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OrthoVellum

© 2026 OrthoVellum. For educational purposes only.

Not affiliated with the Royal Australasian College of Surgeons.

Lunate Fractures

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Lunate Fractures

Comprehensive guide to lunate fractures - diagnosis, classification, association with Kienbock's disease, surgical management, and Orthopaedic exam preparation

complete
Updated: 2024-12-18
High Yield Overview

LUNATE FRACTURES

Keystone Carpal Bone | Risk of AVN | Kienbock's Disease Precursor

Under 3%Of all carpal fractures
DorsalMost common fragment location
AVN RiskUnique vascular pattern
Kienbock'sAssociated disease progression

TEISEN CLASSIFICATION (5 TYPES)

Type I
PatternVolar pole fracture
TreatmentProtected ROM, surgery if displaced
Type II
PatternChip fracture (dorsal or volar)
TreatmentImmobilization, excision if symptomatic
Type III
PatternDorsal pole fracture
TreatmentScrew fixation or excision
Type IV
PatternSagittal body fracture
TreatmentORIF with screws
Type V
PatternTransverse body fracture
TreatmentORIF, high AVN risk

Critical Must-Knows

  • Lunate is keystone of proximal carpal row - central to wrist biomechanics
  • Vulnerable blood supply - 80% receive blood from single vessel
  • Kienbock's disease may follow acute fracture or develop insidiously
  • Negative ulnar variance increases lunate loading and injury risk
  • Body fractures (Type IV/V) have highest risk of AVN and nonunion

Examiner's Pearls

  • "
    Lunate fractures are rare but frequently missed on plain radiographs
  • "
    MRI is essential for diagnosis and assessing vascularity
  • "
    Negative ulnar variance is major risk factor for Kienbock's
  • "
    Displaced body fractures require ORIF to prevent AVN

High-Yield Lunate Fracture Exam Points

The Keystone Bone

The lunate is the central link in the proximal carpal row, articulating with radius, scaphoid, triquetrum, capitate, and hamate. Its integrity is essential for normal wrist kinematics. Injury disrupts the entire wrist biomechanics.

Vascular Vulnerability

80% of lunates receive blood supply from a single dorsal vessel. This makes the bone highly susceptible to avascular necrosis (AVN) following fracture, especially body fractures that disrupt the main blood supply.

Kienbock's Association

Acute lunate fracture can progress to Kienbock's disease (lunate AVN). However, most Kienbock's cases arise from repetitive microtrauma rather than acute fracture. Both share the common endpoint of lunate collapse and carpal arthritis.

Ulnar Variance Factor

Negative ulnar variance (short ulna) concentrates force on the lunate. This is a risk factor for both acute fracture and Kienbock's disease. Ulnar lengthening may be part of treatment in select cases.

At a Glance: Lunate Fracture Management

Fracture TypeLocationManagementKey Consideration
Type I - Volar poleVolar lipImmobilization or screwAssociated with perilunate injury
Type II - ChipDorsal or volarImmobilization, excision if neededUsually good prognosis
Type III - Dorsal poleDorsal lipScrew fixation or excisionAssess for scapholunate injury
Type IV - SagittalThrough bodyORIF with headless screwsModerate AVN risk
Type V - TransverseThrough bodyORIF criticalHigh AVN risk - needs early fixation
Mnemonic

LUNATE - Key Fracture Features

L
Location central
Keystone of proximal carpal row
U
Ulnar variance matters
Negative variance increases lunate load
N
Necrosis risk
80% single vessel supply - high AVN risk
A
Associated injuries
Check for perilunate injury, SL damage
T
Teisen classification
Types I-V based on fracture pattern
E
Early MRI essential
Assess vascularity and occult fractures

Memory Hook:LUNATE - the keystone bone with Necrosis risk requiring early Assessment

Mnemonic

KIEN - Kienbock's Disease Features

K
Keystone collapse
Progressive lunate collapse and fragmentation
I
Insidious onset
Often no acute injury - repetitive microtrauma
E
Early MRI changes
Signal changes precede X-ray findings
N
Negative ulnar variance
Major risk factor - increased lunate load

Memory Hook:KIEN-bock's develops Insidiously with Negative ulnar variance

Mnemonic

BOCK - Management Algorithm

B
Bone vascularity
MRI to assess blood supply - critical for prognosis
O
Offload the lunate
Radial shortening or ulnar lengthening
C
Core decompression
For early disease - preserves bone structure
K
Kinematic fusion
For advanced disease - limited intercarpal fusion

Memory Hook:BOCK management: assess Bone vascularity, Offload, Core decompression, Kinematic fusion

Overview and Epidemiology

Definition

Lunate fractures are fractures of the lunate carpal bone, the central "keystone" of the proximal carpal row. These fractures range from minor avulsion injuries to complete body fractures with significant implications for wrist function.

Epidemiology

  • Incidence: Under 3% of all carpal fractures (very rare)
  • Age distribution: Typically young to middle-aged adults
  • Gender: Male predominance in acute trauma
  • Mechanism: High-energy axial loading or hyperextension

Relationship to Kienbock's Disease

Kienbock's disease (lunate avascular necrosis) may develop:

  • Following acute lunate fracture
  • From repetitive microtrauma without acute fracture
  • Due to inherent vascular compromise

The relationship between acute fracture and Kienbock's is complex, with both potentially representing points on a spectrum of lunate injury.

