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Knee Biomechanics

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Knee Biomechanics

Fundamental biomechanical principles of the knee joint including tibiofemoral mechanics, patellofemoral tracking, ligament function, and clinical implications

complete
Updated: 2025-12-24
High Yield Overview

KNEE BIOMECHANICS

Tibiofemoral Mechanics | Patellofemoral Tracking | Ligament Function

2-3xBW in walking (up to 6x running)
3-4xBW patellofemoral force (stair climbing)
140°flexion ROM (functional arc 0-110°)
20-25mmposterior femoral rollback at 90° flexion

Knee Motion Patterns

Extension (0°)
PatternScrew-home mechanism, tibial ER 10°
TreatmentMaximum stability
Flexion (0-30°)
PatternRolling dominant, minimal translation
TreatmentCritical for gait
Flexion (30-140°)
PatternGliding dominant, femoral rollback
TreatmentSquatting/sitting

Critical Must-Knows

  • Screw-home mechanism: Automatic tibial external rotation 10° during terminal extension locks knee
  • Femoral rollback 20-25mm at 90° flexion prevents posterior impingement, maintained by PCL and posterolateral corner
  • ACL resists 85% of anterior tibial translation, secondary restraints resist remaining 15%
  • Patellofemoral reaction force = 3-4x body weight during stair climbing, 7-8x during squatting
  • Medial femoral condyle contacts 60% of tibial plateau, lateral 40% (medial bias in load distribution)

Examiner's Pearls

  • "
    Q-angle averages 14° males, 17° females (increased angle raises patellofemoral stress)
  • "
    Instant center of rotation shifts posteriorly with flexion (crucial for TKA design)
  • "
    MCL is primary valgus restraint at 30° flexion (ACL at full extension)
  • "
    Coupled motion: Tibial internal rotation with knee flexion, external rotation with extension

Critical Knee Biomechanics Exam Points

Screw-Home Mechanism

Terminal extension locks knee via tibial ER. Occurs due to asymmetric femoral condyles (lateral shorter). Popliteus unlocks knee to initiate flexion.

Femoral Rollback

20-25mm posterior translation at 90° flexion. Maintains extensor mechanism leverage. PCL and posterolateral corner are critical restraints.

ACL Function

Primary restraint to anterior tibial translation (85%). Secondary function resists internal rotation and valgus. Failure mechanism: pivot-shift injury.

Patellofemoral Force

Increases with knee flexion angle. 0.5x BW at 30°, 3-4x BW at 90° (stairs), 7-8x BW at 135° (deep squat). Explains anterior knee pain in flexion.

At a Glance

The knee experiences forces of 2-3× body weight during walking (up to 6× running) and 3-4× body weight patellofemoral force during stair climbing, making biomechanical understanding crucial for clinical practice. The screw-home mechanism automatically externally rotates the tibia 10° during terminal extension due to asymmetric femoral condyles (lateral shorter), locking the knee for standing stability—the popliteus unlocks this to initiate flexion. Femoral rollback (20-25mm at 90° flexion) prevents posterior impingement and maintains extensor mechanism leverage, controlled by the PCL and posterolateral corner. The ACL resists 85% of anterior tibial translation, with secondary restraints handling the remaining 15%; the MCL is the primary valgus restraint at 30° flexion while the ACL assumes this role at full extension. Patellofemoral reaction force increases exponentially with flexion, reaching 7-8× body weight during deep squatting, explaining anterior knee pain in flexion-loading activities.

Mnemonic

APMRFour Major Knee Ligaments and Primary Functions

A
ACL - Anterior translation
Resists anterior tibial translation (85%), secondary restraint to IR and valgus
P
PCL - Posterior translation
Resists posterior tibial translation, maintains femoral rollback
M
MCL - Medial (valgus) stability
Primary valgus restraint at 30° flexion, secondary at 0°
R
LCL/PLC - Lateral (varus) stability
Primary varus restraint, resists posterolateral rotation

Memory Hook:APMR keeps knees stable: Anterior/Posterior translation, Medial/Lateral (Rotational) stability!

