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OrthoVellum

© 2026 OrthoVellum. For educational purposes only.

Not affiliated with the Royal Australasian College of Surgeons.

REVERSE HILL-SACHS LESIONS

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REVERSE HILL-SACHS LESIONS

Comprehensive guide to Reverse Hill-Sachs lesions, including classification, detailed management algorithm, and surgical options like the Modified McLaughlin procedure.

complete
Updated: 2026-01-02
High Yield Overview

REVERSE HILL-SACHS LESIONS

Posterior Instability | Impression Fracture | McLaughlin Lesion

86%Missed on initial X-ray
20-40%Critical defect size
SeizureCommon mechanism
less than 3 weeksAcute vs Chronic

Defect Size Classification

Small (under 20%)
PatternStable after reduction
TreatmentNon-operative / Remplissage
Intermediate (20-45%)
PatternUnstable
TreatmentMod. McLaughlin / Allograft
Large (over 45%)
PatternSignificant bone loss
TreatmentArthroplasty

Critical Must-Knows

  • Often associated with posterior shoulder dislocation (seizures, electrocution)
  • Lightbulb sign on AP X-ray due to internal rotation
  • Critical defect size is greater than 25% (some sources say 20%)
  • Modified McLaughlin transfers subscapularis/lesser tuberosity into defect
  • Chronic locked posterior dislocations are frequently missed

Examiner's Pearls

  • "
    Demonstrate the 'Posterior Drawer' and 'Jerk Test' in viva
  • "
    Axillary lateral view is mandatory to confirm diagnosis
  • "
    Distinguish between 'engaging' and 'non-engaging' lesions
  • "
    Review the 3 'E's of posterior dislocation: Epilepsy, Ethanol, Electricity

Critical Exam Points

The 'Missed' Diagnosis

Posterior dislocations are the most commonly missed major joint dislocation (greater than 50% missed initially). Always check axillary view.

Critical Size

20-25% articular surface. Defects larger than this are unstable and engage with the posterior glenoid rim.

Mechanism

Adduction + Internal Rotation. Seizures and electrocution cause massive muscle contraction (Lat/Pec/Subscap) driving head posterior.

Surgical Timing

less than 3 weeks = Acute. Over 3 weeks = Chronic. Affects reducibility and viability of the head.

Quick Decision Guide

ScenarioDefect SizeTreatmentKey Pearl
Acute (under 3wks), ReducibleSmall (under 20%)Closed Reduction + BraceImmobilize in ER (gunslinger)
Acute/Chronic, UnstableMedium (20-45%)Mod. McLaughlin / AllograftTransfer L.T. into defect
Chronic, Head collapseLarge (over 45%)Hemi / Total ShoulderHead is unsalvageable
Mnemonic

3 EsCauses of Posterior Dislocation

E
Epilepsy
Seizures cause violent internal rotation
E
Electricity
Electrocution shock causing contraction
E
Ethanol
Alcohol withdrawal seizures or trauma

Memory Hook:The 3 Es force the head out the Back!

Mnemonic

SMATreatment Algorithm by Size

S
Small (under 20%)
Stable - Non-op / Remplissage
M
Medium (20-45%)
Mod. McLaughlin or Allograft
A
All (over 45%)
Arthroplasty (Hemi/Total)

Memory Hook:Small, Medium, All-gone (Arthroplasty)

Mnemonic

LIRThe 3 Signs on X-ray

L
Lightbulb Sign
Round head on AP due to internal rotation
I
Impression Fracture
Trough line sign on AP
R
Rim Sign
Widened joint space greater than 6mm

Memory Hook:Look for LIR (Lighthouse in Rain) - Lightbulb, Impression, Rim!

Overview and Epidemiology

Terminology Clarification

Reverse Hill-Sachs Lesion: An impaction fracture of the anteromedial humeral head. Reverse Bankart Lesion: Injury to the posterior glenoid labrum. Both occur in posterior shoulder instability/dislocation.

Pathology

Impaction of the anterior humeral head against the posterior glenoid rim during dislocation.

Chronicity

Often present as "Locked Posterior Dislocation". The head is impacted and cannot be reduced closed.

