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Lateral Ankle Instability

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Lateral Ankle Instability

Comprehensive guide to lateral ankle instability including ATFL/CFL anatomy, examination techniques, Brostrom-Gould repair, and anatomic reconstruction options for Orthopaedic examination.

complete
Updated: 2026-01-02
High Yield Overview

Lateral Ankle Instability

ATFL and CFL Insufficiency

Weakest, injured first (inversion + plantarflexion)ATFL
Injured with severe sprains (inversion + dorsiflexion)CFL
Acute sprains progress to chronic instability20-40%
85-90% good/excellent outcomesBrostrom-Gould

Ankle Sprain Grades

Grade I
PatternLigament stretch, no macroscopic tear
TreatmentRICE, early mobilization
Grade II
PatternPartial ligament tear
TreatmentBrace 4-6 weeks, rehabilitation
Grade III
PatternComplete ligament rupture
TreatmentBrace, rehabilitation ± surgery

Critical Must-Knows

  • ATFL: Weakest lateral ligament. Resists inversion in plantarflexion. Most commonly injured.
  • CFL: Injured in severe sprains. Resists inversion in dorsiflexion. Crosses both ankle and subtalar joints.
  • Anterior Drawer: Tests ATFL integrity. Compare to contralateral side.
  • Talar Tilt: Tests CFL integrity. Performed in neutral dorsiflexion.
  • Brostrom-Gould: Direct ATFL repair + inferior extensor retinaculum augmentation. Gold standard surgical treatment.

Examiner's Pearls

  • "
    ATFL is weakest - always injured first (ACP order)
  • "
    Anterior drawer tests ATFL, talar tilt tests CFL
  • "
    Rehabilitation first - majority stabilize without surgery
  • "
    Brostrom repair is gold standard (85-90% success)
  • "
    Reconstruction for revision, heavy athletes, or generalized laxity

Critical Anatomy

Lateral ligament injury order: ATFL → CFL → PTFL (weakest to strongest).

  • ATFL is weakest - injured with inversion in plantarflexion (most common mechanism).
  • CFL is next - injured with greater force (inversion in dorsiflexion). Crosses ankle AND subtalar joints.
  • PTFL is strongest - only injured in severe ankle dislocations.
  • Remember: ACP = Anterior, Calcaneofibular, Posterior (weakest to strongest).

At a Glance Table

FeatureDetails
DefinitionChronic mechanical and/or functional instability of the lateral ankle complex
Epidemiology23,000 ankle sprains/day (US); 20-40% develop chronic instability
Key LigamentsATFL (most common), CFL, PTFL (rare)
MechanismInversion injury, typically in plantarflexion
Key ExamAnterior drawer (ATFL), Talar tilt (CFL)
First-Line TreatmentRehabilitation: proprioceptive training + peroneal strengthening
Surgical Gold StandardBrostrom-Gould repair (85-90% success)
Reconstruction IndicationsFailed Brostrom, generalized ligamentous laxity, revision, heavy athletes
Mnemonic

Lateral Ligaments - ACP

A
ATFL
Anterior talofibular - weakest, most commonly injured
C
CFL
Calcaneofibular - crosses ankle AND subtalar joints
P
PTFL
Posterior talofibular - strongest, rarely injured

Memory Hook:ACP = Anterior to Posterior = Weakest to Strongest. Like the alphabet, A comes first (injured first).

Mnemonic

Stress Test Interpretation - ATE

A
Anterior Drawer
Tests ATFL - anterior translation of talus
T
Talar Tilt
Tests CFL - inversion stress in neutral dorsiflexion
E
External Rotation
Tests Syndesmosis - NOT lateral ligaments

Memory Hook:ATE my ankle! Know which ligament each stress test assesses.

Mnemonic

SCAR - Lateral Ankle Surgery Complications

S
Stiffness
Especially dorsiflexion - avoid over-tightening repair
C
Chronic pain
May indicate missed OCD or impingement
A
And nerve injury
Superficial peroneal most common
R
Recurrence
5-10% - may need reconstruction

Memory Hook:SCAR reminds you of the main complications that can leave a mark on outcomes.

Overview/Epidemiology

Lateral ankle instability is the most common sports injury worldwide, affecting athletes across all sports and age groups.