Clinical Significance

The lunate occupies a critical position in the wrist:

  • Transmits approximately 50% of carpal load
  • Central to proximal row kinematics
  • Articulates with multiple carpal and forearm bones
  • Injury affects entire wrist function

Understanding the lunate's central role is essential for appreciating the significance of these fractures.

Anatomy/Biomechanics

Osseous Anatomy

Shape and Configuration

  • Crescent-shaped when viewed laterally (lunate = moon-shaped)
  • Volar horn: Projects palmarly, attachment for radiocarpal ligaments
  • Dorsal horn: Smaller, attachment for dorsal intercarpal ligament
  • Proximal surface: Convex, articulates with lunate facet of radius

Articular Surfaces

  • Proximal: Articulates with radius (70%) and TFCC (30%)
  • Radial: Articulates with scaphoid
  • Ulnar: Articulates with triquetrum
  • Distal: Articulates with capitate and often hamate (Type II lunate)

Type I vs Type II Lunates

  • Type I (35%): Single distal facet for capitate only
  • Type II (65%): Two distal facets - capitate and hamate
  • Type II may have altered biomechanics affecting injury patterns

Vascular Anatomy

Lunate bone anatomy and vascular supply patterns showing dorsal and volar vessel entry points
Click to expand
Lunate anatomy demonstrating vascular supply patterns - the majority have single vessel (I-pattern) supply making them susceptible to AVNCredit: Wikimedia Commons (CC BY-SA)

Blood Supply Pattern

The lunate receives blood supply through two main patterns:

Y-Pattern (20%)

  • Dorsal and volar vessels anastomose within bone
  • More protected from AVN

I-Pattern (80%)

  • Single dominant vessel (usually dorsal)
  • No significant intraosseous anastomosis
  • High risk of AVN with any fracture disrupting this vessel

Vessel Entry Points

  • Dorsal: 1-3 vessels entering dorsal pole
  • Volar: 1-2 vessels entering volar pole
  • No intraosseous penetration from radial or ulnar surfaces

Vascular Pattern Determines Prognosis

The I-pattern (80% of lunates) with single dominant vessel explains why body fractures (Type IV/V) carry such high AVN risk. Fracture line disrupts the only blood supply to a significant portion of the bone.

Biomechanics

Load Transmission

  • Lunate transmits approximately 50% of axial wrist load
  • Forces concentrated when ulnar variance is negative
  • Normal lunate tilts volarly 10-15 degrees

Carpal Kinematics

  • Part of proximal row "intercalated segment"
  • Moves with scaphoid and triquetrum as functional unit
  • Disruption leads to carpal instability patterns (DISI/VISI)

Ulnar Variance Impact

  • Negative variance: Increased lunate loading
  • Positive variance: Load shared with TFCC
  • Average: Neutral (0 +/- 1mm)

Understanding the anatomy explains why certain fracture patterns carry higher risks.

Classification Systems

Teisen Classification

The Teisen classification is the most widely used system for acute lunate fractures:

Type I - Volar Pole Fracture

  • Avulsion of volar horn by short radiolunate ligament
  • Often associated with perilunate injury pattern
  • Usually small fragment - may not require fixation
  • Treatment: Immobilization if undisplaced, surgery if part of perilunate

Type II - Chip Fracture

  • Small dorsal or volar avulsion fragment
  • Usually minimal clinical significance
  • Most common acute lunate fracture pattern
  • Treatment: Cast immobilization, excision if persistently symptomatic

Type III - Dorsal Pole Fracture

  • Larger dorsal horn fragment
  • May involve scapholunate ligament attachment
  • Risk of dorsal intercalated segment instability
  • Treatment: Screw fixation if large, excision if small

Type IV - Sagittal Body Fracture

  • Sagittal split through lunate body
  • Moderate risk of AVN
  • Disrupts articular surfaces
  • Treatment: ORIF with headless compression screws

Type V - Transverse Body Fracture

  • Coronal/transverse split through body
  • Highest AVN risk (disrupts dominant vessel)
  • Poor prognosis without treatment
  • Treatment: Urgent ORIF, close monitoring for AVN

This classification guides prognosis and treatment selection.

Lichtman Classification (Kienbock's Disease)

MRI and radiograph showing progressive stages of Kienbock's disease from early AVN to lunate collapse
Click to expand
Lichtman staging of Kienbock's disease - progression from early MRI changes (Stage I) through lunate collapse and arthritis (Stage IV)Credit: PMC (CC BY)

While not specific to acute fractures, this staging is essential for managing lunate pathology:

Stage I

  • Normal radiographs
  • MRI shows decreased T1 signal
  • Bone intact structurally

Stage II

  • Sclerosis visible on radiographs
  • No collapse
  • Bone scan positive

Stage IIIA

  • Lunate collapse begins
  • Carpal height maintained
  • No fixed scaphoid rotation

Stage IIIB

  • Lunate collapse with carpal changes
  • Scaphoid rotation (ring sign)
  • Proximal capitate migration

Stage IV

  • Secondary degenerative arthritis
  • Radiocarpal and intercarpal involvement
  • End-stage disease

Understanding Kienbock's staging helps predict progression after acute lunate injury.

