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Scapulothoracic Dissociation

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Scapulothoracic Dissociation

Comprehensive guide to scapulothoracic dissociation - closed forequarter amputation, neurovascular assessment, Damschen classification, limb-salvage vs amputation decision, and management priorities for orthopaedic exam

complete
Updated: 2024-12-16
High Yield Overview

SCAPULOTHORACIC DISSOCIATION

Closed Forequarter Amputation | High-Energy Trauma | Neurovascular Emergency

94%Brachial plexus injury rate
88%Subclavian/axillary vessel injury
21%Associated mortality
10%Complete flail limb

DAMSCHEN CLASSIFICATION

Type I
PatternMusculoskeletal only
TreatmentConservative possible
Type IIA
PatternVascular injury (viable)
TreatmentUrgent vascular repair
Type IIB
PatternVascular injury (ischemic)
TreatmentEmergency revascularization
Type III
PatternComplete avulsion (flail)
TreatmentConsider amputation

Critical Must-Knows

  • Scapulothoracic dissociation = traumatic disruption of the scapulothoracic articulation
  • Lateral scapular displacement greater than 1.0 on scapular index = diagnostic
  • Brachial plexus avulsion in 94% - often complete (C5-T1) with poor prognosis
  • Subclavian/axillary injury in 88% - may be occult, requires angiography
  • Type III (complete flail limb) often requires forequarter amputation

Examiner's Pearls

  • "
    Called 'closed forequarter amputation' - internal degloving of shoulder girdle
  • "
    High-energy mechanism: MVA, motorcycle, industrial traction injuries
  • "
    CXR shows lateral scapular displacement - measure scapular index
  • "
    Flail limb with absent pulses = emergent angiography + vascular surgery consult

Critical Scapulothoracic Dissociation Exam Points

Life-Threatening Injury

21% mortality from associated injuries. Massive soft tissue disruption with hemorrhage. Polytrauma protocol - ATLS management takes priority over limb. Life before limb.

Neurovascular Emergency

94% brachial plexus injury - often complete avulsion (C5-T1). 88% vascular injury - subclavian or axillary. Occult hemorrhage may cause hemodynamic instability.

Scapular Index

Ratio of distance from midline to medial scapular border: injured/uninjured. Ratio greater than 1.0 is diagnostic. Lateral displacement on CXR is the key finding.

Flail Limb Decision

Complete neurological deficit + vascular injury = flail limb (Type III). Limb salvage rarely successful. Forequarter amputation may be the most functional outcome.

Quick Decision Guide

PresentationClassificationManagementPrognosis
Swelling, bony injury onlyType I (musculoskeletal)Treat bony injuries, observeGood - preserve function
Vascular injury, limb viableType IIA (viable)Urgent vascular repairVariable - depends on neuro
Ischemic limb, neurologically intactType IIB (ischemic)Emergency revascularizationReasonable if neuro intact
Flail limb, no pulses, no functionType III (complete avulsion)Consider forequarter amputationPoor - non-functional limb
Mnemonic

SCAPSCAP - Scapulothoracic Dissociation Features

S
Scapular displacement
lateral on CXR, index greater than 1.0
C
Clavicle/AC joint disruption
associated fractures
A
Arterial injury
subclavian/axillary, occult hemorrhage
P
Plexus avulsion
brachial plexus C5-T1, usually complete

Memory Hook:SCAP for SCAPulothoracic - remember the 4 key components of this devastating injury

Mnemonic

FLAILFLAIL - Indications for Amputation

F
Flaccid paralysis
complete motor loss (0/5 throughout)
L
Loss of sensation
complete sensory deficit
A
Absent pulses
vascular disruption confirmed
I
Irreparable plexus
avulsion (not rupture) injury
L
Limb non-salvageable
despite revascularization attempts

Memory Hook:FLAIL limb = amputation consideration - all 5 features typically present in Type III

Mnemonic

1-2A-2B-3DAMSCHEN - Classification Types

1
Type I
Musculoskeletal injury only (bones, ligaments)
2A
Type IIA
Vascular injury, limb VIABLE (perfused)
2B
Type IIB
Vascular injury, limb ISCHEMIC (not perfused)
3
Type III
Complete avulsion (flail limb)

Memory Hook:Think 1-2-3: Type 2 splits into A (alive) and B (bad ischemia)

Mnemonic

INDEXINDEX - Scapular Index Calculation

I
Injured side
measure midline to medial scapula
N
Normal side
measure midline to medial scapula
D
Divide injured by
normal
E
Elevated ratio greater
than 1.0 = positive
X
X-ray
CXR or CT) for measurement

Memory Hook:INDEX greater than 1.0 = lateral displacement = scapulothoracic dissociation

Overview and Epidemiology

Life-Threatening Injury

Scapulothoracic dissociation is a true orthopaedic emergency with 21% mortality. It represents a spectrum from isolated musculoskeletal injury to complete "closed forequarter amputation." Polytrauma assessment and resuscitation take absolute priority.

