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Swan Neck Deformity

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Swan Neck Deformity

Comprehensive guide to Swan Neck Deformity, covering Nalebuff classification, intrinsic tightness testing, and management strategies.

complete
Updated: 2025-12-20
High Yield Overview

SWAN NECK DEFORMITY

PIPJ Hyperextension + DIPJ Flexion

PIPJHyperextension
DIPJFlexion
RACommon Cause
Oval-8Splint Type

Nalebuff Classification (RA)

Type I
PatternFlexible PIPJ in all positions.
Treatment
Type II
PatternFlexible PIPJ, but Intrinsic Tightness limited.
Treatment
Type III
PatternFixed PIPJ limitation (Stiff).
Treatment
Type IV
PatternJoint Destruction (Arthritis).
Treatment

Critical Must-Knows

  • Characterized by PIPJ Hyperextension and DIPJ Flexion.
  • Driven by dorsal subluxation of the lateral bands (opposite of Boutonniere).
  • Can be Primary (Volar plate laxity/Intrinsic tightness) or Secondary (Mallet finger).
  • Bunnell's Intrinsic Tightness Test discriminates Type I from Type II.
  • Conservative management involves blocking PIPJ hyperextension (Figure-of-8 splint).
  • Surgery ranges from soft tissue balancing (FDS tenodesis) to salvage (Fusion).

Examiner's Pearls

  • "
    If the PIPJ is stiff, tenodesis will NOT work. You must release the contracture first.
  • "
    In Mallet-induced Swan Neck, treating the DIPJ often corrects the PIPJ (if flexible).
  • "
    Intrinsic tightness is defined as less PIP flexion when the MCP is extended vs flexed.

Clinical Imaging

Imaging Gallery

swan-neck-deformity imaging 1
Click to expand
Clinical imaging for swan-neck-deformityCredit: Zarei-Ghanavati S et al., J Ophthalmic Vis Res via PMC3381109 (CC-BY)
swan-neck-deformity imaging 2
Click to expand
Clinical imaging for swan-neck-deformityCredit: Open-i / NIH via PMC5287134 (CC-BY)
swan-neck-deformity imaging 3
Click to expand
Clinical imaging for swan-neck-deformityCredit: Donato Alves T et al., Case Rep Rheumatol via PMC4150410 (CC-BY)

The Intrinsic Trap

The Problem

Intrinsic Tightness In Rheumatoid Arthritis, the intrinsic muscles often become fibrotic and tight. This pulls the lateral bands taut and hyper-extends the PIPJ. If you simply perform a capsulodesis or tenodesis without releasing the intrinsics, the deformity will recur or the finger will be stiff.

The Solution

Bunnell's Test You MUST assess intrinsic tightness. Passive PIP flexion is checked with MCP Extended (tightens intrinsics) and MCP Flexed (relaxes intrinsics). If Flexion with Extended MCP is LESS than Flexion with Flexed MCP, the test is POSITIVE.

Swan neck deformity in fifth finger
Click to expand
Classic swan neck deformity in the 5th finger: note the PIPJ hyperextension and DIPJ flexion. This isolated deformity in a rheumatoid arthritis patient demonstrates the characteristic posture caused by dorsal subluxation of the lateral bands.Credit: Zarei-Ghanavati S et al., J Ophthalmic Vis Res via PMC3381109 (CC-BY)
ConditionPIPJDIPJLateral BandsVolar Plate
Swan NeckHyperextendedFlexedDorsal SubluxedLax/Ruptured
BoutonniereFlexedHyperextendedVolar SubluxedNormal/Contracted
MalletNormalFlexedNormalNormal
JerseyNormalExtended (cannot flex)NormalNormal
Mnemonic

DOVEPathomechanics

D
Dorsal
Lateral bands slide dorsal.
O
Overpull
Extensor mechanism overpull.
V
Volar Plate
Volar plate laxity/failure.
E
Extension
PIPJ Hyperextension.

Memory Hook:The Swan flies like a DOVE.