Epidemiology

Incidence

  • 23,000 ankle sprains per day in the United States
  • 1 per 10,000 person-days in general population
  • Up to 30% of all sporting injuries
  • Most common in basketball, soccer, and volleyball

Natural History

  • 20-40% of acute sprains develop chronic instability
  • Risk factors: incomplete rehabilitation, ligamentous laxity
  • 55% have residual symptoms at 6 months
  • Recurrence rate without treatment: 70-80%

Demographics

  • Peak incidence: 15-35 years (active population)
  • Equal male:female ratio in general population
  • Higher female incidence in specific sports
  • Prior ankle sprain strongest risk factor

Types of Instability

  1. Mechanical Instability: Objective ligament laxity on stress testing
  2. Functional Instability: Subjective feeling of giving way despite normal stress tests
  3. Combined: Most patients have elements of both

Risk Factors for Chronic Instability

  • Previous ankle sprain (strongest predictor)
  • Incomplete or inadequate rehabilitation
  • Generalized ligamentous laxity (Beighton score greater than 4)
  • High-demand sports (basketball, soccer, netball)
  • Hindfoot varus malalignment
  • Cavovarus foot deformity
  • Peroneal weakness or dysfunction

Pathophysiology and Mechanisms

Lateral Ankle Ligament Complex

The lateral ankle ligaments originate from the lateral malleolus and provide stability against inversion and anterior translation of the talus.

Lateral Ankle Ligaments - Detailed Anatomy

LigamentOriginInsertionFunctionInjury Order
Anterior border of lateral malleolusTalar neck (anterolateral)Resists anterior translation + inversion in plantarflexion1st (weakest)
Tip of lateral malleolusLateral calcaneusResists inversion in dorsiflexion; crosses ankle + subtalar2nd
Medial surface of lateral malleolusPosterior talusResists posterior translation; strongest ligament3rd (rarely injured)

ATFL Anatomy (Most Important)

  • Length: 20-25mm
  • Width: 10-12mm
  • Orientation: Horizontal in plantarflexion → becomes vertical in dorsiflexion
  • Variants: 1-3 bands (2 bands most common)
  • Strength: Weakest of the three (58-90N failure load)

CFL Anatomy

  • Length: 20-25mm
  • Crosses two joints: Ankle AND subtalar (unique feature)
  • Orientation: 10-45° posterior to long axis of fibula
  • Key relationship: Lies deep to peroneal tendons
  • Clinical significance: CFL injury affects subtalar stability

Biomechanics of Injury

Injury Mechanism

The typical mechanism is inversion injury in plantarflexion:

  • In plantarflexion, the narrower posterior talus sits in the mortise → less bony stability
  • ATFL becomes the primary restraint → first to fail
  • With continued force, CFL then fails
  • PTFL only fails with severe trauma/dislocation

Dynamic Stabilizers

  • Peroneus longus and brevis: Primary dynamic stabilizers
  • Extensor digitorum longus: Secondary support
  • Proprioceptive afferents: Critical for neuromuscular control
  • Peroneal reaction time: Delayed in chronic instability

Classification Systems

Ankle Sprain Grading

Ankle Sprain Classification

GradePathologyClinical FindingsStabilityTreatment
Microscopic tear, stretch injuryMild swelling, tendernessStableRICE, early mobilization
Partial macroscopic tearModerate swelling, ecchymosisMild laxityBrace 4-6 weeks, physio
Complete rupture ATFL ± CFLSevere swelling, ecchymosisFrank instabilityBrace, physio ± surgery

Grade I and II injuries typically heal with conservative treatment. Grade III injuries may require surgical intervention if chronic instability develops.

Chronic Ankle Instability Classification

Mechanical Instability

  • Objective ligament laxity on examination
  • Positive anterior drawer (greater than 10mm translation)
  • Positive talar tilt (greater than 10° compared to other side)
  • Stress radiograph abnormalities

Functional Instability

  • Subjective feeling of instability/giving way
  • Normal or near-normal stress testing
  • Proprioceptive deficits
  • Peroneal weakness or delayed reaction time

Most patients with chronic instability have components of both mechanical and functional instability requiring comprehensive rehabilitation.

Ligament Injury Pattern Classification

PatternLigaments InvolvedStress TestSurgical Implications
Isolated ATFLATFL onlyAnterior drawer positiveBrostrom repair (ATFL only)
ATFL + CFLBoth ligamentsAnterior drawer + talar tiltBrostrom repair (both ligaments)
Complete lateralATFL + CFL + PTFLGrossly unstableConsider reconstruction

The pattern of ligament injury determines the surgical approach and the extent of repair required.