Definition

  • Traumatic separation of the scapula from the thoracic wall
  • Complete disruption of scapulothoracic articulation
  • Involves AC joint, SC joint, or clavicle fracture
  • Associated soft tissue envelope disruption
  • Often called "closed forequarter amputation"

Epidemiology

  • Rare injury - reported incidence unknown (underdiagnosed)
  • High-energy mechanism required
  • Male predominance (trauma demographics)
  • 21% mortality from associated injuries
  • 10-15% of survivors have complete flail limb

Mechanism

High-energy lateral traction to the upper limb. Common mechanisms include:

  • Motorcycle accidents - arm caught, body continues
  • Motor vehicle accidents - ejection with arm traction
  • Industrial accidents - machinery entrapment
  • Agricultural accidents - PTO (power take-off) injuries

Pathophysiology and Anatomy

Key Anatomical Concept

The scapula is connected to the axial skeleton only by the clavicle (via AC and SC joints). All other attachments are muscular. Scapulothoracic dissociation requires disruption of these bony connections PLUS the extensive muscular envelope, neurovascular structures, and soft tissues.

Structures Disrupted in Scapulothoracic Dissociation

StructureNormal FunctionInjury Pattern
Clavicle/AC/SC jointBone bridge to axial skeletonFracture or dislocation
Trapezius, rhomboidsScapular retraction/elevationComplete rupture
Subclavian/axillary arteryUpper limb perfusionTear, avulsion, intimal injury
Subclavian/axillary veinVenous returnMay cause massive hemorrhage
Brachial plexus (C5-T1)Motor and sensory functionAvulsion (preganglionic) or rupture
Skin/subcutaneous tissueSoft tissue envelopeInternal degloving (Morel-Lavallée)

Brachial Plexus Injury Types

  • Avulsion (preganglionic): Root torn from cord - NOT repairable
  • Rupture (postganglionic): Nerve torn in continuity - potentially repairable
  • Complete plexus (C5-T1): Most common pattern in STD
  • Avulsion features: Horner syndrome, absent SNAP, positive myelogram

Vascular Injury Patterns

  • Complete transection: Obvious, requires repair
  • Intimal tear: May thrombose hours later - occult
  • Pseudoaneurysm: Delayed presentation
  • Always assume vascular injury until proven otherwise

Avulsion vs Rupture

Avulsion (preganglionic) = root torn FROM the spinal cord = NOT repairable = poor prognosis Rupture (postganglionic) = nerve torn BEYOND ganglion = potentially repairable = better prognosis

Clinical clues to AVULSION:

  • Horner syndrome (ptosis, miosis, anhidrosis) - T1 root
  • Rhomboid/serratus paralysis - dorsal scapular and long thoracic nerves
  • Preserved SNAP (Sensory Nerve Action Potential) despite anesthesia - dorsal root ganglion intact

Classification Systems

Damschen Classification (Most Commonly Used)

TypeDescriptionNeurovascular StatusManagement
Type IMusculoskeletal injury onlyIntactConservative/fixation
Type IIAVascular injury, limb viablePerfused, variable neuroUrgent vascular repair
Type IIBVascular injury, limb ischemicNon-perfused, variable neuroEmergency revascularization
Type IIIComplete neurovascular disruptionFlail limbAmputation consideration

Damschen Key Points

Type I is rare in true scapulothoracic dissociation (most have some neurovascular injury). The distinction between IIA and IIB is based on ischemia time - IIB requires emergent intervention. Type III represents complete "internal forequarter amputation."

Zelle Classification (Alternative System)

TypeNeurological StatusVascular Status
Type 1IntactIntact
Type 2AIncomplete plexus injuryIntact or injured
Type 2BComplete plexus injuryIntact or injured
Type 3Complete plexus avulsionMajor vessel disruption

Classification Application

For exam purposes, Damschen is the most commonly cited classification. The key decision points are:

  1. Is there vascular injury? (Type I vs II/III)
  2. Is the limb ischemic? (IIA vs IIB)
  3. Is there complete neurological loss? (Type III = flail limb)

Clinical Assessment

ATLS First

These patients are polytrauma until proven otherwise. Complete ATLS primary and secondary survey before focused limb assessment. Hemorrhagic shock from occult vascular injury is common.

