • Partial tear: Increased signal within tendon
  • Tendinopathy: Thickening, intermediate signal

MRI advantages:

  • Gold standard for soft tissue
  • Multiplanar imaging
  • Can assess muscle quality (atrophy, fatty infiltration)

When to Image?

Acute open lacerations - imaging not needed, go to OR for exploration and repair. Closed rupture or delayed presentation - MRI or ultrasound useful to: (1) Confirm diagnosis, (2) Assess gap size, (3) Evaluate tendon quality, (4) Identify retraction, (5) Plan reconstruction.

Advanced imaging:

CT scan:

  • Limited role
  • Useful for bony avulsion injuries
  • Assesses fracture displacement

Diagnostic local anesthesia:

  • Not typically used
  • May differentiate tendon vs neurological cause of weakness

Management Algorithm

📊 Management Algorithm
extensor tendon injuries foot management algorithm
Click to expand
Management algorithm for extensor tendon injuries footCredit: OrthoVellum
Initial Assessment and Decision

Within 2 weeks of injury

  1. History and examination - establish diagnosis
  2. Wound assessment - clean vs contaminated
  3. Neurovascular status - deep peroneal nerve and dorsalis pedis
  4. Radiographs - exclude fracture
  5. Decision:
    • Clean laceration + intact skin = Primary repair in OR
    • Contaminated wound = Washout, delayed primary repair
    • Closed rupture = Early surgical repair (TA/EHL), conservative vs surgery (EDL)
Delayed Primary Repair

If missed acutely or delayed presentation

  • Tendon ends still identifiable
  • May require tendon advancement or recession
  • Less predictable than primary repair
  • Consider MRI for surgical planning
Late Reconstruction

Chronic injuries

  • Tendon ends retracted, scarred
  • Direct repair usually not possible
  • Options: Tendon graft, tendon transfer, arthrodesis (for mallet toe)
  • Reconstruction vs acceptance of deficit depends on functional demands

Conservative Management:

Indications:

  • Partial lacerations less than 50% with continuity preserved
  • EDL injuries with intact EDB (lesser toes)
  • Peroneus tertius injuries
  • Mallet toe (closed injuries)
  • Medical comorbidities precluding surgery
  • Low functional demands

Principles:

  • Immobilization in boot or cast with ankle in neutral to slight dorsiflexion
  • Duration: 3-4 weeks for partial injuries
  • Serial examination to ensure no progression
  • Physiotherapy for strengthening after immobilization

Outcomes:

  • Partial tears: 70-80% good function without surgery
  • EDL to lesser toes: Minimal functional deficit if EDB intact
  • Mallet toe: 85-90% good outcomes with splinting

Surgical Management:

Indications for surgery:

  • Complete TA laceration or rupture - absolute indication
  • Complete EHL laceration or rupture - strong relative indication
  • EDL lacerations - repair when feasible
  • Multiple tendon injuries
  • Associated nerve or vascular injury
  • Failed conservative management

Goals of surgery:

  1. Restore tendon continuity
  2. Restore length-tension relationship
  3. Achieve stable repair allowing early mobilization
  4. Minimize adhesions
  5. Restore function

Timing decisions:

Surgical Timing - Pros and Cons

TimingAdvantagesDisadvantagesBest For
Primary (less than 48h)Direct repair, no retraction, best outcomesMay need to delay if contaminatedClean lacerations
Delayed Primary (2-14 days)Allows wound optimization, still repairableSome retraction, more difficult repairContaminated wounds after washout
Late (more than 2 weeks)Allows full assessment, planned reconstructionOften needs graft or transfer, worse outcomesMissed injuries, chronic ruptures

Surgical Technique

Pre-operative Planning:

  1. Mark tendon course on skin with ankle in neutral
  2. Identify injury zone from history and examination
  3. Plan incision - curvilinear or longitudinal, avoid skin creases
  4. Consent for possible nerve injury, adhesions, weakness, re-rupture

Setup:

  • Supine positioning
  • Thigh tourniquet (thigh level for ankle/foot injuries)
  • Bump under ipsilateral hip if needed for positioning
  • Image intensifier available but rarely needed
  • Loupe magnification helpful for distal repairs

Incision:

  • Zone 3-4 (leg/ankle): Longitudinal incision centered over tendon
  • Zone 5 (dorsum foot): Curvilinear to avoid crossing joints at right angles
  • Zone 6-8 (toes): Longitudinal over dorsum of toe
  • Extend incision as needed to retrieve retracted tendon ends

Dissection:

  1. Incise skin and subcutaneous tissue
  2. Identify and protect superficial nerves (superficial peroneal branches on dorsum)
  3. Identify deep peroneal nerve - runs with anterior tibial artery beneath retinaculum
  4. Locate tendon ends - use passive ankle/toe motion to aid identification
  5. Retrieve retracted ends - may need proximal and distal extensions
  6. Debride minimal amount of nonviable tendon (preserves length)
  7. Assess tendon quality - healthy tendon should be white and glistening

Zone 4 specific considerations:

  • Retinaculum may need to be partially released to access tendons
  • Preserve retinaculum for later repair (prevents bowstringing)
  • Identify anterior tibial artery and deep peroneal nerve
  • Multiple tendons may be injured - identify all structures

Finding Retracted Tendons

Proximal retraction is common, especially with TA. Techniques to find tendon: (1) Passive motion - plantarflex ankle and watch for tendon movement, (2) "Milking" muscle belly proximally, (3) Extended incision if needed, (4) Contralateral side for reference anatomy.

