Periprosthetic Hip Fracture

Periprosthetic hip fractures are fractures around a total hip arthroplasty. They are increasingly common as more THRs are performed and the population ages.

Risk Factors

Patient: Osteoporosis, female, advanced age, rheumatoid arthritis.

Implant: Revision surgery, cementless stems (higher risk than cemented), osteolysis.

Technical: Cortical perforation, stress risers.

Understanding the anatomy of the proximal femur and hip arthroplasty is essential for classifying and managing periprosthetic fractures.

Proximal Femoral Anatomy

Bone anatomy:

• Greater trochanter: Insertion of hip abductors (gluteus medius, gluteus minimus). Fractures here (Type AG) affect abduction strength.
• Lesser trochanter: Insertion of iliopsoas. Avulsion (Type AL) may indicate underlying osteolysis and stem loosening.
• Femoral shaft: Cortical bone tube surrounding the stem. Bone quality critical for fixation.
• Calcar: Medial cortex at femoral neck base, often resected or resorbed around stems.

Vascular supply:

• Femoral head blood supply disrupted by arthroplasty (not relevant to fracture healing)
• Femoral shaft: Nutrient artery plus periosteal blood supply
• Fracture healing relies on periosteal and endosteal blood supply
• Extensive soft tissue stripping during ORIF may compromise healing

Hip Arthroplasty Components

Femoral stem types:

• Cemented: Cement mantle transmits forces to bone. Fracture through cement or at cement-bone interface. Generally more stable initially.
• Uncemented: Press-fit, relies on bone ingrowth. May have extensive porous coating (fully coated) or proximal coating (proximally coated). Stress shielding can weaken proximal bone over time.

Stem geometry:

• Straight stems: Easier to bypass with long revision stems
• Curved/anatomic stems: May limit revision options
• Modular stems: Allow length adjustment but have junction points that may fail

Biomechanical Considerations

Forces on proximal femur:

• Hip joint reaction force: 3-5x body weight during walking
• Abductor muscle force: Stabilizes pelvis in single-leg stance
• Torsional forces: Rotational stress on femoral shaft
• Bending moments: Anterior bow of femur creates bending stress

Effect of stem on bone stress:

• Stress shielding: Stiff stem bypasses proximal bone, reducing load. Leads to bone resorption (Wolff's law). Common in fully coated stems.
• Stress concentration: Stem tip acts as stress riser. Type C fractures often occur 2-3 cortical diameters below stem tip.
• Cortical windows: Surgical defects (e.g., cerclage wire holes, screw holes from prior surgery) create weak points.

Fracture stability determinants:

• Stem fixation: Loose stem cannot support ORIF. Must revise if loose.
• Bone stock: Poor bone (osteoporosis, osteolysis, radiation) reduces healing potential and screw purchase.
• Fracture pattern: Transverse fractures more stable than comminuted or spiral patterns.

Periprosthetic fractures result from the complex interaction between bone quality, implant characteristics, and mechanical forces.

Mechanisms of Fracture

Intraoperative fractures:

• Occur during primary or revision arthroplasty
• Femoral preparation: Broaching, reaming, or rasping can split cortex
• Implant insertion: Press-fit stems generate hoop stresses that can crack cortex
• Uncemented stems: Higher intraoperative fracture risk (1-3%) vs cemented (0.1-1%)
• Risk factors: Osteoporotic bone, tight canal, oversized components, varus positioning

Postoperative fractures (more common):

• Early (less than 5 years post-THR): Usually trauma-related, often at stress risers
• Late (greater than 5 years post-THR): Combination of bone loss and trauma
• Spontaneous: Severe osteoporosis or osteolysis may result in atraumatic fracture

Bone Changes Around Implants

Stress shielding:

• Stiff femoral stem carries load, bypassing proximal femur
• Reduced mechanical stimulus leads to bone resorption (Wolff's law)
• Proximal bone density decreases up to 30% in first 2 years
• More pronounced with:
  • Fully porous-coated stems (stiffer)
  • Larger diameter stems
  • Younger, more active patients
• Clinical significance: Weakened bone at risk of fracture with minimal trauma