Mnemonic

FLIDNalebuff Classification

F
Flexible
Type I (Mobile).
L
Limited
Type II (Intrinsic Tightness).
I
Immobile
Type III (Stiff).
D
Destroyed
Type IV (Arthritis).

Memory Hook:The lid (FLID) is on the deformity.

Mnemonic

SOFTSurgical Options

S
Splint
Oval-8 (Type I).
O
Oblique
Oblique Retinacular Lig reconstruction.
F
FDS
FDS Tenodesis (limit extension).
T
Tightness
Intrinsic Release (Type II).

Memory Hook:Keep the finger SOFT.

Overview

Definition

Swan Neck deformity consists of hyperextension of the PIPJ and reciprocal flexion of the DIPJ. It is caused by an imbalance where the extensor mechanism subluxates dorsally at the PIPJ.

It is the reverse of a Boutonniere deformity. It requires laxity of the Volar Plate to permit hyperextension. The lateral bands displace dorsal to the axis of rotation of the PIPJ. Once dorsal, they act as extensors of the PIPJ, locking it in hyperextension. This tension is transmitted distally, but because the lateral bands are effectively shortened by the hyperextension, the DIPJ is pulled into flexion by the FDP (Passive Insufficiency). Or, the FDP causes DIPJ flexion because the extensor mechanism has no power at the DIPJ? Actually, the DIPJ flexion is usually passive due to the FDP pull exceeding the extensor pull (which is all used up at the PIPJ).

Pathophysiology and Mechanisms

Extensor Apparatus

  • Lateral Bands: Normally lie dorsal to the axis of rotation of the PIPJ (extensors).
  • Transverse Retinacular Ligament: Prevents dorsal migration. In Swan Neck, this ligament stretches or fails.
  • Triangular Ligament: Becomes tight / shortened.

The interplay of these ligaments creates the balance.

Volar Constraints

  • Volar Plate: Primary restraint to hyperextension. Must be lax or ruptured for deformity to occur.
  • FDS Tendon: Rupture of FDS removes a dynamic stabilizer against hyperextension.

The Volar Plate is the key static restraint.

Pathogenesis

  1. Proximal Cause: Intrinsic tightness (pulls lateral bands dorsal) or Extensor Habitualis.
  2. Joint Cause: RA synovitis distends the joint, stretches the volar plate.
  3. Distal Cause: Mallet finger (loss of distal anchor) allows extensor force to concentrate at the PIPJ (Fowler's mechanism).

The deformity can be primary (PIP) or secondary (DIP).

Classification Systems

Reversible swan neck deformity demonstrating Nalebuff Type I
Click to expand
Four-panel view demonstrating reversible swan neck deformities (Nalebuff Type I): (Top left) lateral view showing PIPJ hyperextension and DIPJ flexion; (Top right) dorsal view showing deformity in multiple fingers; (Bottom left/right) patient making a fist demonstrates full flexion is preserved - hallmark of flexible/mobile Type I.Credit: Donato Alves T et al., Case Rep Rheumatol via PMC4150410 (CC-BY)

Nalebuff Classification (Rheumatoid)

  • Type I: PIPJ is flexible in all positions. No intrinsic tightness.
  • Type II: PIPJ is flexible, but intrinsic tightness is present (Bunnell +ve).
  • Type III: PIPJ flexion is limited (Stiff). X-ray often normal.
  • Type IV: PIPJ is stiff and X-ray shows joint destruction.

This classification guides treatment from splinting (I), release (II), manipulation (III), to fusion (IV).

Clinical Assessment

Preoperative view of swan neck deformity in index finger
Click to expand
Preoperative view demonstrating swan neck deformity of the index finger: PIPJ hyperextension with compensatory DIPJ flexion. This case demonstrates swan neck in cerebral palsy, where spasticity of the intrinsic muscles drives the deformity - an important etiology beyond rheumatoid arthritis.Credit: Open-i / NIH via PMC5287134 (CC-BY)

Examination

  • Deformity: Obvious PIP Hyperextension / DIP Flexion.
  • Passive Correctability: Can you reduce the PIPJ easily? (Type I/II vs III).
  • Impact: Does the finger initiate flexion? Locking?
  • Mallet: Check if the DIPJ can extend passively.