This completes the exposure phase of the operation.

Suture Material:

  • Core suture: Nonabsorbable braided (e.g., Ethibond, FiberWire) number 2 or number 0
  • Epitenon suture: Absorbable or nonabsorbable 4-0 or 5-0
  • Strong suture essential for early mobilization protocols

Core Suture Techniques:

Modified Kessler (4-strand):

  • Traditional technique for larger tendons
  • Grasping suture through tendon substance
  • Exit and re-entry points 1cm from cut end
  • Good strength (30-40N breaking strength)
  • Technique: Pass suture into tendon 1cm from end, out through cut surface, across to other tendon, in through cut surface, out 1cm from end, tie

Krackow (6-strand or more):

  • Strongest technique (50-60N breaking strength)
  • Multiple locking loops along tendon
  • Best for larger tendons (TA, EHL)
  • Preferred for repairs requiring early mobilization
  • Technique: Weave suture in multiple locking loops, typically 3 passes per side, creates 6-strand construct

Simple interrupted:

  • Weaker technique
  • Only for low-demand situations or supplementary repair
  • Not recommended as primary repair

Epitenon/Running Suture:

  • Purpose: Smoothens repair site, adds 10-20% strength, prevents gapping
  • Technique: Continuous circumferential simple or running locked suture
  • Material: 4-0 or 5-0 absorbable or nonabsorbable
  • Essential to complete core suture for optimal strength

Repair Configuration:

End-to-end repair (fresh injuries):

  • Preparation: Minimal debridement, preserve length
  • Technique: Core suture (Krackow or Kessler) plus epitenon
  • Tensioning: Ankle in neutral dorsiflexion, appropriate tension
  • Assessment: No gap at repair site, smooth tendon contour

Tendon advancement (delayed, 2-6 weeks):

  • If gap less than 3cm, may advance tendon
  • TA: Can advance from muscle belly or recede insertion
  • EHL/EDL: Limited advancement without over-lengthening
  • Technique: Core suture with slight over-tensioning to account for stretch

Tendon grafting (chronic, more than 6 weeks):

  • Indications: Gap more than 3cm, poor quality tendon ends
  • Graft options: Hamstring autograft, tibialis posterior, FHL, allograft
  • Technique: Weave graft through proximal and distal stumps, multiple sutures

Zone-Specific Techniques:

Zone 4 (retinaculum):

  • Repair tendon as above
  • Retinaculum repair: Loosely reapproximate to prevent bowstringing but avoid constriction
  • Consider leaving one limb of retinaculum open if tight

Zone 5 (multiple tendons):

  • Repair in order: TA first, then EHL, then EDL
  • Keep tendons in anatomical position
  • Avoid intertendinous adhesions by preserving paratenon where possible

Zone 8 (mallet toe):

  • If open injury: Direct repair of terminal tendon
  • If bony avulsion: K-wire fixation in extension + tendon repair
  • Extension splinting for 6 weeks

Tension and Tenodesis

Correct tensioning is critical. Ankle in neutral dorsiflexion for repair. Too tight = overlengthening = weak dorsiflexion. Too loose = gapping = elongation = weak dorsiflexion. Use contralateral side for reference. Test passive motion before closing to ensure smooth gliding.

The repair technique must balance strength and the need for smooth gliding during rehabilitation.

Wound Closure:

  1. Hemostasis: Meticulous, use bipolar cautery
  2. Retinaculum: Reapproximate loosely if released (absorbable 3-0 suture)
  3. Subcutaneous: Close in layers if needed (3-0 or 4-0 absorbable)
  4. Skin: Monofilament nonabsorbable (4-0 nylon) or absorbable subcuticular
  5. Dressing: Nonadherent, padded, and protective

Immobilization:

  • Below-knee back-slab or boot
  • Ankle in neutral to slight dorsiflexion (5-10 degrees)
  • Toes free for observation and gentle motion
  • Non-weight bearing initially

Alternative Techniques:

Tendon Transfer (chronic TA rupture):

Extensor hallucis longus to TA transfer:

  • Indication: Chronic TA rupture with irreparable tendon
  • Principle: Sacrifice EHL function to restore dorsiflexion
  • Technique:
    1. Harvest EHL at musculotendinous junction
    2. Pass through interosseous membrane to anterior compartment
    3. Weave into TA stump or drill holes in cuneiform
    4. Tension with ankle in neutral dorsiflexion
  • Outcomes: Restores dorsiflexion but loses hallux IP extension