History

  • Mechanism: High-energy lateral traction to arm
  • Time from injury (ischemia time critical)
  • Associated injuries (chest, spine, other limbs)
  • Hand dominance (for functional prognosis)
  • Occupation (manual labor vs sedentary)

Inspection

  • Massive swelling of shoulder girdle
  • Lateral displacement of entire shoulder
  • Skin changes (bruising, abrasions, degloving)
  • Limb position (may hang flaccid)
  • Open wounds (rare - usually closed injury)

Clinical Examination Sequence

Immediate1. Primary Survey (ATLS)

Airway, Breathing, Circulation, Disability, Exposure. Address life-threatening injuries. IV access, resuscitation as needed.

Priority2. Vascular Assessment

Pulses (radial, ulnar, brachial). Capillary refill. Doppler if pulses absent. Hand temperature and color. Document ischemia time.

Complete3. Neurological Examination

Brachial plexus assessment: C5 (shoulder abduction), C6 (wrist extension), C7 (elbow extension), C8 (finger flexion), T1 (finger abduction). Horner syndrome (T1 avulsion).

Secondary4. Musculoskeletal

Clavicle palpation (fracture). AC joint stability. SC joint assessment. Scapular position. Associated limb injuries.

Brachial Plexus Root Assessment

RootMotor FunctionSensoryReflex
C5Shoulder abduction (deltoid)Lateral armBiceps
C6Wrist extension (ECRL/ECRB)Lateral forearm, thumbBrachioradialis
C7Elbow extension (triceps)Middle fingerTriceps
C8Finger flexion (FDP)Medial forearm-
T1Finger abduction (interossei)Medial arm-

Horner Syndrome

Horner syndrome (ptosis, miosis, anhidrosis) indicates T1 root avulsion. This is a preganglionic injury and is NOT repairable. Its presence suggests complete plexus avulsion and poor prognosis for limb function.

Investigations

Essential Investigations

InvestigationKey FindingsUtility
CXRLateral scapular displacement, fractures, hemothoraxInitial screening - measure scapular index
CT AngiographyVascular injury, intimal flap, occlusionGold standard for vascular assessment
Conventional AngiographyDefinitive vascular imaging, allows interventionIf CTA equivocal or intervention planned
MRI/MR MyelographyNerve root avulsion (pseudomeningoceles)Delayed - for surgical planning
Nerve Conduction StudiesDistinguish avulsion vs ruptureDelayed (3 weeks) - preserved SNAP = avulsion

Scapular Index Calculation

Scapular Index = Injured side / Normal side

Measure from midline (spinous processes) to medial border of scapula on CXR or CT scout

  • Index greater than 1.0 = lateral displacement = positive
  • Original description used 1.29 cutoff
  • Any significant asymmetry is concerning

CT Angiography Findings

  • Vessel occlusion - complete cutoff
  • Intimal flap - linear filling defect
  • Pseudoaneurysm - contained rupture
  • Active extravasation - contrast blush
  • Vessel displacement - with hematoma

Nerve Conduction Studies

Performed at 3+ weeks post-injury. The key finding distinguishing avulsion from rupture is:

  • SNAP (Sensory Nerve Action Potential) preserved = AVULSION (dorsal root ganglion intact, sensory fibers still connected to ganglion but disconnected from cord)
  • SNAP absent = RUPTURE (entire nerve disrupted including sensory fibers)

Management

Management Priorities

Life before Limb. ATLS resuscitation takes absolute priority. Hemorrhagic shock is common from occult vascular injury. Once stable, address vascular emergency before definitive orthopaedic management.

📊 Management Algorithm
Management Algorithm for Scapulothoracic Dissociation
Click to expand
Management algorithm emphasizing the Life over Limb principle and the distinction between incomplete injury and complete flail limb.Credit: OrthoVellum

Acute Resuscitation

Emergency Management Algorithm

0-15 minATLS Primary Survey

Airway, Breathing, Circulation, Disability, Exposure. Large bore IV access. Blood products if shocked. Address life-threatening injuries (tension pneumothorax, hemothorax).

