Intrinsic-Minus (Claw) vs Intrinsic-Plus - Two Opposite Deformities
- The intrinsic muscles of the hand (the INTEROSSEI and LUMBRICALS) act through the extensor expansion/lateral bands to FLEX the metacarpophalangeal (MCP) joints and EXTEND the interphalangeal (IP) joints; the lumbricals (origin FDP, insertion lateral band) are weak motors but spindle-rich and important for proprioception/precision pinch.
- INTRINSIC-MINUS (CLAW HAND): when the intrinsics are PARALYSED, MCP flexion and IP extension are lost, so the unopposed long extensors HYPEREXTEND the MCP joints and the unopposed long flexors FLEX the IP joints - the claw posture. The commonest causes are low ULNAR nerve palsy (claws the RING and LITTLE fingers), combined low ulnar+median palsy (claws all four fingers), and worldwide LEPROSY; also Charcot-Marie-Tooth and T1/lower brachial plexus lesions.
- The ULNAR PARADOX: a HIGH (proximal) ulnar nerve lesion produces LESS clawing than a LOW (distal) lesion, because a high lesion also paralyses the FDP to the ring and little fingers, so there is less IP flexion - paradoxically a worse (more proximal) lesion looks less clawed.
- The BOUVIER manoeuvre/test guides treatment of claw: if you passively BLOCK MCP HYPEREXTENSION (hold the MCP flexed) and the patient can then ACTIVELY EXTEND the IP joints, the extensor mechanism is intact (Bouvier-positive) and a STATIC MCP-blocking procedure (capsulodesis, or Zancolli LASSO) will correct the claw; if the IP joints still will not extend, a DYNAMIC transfer inserting into the extensor mechanism is needed.
- INTRINSIC-PLUS is the OPPOSITE deformity: intrinsic TIGHTNESS, CONTRACTURE or SPASTICITY makes the intrinsics overpull, FLEXING the MCP joints and EXTENDING the IP joints (often with thumb adduction). Causes include spasticity (stroke, cerebral palsy, head injury), ISCHAEMIC (Volkmann) contracture, trauma/scarring, rheumatoid disease, and prolonged immobilisation in the wrong position. The BUNNELL intrinsic tightness test detects it: PIP flexion is limited when the MCP is held EXTENDED (which tightens the intrinsics) but improves when the MCP is FLEXED.
- MANAGEMENT differs by deformity: for CLAW, prevent fixed contracture with anti-claw (lumbrical-bar) SPLINTING, correct any fixed PIP contracture, and for established claw use TENDON TRANSFERS (Zancolli lasso, FDS four-tail/Stiles-Bunnell, or Brand-type extensor transfers) to restore MCP flexion +/- IP extension; for INTRINSIC-PLUS, use stretching/splinting, treat spasticity (botulinum toxin), and perform a DISTAL INTRINSIC RELEASE for a fixed contracture.
- “Intrinsics FLEX MCP and EXTEND IP. Minus (paralysis) = claw (MCP hyperextended, IP flexed); Plus (tight/spastic) = MCP flexed, IP extended.
- “Ulnar paradox: HIGH ulnar lesion claws LESS than a LOW lesion (FDP to ring/little also out). Ulnar claw = ring + little fingers.
- “Bouvier test (block MCP hyperextension -> IP extend?) guides claw surgery; Bunnell test (PIP flexion worse with MCP extended) diagnoses intrinsic tightness.
Intrinsics paralysed -> MCP hyperextension + IP flexion. Think ulnar palsy/leprosy. Treat with splinting + tendon transfers.
Intrinsics tight/spastic -> MCP flexion + IP extension. Think spasticity/Volkmann. Treat with stretching, botulinum toxin, intrinsic release.
Intrinsic Muscle Function
The interossei (dorsal = abduct/DAB, palmar = adduct/PAD) and the lumbricals insert into the lateral bands/extensor expansion, giving them a unique action: they FLEX the MCP joints (they pass volar to the MCP axis) and EXTEND the IP joints (they pass dorsal to the IP axes). The lumbricals are unusual in arising from the FDP tendons and inserting on the radial lateral band, linking the flexor and extensor systems; they generate little force but are rich in muscle spindles and contribute to proprioception and precision pinch. Ulnar nerve supplies most intrinsics (all interossei, the ulnar two lumbricals, hypothenar and adductor pollicis); the median nerve supplies the radial two lumbricals and most thenar muscles. When the intrinsics fail, the MCP/IP balance is lost - and the direction of the resulting deformity depends on whether they are too WEAK (minus) or too TIGHT (plus).

Intrinsic-Minus (Claw Hand)
When the intrinsics are paralysed, the long extensors hyperextend the MCP joints and the long flexors flex the IP joints - the claw. A low ULNAR nerve palsy claws the ring and little fingers (the ulnar two lumbricals/interossei), while a combined low ulnar + median palsy claws all four fingers. The ULNAR PARADOX is high-yield: a HIGH ulnar lesion claws LESS than a LOW one, because a high lesion also paralyses the FDP to ring/little, reducing the IP flexion that produces the claw. Associated ulnar signs include Wartenberg's (abducted little finger), Froment's (FPL substitutes for a weak adductor pollicis on pinch) and Jeanne's signs. The commonest worldwide cause of intrinsic-minus claw is LEPROSY; other causes are Charcot-Marie-Tooth disease and T1/lower-trunk brachial plexus lesions.