FHL to FDL transfer + EHL to TA:

  • More complex reconstruction
  • Transfers FHL to FDL first (restores hallux flexion)
  • Then can use EHL for TA reconstruction

Tendon Lengthening:

  • Rarely needed in foot extensors
  • May be used for chronic contractures after failed repair

Arthrodesis:

  • Hallux IP fusion - for irreparable EHL in low-demand patient
  • Mallet toe: DIP fusion alternative to repair in chronic cases

Synthetic Augmentation:

  • Not routinely recommended
  • May use in revision surgery or poor-quality tendon
  • Options: Collagen scaffolds, synthetic mesh

Tendon Transfer Considerations

EHL to TA transfer is last resort for irreparable chronic TA rupture. Patient must accept loss of hallux IP extension. This causes claw hallux and weak push-off. Better option if possible: tendon graft reconstruction to preserve EHL function. Only transfer if graft not feasible.

Closure and immobilization complete the surgical procedure.

Complications

Early Complications (less than 6 weeks):

Re-rupture:

  • Incidence: 5-10% with modern repair techniques
  • Risk factors: Inadequate repair strength, premature weight bearing, poor compliance
  • Presentation: Sudden pain, loss of dorsiflexion, palpable gap
  • Management: Revision repair if early (within 2 weeks), reconstruction if late
  • Prevention: Adequate suture technique (Krackow preferred), protected mobilization protocol

Infection:

  • Incidence: 2-5% (higher with contaminated wounds)
  • Risk factors: Contamination, delayed treatment, diabetes, immunosuppression
  • Presentation: Wound erythema, drainage, fever, pain
  • Management: Wound culture, antibiotics, washout if deep infection
  • Prevention: Prophylactic antibiotics, meticulous wound care, early coverage

Wound dehiscence:

  • Incidence: 5-10%
  • Risk factors: Poor skin quality, tension, infection, poor vascularity
  • Management: Local wound care, delayed closure, skin graft if needed
  • Prevention: Tension-free closure, avoid incisions over bony prominences

Nerve injury:

  • Deep peroneal nerve: Numbness in first web space, weakness of EDB
  • Superficial peroneal nerve: Numbness on dorsum of foot
  • Incidence: 2-5% iatrogenic injury
  • Management: Observation (most neuropraxia recover), neurolysis if needed
  • Prevention: Careful identification and protection during dissection

Vascular injury:

  • Anterior tibial artery: Rare but catastrophic
  • Presentation: Loss of dorsalis pedis pulse, foot ischemia
  • Management: Immediate vascular surgery consultation
  • Prevention: Identify artery during dissection, careful retraction

Compartment syndrome:

  • Incidence: Rare (less than 1%)
  • Risk factors: Extensive trauma, prolonged tourniquet, post-op hematoma
  • Presentation: Severe pain, tense anterior compartment, pain with passive stretch
  • Management: Urgent fasciotomy
  • Prevention: Monitor high-risk patients, avoid overly tight dressings

Re-rupture Prevention

Re-rupture is devastating complication. Prevention requires: (1) Strong repair - Krackow or Kessler core suture plus epitenon, (2) Protected mobilization - Non-weight bearing for 4 weeks, (3) Patient education - compliance with restrictions critical, (4) Graduated return - progressive loading over 8-12 weeks.

Late Complications (more than 6 weeks):

Adhesions and Stiffness:

  • Incidence: 20-30% have some limitation
  • Risk factors: Zone 4 injuries, prolonged immobilization, extensive dissection
  • Presentation: Limited ankle dorsiflexion, painful motion
  • Management: Aggressive physiotherapy, tenolysis if severe and disabling
  • Prevention: Gentle tissue handling, early protected mobilization, retinaculum release

Weakness:

  • Incidence: 10-20% have some residual weakness
  • Causes: Elongation at repair site, muscle atrophy, adhesions
  • Presentation: Reduced dorsiflexion strength compared to contralateral
  • Management: Strengthening exercises, orthotic support, acceptance
  • Prevention: Adequate tensioning at repair, early mobilization, strengthening

Chronic pain:

  • Incidence: 5-10%
  • Causes: Neuroma, adhesions, tendinopathy, complex regional pain
  • Management: Physiotherapy, nerve blocks, neuroma excision if identified
  • Prevention: Protect sensory nerves, gentle tissue handling

Tendon elongation:

  • Incidence: 10-15%
  • Mechanism: Gap formation, suture pull-through, inadequate repair
  • Presentation: Weakness despite intact tendon on imaging
  • Management: Plication or reconstruction if severe
  • Prevention: Strong repair technique, appropriate tensioning

Mallet toe (after Zone 8 injury):

  • Incidence: 5-10% develop fixed deformity
  • Presentation: DIP flexion deformity, nail deformity
  • Management: Splinting, arthrodesis if symptomatic and rigid
  • Prevention: Adequate extension splinting duration (6 weeks minimum)

Functional Limitations:

TA rupture/repair:

  • Difficulty with stairs (especially descending)
  • Tripping risk due to subtle weakness
  • Slapping gait may persist

EHL rupture/repair:

  • Claw hallux
  • Reduced push-off power
  • Shoe wear issues

Claw toe deformity:

  • Occurs with EDL over-pull or FDL tightness
  • Causes metatarsalgia, shoe fitting issues
  • May require flexor tenotomy or transfer

Postoperative Care and Rehabilitation

Immediate Post-operative (0-2 weeks):

Immobilization:

  • Below-knee back-slab or boot
  • Ankle in neutral dorsiflexion
  • Non-weight bearing with crutches
  • Elevate limb above heart level to reduce swelling

Wound care:

  • Keep dressing clean and dry
  • First dressing change at 48-72 hours
  • Inspect wound for signs of infection
  • Suture removal at 10-14 days

Pain management:

  • Simple analgesia (paracetamol, NSAIDs)
  • Avoid excessive opioid use
  • Cryotherapy (ice packs) for swelling

Phase 1: Protected Immobilization (2-4 weeks):

Mobilization:

  • Convert to walking boot (removable)
  • Continue non-weight bearing
  • Begin gentle passive dorsiflexion with boot removed (therapist-supervised)
  • No active dorsiflexion yet (protect repair)

Goals:

  • Maintain ankle ROM in plantarflexion
  • Prevent adhesions without stressing repair
  • Reduce swelling and inflammation

Precautions:

  • Avoid active dorsiflexion
  • Avoid resisted exercises
  • No weight bearing

Phase 2: Early Active Motion (4-6 weeks):

Weight bearing:

  • Begin partial weight bearing in boot at 4 weeks
  • Progress to full weight bearing by 6 weeks
  • Wean from boot as tolerated (typically 6 weeks)

Active exercises:

  • Gentle active dorsiflexion against gravity (no resistance)
  • Ankle circles (plantarflexion, dorsiflexion, inversion, eversion)
  • Toe flexion and extension exercises
  • Intrinsic muscle strengthening

Passive stretching:

  • Passive dorsiflexion stretch (gastrocnemius and soleus)
  • Avoid aggressive stretching

Goals:

  • Restore active ROM
  • Prevent Achilles tendon contracture
  • Begin muscle re-education

Phase 3: Strengthening (6-12 weeks):

Resisted exercises:

  • Resistance band dorsiflexion - start with light resistance
  • Progressive strengthening of TA, EHL, EDL
  • Proprioception exercises (balance board)
  • Heel walking progression

Functional exercises:

  • Gait re-education
  • Stair training
  • Single-leg balance
  • Sport-specific drills (if applicable)

Goals:

  • Restore strength to 80% of contralateral
  • Normal gait pattern
  • Return to daily activities

Phase 4: Return to Sport/Full Activity (3-6 months):

Criteria for return:

  • Full pain-free ROM
  • Strength at least 80-90% of contralateral
  • Normal gait without limp
  • Sport-specific testing passed

Advanced exercises:

  • Plyometrics
  • Agility drills
  • Sport-specific training
  • Running progression

Functional milestones:

Partial Weight Bearing

Begin partial weight bearing in boot. Start gentle active dorsiflexion. No resistance.

Full Weight Bearing

Full weight bearing without boot. Active ROM exercises. Light resistance begins.

Strengthening Phase

Progressive resistance exercises. Proprioception training. Return to work (sedentary).

Return to Activities

Return to unrestricted activities of daily living. Light sports and recreation.

Return to Sport

Full return to competitive sports if criteria met. Continue strengthening program.

Protocol variations:

Tibialis anterior repairs:

  • Longer protection (may extend immobilization to 6 weeks)
  • Critical for gait function
  • More conservative return to sport

EHL/EDL repairs:

  • Can progress faster (less critical loads)
  • Earlier active motion possible
  • Functional demands guide progression

Mallet toe:

  • Extension splinting for 6 weeks continuous
  • Then night splinting for additional 4-6 weeks
  • Buddy taping during mobilization

Rehabilitation Philosophy

Balance is key in extensor tendon rehab: (1) Too aggressive = re-rupture, elongation, failure, (2) Too conservative = adhesions, stiffness, weakness. Modern approach favors protected early motion - passive/active-assisted ROM early, graduated resistance later. Goal: optimal healing AND function.

Expected outcomes:

Excellent (80-85% of cases):

  • Return to baseline function
  • Full or near-full strength
  • Minimal pain or stiffness

Good (10-15%):

  • Mild weakness or stiffness
  • Return to most activities
  • Occasional discomfort

Fair/Poor (5-10%):

  • Persistent weakness
  • Chronic pain or stiffness
  • Activity limitations
  • May need revision or salvage

Rehabilitation is complete when functional goals are achieved and patient satisfied with outcome.