Pathophysiology and Mechanisms

The Engaging Lesion

A Reverse Hill-Sachs lesion is considered "engaging" if the defect falls off the posterior glenoid rim when the arm is internally rotated and flexed (functional position). This causes recurrent instability.

Mechanism of Injury

  • Axial loading of the adducted and internally rotated arm.
  • Violent muscle contraction: The internal rotators (Latissimus Dorsi, Pectoralis Major, Subscapularis) are stronger than external rotators.
  • During a seizure, these muscles overpower the external rotators, forcing the humeral head posteriorly.

The Defect

  • Location: Anteromedial humeral head.
  • Comparison: A reflected image of a standard Hill-Sachs (which is posterolateral).
  • Engagement: As the arm internally rotates, the defect engages the posterior glenoid.

Associated Pathology

  • Reverse Bankart: Posterior labral tear.
  • Posterior Glenoid Rim Fracture: "Reverse Bony Bankart".
  • Lesser Tuberosity Fracture: Can be avulsed by subscapularis.
  • Posterior Capsular Stretch: Typically present in chronic cases.

Hill-Sachs vs Reverse Hill-Sachs

FeatureHill-SachsReverse Hill-Sachs
InstabilityAnteriorPosterior
Location on HeadPosterolateralAnteromedial
Glenoid LesionBankart (Anterior)Reverse Bankart (Posterior)
Position of EngagementAbduction + Ext RotationAdduction + Int Rotation

Classification Systems

Classification by Articular Surface Involvement

Most clinically useful for decision making.

Size% of HeadStabilityTreatment
Smallunder 20%StableClosed Reduction / Neglect
Medium20-45%UnstableReconstruction (McLaughlin)
Largeover 45-50%Grossly UnstableArthroplasty

Gamma Angle Classification

Used to predict instability.

  • Angle between the axis of the defect and the anatomical neck.
  • Greater than 90 degrees: Suggests higher risk of engaging.

Clinical Assessment

History

  • Mechanism: Seizure, shock, fall on flexed/adducted arm.
  • Symptoms: Pain, "locked" shoulder, inability to externally rotate.
  • Missed History: Patient treats it as "frozen shoulder" for months.

Observation

  • Posture: Arm held in adduction and internal rotation (".gunslinger" position).
  • Contour: Flattening of anterior shoulder, prominence of coracoid (posterior fullness).
  • ROM: Block to External Rotation (pathognomonic for locked posterior dislocation).

Specific Tests for Posterior Instability

  1. Posterior Drawer Test: Supine. Axial load + posterior force.
  2. Jerk Test: Seated. Flexion to 90 + Adduction + Axial Load. "Clunk" as head subluxes posteriorly.
  3. Kim Test: Variation of Jerk test for inferior-posterior, labral pathology.
  4. Load and Shift: Assess translation grading (Grade I-III).

ALWAYS compare to the contralateral side.

Neurovascular Exam

  • Axillary Nerve: Sensation over regimental badge area. Deltoid function.
  • Less commonly injured in posterior vs anterior dislocation, but verify.

Investigations

Imaging Protocol

X-rayTrauma Series
  • AP: "Lightbulb sign" (head internally rotated, looks symmetrical). Loss of half-moon overlap.
  • Axillary Lateral: GOLD STANDARD. Shows head posterior to glenoid.
  • Scapular Y: Head posterior to intersection of Y.
CTPre-operative Planning
  • Mandatory for operative planning.
  • Quantify defect size (% of articular surface).
  • Assess glenoid bone loss (Reverse Bony Bankart).
MRISoft Tissue
  • Assess posterior labrum (Reverse Bankart).
  • Assess cuff integrity (subscapularis).
  • Often done if diagnosis unclear or for chronic pain.

The Lightbulb Sign

On AP X-ray, the humeral head is fixed in internal rotation. The greater tuberosity rotates anteriorly, making the head look perfectly round (like a lightbulb) rather than its normal walking-stick appearance.

Management Algorithm

📊 Management Algorithm
Management algorithm for Reverse Hill-Sachs lesions
Click to expand
Treatment Strategy based on Defect Size and ChronicityCredit: OrthoVellum

Acute Management

Goal: Reduce and maintain stability.