15-30 minVascular Assessment

Pulse check. If absent/diminished: emergent CTA or direct to angiography. Vascular surgery consult. Document ischemia time.

When stableNeurological Documentation

Complete brachial plexus examination. Document motor (0-5 scale) and sensory function for each root. Look for Horner syndrome.

When stableImaging

CXR (scapular index), CT chest/shoulder, CTA (vascular injury). MRI delayed for nerve root assessment.

Definitive Management by Type

Management by Damschen Type

TypeVascularNeurologicalOrthopaedic
Type IObservationMonitor/therapyFix fractures, sling
Type IIAUrgent repairAssess after vascularStaged fixation
Type IIBEmergency revascularizationSecondary priorityAfter revascularization
Type IIIMay not change outcomeComplete avulsionConsider amputation

Surgical Decision-Making

Limb Salvage Indications

  • Incomplete neurological injury
  • Viable limb after revascularization
  • Patient preference (informed consent)
  • Potential for some function
  • Good soft tissue envelope

Amputation Considerations

  • Complete flail limb (Type III)
  • Complete brachial plexus avulsion (C5-T1)
  • Non-salvageable vascular injury
  • Severe soft tissue loss
  • Patient preference (some prefer early amputation)

The Flail Limb Dilemma

A flail limb (complete motor/sensory loss, no vascular supply) will remain completely non-functional even after revascularization. Many patients ultimately choose forequarter amputation for:

  • Elimination of a painful, non-functional limb
  • Improved cosmesis with prosthesis
  • Better quality of life
  • Reduced infection/wound complication risk

Early amputation should be discussed but not mandated - patient autonomy is paramount.

Brachial Plexus Reconstruction

Timing

  • Surgery at 3-6 months post-injury
  • Allows nerve regeneration assessment
  • EMG/NCS at 3 weeks and 3 months
  • Earlier surgery (6 weeks) for clean lacerations

Options

  • Nerve graft - sural nerve for short gaps
  • Nerve transfer - expendable donor to denervated muscle
  • Free functioning muscle transfer - if no target muscles
  • Tendon transfers - late reconstruction

Surgical Technique

Multidisciplinary Surgery

Scapulothoracic dissociation requires coordinated multidisciplinary surgical management. Vascular surgery takes priority, with orthopaedic and reconstructive procedures staged appropriately.

Emergent Vascular Surgery:

Indications:

  • Subclavian artery disruption
  • Axillary artery injury
  • Active hemorrhage
  • Limb ischemia with salvageable limb

Approach:

  • Supraclavicular incision for proximal control
  • Infraclavicular/deltopectoral extension as needed
  • Median sternotomy for proximal subclavian access (rare)

Techniques:

  • Primary repair (rare - usually too damaged)
  • Interposition vein graft (saphenous vein)
  • Interposition prosthetic graft (PTFE/Dacron)
  • Temporary shunting if staged procedures needed

Ischemia Time

Warm ischemia time of greater than 6 hours results in near 100% amputation rate. Document time of injury and time of revascularization. Fasciotomies may be required after revascularization.

Brachial Plexus Surgery (3-6 months post-injury):

Indications:

  • Incomplete nerve injury with potential for recovery
  • Postganglionic injury (rupture, not avulsion)
  • Patient motivated for prolonged rehabilitation

Nerve Transfer Options:

  • Spinal accessory to suprascapular (shoulder abduction)
  • Intercostal to musculocutaneous (elbow flexion)
  • Contralateral C7 transfer (rarely used)

Nerve Grafting:

  • Sural nerve grafts for short gaps
  • Multiple cables for larger nerves

Nerve Reconstruction Priority

Priority is elbow flexion (most functional), then shoulder stability, then hand function. Complete C5-T1 avulsion is NOT reconstructable - consider amputation.

Associated Fracture Fixation:

Clavicle:

  • ORIF with plate if displaced
  • Usually delayed until vascular stability achieved
  • Superior plating preferred

Scapula:

  • Often managed non-operatively
  • Fixation if significant displacement affecting shoulder mechanics
  • Posterior approach if needed

Humerus:

  • IM nail or plate depending on location
  • May need staged procedures

AC/SC Joint:

  • Reconstruction if unstable
  • Often delayed/staged

All orthopaedic fixation is secondary to vascular repair and patient stabilization.