The BOUVIER manoeuvre plans surgery: passively block MCP hyperextension (hold the MCP flexed) and ask the patient to extend the fingers. If the IP joints now extend (Bouvier-positive), the extensor mechanism is intact and a STATIC MCP-blocking procedure works - an MCP volar capsulodesis or the Zancolli LASSO (an FDS slip looped around the A1/A2 pulley to flex the MCP). If the IP joints still will not extend (Bouvier-negative), a DYNAMIC transfer that inserts into the lateral bands/extensor mechanism is required - e.g. the FDS four-tail (Stiles-Bunnell) or Brand transfers (ECRB/ECRL + tendon graft) to restore MCP flexion and IP extension. Always correct a fixed PIP flexion contracture first (splint/ release), and prevent contracture early with an anti-claw (lumbrical-bar) splint that holds the MCP flexed.
Intrinsic-Plus Hand
INTRINSIC-PLUS is the opposite of claw: the intrinsics are too tight (contracture) or spastic, so they overpull, FLEXING the MCP joints and EXTENDING the IP joints, often with the thumb adducted. Causes include spasticity (stroke, cerebral palsy, traumatic brain injury), ischaemic (Volkmann) contracture, trauma/scarring of the intrinsic compartments, rheumatoid disease, and prolonged immobilisation in the wrong position (which is exactly why the protective 'intrinsic-plus' position - MCP flexed, IP extended - is used to splint a hand, since it keeps the collateral ligaments at length). The BUNNELL intrinsic tightness test detects intrinsic tightness: hold the MCP in EXTENSION (which stretches the intrinsics) and try to flex the PIP - if PIP flexion is limited with the MCP extended but improves when the MCP is flexed (relaxing the intrinsics), the test is positive for intrinsic tightness. (If PIP flexion is instead worse with the wrist/MCP flexed, suspect extrinsic/extensor tightness.)
- Non-fixed/spastic: stretching and splinting, and for spasticity, botulinum toxin (or systemic/ neurosurgical spasticity management) to reduce intrinsic overpull.
- Fixed contracture: a DISTAL INTRINSIC RELEASE - excising the oblique fibres (the medial portion of the lateral bands/extensor expansion) distal to the MCP - relieves the deforming intrinsic pull while preserving MCP flexion; more proximal releases or muscle slide are options in severe ischaemic contracture.
- Treat the underlying cause (e.g. established Volkmann's) and rehabilitate.
Evidence & Key Studies
A biomechanical and evolutionary perspective on the function of the lumbrical muscle
- The lumbricals originate from the FDP tendons and insert onto the lateral band of the extensor mechanism, linking the flexor and extensor systems.
- They have the smallest physiological cross-sectional area in the upper limb (about one-tenth the motor force of the interossei) but are rich in muscle spindles, contributing to proprioception of the finger joints.
- The radial two lumbricals (median-innervated) appear functionally more important for precision pinch - relevant to the radial sparing seen in ulnar claw.
Surgical reconstruction of irreversible ulnar nerve paralysis in leprosy
- In 25 patients with irreversible ulnar claw, the FDS four-tail (Stiles-Bunnell) and Zancolli lasso procedures were compared.
- The FDS four-tail was more effective at CORRECTING the claw deformity, whereas the Zancolli lasso better restored GRIP STRENGTH.
- Pre-operative PIP extensor lag and longer paralysis time predicted worse outcomes, and swan-neck deformity could develop - reinforcing early treatment and contracture prevention.
According to PubMed, the lumbrical origin/insertion, weak-but-proprioceptive role and the functional importance of the radial two lumbricals come from the cited Wang study, and the comparison of FDS four-tail versus Zancolli lasso for ulnar claw (and the prognostic effect of PIP extensor lag/paralysis duration) from the cited Ozkan study. The intrinsic action (flex MCP/extend IP), the ulnar paradox and the Bouvier/Bunnell tests are standard, well-established hand teaching. (See also our Ulnar Nerve Palsy, Cubital Tunnel, Volkmann's Ischaemic Contracture and Tendon Transfers topics.)
Clinical Decision Scenarios
Practise clinical reasoning and management decisions out loud
“What is a claw hand, what causes it, and what is the ulnar paradox?”
“How do the Bouvier and Bunnell tests help you, and how does management differ between a claw hand and an intrinsic-plus contracture?”
Mnemonics & Memory Aids
MINUS = claw
Hook:Intrinsic-MINUS = claw: MCP up, IP down.
PLUS
Hook:Intrinsic-PLUS = MCP flexed, IP extended (mirror of claw).
Intrinsic function
- Interossei + lumbricals: FLEX MCP, EXTEND IP (via lateral bands)
- DAB (dorsal abduct) / PAD (palmar adduct); lumbricals: FDP->lateral band (proprioception)
- Ulnar = most intrinsics; median = radial 2 lumbricals + thenar
Intrinsic-minus (claw)
- MCP hyperextension + IP flexion (intrinsic paralysis)
- Ulnar palsy (ring/little), combined (all four), leprosy, CMT, T1/plexus
- Ulnar paradox: HIGH lesion claws LESS; signs: Wartenberg, Froment, Jeanne
Intrinsic-plus
- MCP flexion + IP extension (intrinsic tightness/spasticity)
- Spasticity (stroke/CP/TBI), Volkmann, trauma, wrong-position immobilisation
- Bunnell test: PIP flexion limited with MCP extended, improves with MCP flexed
Management
- Claw: anti-claw splint, correct PIP contracture; Bouvier+ -> capsulodesis/Zancolli lasso; Bouvier- -> FDS-4-tail/Brand
- Intrinsic-plus: stretch/splint, botulinum toxin (spastic), distal intrinsic release (fixed)
- Treat the underlying cause; prevent fixed contractures early