Osteolysis:

• Wear particles (polyethylene, metal, ceramic) trigger inflammatory response
• Macrophages release cytokines (TNF-α, IL-1, IL-6) activating osteoclasts
• Progressive bone resorption creates cavitary and cortical defects
• Gruen zones 1 and 7 (proximal medial and lateral) most commonly affected
• Compromises both fracture risk and fixation options

Cortical remodeling:

• Adaptive remodeling around stem alters bone architecture
• Proximal: Cortical thinning from stress shielding
• Distal (stem tip): Cortical thickening from stress concentration
• Hypertrophy: Calcar remodeling, endosteal scalloping
• Net effect: Altered bone quality makes fracture more likely

Biomechanical Factors

Stress concentration at stem tip:

• Stem tip acts as fulcrum during loading
• Bending moment greatest 2-3 cortical diameters below stem tip
• Type C fractures commonly occur at this location
• Longer, stiffer stems increase stress concentration

Torsional stress:

• Hip rotation generates torsional forces on femoral shaft
• Cemented stems: Cement-bone interface can fail in torsion
• Uncemented stems: Bone-implant interface may fail if not well-fixed
• Spiral fractures result from excessive torsion

Cyclic loading:

• Repeated loading cycles cause fatigue micro-damage
• Normally repaired by bone remodeling
• If damage accumulates faster than repair: stress fracture
• May present as impending fracture with progressive pain

Type A (Trochanters)

AG: Greater trochanter fracture. AL: Lesser trochanter fracture.

Usually conservative treatment unless significant displacement affecting abductor function (AG) or suggests stem loosening (AL avulsion from osteolysis).

Type B (Around Stem)

B1: Fracture around stem, stem STABLE, adequate bone stock.

• ORIF with cable plate (cerclage cables + plate).

B2: Fracture around stem, stem LOOSE, adequate bone stock.

• Revision to longer stem +/- ORIF.

B3: Fracture around stem, stem LOOSE, poor bone stock.

• Revision with augmentation (impaction grafting, allograft struts, tumor prosthesis if severe).

Type C (Below Stem)

Fracture distal to stem (not involving prosthesis directly).

Treat as standard femoral shaft fracture: Plate (with cables proximally if needed) or IM nail (if space allows above fracture for nail entry).

Thorough clinical assessment guides diagnosis, classification, and treatment planning.

History

Mechanism of injury:

• Low-energy trauma: Fall from standing height most common. Indicates pathological fracture through weakened bone.
• High-energy trauma: Rare but possible (MVA). May have associated injuries.
• Spontaneous: Severe osteoporosis or osteolysis may cause atraumatic fracture.

Pain characteristics:

• Acute onset: Suggests acute fracture
• Chronic/progressive pain: May indicate impending fracture or loosening
• Pain location: Groin (hip), thigh (shaft), knee (referred from hip)

Functional history:

• Pre-injury mobility: Walking aids, distance, independence
• Time since THR: Recent vs remote (affects loosening likelihood)
• Previous revisions: Higher risk of bone loss and complications
• Reason for THR: Osteoarthritis, fracture, inflammatory arthritis (affects bone quality)

Medical history:

• Osteoporosis: Fragility fractures, DEXA scan results, current treatment
• Medications: Bisphosphonates, steroids, anticoagulation
• Comorbidities: Cardiac, respiratory, renal disease (affects surgical risk)
• Smoking, alcohol: Impair bone healing

Physical Examination

Inspection:

• Deformity: Shortening, rotation (usually external rotation if displaced)
• Swelling: Thigh swelling common, ecchymosis may appear after several days
• Surgical scars: Previous hip incisions (lateral, posterior, anterolateral)
• Leg length: Measure from umbilicus to medial malleolus (compare to contralateral)

Palpation:

• Tenderness: Localized to fracture site (trochanteric, shaft, distal thigh)
• Crepitus: May be palpable with gentle manipulation
• Soft tissue: Check for tense compartments (rare but possible)