Clinical Assessment

History

Key Questions:

  • Initial injury: mechanism, treatment, rehabilitation
  • Symptoms: giving way episodes, pain, swelling
  • Functional limitation: sports, uneven ground, stairs
  • Prior treatment: physiotherapy duration, bracing
  • Red flags: locking (loose body), medial pain (deltoid), persistent swelling (OCD)

Physical Examination

Observation

  • Swelling: lateral ankle, anterolateral gutter
  • Ecchymosis: may indicate recent episode
  • Hindfoot alignment: varus predisposes to lateral sprains
  • Cavus foot: associated with instability
  • Muscle wasting: peroneal atrophy suggests chronic injury

Inspection findings help assess the chronicity and severity of instability.

Key Structures to Palpate

  1. ATFL: Anterior to lateral malleolus (most common tenderness)
  2. CFL: Below lateral malleolus, deep to peroneals
  3. Lateral malleolus: Rule out fracture
  4. Base of 5th metatarsal: Avulsion injury
  5. Anterior process calcaneus: Associated fracture
  6. Peroneal tendons: Subluxation, tendinopathy
  7. Anterior talofibular joint: OCD, impingement

Systematic palpation identifies the primary pathology and associated injuries.

Stress Tests

Anterior Drawer Test (ATFL)

  • Position: Slight plantarflexion, knee flexed
  • Technique: Stabilize tibia, translate talus anteriorly
  • Positive: Greater than 3mm difference vs. contralateral
  • Grading: +1 (3-5mm), +2 (5-10mm), +3 (greater than 10mm)

Talar Tilt Test (CFL)

  • Position: Neutral dorsiflexion
  • Technique: Invert talus in mortise
  • Positive: Greater than 10° tilt or greater than 5° vs. contralateral
  • Note: Also tests subtalar stability

External Rotation Stress Test

  • Tests syndesmosis (not lateral ligaments)
  • Important to exclude high ankle sprain

Always compare to the contralateral side for accurate assessment.

Additional Examination

Peroneal Assessment

  • Strength: Resisted eversion
  • Subluxation: Resisted dorsiflexion/eversion
  • Tendinopathy: Tenderness along tendons

Subtalar Joint

  • Stability: CFL injury affects subtalar
  • Arthritis: Chronic instability sequela

Hindfoot Alignment

  • Varus: Predisposes to lateral sprains
  • Coleman block test: Flexible vs. rigid cavovarus

Generalized Laxity

  • Beighton score: Affects surgical planning
  • Score greater than 4: Consider reconstruction over repair

Comprehensive examination guides treatment selection.

Investigations

Imaging Protocol

Standard Views

  • Weight-bearing AP: Mortise alignment, OCD
  • Mortise view: Lateral clear space
  • Lateral: Talar dome, anterior impingement

Stress Radiographs (If Indicated)

  • Anterior drawer stress: Greater than 10mm translation significant
  • Talar tilt stress: Greater than 10° or greater than 5° vs. contralateral
  • Useful for objective documentation
  • Compare to contralateral side

Standard weight-bearing views are essential first-line imaging.

Indications

  • Diagnostic uncertainty
  • Suspected osteochondral lesion
  • Pre-operative planning
  • Associated pathology (peroneal, syndesmosis)

Key Findings

  • ATFL: Discontinuity, thickening, signal change
  • CFL: Often better seen on axial images
  • OCD: Talar dome lesions (common association)
  • Peroneal tendons: Tears, subluxation

MRI has high sensitivity but clinical examination remains primary diagnostic tool.

CT Scan

  • Osteochondral lesion characterization
  • Bony morphology assessment
  • Pre-operative planning for malalignment correction

Ultrasound

  • Dynamic assessment of ligaments
  • Peroneal subluxation (dynamic)
  • Operator dependent
  • Cost-effective screening

Advanced imaging is reserved for specific clinical scenarios.

Associated Pathology to Exclude

ConditionFrequencyInvestigationClinical Significance
Osteochondral lesion (OCD)20-25%MRI, CTAddress at surgery
Peroneal tendon pathology15-20%MRI, ultrasoundMay need concurrent repair
Anterior impingement10-15%Lateral X-ray, MRIDebride at arthroscopy
Syndesmosis injury5-10%X-ray, MRIDifferent treatment
Subtalar instabilityVariableStress viewsConsider reconstruction

Management Algorithm

📊 Management Algorithm
Lateral Ankle Instability Management Algorithm
Click to expand
Medical sketchnote flowchart showing the treatment pathway from conservative rehabilitation to surgical options.