Mnemonic

LEAPScrew-Home Mechanism Components

L
Lateral condyle shorter
Asymmetric femoral condyles drive external rotation
E
External rotation 10 degrees
Tibia externally rotates during terminal extension
A
Automatic locking
Locks knee in extension for standing stability
P
Popliteus unlocks
Popliteus contracts to internally rotate tibia and unlock knee

Memory Hook:LEAP into extension: Lateral condyle shorter drives External rotation for Automatic locking, Popliteus unlocks!

Overview and Introduction

Knee biomechanics encompasses the study of mechanical forces, motion patterns, and structural relationships within the knee joint complex. Understanding these principles is fundamental to orthopaedic practice, informing diagnosis, surgical technique, rehabilitation, and implant design.

The knee is the largest joint in the body, bearing forces up to 6x body weight during activities. It must balance mobility (0-140° flexion) with stability for weight-bearing. This is achieved through the complex interplay of bone geometry, ligaments, menisci, and surrounding musculature.

Concepts and Principles

Key Biomechanical Principles:

  1. Four-Bar Linkage Model: The ACL and PCL form a crossed linkage system that constrains motion while allowing rollback
  2. Screw-Home Mechanism: Terminal tibial external rotation locks the knee in extension
  3. Coupled Motion: Flexion couples with internal rotation; extension couples with external rotation
  4. Instant Center of Rotation: Shifts posteriorly with flexion, important for implant design

Tibiofemoral Biomechanics

Joint Geometry and Congruency

The tibiofemoral joint is incongruent - the convex femoral condyles articulate with relatively flat tibial plateaus. This geometry allows large range of motion but requires menisci and ligaments for stability.

Femoral Condyles

  • Medial condyle: Longer (60% load), larger radius, extends further distally
  • Lateral condyle: Shorter (40% load), smaller radius, flattens posteriorly
  • Asymmetry: Drives screw-home mechanism in terminal extension
  • Articular surface: Spherical anteriorly, flattens posteriorly

Tibial Plateaus

  • Medial plateau: Concave, larger surface area, more conforming
  • Lateral plateau: Convex or flat, smaller, less conforming
  • Posterior slope: 5-10° posteroinferior (sagittal plane)
  • Role: Minimal intrinsic stability without soft tissues

Clinical Relevance of Geometry

Low native congruency makes knee dependent on soft tissues. Meniscectomy or ligament injury significantly increases contact stress. ACL-deficient knee loses anterior restraint and develops abnormal kinematics. This is why ACL reconstruction restores both stability and normal motion patterns.

Tibiofemoral Motion Patterns

Knee motion combines rolling and gliding (sliding) of femoral condyles on tibial plateaus. The ratio changes with flexion angle.

Rolling vs Gliding Across Flexion Arc

Flexion RangeDominant MotionFemoral Contact PointClinical Significance
0-30° (early flexion)Rolling dominantMoves posteriorly on tibiaGait cycle, stance phase
30-90° (mid flexion)Mixed rolling and glidingProgressive posterior shiftStair climbing, chair rise
90-140° (deep flexion)Gliding dominant20-25mm posterior rollbackSquatting, kneeling

Femoral rollback is the posterior translation of the femoral condyles on the tibial plateau during flexion. This is critical for:

  • Maintaining extensor mechanism moment arm (prevents quadriceps insufficiency)
  • Preventing posterior soft tissue impingement
  • Allowing deep flexion without bony contact

The PCL and posterolateral corner resist excessive rollback. Loss of PCL leads to excessive posterior translation and abnormal kinematics.

Screw-Home Mechanism

The screw-home mechanism is the automatic external rotation of the tibia (10°) during the last 20-30° of extension. This locks the knee in full extension for energy-efficient standing.