Range of motion:

• Hip ROM: Painful and limited. Do not force motion if fracture suspected.
• Knee ROM: Check to rule out ipsilateral knee injury

Neurovascular examination (CRITICAL):

• Femoral artery: Palpate femoral, popliteal, dorsalis pedis, posterior tibial pulses
• Femoral nerve: Test quadriceps strength (knee extension), sensation over anterior thigh
• Sciatic nerve: Test ankle dorsiflexion (deep peroneal), plantarflexion (tibial), sensation
• Document carefully: Baseline neurovascular status essential for medicolegal reasons

Systems Assessment

Medical optimization:

• Cardiorespiratory fitness: Exercise tolerance, functional capacity
• Anemia: Common in elderly, may need pre-op transfusion
• Anticoagulation: Warfarin, DOACs - plan reversal for surgery
• Nutrition: Albumin, protein-calorie malnutrition impairs healing

Frailty assessment:

• Cognitive function: Delirium risk, ability to comply with weight-bearing restrictions
• Social support: Home situation, carers, discharge planning needs
• Goals of care: Some patients may decline surgery if severe comorbidities

The entire algorithm hinges on the B1-versus-B2 decision, and the topic repeatedly says to "confirm stem stability intraoperatively" and "test the stem" - but never describes HOW, which is exactly what an examiner will ask. Radiographs are imperfect for this (Naqvi's B1/B2 kappa was only 0.68), so the definitive judgement is made in theatre.

• Expose enough to see and grip the implant. Through the fracture (or after opening the interval), clear soft tissue off the stem shoulder/trunnion so you can apply a controlled force directly to the implant.
• Apply torque and push-pull, and watch the interface. Apply a rotational (torque) and axial (push-pull) force to the exposed stem or trunnion (directly or via a trial head/impactor) and look for ANY motion at the bone-implant (or cement-bone) interface - visible micromotion, a rock, or blood/fluid "pumping" out of the interface all indicate a loose stem (B2/B3). A stem that is rock-solid with no motion under applied torque is stable (B1).
• Read the fixation type. For a cemented stem also inspect the cement mantle for fracture and a debonded cement-bone or cement-stem interface; for a cementless stem, distinguish a solidly ingrown implant from one that toggles.
• When in doubt, treat as loose. If you cannot test the stem properly, or there is any residual doubt, assume B2 and revise - the topic's own maxim is that it is "safer to revise a stable stem than ORIF a loose one," because ORIF of a loose stem has a near-universal failure rate. Always have revision implants available for any suspected B1/B2 so you can convert without abandoning the case.

Once a stem is confirmed loose (or a well-fixed stem must come out), the vivas invoke the extended trochanteric osteotomy (ETO) ("may need extended trochanteric osteotomy"; follow-up "What is extended trochanteric osteotomy?") - but it is never developed. It is the workhorse exposure for revising a B2/B3 and removing a well-fixed cemented or cementless stem. (Femoral bone-loss grading itself - the Paprosky system referenced in the vivas - is covered in the paprosky-classification topic.)

• What it is. A planned, controlled osteotomy that raises an anterolateral cortical fragment of the proximal femur, INCLUDING the greater trochanter, kept attached to the abductors and vastus lateralis as a hinged, vascularised muscle-osseous flap. It gives direct access to the bone-implant interface and the cement mantle while preserving the abductor mechanism.
• How it is fashioned. The fragment is typically about one-third of the cortical circumference (anterolateral), its length planned to just reach the level of the stem tip / cement while leaving enough intact distal diaphysis (commonly at least 4-6 cm) for the revision stem to gain a scratch-fit. Rounded osteotomy corners reduce the risk of propagation.
• Why use it. It allows safe removal of a well-fixed stem and ALL cement (curettes, osteotomes, cement splitters, ultrasonic tools) without blindly perforating the cortex, and it corrects varus remodelling/deformity to let a straight revision stem pass.
• How it is closed. The long cementless bypass stem (extensively porous-coated or tapered-fluted modular) gains distal fixation beyond the osteotomy; the trochanteric fragment is then reduced and secured with cerclage cables/wires around the stem. Union is reliable (roughly 90% or more).
• Pitfalls. Making the flap too short (poor access), migration/non-union of the fragment, and inadvertent propagation into a fracture - so plan the length and repair carefully.