Treatment Decision Framework

First-Line for ALL Patients

Duration: 3-6 months minimum before considering surgery

Components:

  1. Proprioceptive Training

    • Wobble board exercises
    • Single-leg stance progressions
    • Sport-specific balance training
  2. Peroneal Strengthening

    • Resisted eversion exercises
    • Theraband programs
    • Functional strengthening
  3. Ankle Bracing

    • Semi-rigid brace for sport
    • Lace-up brace for daily activities
    • Taping for specific activities
  4. Activity Modification

    • Avoid uneven terrain initially
    • Gradual return to sport

Success Rate: 70-80% of patients stabilize with rehabilitation alone.

Conservative treatment remains the cornerstone of management.

Indications for Surgery

Primary Indications:

  • Failed 3-6 months of supervised rehabilitation
  • Recurrent giving way episodes despite compliance
  • High-level athlete with mechanical instability
  • Functional limitation affecting quality of life

Relative Indications:

  • Early intervention in elite athletes (controversial)
  • Associated pathology requiring surgery (OCD, impingement)
  • Patient preference after informed consent

Contraindications:

  • Active infection
  • Severe peripheral vascular disease
  • Non-compliance with post-operative protocol
  • Inadequate rehabilitation trial

Surgery is reserved for patients who fail appropriate conservative management.

Surgical Procedures

1. Brostrom-Gould Repair (Gold Standard)

  • Direct repair of ATFL ± CFL
  • Inferior extensor retinaculum augmentation
  • 85-90% good/excellent results

2. Anatomic Reconstruction

  • Indications: Failed Brostrom, generalized laxity, revision
  • Graft options: Peroneus brevis, hamstring allograft
  • Reproduces native anatomy

3. Non-Anatomic Reconstruction

  • Evans procedure, Watson-Jones, Chrisman-Snook
  • Historical procedures - largely abandoned
  • Restrict subtalar motion

4. Arthroscopic Techniques

  • Arthroscopic Brostrom repair
  • Address concomitant pathology
  • Equivalent outcomes to open in select patients

Procedure selection depends on patient factors and tissue quality.

Treatment Algorithm Summary

  1. All patients: Rehabilitation first (3-6 months)
  2. Persistent symptoms: Brostrom-Gould repair
  3. Failed Brostrom/Generalized laxity/Heavy athlete: Anatomic reconstruction
  4. Revision surgery: Consider reconstruction with allograft

Surgical Technique

Brostrom-Gould Repair (Gold Standard)

Patient Positioning

  • Position: Supine with bump under ipsilateral hip
  • Tourniquet: High thigh (250-300mmHg)
  • Leg position: Slight internal rotation for lateral access

Equipment

  • Standard foot and ankle set
  • Suture anchors (2.4-3.0mm)
  • Non-absorbable braided suture
  • Fluoroscopy (optional)

Approach

  • Curvilinear incision over lateral malleolus
  • Length: 4-6cm
  • Identify and protect superficial peroneal nerve branches

Proper positioning and nerve protection are essential for successful repair.

Surgical Steps

Step 1: Exposure

  • Incise through subcutaneous tissue
  • Identify and protect superficial peroneal nerve
  • Identify inferior extensor retinaculum (IER)
  • Open ankle capsule

Step 2: ATFL Identification

  • ATFL lies within anterolateral capsule
  • Often attenuated and scarred
  • Identify talar and fibular insertions

Step 3: Ligament Repair

  • Pants-over-vest imbrication of ATFL remnant
  • Suture anchor at fibular origin (anatomic footprint)
  • Repair with foot in eversion and dorsiflexion
  • Tighten to eliminate anterior drawer

Step 4: Gould Modification (Critical)

  • Advance IER over ATFL repair
  • Anchor to fibula with sutures
  • Provides additional restraint and proprioceptive input

Step 5: CFL Repair (If Needed)

  • Identify CFL deep to peroneal tendons
  • Similar vest-over-pants technique
  • Suture anchor at fibular footprint

Step 6: Closure

  • Irrigate wound
  • Layered closure
  • Below-knee backslab in neutral

The Gould modification (IER augmentation) is critical for improved outcomes.

Anatomic Reconstruction Technique

Indications:

  • Failed Brostrom repair
  • Generalized ligamentous laxity (Beighton greater than 4)
  • Heavy/high-demand athletes
  • Poor quality native tissue

Graft Options:

  • Peroneus brevis (autograft)
  • Hamstring tendon (autograft)
  • Semitendinosus allograft (preferred)
  • Peroneus longus (less common)

Key Principles:

  • Anatomic tunnel placement (fibula, talus, calcaneus)
  • Graft tensioning in neutral position
  • Interference screw or suture anchor fixation
  • Recreate both ATFL and CFL

Modified Brostrom with InternalBrace:

  • Augmentation with suture tape
  • Allows early mobilization
  • Good results in select patients

Reconstruction provides additional stability for high-demand patients.