Gelberman Vascular Classification

This anatomical classification describes blood supply pattern:

Y-Pattern (20%)

  • Multiple entering vessels
  • Intraosseous anastomoses
  • Lower AVN risk

X-Pattern

  • Variant with crossed vessels
  • Moderate anastomosis
  • Intermediate risk

I-Pattern (80%)

  • Single dominant vessel
  • No significant anastomosis
  • Highest AVN risk

Vascular pattern cannot be determined preoperatively but explains the high overall AVN rate after body fractures.

Classification Comparison

Teisen TypeLocationAVN RiskTypical Treatment
Type IVolar poleLowImmobilization or ligament repair
Type IIChip/avulsionVery lowCast, excision if symptomatic
Type IIIDorsal poleLow-moderateScrew fixation or excision
Type IVSagittal bodyModerate-highORIF with screws
Type VTransverse bodyHighUrgent ORIF

Classification guides treatment urgency and prognosis discussion.

Clinical Assessment

History

Mechanism of Injury

  • High-energy fall: FOOSH with axial loading
  • Direct trauma: Rare due to protected position
  • Sports injury: Gymnastics, contact sports
  • Insidious onset: May indicate developing Kienbock's rather than acute fracture

Key History Points

  • Exact mechanism and energy of injury
  • Prior wrist symptoms (may indicate pre-existing Kienbock's)
  • Occupational demands (manual labor, vibration exposure)
  • Hand dominance
  • Duration of symptoms

Physical Examination

Inspection

  • Swelling may be subtle due to deep location
  • No obvious deformity unless associated carpal injury
  • Compare to contralateral wrist

Palpation

  • Lunate fossa tenderness: Palpate with wrist slightly flexed
  • Dorsal lunate: Tender with wrist extended
  • May be difficult to localize with surrounding swelling

Range of Motion

  • Limited wrist flexion and extension
  • Pain with forearm rotation
  • Grip weakness

Neurovascular Assessment

  • Usually preserved
  • Check median nerve (carpal tunnel with swelling)
  • Document baseline for comparison

Special Tests

Watson Test (Scaphoid Shift)

  • Assess for associated scapholunate injury
  • Often positive with perilunate pattern injuries

Ballottement Test

  • Lunotriquetral stability
  • Assess for associated LT injury

Grind Test

  • Axial load with rotation
  • Positive if pain reproduced

Carpal Tunnel Assessment

  • Phalen's and Tinel's tests
  • May be positive with acute swelling

Clinical examination is often non-specific; imaging is essential for diagnosis.

Investigations

Plain Radiographs

Lateral radiograph showing lunate position and carpal alignment assessment
Click to expand
Radiographic assessment of lunate fracture - lateral view is essential for evaluating lunate position and detecting DISI/VISI patternsCredit: Radiopaedia (CC BY-NC-SA)

Standard Views

  • PA view: May show fracture line, sclerosis, or collapse
  • Lateral view: Assess lunate position (DISI/VISI), dorsal fractures
  • Scaphoid view: Additional perspective on carpal relationships

Radiographic Signs of Acute Fracture

  • Fracture line (often subtle or absent)
  • Slight density change
  • Associated carpal malalignment

Signs of Kienbock's Disease

  • Increased lunate density (sclerosis)
  • Loss of carpal height
  • Lunate collapse and fragmentation
  • Secondary arthritis (late)

Ulnar Variance Measurement

  • Measure on neutral rotation PA view
  • Negative variance (short ulna) is risk factor
  • Compare to contralateral if available

CT Scanning

Indications

  • Characterize fracture pattern for surgical planning
  • Assess for occult fracture not seen on X-ray
  • Evaluate associated carpal injuries

Key CT Findings

  • Fracture line orientation (sagittal vs transverse)
  • Fragment size and displacement
  • Articular surface involvement
  • Comminution assessment

MRI

Critical Role in Lunate Pathology

MRI is essential for:

  • Detecting occult fractures missed on X-ray
  • Assessing lunate vascularity
  • Differentiating acute fracture from Kienbock's
  • Evaluating ligamentous injuries

Signal Changes

  • Normal lunate: Isointense to other carpal bones
  • Early ischemia: Decreased T1 signal
  • Established AVN: Low T1 and T2 signal
  • Revascularization: Mixed signals

Gadolinium Enhancement

  • Enhanced signal suggests preserved vascularity
  • Absence of enhancement indicates AVN
  • Helps predict prognosis

Bone Scan

Limited Role

  • Sensitive but not specific
  • Hot lunate in any pathology
  • Superseded by MRI for most indications

Investigations are summarized in the table below.

Imaging Modalities for Lunate Fractures

ModalityPrimary RoleAdvantagesLimitations
Plain X-rayInitial screeningAvailable, low costMisses early/occult fractures
CT scanFracture characterizationBone detail, 3D planningNo vascularity assessment
MRIVascularity, occult injurySoft tissue, blood supplyCost, availability
Bone scanRarely neededSensitive for pathologyNon-specific

MRI is the key investigation for determining treatment and prognosis.