Evidence Base

Primary Repair Outcomes for Extensor Tendon Lacerations

III
Scaduto et al. • Foot Ankle Int (2000)
Key Findings:
  • 92% good to excellent outcomes with primary repair
  • Zone 4 injuries had 30% stiffness rate vs 10% in other zones
  • Core plus epitenon suture superior to simple repairs
  • Complications: 8% re-rupture, 12% adhesions, 5% infection
Clinical Implication: This evidence guides current practice.

Closed Tibialis Anterior Rupture: Surgical vs Conservative

IV
Markarian et al. • Foot Ankle Int (1998)
Key Findings:
  • Surgical repair restored 85% strength vs 60% conservative
  • 100% return to activity in surgical group vs 40% in conservative
  • Average gap at rupture site was 4.2 cm
  • MTJ ruptures most common site (75%)
Clinical Implication: This evidence guides current practice.

Early Active Mobilization vs Immobilization After Extensor Tendon Repair

III
Browne et al. • J Foot Ankle Surg (2011)
Key Findings:
  • Early motion improved final ROM by average 10 degrees
  • No increase in re-rupture rate with early motion protocol
  • Adhesion rate lower in early motion group (15% vs 28%)
  • Patient satisfaction higher with early motion
Clinical Implication: This evidence guides current practice.

Krackow vs Kessler Suture Technique for Extensor Tendon Repair

IV
Thompson et al. • Foot Ankle Spec (2015)
Key Findings:
  • Krackow technique 32% stronger than Kessler
  • Both techniques exceeded minimum strength for early mobilization
  • Epitenon suture adds 15-20% strength
  • Gap formation less with Krackow technique under cyclic loading
Clinical Implication: This evidence guides current practice.

Zone-Specific Outcomes in Foot Extensor Tendon Injuries

III
Anderson et al. • J Orthop Trauma (2017)
Key Findings:
  • Zone 4 worst outcomes due to adhesions under retinaculum
  • Multiple tendon injuries increased complication risk 3-fold
  • EDL repairs to lesser toes had minimal functional improvement
  • TA and EHL repairs critical for functional outcomes
Clinical Implication: This evidence guides current practice.

Clinical Practice Guidelines:

American Academy of Orthopaedic Surgeons (AAOS):

  • No specific guideline for extensor tendon injuries (included in general soft tissue trauma)
  • Recommend early diagnosis and repair for complete tendon disruptions
  • Support early controlled mobilization protocols

Consensus recommendations:

  1. Primary repair within 2 weeks for complete TA and EHL disruptions
  2. Core suture technique (Krackow or Kessler) plus epitenon
  3. Protected early mobilization starting at 2-4 weeks
  4. EDL repairs recommended when feasible but not critical for lesser toes
  5. MRI or ultrasound for diagnosis of closed ruptures

Key controversies:

EDL repair necessity:

  • Some authors advocate repair of all EDL tendons
  • Others suggest selective repair (only if EDB absent or patient high-demand)
  • Consensus: Repair when feasible but low priority if EDB intact

Optimal rehabilitation protocol:

  • Traditional: 6 weeks strict immobilization
  • Modern: Early protected motion starting at 2-4 weeks
  • Consensus shifting toward early motion based on biomechanical and clinical data

Timing of delayed repairs:

  • Some advocate up to 6 months for TA reconstruction
  • Others suggest diminishing returns after 6 weeks
  • Consensus: Best outcomes with repair within 6 weeks; reconstruction possible up to 6 months in motivated patients

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Acute TA Laceration

EXAMINER

"A 32-year-old construction worker presents to ED 4 hours after stepping through a glass window. He has a 6cm laceration across the anterior ankle. On examination, he cannot dorsiflex the ankle against gravity and there is a palpable gap. Neurovascular status is intact. What is your assessment and management?"