Always document the passive range of motion.

Bunnell's Intrinsic Tightness Test

  • Step 1: Extend MCP joint. Passively Flex PIPJ. Note angle (Intrinsic Tight).
  • Step 2: Flex MCP joint. Passively Flex PIPJ. Note angle (Intrinsic Relaxed).
  • Positive: PIP Flexion in Step 1 is less than Step 2.
  • Negative: PIP Flexion is equal (Joint stiffness) or better in Step 1 (Extrinsic tight).

Intrinsic tightness is a key feature of Type II.

Investigations

Radiology

  • Views: AP and Lateral.
  • Findings:
    • Joint Space: Look for narrowing/erosions (Type IV).
    • Alignment: Degree of hyperextension.
    • Fracture: Mallet fracture at DIPJ?

Crucial to rule out joint destruction before offering soft tissue reconstruction.

Management Algorithm

📊 Management Algorithm
Swan neck deformity management algorithm by Nalebuff type
Click to expand
Treatment algorithm: Type I (flexible) - splinting, FDS tenodesis. Type II (intrinsic tight) - intrinsic release. Type III (stiff) - serial casting, capsulotomy. Type IV (destroyed) - fusion or arthroplasty.Credit: OrthoVellum

Splinting (Type I)

  • Device: Figure-of-8 Splint / Oval-8 / Silver Ring Splint.
  • Mechanism: Blocks the last 10-20 degrees of extension (prevents hyperextension) but allows full flexion.
  • Mechanism: Blocks the last 10-20 degrees of extension (prevents hyperextension) but allows full flexion.
  • Outcome: Highly effective for mobile deformities. Patients often wear silver rings permanently as jewelry.

Compliance is very high with silver rings compared to plastic.

Surgical Management

  • Type I (Mobile): FDS Tenodesis (Swallow tail), Dermodesis (skin shortening), Lateral Band relocation.
  • Type II (Tight): Intrinsic Release (Littler). Release of ulnar lateral band.
  • Type III (Stiff): Closed manipulation, Capsulotomy. If fails then Fusion.
  • Type IV (Destroyed): Arthrodesis (Fusion) or Arthroplasty (Silicone).

Surgery must address the underlying cause (Intrinsic vs Joint).

Surgical Considerations

FDS Tenodesis (Swallow Tail)

  • Indication: Type I (Mobile).
  • Technique: One hemislip of the FDS tendon is divided proximally (in palm or P1).
  • Routing: It is passed through the flexor sheath and anchored into P2 bone or looped.
  • Effect: Creates a check-rein that prevents PIP hyperextension.
  • Rehab: Dorsal block splint to protect the tenodesis.

The tenodesis must be tensioned with the PIPJ flexed 20-30 degrees.

Intrinsic Release (Littler)

  • Indication: Type II.
  • Technique: Excision of the oblique fibers of the extensor hood (triangular area).
  • Effect: Preserves MCP flexion (transverse fibers) but removes the extending force on the PIPJ (oblique fibers).

Often combined with synovectomy in RA patients. The goal is to reduce the extending moment arm.

Complications

Surgical Complications

  • Recurrence: Common in RA due to progressive disease; occurs in up to 30% at 5 years.
  • Stiffness: Correction of hyperextension often results in loss of full flexion; trade-off between stability and motion.
  • Infection: Increased risk with silicone implants or immunosuppressive RA medications.
  • Tendon Rupture: FDS tenodesis can fail if tensioned too tightly or if manipulation is forceful.
  • Implant Failure: Silicone arthroplasty fractures over time (7-10 year lifespan).
  • Neuroma: Risk of digital nerve injury with lateral approaches.
  • Flexion Contracture: Over-correction can limit extension; reported in 15% of tenodesis cases.

Careful patient selection and setting realistic expectations are essential.

Complications of Conservative Management

  • Skin Irritation: Ring splints may cause pressure sores if poorly fitted.
  • Progression: Deformity may worsen despite splinting, especially in RA.
  • Functional Decline: Locking episodes interfere with daily activities.
  • Psychological Impact: Visible deformity affects body image and social function.
  • Non-Compliance: Splints may be removed for cosmetic or comfort reasons.