Management Algorithm

📊 Management Algorithm
lunate fractures management algorithm
Click to expand
Management algorithm for lunate fracturesCredit: OrthoVellum

Non-Operative Management

Indications

  • Type I (volar pole) - undisplaced
  • Type II (chip fractures) - most cases
  • Type III (dorsal pole) - small, undisplaced fragments
  • Elderly, low-demand patients with body fractures

Protocol

Immobilization

  • Short arm cast or splint
  • Wrist in neutral position
  • Include thumb if scaphoid concern
  • Duration: 6-8 weeks minimum

Follow-Up

  • Week 2: Clinical review, check cast
  • Week 6: Repeat radiographs
  • Week 8-12: MRI if symptoms persist
  • Monitor for AVN development (may take months)

Transition to Rehabilitation

  • Begin ROM when clinically healed
  • Progressive strengthening
  • Monitor for late complications

Expected Outcomes

  • Type II fractures: Excellent prognosis
  • Type I/III: Good with proper immobilization
  • Body fractures: Higher failure rate with non-operative treatment

Non-operative management requires vigilant monitoring for AVN.

Surgical Indications

Absolute Indications

  • Type V (transverse body) fractures
  • Displaced Type IV (sagittal) fractures
  • Associated perilunate injury
  • Symptomatic nonunion

Relative Indications

  • Large displaced Type III fragments
  • Type IV with minimal displacement in high-demand patient
  • Failed non-operative treatment
  • Persistently symptomatic chip fracture

Patient Factors

Favoring Surgery

  • Young, active patient
  • High occupational demands
  • Body fracture with displacement
  • MRI showing intact vascularity

Favoring Non-Operative

  • Elderly, low-demand patient
  • Significant comorbidities
  • Small avulsion fragments
  • No displacement

The decision balances fracture pattern with patient factors and AVN risk.

ORIF Techniques

Surgical Goals

  1. Anatomic reduction of articular surface
  2. Stable fixation preserving blood supply
  3. Early mobilization to prevent stiffness
  4. Minimize further vascular compromise

Surgical Approaches

Dorsal Approach

  • Indication: Dorsal pole fractures, body fractures
  • Through 3rd/4th extensor compartments
  • Capsulotomy with ligament preservation
  • Best visualization of dorsal lunate

Volar Approach

  • Indication: Volar pole fractures
  • Between FCR and radial artery
  • Capsulotomy preserving radiocarpal ligaments
  • Access to volar horn

Fixation Options

Headless Compression Screws

  • Indication: Body fractures (Type IV/V)
  • 2.0-2.4mm diameter
  • Countersunk beneath cartilage
  • Provides compression across fracture

K-wire Fixation

  • Indication: Small fragments, provisional fixation
  • 1.1-1.25mm wires
  • May require supplemental cast

Fragment Excision

  • Indication: Small chip fractures (Type II)
  • Persistently symptomatic despite immobilization
  • Remove fragment, debride bed

Technical Pearls

  • Minimize soft tissue stripping to preserve blood supply
  • Use fluoroscopy to confirm screw position
  • Consider arthroscopic assistance for visualization
  • Avoid compression that could compromise vessels

Surgical technique prioritizes gentle handling to preserve blood supply.

Management decisions must consider the high risk of AVN with body fractures.

Surgical Technique

Dorsal Approach for Lunate Fracture ORIF

Patient Positioning

  • Supine with arm table
  • Tourniquet on upper arm
  • Consider traction tower for visualization

Incision and Exposure

Skin Incision

  • Dorsal longitudinal over wrist
  • Centered on Lister's tubercle
  • 4-5 cm length

Deep Dissection

  1. Incise retinaculum between 3rd and 4th compartments
  2. Protect EPL tendon (retract radially)
  3. Retract EDC ulnarly
  4. Capsulotomy - ligament-sparing technique preferred

Fracture Reduction

Visualization

  • Flex wrist to expose lunate dorsum
  • Identify fracture pattern
  • Assess vascularity (bleeding from bone)

Reduction Technique

  1. Gentle manipulation of fragments
  2. Provisional K-wire fixation
  3. Confirm reduction with fluoroscopy
  4. Assess articular surface congruity

Fixation

Headless Compression Screw

  1. Select appropriate screw size (2.0-2.4mm)
  2. Start at dorsal cortex
  3. Aim for volar cortex (or far fragment)
  4. Countersink beneath cartilage
  5. Confirm position on multiple fluoroscopy views

Supplemental Fixation

  • K-wire for rotational control if needed
  • Second screw for large body fractures

Closure

  • Repair capsule meticulously
  • Close retinaculum loosely
  • Standard skin closure
  • Splint in neutral position

Gentle handling throughout preserves the tenuous blood supply.