  1. Closed Reduction: Under conscious sedation/GA.
    • Traction + gentle anterior pressure. Avoid force (fracture risk).
  2. Assess Stability:
    • Stable: Immobilize in External Rotation (gunslinger brace) for 4-6 weeks.
    • Unstable: If redislocates or engages in functional ROM to Surgery.

Early reduction minimizes cartilage damage.

Surgical Decision Making (Based on Size)

Defect SizeProcedureRationale
less than 20%Arthroscopic Remplissage / PlicationFill small defect with capsule
20-45%Modified McLaughlinTransfer Lesser Tuberosity into defect
20-45% (Alternative)Allograft ReconFemoral head / Humeral head allograft
over 45-50%Hemi / Total ArthroplastyHead geometry destroyed

Surgical Technique

The Concept

Transferring the Subscapularis tendon (original McLaughlin) or Lesser Tuberosity (modified) into the defect.

  • Effect: Fills the defects and acts as a check-rein preventing internal rotation.

Steps

  1. Approach: Deltopectoral approach.
  2. Identify Defect: Locate the anteromedial impression fracture.
  3. Osteotomy: Lesser tuberosity osteotomy (with subscapularis attached).
  4. Reduction: Reduce the humeral head into the glenoid.
  5. Preparation: Prepare the defect bed (curette/burr) to encourage healing.
  6. Fixation: Secure the lesser tuberosity bone block into the defect using screws or suture anchors.
  7. Result: The bony plug fills the hole and the subscapularis tightens, preventing internal rotation.

Careful protection of the axillary nerve is required.

Segmental Allograft

Used when the defect is large but the joint is salvageable (young patient).

  1. Approach: Deltopectoral.
  2. Graft: Fresh femoral head or humeral head allograft.
  3. Shaping: Graft shaped to match the defect ("snowman" or wedge shape).
  4. Fixation: Countersunk headless compression screws.
  5. Advantage: Restores spherical head shape effectively.

Allograft availability can be a limiting factor.

Hemi/Total Arthroplasty

Reserved for defects over 45-50% or older low-demand patients.

  • Hemiarthroplasty: If glenoid is intact.
  • Total Shoulder (Anatomic): If glenoid is arthritic but cuff intact.
  • Reverse Total Shoulder: If cuff is deficient or in very elderly.

Hemiarthroplasty is less commonly used now due to glenoid erosion risk.

Complications

ComplicationRisk FactorsPrevention/Management
Recurrent InstabilityUndersized graft, Missed engaging lesionEnsure defect is filled/bypassed
OsteoarthritisDamage to cartilage, hardware penetrationCountersink screws, accurate reduction
Subscapularis FailurePoor fixation of osteotomyProtect range of motion (ER) post-op
Hardware ProminenceScrew heads prominentUse headless screws

Postoperative Care

Rehab Protocol (Modified McLaughlin)

0-6 WeeksProtection
  • Sling: Gun-slinger brace (neutral to external rotation).
  • Restrictions: NO Internal Rotation (protects subscap transfer).
  • ROM: Passive ER allowed.
6-12 WeeksMotion
  • Wean sling.
  • Active assist ROM.
  • Begin gentle Internal Rotation stretching.
3-6 MonthsStrengthening
  • Rotator cuff strengthening.
  • Scapular stabilizers.
  • Return to sport/work assessment.

Outcomes and Prognosis

Modified McLaughlin

Good to Excellent results in 75-85% of patients. Low recurrence rate if sized correctly.

Allograft

Technical procedure but shows good survival at 10 years. Risk of graft resorption/necrosis.

Neglected Cases

Associated with poor functional outcomes and rapid progression to OA if left unreduced.

Arthroscopy

Arthroscopic "Reverse Remplissage" involves filling defect with subscapularis via suture anchors. Good for small/medium defects.