EXCEPTIONAL ANSWER
This patient has a complete tibialis anterior tendon laceration requiring urgent surgical repair. My systematic approach would be: First, confirm the diagnosis with examination - loss of active dorsiflexion and palpable gap are diagnostic. Document neurovascular status including deep peroneal nerve function (first web space sensation, EDB function) and dorsalis pedis pulse. Take radiographs to exclude fracture. This is a clean acute laceration presenting within the golden period for primary repair. I would consent for tendon exploration and repair in the operating room. Under tourniquet control, I would extend the incision to visualize both tendon ends, identify and protect the deep peroneal nerve and anterior tibial artery, perform primary repair using a Krackow core suture with nonabsorbable braided suture plus running epitenon. Immobilize in a below-knee back-slab in neutral dorsiflexion. Post-operatively, non-weight bearing for 4 weeks, then protected mobilization with gradual return to activity over 8-12 weeks. I would counsel about risks including infection, nerve injury, re-rupture (5-10%), adhesions, and potential weakness.
KEY POINTS TO SCORE
Recognize TA laceration as surgical emergency requiring primary repair
Document complete neurovascular examination (deep peroneal nerve critical)
Primary repair technique: Krackow or Kessler core plus epitenon
Post-op protocol: 4 weeks non-weight bearing, then progressive mobilization
Counsel realistic expectations - 85-90% return to full function
COMMON TRAPS
✗Missing associated nerve or vascular injury - always document
✗Inadequate exploration - must visualize both tendon ends completely
✗Poor suture technique - simple sutures insufficient, need core plus epitenon
✗Too aggressive rehab - risk re-rupture if loaded before 4 weeks
✗Not explaining functional importance - TA critical for gait, patient must comply
LIKELY FOLLOW-UPS
"What if the deep peroneal nerve was also injured? - Would repair nerve primarily with 8-0 or 9-0 nylon under microscope. Document pre-op deficit. Counsel that nerve recovery takes 3-6 months and may be incomplete."
"How would your management differ if he presented 3 weeks later? - Delayed primary repair still possible within 2-6 weeks but more difficult. May need tendon advancement. MRI useful to assess gap and retraction. Worse prognosis than acute primary repair."
"Describe your post-operative rehabilitation protocol. - Phase 1 (0-4 weeks): Non-weight bearing in boot, gentle passive ROM. Phase 2 (4-6 weeks): Progress to full weight bearing, active ROM. Phase 3 (6-12 weeks): Strengthening with resistance band, proprioception. Phase 4 (3-6 months): Return to work and sport if criteria met."
"What factors affect your suture technique choice? - Tendon size (TA is large, use stronger technique), repair strength needed (early mobilization requires minimum 100N), surgeon familiarity. I prefer Krackow for TA because it provides 50-60N breaking strength and locks securely."
VIVA SCENARIOChallenging

Scenario 2: Closed TA Rupture - Conservative vs Surgical

EXAMINER

"A 58-year-old recreational runner presents with 3-week history of anterior ankle pain and weakness after feeling a 'pop' while running downhill. Examination reveals weak dorsiflexion (3/5 power) and a palpable gap 5cm above the ankle. MRI confirms complete TA rupture at the musculotendinous junction with 4cm gap. He wants to return to running. How would you manage this patient?"

EXCEPTIONAL ANSWER
This is a closed tibialis anterior rupture at the MTJ, presenting in the delayed primary repair window. Given his functional goals (return to running) and the large gap (4cm), I would recommend surgical reconstruction. I would counsel that conservative management may leave him with significant weakness (typically 60% strength of contralateral) which would impair running and increase tripping risk. Surgical repair offers better restoration of strength (85% of contralateral) and higher return to sport rates. The 3-week delay means delayed primary repair - tendon ends still identifiable but may be retracted and require advancement. I would perform surgical exploration through an anterior incision, retrieve both tendon ends (may need proximal extension into muscle belly), debride minimally, perform end-to-end repair with Krackow core suture if possible (tendon advancement if needed to close gap), add epitenon, and immobilize in neutral. Post-operatively, protected mobilization protocol similar to acute repair. Return to running typically 4-6 months. Risks include re-rupture (higher than acute repair, approximately 10-15%), adhesions, persistent weakness, and possible need for tendon graft if gap cannot be closed primarily.
KEY POINTS TO SCORE
Closed TA rupture requires surgical repair for active patients with high functional demands
3-week delay still within delayed primary repair window (2-6 weeks)
MTJ rupture common site - degenerative changes in middle-aged runners
Surgical outcomes superior to conservative for strength and return to sport
4cm gap challenging but may achieve primary repair with advancement
COMMON TRAPS
✗Offering conservative management to active patient - functional outcomes poor
✗Underestimating difficulty of delayed repair - requires skill to mobilize retracted tendon
✗Not getting MRI - essential for surgical planning (gap size, retraction, muscle quality)
✗Promising too much - some residual weakness expected even with surgery
✗Not discussing tendon graft possibility - if gap cannot be closed, need backup plan
LIKELY FOLLOW-UPS
"What if the gap was 8cm and you could not achieve primary repair? - Would perform tendon graft reconstruction using hamstring autograft or allograft. Weave graft through proximal and distal stumps, secure with multiple sutures. Longer immobilization (6 weeks). Outcomes less predictable than primary repair but better than acceptance of deficit."
"Would you ever recommend conservative management for this injury? - Yes, for elderly low-demand patients with significant medical comorbidities precluding surgery, or patients who refuse surgery. Would immobilize in equinus boot for 6 weeks, then progressive strengthening. Counsel to expect permanent weakness and altered gait. AFO may be needed."
"What is the evidence comparing surgical vs conservative management? - Level IV evidence (case series) suggests surgical repair achieves 80-90% strength vs 50-60% conservative. Return to sport 70-80% surgical vs 30-40% conservative. No randomized trials but retrospective comparisons favor surgery for active patients."
"How does TA rupture differ from Achilles tendon rupture? - TA ruptures are less common, typically older patients, often at MTJ rather than mid-substance. Functional deficit less obvious (foot drop vs push-off weakness). Surgical indications similar - active patients benefit from repair. Healing potential similar."
VIVA SCENARIOChallenging

Scenario 3: Zone 5 Multiple Tendon Laceration

EXAMINER

"A 25-year-old presents with a lawn mower injury causing extensive laceration across the dorsum of the foot at the mid-foot level. Wound is contaminated with grass and dirt. On examination, there is a 10cm laceration with obvious injury to multiple structures. She cannot dorsiflex the ankle or extend any toes. Pulses are intact but sensation in the first web space is diminished. How do you proceed?"