Volar Approach for Lunate Fracture ORIF

Indications

  • Volar pole fractures (Type I)
  • Combined volar/dorsal approach for body fractures

Patient Positioning

  • Supine with arm table
  • Arm supinated
  • Tourniquet applied

Incision and Exposure

Skin Incision

  • Over FCR tendon
  • From wrist crease extending 4 cm proximally

Deep Dissection

  1. Incise FCR sheath, retract tendon radially
  2. Identify and protect radial artery
  3. Divide transverse carpal ligament if needed
  4. Capsulotomy between radioscapholunate and long radiolunate ligaments

Fracture Reduction

  • Direct visualization of volar pole
  • Reduce fragment anatomically
  • Provisional K-wire fixation

Fixation

  • Small screws or K-wires for volar pole
  • Consider suture anchor if ligament avulsion
  • Fluoroscopy to confirm position

Closure

  • Repair capsule
  • Standard layered closure
  • Volar splint

The volar approach provides excellent access to volar pole pathology.

Postoperative Protocol

Immediate Postoperative (Day 0-14)

Immobilization

  • Volar or dorsal splint in neutral
  • Immediate finger motion
  • Elevate above heart level

Wound Care

  • First dressing change 48-72 hours
  • Monitor for infection
  • K-wire site care if applicable

Early Phase (Weeks 2-6)

Week 2

  • Suture removal
  • Transition to removable splint
  • Begin gentle active ROM

Week 4

  • Progress ROM exercises
  • Continue splint between exercises

Week 6

  • Repeat radiographs
  • Consider MRI to assess healing and vascularity
  • K-wire removal if used

Rehabilitation Phase (Weeks 6-12)

Week 6-8

  • Progressive ROM
  • Gentle grip strengthening
  • Discontinue splint if stable

Week 8-12

  • Progressive strengthening
  • Functional activities
  • Work conditioning as needed

Long-Term Monitoring

  • Monitor for AVN development (may be delayed)
  • Repeat MRI at 6 months if any concerns
  • Annual clinical review for 2 years

Close monitoring for AVN is essential in the postoperative period.

Surgical technique must minimize further vascular compromise.

Complications

Intraoperative Complications

Iatrogenic Fracture

  • Lunate is small and fragile
  • Risk during screw insertion
  • Prevention: Careful technique, appropriate implant size
  • Management: Additional fixation if occurs

Screw Malposition

  • Joint penetration causes arthritis
  • Prevention: Multiple fluoroscopy views
  • Management: Revise if intra-articular

Vascular Injury

  • Further compromise to tenuous blood supply
  • Prevention: Minimize soft tissue stripping
  • Cannot be assessed intraoperatively

Early Complications

Wound Complications

  • Infection: Unusual but problematic for this small bone
  • Dehiscence: May expose hardware
  • Management: IV antibiotics, debridement if deep

Hardware Problems

  • Screw prominence: May irritate extensor tendons
  • K-wire migration: Remove early
  • Management: Hardware removal once healed

Carpal Tunnel Syndrome

  • May develop with swelling
  • Treatment: Splinting, elevation, decompression if persistent

Late Complications

Avascular Necrosis (Most Critical)

  • Occurs in up to 50% of body fractures
  • May present months after injury
  • Progressive pain, decreased motion
  • Treatment depends on stage (see Kienbock's management)

Nonunion

  • More common with body fractures
  • Related to AVN in many cases
  • Treatment: Bone grafting, revision fixation

Post-Traumatic Arthritis

  • Consequence of AVN or articular malreduction
  • Progressive wrist pain and stiffness
  • Treatment: Activity modification to fusion

Carpal Instability

  • DISI or VISI pattern may develop
  • Related to associated ligament injury
  • May require carpal fusion

Complication Management Summary

ComplicationRisk FactorsPreventionManagement
AVNBody fractures, single vessel supplyEarly fixation, gentle techniqueStage-dependent: core decompression to fusion
NonunionAVN, inadequate fixationStable fixation, bone graftRevision fixation, vascularized graft
ArthritisMalreduction, AVNAnatomic reductionArthrodesis options
Hardware problemsProminent implantsCountersink screwsHardware removal

AVN is the dominant concern in lunate fracture management.

Postoperative Care

Immediate Postoperative Care (0-2 Weeks)

Immobilization

  • Splint in neutral wrist position
  • Allow immediate finger motion
  • Maintain elevation

Pain Management

  • Multimodal analgesia
  • Ice application
  • Elevate extremity

Monitoring

  • Watch for signs of infection
  • Neurovascular checks
  • Swelling assessment

Wound Care

Dressing Changes

  • First change at 48-72 hours
  • Assess wound healing
  • K-wire site cleaning if applicable

Suture Removal

  • 10-14 days postoperatively
  • Apply steri-strips for support

Rehabilitation Phases

Phase 1: Protection (Weeks 0-2)

  • Active finger motion
  • Shoulder and elbow ROM
  • Edema control with elevation and compression

Phase 2: Early Motion (Weeks 2-6)

  • Begin gentle wrist ROM
  • Removable splint between exercises
  • Continue edema management
  • Hand therapy referral

Phase 3: Progressive Loading (Weeks 6-12)

  • Progress to functional activities
  • Light grip strengthening
  • Discontinue splint (usually at week 6)
  • Normal ADLs

Phase 4: Return to Function (Weeks 12+)

  • Progressive strengthening
  • Sport-specific activities
  • Work conditioning
  • Full recovery may take 6-12 months

Follow-Up Schedule

TimepointAssessmentImaging
Week 2Wound checkOptional
Week 6ROM, healingRadiographs
Week 12FunctionRadiographs
Month 6AVN surveillanceMRI recommended
Year 1Long-term outcomeAs needed
Year 2Final reviewIf symptomatic

MRI surveillance for AVN is critical, especially for body fractures.