Evidence Base

Modified McLaughlin vs Allograft

(2010)
Key Findings:
  • Analyzed outcomes of Mod. McLaughlin vs Allograft for defects 25-50%
  • Both techniques yielded satisfactory clinical outcomes/stability
  • No significant difference in recurrence rates
  • McLaughlin is technically easier and uses autograft
Clinical Implication: McLaughlin procedure is the gold standard for medium defects, avoiding the morbidity of allografts while maintaining stability.
Source: Gavriilidis et al. (2010)

Critical Size Limit

(2004)
Key Findings:
  • Defects under 25% are usually stable post-reduction
  • Defects 25-50% require reconstruction
  • Defects over 50% require prosthetic replacement
  • Established the treatment algorithm widely used today
Clinical Implication: Defect size quantification via CT is the single most important factor in determining surgical strategy.
Source: Cicak (2004)

Initial Miss Rate

(Classic)
Key Findings:
  • Reported the classic 'Locked Posterior Dislocation' series
  • Average time to diagnosis was several months
  • Emphasized the Axillary Lateral view as critical for diagnosis
Clinical Implication: Never accept a diagnosis of 'frozen shoulder' in a trauma patient without a confirmed axillary lateral view to rule out posterior dislocation.
Source: Hawkins et al.

Arthroscopic Management

(2012)
Key Findings:
  • described posterior instability repair with engaging reverse Hill-Sachs
  • Arthroscopic plication into the defect
  • Suggests viable for defects up to 30% without open surgery
Clinical Implication: Arthroscopic management is increasingly viable for defects up to 30%, offering a minimally invasive alternative to open McLaughlin.
Source: Bhatia et al. (2012)

Long term Outcomes

(2012)
Key Findings:
  • Followed posterior dislocation patients
  • Recurrent instability rate is LOWER for posterior vs anterior dislocations
  • Main issue is often pain or unrecognized bone defects
Clinical Implication: Long-term prognosis is generally good if bone defects are recognized and treated; recurrence is less common than in anterior instability.
Source: Robinson et al.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: The Missed Diagnosis

EXAMINER

"A 45-year-old male presents with a 'frozen shoulder' for 3 months after a seizure. He cannot externally rotate past neutral. Show me the X-rays you would order and describe the findings."

EXCEPTIONAL ANSWER
I would order a full trauma series, specifically ensuring an **Axillary Lateral** view or a Velpeau view if the patient is in pain. On the AP, I am looking for the **Lightbulb Sign** (loss of the half-moon overlap) and the **Rim Sign** (widened joint space greater than 6mm). On the Axillary view, I expect to see the humeral head located posterior to the glenoid, potentially with an impaction fracture (Reverse Hill-Sachs) on the anteromedial head. Given the 3-month history, this is a **Chronic Locked Posterior Dislocation**.
KEY POINTS TO SCORE
Mandatory Axillary View
Lightbulb Sign description
Chronic Locked Dislocation diagnosis
Block to External Rotation is the key clinical sign
COMMON TRAPS
✗Accepting 'Frozen Shoulder' diagnosis without X-rays
✗Failing to order an Axillary view
✗Missing the diagnosis on AP X-ray
LIKELY FOLLOW-UPS
"How does the duration (3 months) affect your management?"
"What CT findings would determine if the head is salvageable?"
VIVA SCENARIOChallenging

Scenario 2: Surgical Decision Making

EXAMINER

"You confirm a locked posterior dislocation. A CT scan shows a Reverse Hill-Sachs lesion involving 35% of the articular surface. The head is viable. How do you manage this?"

EXCEPTIONAL ANSWER
This is a chronic locked dislocation with a **Medium-sized (35%)** defect. My management goals are reduction and stability. A defect of greater than 25% is unstable. I would perform an **Open Reduction** via a Deltopectoral approach. Since the defect is 35%, I would perform a **Modified McLaughlin Procedure**, where I transfer the Lesser Tuberosity (with subscapularis) into the defect. This fills the bone loss and acts as a check-rein against internal rotation. An alternative would be a segmental allograft, but McLaughlin uses local autograft.
KEY POINTS TO SCORE
Defect size 20-45% = Reconstruction
Modified McLaughlin rationale
Deltopectoral approach
Alternative: Allograft
COMMON TRAPS
✗Suggesting Remplissage alone for a 35% defect (too large)
✗Suggesting Arthroplasty (Head is viable and patient is young)
✗Forgetting to address the posterior labrum if torn
LIKELY FOLLOW-UPS
"Why not just reduce it and immobilize?"
"What acts as the 'Check-Rein' in the McLaughlin procedure?"
VIVA SCENARIOCritical

Scenario 3: The 'Engaging' Lesion

EXAMINER

"Explain the concept of an 'Engaging' Reverse Hill-Sachs lesion and how it differs from an engaging anterior Hill-Sachs."