Technical Pearls

Exposure:

  • Map superficial peroneal nerve before incision
  • Preserve IER for Gould modification
  • Identify both ATFL and CFL

Repair:

  • Tension repair with foot in eversion/dorsiflexion
  • Avoid over-tightening (stiffness)
  • Place anchors at anatomic footprint

Closure:

  • IER advancement is critical (Gould modification)
  • Layered closure prevents wound dehiscence

Pitfalls to Avoid

  • Nerve injury: Always identify superficial peroneal nerve
  • Over-tightening: Causes stiffness and subtalar restriction
  • Missed CFL injury: Assess both ligaments
  • Inadequate IER advancement: Key to Gould modification
  • Wrong anchor placement: Use anatomic footprints

Attention to detail prevents common complications.

Nerve at Risk

The superficial peroneal nerve is at risk with lateral ankle approaches. It typically crosses the surgical field 5-10cm proximal to the tip of the lateral malleolus. Always identify and protect this nerve to avoid sensory deficit.

Complications

Complications by Type

Intraoperative Complications

ComplicationIncidencePreventionManagement
Superficial peroneal nerve injury2-5%Careful dissection, identify before incisionNeurolysis, reassurance
Sural nerve injury1-2%Posterior incision extension awarenessReassurance (usually neuropraxia)
Inadequate tissue quality5-10%Pre-op MRI assessmentConvert to reconstruction
Anchor pull-outRareProper technique, bone quality assessmentRe-anchor or transosseous sutures

Nerve identification is the key to preventing intraoperative complications.

Early Complications (less than 6 weeks)

Wound Complications:

  • Wound dehiscence: 2-5%
  • Superficial infection: 1-3%
  • Deep infection: less than 1%
  • Haematoma: 1-2%

Prevention:

  • Layered closure
  • Appropriate immobilization
  • Early mobilization within protocol
  • DVT prophylaxis

Management:

  • Wound care, antibiotics as needed
  • Return to theatre if deep infection

Early wound complications are minimized with careful surgical technique.

Late Complications (greater than 6 weeks)

Stiffness (5-10%):

  • Usually dorsiflexion restriction
  • Prevention: Early mobilization, avoid over-tightening
  • Treatment: Physiotherapy, manipulation under anaesthesia

Recurrent Instability (5-10%):

  • Failure of repair or stretch-out
  • Risk factors: Non-compliance, generalized laxity
  • Treatment: Revision with reconstruction

Chronic Pain (5-15%):

  • May indicate missed pathology (OCD, impingement)
  • Investigation: MRI, diagnostic arthroscopy
  • Treatment: Address underlying cause

Subtalar Stiffness:

  • More common with non-anatomic reconstructions
  • Brostrom repair preserves subtalar motion

Late complications often require revision surgery.

Postoperative Care

Rehabilitation Protocol

Immobilization Phase

Goals:

  • Wound healing
  • Pain and swelling control
  • Prevent DVT

Protocol:

  • Below-knee backslab or CAM boot
  • Non-weight bearing or touch weight bearing
  • Elevation, ice
  • Ankle pumps (within splint if able)

Follow-up:

  • Wound check at 10-14 days
  • Remove sutures
  • Transition to CAM boot

Early immobilization protects the healing repair.

Protected Mobilization Phase

Goals:

  • Early ROM (avoid inversion)
  • Progressive weight bearing
  • Maintain muscle function

Protocol:

  • CAM boot for ambulation
  • Weight bearing as tolerated in boot
  • Active ROM: Dorsiflexion, plantarflexion (avoid inversion)
  • Isometric peroneal exercises
  • Remove boot for exercises and sleeping (by week 4)

Restrictions:

  • No inversion stress
  • No running or jumping

Protected mobilization allows healing while preventing stiffness.

Strengthening Phase

Goals:

  • Full ROM
  • Progressive strengthening
  • Proprioceptive training initiation

Protocol:

  • Wean from boot
  • Supportive ankle brace for activities
  • Full ROM exercises including inversion
  • Progressive resistance exercises
  • Proprioceptive training: Wobble board, single-leg stance
  • Stationary cycling, swimming

Milestones:

  • Full pain-free ROM by week 8
  • Single-leg stance 30 seconds by week 10

Proprioceptive training is critical for functional recovery.