Outcomes and Prognosis

Functional Outcomes by Fracture Type

Type I-III (Pole/Chip Fractures)

  • Generally good to excellent outcomes
  • Full ROM recovery expected
  • Return to previous activities typical
  • Low AVN risk

Type IV (Sagittal Body)

  • Moderate outcomes
  • Some motion loss common
  • AVN in 30-40%
  • May require secondary procedures

Type V (Transverse Body)

  • Most guarded prognosis
  • AVN in up to 50%
  • Significant motion loss common
  • Often progresses to Kienbock's

Prognostic Factors

Favorable Factors

  • Pole or chip fractures
  • Undisplaced pattern
  • Early diagnosis and treatment
  • MRI showing preserved vascularity
  • Neutral or positive ulnar variance

Unfavorable Factors

  • Body fractures (Type IV/V)
  • Delayed diagnosis
  • Associated carpal injuries
  • MRI showing avascularity
  • Negative ulnar variance

Natural History of Untreated Fractures

Chip Fractures

  • May remain asymptomatic
  • Occasional persistent pain
  • Rarely progress to AVN

Body Fractures

  • High rate of nonunion
  • Progressive AVN development
  • Eventual carpal collapse
  • Secondary arthritis

Long-Term Outcomes

Without AVN

  • Good grip strength recovery (80-90% of contralateral)
  • Near-normal ROM
  • Return to previous occupation and sport

With AVN (Kienbock's Development)

  • Progressive deterioration
  • May require salvage procedures
  • Outcomes depend on stage at intervention:
    • Stage I-II: Core decompression or revascularization may preserve function
    • Stage IIIA: Motion-preserving procedures possible
    • Stage IIIB-IV: Limited wrist fusion or arthroplasty

Prognosis is largely determined by development or avoidance of AVN.

Evidence Base

Level IV
📚 Gelberman RH et al. The Vascularity of the Lunate Bone and Kienbock's Disease
Key Findings:
  • 80% of lunates have I-pattern (single vessel) blood supply
  • Y-pattern (20%) has intraosseous anastomoses
  • Vascular pattern determines AVN risk after fracture
  • Dorsal vessels are predominant blood supply
Clinical Implication: Understanding vascular anatomy explains the high AVN rate after body fractures and guides surgical technique to minimize further vascular compromise.
Source: J Hand Surg Am 1980

Level IV
📚 Teisen H, Hjarbaek J. Classification of Fresh Fractures of the Lunate
Key Findings:
  • Proposed 5-type classification for acute lunate fractures
  • Body fractures (Type IV/V) have highest complication rate
  • Pole fractures generally have good prognosis
  • Classification guides treatment selection
Clinical Implication: The Teisen classification remains the standard for acute lunate fracture description and helps predict prognosis.
Source: J Hand Surg Br 1988

Level V
📚 Lichtman DM et al. Kienbock's Disease: Current Concepts
Key Findings:
  • Negative ulnar variance is major risk factor
  • Early MRI changes precede radiographic findings
  • Stage-dependent treatment algorithm
  • Motion-preserving procedures preferred when possible
Clinical Implication: Understanding Kienbock's disease progression helps manage lunate fractures that may progress to AVN.
Source: J Hand Surg Am 2016

Level IV
📚 Kristensen SS et al. Fractures of the Lunate Bone
Key Findings:
  • Acute lunate fractures are rare (under 3% of carpal fractures)
  • High association with perilunate injury pattern
  • MRI essential for diagnosis of occult fractures
  • Body fractures require surgical fixation
Clinical Implication: Awareness of lunate fracture rarity and associated injuries helps avoid missed diagnosis.
Source: J Hand Surg Br 1987

Level IV
📚 Allan CH et al. Outcomes After Lunate Fracture Fixation
Key Findings:
  • ORIF of body fractures reduces AVN rate
  • Early fixation (under 2 weeks) has better outcomes
  • Headless compression screws provide stable fixation
  • Long-term surveillance for AVN is essential
Clinical Implication: Early surgical fixation of body fractures may reduce AVN development and improve outcomes.
Source: Hand Clin 2019

The evidence supports early fixation for body fractures and long-term AVN surveillance.

Viva Scenarios

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Acute Lunate Fracture Evaluation

EXAMINER

"A 28-year-old manual laborer presents 2 days after a fall from scaffolding. He has wrist pain, and X-rays show a possible lunate abnormality. How do you evaluate this injury?"