Anatomical Basis

Why It Occurs

  • Asymmetric condyles: Lateral condyle shorter than medial
  • Geometry: Tibial ER aligns femoral condyles with tibial plateaus
  • Result: Maximum bony congruency at full extension
  • Stability: Locked position for standing without muscle effort

How to Unlock

  • Popliteus muscle contracts
  • Action: Internally rotates tibia, "unlocks" knee
  • Timing: Must occur before knee can flex from extension
  • Clinical: Popliteus injury causes "unlocking" difficulty

Clinical Testing

Q: How do you demonstrate screw-home mechanism clinically? A: Place patient supine with knee flexed 20°. Passively extend knee while palpating tibial tuberosity. Feel tibial external rotation during terminal extension. Absence suggests ACL or meniscal pathology affecting normal kinematics.

Ligament Function and Load Sharing

Anterior Cruciate Ligament (ACL)

The ACL is the primary restraint to anterior tibial translation, resisting 85% of anterior drawer force at 30° flexion.

Primary Function

  • Anterior translation: Resists 85% of anterior tibial displacement
  • Peak tension: 30° flexion (Lachman position)
  • Secondary functions: Resists internal rotation, valgus stress
  • Bundles: Anteromedial (tight in flexion), posterolateral (tight in extension)

Failure Mechanism

  • Pivot-shift injury: Valgus + internal rotation + anterior translation
  • Non-contact: Deceleration with foot planted, knee near extension
  • Result: Abnormal anterior translation, rotatory instability
  • Compensation: Secondary restraints insufficient alone

Secondary restraints to anterior translation include:

  • Medial and lateral menisci (posterior horns)
  • MCL (medial capsule)
  • Posterolateral corner
  • Iliotibial band

These resist approximately 15% of anterior drawer force combined. ACL reconstruction restores primary restraint and normal kinematics.

Posterior Cruciate Ligament (PCL)

The PCL resists posterior tibial translation and maintains femoral rollback during flexion.

PCL-deficient knees develop excessive posterior translation and loss of femoral rollback, leading to:

  • Patellofemoral pain (loss of extensor mechanism leverage)
  • Increased medial compartment load
  • Difficulty descending stairs or slopes

Collateral Ligaments

MCL vs LCL Function

LigamentPrimary RestraintPeak Load AngleAssociated Structures
MCL (medial)Valgus stress (30° flexion)25° flexion for superficial MCLMedial meniscus, posterior capsule
LCL (lateral)Varus stress (30° flexion)30° flexion for LCLPosterolateral corner, lateral meniscus

At full extension, the ACL and posterior capsule become primary restraints to valgus/varus stress. At 30° flexion, collaterals are isolated and maximally stressed.

Patellofemoral Biomechanics

Patellofemoral Joint Forces

The patella functions as a mechanical lever to increase the moment arm of the quadriceps, improving extension efficiency. However, this creates large compressive forces at the patellofemoral joint.

Force Magnitudes

  • Walking: 0.5x body weight
  • Stair climbing: 3-4x body weight
  • Squatting (90°): 7-8x body weight
  • Deep squat (135°): Up to 8-10x body weight

Why Force Increases

  • Flexion angle: Greater flexion = greater quadriceps force needed
  • Moment arm: Patellar tendon lever arm decreases in deep flexion
  • Contact area: Peak stress at 60-90° flexion
  • Clinical: Explains anterior knee pain with stairs/squats

Patellar Tracking

Normal patellar tracking requires balance between medial and lateral stabilizers.