EXCEPTIONAL ANSWER
This is a complex Zone 5 injury with multiple tendon lacerations, possible deep peroneal nerve injury, and contamination. This requires urgent surgical exploration and washout. First, in the emergency department, I would assess and document neurovascular status completely - dorsalis pedis pulse, deep peroneal nerve function (first web space numbness suggests nerve injury, weak EDB function). Photograph and dress the wound. Give IV antibiotics (cephalosporin) and tetanus prophylaxis. Update tetanus if needed. Take radiographs to exclude fracture or foreign body. Then proceed urgently to operating room for irrigation, debridement, exploration, and delayed primary repair. Under tourniquet, I would extend the wound to allow full visualization. I would identify all injured structures - likely TA, EHL, EDL tendons, and deep peroneal nerve based on anatomy. Copious irrigation (6-9L saline). Debride devitalized tissue carefully. Tag all tendon ends with sutures to maintain identification. Primary repair of TA and EHL is essential. EDL repair when feasible. If deep peroneal nerve divided, would repair primarily with 8-0 or 9-0 nylon. Given contamination, I would consider leaving skin open or loosely approximated for delayed closure at 48-72 hours. Immobilize in neutral dorsiflexion. Post-op IV antibiotics for 24-48 hours, then oral. Monitor for infection. Rehabilitation delayed until wound healed.
KEY POINTS TO SCORE
Complex contaminated injury requires urgent surgical washout
Multiple tendon injuries common in Zone 5 - must identify all structures
Deep peroneal nerve injury suggested by first web space numbness
Tag all tendon ends during initial surgery if delayed repair planned
Contaminated wounds may require delayed closure or second-look surgery
COMMON TRAPS
✗Attempting immediate definitive repair in contaminated wound - risk infection
✗Missing nerve injury - must test and document sensory and motor function
✗Inadequate irrigation - contaminated wounds need copious washout (6-9L minimum)
✗Not tagging tendons - retraction after initial surgery makes identification difficult
✗Single-stage management - contaminated wounds often need staged approach
LIKELY FOLLOW-UPS
"Would you repair all tendons or stage the reconstruction? - In this contaminated case, I would tag all tendon ends but consider staging definitive repair. TA and EHL are priority. Could perform delayed primary repair at 48-72 hours after confirming no infection. EDL could be repaired at same setting or later if needed."
"How do you manage the deep peroneal nerve injury? - Primary repair with 8-0 or 9-0 nylon under loupe magnification or microscope. Document pre-operative deficit. Counsel patient that recovery takes 3-6 months (nerve regenerates 1mm/day). May have permanent sensory loss in first web space and EDB weakness."
"What if you cannot achieve primary closure due to tissue loss? - Options include: (1) Negative pressure wound therapy until granulation, then skin graft, (2) Local flap coverage, (3) Delayed primary closure after swelling resolves. Key is protecting repaired tendons and achieving soft tissue coverage within 7-10 days."
"When would you start rehabilitation after this complex injury? - Would delay aggressive rehabilitation until wound healed (typically 2-3 weeks). Then gentle passive ROM. Active ROM at 4-6 weeks. Strengthening at 8-12 weeks. Overall timeline extended by 2-3 weeks compared to clean acute laceration due to contamination and complexity."

EXTENSOR TENDON INJURIES - FOOT

High-Yield Exam Summary

Four Main Tendons (TALE)

  • •Tibialis Anterior - strongest dorsiflexor, ALWAYS repair
  • •Extensor Hallucis Longus - hallux IP extension, repair recommended
  • •Extensor Digitorum Longus - lesser toe extension, repair when feasible
  • •Peroneus Tertius - weakest, accessory dorsiflexor/everter, repair optional

Zone Classification (8 Zones)

  • •Zone 1-2: Muscle belly and MTJ - good healing, TA rupture common at MTJ
  • •Zone 3: Distal leg - primary repair with core plus epitenon
  • •Zone 4: Ankle/retinaculum - CRITICAL zone, adhesion risk high
  • •Zone 5: Dorsum foot to MTP - multiple tendon injuries common
  • •Zone 6-8: MTP and phalanges - mallet toe at Zone 8

TA Rupture Clinical Features (DROP FOOT)

  • •Dorsiflexion weak or absent (0-2/5 power)
  • •Foot drop and slapping gait
  • •Palpable gap anterior ankle
  • •Cannot heel walk or lift foot against gravity
  • •MTJ rupture most common (Zone 2)