EXCEPTIONAL ANSWER
This is a young patient with high-energy wrist trauma requiring thorough evaluation. **Initial Assessment:** I would first confirm the mechanism - a fall from scaffolding suggests significant axial loading which can cause occult carpal injuries. I would examine the entire upper limb for associated injuries and document neurovascular status, particularly median nerve function. **Plain Radiograph Analysis:** On the PA view, I would assess lunate contour and density, looking for fracture lines or sclerosis. On the lateral, I would check lunate position (DISI or VISI pattern would suggest associated ligamentous injury). I would also measure ulnar variance, as negative variance increases lunate loading. **Advanced Imaging:** Given the clinical suspicion and high-energy mechanism, I would obtain a CT scan to characterize any fracture pattern. More importantly, I would obtain an MRI to assess for occult fracture not visible on CT and to evaluate lunate vascularity with T1-weighted sequences. Decreased T1 signal would indicate early vascular compromise. **Classification:** Using the Teisen classification, I would categorize the fracture as: - Type I-III (pole or chip): Generally good prognosis - Type IV (sagittal body): Moderate AVN risk - Type V (transverse body): High AVN risk requiring surgical fixation **Treatment Decision:** If this is a body fracture (Type IV or V), I would recommend surgical fixation with headless compression screws to optimize reduction and potentially reduce AVN risk. Pole or chip fractures can often be managed non-operatively with cast immobilization.
KEY POINTS TO SCORE
High-energy mechanism warrants thorough evaluation
MRI is essential for vascularity assessment
Teisen classification guides prognosis and treatment
Body fractures require surgical consideration
COMMON TRAPS
✗Missing occult fracture by relying only on X-ray
✗Not assessing ulnar variance as risk factor
✗Failing to evaluate for associated perilunate injury
LIKELY FOLLOW-UPS
"What would you do if the MRI shows decreased T1 signal in the lunate?"
"How does ulnar variance affect your treatment recommendation?"
"What associated injuries would you specifically look for?"
VIVA SCENARIOChallenging

Lunate Fracture with AVN Risk

EXAMINER

"CT and MRI confirm a Type V transverse lunate body fracture in a 35-year-old office worker. MRI shows intact signal on T1 sequences. How do you counsel this patient and plan treatment?"

EXCEPTIONAL ANSWER
This is a high-risk lunate fracture pattern that requires careful management and counseling. **Patient Counseling:** I would explain that a transverse body fracture carries significant risk of avascular necrosis (AVN) - up to 50% without treatment. The good news is that current MRI shows intact blood supply, suggesting we have a window to optimize outcomes with treatment. **Treatment Recommendation:** I would strongly recommend surgical fixation for this Type V fracture. My goals are to achieve anatomic reduction, provide stable fixation with a headless compression screw, and minimize further vascular compromise through gentle surgical technique. **Surgical Approach:** I would use a dorsal approach through the 3rd and 4th extensor compartments. I would perform a ligament-sparing capsulotomy to preserve as much soft tissue blood supply as possible. Using fluoroscopic guidance, I would reduce the fracture and fix it with a 2.0-2.4mm headless compression screw, ensuring it is countersunk beneath the cartilage. **Postoperative Protocol:** Splinting for 2 weeks, then protected ROM in a removable splint. I would obtain repeat MRI at 3-6 months to monitor for AVN development, even if clinical progress seems satisfactory. **Prognosis Discussion:** Despite optimal treatment, there remains a significant AVN risk. If AVN develops, treatment would be stage-dependent - from core decompression for early disease to partial wrist fusion for advanced collapse. I would ensure the patient understands the need for long-term surveillance.
KEY POINTS TO SCORE
Type V fractures have up to 50% AVN rate
Preserved MRI signal is favorable but not protective
ORIF with gentle technique to preserve blood supply
Long-term MRI surveillance is essential
COMMON TRAPS
✗Underestimating AVN risk with reassuring MRI
✗Aggressive soft tissue dissection during surgery
✗Not planning for MRI surveillance postoperatively
LIKELY FOLLOW-UPS
"What is your surgical approach and fixation technique?"
"How would you manage if AVN develops despite optimal treatment?"
"What is your postoperative surveillance protocol?"
VIVA SCENARIOChallenging

Kienbock's Disease Management

EXAMINER

"A patient you treated for a lunate fracture 18 months ago returns with progressive wrist pain. MRI shows Stage IIIA Kienbock's disease. How do you manage this complication?"

EXCEPTIONAL ANSWER
This patient has developed avascular necrosis progressing to Kienbock's disease, which is the most significant complication of lunate fractures. **Stage IIIA Assessment:** Stage IIIA Kienbock's indicates lunate collapse has begun, but carpal height is maintained and the scaphoid has not developed fixed rotation. This is an important stage because motion-preserving procedures may still be possible. **Imaging Review:** I would obtain current radiographs to confirm staging and assess for any progression. CT can help evaluate the degree of lunate fragmentation. MRI would show the extent of necrosis and any revascularization attempts. **Treatment Options:** For Stage IIIA, I would discuss several options: **1. Ulnar Lengthening or Radial Shortening:** If the patient has negative ulnar variance (which is common in Kienbock's), unloading the lunate by correcting variance may halt progression. I would favor radial shortening osteotomy as it is technically more reliable. **2. Core Decompression:** If there is any residual vascularity on MRI, core decompression with or without bone grafting may promote revascularization. However, in established Stage III disease, success rates are lower. **3. Partial Carpal Fusion:** If the lunate is severely fragmented, a limited intercarpal fusion (scaphocapitate fusion or scaphotrapeziotrapezoid fusion) can offload the lunate while preserving some motion. **4. Proximal Row Carpectomy:** If the capitate head cartilage is intact, PRC can provide reasonable motion and pain relief, but I would reserve this for more advanced disease or failed treatments. **My Recommendation:** For this Stage IIIA with negative ulnar variance, I would recommend radial shortening osteotomy as the first-line treatment, combined with core decompression of the lunate. This addresses the mechanical overload while attempting to preserve the bone.
KEY POINTS TO SCORE
Stage IIIA allows motion-preserving procedures
Correct ulnar variance to unload the lunate
Radial shortening preferred over ulnar lengthening
Multiple options exist based on patient factors
COMMON TRAPS
✗Proceeding directly to fusion without considering unloading
✗Not assessing ulnar variance as treatment target
✗Missing the window for motion-preserving surgery
LIKELY FOLLOW-UPS
"What if this patient has neutral ulnar variance?"
"When would you consider proximal row carpectomy?"
"What is the expected outcome with radial shortening osteotomy?"