Medial vs Lateral Patellar Stabilizers

StructureRestraint DirectionContributionFailure Pattern
MPFL (medial patellofemoral ligament)Resists lateral displacement50-60% of medial restraintPatellar dislocation (lateral)
VMO (vastus medialis obliquus)Dynamic medial stabilizerActive stabilization in extensionVMO atrophy worsens tracking
Lateral retinaculumPassive lateral tetherBalanced by medial structuresTightness causes lateral tilt/compression

Q-angle (quadriceps angle) measures the vector of quadriceps pull relative to patellar tendon. Normal values:

  • Males: 14° average
  • Females: 17° average (wider pelvis)

Increased Q-angle raises lateral patellar stress and predisposes to lateral tracking disorders.

Load Distribution and Contact Mechanics

Knee flexion angle and joint reaction force during normal gait cycle
Click to expand
Knee kinematics and loading during normal gait. Left panel: Knee flexion angle (radians) vs time showing characteristic pattern with heel strike and toe off markers - note flexion peaks during swing phase and relative extension during stance. Right panel: Total joint reaction force (N) vs time demonstrating the typical double-peak loading pattern during stance phase, reaching ~2500N (approximately 3x body weight for a 70kg individual). Understanding these loading patterns is fundamental to knee biomechanics and TKA design.Credit: Loi I et al., Front Bioeng Biotechnol 2021 - CC BY
Tibial insert contact pressure distribution during different gait patterns
Click to expand
Tibial contact pressure maps from finite element analysis during gait phases. Panels A-D show contact pressure distribution (MPa) on tibial polyethylene insert surface with maximum pressures of 2.19, 3.80, 3.64, and 7.85 MPa respectively. Color scale: blue (low) to red (high). Note predominantly medial loading pattern in panels A and C, reflecting the 60:40 medial:lateral load distribution. Higher contact stresses occur at loading response and push-off phases of gait cycle.Credit: Loi I et al., Front Bioeng Biotechnol 2021 - CC BY

Tibiofemoral Contact Stress

The menisci are critical for load distribution. They transmit 40-60% of knee joint load and reduce peak contact stress on articular cartilage.

Meniscectomy consequences:

  • Total meniscectomy: 200-300% increase in peak contact stress
  • Partial meniscectomy: 65-100% increase (depends on amount removed)
  • Increased risk of osteoarthritis

Alignment effects:

  • Varus alignment: Shifts load medially (medial compartment overload)
  • Valgus alignment: Shifts load laterally (lateral compartment overload)
  • Neutral alignment: 60% medial, 40% lateral load distribution

Clinical Applications

Total Knee Arthroplasty Design Considerations

Understanding knee biomechanics guides TKA design:

Femoral Component

  • Rollback: Must replicate normal posterior translation
  • J-curve: Matches native femoral condyle geometry
  • Asymmetric condyles: Replicates medial/lateral differences
  • Goal: Restore normal kinematics and ROM

Tibial Component

  • Posterior slope: 0-7° to facilitate rollback
  • Conformity: More conforming = lower contact stress but less mobility
  • Flat vs dished: Trade-off between stability and constraint
  • Polyethylene: Must withstand 3-6x BW cyclically

Cruciate-retaining vs posterior-stabilized TKA:

  • CR-TKA: Preserves PCL, maintains femoral rollback naturally
  • PS-TKA: Resects PCL, uses cam-post mechanism to replicate rollback
  • Both aim to restore 20-25mm posterior translation at 90° flexion

Evidence Base

Femoral Rollback in Native Knee Kinematics

3
Dennis et al • Clin Orthop Relat Res (2005)
Key Findings:
  • Fluoroscopic analysis of healthy knees during weight-bearing
  • Femoral rollback averages 20-25mm from 0° to 90° flexion
  • Rollback maintained by PCL and posterolateral corner
  • Loss of rollback reduces quadriceps moment arm and ROM
Clinical Implication: TKA designs must replicate femoral rollback to restore normal kinematics and prevent quadriceps insufficiency.
Limitation: In vivo fluoroscopy limited to 2D projection, not full 3D analysis.