EXCEPTIONAL ANSWER
An engaging Reverse Hill-Sachs lesion is an anteromedial defect that falls off the posterior glenoid rim when the arm is placed in a functional position of **Adduction and Internal Rotation**. This causes the head to sublux posteriorly. In contrast, a standard engaging Hill-Sachs (anterior instability) engages during **Abduction and External Rotation**. The concept is crucial because engaging lesions require bone-block procedures or Remplissage/McLaughlin to prevent engagement; soft tissue repair alone (Labral repair) will fail.
KEY POINTS TO SCORE
Position of Engagement: Add + IR
Functional implication: Recurrent instability
Treatment implication: Needs bony fill procedure
Comparison to Anterior (Abd + ER)
COMMON TRAPS
✗Confusing the positions of engagement
✗Thinking soft tissue repair is sufficient for engaging lesions
LIKELY FOLLOW-UPS
"How do you test for engagement intra-operatively?"
"What is the 'Track' concept in posterior instability?"

MCQ Practice Points

Nerve Injury

Q: What is the most common nerve injury associated with posterior shoulder dislocation? A: While the Axillary nerve is most common in anterior dislocations, checking it is still mandatory. However, posterior dislocations have a lower rate of nerve injury overall compared to anterior.

Pathognomonic Fracture

Q: Which fracture is pathognomonic for a posterior shoulder dislocation? A: A Lesser Tuberosity fracture (avulsion by the subscapularis tendon) is pathognomonic and indicates a posterior dislocation mechanism.

Radiographic Signs

Q: What is the 'Rim Sign' on an AP shoulder X-ray? A: The Rim Sign is a widening of the glenohumeral joint space greater than 6mm on the AP view, indicating posterior displacement of the head.

Cortical Lines

Q: What is the 'Trough Line Sign'? A: It appears as two parallel lines of dense cortical bone on the AP X-ray, representing the impaction fracture (Reverse Hill-Sachs) of the anteromedial humeral head.

Mechanism

Q: Which muscle group is responsible for posterior dislocation during a seizure? A: The massive internal rotators (Latissimus Dorsi, Pectoralis Major, Subscapularis) overpower the weaker external rotators, driving the head posteriorly.

Australian Context

Epidemiology

Common in AFL and Rugby (direct blow to anterior shoulder or fall on flexed arm). Also classic in 'surfing' wipeouts.

Guidelines

Early specialist referral recommended for all first-time posterior dislocations given high rate of missed diagnosis and bone defects.

REVERSE HILL-SACHS RECAP

High-Yield Exam Summary

Anatomy

  • •Impaction of Anteromedial Head
  • •Engages in Adduction + IR
  • •Posterior dislocation association
  • •Subscapularis tendon involvement

Classification (Size)

  • •Small (under 20%) = Stable
  • •Medium (20-45%) = Unstable/Recon
  • •Large (over 45%) = Arthroplasty
  • •Gamma angle greater than 90 deg = Unstable

Clinical Signs

  • •Locked in Internal Rotation
  • •Prominent Coracoid
  • •Flattened anterior shoulder
  • •Posterior fullness (humeral head)

Imaging

  • •Lightbulb Sign (AP)
  • •Trough Line Sign (Impaction)
  • •Axillary Lateral = Diagnostic
  • •Rim Sign: Widened joint space greater than 6mm

Treatment

  • •Acute less than 3wks + Small = Reduction + Brace
  • •Chronic/Med = Mod. McLaughlin
  • •Large/Collapse = Hemi/Total
  • •Remplissage for small engaging lesions

Complications

  • •Missed diagnosis (common)
  • •Recurrent instability
  • •AVN (late collapse)
  • •Arthritis
Quick Stats
Reading Time54 min
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