Complications

Complications of Scapulothoracic Dissociation

ComplicationIncidencePrevention/Management
Death21%ATLS resuscitation, hemorrhage control
Complete flail limb10-15%May require amputation
Chronic pain (neuropathic)CommonPain management, amputation may help
Compartment syndrome (arm)Risk with revascularizationProphylactic fasciotomy
Wound complicationsHighStaged procedures, VAC therapy
Heterotopic ossificationWith soft tissue traumaIndomethacin, radiation

Reperfusion Injury

After revascularization of an ischemic limb, consider:

  • Prophylactic forearm fasciotomy - especially if ischemia time greater than 6 hours
  • Monitor for compartment syndrome
  • Reperfusion can cause myoglobinuria and acute kidney injury
  • Hydration and urine alkalinization may be needed

Postoperative Care and Rehabilitation

Rehabilitation Phases

Day 0-7ICU/Acute Care

Hemodynamic monitoring. Wound care. Pain management. Vascular graft surveillance (duplex daily). Limb elevation. Compartment monitoring.

Week 1-6Early Rehabilitation

Gentle passive ROM if stable fixation. Edema control. Wound healing. Psychosocial support. Pain team involvement.

Month 2-6Active Rehabilitation

Active ROM as nerve function returns. Strengthening as tolerated. Occupational therapy for ADLs. Reassess for nerve reconstruction.

Month 6+Long-term

Ongoing therapy. Orthotics/prosthetics if needed. Vocational rehabilitation. Chronic pain management.

Rehabilitation After Limb Salvage:

  • Phase 1 (0-6 weeks): Protect vascular repair, passive ROM
  • Phase 2 (6-12 weeks): Active ROM, gentle strengthening
  • Phase 3 (3-6 months): Progressive strengthening, functional activities
  • Phase 4 (6+ months): Return to modified activities

Key Considerations:

  • Nerve recovery takes 12-18 months
  • Motor recovery before sensory
  • Therapy focus on available function

Nerve Recovery

Expect nerve regeneration at approximately 1mm/day (1 inch/month). Proximal injuries (brachial plexus level) take 12-18 months to reach distal targets. Recovery is never complete after severe injury.

Rehabilitation After Forequarter Amputation:

  • Early: Wound care, pain management, psychological support
  • 4-6 weeks: Prosthetic evaluation if desired
  • 6-12 weeks: Prosthetic training (if fitted)
  • Ongoing: Phantom limb pain management, psychosocial support

Prosthetic Options:

  • Cosmetic prosthesis (most common choice)
  • Functional prosthesis (limited utility at this level)
  • No prosthesis (many patients prefer this)

Patient autonomy in prosthetic decisions is paramount.

Pain Management

  • Multimodal analgesia
  • Neuropathic pain medications (gabapentin, pregabalin)
  • Pain team early involvement
  • Chronic pain clinic referral
  • Consider spinal cord stimulator

Psychosocial Support

  • Early psychological assessment
  • PTSD screening and treatment
  • Peer support programs
  • Vocational counseling
  • Family support services

Outcomes and Prognosis

Outcomes by Type

TypeFunctional OutcomeNotes
Type IGoodFull recovery expected if fractures heal
Type IIAVariableDepends on neurological recovery
Type IIBVariableDepends on ischemia time and neuro status
Type IIIPoor limb functionAmputation often provides better QOL

Factors for Poor Prognosis

  • Complete brachial plexus avulsion
  • Delayed revascularization (greater than 6 hours)
  • Associated severe injuries
  • Complete flail limb
  • Preganglionic injury (Horner syndrome)

Factors for Better Prognosis

  • Incomplete plexus injury
  • Early revascularization
  • Type I (musculoskeletal only)
  • Postganglionic injury (repairable)
  • Young patient

Evidence Base

Damschen Original Description

IV
Damschen et al. • Journal of Trauma (1997)
Key Findings:
  • Defined classification system. Type III (complete avulsion) has uniformly poor outcomes. Early amputation may be appropriate.
Clinical Implication: Provides framework for categorizing injury severity and guiding management decisions.

Zelle Classification Study

IV
Zelle et al. • Journal of Bone and Joint Surgery (2004)
Key Findings:
  • Alternative classification focusing on neurological status. Complete plexus injury (Type 2B/3) rarely recovers function.
Clinical Implication: Neurological status is the key determinant of functional outcome.

Limb Salvage vs Amputation Outcomes

IV
Ebraheim et al. • Injury (1988)
Key Findings:
  • Patients with complete plexus avulsion who underwent limb salvage had poor functional outcomes. Many later opted for amputation.
Clinical Implication: Early amputation discussion is appropriate for complete flail limb (Type III).