Return to Sport Phase

Goals:

  • Return to full activity
  • Sport-specific training
  • Long-term stability

Protocol:

  • Jogging progression (week 12)
  • Agility drills (week 14-16)
  • Sport-specific training (week 16-20)
  • Full return to sport (4-6 months)

Return to Sport Criteria:

  • Full pain-free ROM
  • Equal strength to contralateral
  • Completed agility testing
  • Sport-specific drills without symptoms

Long-term:

  • Ankle brace for sport (first 6-12 months)
  • Ongoing proprioceptive maintenance

Gradual return to sport prevents re-injury.

Expected Outcomes

Outcome MeasureBrostrom-GouldAnatomic Reconstruction
Good/Excellent results85-90%80-90%
Return to sport85-95%80-90%
Recurrence rate5-10%5-15%
Time to sport4-6 months6-9 months

Outcomes

Results by Treatment Type

Conservative Treatment

  • 70-80% stabilize without surgery
  • Best results with supervised physiotherapy
  • Proprioceptive training key to success
  • Peroneal strengthening essential
  • Bracing for sport may be permanent

Brostrom-Gould Repair

  • 85-90% good/excellent results
  • Gold standard surgical treatment
  • Preserves subtalar motion
  • Lower revision rate than non-anatomic
  • Return to sport: 4-6 months

Anatomic Reconstruction

  • 80-90% good results
  • Reserved for failures, laxity, heavy athletes
  • Longer rehabilitation (6-9 months)
  • Allograft reduces donor site morbidity
  • Higher revision rate than primary Brostrom

Prognostic Factors

Favourable:

  • Single ligament injury (ATFL only)
  • Good tissue quality
  • Normal hindfoot alignment
  • Compliant patient
  • Supervised rehabilitation

Unfavourable:

  • Combined ATFL + CFL injury
  • Generalized ligamentous laxity
  • Hindfoot varus or cavovarus
  • Associated OCD
  • Poor compliance

Evidence Base

Landmark
📚 Brostrom L - Original Brostrom Repair
Key Findings:
  • Original description of direct ligament repair technique
  • 85% good results at long-term follow-up
  • Foundation for modern lateral ankle stabilization
Clinical Implication: Brostrom repair established as gold standard treatment for chronic lateral ankle instability.
Source: J Bone Joint Surg Am 1966

Level IV
📚 Gould N et al - Gould Modification
Key Findings:
  • Added inferior extensor retinaculum augmentation
  • Improved stability compared to Brostrom alone
  • Provided additional proprioceptive input
Clinical Implication: Gould modification (IER augmentation) should be performed with all Brostrom repairs.
Source: Foot Ankle 1980

Level II
📚 DiGiovanni BF et al - Rehabilitation Effectiveness
Key Findings:
  • 78% of patients with chronic instability avoided surgery with rehabilitation
  • Proprioceptive training was key component
  • Peroneal strengthening improved outcomes
Clinical Implication: All patients should undergo supervised rehabilitation before surgical consideration.
Source: Foot Ankle Int 2008

Level I
📚 Maffulli N et al - Systematic Review
Key Findings:
  • Anatomic procedures superior to non-anatomic
  • Brostrom-Gould most consistent outcomes
  • Non-anatomic procedures restrict subtalar motion
Clinical Implication: Anatomic repairs (Brostrom-Gould) preferred over non-anatomic procedures.
Source: Br Med Bull 2009

Level III
📚 Cordier G et al - Arthroscopic vs Open Brostrom
Key Findings:
  • Comparable functional outcomes between approaches
  • Arthroscopic allows treatment of associated pathology
  • Similar complication rates
  • Steeper learning curve for arthroscopic technique
Clinical Implication: Arthroscopic Brostrom is a viable alternative in experienced hands.
Source: AJSM 2020

Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Recurrent Ankle Sprains in Athlete

EXAMINER

"A 25-year-old basketball player presents with recurrent left ankle 'giving way' episodes despite 6 months of physiotherapy. He reports 3-4 episodes per month during games. Examination shows positive anterior drawer test compared to the contralateral side. What is your assessment and management plan?"

EXCEPTIONAL ANSWER

This is chronic lateral ankle instability with failed conservative treatment. The positive anterior drawer indicates ATFL insufficiency.

Assessment:

  • Confirm rehabilitation was supervised and comprehensive
  • Assess for CFL involvement (talar tilt test)
  • Check hindfoot alignment (varus predisposes)
  • Evaluate for generalized laxity (Beighton score)
  • X-rays to exclude OCD, loose bodies
  • MRI to assess ligament quality and associated pathology

Management:

I would offer Brostrom-Gould repair - direct ATFL repair with inferior extensor retinaculum augmentation. This is the gold standard with 85-90% good/excellent results.