Regular follow-up ensures early detection of progression.

Rehabilitation

Week 0-3
  • Splint: Dorsal block splint (prevents extension past 20 deg).
  • Motion: Active flexion allowed.
Week 3-6
  • Wean: Start gentle active extension.
  • Check: Ensure no recurrence of hyperextension "snap".
Week 6-12
  • Strength: Grip strengthening using putty and Theraputty exercises.
  • Splint: Night splinting often continued for 3-6 months to prevent recurrence.
  • Dexterity: Fine motor tasks (picking up coins, buttons, writing).
  • Goals: Functional ROM (30-80 degrees) is better than Full ROM.
  • Work Simulation: Occupation-specific tasks to prepare for return to work.
Beyond 12 weeks
  • Long-term Splinting: Silver ring splints worn permanently in some cases.
  • Monitoring: Regular hand therapy reviews to detect recurrence early.
  • Disease Control: In RA, coordination with rheumatology for DMARD optimisation.

Prognosis

Expected Outcomes by Treatment

  • Splinting (Type I): Excellent functional outcomes; 80-90% symptom control with ring splints.
  • FDS Tenodesis: Good results in 70-80%; some loss of full flexion expected (10-20 degrees).
  • Intrinsic Release: Effective for Type II; may need combined with tenodesis.
  • Fusion (Type III/IV): Reliable pain relief; functional position (30-45 degrees) preferred.
  • Arthroplasty: Fair function for 7-10 years; silicone fracture is inevitable long-term.

Patient satisfaction depends on realistic expectations and appropriate procedure selection.

Prognosis by Cause

  • Traumatic: Best outcomes; young patients with isolated injury recover well with conservative care.
  • Rheumatoid: High recurrence rate (30% at 5 years); progressive disease limits long-term success.
  • Mallet-Induced: Treating the DIPJ pathology corrects the PIPJ if addressed early (within 3 months).
  • Cerebral Palsy/Spasticity: Poor outcomes; spasticity often overcomes surgical correction.

Underlying disease control is critical for sustained improvement.

Prognostic Factors

  • Duration: Early intervention (less than 6 months) yields better outcomes.
  • Joint Status: Preserved cartilage is essential for soft tissue procedures.
  • Disease Activity: Active RA synovitis predicts recurrence; optimise with DMARDs first.
  • Compliance: Splint adherence determines conservative management success.
  • Age: Younger patients have better tissue quality but higher functional demands.
  • Multiple Digits: Widespread involvement suggests systemic cause with guarded prognosis.

Serial photography helps document progression or improvement.

Evidence Base

Classification Utility

4
Nalebuff and Millender • Orthop Clin North Am (1975)
Key Findings:
  • Classic paper describing the 4 types
  • Tailored treatment algorithm based on mobility and X-ray
  • Emphasized the importance of intrinsic tightness
Clinical Implication: Use Nalebuff to guide surgical choice.

FDS Tenodesis Outcomes

4
Suematsu et al. • J Hand Surg (1995)
Key Findings:
  • Review of superficialis tenodesis for swan neck
  • Corrected widespread deformity in majority
  • Complication of flexion contracture in 15%
  • Effective for Type I/II
Clinical Implication: Effective but risk of stiffness.

Ring Splints

2
Zijlstra et al. • Rheumatology (2005)
Key Findings:
  • RCT comparing silver ring splints to thermoplastic
  • Silver rings had higher compliance and patient satisfaction
  • Both effectively corrected deformity and improved dexterity
Clinical Implication: Silver rings are the gold standard for conservative care.

Spiral Oblique Retinacular Ligament

5
Thompson and Litter • J Hand Surg (1978)
Key Findings:
  • SORL reconstruction uses a tendon graft to link DIP extension to PIP flexion
  • Complex procedure
  • Corrects both deformities theoretically but stretches out over time
Clinical Implication: Elegant concept, unreliable in practice (in RA).