Periprosthetic hip fractures and their treatment carry significant complication risks, both from the fracture itself and the surgical management required.

Medical Complications

Thromboembolic events: DVT/PE risk 2-5% despite prophylaxis. Elderly patients with prolonged immobility at highest risk. Mechanical and pharmacological prophylaxis essential.

Cardiopulmonary complications: MI, pneumonia, respiratory failure more common in elderly patients undergoing revision surgery. Medical optimization and early mobilization critical.

Mortality: 30-day mortality 2-5%, one-year mortality 10-20%. Higher in elderly, comorbid patients. Comparable to native hip fractures. Early surgery and medical optimization improve outcomes.

Successful outcomes require structured postoperative care addressing both the fracture and the underlying prosthesis.

Discharge Planning

Equipment needs: Elevated toilet seat, shower chair, reaching aids, walking frame or crutches.

Home modifications: Remove trip hazards, install grab rails, ensure bedroom/bathroom on same level if possible.

Support services: Consider home care package, meals on wheels, community physiotherapy. Involve occupational therapy.

Patient education: Hip precautions (avoid low chairs, crossing legs), weight-bearing restrictions, signs of complications, when to seek help.

Global Epidemiology

• Periprosthetic femoral fracture is now among the leading indications for revision THA worldwide, rising as the primary arthroplasty population ages.
• Swedish Hip Arthroplasty Register: postoperative incidence approximately 0.4% after primary THA and 2.1% after revision THA, increasing from roughly 1.0 to 1.4 per 1000 primary THRs over the study era.
• Cementless (uncemented) stems carry the higher INTRAoperative fracture risk (hoop stress on press-fit); cemented stems carry relatively more LATE fractures at the cement-tip stress riser.
• Most LATE periprosthetic fractures occur around an already-loose stem (Swedish register), which is why a high proportion of type B fractures prove to be B2/B3.

Side-by-Side Guidance

Registry Insights

• National registries (Swedish/SHAR, NJR England & Wales, AOANJRR Australia, AJRR US, Norwegian, NZJR) all record periprosthetic fracture as a discrete revision indication with rising frequency.
• Implant design influences risk: collarless polished tapered cemented and certain cementless designs differ in fracture pattern and timing; registries track these signals.
• Long cementless bypass stems dominate revision for loose stems; modular proximal femoral replacement is increasingly used for B3 with unreconstructable bone, especially in the frail elderly.

High- vs Limited-Resource Practice

• Well-resourced centres: pre-operative CT, full revision implant inventory (long modular stems, struts, megaprostheses), cell salvage, ortho-geriatric co-management, and tertiary referral pathways.
• Limited-resource settings: emphasis on robust plate-cerclage ORIF for stable stems and standard fixation for type C; revision implants and structural allograft may be scarce, so accurate B1 vs B2 triage and appropriate transfer are critical to avoid predictable ORIF failure on a loose stem.
• Universal priorities everywhere: early surgery, fracture-permitting weight-bearing, infection exclusion, and treatment of the underlying osteoporosis that drove the fragility fracture.

Periprosthetic hip fracture management has several genuinely unresolved questions - useful for the higher viva marks.

Periprosthetic hip fractures around total hip arthroplasty are classified using the Vancouver system based on fracture location, stem stability, and bone stock. The key clinical question is whether the stem is stable or loose - this determines treatment. Type B1 (stable stem, good bone) is treated with ORIF using cable plates. Type B2 (loose stem, good bone) requires revision to a longer stem. Type B3 (loose stem, poor bone) needs revision with bone augmentation (impaction grafting, megaprosthesis). Type A fractures (around trochanters) are usually treated conservatively, while Type C (below stem) is treated as a standard femoral shaft fracture.