Post-operative protocol: Protected weight bearing 4-6 weeks, CAM boot, then progressive rehabilitation with proprioceptive training. Return to sport expected at 4-6 months with ankle brace.

KEY POINTS TO SCORE
Rehabilitation trial mandatory before surgery
Brostrom-Gould is gold standard surgical treatment
Assess for associated pathology (OCD, peroneal issues)
85-90% good/excellent results expected
COMMON TRAPS
✗Operating without adequate rehabilitation trial
✗Using reconstruction when Brostrom is adequate
✗Missing associated pathology (OCD, impingement)
LIKELY FOLLOW-UPS
"When would you consider reconstruction instead of repair?"
"How do you assess tissue quality intraoperatively?"
"What is your return to sport protocol?"
VIVA SCENARIOStandard

Failed Brostrom Repair

EXAMINER

"A 30-year-old female netball player presents 18 months after a Brostrom repair with recurrent instability. She has hypermobile joints (Beighton score 6/9). Stress testing shows persistent ATFL laxity. How do you approach this case?"

EXCEPTIONAL ANSWER

This is a failed Brostrom repair in a patient with generalized ligamentous laxity. The high Beighton score suggests the native tissue repair has stretched out.

Assessment:

  • Review operative notes from index procedure
  • MRI to assess remaining ligament tissue and exclude other pathology
  • Assess hindfoot alignment and exclude cavovarus
  • Evaluate peroneal function

Management:

Given the generalized laxity and failed primary repair, I would recommend anatomic reconstruction rather than revision Brostrom. My preference would be semitendinosus allograft reconstruction to avoid donor site morbidity.

Key principles:

  • Anatomic tunnel placement in fibula, talus, and calcaneus
  • Graft tensioning in neutral dorsiflexion and eversion
  • Interference screw or suture anchor fixation
  • Recreate both ATFL and CFL

Rehabilitation is longer (6-9 months) with protected weight bearing for 6 weeks.

KEY POINTS TO SCORE
Generalized laxity is a risk factor for Brostrom failure
Reconstruction indicated for failed repair, laxity, heavy athletes
Allograft reduces donor site morbidity
Longer rehabilitation than primary repair
COMMON TRAPS
✗Repeat Brostrom in patient with generalized laxity
✗Non-anatomic reconstruction (restricts subtalar motion)
✗Not addressing underlying laxity
LIKELY FOLLOW-UPS
"What graft options would you consider?"
"How does recovery differ from primary Brostrom?"
"What is your long-term prognosis for return to sport?"
VIVA SCENARIOStandard

Acute Severe Ankle Sprain

EXAMINER

"A 22-year-old soccer player sustains an inversion injury during a match. He has significant lateral swelling, ecchymosis, and tenderness over the ATFL. He cannot weight bear. Anterior drawer appears positive but is difficult to assess due to guarding. How do you manage this acute injury?"

EXCEPTIONAL ANSWER

This is an acute Grade III lateral ankle sprain with suspected complete ATFL rupture based on the mechanism, clinical findings, and apparent positive anterior drawer.

Acute Assessment:

  • Ottawa Ankle Rules to determine need for X-ray
  • X-ray to exclude fracture (lateral malleolus, base 5th metatarsal, anterior process calcaneus)
  • Clinical exam under analgesia if needed for accurate assessment
  • Assess syndesmosis (external rotation stress test)

Acute Management:

  • RICE protocol: Rest, Ice, Compression, Elevation
  • Functional bracing: Semi-rigid or lace-up ankle brace
  • Weight bearing as tolerated with crutches
  • Early mobilization: Begin ROM exercises within pain tolerance

Rehabilitation (Critical):

  • Supervised physiotherapy starting early
  • Proprioceptive training from week 2
  • Peroneal strengthening program
  • Sport-specific training before return

Prognosis: 80% of acute sprains heal well with functional treatment. 20-40% may develop chronic instability requiring further intervention.

KEY POINTS TO SCORE
Ottawa Ankle Rules guide imaging decisions
Functional treatment superior to immobilization for acute sprains
Early mobilization and rehabilitation are key
20-40% may develop chronic instability
COMMON TRAPS
✗Over-treating with prolonged immobilization
✗Missing associated fractures
✗Rushing return to sport without rehabilitation
✗Not warning about chronic instability risk
LIKELY FOLLOW-UPS
"When would you consider acute surgical repair?"
"What are the Ottawa Ankle Rules?"
"How long before return to sport?"