Dermodesis

5
Various • Textbooks (2000)
Key Findings:
  • Removing an ellipse of skin from volar PIPJ
  • Shortens the volar skin to act as a check-rein
  • Simple, low risk, but skin stretches over time
  • Good for mild deformity
Clinical Implication: Simple fix for mild cases.

Viva Scenarios

Practice these scenarios to excel in your viva examination

VIVA SCENARIOStandard

Scenario 1: Rheumatoid Assessment

EXAMINER

"A 60-year-old with RA has Swan Neck deformities in fingers 3, 4, 5. She wants surgery because they 'lock'. What is your assessment?"

EXCEPTIONAL ANSWER
I would examine the mobility of the PIPJ. If they are flexible (Type I), I maintain them. I perform Bunnell's test to rule out intrinsic tightness (Type II). I check the X-rays for joint destruction (Type IV). If mobile and no tightness, I would first offer Ring Splints. If she fails those or demands surgery, FDS tenodesis is an option.
KEY POINTS TO SCORE
Classify the deformity (Nalebuff)
Bunnell Test
Conservative first (Splints)
COMMON TRAPS
✗Operating on a stiff joint as if it were mobile
✗Ignoring the rest of the RA hand
LIKELY FOLLOW-UPS
"She has intrinsic tightness. What now?"
"She is Type II. She requires Intrinsic Release (Littler) combined with the tenodesis or skin procedure."
VIVA SCENARIOStandard

Scenario 2: The Mallet Swan

EXAMINER

"A patient presents with a chronic Mallet finger and a secondary Swan Neck deformity. The PIPJ is flexible. How do you treat the Swan Neck?"

EXCEPTIONAL ANSWER
The Swan Neck here is driven by the proximal retraction of the extensor apparatus due to the loss of the distal anchor (Mallet). If the PIPJ is flexible, treating the Mallet finger (restoring the distal anchor) often corrects the Swan Neck. I would splint the DIPJ or perform a tenodermodesis/fusion of the DIPJ. I would NOT operate on the PIPJ initially.
KEY POINTS TO SCORE
Mechanism: Proximal retraction
Treat the primary cause (DIPJ)
Don't touch the PIPJ usually
COMMON TRAPS
✗Doing a complex PIPJ reconstruction for a DIPJ problem
LIKELY FOLLOW-UPS
"Why does this happen?"
"Increased pull on the central slip because the terminal tendon is disconnected."
VIVA SCENARIOStandard

Scenario 3: Post-Traumatic Stiff Swan Neck

EXAMINER

"A young carpenter had a hyperextension injury. Now has a fixed Swan Neck (Type III). X-ray is normal. He needs strength."

EXCEPTIONAL ANSWER
This is a Type III (Stiff). The volar plate is likely scarred or contracted dorsally, and lateral bands adherent. Soft tissue release is difficult. I would attempt manipulation or open volar adhesiolysis. However, if he needs strength and stability, and the joint is damaged/stiff, Arthrodesis (Fusion) of the PIPJ in functional flexion (40 degrees) is the most reliable option for a heavy labourer.
KEY POINTS TO SCORE
Stiffness management
Occupational demands (Carpenter = Fusion)
Unreliability of release
COMMON TRAPS
✗Promising full motion
✗Using an implant (Arthroplasty) in a labourer
LIKELY FOLLOW-UPS
"What position do you fuse?"
"Index 30, Middle 35, Ring 40, Little 45 degrees (Cascade)."
VIVA SCENARIOStandard

Scenario 4: Multiple Digits in RA

EXAMINER

"A 55-year-old woman with well-controlled rheumatoid arthritis presents with Swan Neck deformities in all fingers of her dominant hand. She is an artist and cannot hold a brush. Radiographs show preserved joint spaces. What is your approach?"

