OS TRIGONUM SYNDROME
Accessory Ossicle | Posterior Impingement | Dancers
Related Anatomy
Critical Must-Knows
- Os trigonum = unfused lateral tubercle of posterior talus
- Present in 7-14% of population (bilateral in 50%)
- Symptomatic in dancers and athletes requiring plantarflexion
- Posterior impingement with forced plantarflexion
- Endoscopic excision is treatment of choice
Examiner's Pearls
- "Lateral X-ray shows accessory ossicle behind talus
- "FHL tenosynovitis may be associated
- "Posterior impingement test positive
- "Injection both diagnostic and therapeutic

Critical Os Trigonum Exam Points
Anatomy
Os trigonum = secondary ossification center of lateral talar process that fails to fuse. Appears at 8-13 years, usually fuses at 1 year. Unfused ossicle in 7-14%.
Mechanism
Forced or repetitive plantarflexion compresses os trigonum between tibia and calcaneus. Bone marrow edema develops. Dancers (en pointe) and footballers (kicking) are classic.
FHL
The flexor hallucis longus runs adjacent to the os trigonum. May develop tenosynovitis as part of syndrome. Causes triggering of great toe ("dancer's tendinitis").
Treatment
Conservative: Rest, injection, activity modification. Surgical: Endoscopic or open excision of os trigonum. Address FHL if involved. Excellent return to sport.
OT-DANCEOs Trigonum Features
Memory Hook:OT-DANCE = Os Trigonum in DANCErs - Nutcracker, Conservative, Excision!
Overview and Anatomy
Os trigonum is an accessory ossicle resulting from failure of fusion of the lateral tubercle of the posterior talar process. It is present in 7-14% of the population and bilateral in 50% of those with the ossicle.
Anatomy
The posterior process of the talus has two tubercles: medial and lateral. The lateral tubercle may develop from a secondary ossification center that appears at 8-13 years and usually fuses within 1 year. When it fails to fuse, the os trigonum remains as a separate bone connected to the talus by a synchondrosis.
The flexor hallucis longus (FHL) tendon runs in a groove between the medial and lateral tubercles. This close relationship means FHL tenosynovitis commonly accompanies os trigonum syndrome.
Pathophysiology
Forced or repetitive plantarflexion (as in ballet dancing en pointe or downward kicking in soccer) compresses the os trigonum in a "nutcracker" fashion between the posterior tibia and calcaneus. This microtrauma leads to bone marrow edema, synchondrosis injury, and inflammation.
Clinical Presentation
History
Posterior ankle pain in a dancer or athlete requiring plantarflexion. Pain worse with en pointe position or downward kicking. May describe deep posterior pain. May have triggering or clicking of the great toe if FHL involved.
Examination
Posterior Impingement Test: Passive forced plantarflexion reproduces posterior ankle pain. Highly suggestive.
Palpation: Deep posterior tenderness between Achilles and peroneal tendons.
FHL Assessment: Passive flexion/extension of hallux with ankle in different positions may reveal tenosynovitis or triggering.
Differential
- Achilles tendinopathy (more superficial, insertion pain)
- Retrocalcaneal bursitis
- Peroneal tendinopathy
- Flexor hallucis longus tendinopathy (may coexist)
Diagnosis and Management

Imaging
Lateral Radiograph: Shows os trigonum as a separate ossicle posterior to the talus. May see enlarged Stieda process if the tubercle is fused but prominent.

MRI: Shows bone marrow edema in os trigonum and adjacent talus. Shows FHL tenosynovitis. Soft tissue inflammation.
CT: Useful for showing degenerative changes at the synchondrosis and surgical planning.

Nuclear Medicine (SPECT-CT): Can confirm symptomatic os trigonum with increased uptake localizing to the ossicle.