ACL Load Sharing and Secondary Restraints

3
Butler et al • J Biomech (1980)
Key Findings:
  • ACL resists 85% of anterior drawer force at 30° flexion
  • Secondary restraints (menisci, MCL, capsule) resist remaining 15%
  • ACL failure results in anterior translation exceeding 5mm
  • Secondary restraints alone cannot prevent abnormal kinematics
Clinical Implication: ACL reconstruction is necessary to restore primary anterior restraint and normal knee kinematics in active patients.
Limitation: Cadaveric study, does not account for dynamic muscle stabilization.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Knee Kinematics and Screw-Home Mechanism

EXAMINER

"Examiner asks: Describe the screw-home mechanism of the knee. Why does it occur and what is its clinical significance?"

EXCEPTIONAL ANSWER
The screw-home mechanism is the automatic external rotation of the tibia during the last 20-30 degrees of knee extension. This occurs due to the asymmetry of the femoral condyles - the lateral condyle is shorter than the medial condyle. As the knee extends, the lateral femoral condyle reaches full extension before the medial condyle, causing the tibia to externally rotate approximately 10 degrees to complete extension. Clinically, this locks the knee in full extension for energy-efficient standing. The popliteus muscle unlocks the knee by internally rotating the tibia to initiate flexion. Disruption of this mechanism may occur with ACL or meniscal injuries, leading to abnormal kinematics.
KEY POINTS TO SCORE
Define screw-home: automatic tibial ER 10° in terminal extension
Anatomical basis: asymmetric femoral condyles (lateral shorter)
Function: locks knee for standing stability
Popliteus unlocks knee to initiate flexion
COMMON TRAPS
✗Confusing with coupled rotation (flexion-extension)
✗Forgetting to mention popliteus role in unlocking
✗Not explaining clinical significance for standing
LIKELY FOLLOW-UPS
"What happens to screw-home after ACL injury?"
"How would you test for screw-home clinically?"
"What role does popliteus play in knee stability?"
VIVA SCENARIOChallenging

Scenario 2: ACL Function and Biomechanics

EXAMINER

"Examiner shows fluoroscopy of ACL-deficient knee and asks: Explain the biomechanical functions of the ACL and what happens when it is torn."

EXCEPTIONAL ANSWER
The ACL has both primary and secondary biomechanical functions. Its primary function is to resist anterior translation of the tibia on the femur, accounting for 85% of the restraint to anterior drawer force at 30 degrees of flexion. Secondary functions include resisting internal tibial rotation and valgus stress. The ACL has two bundles: the anteromedial bundle is tight in flexion, while the posterolateral bundle is tight in extension. When torn, patients develop anterior translation and rotatory instability, manifesting as the pivot-shift phenomenon. Secondary restraints including the menisci, MCL, and posterolateral corner can only resist 15% of anterior drawer force, which is insufficient to prevent abnormal kinematics. This is why ACL reconstruction is recommended for active patients.
KEY POINTS TO SCORE
Primary function: resist 85% of anterior tibial translation
Secondary functions: resist IR and valgus
Two bundles with reciprocal tightening patterns
Secondary restraints insufficient when ACL torn
COMMON TRAPS
✗Saying ACL only prevents anterior translation (forget rotatory control)
✗Not quantifying 85% contribution
✗Forgetting to mention secondary restraints and their inadequacy
LIKELY FOLLOW-UPS
"What is the pivot-shift test and why does it occur?"
"What structures act as secondary restraints?"
"How does ACL reconstruction restore kinematics?"

MCQ Practice Points

Load Distribution Question

Q: What percentage of knee joint load is transmitted through the menisci in a healthy knee? A: 40-60% - The menisci are critical load distributors. Total meniscectomy increases peak contact stress by 200-300%, significantly raising osteoarthritis risk.

ACL Load Sharing Question

Q: What percentage of anterior drawer restraint does the ACL provide at 30° flexion? A: 85% - The ACL is the primary anterior restraint. Secondary restraints (menisci, MCL, capsule) resist only 15%, which is insufficient to prevent abnormal kinematics when ACL is torn.