EXCEPTIONAL ANSWER
This is a challenging case of multiple Type I or II deformities. Given preserved joints and her occupation, I would start with comprehensive hand therapy and silver ring splints for all affected fingers. If intrinsic tightness is present (Bunnell positive), staged intrinsic releases may be considered. However, operating on multiple fingers simultaneously risks stiffness in the entire hand. I would counsel her that conservative management with splints offers the best function-to-risk ratio. If surgery is pursued, I would stage it - operating on 1-2 fingers at a time with intervals of 6-8 weeks.
KEY POINTS TO SCORE
Prioritise function over correction
Ring splints for multiple digits
Staged surgery if operative
Whole-hand perspective in RA
COMMON TRAPS
✗Operating on all fingers at once (catastrophic stiffness)
✗Promising full correction (unrealistic in RA)
✗Ignoring thumb and wrist pathology
LIKELY FOLLOW-UPS
"She wants surgery on all fingers. What do you say?"
"I would counsel strongly against simultaneous surgery. Staged procedures over 6-12 months allow assessment of outcomes and adjustment of technique. Operating on all digits risks losing the entire hand to stiffness."

MCQ Practice Points

Diagnosis

Q: What constitutes a positive Bunnell test? A: Decreased PIP flexion when the MCP joint is extended (vs flexed).

Anatomy

Q: Which ligament normally prevents dorsal subluxation of the lateral bands? A: Transverse Retinacular Ligament.

Classification

Q: In Nalebuff Type II, what is the defining feature? A: Intrinsic muscle tightness.

Treatment

Q: What is the primary function of a Figure-of-8 splint in Swan Neck? A: To block PIP hyperextension while allowing flexion.

Surgical Indication

Q: What is the surgical treatment for Nalebuff Type II Swan Neck? A: Intrinsic release (Littler procedure) to address the underlying intrinsic muscle tightness.

Secondary Swan Neck

Q: How does a Mallet finger cause Swan Neck deformity? A: Loss of the terminal tendon anchor causes proximal retraction of the extensor mechanism, concentrating extension force at the PIPJ.

Australian Context

Splinting and Therapy:

  • Silver Ring Splints (Oval-8, Murphy) are often imported from the USA or custom-made by specialized jewelers.
  • Australian Hand Therapy Association (AHTA) members provide specialized splinting services.
  • NDIS may fund splints and hand therapy for eligible patients with permanent disability from RA.
  • Medicare rebates available for hand therapy under Enhanced Primary Care (EPC) referrals.

Epidemiology and Access:

  • Rheumatoid arthritis affects approximately 1.9% of Australians, with higher prevalence in older females.
  • Indigenous Australians have higher rates of untreated trauma leading to chronic deformities (Mallet to Swan Neck progression).
  • Hand surgeons with specialized RA experience are concentrated in major metropolitan centres.
  • Public hospital waiting lists for elective hand surgery can be Category 3 (up to 365 days).

Telehealth Considerations:

  • Assessment of intrinsic tightness (Bunnell's test) is difficult via video consultation.
  • Pre-operative counselling can be effectively done remotely.
  • Post-operative follow-up may use hybrid models with local allied health support.

Occupational and Legal:

  • WorkCover claims arise from repetitive strain or crush injuries causing secondary deformity.
  • Return-to-work programs should involve hand therapist and occupational physician collaboration.
  • Functional capacity evaluations may be required for workers' compensation cases.

Multidisciplinary Care:

  • Rheumatology, Hand Surgery, and Hand Therapy must communicate regarding disease control.
  • Arthritis Australia provides patient support groups and educational materials.
  • Climate considerations: Heat can reduce splint compliance; breathable materials preferred.

Cost Considerations:

  • Silver ring splints cost $100-200+ each (not PBS subsidised).
  • Silicone arthroplasty implants have significant cost implications in public hospital budgets.
  • Early referral and conservative management reduce overall healthcare costs.

High-Yield Exam Summary

Classification (Nalebuff)

  • •Type I: Mobile
  • •Type II: Intrinsic Tight
  • •Type III: Stiff
  • •Type IV: Arthritis

Management

  • •Splint: Oval-8 / Ring
  • •Sx I: Tenodesis
  • •Sx II: Intrinsic Release
  • •Sx III/IV: Fusion

Mechanics

  • •Volar Plate Laxity
  • •Lateral Bands Dorsal
  • •Bunnell Test Positive
  • •Mallet drive
Quick Stats
Reading Time60 min
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