Vascular Injury Management

IV
Multiple Case Series • Various (1990-2010)
Key Findings:
  • Ischemia time greater than 6 hours associated with worse outcomes. Prophylactic fasciotomy recommended after delayed revascularization.
Clinical Implication: Emergent vascular repair is critical. Plan for compartment syndrome.

Brachial Plexus Reconstruction Timing

IV
Terzis and Kostopoulos • Microsurgery (2007)
Key Findings:
  • Optimal timing for plexus reconstruction is 3-6 months. Earlier surgery for sharp injuries. Nerve transfers expanding options.
Clinical Implication: Don't rush to plexus surgery - allow time for nerve recovery assessment.

Exam Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

EXAMINER

"A 28-year-old motorcyclist is brought to ED after high-speed collision. His left arm is massively swollen and hangs flaccid. There are no pulses in the left upper limb and complete motor/sensory loss. CXR shows lateral displacement of the left scapula. How do you proceed?"

EXCEPTIONAL ANSWER
**Initial Assessment:** I would approach this as a polytrauma patient following ATLS principles: **Primary Survey:** - Airway: Assess patency, C-spine control - Breathing: Inspect chest, consider hemothorax - Circulation: Two large-bore IVs, commence resuscitation if shocked - Disability: GCS, pupil assessment - Exposure: Full examination looking for other injuries **Focused Limb Assessment:** Once stable, I would assess the left upper limb: - **Vascular**: Absent pulses, cool limb, delayed capillary refill - confirms vascular injury - **Neurological**: Complete motor (0/5) and sensory loss - suggests complete plexus injury - **Look for Horner syndrome** (ptosis, miosis, anhidrosis) - would indicate T1 avulsion **Investigations:** - CXR: Lateral scapular displacement, calculate scapular index (injured/normal greater than 1.0) - Emergent CT Angiography: Identify vascular injury site - CT chest/shoulder: Associated injuries, fractures **Classification:** Based on ischemic limb + likely complete neurological loss = **Damschen Type IIB or III** **Management:** 1. Vascular surgery consult - emergent revascularization 2. Document ischemia time (critical for prognosis) 3. If revascularization successful and limb viable, plan for: - Prophylactic fasciotomy (especially if ischemia greater than 6 hours) - Staged bony fixation 4. If complete flail limb confirmed (Type III), early discussion of forequarter amputation may be appropriate **Follow-up Answer (Complete Plexus with Horner):** The presence of **Horner syndrome indicates T1 root avulsion** - a preganglionic injury that is NOT repairable. Combined with complete C5-T1 motor/sensory loss, this confirms **complete brachial plexus avulsion (Type III scapulothoracic dissociation)**. **Options:** 1. **Limb salvage with acceptance of flail limb**: Maintain vascular supply, staged wound care, eventual nerve reconstruction attempts - but function will be minimal at best 2. **Forequarter amputation**: Removes painful, non-functional limb. Allows prosthetic fitting. Often provides better quality of life. 3. **Nerve reconstruction**: Limited options with complete avulsion. Intercostal nerve transfers for elbow flexion possible but results modest. I would have a frank discussion with the patient about: - The poor prognosis for limb function - The possibility of chronic neuropathic pain - The option of early amputation vs limb salvage - This is ultimately a patient-centered decision
KEY POINTS TO SCORE
ATLS primary survey first - life before limb
This is likely Type IIB or III scapulothoracic dissociation
Emergent CTA or angiography for vascular assessment
Document neurological status (likely complete plexus injury)
Vascular surgery consult for revascularization
COMMON TRAPS
✗Missing associated injuries in polytrauma
✗Focusing on limb before life (ATLS)
✗Not calculating Scapular Index
VIVA SCENARIOStandard

EXAMINER

"Describe the scapular index and how you would calculate it on a chest X-ray. What is the diagnostic threshold?"