Femoral Rollback Question

Q: How much posterior femoral translation (rollback) occurs at 90° knee flexion? A: 20-25mm - Femoral rollback is critical for maintaining extensor mechanism leverage and preventing posterior impingement. PCL and posterolateral corner resist excessive rollback.

Australian Context

Australian Epidemiology and Practice

Knee Biomechanics in Australian Practice:

  • Knee biomechanics forms a core component of FRACS Basic Science examination
  • Understanding ACL function, screw-home mechanism, and patellofemoral mechanics is essential for both Part I and Part II examinations
  • AOANJRR data demonstrates over 65,000 primary TKAs performed annually in Australia, making understanding of knee kinematics critical for surgical planning

RACS Orthopaedic Training Relevance:

  • FRACS curriculum emphasizes four-bar linkage model, instant center of rotation, and coupled motion concepts
  • ACL biomechanics examination questions focus on load sharing percentages (85% primary, 15% secondary restraints)
  • Understanding femoral rollback (20-25mm) and its implications for TKA design is frequently examined
  • Screw-home mechanism and popliteus function are common viva topics

Australian Knee Registry Data:

  • AOANJRR tracks TKA outcomes with particular attention to kinematic alignment vs mechanical alignment techniques
  • Registry data informs choice between CR-TKA and PS-TKA based on long-term revision rates
  • Australian surgeons contribute to global understanding of knee kinematics through research at major centres

Clinical Application in Australian Practice:

  • Major trauma centres employ fluoroscopic gait analysis for complex knee pathology assessment
  • Sports medicine centres around Australia utilise biomechanical analysis for ACL injury prevention programs
  • Victorian Institute of Sport and Queensland Academy of Sport integrate knee biomechanics into athlete screening protocols

PBS Considerations:

  • Physiotherapy for knee rehabilitation is subsidised under Medicare chronic disease management plans
  • Hyaluronic acid injections for knee OA management available with appropriate clinical indication

KNEE BIOMECHANICS

High-Yield Exam Summary

Tibiofemoral Motion

  • •Screw-home mechanism = 10° tibial ER in terminal extension (locks knee)
  • •Femoral rollback = 20-25mm posterior at 90° flexion (PCL maintains)
  • •Rolling dominant 0-30°, gliding dominant 30-140°
  • •Functional ROM for ADLs = 0-110° flexion

Ligament Functions

  • •ACL: 85% anterior translation restraint, secondary IR/valgus
  • •PCL: 95% posterior translation restraint, maintains rollback
  • •MCL: Primary valgus restraint at 30° flexion
  • •LCL/PLC: Primary varus restraint, posterolateral stability

Patellofemoral Mechanics

  • •PF force: 0.5x BW walking, 3-4x BW stairs, 7-8x BW squatting
  • •Q-angle: 14° males, 17° females (higher = more lateral stress)
  • •MPFL provides 50-60% medial restraint to patellar dislocation
  • •Contact stress peaks at 60-90° flexion

Load Distribution

  • •Menisci transmit 40-60% of joint load
  • •Total meniscectomy increases peak stress 200-300%
  • •Medial:lateral load ratio = 60:40 in neutral alignment
  • •Varus shifts load medially, valgus shifts laterally

TKA Design Principles

  • •Must replicate 20-25mm femoral rollback at 90° flexion
  • •Tibial slope 0-7° facilitates rollback
  • •CR-TKA preserves PCL, PS-TKA uses cam-post mechanism
  • •Goal: restore normal kinematics and ROM 0-120°

Key Numbers

  • •Joint forces: 2-3x BW walking, up to 6x BW running
  • •Popliteus unlocks knee (IR tibia) to initiate flexion
  • •PCL is 2x stronger than ACL
  • •Secondary restraints resist only 15% anterior drawer
Quick Stats
Reading Time58 min
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