Surgical Repair Technique

  • •Core suture: Krackow (strongest, 50-60N) or Kessler (30-40N)
  • •Add running epitenon (adds 15-20% strength)
  • •Ankle in neutral dorsiflexion for repair
  • •Primary repair ideal within 2 weeks
  • •Delayed primary 2-6 weeks, reconstruction more than 6 weeks

Rehabilitation Protocol

  • •0-4 weeks: Non-weight bearing, gentle passive ROM
  • •4-6 weeks: Progress to full weight bearing, active ROM
  • •6-12 weeks: Strengthening with resistance, proprioception
  • •3-6 months: Return to sport if criteria met (80-90% strength)

Key Complications

  • •Re-rupture 5-10% - prevented by adequate suture technique and compliance
  • •Adhesions 20-30% (especially Zone 4) - early motion helps prevent
  • •Infection 2-5% - higher with contaminated wounds
  • •Deep peroneal nerve injury - assess first web space sensation and EDB
  • •Weakness 10-20% - some residual deficit expected

Exam Traps to Avoid

  • •Not documenting neurovascular exam (deep peroneal nerve critical)
  • •Offering conservative management for active patient with TA rupture
  • •Using inadequate suture technique (simple sutures insufficient)
  • •Too aggressive rehab (risk re-rupture before 4 weeks)
  • •Not recognizing Zone 4 as critical zone for adhesions

High Yield Exam Points

  • •TA provides 80% of dorsiflexion - ALWAYS repair
  • •Zone 4 (retinaculum) worst outcomes due to adhesions
  • •Krackow suture strongest technique (50-60N breaking strength)
  • •Early protected motion superior to prolonged immobilization
  • •EDL repair optional if EDB intact (lesser toe function preserved)
  • •Surgical repair achieves 85% strength vs 60% conservative for TA
  • •Deep peroneal nerve runs with anterior tibial artery at ankle

Australian Context

Epidemiology in Australia:

  • Extensor tendon injuries common in rural areas (farm machinery, lawn mowers)
  • Work-related injuries covered under WorkCover in most states
  • Laceration injuries peak during summer months (outdoor activities, footwear)

Public System Coverage:

  • Tendon repair procedures covered under public hospital system
  • Complex tendon repairs with nerve repair fully funded
  • Tendon graft or reconstruction covered under public system

Pharmaceutical Benefits Scheme (PBS):

  • Post-operative antibiotics: Cephalexin 500mg QID (covered under PBS)
  • Pain management: Paracetamol/codeine combinations (PBS listed)

eTG (Therapeutic Guidelines) Recommendations:

  • Surgical prophylaxis: Cephazolin 2g IV pre-op (or clindamycin if penicillin allergic)
  • Post-operative: Continue oral antibiotics for 5-7 days if contaminated wound
  • Tetanus: Update if more than 10 years since last dose (or 5 years if contaminated)

Workers Compensation:

  • Extensor tendon injuries often work-related
  • Covered under WorkCover/WorkSafe in respective states
  • Typical return to work: Light duties at 6-8 weeks, full duties at 12-16 weeks
  • Hand-dominant occupations (e.g., construction, manual labor) may require longer rehabilitation

Rehabilitation Services:

  • Public hospital physiotherapy services available but may have waitlists
  • Private physiotherapy (Medicare rebate via EPC with GP referral, max 5 visits)
  • NDIS coverage if injury results in permanent disability (rare)

Australian Clinical Practice:

  • Early surgical repair advocated by Australian Orthopaedic Association
  • Growing trend toward early protected mobilization (follows international evidence)
  • Regional variations in access to specialist foot and ankle surgeons
  • Tertiary centers (e.g., Royal Adelaide, Royal Melbourne) offer complex reconstruction

Medicolegal Considerations:

  • Informed consent must cover: infection, nerve injury, re-rupture, adhesions, weakness
  • Document neurovascular status pre-operatively (deep peroneal nerve important)
  • Work-related injuries may involve medico-legal assessment for permanent impairment
  • Permanent impairment typically less than 5% whole person impairment for good outcome

Rural and Remote Australia:

  • Access to specialist care limited in remote areas
  • Telemedicine consultations increasingly utilized
  • Initial management in regional centers, complex cases referred to metropolitan hospitals
  • Royal Flying Doctor Service may be involved in remote trauma transfers

References

  1. Scaduto AA, Cracchiolo A. Lacerations and ruptures of the flexor or extensor hallucis longus tendons. Foot Ankle Clin. 2000;5(3):725-736.

  2. Markarian GG, Kelikian AS, Brage M, et al. Anterior tibialis tendon ruptures: an outcome analysis of operative versus nonoperative treatment. Foot Ankle Int. 1998;19(12):792-802.

  3. Browne K, Barnett S, Blundell C. Early controlled mobilisation after extensor tendon repair in the foot and ankle: a systematic review. J Foot Ankle Surg. 2011;50(3):315-320.

  4. Thompson SA, Whitehouse MR, Roberts S, Atkins RM. The strength of extensor tendon repair using different suture techniques: a biomechanical study. Foot Ankle Spec. 2015;8(6):452-458.

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