EXCEPTIONAL ANSWER
**Scapular Index Calculation:** The scapular index is a radiographic measurement used to identify lateral displacement of the scapula, which is diagnostic of scapulothoracic dissociation. **Method:** 1. On an AP chest X-ray (or CT scout), identify the **midline** (spinous processes) 2. Measure the distance from midline to the **medial border of each scapula** 3. Calculate the ratio: **Injured side distance / Uninjured side distance** **Interpretation:** - **Ratio greater than 1.0** = lateral displacement of the injured scapula = POSITIVE for scapulothoracic dissociation - The original description by Oreck used a cutoff of **1.29** - In practice, any significant asymmetry (greater than 1.0) warrants further investigation **Pitfalls:** - Patient rotation can cause false asymmetry - Pre-existing scapular pathology may affect measurement - May be obscured by massive swelling or associated injuries - Best measured on CT if CXR unclear **Other Radiographic Signs:** 1. **Clavicle fracture** - commonly associated (laterally displaced fragment) 2. **AC joint widening** - acromioclavicular separation 3. **SC joint dislocation** - sternoclavicular disruption (rare) 4. **First/second rib fractures** - suggest high-energy mechanism 5. **Scapular fractures** - particularly body or spine 6. **Hemothorax/pneumothorax** - from associated chest trauma 7. **Widened mediastinum** - suggests great vessel injury (aorta) On **CT**: - Lateral displacement more clearly visualized - Hematoma surrounding neurovascular structures - Muscular disruption (trapezius, rhomboids) - Soft tissue stranding indicating degloving
KEY POINTS TO SCORE
Ratio of injured side to uninjured side
Measure midline to medial scapular border
Ratio greater than 1.0 indicates lateral displacement
Use AP chest X-ray or CT scout
Any significant asymmetry is concerning
COMMON TRAPS
✗Inaccurate measurement due to patient rotation
✗Confusing with isolated scapula fractures
✗Missing contralateral comparison
VIVA SCENARIOAdvanced

EXAMINER

"A patient with Type III scapulothoracic dissociation asks about the option of keeping his arm versus amputation. How would you counsel him?"

EXCEPTIONAL ANSWER
**Counseling the Patient:** This is a complex shared decision-making scenario. I would approach it with empathy and clear, honest information. **Opening:** "I understand this is an incredibly difficult situation. Let me explain what we know about both options so you can make the decision that's right for you." **Explaining the Flail Limb:** "Your injury has damaged all the nerves that control your arm and hand. Even though we've restored blood flow, the nerves are not able to be repaired. This means: - You will have no voluntary movement in your arm or hand - You will have no sensation - Your arm will hang at your side and require support - You may experience significant nerve pain (neuropathic pain) - The arm will require ongoing care to prevent injury to insensate skin" **Option 1 - Limb Preservation:** - "We can keep your arm alive and attached" - "Some people prefer this for psychological or cultural reasons" - "The arm will be cosmetically present but non-functional" - "There is a small possibility of nerve transfers for limited function (elbow flexion)" - "Risks: chronic pain, skin breakdown, infection, potential later amputation" **Option 2 - Forequarter Amputation:** - "This involves removing the arm at the shoulder level" - "It eliminates the non-functional limb and often the associated pain" - "Many patients report improved quality of life after adjustment" - "Prosthetic options exist but have limitations" - "This is a major psychological adjustment" **Key Counseling Points:** 1. "There is no right or wrong answer - this is your decision" 2. "Many patients with similar injuries have chosen amputation and report they're glad they did" 3. "You don't have to decide immediately - but prolonged delay increases risks of the flail limb" 4. "I recommend you speak with a psychologist who specializes in limb loss" 5. "I can connect you with patients who have faced similar decisions" **Multidisciplinary Involvement:** - Psychology/psychiatry consultation - Occupational therapy assessment - Pain medicine consultation - Peer support (amputee mentorship) **Functional Limitations of Flail Limb:** - No grip or pinch - No elbow flexion/extension - No shoulder movement - Requires sling or support at all times - Insensate skin prone to injury - Dependent edema without muscle pump - Shoulder subluxation/pain from weight - Difficulties with dressing, hygiene, sleep - Social/psychological burden of visible disability - Neuropathic pain (often severe)
KEY POINTS TO SCORE
Complete flail limb has poor functional prognosis
Chronic neuropathic pain is common
Amputation may provide better quality of life
Patient autonomy is paramount
Multidisciplinary discussion recommended
COMMON TRAPS
✗Mandating amputation against patient wishes
✗Promising functional recovery of flail limb
✗Ignoring psychological impact and phantom pain

MCQ Practice Points

High-Yield MCQ Topics

TopicKey PointExam Trap
Scapular indexGreater than 1.0 = positiveDon't confuse with absolute measurement
ClassificationDamschen I/IIA/IIB/IIIKnow all 4 types and what distinguishes them
Horner syndromeT1 avulsion (preganglionic)Indicates poor prognosis - NOT repairable
Preserved SNAPIndicates avulsion NOT ruptureCounter-intuitive finding
First managementATLS resuscitationNOT immediate surgery for the limb

SNAP Question

Q: A patient with complete brachial plexus injury undergoes nerve conduction studies at 4 weeks. SNAP is preserved. What does this indicate?