MCQ Practice Points

MCQ: Weakest Lateral Ligament

Q: Which lateral ankle ligament is injured first during an inversion injury?

A: The ATFL (anterior talofibular ligament) is the weakest and most commonly injured. It is injured with inversion in plantarflexion - the most common ankle sprain mechanism. Remember ACP order: Anterior → Calcaneofibular → Posterior (weakest to strongest).

MCQ: Stress Test Interpretation

Q: A patient has a positive talar tilt test but negative anterior drawer. Which ligament is injured?

A: The CFL (calcaneofibular ligament) is injured. The talar tilt test assesses CFL integrity (inversion stress in dorsiflexion), while the anterior drawer tests ATFL. This pattern is unusual as ATFL is typically injured first, so consider other pathology.

MCQ: Gould Modification

Q: What does the Gould modification add to the original Brostrom repair?

A: The Gould modification adds augmentation with the inferior extensor retinaculum (IER). The IER is advanced over the ATFL repair and anchored to the fibula, providing additional restraint and improved proprioceptive input. This is now standard with all Brostrom repairs.

MCQ: Reconstruction Indications

Q: When is anatomic reconstruction preferred over Brostrom repair for lateral ankle instability?

A: Reconstruction is indicated for: (1) Failed Brostrom repair, (2) Generalized ligamentous laxity (Beighton score greater than 4), (3) Heavy/high-demand athletes, and (4) Poor quality native tissue. These patients have higher failure rates with direct repair.

MCQ: CFL Unique Feature

Q: What is unique about the calcaneofibular ligament compared to other lateral ankle ligaments?

A: The CFL crosses two joints - both the ankle joint AND the subtalar joint. This means CFL injury affects subtalar stability as well as ankle stability. The ATFL and PTFL only cross the ankle joint.

Australian Context

Australian Guidelines and Practice

Sports Medicine Australia (SMA) Guidelines:

  • Functional treatment recommended for acute ankle sprains
  • Immobilization reserved for Grade III injuries or non-compliance
  • Supervised rehabilitation superior to unsupervised
  • Return to sport based on functional criteria, not time alone

Referral Pathways

When to Refer to Orthopaedic Surgeon:

  • Failed 3-6 months of supervised rehabilitation
  • Recurrent instability affecting function or sport
  • Associated pathology (OCD, syndesmosis injury)
  • Elite athlete with mechanical instability

When to Refer to Sports Physician:

  • Initial management of acute sprains
  • Supervision of rehabilitation programs
  • Assessment of return to sport readiness

Australian Epidemiology

  • Ankle sprains account for 15-20% of sports injuries in Australia
  • Higher incidence in netball (most common sport injury in women)
  • Football codes (AFL, NRL, Rugby Union) have significant ankle injury burden
  • Economic cost: Significant healthcare and lost productivity burden

Physiotherapy Standards (Australian Physiotherapy Association)

Rehabilitation Components:

  1. Early Phase: Pain management, protected mobility
  2. Middle Phase: ROM, strengthening, proprioception
  3. Late Phase: Sport-specific training, return to play assessment
  4. Maintenance: Ongoing proprioceptive work, bracing for sport

LATERAL ANKLE INSTABILITY

High-Yield Exam Summary

ANATOMY - ACP

  • •ATFL: Weakest, injured first, resists inversion in plantarflexion
  • •CFL: Crosses ankle AND subtalar, injured second
  • •PTFL: Strongest, only injured in dislocations
  • •ACP order = Anterior to Posterior = Weakest to Strongest

EXAMINATION

  • •Anterior drawer = ATFL (anterior translation)
  • •Talar tilt = CFL (inversion in dorsiflexion)
  • •External rotation = Syndesmosis (NOT lateral ligaments)
  • •Always compare to contralateral side

TREATMENT ALGORITHM

  • •FIRST: Rehabilitation 3-6 months (majority stabilize)
  • •SECOND: Brostrom-Gould repair (85-90% success)
  • •THIRD: Reconstruction if failed Brostrom/laxity/heavy athlete
  • •Gould modification = IER augmentation (always do this)

KEY NUMBERS

  • •20-40%: Acute sprains → chronic instability
  • •70-80%: Stabilize with rehabilitation alone
  • •85-90%: Good results with Brostrom-Gould
  • •4-6 months: Return to sport after Brostrom

Self-Assessment Quiz

Quick Stats
Reading Time92 min
Related Topics

Ankle Impingement Syndromes

Anterior Ankle Impingement

Anterior Tibial Tendon Rupture

Baxter's Nerve Entrapment