Diagnostic Injection: Local anesthetic to the posterior ankle. Greater than 70% pain relief confirms diagnosis.
Rest: Avoid plantarflexion activities.
Activity Modification: Modify dance or sport technique temporarily.
Injection: Corticosteroid to the posterior ankle may provide relief (weeks to months).
Physiotherapy: Address any muscular imbalances. Avoid excessive plantarflexion.
Conservative treatment may be sufficient for some patients but dancers/athletes often require surgery to return to full activity.
Evidence Base
- Endoscopic treatment technique described
- Two-portal posterior ankle approach
- Good outcomes in dancers/athletes
- Foundation for modern treatment
Exam Viva Scenarios
Practice these scenarios to excel in your viva examination
Scenario 1: Ballet Dancer with Posterior Pain
"A 20-year-old ballet dancer has posterior ankle pain when going en pointe. Lateral X-ray shows an os trigonum. How do you manage her?"
Scenario 2: Footballer with Posterior Ankle Pain - Differential Diagnosis and Determining if Os Trigonum is Symptomatic
"You are seeing a 24-year-old semi-professional footballer in your sports injury clinic who has been referred by the team physiotherapist for persistent posterior right ankle pain. He describes a 6-month history of progressive aching pain in the back of his right ankle, particularly after training sessions and matches. The pain is worse with running and kicking activities. He initially thought it was Achilles tendon pain and has tried eccentric loading exercises and a period of rest without significant improvement. He now reports the pain is deep in the back of the ankle rather than at the Achilles insertion. He is frustrated as it is affecting his performance and he has missed several matches. On examination, he has tenderness to deep palpation in the posterior ankle between the Achilles tendon and peroneal tendons. There is mild fullness in this area but no obvious swelling. His Achilles tendon appears normal with no nodularity or insertion tenderness. The retrocalcaneal bursa is non-tender. You perform a posterior impingement test (passive forced plantarflexion) which reproduces his deep posterior ankle pain. You also note he has some discomfort with resisted plantarflexion of the hallux (testing FHL function), but no triggering or clicking of the great toe. Active and passive ankle range of motion is full and pain-free except at extremes of plantarflexion. He has brought his plain radiographs from the sports medicine physician who initially saw him. The lateral ankle X-ray report states: 'Normal alignment. No fracture. Accessory os trigonum noted posterior to the talus. Achilles tendon insertion appears normal. No bony spurs.' The radiologist has noted in brackets: 'Os trigonum is a normal variant present in 7-14% of the population and is usually asymptomatic.' The patient is confused by this report and asks: (1) The radiologist says this bone is normal and usually doesn't cause problems - so is this my problem or is something else causing my pain? (2) How do we know if this os trigonum is the cause of my pain or just an incidental finding? (3) If it is the problem, what are my treatment options and how long will I be out of football? (4) Is there any risk of making it worse if I keep playing through the pain?"
Scenario 3: Elite Ballet Dancer with Combined Os Trigonum and FHL Pathology - Complex Surgical Planning and Bilateral Disease
"You are seeing a 22-year-old professional ballet dancer in your complex foot and ankle clinic. She is a principal dancer with a major ballet company and has been referred by the company physician for evaluation of bilateral posterior ankle pain that is significantly affecting her ability to perform. Her RIGHT ankle symptoms started 18 months ago with intermittent pain when en pointe (extreme plantarflexion on the tips of her toes). Over time, this has progressed to constant aching pain and she now describes two distinct components: (1) Deep posterior ankle pain, particularly when going en pointe or during relevé (rising onto the ball of the foot), and (2) Triggering and catching of her great toe, particularly when pushing off from en pointe position. She describes that sometimes her great toe 'gets stuck' in a flexed position and she has to manually extend it - this is extremely problematic during performances. She has also developed similar but less severe symptoms in her LEFT ankle over the past 6 months. She is understandably very anxious as her career is at stake - she is scheduled to perform the lead role in Swan Lake in 6 months and needs to be at full capacity. She has tried extensive conservative management including rest (took 3 months off from performing), custom orthotics, multiple physiotherapy programs, and two series of corticosteroid injections (one in each ankle) which provided only 2-3 weeks of relief each time. She has been seen by another orthopaedic surgeon who recommended surgery but didn't give her clear advice about what exactly needed to be done or the timeline for return to dance. On examination of the RIGHT ankle: There is deep posterior ankle tenderness between the Achilles and peroneal tendons. Forced passive plantarflexion reproduces her deep posterior pain (positive impingement test). When you ask her to actively flex and extend her great toe, you can clearly see and feel TRIGGERING of the FHL tendon with a palpable snap in the posterior ankle. Resisted plantarflexion of the hallux is painful. Passive forced extension of the hallux with the ankle in plantarflexion also reproduces posterior ankle pain (stretches the FHL). Her LEFT ankle examination is similar but less severe - positive impingement test, mild FHL triggering but not as pronounced. You review the MRI scans she brought: RIGHT ankle MRI report: 'Os trigonum present with extensive bone marrow edema (high T2 signal) throughout the ossicle and at the synchondrosis with the talus. Moderate joint effusion in posterior ankle. Flexor hallucis longus tendon is markedly thickened (diameter 8mm, normal less than 6mm) with high T2 signal within the tendon consistent with tendinopathy. There is fluid surrounding the FHL tendon sheath extending from the posterior ankle to the level of the sustentaculum tali (tenosynovitis). The FHL appears stenosed/constricted in the fibro-osseous tunnel between the medial and lateral tubercles of the posterior talus. Findings consistent with os trigonum syndrome with severe FHL tendinopathy and stenosing tenosynovitis.' LEFT ankle MRI report: 'Os trigonum present with mild bone marrow edema. Mild FHL tendinopathy and tenosynovitis, less severe than right side. No significant stenosis.' The patient has multiple questions: (1) The MRI mentions both the os trigonum and the FHL tendon problems - which one is causing my triggering, and do you need to treat both? (2) I have pain in both ankles - do I need surgery on both? If yes, can you do both together or do I need two separate operations? (3) What exactly does the surgery involve and how long will I be unable to dance? I need to know if I can make my Swan Lake performance in 6 months. (4) What are the risks to my FHL tendon? I've read online about dancers who had surgery and developed weakness in their great toe which affected their ability to dance en pointe. (5) Is there any chance this could end my dancing career? The previous surgeon didn't give her clear answers and she is extremely anxious."
OS TRIGONUM SYNDROME
High-Yield Exam Summary
Key Facts
- •Unfused lateral tubercle (7-14%)
- •Bilateral in 50%
- •Dancers and athletes affected
- •Nutcracker with plantarflexion
FHL Involvement
- •Runs adjacent to os trigonum
- •Tenosynovitis common
- •Triggering of great toe
- •Address during surgery
Diagnosis
- •Lateral X-ray: Ossicle posterior to talus
- •MRI: Bone marrow edema
- •Posterior impingement test
- •Injection confirms diagnosis
Treatment
- •Conservative: Rest, injection
- •Surgical: Endoscopic excision
- •Address FHL if involved
- •85-95% return to sport