A: Preganglionic avulsion injury (NOT repairable). When SNAP is preserved despite clinical sensory loss, it indicates the dorsal root ganglion is intact but disconnected from the spinal cord. The sensory nerve still conducts because the injury is proximal to the ganglion. This is counter-intuitive but a classic exam question.

Scapular Index Question

Q: What chest X-ray finding suggests scapulothoracic dissociation?

A: Scapular index greater than 1.0 (lateral scapular displacement). Calculate by comparing the distance from spine to medial scapular border on injured vs uninjured side. A ratio greater than 1.0 indicates abnormal lateral displacement of the injured scapula.

Horner Syndrome Question

Q: A polytrauma patient has ptosis, miosis, and anhidrosis on one side. Which brachial plexus root avulsion does this suggest?

A: T1 avulsion (preganglionic). Horner syndrome indicates disruption of sympathetic fibers traveling through T1. This is a poor prognostic sign indicating preganglionic injury (not surgically repairable). It suggests high-energy mechanism with severe nerve damage.

Management Priority Question

Q: A patient presents with suspected scapulothoracic dissociation after a motorcycle accident. What is the first management priority?

A: ATLS resuscitation - Life before Limb. These injuries have 21% mortality from associated vascular injury and hemorrhagic shock. After stabilization, address vascular emergency (subclavian/axillary artery) before definitive orthopaedic management.

Classification Question

Q: What distinguishes Damschen Type IIA from Type IIB scapulothoracic dissociation?

A: Vascular status. Both have neurological injury, but Type IIA has intact vascular supply while Type IIB has vascular injury requiring repair. Type I has musculoskeletal injury only, Type III has complete flail limb (complete vascular + neurological disruption).

Flail Limb Question

Q: A Type III scapulothoracic dissociation results in a 'flail limb'. What management options should be discussed with the patient?

A: Limb preservation vs forequarter amputation. A flail limb has no motor or sensory function. Many patients ultimately choose amputation because it: eliminates a painful non-functional limb, improves quality of life, reduces infection/wound risks. Patient autonomy is paramount - amputation should be discussed but never mandated.

Australian Context

Trauma System

  • Scapulothoracic dissociation requires Major Trauma Centre care
  • Retrieval services for transfer if needed
  • Multidisciplinary team: Ortho, Vascular, Plastics, ICU
  • National Trauma Registry data collection

Rehabilitation

  • State-based Lifetime Care schemes for catastrophic injury
  • NDIS for ongoing support needs
  • Prosthetic funding through these schemes
  • Access to specialized rehabilitation units

Transfer Decisions

Any suspected scapulothoracic dissociation should be transferred to a Major Trauma Centre with:

  • Level 1 trauma capability
  • Vascular surgery 24/7
  • Microsurgery/Plastics capability
  • ICU capacity
  • Brachial plexus surgery expertise (may be at specialized centre)

SCAPULOTHORACIC DISSOCIATION

High-Yield Exam Summary

Key Numbers

  • •Scapular Index greater than 1.0 = positive diagnosis
  • •94% have brachial plexus injury
  • •88% have vascular injury
  • •21% mortality rate
  • •6 hours ischemia threshold for poor outcomes

Diagnosis

  • •High-energy traction mechanism (motorcycle, MVA)
  • •Massive shoulder girdle swelling
  • •Lateral scapular displacement (scapular index)
  • •Absent/diminished distal pulses
  • •Brachial plexus deficit (C5-T1)

Damschen Classification

  • •Type I: Musculoskeletal injury only
  • •Type IIA: Neuro injury, vascular intact
  • •Type IIB: Neuro + vascular injury
  • •Type III: Complete flail limb (total disruption)

Management Priorities

  • •ATLS resuscitation (Life before Limb)
  • •CTA for vascular assessment
  • •Emergent revascularization if ischemic
  • •Document neuro status before intervention
  • •Type III: Discuss forequarter amputation

High-Yield Exam Points

  • •Preserved SNAP = preganglionic avulsion (NOT repairable)
  • •Horner syndrome = T1 avulsion (poor prognosis)
  • •Complete plexus avulsion = consider amputation
  • •Know scapular index calculation method
Quick Stats
Reading Time98 min
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