MCQ Practice Points

Vascular Anatomy

Q: What percentage of lunates have a single-vessel (I-pattern) blood supply? A: Approximately 80% of lunates receive blood supply from a single dominant vessel with no significant intraosseous anastomosis. This explains the high rate of AVN following body fractures.

AVN Risk by Fracture Type

Q: Which Teisen classification type has the highest risk of avascular necrosis? A: Type V (transverse body) fractures have the highest AVN risk, up to 50%. The transverse fracture line disrupts the dominant blood vessel entering from the dorsal or volar surface.

Ulnar Variance Association

Q: What ulnar variance pattern is associated with increased lunate fracture and Kienbock's disease risk? A: Negative ulnar variance (short ulna relative to radius) concentrates axial load on the lunate. This is a major risk factor for both acute fracture and progressive AVN.

Keystone Position

Q: What percentage of wrist axial load is transmitted through the lunate? A: Approximately 50% of axial load passes through the lunate, making it the "keystone" of the proximal carpal row. This explains why lunate injury significantly affects overall wrist function.

MRI Findings

Q: What MRI finding indicates early avascular necrosis of the lunate? A: Decreased T1 signal relative to other carpal bones indicates early AVN, as fat in the bone marrow is replaced by edema or necrotic tissue. T2 signal may be variable depending on the stage.

Kienbock's Staging

Q: What distinguishes Stage IIIA from Stage IIIB Kienbock's disease? A: Stage IIIA has lunate collapse but maintained carpal height and no fixed scaphoid rotation. Stage IIIB shows scaphoid rotation (ring sign on X-ray) and proximal capitate migration, indicating more advanced carpal collapse.

Understanding these key concepts will help with exam success.

Australian Context

Lunate fractures are uncommon injuries managed across Australian trauma centers. High-energy mechanisms including workplace falls in construction and mining industries, as well as motor vehicle and motorcycle accidents, account for most acute presentations.

The Australian orthopaedic community follows international consensus regarding the importance of MRI for diagnosis and vascularity assessment. Most major centers have ready access to MRI, though regional patients may experience delays that could impact early intervention.

Kienbock's disease management in Australia aligns with international practice, with radial shortening osteotomy being the preferred unloading procedure when indicated. Access to hand surgery subspecialists with experience in wrist reconstruction is available in capital cities, with telehealth consultation supporting regional practitioners.

Rehabilitation services through hand therapy are widely available in metropolitan areas. Workers compensation pathways support appropriate treatment and rehabilitation for occupationally-acquired injuries.

The Australian healthcare system provides comprehensive coverage for lunate fracture treatment through both public and private pathways, ensuring access to appropriate surgical care and long-term surveillance for AVN development.

Lunate Fractures - Rapid Recall

High-Yield Exam Summary

Definition & Key Concepts

  • •Keystone of proximal carpal row
  • •Under 3% of all carpal fractures
  • •80% have single-vessel blood supply
  • •50% of wrist axial load through lunate
  • •Negative ulnar variance increases risk

Teisen Classification

  • •Type I: Volar pole fracture
  • •Type II: Chip fracture (dorsal/volar)
  • •Type III: Dorsal pole fracture
  • •Type IV: Sagittal body fracture
  • •Type V: Transverse body - highest AVN risk

Imaging Strategy

  • •X-ray: May miss fracture, assess ulnar variance
  • •CT: Characterize fracture pattern
  • •MRI: Essential for vascularity assessment
  • •Decreased T1 signal = early AVN

Treatment Algorithm

  • •Type I-III: Often non-operative (cast 6-8 weeks)
  • •Type IV/V: ORIF with headless screws
  • •Dorsal approach for most fractures
  • •Gentle technique to preserve blood supply

AVN & Kienbock's Management

  • •Stage I-II: Core decompression, revascularization
  • •Stage IIIA: Radial shortening, limited fusion
  • •Stage IIIB: PRC or limited fusion
  • •Stage IV: Salvage fusion or arthroplasty

Complications & Outcomes

  • •AVN: Up to 50% in Type V fractures
  • •Nonunion: Related to AVN in many cases
  • •Long-term MRI surveillance essential
  • •Prognosis depends on AVN development
Quick Stats
Reading Time99 min
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